The Most Important Article I Have Ever Written!
The blood “knows” that!
We’re better off not to interfere.
Now, back to the blood and Interstitium fluids.
On March 18th and 19th, of this year we announced a universal cure for cancer at the 14th Annual Global Summit on Oncology and Cancer!
This universal cure for cancer is based upon the research and findings of Dr. Robert O Young, published, January 9th, 2019, in the ACTA Scientific Cancer Biology, medical journal, Volume 3 Issue 2 , 2019.
Cystic Fibrosis and Pulmonary Adenocarcinoma Lung Cancer both Metabolic and Dietary Acidic Conditions of the Interstitial Fluids of the Interstitium!
A Universal Cure for Cancer!
On March 18th and 19th, of this year we announced a universal cure for cancer at the 14th Annual Global Summit on Oncology and Cancer!
This universal cure for cancer is based upon the research and findings of Dr. Robert O Young and Dr. Galina Migalko, published, January 9th, 2019, in the ACTA Scientific Cancer Biology, medical journal, Volume 3 Issue 2 , 2019.
Cystic Fibrosis and Pulmonary Adenocarcinoma Lung Cancer both Metabolic and Dietary Acidic Conditions of the Interstitial Fluids of the Interstitium
Cystic fibrosis (CF) [1,2] and Pulmonary Adenocarcinoma (PAC)  have similar symptomologies and are chronic, progressive, and frequently fatal acidic conditions of the respiratory system (lungs), lymphatic system (lymph nodes), intestines, pancreas, uri-nary tract system, reproductive organs and the skin as the alkaloid glands (the salivary glands, stomach, and small and large intestines) produce and secrete alkaline compounds, such as sodium bicarbonate to buffer and preserve the alkaline design of the body and the specific organs and glands affected. These metabolic and dietary acidic conditions resulting in the build-up of mucous  can affect any organ or organ system but primarily affects the respiratory, lymphatic system, digestive, and reproductive tracts in children and young adults with CF and the lungs and surrounding lymph nodes in PAC. I have suggested from my own clinical research that both of these conditions are the result of latent tissue acidosis (LTA) in the interstitial fluids of the Interstitium or the fluids that surround every cell, created from metabolism, diet, thoughts and environment and may be successfully treated and reversed with an alkaline lifestyle and diet (ALD) .\
Introduction and Historical Perspective:
According to the Cystic Fibrosis Foundation, about 30,000 Americans have CF. This condition occurs mostly in whites whose ancestors came from northern Europe, although it cuts across all races and ethnic groups. About 3,500 babies are born with this acidic condition each year in the United States. Moreover, about one in every 30 Americans suffer from CF [1,3].Nearly 40% of lung cancers in the US are adenocarcinoma, which usually originates in peripheral lung tissue . Most cases of adenocarcinoma are associated with smoking; however, among people who have smoked fewer than 100 cigarettes in their life-times (“never-smokers”) , adenocarcinoma is the most common form of lung cancer . Its incidence has been increasing in many developed Western nations in the past few decades, where it has become the most common major type of lung cancer in smokers (replacing squamous cell lung carcinoma) and in lifelong non-smokers . According to the Nurses’ Health Study, the risk of adenocarcinoma of the lung increases substantially after a long duration of previous tobacco smoking, with a previous smoking du-ration of 30 to 40 years giving a relative risk of approximately 2.4 compared to never-smokers, and a duration of more than 40 years giving a relative risk of approximately 5 . CF and PAC have similar symptomologies and are often accom-panied by the following signs and symptoms:o Thick, viscous mucus in the lungs caused by the glandular secretion of sodium bicarbonate in the chelation of ex-cess dietary and/or metabolic acids [3,9,10].Signs and Symptoms of CF and PACCitation:Robert Young. “Cystic Fibrosis and Pulmonary Adenocarcinoma Lung Cancer both Metabolic and Dietary Acidic Conditions”.
To read the entire article go to:
Citation: Acta Scientific Cancer Biology 3.2 (2019): 11-20.
The following pictures are PET Scans of Dawn Kali. These pictures show the before and after PET scans provided by her oncologist Dr. Smith in July and then again in October of 2013. You will see that Dawn Kali, with sure PET scan evidence that her cancer went into remission completely in October of 2013. There are three bright areas on the spine and one bright area on the hip showing active cancer and bone loss. In October of 2013 the PET scans were done again by Dr. Smith showing no cancer anywhere in Dawn’s body. She was in complete remission following the pH Miracle Protocol and taking Herceptin
In the above PET scan taken October, 2013 there is no evidence of cancer in Dawn’s lower extremities, including her hip bones.
The two PET scans above show total remission of the Dawn’s bone cancer, diagnosed by Dr. Smith. Dr. Smith declares Dawn cancer-free in October of 2013. Look at the date above showing October 2013 as the month Dawn was in complete remission from her metastatic HER2 positive State 4 breast cancer originally diagnosed in 2007. Dawn has outlived her prognosis for this type of cancer.
Why were these before and after PET scans showing the reversal of Dawn’s cancer in October not shown to the Jury?
Where is the fraud? Dawn was seeing her doctor every 3 months for check-ups and never paid The pH Miracle Center for ANY non-invasive scans or treatments.
Follow the money? Dawn was making over $150,000 a year by heading up the pH Miracle Coaching program.
Dawn Kali talks about her inflammatory ductal cell carcinoma breast cancer diagnosis openly with Dr. Robert O. Young in 2013. This is one of the many times when Dawn Kali is actually telling the truth. Dawn also declared herself cancer free in December, 2013 in a recorded pH Miracle training of 30 potential pH Miracle Coaches.
Listen to the following two audios with Carolyn and Dawn hosting a Conference call for a training they were doing for around 30 pH Miracle Coaches. The dates of these Conference calls were June 2012 and August 2013.
MARCH 21st at 6:00pm
1730 Shattuck Ave (@ Virginia)
MARCH 26th at 11:00am
Dr. Steven Finnell, Doctor of Chiropractic
193 Blue Ravine Road #245,
Watch for upcoming presentations in Healdsburg, San Francisco, Marin and other locations TBA soon!
Below, a note from Caroline Robitaille (Dawn Kali is now partnering with Caroline Robitaille who has led many people to better health with amazing results.)
“As many of you know, Charles and I started implementing a healthier way of life almost a year ago. We wanted to take charge of our own health and not leave it to a future of foreseeable medication and/or surgeries. Since then I’ve lost a lot of weight, no more aches and pains… including a back issue that showed up a few years ago. Never in my life have I felt this fantastic! And then there is Charles… he has completely reversed his diabetes; his numbers went from high 300’s to a low of 89. His arthritis is gone, no more aches and pains… including a back issue that showed up for him last year as well. We experienced these positive changes all within a few months. And we’re still moving towards optimum health! We have both had our “Live Blood Analysis” done with Dawn Kali, so that we could first identify our problem areas and then watch the progress. And just for the heck of it… we also had our blood work done with our medical doctors and everything has not only improved but we are in the healthy range in all areas. Oh and the really fun part is that recently Charles went to have his eyes examined because he was feeling like it was time for new glasses. It was true; he needed new glasses all right….BECAUSE FOR THE FIRST TIME EVER HIS EYE SIGHT HAD IMPROVED!”
Now, as of January 1st I have had the privilege of coaching over 40 people towards optimum health and it’s been an amazing experience for me and them! J They too have improved in additional areas such as; cholesterol, fibromyalgia, headaches have disappeared, increased energy, mental clarity, skin rashes have disappeared, whitened teeth, brighter eyes, shiny hair, reports of feeling like they have a new lease on life… just to name a few.
I know I sound like an infomercial but it’s only because I am so passionate about this. I’ve seen first hand how a healthy diet and lifestyle can significantly change everything in a relatively short amount of time and I want to share it with you! It’s easy, it’s fun and it’s life changing. No magic pills – just learning how out-of-control our diets, stress levels and lifestyles have become – with simple ways to make positive changes.
A few years ago, Dawn Kali, a young woman who was faced with Breast Cancer was told by her doctors that she needed aggressive chemotherapy, radiation and multiple surgeries. Instead her research led her to alternative ways and Dr. Robert Young. Following his protocol she was able to avoid conventional treatments and heal herself. Today, she is cancer free and has a beautiful little three month old baby boy. Dawn then trained under Dr. Robert Young as a Microscopist and is now performing ‘Live Blood Analysis’ that can show you how to detect problems at the preventative stages.
We have recently joined forces to spread the word and taking our message on the road! I would love to see you, family and friends at one of our talks if you think you have time to learn more about improving your health. J We have two upcoming presentations but there are more to follow in Marin, Healdsburg and San Francisco. J These presentations are free and informative. J Come listen to Dawn Kali and I tell our stories while we show you what simple changes you can make for a healthier you and you will see what healthy blood vs. unhealthy blood looks like!”
For more information contact me at Caroline.Robitaille@comcast.net or 925.683.1690
After sequencing his own genome, pioneer genomic researcher Craig Venter remarked at a leadership for the twenty-first century conference, “Human biology is actually far more complicated than we imagine. Everybody talks about the genes that they received from their mother and father, for this trait or the other. But in reality, those genes have very little impact on life outcomes. Our biology is way too complicated for that and deals with hundreds of thousands of independent factors. Genes are absolutely not our fate. They can give us useful information about the increased risk of a disease, but in most cases they will not determine the actual cause of the disease, or the actual incidence of somebody getting it. Most biology will come from the complex interaction of all the proteins and cells working with environmental factors, not driven directly by the genetic code” (http://indiatoday.digitaltoday.in/index.php?
This statement is very important because looking to the human genome for solutions to most chronic illnesses, including the diagnosis, prevention, and treatment of cancer, is overemphasized in today’s world. Observational studies, however, have indicated that as we migrate from one country to another, our chances of being diagnosed with most chronic illnesses are determined not by the country we come from but by the country we migrate to (1–4). In addition, studies with identical twins have suggested that genes are not the source of most chronic illnesses. For instance, the concordance between identical twins for breast cancer was found to be only 20% (5). Instead of our genes, our lifestyle and environment account for 90–95% of our most chronic illnesses.
Cancer continues to be a worldwide killer, despite the enormous amount of research and rapid developments seen during the past decade. According to recent statistics, cancer accounts for about 23% of the total deaths in the USA and is the second most common cause of death after heart disease (6). Death rates for heart disease, however, have been steeply decreasing in both older and younger populations in the USA from 1975 through 2002. In contrast, no appreciable differences in death rates for cancer have been observed in the United States (6).
By 2020, the world population is expected to have increased to 7.5 billion; of this number, approximately 15 million new cancer cases will be diagnosed, and 12 million cancer patients will die (7). These trends of cancer incidence and death rates again remind us of Dr. John Bailer’s May 1985 judgment of the US national cancer program as a “qualified failure,” a judgment made 14 years after President Nixon’s official declaration of the “War on Cancer.” Even after an additional quarter century of extensive research, researchers are still trying to determine whether cancer is preventable and are asking “If it is preventable, why are we losing the war on cancer?” In this review, we attempt to answer this question by analyzing the potential risk factors of cancer and explore our options for modulating these risk factors.
Cancer is caused by both internal factors (such as inherited mutations, hormones, and immune conditions) and environmental/acquired factors (such as tobacco, diet, radiation, and infectious organisms; Fig. 1). The link between diet and cancer is revealed by the large variation in rates of specific cancers in various countries and by the observed changes in the incidence of cancer in migrating. For example, Asians have been shown to have a 25 times lower incidence of prostate cancer and a ten times lower incidence of breast cancer than do residents of Western countries, and the rates for these cancers increase substantially after Asians migrate to the West (http://www.dietandcancerreportorg/?p=ER).
The role of genes and environment in the development of cancer. A The percentage contribution of genetic and environmental factors to cancer. The contribution of genetic factors and environmental factors towards cancer risk is 5–10% and 90–95% respectively. B Family risk ratios for selected cancers. The numbers represent familial risk ratios, defined as the risk to a given type of relative of an affected individual divided by the population prevalence. The data shown here is taken from a study conducted in Utah to determine the frequency of cancer in the first-degree relatives (parents + siblings + offspring). The familial risk ratios were assessed as the ratio of the observed number of cancer cases among the first degree relatives divided by the expected number derived from the control relatives, based on the years of birth (cohort) of the case relatives. In essence, this provides an age-adjusted risk ratio to first-degree relatives of cases compared with the general population.
C Percentage contribution of each environmental factor. The percentages represented here indicate the attributable-fraction of cancer deaths due to the specified environmental risk factor.
The importance of lifestyle factors in the development of cancer was also shown in studies of monozygotic twins (8). Only 5–10% of all cancers are due to an inherited gene defect. Various cancers that have been linked to genetic defects are shown in Fig. 2. Although all cancers are a result of multiple mutations (9, 10), these mutations are due to interaction with the environment (11, 12).
These observations indicate that most cancers are not of hereditary origin and that lifestyle factors, such as dietary habits, smoking, alcohol consumption, and infections, have a profound influence on their development (13). Although the hereditary factors cannot be modified, the lifestyle and environmental factors are potentially modifiable. The lesser hereditary influence of cancer and the modifiable nature of the environmental factors point to the preventability of cancer. The important lifestyle factors that affect the incidence and mortality of cancer include tobacco, alcohol, diet, obesity, infectious agents, environmental pollutants, and radiation.
Smoking was identified in 1964 as the primary cause of lung cancer in the US Surgeon General’s Advisory Commission Report (http://profiles.nlm.nih.gov/NN/Views/AlphaChron/date/10006/05/01/2008), and ever since, efforts have been ongoing to reduce tobacco use. Tobacco use increases the risk of developing at least 14 types of cancer (Fig. 3). In addition, it accounts for about 25–30% of all deaths from cancer and 87% of deaths from lung cancer. Compared with nonsmokers, male smokers are 23 times and female smokers 17 times more likely to develop lung cancer.
(http://www.cancer.org/docroot/STT/content/STT_1x_Cancer_Facts_and_Figures_2008.asp accessed on 05/01/2008).
The carcinogenic effects of active smoking are well documented; the U. S. Environmental Protection Agency, for example, in 1993 classified environmental tobacco smoke (from passive smoking) as a known (Group A) human lung carcinogen.
(http://cfpub2.epa.gov/ncea/cfm/recordisplay.cfm?deid=2835 accessed on 05/01/2008).
Tobacco contains at least 50 carcinogens. For example, one tobacco metabolite, benzopyrenediol epoxide, has a direct etiologic association with lung cancer (14). Among all developed countries considered in total, the prevalence of smoking has been slowly declining; however, in the developing countries where 85% of the world’s population resides, the prevalence of smoking is increasing. According to studies of recent trends in tobacco usage, developing countries will consume 71% of the world’s tobacco by 2010, with 80% increased usage projected for East Asia.
(http://www.fao.org/DOCREP/006/Y4956E/Y4956E00.HTM accessed on 01/11/08)
The use of accelerated tobacco-control programs, with an emphasis in areas where usage is increasing, will be the only way to reduce the rates of tobacco-related cancer mortality.
Percentages represent the cancer mortality attributable to alcohol and smoking in men and women as reported by Irigaray et al. (see 13).
How smoking contributes to cancer is not fully understood. We do know that smoking can alter a large number of cell-signaling pathways. Results from studies in our group have established a link between cigarette smoke and inflammation. Specifically, we showed that tobacco smoke can induce activation of NF-κB, an inflammatory marker (15,16). Thus, anti-inflammatory agents that can suppress NF-κB activation may have potential applications against cigarette smoke.
We also showed that curcumin, derived from the dietary spice turmeric, can block the NF-κB induced by cigarette smoke (15). In addition to curcumin, we discovered that several natural phytochemicals also inhibit the NF-κB induced by various carcinogens (17). Thus, the carcinogenic effects of tobacco appear to be reduced by these dietary agents. A more detailed discussion of dietary agents that can block inflammation and thereby provide chemopreventive effects is presented in the following section.
The first report of the association between alcohol and an increased risk of esophageal cancer was published in 1910 (18). Since then, a number of studies have revealed that chronic alcohol consumption is a risk factor for cancers of the upper aerodigestive tract, including cancers of the oral cavity, pharynx, hypopharynx, larynx, and esophagus (18–21), as well as for cancers of the liver, pancreas, mouth, and breast (Fig. 3). Williams and Horn (22), for example, reported an increased risk of breast cancer due to alcohol. In addition, a collaborative group who studied hormonal factors in breast cancer published their findings from a reanalysis of more than 80% of individual epidemiological studies that had been conducted worldwide on the association between alcohol and breast cancer risk in women. Their analysis showed a 7.1% increase in relative risk of breast cancer for each additional 10 g/day intake of alcohol (23). In another study, Longnecker et al., (24) showed that 4% of all newly diagnosed cases of breast cancer in the USA are due to alcohol use. In addition to it being a risk factor for breast cancer, heavy intake of alcohol (more than 50–70 g/day) is a well-established risk factor for liver (25) and colorectal (26,27) cancers.
There is also evidence of a synergistic effect between heavy alcohol ingestion and hepatitis C virus (HCV) or hepatitis B virus (HBV), which presumably increases the risk of hepatocellular carcinoma (HCC) by more actively promoting cirrhosis. For example, Donato et al. (28) reported that among alcohol drinkers, HCC risk increased linearly with a daily intake of more than 60 g. However, with the concomitant presence of HCV infection, the risk of HCC was two times greater than that observed with alcohol use alone (i.e., a positive synergistic effect). The relationship between alcohol and inflammation has also been well established, especially in terms of alcohol-induced inflammation of the liver.
How alcohol contributes to carcinogenesis is not fully understood but ethanol may play a role. Study findings suggest that ethanol is not a carcinogen but is a cocarcinogen (29). Specifically, when ethanol is metabolized, acetaldehyde and free radicals are generated; free radicals are believed to be predominantly responsible for alcohol-associated carcinogenesis through their binding to DNA and proteins, which destroys folate and results in secondary hyperproliferation. Other mechanisms by which alcohol stimulates carcinogenesis include the induction of cytochrome P-4502E1, which is associated with enhanced production of free radicals and enhanced activation of various procarcinogens present in alcoholic beverages; a change in metabolism and in the distribution of carcinogens, in association with tobacco smoke and diet; alterations in cell-cycle behavior such as cell-cycle duration leading to hyperproliferation; nutritional deficiencies, for example, of methyl, vitamin E, folate, pyridoxal phosphate, zinc, and selenium; and alterations of the immune system. Tissue injury, such as that occurring with cirrhosis of the liver, is a major prerequisite to HCC. In addition, alcohol can activate the NF-κB proinflammatory pathway (30), which can also contribute to tumorigenesis (31). Furthermore, it has been shown that benzopyrene, a cigarette smoke carcinogen, can penetrate the esophagus when combined with ethanol (32). Thus anti-inflammatory agents may be effective for the treatment of alcohol-induced toxicity.
In the upper aerodigestive tract, 25–68% of cancers are attributable to alcohol, and up to 80% of these tumors can be prevented by abstaining from alcohol and smoking (33). Globally, the attributable fraction of cancer deaths due to alcohol drinking is reported to be 3.5% (34). The number of deaths from cancers known to be related to alcohol consumption in the USA could be as low as 6% (as in Utah) or as high as 28% (as in Puerto Rico). These numbers vary from country to country, and in France have approached 20% in males (18).
In 1981, Doll and Peto (21) estimated that approximately 30–35% of cancer deaths in the USA were linked to diet (Fig. 4). The extent to which diet contributes to cancer deaths varies a great deal, according to the type of cancer (35). For example, diet is linked to cancer deaths in as many as 70% of colorectal cancer cases. How diet contributes to cancer is not fully understood. Most carcinogens that are ingested, such as nitrates, nitrosamines, pesticides, and dioxins, come from food or food additives or from cooking.
Heavy consumption of red meat is a risk factor for several cancers, especially for those of the gastrointestinal tract, but also for colorectal (36–38), prostate (39), bladder (40), breast (41), gastric (42), pancreatic, and oral (43) cancers. Although a study by Dosil-Diaz et al., (44) showed that meat consumption reduced the risk of lung cancer, such consumption is commonly regarded as a risk for cancer for the following reasons. The heterocyclic amines produced during the cooking of meat are carcinogens. Charcoal cooking and/or smoke curing of meat produces harmful carbon compounds such as pyrolysates and amino acids, which have a strong cancerous effect. For instance, PhIP (2-amino-1-methyl-6-phenyl-imidazo[4,5-b]pyridine) is the most abundant mutagen by mass in cooked beef and is responsible for ~20% of the total mutagenicity found in fried beef. Daily intake of PhIP among Americans is estimated to be 280–460 ng/day per person (45).
Nitrites and nitrates are used in meat because they bind to myoglobin, inhibiting botulinum exotoxin production; however, they are powerful carcinogens (46). Long-term exposure to food additives such as nitrite preservatives and azo dyes has been associated with the induction of carcinogenesis (47). Furthermore, bisphenol from plastic food containers can migrate into food and may increase the risk of breast (48) and prostate (49) cancers. Ingestion of arsenic may increase the risk of bladder, kidney, liver, and lung cancers (50). Saturated fatty acids, trans fatty acids, and refined sugars and flour present in most foods have also been associated with various cancers. Several food carcinogens have been shown to activate inflammatory pathways.
According to an American Cancer Society study (51), obesity has been associated with increased mortality from cancers of the colon, breast (in postmenopausal women), endometrium, kidneys (renal cell), esophagus (adenocarcinoma), gastric cardia, pancreas, prostate, gallbladder, and liver (Fig. 5). Findings from this study suggest that of all deaths from cancer in the United States, 14% in men and 20% in women are attributable to excess weight or obesity. Increased modernization and a Westernized diet and lifestyle have been associated with an increased prevalence of overweight people in many developing countries (52).
Various cancers that have been linked to obesity. In the USA overweight and obesity could account for 14% of all deaths from cancer in men and 20% of those in women (see 51).
Studies have shown that the common denominators between obesity and cancer include neurochemicals; hormones such as insulinlike growth factor 1 (IGF-1), insulin, leptin; sex steroids; adiposity; insulin resistance; and inflammation (53).
Involvement of signaling pathways such as the IGF/insulin/Akt signaling pathway, the leptin/JAK/STAT pathway, and other inflammatory cascades have also been linked with both obesity and cancer (53). For instance, hyperglycemia, has been shown to activate NF-κB (54), which could link obesity with cancer. Also known to activate NF-κB are several cytokines produced by adipocytes, such as leptin, tumor necrosis factor (TNF), and interleukin-1 (IL-1) (55). Energy balance and carcinogenesis has been closely linked (53). However, whether inhibitors of these signaling cascades can reduce obesity-related cancer risk remains unanswered. Because of the involvement of multiple signaling pathways, a potential multi-targeting agent will likely be needed to reduce obesity-related cancer risk.
Worldwide, an estimated 17.8% of neoplasms are associated with infections; this percentage ranges from less than 10% in high-income countries to 25% in African countries (56, 57). Viruses account for most infection-caused cancers (Fig. 6). Human papillomavirus, Epstein Barr virus, Kaposi’s sarcoma-associated herpes virus, human T-lymphotropic virus 1, HIV, HBV, and HCV are associated with risks for cervical cancer, anogenital cancer, skin cancer, nasopharyngeal cancer, Burkitt’s lymphoma, Hodgkin’s lymphoma, Kaposi’s sarcoma, adult T-cell leukemia, B-cell lymphoma, and liver cancer.
Various cancers that have been linked to infection. The estimated total of infection attributable cancer in the year 2002 is 17.8% of the global cancer burden. The infectious agents associated with each type of cancer is shown in the bracket. HPV Human papilloma virus, HTLV human T-cell leukemia virus, HIV human immunodeficiency virus, EBV Epstein–Barr virus (see 57).
In Western developed countries, human papillomavirus and HBV are the most frequently encountered oncogenic DNA viruses. Human papillomavirus is directly mutagenic by inducing the viral genes E6 and E7 (58), whereas HBV is believed to be indirectly mutagenic by generating reactive oxygen species through chronic inflammation (59–61). Human T-lymphotropic virus is directly mutagenic, whereas HCV (like HBV) is believed to produce oxidative stress in infected cells and thus to act indirectly through chronic inflammation (62, 63). However, other microorganisms, including selected parasites such as Opisthorchis viverrini or Schistosoma haematobium and bacteria such as Helicobacter pylori, may also be involved, acting as cofactors and/or carcinogens (64).
The mechanisms by which infectious agents promote cancer are becoming increasingly evident. Infection-related inflammation is the major risk factor for cancer, and almost all viruses linked to cancer have been shown to activate the inflammatory marker, NF-κB (65). Similarly, components of Helicobacter pylorihave been shown to activate NF-κB (66). Thus, agents that can block chronic inflammation should be effective in treating these conditions.
Environmental pollution has been linked to various cancers (Fig. 7). It includes outdoor air pollution by carbon particles associated with polycyclic aromatic hydrocarbons (PAHs); indoor air pollution by environmental tobacco smoke, formaldehyde, and volatile organic compounds such as benzene and 1,3-butadiene (which may particularly affect children); food pollution by food additives and by carcinogenic contaminants such as nitrates, pesticides, dioxins, and other organochlorines; carcinogenic metals and metalloids; pharmaceutical medicines; and cosmetics (64).
Various cancers that have been linked to environmental carcinogens. The carcinogens linked to each cancer is shown inside bracket. (see 64).
Numerous outdoor air pollutants such as PAHs increase the risk of cancers, especially lung cancer. PAHs can adhere to fine carbon particles in the atmosphere and thus penetrate our bodies primarily through breathing. Long-term exposure to PAH-containing air in polluted cities was found to increase the risk of lung cancer deaths. Aside from PAHs and other fine carbon particles, another environmental pollutant, nitric oxide, was found to increase the risk of lung cancer in a European population of nonsmokers. Other studies have shown that nitric oxide can induce lung cancer and promote metastasis. The increased risk of childhood leukemia associated with exposure to motor vehicle exhaust was also reported (64).
Indoor air pollutants such as volatile organic compounds and pesticides increase the risk of childhood leukemia and lymphoma, and children as well as adults exposed to pesticides have increased risk of brain tumors, Wilm’s tumors, Ewing’s sarcoma, and germ cell tumors. In utero exposure to environmental organic pollutants was found to increase the risk for testicular cancer. In addition, dioxan, an environmental pollutant from incinerators, was found to increase the risk of sarcoma and lymphoma.
Long-term exposure to chlorinated drinking water has been associated with increased risk of cancer. Nitrates, in drinking water, can transform to mutagenic N-nitroso compounds, which increase the risk of lymphoma, leukemia, colorectal cancer, and bladder cancer (64).
Up to 10% of total cancer cases may be induced by radiation (64), both ionizing and non-ionizing, typically from radioactive substances and ultraviolet (UV), pulsed electromagnetic fields. Cancers induced by radiation include some types of leukemia, lymphoma, thyroid cancers, skin cancers, sarcomas, lung and breast carcinomas. One of the best examples of increased risk of cancer after exposure to radiation is the increased incidence of total malignancies observed in Sweden after exposure to radioactive fallout from the Chernobyl nuclear power plant. Radon and radon decay products in the home and/or at workplaces (such as mines) are the most common sources of exposure to ionizing radiation. The presence of radioactive nuclei from radon, radium, and uranium was found to increase the risk of gastric cancer in rats. Another source of radiation exposure is x-rays used in medical settings for diagnostic or therapeutic purposes. In fact, the risk of breast cancer from x-rays is highest among girls exposed to chest irradiation at puberty, a time of intense breast development. Other factors associated with radiation-induced cancers in humans are patient age and physiological state, synergistic interactions between radiation and carcinogens, and genetic susceptibility toward radiation.
Non-ionizing radiation derived primarily from sunlight includes UV rays, which are carcinogenic to humans. Exposure to UV radiation is a major risk for various types of skin cancers including basal cell carcinoma, squamous cell carcinoma, and melanoma. Along with UV exposure from sunlight, UV exposure from sun beds for cosmetic tanning may account for the growing incidence of melanoma. Depletion of the ozone layer in the stratosphere can augment the dose-intensity of UVB and UVC, which can further increase the incidence of skin cancer.
Low-frequency electromagnetic fields can cause clastogenic DNA damage. The sources of electromagnetic field exposure are high-voltage power lines, transformers, electric train engines, and more generally, all types of electrical equipments. An increased risk of cancers such as childhood leukemia, brain tumors and breast cancer has been attributed to electromagnetic field exposure. For instance, children living within 200 m of high-voltage power lines have a relative risk of leukemia of 69%, whereas those living between 200 and 600 m from these power lines have a relative risk of 23%. In addition, a recent meta-analysis of all available epidemiologic data showed that daily prolonged use of mobile phones for 10 years or more showed a consistent pattern of an increased risk of brain tumors (64).
Fruits, vegetables, spices, condiments and cereals with potential to prevent cancer. Fruits include 1 apple, 2apricot, 3 banana, 4 blackberry, 5 cherry, 6 citrus fruits, 7 dessert date, 8 durian, 9 grapes, 10 guava, 11 Indian gooseberry, 12 mango, 13 malay apple, 14 mangosteen, 15 pineapple, 16 pomegranate. Vegetables include 1artichok, 2 avocado, 3 brussels sprout, 4 broccoli, 5 cabbage, 6 cauliflower, 7 carrot, 8 daikon 9 kohlrabi, 10onion, 11 tomato, 12 turnip, 13 ulluco, 14 water cress, 15 okra, 16 potato, 17 fiddle head, 18 radicchio, 19komatsuna, 20 salt bush, 21 winter squash, 22 zucchini, 23 lettuce, 24 spinach. Spices and condiments include 1 turmeric, 2 cardamom, 3 coriander, 4 black pepper, 5 clove, 6 fennel, 7 rosemary, 8 sesame seed, 9 mustard, 10 licorice, 11 garlic, 12 ginger, 13 parsley, 14 cinnamon, 15 curry leaves, 16 kalonji, 17 fenugreek, 18camphor, 19 pecan, 20 star anise, 21 flax seed, 22 black mustard, 23 pistachio, 24 walnut, 25 peanut, 26 cashew nut. Cereals include 1 rice, 2 wheat, 3 oats, 4 rye, 5 barley, 6 maize, 7 jowar, 8 pearl millet, 9 proso millet, 10 foxtail millet, 11 little millet, 12 barnyard millet, 13 kidney bean, 14 soybean, 15 mung bean, 16 black bean, 17 pigeon pea, 18 green pea, 19 scarlet runner bean, 20 black beluga, 21 brown spanish pardina, 22green, 23 green (eston), 24 ivory white, 25 multicolored blend, 26 petite crimson, 27 petite golden, 28 red chief.
Join Robert O Young PhD and Galina Migalko MD in Dubai on December 5th and 6th, 2018 for the Annual Conference on Bacterial, Viral and Infectious Diseases. They will be Key Note Speakers and doing a workshop on the New Biology.
For more information and to register go to: https://bacterialdiseases.infectiousconferences.com/organizing-committee.php
The following is the abstract for Dr. Young’s lecture:
Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner
There is now over 100 years of documented history and research on the Polio virus and whether or not its treatment by inoculation has been successful in eradicating Polio. I am suggesting in this article and in my lecture that there are significant findings based on historical and past and current research, including my own that the viral theory of Polio and possibly other modern-day diseases, such as Post-Polio Syndrome, Polio Vaccine-Induced Paralysis, Legionnaires, CNS disease, Cancer, HIV/AIDS and now Zika may be caused by acidic chemical poisoning from DDT (dichloro-diphenyl-trichloroethane) and other related DDT pesticides, acidic vaccinations, and other factors including lifestyle and dietary factors rather than from a lone infectious virus. I will present ten historical graphs outlining the history of Polio, the production of DDT, BHC, Lead, Arsenic, Polio vaccinations and the author’s theory that chemical poisoning, vaccination, and lifestyle and dietary choices are a more likely causes for the symptoms of Polio, neurological diseases, Cancer, HIV/AIDS and now Zika.
1. L. N. Kolonel, D. Altshuler, and B. E. Henderson. The multiethnic cohort study: exploring genes, lifestyle and cancer risk. Nat. Rev. Cancer. 4:519–27 (2004) doi:10.1038/nrc1389. [PubMed]
2. J. K. Wiencke. Impact of race/ethnicity on molecular pathways in human cancer. Nat. Rev. Cancer. 4:79–84 (2004) doi:10.1038/nrc1257. [PubMed]
3. R. G. Ziegler, R. N. Hoover, M. C. Pike, A. Hildesheim, A. M. Nomura, D. W. West, A. H. Wu-Williams, L. N. Kolonel, P. L. Horn-Ross, J. F. Rosenthal, and M. B. Hyer. Migration patterns and breast cancer risk in Asian-American women. J. Natl. Cancer Inst.85:1819–27 (1993) doi:10.1093/jnci/85.22.1819. [PubMed]
4. W. Haenszel and M. Kurihara. Studies of Japanese migrants. I. Mortality from cancer and other diseases among Japanese in the United States. J. Natl. Cancer Inst.40:43–68 (1968). [PubMed]
5. A. S. Hamilton and T. M. Mack. Puberty and genetic susceptibility to breast cancer in a case-control study in twins. N. Engl. J. Med.348:2313–22 (2003) doi:10.1056/NEJMoa021293. [PubMed]
6. A. Jemal, R. Siegel, E. Ward, T. Murray, J. Xu, and M. J. Thun. Cancer statistics, 2007. CA Cancer J. Clin.57:43–66 (2007). [PubMed]
7. F. Brayand, and B. Moller. Predicting the future burden of cancer. Nat. Rev. Cancer. 6:63–74 (2006) doi:10.1038/nrc1781. [PubMed]
8. P. Lichtenstein, N. V. Holm, P. K. Verkasalo, A. Iliadou, J. Kaprio, M. Koskenvuo, E. Pukkala, A. Skytthe, and K. Hemminki. Environmental and heritable factors in the causation of cancer—analyses of cohorts of twins from Sweden, Denmark, and Finland. N. Engl. J. Med.343:78–85 (2000) doi:10.1056/NEJM200007133430201. [PubMed]
9. K. R. Loeb, and L. A. Loeb. Significance of multiple mutations in cancer. Carcinogenesis. 21:379–85 (2000) doi:10.1093/carcin/21.3.379. [PubMed]
10. W. C. Hahn, and R. A. Weinberg. Modelling the molecular circuitry of cancer. Nat. Rev. Cancer. 2:331–41 (2002) doi: 10.1038/nrc795. [PubMed]
11. L. A. Mucci, S. Wedren, R. M. Tamimi, D. Trichopoulos, and H. O. Adami. The role of gene-environment interaction in the aetiology of human cancer: examples from cancers of the large bowel, lung and breast. J. Intern. Med.249:477–93 (2001) doi:10.1046/j.1365-2796.2001.00839.x. [PubMed]
12. K. Czene, and K. Hemminki. Kidney cancer in the Swedish Family Cancer Database: familial risks and second primary malignancies. Kidney Int.61:1806–13 (2002) doi:10.1046/j.1523-1755.2002.00304.x.[PubMed]
13. P. Irigaray, J. A. Newby, R. Clapp, L. Hardell, V. Howard, L. Montagnier, S. Epstein, and D. Belpomme. Lifestyle-related factors and environmental agents causing cancer: an overview. Biomed. Pharmacother.61:640–58 (2007) doi:10.1016/j.biopha.2007.10.006. [PubMed]
14. M. F. Denissenko, A. Pao, M. Tang, and G. P. Pfeifer. Preferential formation of benzo[a]pyrene adducts at lung cancer mutational hotspots in P53. Science. 274:430–2 (1996) doi:10.1126/science.274.5286.430.[PubMed]
15. R. J. Anto, A. Mukhopadhyay, S. Shishodia, C. G. Gairola, and B. B. Aggarwal. Cigarette smoke condensate activates nuclear transcription factor-kappaB through phosphorylation and degradation of IkappaB(alpha): correlation with induction of cyclooxygenase-2. Carcinogenesis. 23:1511–8 (2002) doi: 10.1093/carcin/23.9.1511. [PubMed]
16. S. Shishodiaand, and B. B. Aggarwal. Cyclooxygenase (COX)-2 inhibitor celecoxib abrogates activation of cigarette smoke-induced nuclear factor (NF)-kappaB by suppressing activation of IkappaBalpha kinase in human non-small cell lung carcinoma: correlation with suppression of cyclin D1, COX-2, and matrix metalloproteinase-9. Cancer Res. 64:5004–12 (2004) doi:10.1158/0008-5472.CAN-04-0206. [PubMed]
17. H. Ichikawa, Y. Nakamura, Y. Kashiwada, and B. B. Aggarwal. Anticancer drugs designed by mother nature: ancient drugs but modern targets. Curr Pharm Des. 13:3400–16 (2007) doi:10.2174/138161207782360500. [PubMed]
18. A. J. Tuyns. Epidemiology of alcohol and cancer. Cancer Res. 39:2840–3 (1979). [PubMed]
19. H. Maier, E. Sennewald, G. F. Heller, and H. Weidauer. Chronic alcohol consumption—the key risk factor for pharyngeal cancer. Otolaryngol. Head Neck Surg.110:168–73 (1994). [PubMed]
20. H. K. Seitz, F. Stickel, and N. Homann. Pathogenetic mechanisms of upper aerodigestive tract cancer in alcoholics. Int. J. Cancer. 108:483–7 (2004) doi:10.1002/ijc.11600. [PubMed]
21. R. Doll, and R. Peto. The causes of cancer: quantitative estimates of avoidable risks of cancer in the United States today. J. Natl. Cancer Inst. 66:1191–308 (1981). [PubMed]
22. R. R. Williams, and J. W. Horm. Association of cancer sites with tobacco and alcohol consumption and socioeconomic status of patients: interview study from the Third National Cancer Survey. J. Natl. Cancer Inst.58:525–47 (1977). [PubMed]
23. N. Hamajima et al. Alcohol, tobacco and breast cancer—collaborative reanalysis of individual data from 53 epidemiological studies, including 58,515 women with breast cancer and 95,067 women without the disease. Br. J. Cancer. 87:1234–45 (2002) doi:10.1038/sj.bjc.6600596. [PMC free article] [PubMed]
24. M. P. Longnecker, P. A. Newcomb, R. Mittendorf, E. R. Greenberg, R. W. Clapp, G. F. Bogdan, J. Baron, B. MacMahon, and W. C. Willett. Risk of breast cancer in relation to lifetime alcohol consumption. J. Natl. Cancer Inst.87:923–9 (1995) doi:10.1093/jnci/87.12.923. [PubMed]
25. F. Stickel, D. Schuppan, E. G. Hahn, and H. K. Seitz. Cocarcinogenic effects of alcohol in hepatocarcinogenesis. Gut. 51:132–9 (2002) doi:10.1136/gut.51.1.132. [PMC free article] [PubMed]
26. H. K. Seitz, G. Poschl, and U. A. Simanowski. Alcohol and cancer. Recent Dev Alcohol. 14:67–95 (1998) doi:10.1007/0-306-47148-5_4. [PubMed]
27. H. K. Seitz, S. Matsuzaki, A. Yokoyama, N. Homann, S. Vakevainen, and X. D. Wang. Alcohol and cancer. Alcohol Clin. Exp. Res.25:137S–143S (2001). [PubMed]
28. F. Donato, U. Gelatti, R. M. Limina, and G. Fattovich. Southern Europe as an example of interaction between various environmental factors: a systematic review of the epidemiologic evidence. Oncogene. 25:3756–70 (2006) doi:10.1038/sj.onc.1209557. [PubMed]
29. G. Poschl, and H. K. Seitz. Alcohol and cancer. Alcohol Alcohol. 39:155–65 (2004) doi:10.1093/alcalc/agh057. [PubMed]
30. G. Szabo, P. Mandrekar, S. Oak, and J. Mayerle. Effect of ethanol on inflammatory responses. Implications for pancreatitis. Pancreatology. 7:115–23 (2007) doi:10.1159/000104236. [PMC free article][PubMed]
31. B. B. Aggarwal. Nuclear factor-kappaB: the enemy within. Cancer Cell. 6:203–208 (2004) doi:10.1016/j.ccr.2004.09.003. [PubMed]
32. M. Kuratsune, S. Kohchi, and A. Horie. Carcinogenesis in the esophagus. I. Penetration of benzo(a) pyrene and other hydrocarbons into the esophageal mucosa. Gann. 56:177–87 (1965). [PubMed]
33. C. La Vecchia, A. Tavani, S. Franceschi, F. Levi, G. Corrao, and E. Negri. Epidemiology and prevention of oral cancer. Oral Oncol.33:302–312 (1997). [PubMed]
34. P. Boffetta, M. Hashibe, C. La Vecchia, W. Zatonski, and J. Rehm. The burden of cancer attributable to alcohol drinking. Int. J. Cancer. 119:884–887 (2006) doi:10.1002/ijc.21903. [PubMed]
35. W. C. Willett. Diet and cancer. Oncologist. 5:393–404 (2000) doi:10.1634/theoncologist.5-5-393.[PubMed]
36. S. A. Bingham, R. Hughes, and A. J. Cross. Effect of white versus red meat on endogenous N-nitrosation in the human colon and further evidence of a dose response. J. Nutr.132:3522S–3525S (2002).[PubMed]
37. A. Chao, M. J. Thun, C. J. Connell, M. L. McCullough, E. J. Jacobs, W. D. Flanders, C. Rodriguez, R. Sinha, and E. E. Calle. Meat consumption and risk of colorectal cancer. JAMA. 293:172–182 (2005) doi:10.1001/jama.293.2.172. [PubMed]
38. N. Hogg. Red meat and colon cancer: heme proteins and nitrite in the gut. A commentary on diet-induced endogenous formation of nitroso compounds in the GI tract. Free Radic. Biol. Med.43:1037–1039 (2007) doi:10.1016/j.freeradbiomed.2007.07.006. [PubMed]
39. C. Rodriguez, M. L. McCullough, A. M. Mondul, E. J. Jacobs, A. Chao, A. V. Patel, M. J. Thun, and E. E. Calle. Meat consumption among Black and White men and risk of prostate cancer in the Cancer Prevention Study II Nutrition Cohort. Cancer Epidemiol. Biomarkers Prev. 15:211–216 (2006) doi:10.1158/1055-9965.EPI-05-0614. [PubMed]
40. R. Garcia-Closas, M. Garcia-Closas, M. Kogevinas, N. Malats, D. Silverman, C. Serra, A. Tardon, A. Carrato, G. Castano-Vinyals, M. Dosemeci, L. Moore, N. Rothman, and R. Sinha. Food, nutrient and heterocyclic amine intake and the risk of bladder cancer. Eur. J. Cancer. 43:1731–1740 (2007) doi:10.1016/j.ejca.2007.05.007. [PubMed]
41. A. Tappel. Heme of consumed red meat can act as a catalyst of oxidative damage and could initiate colon, breast and prostate cancers, heart disease and other diseases. Med. Hypotheses. 68:562–4 (2007) doi:10.1016/j.mehy.2006.08.025. [PubMed]
42. L. H. O’Hanlon. High meat consumption linked to gastric-cancer risk. Lancet Oncol. 7:287 (2006) doi:10.1016/S1470-2045(06)70638-6. [PubMed]
43. T. N. Toporcov, J. L. Antunes, and M. R. Tavares. Fat food habitual intake and risk of oral cancer. Oral Oncol. 40:925–931 (2004) doi:10.1016/j.oraloncology.2004.04.007. [PubMed]
44. O. Dosil-Diaz, A. Ruano-Ravina, J. J. Gestal-Otero, and J. M. Barros-Dios. Meat and fish consumption and risk of lung cancer: A case-control study in Galicia, Spain. Cancer Lett.252:115–122 (2007) doi:10.1016/j.canlet.2006.12.008. [PubMed]
45. S. N. Lauber, and N. J. Gooderham. The cooked meat derived genotoxic carcinogen 2-amino-3-methylimidazo[4,5-b]pyridine has potent hormone-like activity: mechanistic support for a role in breast cancer. Cancer Res.67:9597–0602 (2007) doi:10.1158/0008–5472.CAN-07-1661. [PubMed]
46. D. Divisi, S. Di Tommaso, S. Salvemini, M. Garramone, and R. Crisci. Diet and cancer. Acta Biomed. 77:118–123 (2006). [PubMed]
47. Y. F. Sasaki, S. Kawaguchi, A. Kamaya, M. Ohshita, K. Kabasawa, K. Iwama, K. Taniguchi, and S. Tsuda. The comet assay with 8 mouse organs: results with 39 currently used food additives. Mutat. Res.519:103–119 (2002). [PubMed]
48. M. Durando, L. Kass, J. Piva, C. Sonnenschein, A. M. Soto, E. H. Luque, and M. Munoz-de-Toro. Prenatal bisphenol A exposure induces preneoplastic lesions in the mammary gland in Wistar rats. Environ. Health Perspect.115:80–6 (2007). [PMC free article] [PubMed]
49. S. M. Ho, W. Y. Tang, J. Belmonte de Frausto, and G. S. Prins. Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4. Cancer Res.66:5624–32 (2006) doi:10.1158/0008-5472.CAN-06-0516.[PMC free article] [PubMed]
50. A. Szymanska-Chabowska, J. Antonowicz-Juchniewicz, and R. Andrzejak. Some aspects of arsenic toxicity and carcinogenicity in living organism with special regard to its influence on cardiovascular system, blood and bone marrow. Int. J. Occup. Med. Environ. Health. 15:101–116 (2002). [PubMed]
51. E. E. Calle, C. Rodriguez, K. Walker-Thurmond, and M. J. Thun. Overweight, obesity, and mortality from cancer in a prospectively studied cohort of U.S. adults. N Engl J Med. 348:1625–1638 (2003) doi:10.1056/NEJMoa021423. [PubMed]
52. A. Drewnowski, and B. M. Popkin. The nutrition transition: new trends in the global diet. Nutr. Rev.55:31–43 (1997). [PubMed]
53. S. D. Hursting, L. M. Lashinger, L. H. Colbert, C. J. Rogers, K. W. Wheatley, N. P. Nunez, S. Mahabir, J. C. Barrett, M. R. Forman, and S. N. Perkins. Energy balance and carcinogenesis: underlying pathways and targets for intervention. Curr. Cancer Drug Targets. 7:484–491 (2007) doi:10.2174/156800907781386623. [PubMed]
54. A. Nareika, Y. B. Im, B. A. Game, E. H. Slate, J. J. Sanders, S. D. London, M. F. Lopes-Virella, and Y. Huang. High glucose enhances lipopolysaccharide-stimulated CD14 expression in U937 mononuclear cells by increasing nuclear factor kappaB and AP-1 activities. J. Endocrinol.196:45–55 (2008) doi:10.1677/JOE-07-0145. [PubMed]
55. C. H. Tang, Y. C. Chiu, T. W. Tan, R. S. Yang, and W. M. Fu. Adiponectin enhances IL-6 production in human synovial fibroblast via an AdipoR1 receptor, AMPK, p38, and NF-kappa B pathway. J. Immunol.179:5483–5492 (2007). [PubMed]
56. P. Pisani, D. M. Parkin, N. Munoz, and J. Ferlay. Cancer and infection: estimates of the attributable fraction in 1990. Cancer Epidemiol. Biomarkers Prev.6:387–400 (1997). [PubMed]
57. D. M. Parkin. The global health burden of infection-associated cancers in the year 2002. Int. J. Cancer. 118:3030–3044 (2006) doi:10.1002/ijc.21731. [PubMed]
58. S. Song, H. C. Pitot, and P. F. Lambert. The human papillomavirus type 16 E6 gene alone is sufficient to induce carcinomas in transgenic animals. J. Virol.73:5887–5893 (1999). [PMC free article] [PubMed]
59. B. S. Blumberg, B. Larouze, W. T. London, B. Werner, J. E. Hesser, I. Millman, G. Saimot, and M. Payet. The relation of infection with the hepatitis B agent to primary hepatic carcinoma. Am. J. Pathol.81:669–682 (1975). [PMC free article] [PubMed]
60. T. M. Hagen, S. Huang, J. Curnutte, P. Fowler, V. Martinez, C. M. Wehr, B. N. Ames, and F. V. Chisari. Extensive oxidative DNA damage in hepatocytes of transgenic mice with chronic active hepatitis destined to develop hepatocellular carcinoma. Proc. Natl. Acad. Sci. U S A. 91:12808–12812 (1994) doi:10.1073/pnas.91.26.12808. [PMC free article] [PubMed]
61. A. L. Jackson, and L. A. Loeb. The contribution of endogenous sources of DNA damage to the multiple mutations in cancer. Mutat. Res.477:7–21 (2001) doi:10.1016/S0027-5107(01)00091-4. [PubMed]
62. N. De Maria, A. Colantoni, S. Fagiuoli, G. J. Liu, B. K. Rogers, F. Farinati, D. H. Van Thiel, and R. A. Floyd. Association between reactive oxygen species and disease activity in chronic hepatitis C. Free Radic. Biol. Med.21:291–5 (1996) doi:10.1016/0891–5849(96)00044-5. [PubMed]
63. K. Koike, T. Tsutsumi, H. Fujie, Y. Shintani, and M. Kyoji. Molecular mechanism of viral hepatocarcinogenesis. Oncology. 62(Suppl 1):29–37 (2002) doi:10.1159/000048273. [PubMed]
64. D. Belpomme, P. Irigaray, L. Hardell, R. Clapp, L. Montagnier, S. Epstein, and A. J. Sasco. The multitude and diversity of environmental carcinogens. Environ. Res.105:414–429 (2007) doi:10.1016/j.envres.2007.07.002. [PubMed]
65. Y. S. Guan, Q. He, M. Q. Wang, and P. Li. Nuclear factor kappa B and hepatitis viruses. Expert Opin. Ther. Targets. 12:265–280 (2008) doi:10.1517/14728126.96.36.1995. [PubMed]
66. S. Takayama, H. Takahashi, Y. Matsuo, Y. Okada, and T. Manabe. Effects of Helicobacter pylori infection on human pancreatic cancer cell line. Hepatogastroenterology. 54:2387–2391 (2007). [PubMed]
67. K. A. Steinmetz, and J. D. Potter. Vegetables, fruit, and cancer prevention: a review. J. Am. Diet Assoc.96:1027–1039 (1996) doi:10.1016/S0002–8223(96)00273-8. [PubMed]
68. P. Greenwald. Lifestyle and medical approaches to cancer prevention. Recent Results Cancer Res.166:1–15 (2005). [PubMed]
69. H. Vainio, and E. Weiderpass. Fruit and vegetables in cancer prevention. Nutr. Cancer. 54:111–42 (2006) doi:10.1207/s15327914nc5401_13. [PubMed]
70. L. W. Wattenberg. Chemoprophylaxis of carcinogenesis: a review. Cancer Res. 26:1520–1526 (1966).[PubMed]
71. B. B. Aggarwal, and S. Shishodia. Molecular targets of dietary agents for prevention and therapy of cancer. Biochem. Pharmacol.71:1397–1421 (2006) doi:10.1016/j.bcp.2006.02.009. [PubMed]
72. H. Nishino, M. Murakosh, T. Ii, M. Takemura, M. Kuchide, M. Kanazawa, X. Y. Mou, S. Wada, M. Masuda, Y. Ohsaka, S. Yogosawa, Y. Satomi, and K. Jinno. Carotenoids in cancer chemoprevention. Cancer Metastasis Rev.21:257–264 (2002) doi:10.1023/A:1021206826750. [PubMed]
73. K. B. Harikumar, and B. B. Aggarwal. Resveratrol: A multitargeted agent for age-associated chronic diseases. Cell Cycle. 7:1020–1037 (2008). [PubMed]
74. G. L. Russo. Ins and outs of dietary phytochemicals in cancer chemoprevention. Biochem. Pharmacol. 74:533–544 (2007) doi:10.1016/j.bcp.2007.02.014. [PubMed]
75. R. Agarwal, C. Agarwal, H. Ichikawa, R. P. Singh, and B. B. Aggarwal. Anticancer potential of silymarin: from bench to bed side. Anticancer Res. 26:4457–98 (2006). [PubMed]
76. E. G. Rogan. The natural chemopreventive compound indole-3-carbinol: state of the science. In Vivo. 20:221–228 (2006). [PubMed]
77. N. Juge, R. F. Mithen, and M. Traka. Molecular basis for chemoprevention by sulforaphane: a comprehensive review. Cell Mol Life Sci. 64:1105–27 (2007) doi:10.1007/s00018-007-6484-5. [PubMed]
78. L. Chen, and H. Y. Zhang. Cancer preventive mechanisms of the green tea polyphenol (−)-epigallocatechin-3-gallate. Molecules. 12:946–957 (2007). [PMC free article] [PubMed]
79. P. Anand, C. Sundaram, S. Jhurani, A. B. Kunnumakkara, and B. B. Aggarwal. Curcumin and cancer: An “old-age” disease with an “age-old” solution. Cancer Lett. in press (2008). [PubMed]
80. F. Khanum, K. R. Anilakumar, and K. R. Viswanathan. Anticarcinogenic properties of garlic: a review. Crit. Rev. Food Sci. Nutr.44:479–488 (2004) doi:10.1080/10408690490886700. [PubMed]
81. G. Sethi, K. S. Ahn and B. B. Aggarwal. Targeting NF-kB activation pathway by thymoquinone: Role in suppression of antiapoptotic gene products and enhancement of apoptosis. Mole Cancer Res. in press (2008). [PubMed]
82. Y. J. Surh. Anti-tumor promoting potential of selected spice ingredients with antioxidative and anti-inflammatory activities: a short review. Food Chem. Toxicol.40:1091–1097 (2002) doi:10.1016/S0278-6915(02)00037-6. [PubMed]
83. Y. Shukla, and M. Singh. Cancer preventive properties of ginger: a brief review. Food Chem. Toxicol.45:683–690 (2007) doi:10.1016/j.fct.2006.11.002. [PubMed]
84. M. M. al-Harbi, S. Qureshi, M. Raza, M. M. Ahmed, A. B. Giangreco, and A. H. Shah. Influence of anethole treatment on the tumour induced by Ehrlich ascites carcinoma cells in paw of Swiss albino mice. Eur. J. Cancer Prev.4:307–318 (1995) doi:10.1097/00008469-199508000-00006. [PubMed]
85. C. K. Sen, K. E. Traber, and L. Packer. Inhibition of NF-kappa B activation in human T-cell lines by anetholdithiolthione. Biochem. Biophys. Res. Commun.218:148–53 (1996) doi:10.1006/bbrc.1996.0026.[PubMed]
86. R. A. Lubet, V. E. Steele, I. Eto, M. M. Juliana, G. J. Kelloff, and C. J. Grubbs. Chemopreventive efficacy of anethole trithione, N-acetyl-L-cysteine, miconazole and phenethylisothiocyanate in the DMBA-induced rat mammary cancer model. Int. J. Cancer. 72:95–101 (1997) doi:10.1002/(SICI)1097-0215(19970703)72:1<95::AID-IJC14>3.0.CO;2-9. [PubMed]
87. Y. Nakagawa, and T. Suzuki. Cytotoxic and xenoestrogenic effects via biotransformation of trans-anethole on isolated rat hepatocytes and cultured MCF-7 human breast cancer cells. Biochem. Pharmacol.66:63–73 (2003) doi:10.1016/S0006-2952(03)00208-9. [PubMed]
88. S. Lam, C. MacAulay, J. C. Le Riche, Y. Dyachkova, A. Coldman, M. Guillaud, E. Hawk, M. O. Christen, and A. F. Gazdar. A randomized phase IIb trial of anethole dithiolethione in smokers with bronchial dysplasia. J. Natl. Cancer Inst.94:1001–1009 (2002). [PubMed]
89. S. Shishodia, and B. B. Aggarwal. Diosgenin inhibits osteoclastogenesis, invasion, and proliferation through the downregulation of Akt, I kappa B kinase activation and NF-kappa B-regulated gene expression. Oncogene. 25:1463–1473 (2006) doi:10.1038/sj.onc.1209194. [PubMed]
90. R. Ghosh, N. Nadiminty, J. E. Fitzpatrick, W. L. Alworth, T. J. Slaga, and A. P. Kumar. Eugenol causes melanoma growth suppression through inhibition of E2F1 transcriptional activity. J. Biol. Chem.280:5812–5819 (2005) doi:10.1074/jbc.M411429200. [PubMed]
91. K. Sukumaran, M. C. Unnikrishnan, and R. Kuttan. Inhibition of tumour promotion in mice by eugenol. Indian J. Physiol. Pharmacol.38:306–308 (1994). [PubMed]
92. K. Imaida, M. Hirose, S. Yamaguchi, S. Takahashi, and N. Ito. Effects of naturally occurring antioxidants on combined 1,2-dimethylhydrazine- and 1-methyl-1-nitrosourea-initiated carcinogenesis in F344 male rats. Cancer Lett.55:53–59 (1990) doi:10.1016/0304-3835(90)90065-6. [PubMed]
93. M. Pisano, G. Pagnan, M. Loi, M. E. Mura, M. G. Tilocca, G. Palmieri, D. Fabbri, M. A. Dettori, G. Delogu, M. Ponzoni, and C. Rozzo. Antiproliferative and pro-apoptotic activity of eugenol-related biphenyls on malignant melanoma cells. Mol Cancer. 6:8 (2007) doi:10.1186/1476-4598-6-8.[PMC free article] [PubMed]
94. S. S. Kim, O. J. Oh, H. Y. Min, E. J. Park, Y. Kim, H. J. Park, Y. Nam Han, and S. K. Lee. Eugenol suppresses cyclooxygenase-2 expression in lipopolysaccharide-stimulated mouse macrophage RAW264.7 cells. Life Sci. 73:337–348 (2003) doi:10.1016/S0024–3205(03)00288-1. [PubMed]
95. H. P. Deigner, G. Wolf, U. Ohlenmacher, and J. Reichling. 1¢-Hydroxyeugenol- and coniferyl alcohol derivatives as effective inhibitors of 5-lipoxygenase and Cu(2+)-mediated low density lipoprotein oxidation. Evidence for a dual mechanism. Arzneimittelforschung. 44:956–961 (1994). [PubMed]
96. C. J. Rompelberg, M. J. Steenwinkel, J. G. van Asten, J. H. van Delft, R. A. Baan, and H. Verhagen. Effect of eugenol on the mutagenicity of benzo[a]pyrene and the formation of benzo[a]pyrene-DNA adducts in the lambda-lacZ-transgenic mouse. Mutat. Res.369:87–96 (1996) doi:10.1016/S0165-1218(96)90052-X. [PubMed]
97. D. P. Richardson. The grain, the wholegrain and nothing but the grain: the science behind wholegrain and the reduced risk of heart disease and cancer. Nutr. Bull.25:353–360 (2000) doi:10.1046/j.1467-3010.2000.00083.x.
98. H. E. Miller, F. Rigelhof, L. Marquart, A. Prakash, and M. Kanter. Antioxidant content of whole grain breakfast cereals, fruits and vegetables. J. Am. Coll. Nutr.19:312S–319S (2000). [PubMed]
99. J. L. Slavin, D. Jacobs, and L. Marquart. Grain processing and nutrition. Crit. Rev. Food Sci. Nutr.40:309–326 (2000) doi:10.1080/10408690091189176. [PubMed]
100. L. Chatenoud, A. Tavani, C. La Vecchia, D. R. Jacobs, Jr, E. Negri, F. Levi, and S. Franceschi. Whole grain food intake and cancer risk. Int. J. Cancer. 77:24–8 (1998) doi:10.1002/(SICI)1097-0215(19980703)77:1<24::AID-IJC5>3.0.CO;2-1. [PubMed]
101. D. R. Jacobs, Jr, L. Marquart, J. Slavin, and L. H. Kushi. Whole-grain intake and cancer: an expanded review and meta-analysis. Nutr. Cancer. 30:85–96 (1998). [PubMed]
102. L. Marquart, K. L. Wiemer, J. M. Jones, and B. Jacob. Whole grains health claims in the USA and other efforts to increase whole-grain consumption. Proc. Nutr. Soc.62:151–160 (2003) doi:10.1079/PNS2003242. [PubMed]
103. M. Eastwood, and D. Kritchevsky. Dietary fiber: how did we get where we are? Annu. Rev. Nutr.25:1–8 (2005) doi:10.1146/annurev.nutr.25.121304.131658. [PubMed]
104. A. McIntyre, P. R. Gibson, and G. P. Young. Butyrate production from dietary fibre and protection against large bowel cancer in a rat model. Gut. 34:386–391 (1993) doi:10.1136/gut.34.3.386.[PMC free article] [PubMed]
105. J. L. Slavin, D. Jacobs, L. Marquart, and K. Wiemer. The role of whole grains in disease prevention. J. Am. Diet Assoc.101:780–5 (2001) doi:10.1016/S0002-8223(01)00194-8. [PubMed]
106. K. S. Ahn, G. Sethi, K. Krishnan, and B. B. Aggarwal. Gamma-tocotrienol inhibits nuclear factor-kappaB signaling pathway through inhibition of receptor-interacting protein and TAK1 leading to suppression of antiapoptotic gene products and potentiation of apoptosis. J. Biol. Chem.282:809–820 (2007) doi:10.1074/jbc.M610028200. [PubMed]
107. F. H. Sarkar, S. Adsule, S. Padhye, S. Kulkarni, and Y. Li. The role of genistein and synthetic derivatives of isoflavone in cancer prevention and therapy. Mini Rev. Med. Chem.6:401–407 (2006) doi:10.2174/138955706776361439. [PubMed]
108. K. W. Lee, H. J. Lee, Y. J. Surh, and C. Y. Lee. Vitamin C and cancer chemoprevention: reappraisal. Am. J. Clin. Nutr.78:1074–1078 (2003). [PubMed]
109. B. A. Ingraham, B. Bragdon, and A. Nohe. Molecular basis of the potential of vitamin D to prevent cancer. Curr. Med. Res. Opin.24:139–149 (2008) doi:10.1185/030079907X253519. [PubMed]
110. F. W. Booth, M. V. Chakravarthy, S. E. Gordon, and E. E. Spangenburg. Waging war on physical inactivity: using modern molecular ammunition against an ancient enemy. J. Appl. Physiol.93:3–30 (2002).[PubMed]
111. G. A. Colditz, C. C. Cannuscio, and A. L. Frazier. Physical activity and reduced risk of colon cancer: implications for prevention. Cancer Causes Control. 8:649–67 (1997) doi:10.1023/A:1018458700185.[PubMed]
112. A. R. Shors, C. Solomon, A. McTiernan, and E. White. Melanoma risk in relation to height, weight, and exercise (United States). Cancer Causes Control. 12:599–606 (2001) doi:10.1023/A:1011211615524.[PubMed]
113. A. Tannenbaum, and H. Silverstone. The initiation and growth of tumors. Introduction. I. Effects of underfeeding. Am. J. Cancer. 38:335–350 (1940).
114. S. D. Hursting, J. A. Lavigne, D. Berrigan, S. N. Perkins, and J. C. Barrett. Calorie restriction, aging, and cancer prevention: mechanisms of action and applicability to humans. Annu. Rev. Med.54:131–152 (2003) doi:10.1146/annurev.med.54.101601.152156. [PubMed]
115. M. H. Ross, and G. Bras. Lasting influence of early caloric restriction on prevalence of neoplasms in the rat. J. Natl. Cancer Inst.47:1095–1113 (1971). [PubMed]
116. D. Albanes. Total calories, body weight, and tumor incidence in mice. Cancer Res.47:1987–92 (1987).[PubMed]
117. L. Gross, and Y. Dreyfuss. Reduction in the incidence of radiation-induced tumors in rats after restriction of food intake. Proc. Natl. Acad. Sci. U S A. 81:7596–7598 (1984) doi:10.1073/pnas.81.23.7596. [PMC free article] [PubMed]
118. L. Gross, and Y. Dreyfuss. Prevention of spontaneous and radiation-induced tumors in rats by reduction of food intake. Proc. Natl. Acad. Sci. U S A. 87:6795–6797 (1990) doi:10.1073/pnas.87.17.6795.[PMC free article] [PubMed]
119. K. Yoshida, T. Inoue, K. Nojima, Y. Hirabayashi, and T. Sado. Calorie restriction reduces the incidence of myeloid leukemia induced by a single whole-body radiation in C3H/He mice. Proc. Natl. Acad. Sci. U S A. 94:2615–2619 (1997) doi:10.1073/pnas.94.6.2615. [PMC free article] [PubMed]
120. V. D. Longo, and C. E. Finch. Evolutionary medicine: From dwarf model systems to healthy centenarians? Science. 299:1342–1346 (2003) doi:10.1126/science.1077991. [PubMed]
Br J Cancer. 1999 Jun;80(7):1005-11.
The extracellular (interstitial) pH (pHe) of solid tumours is significantly more acidiccompared to normal tissues. In-vitro, low pH reduces the uptake of weakly basic chemotherapeutic drugs and, hence, reduces their cytotoxicity. This phenomenon has been postulated to contribute to a ‘physiological’ resistance to weakly basic drugs in vivo. Doxorubicin is a weak base chemotherapeutic agent that is commonly used in combination chemotherapy to clinically treat breast cancers. This report demonstrates that MCF-7 human breast cancer cells in vitro are more susceptible to doxorubicin toxicity at pH 7.4, compared to pH 6.8. Furthermore 31P-magnetic resonance spectroscopy (MRS) has shown that the pHe of MCF-7 human breast cancer xenografts can be effectively and significantly raised with sodium bicarbonate in drinking water. The bicarbonate-induced extracellular alkalinization leads to significant improvements in the therapeutic effectiveness of doxorubicin against MCF-7 xenografts in vivo. Although physiological resistance to weakly basic chemotherapeutics is well-documented in vitro and in theory, these data represent the first in vivo demonstration of this important phenomenon.
One of the 3 golf ball sized lumps in my breast that disappeared after changing to a pH alkaline diet.
“Do you have a health condition you think is incurable? Do you want to lose weight and keep it off permanently? Do you want to reverse aging? Do you do everything you can to be healthy but still don’t feel quite right? The alkaline diet could cure all this and more; but is it too good to be true?”
“The alkaline diet is quickly becoming popular with backing of celebrates like Kate Moss, Gwyneth Paltrow, Jennifer Aniston, Linda Gray, Bill Clinton, Larry Hagman, and Kirsten Dunst. In 2003 Cris Carr, former Budweiser girl, made a move documentary on her battle with cancer and how she reversed the cancer with an alkaline diet. You may have heard about the alkaline diet on the news or in one of several interviews on the Oprah Winery show. You can find testimonies of people all over the internet that completely reversed every day illnesses as well as cancer, HIV MS, diabetes type1&2, and other chronic diseases.”
“How does it work? The alkaline diet works on the premise that our bodies are self healing. In order for the body to heal itself it needs the right tools one being the correct pH, others being sufficient nutrients, water, and exercise. The main thing that affects our pH is our diets. By eating alkalizing foods and minimizing acidic foods our bodies can begin to heal, prevent sickness, and help protect from external acid factors like stress and radiation. To maintain a good pH in our bodies we need to eat at least 70% alkaline foods and no more than 30% mildly acidic foods. Alkaline foods include most cooked and raw vegetables, some beans, and few fruits, grains, & nuts. Acidic foods include meat, dairy, sugar, processed foods, coffee, and most fruits, grains, and nuts.”
“Sound too hard? Well, you don’t have to jump right in. Most people have better results by making slow gradual changes to their diet. Some people only need to make a couple of small changes to start seeing results. There are also many tasty alkaline versions of acidic foods; so don’t worry about felling deprived.”
“So does the alkaline lifestyle and diet really work? Apparently it does from all the testimonies on the internet. I tried it myself in 2006 when I found out I had three breast tumors that my doctor told me had to be surgically removed. Within six months the cancerous tumors were gone, and so were my allergies, chronic knee & back pain, and my problem with vertigo that my doctors could not explain or treat. I also have more energy and I don’t get colds anymore. I have been on the pH alkaline diet since 2006 and continue to maintain excellent health. I have met many people that have completely reversed their health problems with the pH alkaline diet. I also know a couple of people that it did not work completely for but it did drastically improve their health. Many people give up on alkalizing before it has a chance to work because they feel deprived. They think they can only eat salad; but this is not true.”
“Supplementation is also important as there are some vitamins and minerals than can be hard to get on an alkaline diet. There are also many supplements that can make alkalizing quicker and easier. The pH alkaline diet can be hard and take a long time to get results if you don’t know enough about it. So it is best to read up on it and get a good pH coach. There is very little clinical research on the pH alkaline diet and its effects on specific disease conditions. However, an article published in PubMed says there supporting research that shows the pH alkaline diet can support health and reverse disease but more research is needed http://www.ncbi.nlm.nih.gov/pubmed/22013455.”
“It will be many years before clinical research can be done on the pH alkaline diet with every health problem. So it is best to consult a health professional before changing your diet especially if you have a chronic disease.”
“Some health problems with supporting clinical studies on the alkaline diet & treatments include cancer, low back pain, bone loss, and increased lean tissue mass in older adults:”
“In a study published in PubMed a high pH treatment was tested on over 30 humans with cancer. In each case the cancer disappeared. http://www.ncbi.nlm.nih.gov/pubmed?term=6522424”
Supplementation with alkaline minerals reduces symptoms in patients with chronic lower back pain. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3195546/?tool=pubmed”
“Increasing the alkaline content of the diet may slow bone loss in healthy older adults. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2630872/”
“Alkaline diets favor lean tissue mass in older adults. http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2597402/”
Have you heard about the ravages of acid rain in Australia and the loss of the coral reef or in Alaska and the loss of millions of pine trees or maybe you have heard about the oceans and the pH dropping because of acid rain. The cause is the result of toxic acidic carbon emissions in the global environment. Acid rain damages the leaves and needles on trees, reduces a tree’s ability to withstand cold, drought, disease and pests, and even inhibits or prevents plant reproduction. The oceans of the World are dying because of acidic carbon emissions from cars and cows. In an effort for the Earth and the oceans to stay alive and combat increased acidic pollution, as tree roots pull important nutrients such as calcium and magnesium from the soil and calcium and the oceans are pulling calcium and magnesium from the coral reefs and sodium from the ocean water increasing acidity. The extraction of alkaline minerals from the soil and water is necessary for all living things on the earth and oceans to stay alive and avoid sudden death. These alkaline nutrients help to balance the increased effects of acid rain, but as they become depleted from the soil or from the ocean, the trees’ and marine life’s ability to survive is strained and placed in certain danger of extinction. Just look at the pictures below and see what is happening to the forests of Denali, Alaska and the great barrier reef in Queensland, Australia. The forests in Alaska and the great barrier reef in Queensland, Australia are both headed towards irreversible extinction because of acid rain.
What if I told you that most ALL people living today are unknowingly doing similar things to their body? A highly acidic lifestyle and diet is like acid rain in our blood, interstitial fluids and intracellular fluids that constitutes over 65% of the whole body. While the body has an alkaline buffering system (headed up by the stomach) in place to ensure that the blood and the interstitial fluids stay slightly alkaline at 7.365 pH, the depletion of alkaline minerals from the bones, muscles and other parts of your body may leave YOU vulnerable to health issues leading to ALL sickness and disease.
The pH (potential of hydrogen) is the measurement of acid (a measurement of hydrogen ions or protons) or alkalinity (a measurement of reduced hydrogen or electrons) on a scale from 0 to 14 with a midpoint of 7. The lower the number the higher the acidity (or the greater the concentration of hydrogen ions or protons) based upon a logarithm to the power of negative 10! For example, the pH of a healthy ocean environment free from acid rain would be 8.350. If the ocean pH drops 1 point due to acid rain to a pH of 7.350, which is a 10 times drop in pH, all life as we know it in the oceans would die. In fact, if the ocean pH drops from 8.350 to 8.100, which is a .235 drop, ALL life in the oceans would die! That is all it takes for ALL marine life to cease in our Oceans! JUST a small drop of 2/10’s of 1 point for ALL life to end! Here is another very important example that I truly want you to understand. The healthy pH of the human blood and interstitial fluids which makes up 80 percent of ALL body fluids is 7.365. This pH of the blood and interstitial fluids is a dynamic and is always changing. How do I know this? Because Dr. Galina Migalko, MD, NMD and I are the only scientist in the World measuring and comparing the pH and chemistries of the blood against the pH and chemistries of the interstitium. This is critical to truly understand when you are moving toward metabolic alkalosis or metabolic acidosis and preventing and/or reversing any sickness and disease as well as determining the efficacy of any non-invasive or invasive treatments. In other words, are the treatments for any sickness and disease making you sicker or better, whether conventional or traditional? This can now be measured and determined with certainty.
So why does the body, primarily the stomach work so hard to maintain the delicate pH of the blood and interstitial fluids of the interstitium? Here is the most important answer YOU will read in YOUR life! If the blood and interstitial fluids drop below 7.100 from the ideal healthy pH of 7.365 you would go into a coma. When the blood and interstitial fluid pH drops to 6.900 you are DEAD! From what? Not global warming but from body warming or in other words acidosis! The key to avoid death is to maintain the alkaline design of the blood and interstitial fluids at a precise pH of 7.365 which can be measured without drawing one drop of blood or interstitial fluid. The technology is here and the science is real!
This is the common denominator for ALL sickness and disease – ALL sickness and disease are caused by acidosis or acid rain or body warming! Therefore, there are NO specific diseases, there are ONLY specific disease or sickness conditions. All sickness and disease is caused by acid rain from within and is exactly what is happening in the oceans, the soils of our planet and in all humanity. Planetary and human sickness and disease is on the rise because of personal acidic lifestyles and dietary choices and because of ignorance. Name any disease and that disease or sickness is caused by metabolic, respiratory, gastrointestinal or environmental acidosis.
Check out this YouTube video on the 7 signs YOU and TOO Acidic
I hope you can see NOW how important it is to understand and then monitor your pH daily by having your your blood and interstitial fluids tested. Unfortunately, this new science and technology for testing the pH of the blood and interstitial fluids is limited Worldwide. (For more information concerning the testing of the blood and interstitial fluids or to make an appointment email: email@example.com) In the meantime, there is a simple, inexpensive and noninvasive way for testing the fluids of the interstitium, but not of the blood, for those of you who desire to monitor your interstitial fluid pH daily. You can test the pH of the morning urine, since this urine is a product of the interstitium and NOT of the blood, by using special pHydrion strips (www.phoreveryoung.com). When you measure the pH of your urine using these special pHydrion strips it is important to achieve each morning a pH of at least 7.300 by following the suggested lifestyle and diet as described below. When you are testing your morning urine, which is the most acidic time of the day, you are testing the pH of the interstitial fluids which makes up over 60 percent of the body fluids (25 liters). You can also test your saliva using the same special pHydrion strips. When you are testing your saliva pH you are testing your body reserves available for buffering acid rain. Both the urine and saliva pH should be at least 7.300 and must be tested daily as you follow the pH Miracle alkaline lifestyle and diet in order to achieve an ideal pH for “Perfect Health!”
An acidic pH of the blood and then interstitial fluids is what causes acid reflux—a condition in which the stomach creates when it is trying to buffer dietary acids from your toxic acidic food or drink ingested or metabolic acids from all functions of the body or respiratory acids from your respiratory system to maintain the pH of the blood and interstitial fluids at a delicate pH of 7.365. The following is the stomach chemistry as it creates sodium bicarbonate to buffer excess acid rain on your blood, interstitial fluids and intercellular fluids: H20 (water) + NaCl (salt) + C02 (carbon dioxide) = NaHC03 (sodium bicarbonate) + HCL (hydrochloric acid).
This may be the first time you have ever heard this, but I have been saying this for many years, “the stomach DOES NOT DIGEST FOOD it ALKALIZES FOOD and protects ALL of our body fluids, organs and tissues from dietary, metabolic, respiratory and environmental acidosis! In other words, the stomach is an organ of contribution and NOT an organ of digestion. Eat any food without chewing it, like a piece of corn and see what happens. The corn comes out of your anus the same way it went into your mouth. The stomach digests nothing. The hydrochloric acid in your stomach is a waste product of sodium bicarbonate production for buffering acid rain or acidic waste from what you eat, what you drink, what you breath and what you think. This is why when an athlete goes into lactic acidosis they throw-up to rid their body of all the hydrochloric acid build-up in the gastric pits of the stomach. You see the body is working hard to buffer the increased lactic acid from increased metabolism so the athlete doesn’t die from acidic rain from a declining pH in the blood and interstitium. Even when a pregnant woman throws-up (generally in her first trimester) her stomach is producing sodium bicarbonate to buffer the acidic loads in her and her unborn child’s blood and interstitium. The increased need for alkalinity during pregnancy is significant and is NOT understood or even considered by medical savants. They think, unknowingly that the body just takes care of the pH of the blood and tissues and that what you eat, what you drink, what you breath, and what you think cannot effect this delicate pH balance. You see, morning sickness is nothing more than increased acids from diet, respiration and metabolism! It requires twice the energy to make a baby and with that the pregnant Mother has increased acid rain. So I want you to understand that the stomach’s main purpose is to maintain the alkaline design of the body to keep it alive. That is IT! Get IT?
To learn more about the physiology of the stomach read the following book. You can order this book online at the following link: http://www.drrobertyoung.com
a) The parietal or cover cells of the stomach split the sodium chloride of the blood. The sodium is used to bind with water and carbon dioxide to form the alkaline salt, sodium bicarbonate or NaHCO3. The biochemistry is: H20 + CO2 + NaCl = NaHCO3 + HCL. This is why I call the stomach an alkalizing organ NOT an organ of digestion. The stomach DOES NOT digest the food or liquids you ingest it alkalizes the food and liquid you ingest.
b) For each molecule of sodium bicarbonate (NaHCO3) made, a molecule of hydrochloric acid (HCL) is made and secreted into the so-called digestive system – specifically, the stomach (the gastric pits in the stomach) – to be eliminated. Therefore HCL is an acidic waste product of sodium bicarbonate production created by the stomach to alkalize the food and liquids ingested and to maintain the delicate pH of the blood and interstitial fluids at a pH of 7.365.
c) The chloride ion from the sodium chloride (salt) binds to an acid or proton forming HCL as a waste product of sodium bicarbonate production. HCL has a pH of 1 and is highly toxic to the body and the cause of indigestion, acid reflux, ulcers and cancer. In fact HCL is in all pharmaceuticals and most dietary nutritional supplements.
d) When large amounts of acids, including HCL, enter the stomach from a rich animal protein or dairy product meal, such as meat and cheese, acid is withdrawn from the acid-base household. The organism would die if the resulting alkalosis – or NaHCO3 (base flood) or base surplus – created by the stomach was not taken up by the alkalophile glands (pancreas, gallbladder, Lieberkuhn glands in the liver and the Brunner glands between the pylorus and the junctions of the bile and pancreatic ducts), that need these quick bases in order to build up their strong sodium bicarbonate secretions. These glands and organs, once again are the stomach, pancreas, Brunner’s glands (between the pylorus and the junctions of the bile and pancreatic ducts, Lieberkuhn’s glands in the liver and its bile with its strong acid binding capabilities which it has to release on the highly acidic meat and cheese to buffer its strong acids of nitric, sulphuric, phosphoric, uric and lactic acids.
e) When a rich animal protein and dairy product meal is ingested, the stomach begins to manufacture and secrete sodium bicarbonate (NHCO3) to alkalize the acids from the food ingested. This causes a loss in the alkaline reserves and an increase in acid and/or HCL found in the gastric pits of the stomach. These acids and/or HCL are taken up by the blood which lowers blood plasma pH. The blood eliminates this increase in gastrointestinal acid by throwing it off into the Pishinger’s spaces or what recent scientist are calling the Interstitium pictured below.
f) The space enclosed by these finer and finer fibers is called the Pishinger’s space, or the spaces of the interstitium that contains the fluids that bath and feed each and every cell while carrying away the acidic waste from those same cells. There is no mention of this organ in American physiology or medical school text books. There is mention of the space but not of any organ that stores acids from metabolism, respiration, environment and diet, like the kidney. I call this organ the “pre-kidney” because it stores metabolic respiratory, environmental and gastrointestinal acids until they can be buffered and eliminated via the skin, urinary tract, or bowels.
g) After a rich animal protein or dairy product meal, the urine pH becomes alkaline.The ingestion of meat and cheese causes a reaction in acidic fashion in the organism by the production of sulfuric, phosphoric, nitric, uric, lactic, acetylaldehyde and ethanol acids, respectively, but also through the formation and excretion of base in the urine. Therefore eating meat and cheese causes a double loss of bases leading to tissue acidosis and eventual disease, especially inflammation and degenerative diseases.
h) During heavy exercise, if the the resulting lactic acid was not adsorbed by the collagen fibers, the specific acid catchers of the body, the organism would die. The total collection of these fibers is the largest organ of the body called SCHADE, the colloidal connective tissue organ or the interstitium. NO liquid exchange occurs between the blood and the parenchyma cells, or in reverse, unless it passes through this connective tissue organ or the interstitium. This organ connects and holds everything in our bodies in place. This organ is composed of ligaments, tendons, sinew, and the finer fibers that become the scaffolding that holds every single cell in our bodies in place. When acids are stored in this organ (just discovered by American science in 2018. Dr. Robert O. Young with Dr. Galina Migalko published their pH findings of the blood, interstitial fluids of the Interstitium and the intracellular fluids in 2015. Their publication is pictured below), which includes the muscles, inflammation and pain develop. The production of lactic acid is increased with the ingestion of milk, cheese, yogurt, butter and especially ice cream.
That is why I have stated for years, “acid is pain and pain is acid.” You cannot have one without the other. This is the beginning of latent tissue acidosis leading to irritation, inflammation and degeneration of the cells, tissues and organs.
i) The more acidity created from eating meat, cheese, milk or ice cream the more gastrointestinal acids are adsorbed into the the collagen fibers to be neutralized and the less sodium bicarbonate or NaHCO3 that is taken up by the alkalophile glands. The larger the potential difference between the adsorbed acids and the amount of NaHCO3 generated with each meal, the more or less alkaline are the alkalophile glands like the pancreas, gallbladder, pylorus glands, blood, etc. The acid binding power of the connective tissue, the blood, and the alkalophile glands depends on its alkali reserve, which can be determined through blood, urine, and saliva pH testing, including live and dried blood analysis. (Currently we are the only two scientist in the World that are doing non-invasive testing of the stomach, blood, interstitium and intracellular fluid pH with results in less than 15 minutes) The saliva pH is an indication of alkali reserves in the alkalophile glands and the urine pH is an indication of the pH of the fluids that surround the cells or the Pishinger’s space.
j) The iso-structure of the blood maintains the pH of the blood by pushing off gastrointestinal or metabolic acids into the connective tissue or the Pishinger’s space or the Interstitium. The blood gives to the urine the same amount of acid that it receives from the tissues and liver so it can retain its iso-form. A base deficiency is always related to the deterioration of the deposit ability of the connective tissues or the Pishinger’s space or interstitial fluid spaces. As long as the iso-structure of the blood is maintained, the urine – which originates from the blood – remains a faithful reflected image of the acid-base regulation, not of the blood, but of the tissues. The urine therefore is an excretion product of the connective tissues or the interstitium, not the blood. So when you are testing the pH of your urine, you are testing the pH of the tissues or the interstitial fluids of the Interstitium.
k) A latent “acidosis” is the condition that exists when there are not enough bases in the alkalophile glands because they have been used up in the process of neutralizing the acids adsorbed to the collagen fibers. This leads to compensated “acidosis.” This means the blood pH has not changed but other body systems have changed. This can then lead to decompensated “acidosis” where the alkaline reserves of the blood are used up and the pH of the blood is altered. Decompensated “acidosis” can be determined by testing the blood pH, urine pH and the saliva pH. The decrease in the alkaline reserves in the body occurs because of hyper-proteinization, (eating Meat and Cheese!)or too much protein, and hyper-carbonization, or too much sugar. This is why 80 to 90 year old folks are all shrunk up and look like prunes. They have very little or no alkaline reserves in their alkalophile glands. When all the alkaline minerals are gone, so are you and your battery runs down. The charge of your cellular battery can be measured by testing the ORP or the oxidative reduction potential of the blood, urine or saliva using an ORP meter. As you become more acidic this energy potential or ORP increases.
l) If there is not enough base left over after meat and cheese or surgary meal, or enough base to neutralize and clear the acids stored in the connective tissues or interstitium, a relative base deficiency develops which leads to latent tissue acidosis.When this happens the liver and pancreas are deficient of adequate alkaline juices to ensure proper alkalization of the food in your stomach and small intestine.
m) Digestion or alkalization cannot proceed without enough of these alkaline juices for the liver and pancreas, etc., and so the stomach has to produce more acid in order to make enough base, ad nauseam, and one can develop indigestion, nausea, acid reflux, GERD, ulcers, esophageal cancer and stomach cancer. All of these symptoms are not the result of too much acid or HCL in the stomach. On the contrary, it is the result of too little base in the form of sodium bicarbonate!
n) Therefore the stomach is NOT an organ of digestion as currently taught in ALL biology and medical texts, BUT an organ of contribution or deposit. It’s function is to deposit alkaline juices to the stomach to alkalize the food and to the blood to carry to the alklophile glands!!!!
o) There is a daily rhythm to this acid base ebb and flow of the fluids of the body. The stored acids are mobilized from the connective tissues and Pishinger’s spaces or the spaces of the interstitium while we sleep.
These acids reach their maximum (base tide) concentration in this fluid, and thereby the urine (around 2 a.m. is the most acidic). The acid content of the urine directly reflects the acid content of the fluid in the Pishinger’s spaces, the interstitial fluid compartments of the body. On the other hand, the Pishinger’s spaces become most alkaline around 2 p.m. (the base flood) as then the most sodium bicarbonate (NaHCO3) is being generated by the cover cells of the stomach to alkalize the food and drink we have ingested.
p) If your urine is not alkaline by 2 p.m. you are definitely in an ACIDIC condition and lacking in alkaline reserves. The pH of the urine should run between 6.8 and 8.4 but ideally 7.2 or greater.
q) After a high protein meal or meat or cheese, the free acids formed such as sulfuric, phosphoric, uric, and nitric acids stick to the collagen fibers to remove them from the blood and protect the delicate pH of the blood at 7.365. The H+ or proton ions from these acids are neutralized by the next base flood, the sodium bicarbonate produced after the meal. The H+ or proton ion combines with the carbonate or HCO3, converts to carbonic acid, H2CO3, which converts to CO2 and H2O. The sulfuric and other acids from proteins are neutralized as follows where the HR represents any acid with the R as its acid radical (SO4, PO4, or NO3) HR + NaHCO3 <=> H2O + NaR (Ca, Mg, K)+ CO2.
r) Medical doctors are not taught the above science in medical school and therefore do not understand the complex chemistry between the stomach, blood and interstitium or even recognize the effects of an acidic lifestyle and diet leading to latent tissue acidosis in the largest organ of the body called the Interstitium. They understand and recognize compensated acidosis and decompensated acidosis in the blood but do not know about or even understand a single thing about the Interstitium. In compensated acidosis, breathing increases in order to blow off more carbonic acid which decreases PCO2 because of the lowered carbonate or HCO3. When the breathing rate can no longer get any faster and when the kidneys can no longer increase its’ function to keep up with the acid load, then the blood pH starts to change from a pH of 7.365 to 7.3 then to 7.2. At a blood pH of 6.95 the heart relaxes and the client goes into a coma or dies.
s) Metabolism of a normal adult diet results in the generation of 50 to 100 meq of H+ or proton per day, which must be excreted if the urine acid-base balance is to be maintained. A meq is a milliequivalent which is an expression of concentration of substance per liter of solution, calculated by dividing the concentration in milligrams per 100 milliliters by the molecular weight. This process involves two basis steps; 1) the reabsorption of the filtered sodium bicarbonate or NaHCO3 and, 2) excretion of the 50 to 100 meq of H+ or proton produced each day by the formation of titratable acidity and NH4+ or ammonium. Both steps involve H+ or proton secretion from the cells of the kidney into the urine.
t) Sodium bicarbonate (NaHCO3) must be reabsorbed into the blood stream, since the loss of NaHCO3 will increase the net acid load and lower the plasma NaHCO3 concentration. The loss of NaHCO3 in the urine is equivalent to the addition of H+ to the body since both are derived from the dissociation of H2CO3 or carbonic acid.
u) The biochemistry is: CO2 + H2O = H2CO3 = HCO3 + H+. The normal subject must reabsorb 4300 meq of NaHCO3 each day! The secreted H+ or proton ions are generated within the kidney cells from the dissociation of H2O or water. This process also results in the equimolar production OH- or hydroxyl ions. The OH- ions bind to the active zinc-containing site of the intracellular carbonic anhydrase; they then combine with CO2 to form HCO3- ions which are released back into the kidney cells and returned to the systemic circulation. Second, the dietary acid load is excreted by the secretion of H+ or proton ions from the kidney cells into the urine. These H+ or proton ions can do one of two things: the H+ or proton ions can be combined with the urinary buffers, particularly HPO4, in a process called titratable acidity (The biochemistry is: H+ + HPO4 = H2PO4), or the phosphate buffering system or the H+ or proton ions can combine with ammonia (NH3) to form ammonium as follows: NH3 + H+ = NH4.
v) This ammonia is trapped and concentrated in the kidney as ammonium which is then excreted in the urine.
w) In response to acid load, 36% of the H+ or proton goes intracellular in exchange for the release of Na+ (sodium) into the blood stream. 15% of the acid goes intracellular in exchange for K+ (potassium) – common in diabetics. 6% of the H+ or proton or acid goes directly into the cell to be buffered by intracellular processes. 43% is buffered by the interstitium as NaHCO3- or sodium bicarbonate combining with H+ or proton to form H2CO3 or carbonic acid which breaks down to CO2 or carbon dioxide to be released by the lungs. 10% of CO2 or carbon dioxide is excreted through the lungs and 90% is used by the body to reabsorb alkaline minerals and make sodium bicarbonate for buffering gastrointestinal, respiratory, enivronmenta and metabolic acids.
The biochemistry is: CO2 + H2O = H2CO3 = HCO3 + H+.
You can order the following book on sodium and potassium bicarbonate at: http://www.phoreveryoung.com or https://www.amazon.com/gp/product/B01JLHJ1Y8/ref=dbs_a_def_rwt_hsch_vapi_taft_p3_i9
x) Of all the ways the body can buffer metabolic and dietary acids, the excretion of protein (the eating of meat and cheese) generated acid residues is the only process that does not add sodium bicarbonate back into blood circulation. This creates a loss of bases which is the forerunner of all sickness and disease. In the long run, the only way to replace these lost bases is by eating more alkaline electron-rich green foods and long-chain polyunsaturated fats. Eating meat and cheese is definitely hazardous to your health. That is why I say, “a cucumber a day keeps the doctor away while eating meat, cheese and even an apple creates more excess acid in the colloidal connective tissues of the Schade or the Interstitium, leading to latent tissue acidosis and then sickness, disease and finally death.
y) With over 30 years of research and testing over 500,000 samples of blood and over 1,000,000 samples of urine and saliva I have come to the conclusion that the Human Body is an acid producing organism by function – yet, it is an alkaline organism by design. Eating animal protein, especially meat and cheese and sugar from any source are deadly acidic choices – unless you interested in becoming sick, tired and fat over time.
z) Bottom line – the pH Miracle Lifestyle and Diet is a program that focuses on the foundational principal that the body is alkaline by design and yet acidic by function. These are my two greatest discoveries. This make this program the ultimate program for preventing and reversing aging and the onset of sickness and dis-ease. I would say that the pH Miracle Lifestyle and Diet is the diet for a longer healthier life free from all sickness and disease. That is why you are seeing a slew of celebrities (Harry and Meghan, Tom Brady, Rhianna, Elle Macpherson, Gwyneth Paltrow, David Beckham, NeNe, Tony Robbins, just to name a few) can attest to the benefits of a pH Miracle alkaline lifestyle and diet and the drinking of alkaline water for improving the quality of their skin, hair and body and to avert over-acidity which often leads to breakouts of the skin and many other health challenges.
My research has linked acidity to every sickness and disease, including enervation, irritation, catarrh, inflammation, induration, ulceration and degeneration. People do not die from disease they die from the inability to maintain the alkaline design of their body. The key to living a long and healthy life is managing the alkaline design of the body. For example pain equals acid and acid equals pain. You cannot have pain with acid. It is that simple! Remove the acid and you remove the pain.
The following are 20 suggestions on how to manage the alkaline design of your body and to increase your energy, vitality and quantity and quantity of life which is in your complete control! YOU determine YOUR Destiny!
1. Start your day with a large glass of 9.5 alkaline water with the juice of a whole, freshly-squeezed lemon. While lemons are wrongly considered acidic, they are NOT! They are loaded with sodium bicarbonate which means they contribute to your alkaline reserves and protect the blood and interstitium from acid rain.
Get weekly alkaline tips of the day for leading a long and healthy and compassionate alkaline life when you sign-up as a member of our pH Miracle Fan Club on our facebook page at: https://www.facebook.com/groups/50864627953/
2. Better yet, invest in a water filtration system that alkalinizes the water and increases the pH of the water to a 9.5 or greater. Pure water found in nature, which is hard to come by now thanks to acid rain, is quite alkaline. If you’re already drinking purified water, you can also purchase water alkalinizing drops to add to your water bottle and to raise the pH of your water to pH or 9.5 or greater. Here is the link to purchase alkaline pH drops for you water: https://store.phoreveryoung.com/collections/supplements/products/activator-by-ph-miracle-2-fl-oz-59-14ml
3. Eat a large green vegetable salad tossed in alkalizing lemon juice and olive oil. Greens are among the best sources of alkaline minerals like calcium and are high in chlorophyll for building hemoglobin and red blood cell counts.
4. Drink raw organic almond milk. Almonds are packed with natural alkaline minerals like calcium, magnesium and potassium which can help to balance out acidity while buffering another acid called glucose or blood sugar.
5. Drink an Avocado smoothie daily. Using a Vita-mix blender you can blend an avocado with spinach greens, cucumber, celery, ginger and almond milk for an incredible alkalizing and energizing green shake.
6. Add green powder like wheat grass, barley grass, moringa grass or other greens to your daily diet since these foods that are highly alkalizing and energizing. It’s easy to throw a tablespoon of these greens into your Avocado based almond milk smoothie. To order the best green powder in the World go to: https://store.phoreveryoung.com/collections/supplements/products/innerlight-supergreens
7. Take a brisk walk, bicycle ride, swim, rebound or some other exercise for at least 30 minutes everyday. Exercise helps move acidic waste products out of the interstitium and through the pores of the skin via perspiration.
8. Breathe deeply. Ideally, choose a spot that has fresh, oxygen-rich air. And, sorry, air filled with Febreze, Glade and all the other so-called “air fresheners,” is not what I’m talking about here. Take a deep breath in through your nose and then switch to breathing through your mouth without letting go of your first inhalation through your nose.
9. Go for Meatless and Eggless Mondays. Better yet, opt for meat-free Tuesdays, Wednesdays and other days throughout the week. During the chewing of meat, acid residues like uric acid, nitric acid, sulphuric acid and phosphoric acid residues are left behind for the stomach to address. There is zero health benefits from eating the flesh of another living being. All flesh is acidic and causes a double-loss of alkalinity in the blood and interstitium.
10. Skip the sugar-laden soda and drink some iJuice Wheat Grass Juice.(www.ijuicenow.com) Sugar is one of the most acidic foods we consume. Sugar is a waste product of metabolism and fermentation. You need over 30 glasses of alkaline water at a pH of 8.4 just to neutralize the acidity (sugar and carbonic acid) of ONE can or bottle of soda.
11. Skip the artificially-sweetened diet beverages and other diet products. They contain artificial sweeteners like aspartame (now known as NeoTame), sucralose (also known as Splenda) or saccharin (also known as SugarTwin) and they all cause body warming and acid rain inside your body.
12. Add more green fruit and vegetables to your diet. No, fried potatoes don’t count, including sweet potatoes. Asparagus, green peppers, green string beans, kale, spinach, beet tops, carrot tops, wheat grass, barley grass, broccoli, cucumber, avocado, and lime and other green fruit and vegetables are also excellent choices for supporting the alkaline design of the body.
13. Instead of slathering your vegetables in acid-forming butter, drizzle alkaline flaxseed oil, hemp seed oil, and/or green olive oil over them.
14. Sprout it out. Add more sprouts to your daily diet like bean sprouts, sunflower seed sprouts and broccoli sprouts. They are extremely alkalizing and supercharged with nutrients and energy-boosting electrons.
15. Skip ALL desserts or reserve them as occasional treats instead of daily habits. Sugar consumption has been linked to a whole host of health problems and is best minimized or eliminated. If you are in body warming then removing all acidic foods and drinks are a must.
16. Avoid all alcoholic beverages or so-called nutritional supplements that contain alcohol. Alcohol is a devastating acid that causes pancreatic and liver cancer.
17, Avoid corn and peanuts because they are loaded with bacteria, yeast and mold and the cancer causing acid lactic acid.
18. No acidic beverages like coffee, black or green tea or chocolate. They all contain food acids that robs your body of its alkaline reserves causing many diseases, including cancer.
19. Stay far away from vinegar. Vinegar is pure acid and steals years off your life! Do not believe the so-called health experts to state the vinegar is good for digestion. Remember this very important point. There is only one instrument in the human body that can digest or breakdown food and the is your teeth. When you pour vinegar into your body all you have done is poison yourself. The stomach has to rob alkalinity from the blood, interstitium, organs and glands to buffer this highly toxic chemical setting the stage for enervation, inflammation, induration, ulceration , degeneration and finally death. Vinegar is death in a bottle.
20. Test your urine and saliva and drink pHour Salts every morning. Your ideal pH of your urine and saliva should be at least 7.300. If your pH is lower than 7.300 take a scoop of pHour salts in a small glass of alkaline water. Ideally, you should drink a glass of phour salts which contains sodium bicarbonate, potassium bicarbonate, magnesium chloride and calcium at least 3 times daily. To order pHour salts go to: https://store.phoreveryoung.com/collections/supplements/products/phour-salts-per-case
You can also order saliva and urine testing strips at the following link: https://store.phoreveryoung.com/products/phydrion-strips-5-5-8-0?variant=2085775876
To learn more about the work, research and discoveries of Robert O Young go to the following websites: http://www.drrobertyoung.com, http://www.phmiracleretreat.com, http://www.ijuicenow.com, http://www.innerlightblue.com and http://www.phmiracleproducts.com
To learn more read The pH Miracle, The pH Miracle revised and updated, The pH Miracle for Diabetes, The pH Miracle for Weight Loss, The pH Miracle for Cancer and Sick and Tired, just to name a few of Robert O Young’s published books. To order any of these books go to: http://www.phoreveryoung.com
Come listen and learn from Key Note Speakers, Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner and Galina Migalko MSc, MD, NMD, in four different countries around the World as they lecture on non-invasive medical diagnostics, the interstitium, pH, nutrition and their break-through research on prevention and non-invasive treatments for cancer, diabetes, heart disease, arthritis, osteoporosis, lupus, multiple sclerosis, infections, and many more acidic-caused diseases.
To pre-register for one or more World Conferences please email firstname.lastname@example.org and receive an additional 10 to 20 percent discount on the listed early-bird pricing. You can also register by phone by calling 760 484 1075.
When you enroll in one of our Conferences you will receive a credit for a live and dried blood cell analysis, valued at 1200 euros.
Please check out the Countries, Cities, Dates and Pricing below!
For decades now, many scientists have been raising red flags that chemotherapy can oftentimes do more harm than good, and in a surprisingly large number of cases, it simply does not work.
Dr. Hardin B. Jones, a former Professor of Medical Physics and Physiology at Berkeley, California, studied the impact of chemotherapy, radiation, and surgery on the survival rates of cancer patients, and found that instead of prolonging lives, these treatments actually make the patients die almost four times sooner. This was found more than 40 years ago, and yet not much has changed in the way the hospitals treat cancer.
For terminal patients as well, a 2016 study in the peer-reviewed journal of the American Cancer Society CANCER found that living at home instead of being treated at the hospital prolonged their lives by about 45%.
And now, a new study was just published earlier this month that analyzed 30-day mortality rates caused by the treatment itself: chemotherapy and chemotherapy plus radiation.
The study, published in The Lancet’s Oncology, looked at 23,228 breast and 9,634 lung cancer patients in England.
The results showed high death rates linked to the treatment, increased use of SACTs (systematic anticancer therapies – cytotoxic chemotherapy). The researchers stated it was due “poor clinical decision making.”
“Patients dying within 30 days after beginning treatment with SACT are unlikely to have gained the survival or palliative benefits of the treatment, and in view of the side-effects sometimes caused by SACT, are more likely to have suffered harm,” states the study.
The researchers write that while there a few patients who may have benefited from SACTs, there were too many who were harmed by it, or even killed by the treatment.
In 2014 , the year the study was researched, almost 1,400 patients in England died within 30 days of their first chemotherapy treatment. In some hospitals, the mortality rate was significantly higher than in the others: up to 51% of breast cancer patients died in Milton Keynes (although the number of total patients was small), and up to 29% of lung cancer patients died in Lancashire Teaching Hospitals.
“Simply reducing doses of or avoiding SACT altogether would reduce or eliminate instances of treatment-related early mortality.”
The study was done after noticing a clear lack of data analyzing the risk versus gain of using chemotherapy and mortality rates caused by it in the first 30 days of treatment. As the researchers state, this is the first time this topic has been brought up and investigated at a national level.
The “million dollar questions” (or perhaps “billion dollar” since this is the cancer treatment industry we’re talking about) raised by the researchers included: is chemotherapy use still advocated based on small clinical studies — most sponsored by the pharmaceutical industry?
And it has never been fully studied how well it works in a real hospital setting?
The study points out two problems. One is that mortality rate is high due to poor clinical decisions. Perhaps because in most hospitals chemotherapy is given in a one-size-fits all manner or a fixed-dose with no consideration to a patient’s health history, and characteristics such as weight and age.
In England’s database, it was found that for many patients, the doctors did not include why the chemotherapy was given, and the overall health of the patient and the severity of their cancers was never recorded.
Both of these factors, as the study points out, could significantly change the outcome for these patients.
In America, another study found that doctors gave palliative chemotherapy to terminal cancer patients, and in at least two-third of cases, the patient did not know that the treatment could not cure them, but it only alleviated some symptoms, such as pain.
Knowing that the treatment could not help them in the end, would they have searched for alternative options that may have worked better?
The second issue the study found is that mortality rates are hard to analyze because they are not well or properly recorded.
In England’s case, many dates of deaths were simply missing from the national database. Some were documented twice, and the two dates did not match.
In America, the national statistic of cancer mortality comes from the death certificates. This is what the National Center for Health Statistics (NCHS) along with the doctors use to see how many people are die from cancer, and how many people die from the treatment. Unfortunately, the National Cancer Institute reports “cancer” as the cause of death for almost all cancer patients, regardless of what actually caused it, as pointed out in a 2002 article.
This article raised concern that “cancer death rates are systematically underestimated, in that many patients who die as a result of cancer treatment do not have cancer recorded as the underlying cause of death.”
For example, they studied patients who died within one month after a cancer-surgery between 1994 and 1998, and 41% of these deaths were not properly recorded. The authors write that cancer treatment was the likely cause of death.
“…Many deaths subsequent to 1 month after cancer-directed surgery may be similarly miscoded.”
Unfortunately, even though the study points out that many death certificates quote some condition other than cancer as the cause of death (such as liver failure), they want the cause of death to be just “cancer.” That would lead to further incorrect data as many patients do die from cancer treatments, and liver failure that would not happen from cancer, is often caused by the toxic chemo drugs. But what this study does show us, is that because of a 1999 revision in the International Statistical Classification of Diseases and Related Health Problems (ICD), instead of a cancer or cancer-related death, death certificates now have to show the “underlying” cause of each death, such as: thromboembolism (blood vessel obstruction), infections, organ failures, and hemorrhage (excessive bleeding).This leads to improper classification and underestimated cancer death rates and statistics.
That is hard to say until every doctor and hospital begins classifying the causes of deaths correctly. Until then, it is important to keep studies like this in mind when considering pros and cons of starting a chemotherapy treatment.
There are more and more alternative options out there – and for many, they do work. (Just look up testimonials from the Nutritional Oncology Research Institute, or even studies on carotenoids antioxidants derived from natural sources, and even IV curcumin and sodium bicarbonate).
“I think it’s important to make patients aware that there are potentially life threatening downsides to chemotherapy. And doctors should be more careful about who they treat with chemotherapy,” writes one of the study’s co-authors, Professor David Dodwell, Institute of Oncology, St James Hospital, Leeds, UK.
This article is for informational purposes only. Consult a doctor before beginning any treatment. See our full disclaimer here.
Want to learn more about the most effective ways to prevent and reverse cancer? Read The pH Miracle revised and updated and The pH Miracle for Cancer –
5) Alkalizing Nutritional Therapy in the Prevention and Treatment of Any Cancerous Condition – https://www.amazon.com/Alkalizing-Nutritional-Prevention-Treatment-Cancerous-ebook/dp/B01JKCXJRY/ref=la_B001ILKCSU_1_14?s=books&ie=UTF8&qid=1528429047&sr=1-14&refinements=p_82%3AB001ILKCSU
6) The pH Miracle for Cancer: Discover the Truth about the Cause, Prevention, Treatments, and Reversal of ALL Types of Cancers – https://www.amazon.com/PH-Miracle-Cancer-Prevention-Treatments-ebook/dp/B01JJX1Q8S/ref=la_B001ILKCSU_1_6?s=books&ie=UTF8&qid=1528428158&sr=1-6&refinements=p_82%3AB001ILKCSU
7) Using Sodium and Potassium Bicarbonates in the Prevention and Treatment of All Sickness and Disease – https://www.amazon.com/Potassium-Bicarbonates-Prevention-Treatment-Sickness-ebook/dp/B01JLHJ1Y8/ref=la_B001ILKCSU_1_30?s=books&ie=UTF8&qid=1528429161&sr=1-30&refinements=p_82%3AB001ILKCSU
Sugar is a sad staple in the modern diet. It is controversial, also, with many people saying we’ve overdramatized the detriment from sugar. Many experts noting that “sugar is found in natural foods.” While this does hold true, it is unlikely the sugar in apples is causing our health infrastructure to fall apart. It is more likely the refined, dense sugars found in processed foods. Nothing wrong with a tomato, but ketchup is an entirely different store because it condenses the sugar experience generally in the cancer causing form of high fructose corn syrup..
Sugar is making us fat and sick. Sugar is making us diabetic. And now sugar might be giving women breast cancer. According to a study at The University of Texas MD Anderson Cancer Center, that’s exactly what might be happening.
The findings, published in the Jan. 1 online issue of Cancer Research,demonstrated dietary sugar’s effect on an enzymatic signaling pathway known as 12-LOX (12-lipoxygenase).
“We found that sucrose intake in mice comparable to levels of Western diets led to increased tumor growth and metastasis, when compared to a non-sugar starch diet,” said Peiying Yang, Ph.D., assistant professor of Palliative, Rehabilitation, and Integrative Medicine. “This was due, in part, to increased expression of 12-LOX and a related fatty acid called 12-HETE.”
Past studies have shown a connection between sugar intake and breast cancer, but much of that was focused on sugar causing inflammation, which inevitably stimulates cancer. This current connection, however, as reported by manderson.org, is much more direct.
“The current study investigated the impact of dietary sugar on mammary gland tumor development in multiple mouse models, along with mechanisms that may be involved,” said co-author Lorenzo Cohen, Ph.D., professor of Palliative, Rehabilitation, and Integrative Medicine. “We determined that it was specifically fructose, in table sugar and high-fructose corn syrup, ubiquitous within our food system, which was responsible for facilitating lung metastasis and 12-HETE production in breast tumors.”
Sugar isn’t good for us because it is an strong acid. We can debate this data in terms of legitimacy, but it is difficult to debate the overall dire health implications caused from over-abundance of sugar being ingested. Due to this, it is hardly a point worth debating: Lower your acidic sugar intake, choose natural high fiber low- sugar sources from cucumber, avocado, celery, tomato, lemon, lime, peppers, broccoli, spinach, grapefruit, and coconut. You will be healthier because you will be less acidic and more alkaline according to my published research.
Citation: Young RO (2015) Alkalizing Nutritional Therapy in the Prevention and Reversal of any Cancerous Condition. Int J Complement Alt Med 2(1): 00046. DOI: 10.15406/ijcam.2015.02.00046
This study, however, is a one of a kind study linking sugar directly to breast and lung cancer. In some ways, it is ground-breaking. From my scientific viewpoint, it is simply another research study validating my 30 years of research that we need to limit any refined sugars, complex locarbohydrates, high-sugar fruit and vegetables. Especially, stay away from sucrose, maltose, lactose, fructose, and any other sugar that ends in “ose.” Stay organic and plant-based with high fiber, low sugar foods and you will lower your risk not only for breast and lung cancer but many more western-diet ailments.
Sugar has long been associated with cancer. The pH Miracle diet (a low carbohydrate and low protein diet) has been linked to preventing and reversing cancer. The core component being that cancer cells thrive from sugar, which stands to reason that cutting the sugar supply off from what you eat and what you drink may cause cancer cells to shrink and die.
First, understand that sugar is a cycle of addiction exactly the same as cigarette and alcohol addiction. Once you cut ALL sugars out of your diet, you will stop craving them. My research and the research of others backs ups this claim. Low carbohydrate dieters often talk about not “being hungry.” And the truth is, it isn’t that they aren’t hungry, it is just that their body only craves what it needs to survive and thrive. Second, reach for high fiber low carbohydrate solutions. Leafy green salads, sprouts, and grasses, low sugar fruit and vegetables are a great example of a low-carb, high fiber experience. This will release electrical energy in the form of electrons into your body slowly so that you don’t bottom out and end up craving a donut. I like to start my day off with a green drink of spinach, celery and cucumber and a shake with avocado, spinach, celery, cucumber and salt. You may even want to try a mixed green salad with green olive oil and lemon instead of s sugar-cereal or oatmeal. Give it a try and feel the energy difference.
It’s been almost 20 years since I met my first disappearing patient — a nurse in her early 40s, let’s call her Kate. Kate was diagnosed with breast cancer. As a nurse, she had seen the results of breast cancer treatments. She was terrified, and determined. She was not heading for surgery, nor chemotherapy, nor radiation.
But Kate worked in a hospital. She worked with the doctors who diagnosed her cancer, and she worked with the surgeon, who wanted to schedule her into surgery “as soon as possible.”
The first thing Kate did was slow down. She did some research. It didn’t take her long to remind herself that in Canada, and in the USA, the treatments for cancer are akin to law. No hospital would dare deviate from the deadly three (cut, poison, burn).
Kate’s cancer was not large. She had been tested for cancer last year and no cancer was found. She knew it took many years for cancers to develop. At first, she was furious, “If it is here today, it must have been here last year. Why didn’t you find it last year?” It had not metastasized. It was not growing rapidly and was not affecting her health in any way. In theory, she had lots of time. So, she took some time.
But Kate didn’t look for magic cures. She didn’t search for the latest “cancer medicine.” She wasn’t interested in curing herself. She knew she was a nurse, not a doctor. She searched instead for the “cured” – patients who were diagnosed with cancer, and no longer had cancer. She knew from her work in the hospital, from conversations with patients, and with some staff, that these people existed — but from the perspective of the medical establishment, they seemed to disappear.
It didn’t take her long to find some patients who claimed they were cured. They hadn’t disappeared from life. They were eating, drinking, loving, and living full healthy and prosperous lives. But according to the medical records, they didn’t exist. They were “never cured.”
The medical system treated their cures as “anecdotal.” It ignored them. There was no attempt by any doctors to understand what happened to these cancer patients. They were no longer sick. The medical system looks after sick patients, treats sick patients. These patients were not sick.
Kate looked and listened. Her interest was not clinical science vs. anecdotal evidence. Her interest was personal. She talked, listened, compared stories. From several, she learned about a clinic that did not claim to cure cancer. It did not use medicines to treat cancers. But patients were cured, somehow. This clinic was not in Canada. It was not in the USA. She would have to go to Mexico to learn more.
There are lots of alternative treatment clinics in Mexico. Are some of them valid, using important techniques to cure cancers? Are some of them scams, wanting to take money from desperate clients? Do some of them have a cure that works sometimes, but might not work for her? Kate didn’t know. She did more research. She called the clinic.
The staff did not claim to cure cancer. Claiming to cure cancer is dangerous, even for a clinic outside of North America. They suggested Kate visit the clinic and see what happens there, no charge for a visit, but she would need to pay for her travel to Mexico. Kate had done her research. She had met and talked to patients whose cancers had disappeared.
Kate made her decision. She was familiar with cancer diagnosis techniques in Canada. She had undergone a physical examination, a mammogram, that detected a lump in her breast. Then she had a biopsy, where tissue was taken from the lump and was sent to a lab for analysis. The lab technician tested and examined the sample and issued a diagnosis “cancer” or “not cancer.” Once the diagnosis is issued, everybody swings into action. Kate knew that the mammogram had a high false positive rate and a false negative rate. Many people who are diagnosed with a “possible cancer” by a mammogram do not actually have cancer. She was also aware that cancer biopsies have a false positive rate and a false negative rate, as well. Her work in the hospital, with real patients, had made this very clear.
She didn’t really know for certain if she had cancer. Her surgeon, on the other hand, was still pressing her to schedule treatment.
Kate knew one thing. She had time. She cashed out some savings and booked a “holiday” in Mexico.
At the clinic, Kate was surprised that there was no “cancer diagnosis.” They did check the presence and size of the lump on her breast. But they didn’t repeat the biopsy. The clinic read her diagnostic reports, but did not investigate them further. There was instead a very thorough analysis completed by a suite of doctors. It took two full days of tests and interviews, if I remember correctly.
Kate was asked about her family’s medical histories. She gave blood samples. She was questioned extensively about her diet, about what she eats on a regular basis. What foods does she like and eat often. What foods does she not like and never eat. Doctors examined her lungs, her heart, liver, and other bodily organs with various tests. Her immune system was tested. Extensive interviews about her life, her work, her relationships, and more.
At the time I talked to Kate, I didn’t realize that she was not getting a “medical analysis,” she was actually getting a “healthicine analysis.” Her tests and questions fit perfectly to the hierarchy of healthicine: genetics, nutrition, cells, tissues, organs, bodily systems, body, mind, spirit, and community.
Kate’s genetics were analyzed through family history. There may have been further genetic analysis, I don’t remember all of the details. Her nutritional status was analyzed, not just by analyzing what she ate, and what she preferred to eat, but also by studying what she didn’t like to eat, what she deliberately never ate, what foods she believed she was allergic to. Her cells and tissues were analyzed directly, through blood samples and physical examination, and indirectly through medical history and other tests. Many of her organs were tested for healthiness. Her bodily systems, immune system, circulatory system, respiratory system, hormonal systems and more were analyzed and assessed. Her physical body was measured, weighed, and examined. Her mental health was assessed, as well as her spiritual healthiness. She was in good spirits, even in light of a potentially life threatening illness. Her community health was analyzed as well. Her family, her relationships with her children, her spouse, her parents, her work community, and more.
After a few days, Kate met with a group of doctors to discuss her health, not her illness, her healthiness. Diagnosing illness is difficult. Analyzing healthiness is more complex. It took several doctors and several hours for Kate to learn and understand what they had learned about her healthinesses and her unhealthinesses.
They then “prescribed” two weeks, if I remember correctly, of healthiness training, tailored to Kate’s specific situation. She spent the next two weeks at the clinic, learning to be healthier, not learning how to be “healthier in principle,” rather – learning what Kate needed to do to make her diet, her body, her mind, her spirits, and even her relationships with her communities healthier. She could not change her work community. But she could change how she reacted to and interacted with it – to improve her own health. After two weeks of learning at the clinic, her breast lump had started to shrink.
Kate went back to Canada, to put her learning into action. The lump disappeared. Her diagnosis was still there on paper. But her “cancer” had disappeared. She was retested at her hospital and no cancer was found.
Then Kate began to disappear.
When the surgeon asked again, she explained that she was not going to surgery. The surgeon looked away. He refused to look her in the eye after that.
But Kate didn’t disappear from her family. She went back to her family. She didn’t disappear from her job. She went back to her job. She disappeared from the cancer system. Her cancer disappeared, so, as a cancer patient, she disappeared.
Was she cured? We don’t know. There is no useful definition of a cancer cure. No medical or scientific test that can prove a patient has been cured of cancer. Our cancer treatment statistics have no count for people who are cured of cancer. Patients that are cured, whether they are cured with medicines or not, are not counted. No breast cancer patients are officially cured by medicine. If their cancer goes away without treatment, they disappear from statistics. If their cancer is killed by radiation, chemotherapy or surgery, they are not cured, they are a “survivor.” Everyone knows that cancer survivors are always waiting for the cancer to reappear. Their symptoms are in remission, but their cancer is not cured. They are not cured. With no proof of a cure, it might just be hidden.
Kate no longer has cancer. She paid, from her own pocket, for her trip to a clinic in Mexico. After the trip, her cancer disappeared. She had medical insurance. But her insurance wouldn’t pay for her trip. Insurance pays for treatments, not for cures. It pays for treatments, even if they fail. But it does not pay for success. Success disappears.
There are two ways for a cancer patient to disappear. You might be cured by health. Or you might be cured by a medicine that is not approved. In both cases, the medical system will ignore the cure, and ignore the patient.
In healthicine, there are no incurable diseases. If it is not curable – it is not a disease, it is a handicap, a disability, a deficiency, or simply an attribute of the person. All diseases can be cured by definition.
I have since met several cancer patients who have disappeared, and not just cancer patients. Maybe you have too? I’ve met more by internet, email, etc. There is no way for me to determine if a disappeared patient actually had cancer, if their treatment cured their cancer, if their body cured their cancer or if they still have cancer. We can only tell if there is another cancer diagnosis. Nothing can be told from the absence of a diagnosis.
There is no way for any doctor to tell either. There are no tests for a cancer cure. There is no way to recognize, much less document a cancer cure. There are no statistics for cancers cured.
Many cured patients don’t disappear quietly. They speak out. They write books and newspaper articles. They blog. But it doesn’t matter. They still don’t count. Once cured, they disappear. The medical system does not study their cases, does not study their diagnosis, does not study their cures. For chronic diseases, like cancer, arthritis, diabetes, heart disease, even obesity, and many more, there are no techniques to document “cured patients.” As a result, there are no statistics for “cured patients” of any chronic illness.
Once they are cured, they disappear. Health doesn’t cure illness, it disappears illness. And medicine doesn’t count people who have disappeared.
To your health