It is known that normal cell activity includes genetic repair. Both enzymes and proteins must be involved. What is the mechanism?
The Pleomorphic Cycle
I suggest a developmental cycle in vivo consisting of three macro stages:  a primitive stage comprising the repair proteins complexes;  an intermediate, or bacterial, stage including filterable forms such as the cell wall deficient forms described by Lida Mittman, PhD. [in Cell Wall Deficient Forms, Stealth pathogens]; and  a culmination stage consisting of yeast and fungal phases, and then mold, the and phase. The usual course of development would be from microzyma to repair proteins, and then to bacterium, etc. However, under certain conditions, such as, for example, it is highly likely that the microzymas can skip the primitive stage and become bacteria directly. Although these transformations are as astounding as that of a larva to a butterfly, what is equally impressive under observation is in the rapidity with which they can take place — in minutes, even seconds, sometimes. By the same token, when provoked by acidic conditions and the cycle proceeds to yeast, fungus and then mold, it may occur so rapidly that the bacterial stage, if that happens, has no time to be of any significance.
The aforementioned casual [alarming] situation, or modification of the median, is chronic tissue acidification [pH imbalance] and oxygen deprivation in the blood and tissues due to acidic forming foods, adverse lifestyle, emotional stress, and environmental stress. This is not an oversimplification! Acidification/hypoxia biochemically signals a dead host to the microzymas, while creating collapsed areas [dead zone’s] of the colloidal system in the intercellular fluid, and it is the primary physiological disease condition at which the symptoms commonly called specific diseases arise.
The term “virus” is the Latin word for poison, and gives us insight into the immediate cause of disease symptoms — poison is: exotoxins and mycotoxins, and a toxin, exotoxins, and toxins from environmental sources, [many of which are primary or secondary mycotoxins.]. Orthodox medicine is well aware that it is bacterial toxins more than the bacteria them self. [They feed in-house], that caused the symptoms referred to as infectious disease. Little if any emphasis is placed on this fine, but important distinction. Always, the germ is emphasized. There is little too, no awareness [or knowledge that], either, of the same role played by acidic toxins of the culminate microforms of the pleomorphic cycle. Their action and the body’s response to them are frequently ascribe to viruses, which do not produce toxins because they are the toxin or acid, but are said to wreck havoc by a number of other means. However, if they participate in symptom at Genesis in a host it is because they are stimulated to evolve into more complex, toxic genetic forms.
Protein inactivation is probably being done by fermentation or by acidic toxins from fermentation, while “poliovirus” is produced in the cell to reverse the damage.
Highly unlikely. The lysing is more likely caused by acidic mycotoxicosis, or by free radicals released in response to mycotoxic stress, or from other sources [I lysine radiation, for example]. Repair particles are residual after cell wall disruption.
The function of immune cells are damaged by bacterial or fungal waste products/acidic and/or overworked by toxic acidic overload, preventing proper cleanup and elimination of disharmonious, symptomgenic elements.
Metabolic derangement has occurred prior to the appearance of repair proteins, due to toxic overload in the cell. It is more likely that the proteins attempt to prevent cell transformation, and that cancerous development is cell conversion from primarily oxidative to wholly fermentation of metabolism, mediated by yeast, fungus and mold.
See answer to number 1 above.
a] Adsorption and penetration of the cell. The viral particle binds to the host cell membrane. This is unusually a specific interaction in which a viral encoded protein on the capsid or a glycoprotein embedded in the virion envelope binds to a host cell membrane receptor and is then internalized. This internalization occurs by endocytosis or by fusion of the virion envelope with the host cell membrane.
b) Uncoating of the virus, so that the nucleic acid can be released from the capsid into the nucleus or cytoplasm.
Repair work may require uncoating. An uncoated “virus” in the saddle plasma, may have, from the nucleus and not yet have a code, as in the case of hepatitis B , according to medical science.
Protein complex is produced in response to an alarming acidic situation — fermentation and mycotoxic stress — are capable of self-reported replication. As suggested by Bechamp, the microzyma is specific for each organ, therefore specific repair proteins will be needed for specific cells that make a specific organ that are being disturbed by dietary and/or metabolic acidic waste products. There is the question of why the great numbers in some cases. One possibility is simply over reaction; for example, fever can be extreme. Why? To remove dietary, metabolic acids or acids from bacteria, yeast, fungus and/or mold.
d) And finally, release of virions from the host cell either by budding or lysis.
Is it not living in a continual mistake to look upon diseases, as we do now, as separate entities, which must exist, like cats and dogs, instead of looking upon them as conditions, like a dirty and a clean condition, and just as much under our own control; or rather, as the reactions of kindly nature against the conditions in which we have placed ourselves?
Since then, I have seen it with my eyes and smelt it with my nose smallpox growing up in the first specimens, ear in close rooms or in overcrowded wards, where it could not by any possibility have been ‘caught’, but must have begun. Nay, more, I have seen diseases begin, grow up, and pass into one another . . . . I have seen, for instance, with a little overcrowding, continued fever grow up; and with a little more, typhoid fever; and when little more, typhus, and all in the same ward or hut.
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