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A Finger on the Magic of Life-Antoine Bechamp, 19th Century Genius (1816 – 1908)

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The Magic Eraser

There have been several notable occasions in history when persons offering invaluable contributions to the advancement of human understanding have been ignored, ridiculed and even persecuted in their time. In most cases, however, their work has subsequently been given a deserved measure of recognition. Some great ones, though, have not enjoyed such rejuvenation and have “suffered the slings” of obscurity.

So it is with Antoine Bechamp. Had the profound voice of his science not been silenced, much of humankind may have been spared the worst aspects of the infectious stresses of the 20th century. Since the case can be made that the approved but improper and dangerous treatment of infectious “diseases” over the last century has in large part given rise io the present epidemic wave of degenerative “disease,” including cancer and AIDSyndroine, we might have been spared these miseries as well. At the least, we would have understood much more clearly why we have them. Fortunately, however, Bechamp’s work has been kept alive by small, successive bands of truth-seekers.

The adoption by science of Louis Pasteur’s germ theory as the whole truth, without regard to the subtleties and deep insight of Bechamp’s microzymian principle, represents one paraphrased: “There is no medical doctrine so potentially dangerous as a partial truth implemented as whole truth.” Any medical professional, bioscientist, health care practitioner, or lay person for that matter, who wishes to gain insight into the origins and nature of infectious and chronic illness, against the backdrop of a marvelous view of the life process, must consider Bechamp. And they must entertain one of the most important concepts to come out of his illustrious career-microbiological pleomorphism as it relates to disease and its symptoms.

There are four books written about him of which this writer is aware (although there are very likely more) and many works published by him. Of the ones by him, all except one are in the original French. Fortunately, his last book, The Blood and Its Third Anatomical Element, was translated into English in 1911 by Montague R. Leverson, M.D., Ph.D., M.A., although it has been difficult to obtain. Of the two major books about him, one is in French and the other (E. Douglas Hume’s Bechamp or Pasteur?) is also rare. The other two books about Bechamp are by R.B. Pearson. The Hume book, one Pearson book, and The Blood are once again available as reproductions in the U.S. after a hiatus of several years.

Bechamp considered The Blood his crowning work, and therein he describes an amazing microanatomical entity and its participation in the clotting process. He also includes details of his work and his experiences with the plagiarisms and “pettifogging ratiocinations” of Louis Pasteur. The French book about him, which author Christopher Bird praised highly to this writer, is by Marie Nonclercq, entitled Antoine Bechamp, 1816- 1908: L’Homme et le Savant, Originalite et Fecondite de Son Oeuvre. The latter part means, The Man and the Scientist, the Originality and Productivity of His Work. According to Bird, in an account given at a 1991 facts that did not set well with reigning theory, many questions arose … as I read essays and books, of a heretical nature, one could say, written by researchers whose names I never heard mentioned in my classes.  

Twenty years ago, the World Health Organization proudly declared recently the discovery that the single greatest factor in heart disease is a vitamin E deficiency.

Also perversely awe-inspiring is the fact that a person of Bechamp’s extraordinary accomplishments has been written out of history books, textbooks and all encyclopedias. It is sobering to consider the required degree of authoritarian control over key academic elements in our culture. It is not my intention to belabor the politics, but as the wonders of Bechamp’s work unfold to the mind, the question simply arises, “Why is this not common knowledge?” Yet, we must be grateful that his “erasure” was far from complete.

It is difficult to do full justice to Bechamp without recourse to a book. His work was incessant and prodigious, and his observations prolific. I will attempt to convey some essentials of his biological work-only a part of the picture, as the total output includes chemistry, medicine and pharmacy. He left a remarkable legacy of scientific insight that borders on the spiritual, yet died in relative obscurity with virtually no recognition by peers or the public. Having outlived his wife, his beloved associate Professor Estor, and his four children, he had to endure those hard lessons of life in addition to the one of professional anonymity. However, in keeping with his extraordinary mind, he never lost conviction that the truth would come to light, as would his role in its revelation.

I’m not sure why, when his life touched mine through E. Bechamp is known among a coterie of modern and contemporary admirers, and his work has been followed up, knowingly or not, by perhaps a total of 50 scientists. This group includes such names as Gunther Enderlein; Wilhelm Reich; Royal Raymond Rife; the courageous Australian team of Glen Dettman, Ph.D. and Archie Kalokerinos, M.D. (who for many years published information in the Toorak Times, an Australian newspaper); and Gaston Naessen, including myself, who have brought the Bechampian locomotive to a full head of steam.

It is at once unbelievable and understandable that the superficial dogma of Louis Pasteur could have prevailed over Bechamp’s insights in the 19th century French Academy of Science. Unbelievable because of the meticulous documentation and presentation Bechamp made of his prolific work. Understandable because Pasteur stole enough of the truth to make it pass, while having on his side upper class connections and a doctrine that more suited the cultural (especially religious) moods of the time. Abetting, if not creating, an atmosphere repressive to truth was a mood of impassioned ignorance among ecclesiastic authorities at the University of Lille, where Bechamp had moved in 1875 to teach. In a manner similar to that which devastated Galileo, they vigorously opposed the “heresy” of the microzymian view. Heightening the poignancy of this tragedy was the depth of that ignorance, which was unable to realize that the view was not heretical at all. In fact, Bechamp was a devout Christian who felt his inquiries merely to be revealing the Creator’s modus. But it is perversely awe-inspiring to see such bias having persisted for a century, supported by the structure of authority in bioscience, so that Bechamp’s principles have not yet (2015) been given fair examination in the mainstream.

Things may soon change-for a number of reasons, not the least of which is that research in the medical literature is now burning a raging blaze below the lofty suite in which the few powerful controllers lurk. They will soon have to surrender themselves at the window, or be consumed by the flames. Of course, one way in which they surrender is to rediscover the truth, that is, claim credit for making scientific “discoveries” about matters long ignored or repressed by them and long held as principle in alternative venues. For example, “science” has just discovered that antioxidants are good for asthma, especially vitamins C and E. And after the tireless, definitive work on vitamin E by the Shute brothers probably 20 years ago, the World Health Organization proudly declared recently the discovery that the single greatest factor in heart disease is a vitamin E deficiency.

Also perversely awe-inspiring is the fact that a person of Bechamp’s extraordinary accomplishments has been written out of history books, textbooks and all encyclopedias. It is sobering to consider the required degree of authoritarian control over key academic elements in our culture. It is not my intention to belabor the politics, but as the wonders of Bechamp’s work unfold to the mind, the question simply arises, “Why is this not common knowledge?” Yet, we must be grateful that his “erasure” was far from complete.

It is difficult to do full justice to Bechamp without recourse to a book. His work was incessant and prodigious, and his observations prolific. I will attempt to convey some essentials of his biological work-only a part of the picture, as the total output includes chemistry, medicine and pharmacy. He left a remarkable legacy of scientific insight that borders on the spiritual, yet died in relative obscurity with virtually no recognition by peers or the public. Having outlived his wife, his beloved associate Professor Estor, and his four children, he had to endure those hard lessons of life in addition to the one of professional anonymity. However, in keeping with his extraordinary mind, he never lost conviction that the truth would come to light, as would his role in its revelation.

I’m not sure why, when his life touched mine through E. Douglas Hume’s historical biography, such a strong feeling arose in me-the need to “exonerate” him, to bring his name and work to their deserved place of honor in history. Part of it, I’m sure, as with M. Nonclercq, is realizing the health benefits society might reap from understanding him, not to mention the inspiring, if not magical, insight into life and being that his views represent. But I’m still not quite sure why I want to be able to say (if in some way my various expositions about him over the last decade, added to the voices of others who have seen with his eyes, contribute to open re-evaluation of his science), “There, Antoine! Rest in peace, my friend.”

Principles of Micromorphology

While some of the ideas Bechamp addressed predated him, they had not been so clearly described, fully developed, or strongly supported by experimentation. It is said there is nothing new under the sun. If true, it may be because all things, or situations, exist at once in the Creation. It is a matter of perspective, much like looking at a tapestry. Bechamp’s perspective allows us to step back from tight focus and see the loose threads of the germ theory amidst a harmonious and astounding pattern of the life process. He had his “finger” on the magic of life. According to Hume, the essence of what he brought to us was as follows:

First, he demonstrated that the air is filled with microscopic organisms capable of fermenting any suitable medium on which they happen to land. He showed that the chemical change is carried out by a soluble ferment produced by the organism, and this ferment is analogous to the digestive juices of the stomach. Thus, he identified fermentation as a digestive process. (Dr. Young theorizes that all decomposition, even the rusting of steel, is mediated by ferments. It is known, for example that bacteria decompose rock into soil. Microorganisms are at or near the foundation of all life and life processes on Earth. For example, fungal forms are indispensable parts of the roots of most plants, including the largest trees.)

Secondly, the most profound conclusion to which Bechamp’s untiring and painstaking research led him is that there is an independently living microanatomical element in the cells and fluids of all organisms. This element precedes life at the cellular level, even the genetic level, and is the foundation of all biological organization. What originally piqued Antoine’s procreative curiosity was the discovery, somewhat by accident, that pure chalk from geological deposits at least 11 million years old would liquefy starch and ferment sugar solutions, while man-made chalk would not. After years of work tracking down the cause (fermentation was not understood at the time), he attributed the action to the living remains of organisms long dead. He called this tiny living element a “microzyma,” or small ferment.

Thirdly, he claimed that microzymas routinely become forms normally referred to as bacteria, and that bacteria can revert or devolve to the microzymian state. (This is the principle of pleomorphism, which is central to understanding the appearance of “infectious” and degenerative disease symptoms in the body.)

Fourthly, he explained that atmospheric germs are not fundamental species, but are either microzymas, or their evolutionary forms, set free from their former vegetable or animal habitat by the death of that “medium.”

Bechamp explained: “The microzyma is at the beginning and end of all organization. It is the fundamental anatomical element whereby the cellules, the tissues, the organs, the whole of an organism are constituted.” He referred to microzymas as the builders and destroyers of cells. The quotation emphasizes the constructive aspect of microzymian activity and purpose, but it is the destructive aspect, or the “end of all organization,” which concerns us in disease. He always found microzymas remaining after the complete decomposition of a dead organism, and concluded that they are the only non-transitory biological elements. In addition, they carry out the vital function of decomposition, or they are the precursors of beings (bacteria, yeasts and fungi) which do so. Thus, he clearly presented the idea that the physical life of higher biological forms arises from, is dependent upon, and is recycled by, microscopic beings. Simple, immediate proof of dependence is the indispensable bacterial population in the human GI tract. And it adds piquancy to the whole matter to consider that our digestive and metabolic associates are plants.

The crucial “catabolic” aspect of microzymian behavior enters the picture when the body becomes diseased, for, according to Bechamp:

In a state of health, the microzymas act harmoniously and our life is, in every meaning of the word, a regular fermentation. … In a condition of disease, the microzymas which have become morbid, determine in the organism special changes . . . which lead alike to the disorganization of the tissues, to the destruction of the cellules and to their vibrionien evolution during life.

The microzyma is an organized (insoluble) ferment: a living element. Controlled fermentation is a vital physiological process. For example, it is utilized as a means of breaking down toxins in intercellular fluid and the lymphatics. Also, some commercial dietary fiber products contain acacia and slippery elm. These soluble fibers ferment in the gut, resulting in short-chain fatty acids such as butyrate and acetate, which are highly beneficial to the colon wall. Bechamp published a paper (still in French) about the role of microzymas in the production of salivary diastase (ptyalin). Since there are microzymas in every cell, in the blood and intercellular milieu, it is likely that many vital substances, mostly enzymes, are produced by them or by their complexes.

Bechamp said that the process of cellular breakdown is mediated by microzymian fermentation-even in a healthy body. Though there is renewal happening as well, breakdown fermentation (aging) eventually takes over, greatly increasing in intensity upon death. When oxidative metabolism ceases and a body dies, negative surface charges are lost and the terrain goes acid. Microzymas respond to biochemical signals, the most important being pH. The condition of disease is a milieu which presents to the microzymas a premature biochemical signal that the organism is dead. They consequently change their function and evolve into forms capable of more vigorous fermentative breakdown-forms that reflect disease-what Bechamp called “morbidly evolved microzymas.” If the host pays no attention while it is still feasible to adjust, s/he will be recycled sooner than would otherwise be the case.

And further:

“… In disease, it is the elementary tissues or cellules that are affected….

It should result therefrom that tissue and cellular pathology are in reality microzymian pathology. In disease, the cellules have been seen to change, be altered and destroyed, and these facts have been noted. But if the cellule were the vital unit living per se, it would know neither destruction nor death, but only change. If then the cellule can be destroyed and die, while the microzyma can only change, it is because the microzyma is really living per se, and physiologically imperishable, even in its own evolutions, for, physiologically nothing is the prey of death; on the contrary, experience daily proves that everything is the prey of life, that is to say, of what can be nourished and can consume.”

Further Conclusions by BeChamp

“That there is produced in the organisms of all living beings, including man, in some part and at a given moment, alcohol, acetic acid, and other compounds that are the natural products of the activity of organized ferments, and that there is no other natural cause of this production than the microzymas of the organism. [Emphasis added. Here is where, in a compromised terrain, the culminate forms described by Dr. Young in the main text of his book Sick and Tired, could play a role. As described by Bechamp-i.e., in an apparently healthy organism-it would likely be the initial development phase.] The presence of alcohol, acetic acid, etc. in tissues reveals one of the causes, independent of the phenomenon of oxidation, of the disappearance of sugar in the organism, and of the disappearance of the gluco-genic matters and that which Dumas called the respiratory foods.”

“That, without the concurrence of any outside influence except that of a suitable temperature, fermentation will go on in a part withdrawn from an animal, such as the egg, milk, liver, muscle, etc., or, in the case of plants, in a germinating seed, or in a fruit which ripens when detached from the tree, etc. The fermentable matter that disappears earliest in an organ after death is the glucose, gluco-genic matter or some other of the compounds called carbohydrate, that is to say respiratory food. And the new compounds that appear are the same as produced in the alcoholic, lactic acid and butyric fermentations of the laboratory; or, during life, alcohol, acetic acid, lactic or sarcolactic acid, etc.”

“That the microzymas, after or before their evolution into bacteria, attack albuminoid or gelatinous matters only after the destruction of the … carbohydrates.”

“That the microzymas and bacteria, having effected the transformations before mentioned, do not die in a closed apparatus in the absence of oxygen; they go into a state of rest, as does the beer yeast in an environment of the products of the decomposition of the sugar, which products it formed.”

“That . . . the necessary destruction of the organic matter of an organism is not left to the chance of causes foreign to that organism, and that when everything else has disappeared, bacteria-and finally the microzymas resulting from their reversion-remain as evidence that there was nothing primarily living except themselves in the perished organism. And these microzymas, which appear to us as the residuum of what lived, still possess some activity of the specific kind that they possessed during the life of the destroyed being.”

Microzymas Unique to Each Organism and Organ

The microzymas were too minute to differentiate with the microscope (even for today’s equipment), and Bechamp knew he was not going to see them in detail. His brilliance shows again, however: “The naturalist will not be able to distinguish them by description, but the chemist and also the physiologist will characterize them by their function.” Having masterful skill in chemistry, he utilized that ability, accompanied by ingenious use of the polarimeter, to draw many of his conclusions. He was led to conclude that an organism’s microzymas are unique to it, and are not interchangeable with those of another. He went further to say that even within a single organism, each organ and tissue has functionally unique microzymas, and that, for example, those of the kidney do not belong in the liver. What, therefore, did he have to say about inoculation?

The most serious, even fatal, disorders may be provoked by the injection of living organisms into the blood; organisms which, existing in the organs proper to them, fulfill necessary and beneficial functions-chemical and physiological-but injected into the blood, into a medium not intended for them, provoke redoubtable manifestations of the gravest morbid phenomena.

“. . . Microzymas, morphologically identical, may differ functionally, and those proper to one species cannot be introduced into an animal of another species, nor even into another center of activity in the same animal, without serious danger.”

How much more foolhardy is it then, when vaccinal microzymas are not only from another species, but are already morbidly evolved and are accompanied by preservatives, formaldehyde, and other chemicals? There is no sanity whatever to this practice. The best that can be said about it is that it may prevent, against the odds, the appearance of varying sets of symptoms. But this is at the price of weakening the immune system, toxifying the body, and possibly setting the stage for degenerative symptoms later in life-all the while doing absolutely nothing for, except perhaps worsening, the underlying disease condition.

As indicated in the above quotation concerning “granulations of the protoplasm,” it would seem that microzymas are also closely related to, and perhaps precursors of, genetic molecules. In an August 8, 1977 address to the (now defunct) International Academy of Preventive Medicine, Drs. Dettman and Kalokerinos had the following to say:

“It became increasingly apparent to us that the problems relating to infection and immunization were, to say the least, oversimplified by organized medicine. Perhaps Bechamp was thinking in advance of our modern molecular biologists who refer to genes controlling enzymes! We wondered whether Bechamp’s writing anticipated, in some respects, the discovery of RNA and DNA? It now appears to us that the experimental data described in Bechamp’s work has, in part, been independently and unknowingly repeated by Professor Bayev of the USSR Academy of Sciences.”

In a personal communication with Prof. Bayev (1974) concerning the common factors of his and Bechamp’s work, Bayev states:

“Self-restoration of the molecule from its parts was obtained with pure transfer RNA from baker’s yeast. It is a rather simple organic substance of molecular weight 30,00 daltons. Its chemical structure is now identified exactly. I think the microzyma by Bechamp has a more complex chemical nature than a simple organic molecule, but our experiments with transfer RNA molecules prove that self-restoration is possible already at the molecular level.” [Emphasis added.]

Finally, might we not ask ourselves how much our uncritical acceptance of Pasteur’s work has retarded the development of medical science to this day? In our own work we found that when we became aware of Bechamp’s arguments we were better able to understand some of the puzzles of ourfindings with Aboriginal infant death in Australia, which initially led us into conflict with the prevailing medical models of disease and immunization. We feel that we have gone too far to turn back, and that we need the help of all health care professionals who dare to think for themselves in working through the tangled web of relationships that govern disease-immunization- nutrition interactions.

Bechamp and Pasteur

Bechamp never denied that the so-called germs of the air or other causes may be contributory, either to decomposition or illness, but only that these have not been expressly created, nor are they needed, for these purposes. As noted, the germs of the air are nothing other than microzymas or their evolved forms from fermentatively destroyed organisms. Their destructive or morbid influence may be added to that already faced by the organism’s endogenous microzymas, which may or may not have initiated morbid evolution. This is a crucial departure from germ theory. That is, without the predisposition of inherent microzymas-which condition is engendered primarily by a faulty internal environment-the germs of the air, or those of other sick bodies, will not produce illness in a person. One can see how this holistic view confers responsibility and power on the individual, as opposed to making him a victim to be saved (by a medical science powerless to do so). In addition to microzymas in the atmosphere, “The spores of the entire microscopic flora may intrude, as well as all the molds that may be born of these spores.”

In the earlier phase of his career, as Professor of Medical Chemistry and Pharmacy at the Faculty of Medicine at Montpellier University, Bechamp and his tireless colleague Professor Estor had many opportunities to test microzymian theory in practice. Examination of an amputated arm and many examinations of frozen plants during a particularly cold winter, convinced them that upon injury, bacteria developed internally without any outside influence. Bruising an apple without breaking the skin is an example; the broken cells will autoferment. This is one basis for the surgical cleaning of wounds.

Pasteur, on the other hand, a non-physician and proponent of the germ theory, seems to have lacked a certain understanding of living systems. He considered the body to be a collection of inert chemicals, and therefore after death he expected nothing living in it. When life would inevitably appear in dead organisms, he had to draw the conclusion that it resulted from invasion from without by the beings whose existence had been taught to him and the world by Bechamp. Either he saw but would not admit, or he simply could not fathom, that microorganisms are already inherent to humans and every other organized medium on the planet, all of which contain, are composed of, and have developed from, microzymas. Unfortunately, the persuasiveness of Pasteur’s superficial conclusions held sway over the deeper, rather elusive, complex, profound, even mystical workings of life and pathology.

Bechamp:

Long before Davaine considered the inside of the organism to be a medium for the development of inoculated bacteria, Raspail said,

“The organism does not engender disease: it receives it from without..

. . Disease is an effect of which the active cause is external to the organism.” In spite of this, the great physicians affirm, in Pidoux’ happy words, “Disease is born of us and in us.”

But M. Pasteur, following Raspail . . . maintains that physicians are in error: the active cause for our maladies resides in disease-germs created at the origin of all things, which, having gained an invisible entry into us, there develop into parasites. For M. Pasteur, as for Raspail, there is no spontaneous disease; without microbes there would be no sickness, no matter what we do, despite our imprudences, miseries or vices! The system, neither new nor original, is ingenious, very simple in its subtlety, and, in consequence, easy to understand and to propagate. The most illiterate of human beings to whom one has shown the connection between the acarus and the itch understands that the itch is the disease of the acarus. Thus it comes about that it has seduced many people who give unthinking triumph to it. Above all, men of the world are carried away by a specious, easy doctrine, all the more applicable to generalities and vague explanations in that it is badly based upon proved and tried scientific demonstrations.

Much of Pasteur’s refusal to accept microzymian theory may have arisen from pure rivalry which came into focus when Bechamp solved, right under the Pasteur’s nose, a disease crisis threatening the French silkworm industry. Since the two must have known each other previously, we must be open-minded enough to allow that Bechamp, though concerned for his country’s important industry, may have indulged himself in a little one- upmanship in his embarrassment of Pasteur, who gained more privilege from social connection than from earned merit (thus, in most books, Pasteur is given credit for solving the crisis). If so, it may have cost Bechamp dearly, because it earned him the eternal resentment of the volatile chemist, who took every future opportunity to oppose his tormentor. And it was primarily the “specious easiness” of germ theory that allowed Pasteur to get away with it, because few scientists of the time were sufficiently skilled to probe deeply enough beneath the superficialities. Few possessed enough knowledge or insight to understand the elusive complexities. And Bechamp warned against facile judgments when he wrote in 1869:

“In typhoid fever, in gangrene, in anthrax, the existence has been proved of bacteria in tissue and in the blood, and one was very much disposed to take them for granted as cases of ordinary parasitism. It is evident, after what we have said, that instead of maintaining that the affection has had as its origin and cause the introduction into the organism of foreign germs with their consequent action, one should affirm that it only has to do with an alteration of the function of the microzymas, an alteration indicated by the change that has taken place in their form.”

Again:

“An egg contains nothing organized except microzymas; everything in the egg, from the chemical point of view, will be necessary for the work of the microzymas; if in this egg its ordered procedure should be disturbed by a violent shaking, what happens? The albuminoid substances and the bodies of fat remain unchanged, the sugar and the glycogen disappear, and in their place are found alcohol, acetic acid and butyric acid; a perfectly characterized fermentation has taken place there. That is the work of the microzymas, the minute ferments, which are the agents and the cause of all observed phenomena. And when the bird’s egg has accomplished its function, which is to produce a bird, have the microzymas disappeared? No, they may be traced in all the histological elements; they pre-exist-one finds them again during the functioning and the life of the elements; one will find them yet again after death; it is by them that the tissues are made alive.”

“The part of organized beings essentially active and living, according to the physiologists, is the granular protoplasm. We went a step farther and said it is the granulations of the protoplasm, and though for their perception a sort of spiritual insight is required, we have based our conclusions upon experimental proofs of the most varied and positive nature. Bichat looked upon the tissues as the elements of the bodies of higher animals. With the help of the microscope, very definite particles, cells, were discovered, and were regarded in their turn as elementary parts, as the last term of the analysis. . . . We have said in our turn: The cell is an aggregate of a number of minute beings having an independent life, a separate natural history. Of this natural history we have made a complete description.”

Bechamp apparently had a good sense of place in the scientific pursuit (“in our turn”) of the ever-retreating Ultimate Secret. He realized that the truth of empiricism is for the time, or is in the process of evolving. No doubt he would willingly have given up microzymian theory in face of right evidence of a newer observation. I am presenting science with a newer, though highly correlative, observation. For, as Bechamp attributed all fermentation in the body to microzymas, we now are able to see that it is also carried out by higher evolutionary forms-yeast and fungus. He would have been open to the idea that bacteria also evolve, and that there may even be a step or two between microzymas and bacteria, e.g., viruses. However, as I have suggested,  functionally the virus form is very likely something other than what it is thought to be in the mist-ified Pasteurian version of bioscience.

In this article the distinction has repeatedly been made between the disease condition and its symptoms. This idea is inherent in microzymian principle, and it is interesting that Bechamp alludes to the source of the disease condition as “imprudences, miseries or vices.” This is a close approximation in different terms of the holistic gamut of precursors to physiological ill-being: improper diet, emotional upheaval and various self­destructive behaviors. Yet it is a testimony to the power and skill of the propagandists of mainstream medicine and the Pasteurian decalogue itself that serious illness remains such a mystery in the mind of the masses.

Cosmic Microzymas

It is also interesting to hear the scientist speak of “spiritual insight.” And it is interesting as well to consider microzymas in terms of Eastern modes of spiritual thought, such as yoga, in which it is felt that our creation is an ongoing process. That is, life was not put here and simply proceeds, but it, and we, are coming into being in the moment. Thus, there is constant “turnover,” or renewal and healing. In this scenario, the microzyma may be seen as an early, if not the primary, transmutation from the fine vibrations of the Cosmic Life Force into a denser form or pattern of life-something not explainable by biochemistry, certainly. Due to the colloidal nature of these nascent elements, they carry high levels of energy and may also be receptive to frequencies of light and radiation asactivating or informational signals. During formation, or once formed, they may be stimulated by cosmic energy, which comes directly into our being, which provides energy that cannot be accounted for in the Krebs cycle, which is ionizing, and which has been interpreted as carrying part of the holographic human archetypal information. Is the microzyma Colloidal Intelligence, or a modus of the Creative Intelligence-a living transducer for the Idea in Consciousness, which it translates into the cellular anatomy? It was said earlier that microzymas respond to the pH of the surrounding medium, reforming when appropriate. However, the chemical aspect may be just an obvious way for us to qualify the situation. Perhaps the change in pH alters vibrations or resonant frequencies, changing the microzymian quality of reception, transmittal or transduction of the Life Force and cosmic rays. 

Bechamp said the microzyma is imperishable. Canadian microscopist Gaston Naessens says his analogous somatid particles have survived carbonizing temperatures, 50,000 rems of radiation, and all acids. If these claims are true, could such imperishability stem from being at the interface of energy/matter and Consciousness, i.e., from the imperishability and constant materialization of life itself? It may therefore be that only the Mystery of life stands prior to the microzymian patterns.

Elaborate Colonies

An interesting corollary to microzymian principle is the idea presented by Lynn Margulis and Dorion Sagan in their book Micro-Cosmos-that all higher forms of life are elaborate colonies of microforms that have undergone a natural assimilation into the more complex whole, thus becoming cells or cooperative parts of cells. Some forms have not, or not yet, become assimilated into tissue, and so appear as separate symbionts. The intestinal bacteria are an excellent example. Based on this theory, an entertaining conjecture is that since the primordial, colonizing forms are plant life, animals don’t exist per se, so that humans are complex, mobile, talking vegetation.

Unfortunately, Micro-Cosmos lacks the insight microzymian principle might bring to it. It fails to recognize life prior to the cell, and therefore cannot consider what may be the primary orchestrative tools of the colonization process. It discusses DNA repair enzymes with no suggestion as to their origin.

This article also does not take into account the rapid functional changes of microforms in response to terrain imbalance, and is mystified by cancer:

“It is as if the uneasy alliances of the symbiotic partnerships that maintain the cells disintegrate. The symbionts fall out of line, once again asserting their independent tendencies. . . . The reasons, of course, are not all that clear, but cancer seems more an untimely regression than a disease.”  Here is what seems a struggle with the bonds of the Pasteurian decalogue. The symbionts falling out of line might easily have been expressed, “The microzymas change their function.” 

Confirmation of Bechamp

There have been many modern and contemporary confirmations of various aspects of Bechamp’s work. One of the earliest and most piquant was reported in an article in The Times, a London newspaper, on April 8, 1914. A French bacteriologist, Mme. Henri, had succeeded in transforming an anthrax bacillus into a coccus form having entirely different functional properties. It could easily have been explained by Professor Bechamp, who sat virtually unrecognized at the London Medical Congress in 1881, where plagiarist Pasteur appeared amidst outbursts of cheering as his country’s representative, and where, as reported in The Times, August 8, 1881, he categorically denied the pleomorphism of B. anthracis.

Pasteur also jumped to the conclusion that each kind of germ produces one specific fermentation, while Bechamp proved that a microorganism might vary its fermentation effect in conformity with the surrounding medium. Bechamp’s assertion that these microforms, under varying conditions, might even change their shape was proved conclusively by F. Loehnis and N.R. Smith of the U.S. Department of Agriculture in 1916 {Journal of Agricultural Research, July 31,1919, p. 675).

And, for evidence that the biological terrain is the determinant factor over the mere presence of a symptogenic microform, we may return to Kalokerinos and Dettman:  

It should come as no surprise to discover that almost every pathogen may be isolated from the majority of so-called “healthy” people: Candida is such an example, and here we quote from the Manual of Clinical Mycology (Conant, Smith, Baker & Calloway, 1971): “Since pathogenic strains of C. albicans can be isolated from (1) normal skin, (2) normal oral and vaginal mucous membranes and (3) stools of normal individuals, it is obvious that most infections have an endogenous source, and the determination of the source of the infection is as difficult as it is with Staphylococcus aureus infections.

This revelation also highlights a recent example of the false conclusions to which one is led by germ theory: The news in research on atherosclerosis is that scientists have isolated a chlamydia-type organism in the plaque, and have concluded that it is the cause of this symptom. The plan is to use antibiotics to combat this “pathogen.” There is only one guarantee in this folly: at the very best they may achieve atherosclerosis without the chlamydia. At worst, they will exacerbate the mounting crisis in health caused by a half-century of antibiotic abuse.

R.R. Rife

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Perhaps the most profound confirmation of pleomorphism was executed by another nearly obliterated genius, this time an American microscopist with the alliterative name of Royal Raymond Rife. His story was told in an impressive piece of work called “The Rife Report” by investigative reporter Barry Lynes. It has been published in book form as The Cancer Cure That Worked!, which is highly recommended from several standpoints-for its revelations about Rife’s research and technology, which would be astounding for these times, never mind for the late 1920s to mid-30s; for a wonderful background on many pioneering figures in biology; for anyone interested in a deeper understanding of where medicine has gone in the United States; and not least, for a wonderful Foreword by John W. Mattingly of Colorado State University, whose writing has always been an inspiration whenever encountered.

Rife’s extraordinary microscope (with 31,000 diameters resolution), reported on in great detail in the Feb. 1944 Journal of the Franklin Institute (Vol. 237, No. 2), was capable of detail and clarity surpassing the newly emerging electron microscopes. Its use of prismatically dispersed natural light frequencies, rather than electron beams and acid stains, allowed clear views of living subjects. Weighing 200 pounds, standing 2 feet high, and consisting of 5,682 (!) parts, the Rife Universal Microscope was an unsung wonder of the world, and the world has thus far been robbed of this absolutely elegant design.

In 1920 Rife began doing research in the electronic treatment of “disease,” specifically to find a way to destroy the tubercle bacillus by means of radio frequency (r.f.) radiation. Attempts to do so were trial and error because the organism’s resonant frequency was unknown. Lynes tells us that when the frequency was finally found and the bacteria killed, the subjects (poor guinea pigs!) died of toxicity. Rife reasoned that there was a viral form in the bacteria that survived the beam because it had a different frequency. But the virus was beyond the reach of his current microscope, which relied on chemical stains. Through an intuitive flash, he “conceived first the idea and then the method of staining the virus with light.’’’’ The idea was based on the principle of resonant frequency. Each microorganism has its own fundamental frequency of light, something Bechainp apparently took advantage of with his polarimeter. Rife arrived at the conclusion that light could be used, instead of fatal chemicals, to “stain” the subject. This was brilliant. Equally brilliant was its execution. A brief, partial description of the instrument, taken from the Journal’s review, is irresistible:

The entire optical system-lenses and prisms, as well as the illuminating units-are made of block-crystal quartz. The illuminating unit used for examining the filterable forms of disease organisms contains fourteen lenses and prisms, three of which are in the high-intensity incandescent lamp, four in the Risley prism, and seven in the achromatic condenser, which incidentally has an aperture of 1.40. Between the source of light and the specimen are subtended two circular, wedge-shaped, block-crystal quartz prisms for the purpose of polarizing the light passing through the specimen, polarization being the practical application of the theory that light waves vibrate in all planes perpendicular to the direction in which they are propagated. When light comes into contact with a polarizing prism, it is split into two beams, one of which is refracted to such an extent that it is reflected to the side of the prism, without, of course, passing through the prism, while the second ray, bent considerably less, is enabled to pass through the prism to illuminate the specimen. When the quartz prisms on the Universal Microscope, which may be rotated with vernier control through 360 degrees, are rotated in opposite directions, they serve to bend the transmitted beams at variable angles of incidence while, at the same time, since only a part of a band of color is visible at one time, a small portion of the spectrum is projected up into the axis of the microscope. It is possible to proceed this way from one end of the spectrum to the other-infra-red to ultra-violet. Now, when that portion of the spectrum is reached in which both the organism and the color band vibrate in exact accord with one another, a definite, characteristic wavelength is emitted by the organism. In the case of the filter­passing form of the Bacillus typhosus, for instance, a blue light is emitted, and the plane of polarization is deviated plus 4.8 degrees. … A monochromatic beam of light corresponding exactly to the frequency of the organism is then sent up through the specimen and the direct, transmitted light, enabling the observer to view the organism stained in its true chemical color and revealing its own structure in a field which is brilliant with light.

Recall that Bechamp said the chemist would identify microzymas by their function. Their evolved forms would also have a chemical function, or in this case, a signature. Thus, we evolved scientifically from analysis based on light polarizations to that based on the emission of light frequencies, which Rife referred to as the organism’s “true chemical refractive index.”

The Journal then explains that instead of light rays from the specimen passing through the objective and converging, they pass through a series of special prisms which keep the rays parallel:

It is this principle of parallel rays in the Universal Microscope, and the shortening of projection distance between the prisms, plus the fact that three matched pairs of ten-millimeter, seven-millimeter and four-millimeter objectives in short mounts are substituted for oculars, which make possible not only the unusually high magnification and resolution, but which serve to eliminate all distortion as well as all chromatic and spherical aberration….The coarse adjustment, a block thread screw with forty threads to the inch, slides in a one and one-half inch dovetail which gibs directly onto the pillar post. The weight of the quadruple nosepiece and the objective system is taken care of by the intermediate adjustment at the top of the body tube. The stage, in conjunction with a hydraulic lift, acts as a lever in operating the fine adjustment. A six-gauge screw having a hundred threads to the inch is worked through a gland into a hollow glycerine-filled post, the glycerine being displaced and replaced as the screw is turned, allowing a five to one ratio on the lead screw. This, accordingly, assures complete absence of drag and inertia. The fine adjustment being seven hundred times more sensitive than that of ordinary microscopes, the length of time required to focus ranges up to one hour and a half.

A major upshot of Rife’s work was his ability, through several pleomorphic stages, to transform a virus he found in cancer tissue into a fungus, plant the fungus in an asparagus- based medium, and produce a bacillus E. coli, the type of microform indigenous to the human intestine. This was repeated hundreds of times. By this accomplishment, Rife showed that the pleomorphic capacity of microforms goes beyond the bacterial level to the fungal level. Dr. Young has observed this cycle, and is suggesting that its progression to the last stage-mold-is critical. And he includes in this cycle the very important stages intermediate to microzymas and bacteria, the protein complexes usually referred to as viruses, and their immediate descendants, the cell-wall deficient forms detailed by Lida Mattman, Ph.D.

What’s more, Rife identified 10 families in the whole spectrum of microlife. Within each family, any form/member could become any other. Also, the fact that organisms have resonant frequencies allowed Rife to further develop his r.f. “beam ray,” which helped rid the body of cancer symptoms.

Apparently, Rife was not aware of Bechamp. Had he been (he was about 20 years old when Bechamp died on the other side of the Atlantic), a light of another frequency might have been thrown on his research,  What a marvelous and beneficial revelations might have arisen with Rife’s technology guided by Bechamp’s vision?  However, even though saddled i the beginning with a germ-theory mindset, he managed to rise above its worst effects.  Demonstrating an instinctive understanding of the disease process, Rife made the following statement: ” We do not wish at this time to claim that we have cured cancer, or any disease, for that matter.  But we can say that  these waves, or this ray, as the frequencies might be called, have been shown to possess the power of devitalizing disease organisms, or ‘killing’ them when tuned to an exact wavelength, or frequency, for each different organism.”  And again: “In reality, it is not the bacteria themselves that produce the disease, but the chemical constituents of these microorganisms enacting upon the unbalanced cell metabolism of the human body that in actuality produce the disease.  We also believe if the metabolism .  . . is perfectly balance or poised, it is susceptible to no disease.”

While he was making the classic error (perhaps a semantic one) of referring to symptoms as the disease, he seemed aware that disease-associated microorganisms do not originally produce the condition which has supported their morbid evolution in the animal or human body.  This fine, but critical, distinction is missing in the views of all the researchers reported on in Lynes’ book.  Even as they stood opposed to the orthodoxy, they still pursed these morbidly evolved symptoms with the intent of curing the visible, or diagnosed “disease.”

When Rife first destroyed the tubercle bacillus, the guinea pigs died of toxic poisoning. Could that poison have been bacterial debris, including endotoxin, and the death a severe Herxheimer reaction? Rife went on to search for a virus he assumed was released when the bacteria died, but if he had understood what Bechamp explained and what I am emphasizing now, he would have known that the organism’s microzymas were thus set free in the medium. And we can now understand that there was no virus per se, but only variously complexed microzymas.

As a poignant insight into the passion of a man of brilliance whose revelations were denied to the world by avarice, Lynes presents a report given in 1958 by one of Rife’s co­workers, who had known him from the early days of his career:

“He finally got to a point where from years of isolation and clarification and purification of these filterable forms, he could produce cancer in the guinea pigs in two weeks. He tried it on rats, guinea pigs and rabbits, but he found finally that he could confine his efforts to guinea pigs and white rats, because every doggone one was his pet. And he performed on them . . . the most meticulous operations you ever want to see in all your born days. No doctor could ever come near to it.

“He had to wear a big powerful magnifying glass. He performed the most wonderful operations you ever saw. Completely eradicating every tentacle out from the intestines, and sewed the thing up and it got well and didn’t know anything about it at all. Did it not once but hundreds of times. This is a thing that again and again I wish was published. I wish with all my heart that all the detailed information that he developed could be published because the man deserves it.”

“He finally got these cultures on the slide. He could look through this thing and you could see them swimming around absolutely motile and active.”

Then he’d say, ‘Watch that.’ He’d go turn on the frequency lamps. When it got to a certain frequency, he’d release the whole doggone flood of power into the room. The doggone little things would die instantly.’

“He built the microscopes himself. He built the micro-manipulator himself. And the micro-dissector and a lot of other stuff.”

“I’ve seen Roy sit in that doggone seat without moving, watching the changes in the frequency, watching when the time would come when the virus in the slide would be destroyed. Twenty-four hours was nothing for him. Forty-eight hours. He had done it many times. Sat there without moving. He wouldn’t touch anything except a little water. His nerves were just like cold steel. He never moved. His hands never quivered.

“Of course he would train beforehand and go through a very careful workout afterward to build himself up again. But that is what I would call one of the most magnificent sights of human control and endurance I’d ever seen.

“I’ve seen the cancer virus. I have seen the polio virus. I’ve seen the TB virus. Here was a man showing people, showing doctors, these viruses of many different kinds of diseases, especially those three deadly ones-TB, polio and cancer.

“Time and time again since that time some of these medical men have made the proud discovery that they had isolated we will say one of the viruses of cancer, had isolated one of the viruses of polio. Why, that was one of the most ridiculous things in the world. Thirty-five years ago Roy Rife showed them these things.

“These machines demonstrate that you could cure cancer- all crazy notions of usurping the rights of the AMA notwithstanding. They definitely could take a leaf out of Roy Rife’s book and do an awful lot of good to this world for sickness and disease. As a consequence, we have lost millions of people that could have been healed by Rife’s machines.”

“I like Roy Rife. I’ll always remember Roy as my Ideal. He had a tremendous capacity for knowledge and a tremendous capacity for remembering what he had learned. He definitely was my Ideal. Outside of old Teddy Roosevelt, I don’t know of any man any smarter than him and I’ll bank him up against a hundred doctors because he did know his stuff with his scientific knowledge in so many lines. He had so many wrinkles that he could have cashed in and made millions out of it if he had wanted to and I do mean millions of dollars. Which would have benefited the human race, irrespective of this tremendous thing that he built which we call the Rife ray machine.”

“In my estimation Roy was one of the most gentle, genteel, self-effacing, moral men I ever met. Not once in all the years I was going over there to the lab, and that was approximately 30 years, did I ever hear him say one word out of place.”

“All the doctors used to beat a path to Rife’s lab door and that was a beautiful lab at one time. It was beautifully arranged inside. The equipment was just exactly right; his study was just wonderful. It was a place of relics and the atmosphere could not be duplicated anywhere.”

(It is noteworthy that even though Rife entered the realm of vivisection, he at least showed the compassion to fix the damaged animals.)

More Cosmic Tones

For some time there have been “Rife instruments” on the market, using his frequencies in an electrode-pad configuration, and sold for research purposes. But that r.f. beam ray, that was the “magic,” technologically speaking, at least. And now an instrument has appeared claiming to be a re-creation of the original (see “Revival and Caution” below). Rife would probably have been the first to question whether the beam deals with the underlying disease condition. In this respect, I would like to suggest a consideration of the beam in terms of both the microzyma and the yogic principle of the chakras.

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In yoga, the chakras (“wheels” or vortices of energy) are said to be the “organs” of the subtle body (the energy blueprint of the being). They are tuned to light frequencies corresponding to the colors of the rainbow. One’s personality, physical and physiological qualities, and even the health of the individual are said to arise from their infinitely complex configurations and their interactions with other fields. They are also spiral vortices through which the meridians of acupuncture flow. By way of the neurolymphatic reflexes and neurovascular points of the body, these flowing energies are intimate with the systems, organs, cells and chemistry of the physiology.

In terms of what was suggested earlier about the cosmic microzyma, consider what Christopher Hills, yogi and physicist, has written:

“… (It is) very likely that the chromosome, when exerting its biochemical effects in replication is NOT an indivisible unit with all its many constituents, in a precise, unchanging hereditary chemical pattern existing from one generation to the next. It is, of course, subject to evolutionary CHANGE. Yet in their function, these chromosomes have to be capable of precise replication, so they must spontaneously aggregate into patterns of LIFE (consciousness of form), which is characterized by the chemical environment in the nucleus of the cell. Any change in this immediate environment, such as a change in the specific frequency of a sharply selected energy, of radiation, of light, of electromagnetic waves or of sound, may alter not only the structural relationship of the molecules in the cell nucleus, but also their biochemical and genetic activity.”

(Taken from pp. 813-814 of Nuclear Evolution, a work on the physics of Consciousness published in 1977, almost one century after Bechamp created the name “microzyma.”)

“Consequently, what if the Rife beam, in addition to its resonant effect on microforms, was influencing the frequency balance of the chakras or the balance and freedom of flow in the meridians, perhaps doing what might be called R.F. Acupuncture, and perhaps ultimately “tuning” the microzymas? This might constitute a sufficient rebalancing of the being, or an altering of its vibrational condition, to be considered curative; and it might be maintained if the individual were subsequently to nurture their psychobiological terrain, which includes “the chemical environment in the nucleus of the cell.”

Leading the Horse to Water

What more could the scientific world have been waiting for than what Rife showed it? Significantly, he was not working in a vacuum but had the attention and support of respected biomedical scientists and doctors, including Dr. Edward C. Rosenow of the Mayo Clinic; Dr. Arthur I. Kendall, Director of Medical Research at Northwestern U. Medical School; and Dr. Milbank Johnson, member of the board of directors at Pasadena Hospital in California. As Lynes informs us, newspapers reported on Rife’s work, including significant clinical success. And as noted, no less a prestigious organization than the Franklin Institute did a detailed report on him. But, not only did the medical establishment (AMA) turn its back on Rife and his safe, effective means of eradicating cancer symptoms, but it systematically conspired to destroy him-which it did not once, but twice. Thus, Bechamp and then his unwitting supporter, Rife, geniuses of the caliber of Copernicus, Galileo and Lavoisier, were rubbed into obscurity. (While on this note, we might remember another genius pleomorphist, Wilhelm Reich, who died miserably in an American prison for attempting to bring truth to light.)

It didn’t take much to see that if Pasteur’s noxious poisons could garner even a semblance of success, the monetary potential would be stupendous. Thus, his greatest claim to fame ought to have been the inauguration of the “calamitous prostitution of science and medicine to commercialism” (Hume). Research facilities modeled after the one opened in 1888 in Paris, and used for brutal experimentation on living animals, as well as the production and sale of vaccine drawn from sickened bodies, came into existence all over the world. Bechamp’s brilliant expositions took second place to the dawning of a “new” era. It was the era of stone-hearted torture of fellow creatures and cruelty to our own species. It was the era in which bacterial disease symptoms were supplanted over time with a second wave of modern chronic fungal “infection.” Surfing this wave of degenerative mycotic infestation-officially unacknowledged as such-partially comprising heart disease, cancer, diabetes, so-called autoimmune disease and AIDSyndrome, were the profiteers, supported by arrogant, single-minded adherence to a scientifically and philosophically flawed, superficially plausible, and financially exploitable model of life and health.

Lynes tells us that Rife found himself in the path of Morris Fishbein, the Hitlerian ruler who headed the AMA from the mid-1920s until 1949, when he was forced from his position by a revolt among doctors. In Chicago, Fishbein had gotten wind of a clinic in San Diego using Rife’s beam-ray method of eliminating cancer symptoms. When refused a buy-in, he used his influence to bring the manufacturing company down in court for operating without a license. This blow to medicine in the late 1930s was a major step in suppressing the knowledge of pleomorphism, the mind-boggling Rife Universal Microscope, and the amazing radio frequency beam instrument used in the clinical setting.

In the second wave of suppression, the establishment (FDA) “Elliot-Nessed” a factory established in the 1950s by Rife and associate John Crane to manufacture the beam ray instrument. Everything was destroyed, records confiscated, and every practitioner possessing a unit was pursued and forced to surrender it as illegal.

Many other courageous individuals have been a part of the process of bringing the hidden truth about microorganisms and their symptogenic properties to light. One of the most significant is Dr. Virginia Livingston-Wheeler. Though she is discussed in the main text, she deserves another mention as a key figure who also faced suppression-the stress of being made invisible-by the sciomeds (power structure of scientific medicine). She published a book in 1983, The Conquest of Cancer, and, according to Lynes, wrote many articles and made presentations to science societies, including the New York Academy of Science, and international conferences. Lynes reports that she once returned from a presentation at an international symposium in Rome to find that her research funds with a major hospital had been canceled and the laboratory closed. During the four or five decades following the first establishment backlash at Rife, several other scientists, including Dr. Eleanor Alexander-Jackson, Dr. Irene Cory Diller, and Lida Mattman, Ph.D. (cell-wall deficient forms), stood in the face of intimidation to continue the valiant, yet feeble, tradition of unbiased biomedical science.

Revival and Caution

There is now afoot, as recently shown on the television show “Strange Universe” (March, 1997), a movement to revive the Rife beam-tube technology. Equipment was shown, as were moving pictures of the lysis of several unidentified microorganisms implied to be culprits in disease. Testimonies were given by a few people saying that they, or people they knew, have been helped by this beam ray. While this is an interesting and promising development, a note of caution is very much in order, so that folks do not end up like Rife’s guinea pigs, being put to death by a violent Herxheimer reaction. I believe the approach I recommended by is safer-more holistic and harmonically based in that we make the environment dissatisfactory to these symptoms of disease, so that instead of exploding on the spot and spewing poisons, they simply “pack their bags and leave.” That is, they will, of themselves, devolve into stages of the pleomorphic cycle consistent with the frequencies natural to a harmonious terrain; or will become so devitalized that the immune system can easily trash them.

It is hoped that this overview has given a provocative taste of what lies obscured in the history of biology. The reader is encouraged to explore the Hume and Lynes books especially, as well as that of the beacon of 19th-century bioscience, Antoine Bechamp: The Blood and Its Third Anatomical Element. 

A Note of Emphasis:

In this writer’s opinion, it is a poverty of compassion, the utmost arrogance, faultiness of perspective, and an error of science to inflict self-generated human miseries on innocent animal species in research laboratory experiments. Each year some 100 million animals are killed. Though many such experiments are used as references in this book, this is not a sanction. It is done to show the kind of results being ignored by “authorities” who believe in these methods, to accommodate professionals who live by them, to appease reductionist minds, and to suggest that enough is enough.  Human development and quality of life are unlikely to improve in any way by this torture of fellow creatures, unless such change occurs in the heart to make such practice unthinkable. The benefit to science and society is highly speculative and frequently negative. Let the experiments be done on human volunteers, whose physiology at least lends some logic to the process. Thalidomide was animal tested. Aspirin will kill a cat. Sheep can eat arsenic.

The habitual basis for vivisection is not founded in true science, but in profound alienation from nature and detachment from the nature of being. It continues out of species prejudice and an egocentric machismo that feeds on conquering nature via destructive analysis. It continues out of a merry-go-round intent to keep laboratories busy, researchers working, and to keep the research supply industry rolling in money. And it continues out of the habitual ignore-ance of the principles of wellness, which have long been in place in many forms. The fault for our rampant “diseases” may be ascribed to such ignorance and not laid at the feet of helpless animals, who play no part except to suffer for us and to die by the hundreds of millions. This is an insult to the Creation, not to mention an ecological disaster from the disposal of bodies. And to make matters worse, much of the research is based upon biased and erroneous science.

But the bottom line is, though we have the power over these creatures to inflict our cruelty on them, to do so may have dire consequences, given a Universe that operates on balance. Individuals of compassion and conscience may wish to consider opposing, through words and actions, this Frankensteinian madness. 

General References 

[1]  Bechamp, Pierre Jacques Antoine. The Blood and Its Third Anatomical Element .(Montague R. Leverson, translator). London: John Ouseley Limited, 1912.

[2]  Bird, Christopher. Gaston Naessens. Tiburon, Cal.: H.J. Kramer, Inc., 1991.

[3]  Bird, Christopher. To Be or Not to Be?. A paper presented in an address to L’Orthobiologie Somatidienne Symposium 1991, Sherbrooke, Quebec, hosted by Gaston Naessens.

[4]  Hills, Christopher. Nuclear Evolution. Boulder Creek, Cal.: University of the Trees Press, 1977 (out of print).

[5]  Hume, E. Douglas. Bechamp or Pasteur? Ashingdon, Rochford, Essex, England: The C.W. Daniel Co. Ltd., 1923.

[6]  Kalokerinos, A. and Dettman, G. Second Thoughts About Disease/ A Controversy and Bechamp Revisited. Warburton, Victoria, Australia: Biological Research Institute [booklet published from an article in Journal of the International Academy of Preventive Medicine-, July 1977; 4(1)].

[7]  Lynes, Barry. The Cancer Cure That Worked! Fifty Years of Suppression. Queensville, Ontario, Canada: Marcus Books, 1987.

[8]  Margulis, Lynn and Sagan, Dorion. Micro-Cosmos. New York: Summit Books, 1986.

[9]  R.O. Young, Sick and Tired, Reclaim Your Inner Terrain.  Woodland Publishing, Orem, Utah, 1999.

[10]  R.O. Young, S.R. Young, The pH Miracle, Hachette Publishing, New York, New York, 2010.

 

 

Pathological Blood Coagulation and the Mycotoxic Oxidative Stress Test

 Robert Young PhD

Naturopathic Practitioner – The pH Miracle Ti Sana Detox Medical Spa and Universal Medical Imaging Group

Abstract

Historical analysis suggests that conventional understandings of Disseminated Intravascular Coagulation (DIC) may be misguided; further examination may be necessary.  Here, a theoretical analysis provides an alternative explanation for DIC pathology; it is suggested that the cause and mechanics of DIC are largely due to the proliferation of several intravascular microforms and their associated metabolic toxic acidic waste products — Mycrozymian Acidic Toxins (MAT) and Exotoxic-Mycotoxic-Producing Microorganisms (EMPO).  The Mycotoxic Oxidative Stress Test (MOST) is presented here as an easy, inexpensive and non-invasive alternative to conventional measurements for the detection of intravascular  acidic toxins, DIC  and oxidative stress.

Introduction and Historical Perspective

More than 150 years ago, British physician T. W. Jones asked the question, “Why does the blood circulating in the vessels not coagulate?”[1]  though a general answer to this question is now obvious, the biochemical mechanisms involved in how the blood coagulates (clots) are complex and varied, and all the intricacies have not yet been explained. A. Trousseau, recognized that the blood of cancer patients is in a hyper-coagulable state in the process of coagulation, even while confined in the blood vessels.[2]  The name given to this discovery is still in use today, as “Trousseau’s Syndrome.”[2]  Early in his career, Rudolph Virchow, the Father of Pathology, was interested in thrombosis and embolism.  He speculated that intravascular blood could be altered so it would clot as a result of a stimulus too weak to clot normal blood.[3]  In 1856 Virchow delivered a lecture setting forth this concept.

Although the concept of partial clotting within vessels reaches back to the beginnings of modern medicine, much of the discovery of its biochemical mechanisms – the activation of clotting factors – has been left to chance.  The admission of a patient to the hospital with an unceplained bleeding disorder challenged researchers to discover the cause of hemorrhaging.  Analysis of blood from normal persons helped in the study of the patient with the blood disorder. A new clotting factor was hereby discovered which was missing from the  patient’s blood.  For this reason, several clotting factors have been named after the individuals in which they were missing: e.g., Christmas factor (factor IX)[4], Hageman factor (factor XII)[4].

In this article, the causes of pathological (intravascular) clotting will be described, as will various methods of detecting this condition, especially a blood test I call the Mycotoxin Oxidative Stress Test (MOST).

The Mechanics of Blood Coagulation

Blood clotting is a highly detailed chemical-mechanism involving many distinct components.  The problem for the hematologist hs been to understand it at the biochemical level.  Undoubtedly, efforts to fully understand blood clotting will continue for many more years.

Recalling Antione Bechamp’s[8] and Gunther Enderlein’s[9] research into the sub cellular living elements and combining this with what is known of colloidal flocculation[6], it is suggested that the clotting of blood begins with the end-linking (polymerizing) of the fundamental protein unit called by Bechamp the microzyma[8].  A chain of these living units constitutes fibrinogen, which is still dispersed 9micro-hetergenous0 in the blood, and it may or may not be further processed.  If processing continues, it will be either by continued end-linking or by cross-linking.  End-linked fibrinogen is referred to here as fibrin monomer, which I have suggested is a repair protein also dispersed in the blood. Due to a number of blood clotting factors, the process may continue until the excess fibrin monomer and/or until fibrin becomes excessively end-linked.

Cross-linking the polymerized strands to form a three-dimensional network results in what is called the hard clot (fibrin – the major protein of clotting blood).  Factor XIII, which instigates the forming of these blood networks. is always present but latent in the blood, and must be activated before the formation can occur.  Persons who are producing fibrin monomer or excessively linked fibrinogen are said to be in a hyper-coagulable state, while those having diminished  ability to form clots are in a hypo-coagulated state.  It is the activation of the colloidal clotting factors which is so complex.  Blood clotting may occur through many pathways and be initiated by many different stimuli.  Regardless of initiation factors, the process is a sequence of events in which the activation of one factor triggers another, until, after a series of discrete steps, fibrin is formed.

When blood is clotted prematurely, and the factors involved are consumed (incorporated into) the body recognizes a deficiency of clotting agents and generates more.  Thus, people with a tendency to clot excessively will alternate between a hyper coagulable state and a hypo-coagulatable state.  When in the hypo coagulated state, such people hemorrhage until the deficient clotting factors are replaced.[4]  When only fibrin monomer or excessively linked fibrinogen is formed (no cross-linking), it is quite subtle and may go undetected.  It may be detected by a change in blood viscosity (sedimentation rate), by the Mycotoxic Oxidative Stress Test (described later), or by other more subtle means.  If strands of fibrinogen are cross-linked, however, a suggicient amount of insoluble precipitate of fires may result, and these can be detected microscopically using a phase contrast and dark-field microscopy in prepared slides of fresh tissue or blood.  An excessive formation of fibrin leads to  an impairment in circulation, and eventual organ failure usually results.[5]

With this background, we are in a position to consider a standard medical term: disseminated intravascular coagultion (DIC).[6]  This term encompasses the hyper coagulable state, i refer to as pathological blood coagulation which consists of both insoluble and excess dispersed polymers of colloidal proteins.

Key Ingredients of Pathological Blood Coagulation

Before discussing DIC in more detail, it si necessary to introduce its fur important ingredients according to this view – mycotoxins, endotoxins, exotoxins, and tissue factor.  Any of these elements, or any combination of them, can play a major role in initiating unwanted DIC.[6]  However, mycotoxins or the acids from yeast have been found to be the underlying element which instigates and intensifies the participation of the other three.[6]  Each will now be described in turn and brought into the clotting picture.

(Micrograph 1: left, shows normal hyper-coagulated blood in a healthy blood clot sample and right, hypo coagulated blood in an unhealthy blood clot sample)

Mycotoxins and Metabolism by Fermentation

As discussed in the main text of my published book, Sick and Tired book[7 ]. acidification of blood and body tissues and organs and the accompanying lack of oxygen lead to pathological metabolic fermentation, which is carried out primarily by yeast and mold.  Such pathological microorganisms, or their precursors, ar inherent to the human body and to all higher organisms.  Their precursors according to Bechamp, the microzymas, carry on a nominal and homeostatic fermentation themselves. under healthy conditions.[8]  The primary function of yeast and mold is to decompose the body upon the death of the animal or human organism.  Their premature overgrowth indicates a biochemical environment akin to death.  During pathological metabolic fermentation, high concentrations of several acidic substances called mycotoxins are created.  They are highly damaging, always acidic, metabolic products.  If not immediately buffered by specific antioxidants, such as hydrogen peroxide and the hydroxyl free-radical, mycotoxins can seriously disrupt the physiology by disrupting normal metabolism and by penetrating blood and body cells and poisoning them.  As will be seen, they interact with many of the mechanisms for DIC in various pathological symptomologies.

In my published article called The Finger on the Magic of Life: Antoine Bechamp, 19th Century Genius (1816-1908),  I discuss pleomorphism in some detail.[7] Understanding this phenomenon – the rapid evolution of microorganisms across traditional taxonomic  lines is helpful in getting a complete picture of DIC.  Briefly stated, collodial living microzymas evolve intracellularly into more complex forms (microorganisms), beginning with a healthy primitive stage comprising of repair proteins.  As the disease condition worsens, morbid intermediate forms (filterable bacteria or viruses, cell-wall deficient forms and full bacteria) develop from repair proteins, or directly from microzymas.  A third macrostage comprises the commonly recognized culminate microorganisms which are yeast, fungus to mold.  In terms of pleomorphism, all of these microorganisms represent a single family of variously functioning forms.[8]  The culminate forms produce the lions share of acids, which are mycotoxins and the primary focus of my research.[7][8][9]  For convenience, bacteria, yeast, fungus and mold that produce acidic metabolic wastes and protein cellular fragments called exotoins, endotoxins and mycotoxins will here be referred to collectively ash EMPO, or exotoxic, mycotoxic-producing microorganisms.

What follows is a shortened description or the description and origin of several exotoxins and mycotoxins, referred to collectively microzymian acidic toxins of MAT, which are involved in the processes leading to DIC.  The bio-effects, or the pathology of cellular fermentation, of these toxic metabolites are know as mycotic illness, mycotoxicosis, or mycotoxic stress as seen in the MOST and described and published by Dr. Bolin in the 1940’s.[10]

One such metabolic product is acetyl aldehyde, which is formed by  cellular breakdown of food, especially carbohydrate and the birth of  EMPO.  Acetyl aldehyde can also break down into a secondary substance know as ethyl alcohol.  Although acetyl aldehyde presents an immediate hazard to health and well-being, nature has provided a means of buffering of neutralizing this acidic by-product of cellular digestion and fermentation almost as soon as it is created.[11] The controls of acetyl aldehyde (and ethyl alcohol) are the sulfur amino acids, cysteine, taurine, methionine and the peptide glutathione which is found in red blood cells and almost all cells utilizing oxygen.[12]  In an attempt to buffer or neutralize MAT, the body will also bind or chelate both fats and minerals to them.[12]

Another member of the MAT family is uric acid, which is formed by the digestion of protein and the creation of EMPO.[13]  Uric acid can also break down into secondary substance, on of which is alloxan.[14] This has been shown to damage the insulin-producing pancreatic beta cells leading to diabetes [Refer to Tables 1 and 2]

A shortage of alkalizing nutrients or an excess of MAT initi­ates an immune response in which a special class of free radicals which I call microzymian oxidative buffering species (MOBS) are released.[15] These oxygen metabolites carry unpaired electrons and are intended to disrupt bacteria, yeast, fungus and mold, and buffer exotoxins, endotoxins, and mycotoxins. Current medical savants believe that they can disrupt just about any­thing they contact, including healthy cells and tissue: this is not accurate. The fact is that MOBS carriers a nega­tive surface-charge and repel healthy cells, which also have a negative surface-charge. [16] It is the positively surface-charged bacteria, yeast/fungus, mold, exotoxins, endotoxins, and myco­toxins that MOBS bind too.[17]  This aspect gives some insight into autoimmune phenomena, which are not, as is often maintained, the result of an overburdened immune system. They result either as a side-effect of the immune system’s attempt to remove foreign or toxic ele­ments, or as a direct attempt by the immune system to remove cells or tissue rendered useless or disturb­ing to the body by MAT.

In every degenerative symptomatology I have studied, I have found excessive MAT and MOBS (see Tables 1-3). Some of these degenerative symptoms and their underlying disease conditions, including cancer are described in my recently published paper on a deficiency on alkaline nutrition and cancer. [15] But the fact that myco­toxins cause harm to humans and other animals is purely a secondary effect, since, as noted, the prima­ry function of the microorganism is not to cause illness. We know from the fossil record that pleomorphic microforms existed long before animals.[19] In fact, humans and animals developed in terms of micro­organisms.[20] The reverse, however, is not true. Since micro­organisms appeared first in the developmental sequence, they are not physiologically aware of humans and animals. There is much evidence that human and animal physiologies are highly aware of, and respond to MAT – these acidic compounds signaling the presence of bacteria, yeast, fungi and/or mold or  EMPO.[21].

Endotoxins

Also involved in the process leading to DIC are endotoxins, substances endogenous to symptogenic (i.e., “pathogenic” in orthodox terms) bacteria. Endotoxins are a family of related substances having certain common characteristics, but differing from one bacterial form (or strain) to another. Endotoxins are lipopolysaccharides (LPS). LPS form a widely diversified group because of (1) the number of long- chain fatty acids composing lipids; (2) the number of individual sugars as well as their modes of linkage to one another; (3) the branching of sugar chains; and (4) the number of possible arrangements of these units. Endotoxins also contain proteins, further com­pounding the structural diversity.[22]

One theory on endotoxin states that its purpose is to act as a semi-permeable membrane for the bac­terium, limiting and regulating substances entering the organism.[22] Endotoxin resides solely on or near the interior surface of the cell membrane and is shed into the surrounding medium only upon the death of the bacterium. Thus, as these microforms die off, or are lysed by bodily activity, endotoxin is released. (This fact may well be an explanation for the Herxheimer reaction, in which a patient becomes worse following the administration of toxic drugs or other forms of treatment that drastically alter the associated organ­ism.[23]) Another endotoxin theory states that LPS are a constituent of the membrane, and as the organism grows, endotoxin fragments are repeatedly sloughed off into the medium. This phenomenon has been observed in the digestive tract.[24] Since bacterial translocation into the blood is not only possible but common where epithelial hyperpermeability exists, one can assume that the process will continue there. Both theories may be correct if we think of the first one as true of “adult” forms, and the second as true of newly developed and expanding ones.

Basic to the structure of an endotoxin is the lipid common to all forms, designated lipid A, to which is attached a “core” polysaccharide, identical for large groups of bacteria. To the core polysaccharide is attached the O-antigen, consisting of various lengths of polysaccharide chains which are chemically unique for each type of organism and LPS. These chains pro­vide endotoxin specificity.[25] Experiments conducted over many years indicate that most, if not all, of the toxic effects of an endotoxin may be attributed to the lipid portion, and it is sometimes used per se in experiments rather than the entire molecule.[26] An important additional feature of lipid A is its phos­phate content. Each phosphate group carries a nega­tive charge, and since lipid A is a rather large mole­cule, it provides, essentially, a negatively charged sur­face. The importance of this will be seen shortly.

Exotoxins

These are the metabolic excretions of bacteria. While endotoxin’s ongoing effect is, in a manner of speaking, in the background, exotoxins, like myco­toxins, present a double-edged sword. Not only do they initiate DIC, but they produce, or influence the body to produce, the various and numerous infec­tious symptomatologies, such as typhoid fever, diph­theria, etc. (See “Vaccination Reconsidered” in Section 4 of the Appendix of Sick and Tired for details on the action of diphtheria toxin.)[7] By comparison, mycotoxins not only initiate DIC, but there is much evidence to sug­gest that they produce, or influence the body to pro­duce, degenerative symptomatologies, such as arthri­tis, diabetes, etc., and cancer and AIDS as well.

Tissue Factor

Crucial to the understanding of DIC is recogni­tion of the role of tissue factor (TF), formerly known as thromboplastin. This transmembrane lipoprotein exists on the surface of platelets, vas­cular endothelial cells, leukocytes, monocytes, and most cells producing EMPO.[27] It plays a major role in several biochemical mechanisms leading to DIC.

TF is the primary cell-bound initiator of the blood coagulation cascade. Its gene is activated in wound healing and other conditions. By itself it is capable of initiating clotting, but also becomes active when complexed with factor VII or activated factor VII (Vila).[28] TF has been described as the receptor for factor VII because of the close association between the two proteins and because it causes a shape change (conformational) in factor VII, allowing it to attain activity. Both factor Vila and the TF/VII com­plex activate factors IX and X, which initiate the clotting cascade and the formation of thrombin.[29]

Development of Disseminated
Intravascular Coagulation
(DIC)

DIC Induced by MAT and Tissue Factor

An infusion of toxins into the blood has a direct effect on TF gene expression in leukocytes. Contact of MAT, endotoxins (lipid A), or exotoxins with leukocytes, activates proteins that bind to DNA nucleotide sequences, thereby activating the TF gene.[30] (See Tables 4-6.)

Endothelial cells damaged in culture by exotoxins, endotoxins, or mycotoxins attract polymorphonuclear leukocytes (PMNs), which adhere to the damaged cells. Once the leukocytes are bound, they can still have their TF gene activated if it hasn’t yet occurred, and they may release MOBS in response to toxins and to organisms of disease, possibly creating further dis­turbances. (Cellular disorganization then releases acti­vating proteins into the blood, which is discussed in more detail later.) Research shows that exotoxic and mycotoxic stress resulting in bound PMNs can be blocked by “antioxidants.”[31] These might better be called anti-exotoxins or antimycotoxins. Both observa­tion and study have led the author to conclude that cellular disorganization is initiated and primarily caused by fermentation pathology, not, as is the cur­rent belief, by the MOBS, or free radicals, generated to destroy toxins and microorganisms. MOBS or free radicals, because of their negative charge, are released to chelate or bind EMPO and MAT. It is suggested by current savants that free radical tissue damage is the secondary, “shotgun” effect of intense immune response to EMPO toxification and MAT-damaged cells. This could not be the case since healthy cells or their membranes carry a negative charge and would resist any electromagnetic attraction because of simi­lar charge. The concentration and instability of MAT generated in a compromised terrain, as opposed to the fleeting existence of free radicals, especially exoge­nous ones, also lead to this conclusion.

Endothelial cells grown in culture can be induced to express tissue factor. In one experiment, no procoagulant activity could be detected in the absence of toxins. However, the addition of mycotoxins from Aspergillus niger or Micrococcus neoformas (Mucor racemosus Fresen) resulted in procoagulant activity which reached a maximum in four to six hours and was dose-dependent. The same experiment was applied using E. coli and Salmonella enteritidis endo­toxin with a similar result.[32] A single intravenous injection of a mycotoxin from Aspergillus niger into experimental animals resulted in circulating endothelial cells within five minutes. In other exper­iments with the mycotoxin, detachment of endothe­lial cells from the basement membrane was noted.[33] (See Table 8.)

Removal of endothelial cells has dire conse­quences from two standpoints: First, the surface of these cells is covered with a specific prostaglandin (PGI2) known as prostacyclin. If blood contacts a surface not covered with PGI2, it will clot. For example, surfaces devoid of this prostaglandin are formed whenever a vessel is cut or punctured. An abrasion or other injury may also expose a surface on which PGI2 is lacking. The removal of endothelial cells by exotoxins or mycotoxins creates a surface devoid of PGI2, leading to blood clotting (see Table 7). Secondly, disorganization of endothelial cells cre­ates increased levels of EMPO and MAT which are attracted to an exposed surface (basement mem­brane) which expresses a negative charge. This also leads to clotting.

DIC Induced by Electrostatic Attraction

It was discovered in 1964 that blood will clot sim­ply from contacting a negatively charged surface.[34] Previously it was believed that the clotting process comprised a cascade of enzyme activity in which one activated the next, etc. The discovery that blood could be clotted simply by contacting a negatively charged surface ruled out the purely enzyme hypoth­esis. Only some of the known clotting factors have been shown to be enzymes.[35] As a result of this sur­prising discovery, detailed research was conducted in an attempt to describe the process. In some experi­ments, the negatively charged surfaces of selected, finely divided, inorganic crystals, including alu­minum oxide, barium sulfate, jeweler’s rouge, quartz, and titanium oxide, were considered.[36]

The clotting factor eventually shown to be activat­ed when whole blood contacted negatively charged surfaces was factor XII, also known as the Hageman factor. This is a positively charged protein migrating in an electric field (electrophoresis) toward the anode.[37] It is believed that factor XII is normally in the shape of a hairpin which binds to the negatively charged sur­face at the bend. Electrostatic attraction forces the two arms to lie flat on the surface, thereby exposing the inner faces and activating the molecule.

It was discovered that if the negatively charged particles were smaller than the clotting factor itself, activation was minimal. Or, if the concentration of clotting factor was too great, there was little or no activation.[38] Both of these observations indicated that the process was one of electrostatic attraction between the negatively charged surface and the clot­ting factor, which is a “basic” protein, that is, posi­tively charged.[39]

Activation of factor XII allows the activation of factor XI, which then activates factor IX. Thus, the blood clotting cascade continues to the formation of fibrin in the normal manner.[40] However, due to a series of activations begun by contact of factor XII with a negatively charged surface, trace amounts of factor Xa also show up in the blood. Factor VII is activated to Vila by factor Xa. Factor Vila then acti­vates factors IX and X, leading to the formation of thrombin. Factor Xa, with co-factor Va, continues the clotting cascade until fibrinogen is activated, leading to fibrin formation.[41] (See Table 5.)

As discussed earlier in terms of prostacyclin, beneath endothelial cells is another surface—the basement membrane. Called the extracellular matrix, it is a thin, continuous net of specialized tis­sue between endothelial cells and the underlying connective tissue. It has four or more main con­stituents, including proteoglycans (protein/polysac- charide).[42] The removal of endothelial cells by’MAT exposes this membrane, which is negatively charged by virtue of its sulfonated polysaccharides in the pro­teoglycans. This brings a reduced negatively charged surface into direct contact with the blood, which activates factor XII and the clotting cascade.[43]The positively charged toxic components of MAT also activate factor XII, as do disturbed disorganized cells, yeast/fungus cells, moldy cells, and the phos­phate groups in the lipid A component of endotoxin. (See Tables 2-5.)

To summarize this section, exotoxic, mycotoxic, and oxidative stress resulting from the overgrowth of bacteria, yeast/fungus, and then mold, has multiple actions, all leading to disseminated intravascular coagulation:

MAT activation of tissue factor gene in leukocytes; subsequent activation of factors VII, IX, and X, resulting in the blood clotting cascade.

MAT activation of tissue factor gene in endothelial cells, again leading to the clotting cascade.

MAT damage to endothelial cells, resulting in neu­trophil attraction, with TF gene activation and generation of MOBS, which, in turn, neutralize MAT, protecting healthy endothelial cells or the basement membrane and supporting the janitorial services of the leukocytes.

Removal of negatively charged endothelial cells by positively charged exotoxins, endotoxins, and mycotoxins, creating a surface devoid of PGI2, also exposes the negatively charged basement membrane, leading to the activation of factor XII and initiation of the clotting cascade. Positively charged components of EMPO, exotoxins and mycotoxins, and several other elements, including the lipid A component of bacterial endotoxin, also activate factor XII and the clotting cascade.

Endothelial Cells as Antithrombotics or Procoagulants

Normal, resting (unstimulated) endothelial cells show antithrombotic activity in several ways: (1) by the inhibition of prostacyclin (platelet adhesion and aggregation); (2) the inhibition of thrombin genera­tion; and (3) the activation of the fibrinolytic system, leading to clot lysis.[45] We will take a brief look at the thrombin aspect.

On the surface of endothelial cells is a protein called thrombomodulin, which acts as a receptor for thrombin. When bound to thrombomodulin, throm­bin can activate protein C. Activated protein C then catalyzes the proteolytic cleavage of factors Va and Vila, thereby destroying their participation in blood clotting. Thus thrombin, which normally activates fib­rinogen, plays an opposite role in this case and inhibits the clotting process.[46,47] (See Table 7.)

On the other side of the coin, the endothelial cell becomes a procoagulant agent when acted on by cer­tain lymphokines, such as interleukin-1. Not only can interleukin-1 induce TF gene expression, but it also suppresses transcription of the thrombomodulin gene in endothelial cells. As in other situations, the lymphokine-activated endothelial cell expresses TF on its surface as a result of TF gene activation. This leads to the production of thrombin and the trigger­ing of the blood clotting cascade.[48] (See Table 5.) Many lymphokines also stimulate adhesion of leuko­cytes to endothelial cells damaged by MAT, resulting in recycling of the cells by MOBS, as described later.

DIC Induced by Intracellular Exotoxic, Mycotoxic, Oxidative Stress by Bacteria, Yeast/Fungus and/or Mold

Any cell which has gone from an oxidative to a fer­mentative state can biochemically cause macrophage production of the lymphokine tumor necrosis factor (TNF). This protein has been shown to activate the gene for TF in fermenting cells, which are so behaved due to morbid evolution of bacteria, yeast/fungus, and then mold.[49,50] In the author’s view, a cell having been switched entirely to fermentation metabolism as a result of a physical or emotional disturbance of that cell, is what constitutes cancer (see Tables 5 and 13). (One might argue that this definition does not fit all “forms” of cancer, such as leukemia, for example. This is because leukemia is not cancer, but an immune response to the rise in EMPO and MAT in the body, and a relatively easy compensation to correct.)

The surface of many disorganizing or fermented cells (cancer cells) is characterized by small projec­tions in the plasma membrane which pinch off, becoming free vesicles containing toxins as well as TF complexed with factor VII. These vesicles can aggre­gate and/or lodge anywhere, ultimately releasing their contents. Also, the presence of excessive amounts of TF/factor VII complexes on the surface of fermented cells allows the formation of a fibrin net around the cell and around the entire mass of cells (tumor). This seems to be an attempt by the body to encapsulate and contain the mass. However, fermented cells do escape from the primary fibrin net, perhaps due to some electromagnetic effect, and become free-float­ing in the circulation. They may thus lodge elsewhere and instigate the fermentation of other cells by fungal penetration or by poisoning them and provoking a morbid evolution of their inherent microzymas.

Because of the surrounding fibrin net, these mobi­lized fermenting cells are protected from collection by the immune system while in transit.[51,52] (See Table 4.) The blockage or dissolution of fibrin net forma­tion by an anticoagulant such as heparin allows freed, fermenting (metastasizing) cells to be dismantled by natural killer cells and other immune cells (see Tables 5, 12 and 13).

DIC Induced by MAT/EMPO and Immune System Response (Release of MOBS)

Unsaturated fatty acids are highly susceptible to EMPO as well as MAT. Linoleic acid, a long-chain fatty acid present in white cells, has 18 carbons and 2 unsaturations. Subjected to MAT, linoleic acid binds the exotoxin, endotoxin, or mycotoxin, there­by forming an epoxide at the first unsaturation.[53] Research has revealed that this compound, named leukotoxin, is highly disturbing to other cells. It caus­es platelet lysis, thereby releasing TF and initiating DIC.[54] (See Table 10.) The fact that MAT result in fermented fats lends further credence to the sugges­tion that the initial and primary degenerative damage to structures and substances in the body is caused by exotoxins and/or mycotoxins, and that damage by MOBS, or by other free radicals, is not possible.

Another mechanism leading to DIC is the release of a special glycoprotein, sialic acid, from the terminal ends of cell-membrane polysaccharides, where it is always found. Polysaccharides play a highly significant role in biochemical processes, with both enzymes and membrane receptors recognizing various groupings of specific sugars linked in highly specific ways.

Immediately preceding the release of sialic acid in the polysaccharide chain is the sugar galactose. The sialic acid/galactose arrangement is utilized as a biolog­ical indicator of cellular and molecular aging. As cells age, sialic acid is naturally expressed from the terminal ends of polysaccharides, thereby exposing galactose. A membrane-bound enzyme from the liver, galactose oxi­dase, recognizes galactose and eventually disorganizes it, disrupting cell function integrity and hastening demise. Aged red blood cells, which have expressed a significant amount of sialic acid, are removed from the blood by this process. (I theorize that the biological ter­rain may be at work in normal cell aging. That is, the rate at which sialic acid is expressed is determined by the levels of corrosive acids in the system and the body’s ability to remove them, although there are no doubt intracellular factors at work as well.)

I suggest from my years of  clinical research  that cellular breakdown is compounded by the fermentation of the galactose by the microzyma. This is a process that begins from within and not necessarily from without. Not only does this action create more sialic acid, it creates other toxic waste products such as acetic aldehyde, alcohol, uric acid, oxalic acid, etc. The increase in cellular disturbances and fermenta­tion of the galactose creates biochemical signals for more galactose oxidase. This leads to greater cellular disorganization and developmental morbidity, espe­cially in the red blood cells, and a rise in the level of detrital serum proteins, which encourages clotting. From this perspective, diabetes, arthritis, atheroscle­rosis and other symptomatologies become more clearly “degenerative” (see Tables 2-5, 12 and 13).

Fibrinogen is a rather elaborate protein having the structure of three beads on a string. Expressed on the end beads is sialic acid, which indicates the beginning of disorganization of the fibrinogen and a declining negative charge to the positive. Prior to the declining charge and the expression of sialic acid on the end beads, fibrinogen, which is negatively charged, will not polymerize the healthy blood due to mutual repulsion. However, fibrinogen will poly­merize to damaged cells, EMPO, MAT and other positively charged areas of the body for repair pur­poses. Thus, as more and more sialic acid is expressed, there will be a significant reduction in the charge of the fibrinogen, acting as the primary requirement for the polymerization of fibrinogen (hypercoagulable state). The resulting polymer, fib­rin monomer, is the protein chain used in the repair of cells and clotting of blood.[55] End-linking will take place after the release of sialic acid (positive charge) by whatever means.

With this background, it is interesting to note that blood taken from persons suffering from anxiety is expressing sialic acid from fibrinogen, and is halfway toward clotting. Hormones released during anxiety states are easily fermented, giving more momentum to MAT and thereby resulting in this important change in fibrinogen. It leads to a clotting pattern characteristic of anxiety stress, and is readily identi­fied in the MOST. As can be seen in this picture, the pattern is a “snowstorm” of protein polymeriza­tions measuring from 2 to 10 microns.

allergiesbefore

 

 

 

 

 

 

 

[Micrograph 2: An Anxiety Profile showing a ‘snowstorm’ of 2 to 10 micron protein polymerizations starting from the center of the clot and moving out towards the edge]

As mentioned earlier, despite the attempt by the body to neutralize EMPO and MAT, an excess will initiate the release of MOBS by immune cells. A major MOBS is superoxide, designated chemically as O 2. It may exist alone or be attached to another ele­ment, such as potassium (KO’2) or sulfur (SO). Again, however, nature has provided a means of pro­tecting healthy cells—their negative charge[1]. Another protection against superoxide is the enzyme superox­ide dismutase (SOD), also found in all healthy cells.

A second member of the MOBS family is hydro­gen peroxide (H202). This molecule is very unstable and tends to react rapidly with other biological mol­ecules, damaging them. The release of hydrogen per­oxide in the body is a response to the overgrowth of decompositional organisms in a declining pH (com­promised biological terrain). The control for healthy cells against hydrogen peroxide is their negative charge and the protective enzyme catalase, one of the most efficient enzymes known.

When leukocytes and other white blood cells are stimulated by the presence of bacteria, yeast/fungus and mold, they treat these organisms as foreign par­ticles to be eliminated. During and prior to phagocy­tosis, the foregoing oxidative cytotoxins, along with the hydroxyl radical (OH’), are generated and released specifically for neutralizing microforms or harmful substances. This release is referred to as an “oxidative burst.” As a result of fermentation and the production of exotoxins and mycotoxins that fer­ment galactose from cells, the immune system is activated. An oxidative burst is released to neutralize the morbid microforms and mycotoxicity.[56] Like other biological processes faced with constantly alarming situations, the continued release of MOBS can get out of control. This may damage endothelial cells, the basement membrane, or other body ele­ments, and this activates fibrinogen to fibrin monomer (repair protein), leading to DIC [see Table 9]. Interestingly, the white blood cells capable of neutralizing MAT through MOBS production are the same ones capable of phagocytosis, the process by which foreign matter, waste products and microor­ganisms are collected and dumped in the liver.[57]

To summarize this section, pathological microforms and their acids create DIC by a number of pathways:

Leukotoxin (linoleic acid bound to mycotoxin) is highly toxic to cells. It causes platelet lysis, there­by releasing TF and initiating DIC.

The expression or release of sialic acid residues from healthy cells that have been disturbed allows for the fermentation of galactose, creating exotox­ins and mycotoxins, biochemically activating galactose oxidase, which further disturbs and dis­organizes healthy cells. This cycle loads the blood with debris.

EMPO and MAT disturb fibrinogen, which releas­es sialic acid and reduces the charge, allowing it to polymerize into fibrin monomer and fibrin nets.

The presence of exotoxins, endotoxins, and myco­toxins and their poisoning of cells activates the immune system. White blood cells generate MOBS (e.g., superoxide [0′2] or hydrogen perox­ide [H202]). These substances bind to and neu­tralize EMPO and MAT. MOBS are repelled by healthy endothelial cells and the basement mem­brane because of their negative charge. Cellular disturbances and disorganization stimulate the generation of fibrin monomer for repair purposes, leading to DIC.

Detection of Disseminated Intravascular Coagulation

The Sonodot Analyzer

The Sonoclot Coagulation Analyzer provides a reaction-rate record of fibrin and clot formation with platelet interaction. An axially vibrating probe is immersed to a controlled depth in a 0.4 ml sample of blood. The viscous drag imposed upon the probe by the fluid is sensed by the transducer. The electronic circuitry quantifies the drag as a change in electrical output. The signal is transmitted to a chart recorder which provides a representation of the entire clot for­mation, clot contraction and clot lysis processes. The analyzer is extremely sensitive to minute changes in visco-elasticity and records fibrin formation at a very early stage. The Sonoclot has been evaluated scientif­ically and shown to provide an accurate measurement of the clotting process.[58,59]

One application of the Analyzer has been the development of a test to distinguish non-advanced breast cancer from tumors that are benign. The ratio­nale for the test is the hypercoagulable state seen in cancer patients (Trousseau’s Syndrome), resulting from the generation of TF by leukocytes (mono­cytes).[60] (See Table 4.)

Fibrin Degradation
Products and Fibrin Monomer

DIC can be seen as a two-step process. First, fib­rinogen, which is always present in the blood, is acti­vated by any of several mechanisms. This activation leads to an automatic polymerization (chain forma­tion) resulting in fibrin monomer. This is not apparent in a microscope unless the blood is allowed to clot, as in the MOST.[61,62] The second step is the precipitation or deposition of fibrin (hard clot) by several other mechanisms. One of these is the formation of cross­links through the action of factor XIII. Another such mechanism may be poor circulation in an organ already blocked by deposited fibrin. The deposition of precipitated fibrin may be detected microscopically in tissue sections and diagnosed as DIC.[62]

Because fibrin monomer is not readily detected, a chemical test for it is of immense value in diagnosing DIC. Research has indicated that its detection may be very useful in the early diagnosis of DIC and MAT.[63] There are three fundamental physiologic areas related to blood clotting: (1) the prevention of blood clotting, (2) the clotting of blood, and (3) the removal of clotted blood once it has formed.

Enzymes are present that are capable of removing (lysing) clotted blood, one of which is plasmin. Another enzyme, plasminogen, is always present in the blood, but is inactive as a proteolytic agent. Plasminogen acti­vator converts plasminogen to plasmin, which can degrade deposited fibrin. This process is not specific for fibrin, however, and other proteins may be affected. When fibrin is degraded (fibrinolysis), fibrin monomer, as well as several other products, are formed. Commercial kits are available for the analysis of fibrin degradation. This test is an indirect measure of the pres­ence of DIC and MAT.[64]

Other tests include:

Protamine Sulfate: Protamine sulfate is a heparin binder sometimes used in surgery for excessive bleed­ing. The test, which indicates fibrin strands and fibrin degradation products, is conducted in a test tube, with fibrin monomer and fibrin forming early and polymer­ization of fibrin degradation products occurring later.[65] Ethanol Gelation: A white precipitate is formed by the addition of ethanol to a solution in a test tube containing fibrin monomer as a degradation product of fibrin, indicating DIC and MAT.[66]

The Mycotoxic Oxidative Stress Test (MOST)

Up to now, blood chemistries have been the prima­ry mode of diagnosis or analysis for the presence of pathology. In the view presented here, the bright-field microscope, is used to easily and inexpensively reveal a disease state as reflected by changes in certain aspects of blood composition and clotting ability. DIC is char­acterized by the abnormal presence in the blood of fib­rin monomer. When allowed to clot, blood containing such an abnormal artifact will exhibit distortions of normal patterns. The presence in the blood of soluble fragments of the extracellular matrix and soluble fibronectin, as well as other factors, will also create abnormal blood clotting patterns as described below.

A small amount of blood from a fingertip is con­tacted with a microscope slide. A series of drops is allowed to dry and clot in a normal manner. Under the compound microscope, the pattern seen in healthy subjects is essentially the same—a dense mat of red areas interconnected by dark, irregular lines, completely filling the area of the drop. The blood of people under mycotoxic/oxidative stress exhibits a variety of characteristic patterns which deviate from nor­mal, but with one striking, common abnormality: “clear” or white areas, in which the fibrin net/red blood cell conglomerate is missing.

BowelCancerLive Blood Dried Blood_0166

 

 

 

 

 

 

 

 

[Micrograph 3; An abnormal clot with striking ‘clear’ or white areas or protein polymerization as seen in the hyper coagulated blood of a patient with lower bowel imbalances]

Why the fibrin net is missing may be understood from the following: Two peptides—A and B—in the central protein bead of the fibrinogen structure become bound in the cross-linking process. There are two ways this can be configured: (1) Thrombin is capable of activating peptides A and B, resulting in the formation of a polymer loosely held together only by hydrogen bonds; (2) With peptides A and B acti­vated normally, the resulting hard clot is insoluble, indicating that the peptides are linked by covalent bonds. The difference in bonds results from factor XIII, an enzyme which links the two fibrin strands with a glutamine-lysine peptide bond.

Additional research has shown that the release of sialic acid from fibrinogen inhibits the action of factor XIII, resulting in a soft, white clot. In addition, acetic aldehyde has been shown to inactivate factor XIII directly. The soft clotting, compounded by other polymeric aggregations (described below), results in clear areas in the dry specimens. In the opposite extreme, high serum levels of calcium, for the pur­pose of neutralizing MAT, activates factor XIII, lead­ing to excessive cross-linking of fibrin to form a clot harder than normal. This is reflected in the MOST pattern characteristic of definite hypercalcemia— that of a series of cracks in the clot radiating outward from the center, resembling the spokes of a wheel. High serum calcium is the body’s attempt to com­pensate for the acidity of mycotoxic stress by pulling this alkalizing mineral from bone into the blood. This demand creates endocrine stress in turn, because reabsorption of bone is mediated by parathormone (PTH). Therefore, this clotting pattern indicates cal­cium deficiency and thyroid/parathyroid imbalance.

calciumpattern

 

 

 

 

 

 

 

[Micrograph 4: A mineral deficiency or more specifically a calcium deficiency pattern associated with an imbalance of they thyroid and/or parathyroid}

Advanced research has shown that there are seven carbohydrate chains in fibrinogen (each terminated by sialic acid). A second action of factor XIII is to ferment a large amount of carbohydrate during clot­ting. Because carbohydrate is most often water solu­ble, the loss of this material undoubtedly adds to the insolubility of a clot, while pathological retention contributes to the softness of the abnormal clot.

Clinical experience demonstrates that the MOST is a reliable indicator of exotoxic and mycotoxic stress and, concurrently, of various disorganizing symptoma­tologies associated with fermentative and oxidative processes. As various cellular degradation occurs, the blood-borne phenomena which accompany such symptoms as diabetes, arthritis, heart attack, stroke, atherosclerosis and cancer show up in the MOST, often with sialic acid beads in the clear areas of poly­merized proteins. (Determination of the liberation of sialic acid from carbohydrate has been approved by the U.S. Food and Drug Administration as an accept­ed indicator for cancer, and is clinically available.)

sialicacid

[Micrograph 5: Sialic acid beads are seen inside the protein
polymerization of the hypocoagulated blood as black dots]

The extent and shape of the clear areas are reflec­tive of particular symptomatologies which have arisen from the way in which the disease condition manifests in a given individual. This observation is borne out by having the patient undergo appropriate alkalizing therapy. With success of treatment based on the patient’s freedom from symptoms, sense of well-being, and live blood exams discussed in the main text of Sick and Tired, Reclaim Your Inner Terrain, Appendix C,[7] repeated analysis with the MOST reveals a progressively improving clotting pattern.

[Micrographs 6 and 7: Medically diagnosed cancer patient with large polymerized protein pools (PPP) in the hypo-coagulated blood above. In the picture below PPP’s have significantly reduced in size and the blood is moving to a more hyper-coagulated state as a result of reducing acid loads with an alkaline lifestyle and diet (7, 70)]

Because of its very nature, the MOST is emi­nently suited to reveal and measure the presence in the blood of abnormal substances, clotting factors, and disorganization of cells due to an inverted way of living, eating, and thinking, which gives rise to MAT. The MOST indicates both the direct and indirect activity of MAT on blood clotting, endothelium, and the extracellular matrix (described next), as well as on biochemical pathways, including hormonal ones. The generation of excessive MOBS in response to EMPO and MAT, the inability that accompanies all degenerative symptoms to neutralize or eradicate EMPO and MAT, and the recognized hyper- and hypocoagulable states seen in various symptomatolo­gies, will beyond doubt be revealed in the MOST.

Aspergillusnigercrystal

 

 

 

 

 

[Micrograph 8 and 9: Medically Diagnosed HIV/AIDS micrograph showing above an Aspergullus niger mold crystal using dark field microscopy and below a hypocoagulated blood clot with systemic protein polymerizations measuring in excess of 40 microns using bright field microscopy}

HIV

 

 

 

 

 

 

As mentioned, hormones are easily fermented, and this will show up as a hypocoagulated blood pattern in the MOST. It is my opinion, this hypocoagulated blood appears in the MOST as misty clouds of protein polymerizations throughout the clot, as seen in the accompanying picture.

poorfibrin

[Micrograph 10: Poor fibrin interconnection in the clot associated with endocrine or hormonal imbalance]

The MOST from Solubilized Extracellular Matrix

There is now a clearer picture of the biochemical rationale for correlating abnormal blood clotting patterns with the presence of degenerative symptoms.  A link between symptoms and the distorted clotted blood patterns has been delineated in the MOST.
Another reason for the abnormal clotting patterns accompanying pathological states, in addition to insufficient bonding of fibrinogen peptides as seen in the MOST, is presence in the blood of water-soluble fragments of the extracellular matrix.

Extracellular Matrix Degradation by MAT

The extracellular matrix (EM) is a three-dimen­sional gel, binding cells together and composed of five or more major constituents: collagen (protein), hyaluronic acid (polysaccharide), proteoglycans (pro- tein/polysaccharide), fibronectin and laminin. Also included are glycosaminoglycans and elastin.[67] In every degenerative disease studied by this author, evidence has been found for MAT activity destruc­tive of EM.

One of the proteolytic enzymes activated in response to EMPO and MAT is alpha-1 antitrypsin (capable of neutralizing MAT), normally not active in the presence of the enzyme trypsin. The active por­tion of this anti-exotoxin and antimycotoxin contains the amino acid methionine, which includes a C-S-C linkage. When chelated by the hydroxyl radical (one of the MOBS oxidants), methionine’s central sulfur atom acquires one or two oxygen atoms (forming the sulfone or sulfoxide respectively). The fermentation of methionine is a secondary effect of immune response to an alarming situation, intended to neutral­ize MAT and prevent degradation of the EM. Once alpha-1 antitrypsin is exhausted, MAT will have more access to the EM. If the EM is damaged beyond repair, then the enzyme trypsin is released to disorganize and recycle the cells involved.[68]

A similar scenario holds for the enzymes collage- nase and elastase. Thus, the absence of alpha-1 antitrypsin in the presence of EMPO and MAT activates three enzymes which degrade the extracellular matrix. Degradation of the EM by enzymes and MAT puts into the blood the water-soluble fragments (proteins and glycoproteins) of normally insoluble EM components (see Table 11). The presence of these fragments modifies the normal clotting pattern (described below), as seen in the M/OST, and is therefore an indication of EM degradation, which is always found with degenerative symptoms. (Also present is fibrin monomer, which has been found in the blood of patients suffering from collagen dis­ease.[69] See Table 11.)

Fibronectin is a molecule in EM having several binding sites for various long-chain molecules— heparin (a sulfonated polysaccharide) and collagen, for example. As such, it functions as a cellular glue, bind­ing cells together as well as various components of the EM. A soluble form of fibronectin is normally found free in the blood, and enters into the formation of a blood clot through the action of factor XIII. This form of fibronectin binds to fibrin. Elevated, bound-serum fibronectin results from EM fragmentation by MAT, and accompanies degenerative symptoms such as arthritis and emphysema (collagen diseases).

Water-soluble fragments of the EM bound by fibronectin form a three-dimensional network or gel in the pathologically clotted blood (fibrin and com­ponents of the blood clotting cascade). Since fibronectin binds to both fibrin and collagen, the two polymeric networks are superimposed and intermin­gled, resulting in a modification of the normal clot­ting pattern. Exactly how the pattern is modified depends upon the nature of the collagen abnormally present, the nature and extent of hyaluronate pre­sent, and the degree to which EM fibronectin has been released by MAT.

Conclusion

Thus, it is easily seen that there are many forms which the pattern of clotted blood may take, depending on the individual and the internal terrain that produced the modifying substances. The MOST reveals not only the presence of exotoxic and mycotoxic stress, but indicates as well the nature of the symptom(s) resulting from the stress (see Table 12). Since MAT underlie the entire complex of events which degrade the extracellular matrix, I must conclude that the absence of these exotoxins, endotoxins and mycotoxins would provide substantial improvements in tissue integrity and the overall physiology and functionality of the organism or animal and human.

­

­

References

[1]  Jones, T.W., “Observations on some points in the anatomy, physiology and pathology of the blood.”  British Foreign Medical Review, 1842. 14 : 585.

[2] Trousseau, A., Phlegmasis alba delens. “Clinque Medicale de L’Hotel Dieu de Paris.”, 1865, 3:94

[3]  Virchow, R., “Hypercoagulability: A review of its development, clinical application, and recent progress.”  Gesammelte Abhandlungen our Wussenschaftlichen Medizin, 1856, 26:477.

[4]  Rapaport, S.I., “Blood Coagulation and its Alterations in Hemorrhagic, and Thrombotic Disorders.”  The Western Journal of Medicine, 1993; 158: 153.

[5]  Hamilton, P.J. et al., “Disseminatied Intravascular Coagulation: A Review.”  Journal of Clinical Pathology, 1978, 31: 609

[6] The Harper Collins Illustrated Medical Dictionary, 1994, p.13.

[7] Young, RO, “Sick and Tired, Reclaim Your Inner Terraine,” Woodland Publishing, 1999.

[8] BeChamp, A., “The Blood and Its Third Anatomical Element,”  Hikari Omni Publishing, 1999.

[9]  Schwerdtle, C, Arnoul, F, Enerlein, G, “Introduction to Darkfield Diagnostics”, Semmelweis-Verlag (2006).

[10]  Hawk, BO, Thoma, GE, Inkley, JJ, The Evaluation of the Bolen Test as a Screening Test for Malignancy*, cancerres.aacrjournals.org on December 5, 2015. © 1951 American Association for Cancer Research.

[11]  Uchida, K., “Role of Reactive Aldehyde in Cardiovascular Diseases”,  Labortory of Food and Biodynamics, Nagoya University Graduate School of Bioagricultural Sciences, Nagoya, Japan , Free Radical Biology and MedicineVolume 28, Issue 12, 15 June 2000, Pages 1685–1696

 [12] Chang JCvan der Hoeven LHHaddox CH, “Glutathione reductase in the red blood cells”,  Ann Clin Lab Sci. 1978 Jan-Feb;8(1):23-9.

[13] Kutzing, MK, Firestein, BL, “Altered Uric Acid Levels and Disease States”, Department of Cell Biology and Neuroscience (M.K.K., B.L.F.), Graduate Program in Biomedical Engineering (M.K.K.), Rutgers University, Piscataway, New Jersey. Address correspondence to: Dr. Bonnie L. Firestein, Department of Cell Biology and Neuroscience, Rutgers University, Piscataway, NJ 08854-8082. E-mail: firestein@biology.rutgers.edu

[14] Claudino, M,. Ceolin,,DS, Alberti, S.,  Cestari, TM,  Spadella, CT, Fischer Rubira-Bullen, IR, Gustavo Pompermaier Garlet, Gerson Francisco de Assis, ” Alloxan-Induced Diabetes Triggers the Development of Periodontal Disease in Rats”,  Published: December 19, 2007. DOI: 10.1371/journal.pone.0001320

[15] Young RO (2015), “Alkalizing Nutritional Therapy in the Prevention and Reversal of any Cancerous Condition. Int J Complement Alt Med 2(1): 00046. DOI: 10.15406/ijcam.2015.02.00046

[16] Heloise Pöckel FernandesCarlos Lenz Cesar, and  Maria de Lourdes Barjas-Castro, “Electrical properties of the red blood cell membrane and immunohematological investigation”, Rev Bras Hematol Hemoter. 2011; 33(4): 297–301. doi:  10.5581/1516-8484.20110080 PMCID: PMC3415751

[17] Harris, JO, “The Relationship Between the Surface Charge and the Absorption of Acid Dyes by Bacterial Cells”, Department of Bacteriology, Kansas Agricultural Experiment Station, Manhattan,Kansas, Received for publication March 3, 195.

[18] Young, RO, “Metabolic and Dietary Acids are the Fuel That Lights the Fuse that Ignites Inflammation that Leads to Cancer”. https://www.linkedin.com/pulse/metabolic-dietary-acids-fuse-ignites-inflammation-causes-young. 2015.

[19] Snaders, R, “Did Bacteria Spark Evolution of Multicellular Life?” Berkeley News, Research, Science and Environment,  October 24, 2012.

[20] Wenner, M, “Humans Carry More Bacterial Cells than Human Ones”. Scientific American, November 30th, 2007.

[21} Animals and humans respond to MAT as a poison.

[22]  Morrison, D.C. et al. The effects of bacterial endotox­ins on host mediation systems. American Journal of Pathology, 1978; 93: 526.

[23]  Ibid.

[24]  Ibid.

[25]  Van Deventer, S.J.H. et al. Intestinal Endotoxemia. Gastroenterology, 1988; 94(3): 825-831.

[26]  Morrison, D.C. et al., op. cit.

[27]  Ibid.

[28]  Hu, T. et al. Synthesis of tissue factor messenger RNA and procoagulant activity in breast cancer cells in response to serum stimulation. Thrombosis Research, 1993; 72: 155.

[29]  Rapaport, op. cit. (Ref. 4).

[30]  Ibid.

[31]  Mackman et al. Lipopolysaccharides—mediated tran­scriptional activation of the human tissue factor gene in THP-1 monocytic cells requires both activator protein 1 and nuclear factor kappa B binding sites. Journal of Experimental Medicine, 1991; 174: 1517.

[32]  Yamada, O. et al. Deleterious effects of endotoxins on cultured endothelial cells: An in vitro model of vascular injury. Inflammation, 1981; 5: 115.

[33]  Colucci, M. et al. Cultured human endothelial cells: An in vitro model of vascular injury. Journal of Clinical Investigation, 1983; 71: 1893.

[34]  Cho, T.H. et al. Effects of Escherichia coli toxin on structure and permeability of myocardial capillaries.

[35]  Acta Pathologica Japonica, 1991; 41: 12.

[36]  Rapaport, op. cit. (Ref. 4).

[37]  Ibid.

[38]  Margolis, J. The interrelationship of coagulation of plasma and release of peptides. Annals of the New York Academy of Sciences, 1963; 104: 133.

[39]  23-25. Ibid.

[40]  Morrison, D.C. et al., op. cit.

[41]  Rapaport, op. cit. (Ref. 4).

[42]  Alberts, B. et al, eds. Molecular Biology of the Cell. New York: Garland Publishing, Inc., 1989 (2nd ed.), p. 818.

[43]  Rapaport, op. cit. (Ref. 4).

[44] Bertz, A., et al. Modulation by cytokines of leukocyte endothelial cell interactions. Implications for thrombo­sis. Biorheology, 1990; 27: 455.

[45]  Rapaport, op. cit. (Ref. 4).

[46]  Nachman, R.L. et al. Hypercoagulable states. Annab of Internal Medicine, 1993; 119: 819.

[47]  Ibid.

[48]  Tallman, M.S., et al. New insights into the pathogene­sis of coagulation dysfunction in acute promyelocytic leukemia. Leukemia and Lymphoma, 1993; IT. 27.

[49]  Silberberg, J.M., et al. Identification of tissue factor in two human pancreatic cancer cell lines. Cancer Research, 1989; 49: 5443.

[50]  Grimstad, I.A. et al. Thromboplastin release, but not content, correlates with spontaneous metastasis of can­cer cells. International Journal of Cancer, 1988; 41: 427.

[51]  Gunji, Y. et al. Role of fibrin coagulation in protection of murine tumor cells from destruction by cytotoxic cells. Cancer Research, 1988; 48: 5216.

[52]  Sugiyama, S. et al. The role of leukotoxin (9, 10- epoxy-12-octadecenoate) in the genesis of coagulation abnormalities. Life Sciences, 1988; 43: 221.

[53]  Ibid.

[54]  White, A. et al, eds. Principles of Biochemistry. McGraw-Hill Book Co., New York, 1964, p. 648.

[55]  Mueller, H.E. et al. Increase of microbial neu­raminidase activity by the hydrogen peroxide concen­tration. Experientia, 1972; 23: 397.

[56]  Young, Robert O. Fermentology and oxidology. The study of fungus-produced mycotoxic species and the activation of the immune system and release of microzymian oxidative buffering species (MOBS). Self- published: InnerLight Biological Research Foundation, Alpine, Utah, 1994.

[57]Chandler, WL. et al. Evaluation of a new dynamic vis­cometer for measuring the viscosity of whole blood and plasma. Clinical Chemistry, 1986; 32: 505.

[58]  Saleem, A. et al. Viscoelastic measurement of clot for­mation: A new test of platelet function. Annals of Clinical and Laboratory Science, 1983; 13: 115.

[59]  Spillert, C.R. et al. Altered coagulability: An aid toselective breast biopsy. Journal of the National Medical Association, 1993; 85: 273.

[60]  Bowie, E.J. et al. The clinical pathology of intravascular coagulation. Bibliotheca Haematologica, 1983; 49: 217.

[61]  Muller-Berghaus, G. et al. The role of granulocytes in the activation of intravascular coagulation and the pre­cipitation of soluble fibrin by endotoxin. Blood, 1975; 45: 631.

[62]  Bick, R.L. Disseminated intravascular coagulation. Hematology/Oncology Clinics of North America, 1993; 6: 1259.

[63]  Bredbacka, S. et al. Laboratory methods for detecting disseminated intravascular coagulation (DIC): New aspects. Acta Anaesthesiologica Scandinavica, 1993; 37: 125.

[64]  Sigma Diagnostics, St. Louis, MO 63178; tel: 314- 771-5765.

[65]  Nachman, R.L. et al. Detection of intravascular coag­ulation by a serial-dilution protamine sulfate test. Annals of Internal Medicine, 1971; 75: 895.

[66]  Breen, F.A. et al. Ethanol gelation: A rapid screening test for intravascular coagulation. Annals of Internal Medicine, 1970; 69: 1197.

[67] Hay, E.D., ed. Cell Biology of Extracellular Matrix. New York: Plenum Press, 1981, p. 653.

[68]  Carp, H. et al. In vitro suppression of serum elastase- inhibitory capacity by ROTS generated by phagocytos- ing polymorphonuclear leukocytes. Journal of Clinical Investigation, 1979; 63: 793.

[69]  Wilson, C.L. The alternatively spliced V region con­tributes to the differential incorporation of plasma and cellular fibronectins into fibrin clots. Journal of Cell Biology, 1992; 119: 923.

[70] Young, RO, Young, SR, “The pH Miracle Revised and Updated”, Hachette Publishing, 2010.

Tables

Table 1

Expression of Sialic Acid/Galactose [MAT] from Cell and Protein Degeneration (From All Serum Proteins, RBC/WBC and Other Cell Surfaces)

  1.  Carbohydrate, Proteins, and Fats From Diet, Body Cells or Reserves
  2. As cells breakdown or ferment they give birth to bacteria, yeast, fungus and mold [EMPO] and their associated metabolic acidic waste [MAT]
  3. Exotoxins, Endotoxins, and Mycotoxins [MAT]
  4. Acetyl Aldehyde, Ethyl Alcohol, Uric Acid, Alloxan, Lactic Acid are examples of MAT
  5. MAT  Ferments Other Body Cells and their Extracellular Membranes and Proteins
  6. MAT Modifies Glycoprotein
  7. Binds to liver Galactosidase
  8. Creating an Increase in Cell and Protein Fermentation and Degeneration and Increased Amounts of Exotoxins, Endotoxins and Mycotoxins [MAT]

Table1a

Table 2

Expression of Sialic Acid [MAT] From the Fermentation of Degeneration of Insulin Producing Pancreatic Beta-Cells in Type I, Type II and Type III Diabetes

  1. Pancreatic Insulin producing Beta-Cells with no or minimal Surface Sialic Acid [MAT]A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Diet
  2. Normal regulation of Insulin Production
  3. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choicesdd
  4. Leads to cellular fermentation and degeneration and the birth of EMPO
  5. This lead to increased abnormal amounts of MAT that the immune system, the alkaline buffering system and the elimination organs has to deal with
  6. Fermenting and degenerating Insulin Producing Beta Cells
  7. Giving Rise to Surface Cell Sialic Acid [MAT}
  8. Increased Amounts of Sialic Acid Activates the Immune Response [MOBS] and Sialidase [AB]
  9. Leads to Lowered or No Insulin Production
  10. Symptoms of Type I, Type II or Type III Expressed
  11. The insulin producing beta cells of the Islets of Langerhans express silica acid on their surface as a break down metabolite.  I have suggested that when insulin producing beta cells are physically disturbed by MAT they begin to disorganize and express sialic acid on the surface of the cell.  This indicates the death of the cell and insulin production will stop.

Table2a

Table 3

HIGH BLOOD PRESSURE, ATHEROSCLEROSIS, HEART ATTACKS, STROKES, and CONGESTIVE HEART FAILURE

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This lead to increased abnormal amounts of MAT that activates the immune system to chelate the MAT.
  4. Increased amounts of MAT will cause endothelial breakdown and the expression of Sialic acid.
  5. Increased Amounts of Sialic Acid and damage to the endothelial will cause a reduction in the negative surface-charge leading to the release of Glycoproteins.
  6. The release of Glycoproteins will cause the activation of Factor XII and the blood clotting cascade.
  7. This cause the creation and formation of fibrin monomers and the increase of Platelet Deposition out of the red blood cells for clotting purposes
  8. The immune system will activate and MOBS will be released as well as sodium bicarbonate, calcium, lipids and other alkaline buffers to reduce metabolic acidity.
  9. The build-up of fibrin monomers in the clotting cascade will lead to fibrin nets and clots causing an increase in blood pressure and the risk of blockages potentially causing a Stroke or Heart Attack.

Table3a

Table 4

DISSEMINATED INTRAVASCULAR COAGULATION RESULTING
FROM INTRACELLULAR DISORGANIZATION OR FERMENTATION WHICH GIVES RISE TO MAT
 AND EMPO

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This lead to increased abnormal amounts of MAT that activates the Tumor Necrosis Factor (TNF).
  4. Increased amounts of TNF activates the Tissue Factor Gene (TF)
  5. Increased Amounts of TF causes the release of Thromboplastin.
  6. The release of Thromboplastin activates the release of clotting Factors VII (VIIa) and trace amounts of Factor Xa into the blood.
  7. This activates the release of Factors IX and X to IXa and the increase of Factor Xa.
  8. The activation of the blood clotting cascade leads to Disseminated Intravascular coagulation and the clotting or thickening of the blood inside the blood vessels.
  9. The DIC or hyper-coagulation will mask the fermentation of healthy cells to unhealthy cells or cancerous cells.
  10. As the unhealthy cells or cancerous cells increase the body will go into preservation mode and begin forming fibrin nets to encapsulated these unhealthy cells to protect healthy body cells.
  11. As body and blood cells breakdown from MAT this causes an increase of MAT and EMPO leading to systemic latent tissue acidosis and a potential metastatic cancerous condition.

Table4a

 Table 5

DISSEMINATED INTRAVASCULAR COAGULATION RESULTING
IN CELLULAR DISORGANIZATION OR FERMENTATION/OXIDATON AND THE INCREASE OF MAT AND EMPO

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices.
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This lead to increased abnormal amounts of MAT that activates the Tumor Necrosis Factor (TNF).
  4. Increased amounts of TNF activates the Tissue Factor Gene (TF)
  5. Increased Amounts of TF causes the release of Thromboplastin.
  6. The release of Thromboplastin activates the release of clotting Factors VII and Factor Xa in the blood.
  7. This activates the release of Factors IX and X to IXa and the increase of Factor Xa.
  8. The activated blood clotting cascade leads to Disseminated Intravascular coagulation and the clotting or thickening of the blood inside the blood vessels.
  9. The DIC or hyper-coagulation will mask the fermentation of healthy cells to unhealthy cells or cancerous cells.
  10. As the unhealthy cells or cancerous cells increase the body will go into preservation mode and begin forming fibrin nets to encapsulated the unhealthy cells.
  11. This leads to tumor formation of the unhealthy or cancerous cells.
  12. As the body and blood cells breakdown this causes an increase of MAT and EMPO leading to an increased risk of  systemic metastatic cancer.

Table5aTable 6

ENDOTHEIAl CELL CONVERSION FROM AN
ANTITHROMBOTIC STATE TO A PROCOAGULANT STATE
CELLULAR DISORGANIZING PATHWAY

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This leads to increased abnormal amounts of MAT that damages the protective endothelial cover cells leading to a reduction of PGI2
  4. The absence of PGI2 causes the release of Interleukin-1 and/or Tumor Necrosis Factor (TNF).
  5. In addition the loss of protective endothelial cover cells leads to Tissue Factor Gene Activation and the release of Thrombin causing a pro-coagulate state leading to DIC
  6. Another pathway to DIC would be the loss of protective endothelial cover cells and the absence of PGI2 causes the suppression of Thromomodulin, Protein C leading to procogradulation and DIC.

Talble6

 Table 7

ENDOTHELIAL CELL CONVERSION
FROM AN ANTITHROMBOTIC STATE
(NORMAL PATHWAY)

Table7

Table 8

MECHANISM OF DISSEMINATED INTRAVASCULAR COAGULATION GENERATED BY MAT

Table8Table 9

ACTIVATION OF SIALIDASE AND MICROZYMIAN OXIDATIVE BUFFERING SPECIES (MOBS) BY EMPO AND MAT

Table9

Table 10

DISSEMINATED INTRAVASCULAR COAGULATION RESULTING FROM PHAGOCYTIC OXIDATIVE BURST

Table10

Table 11

MOST BLOOD TEST and DISSEMINATED INTRAVASCULAR COAGULATION WITH SOLUBILIZED EXTRACELLULAR MATRIX

Table11

Table 12

TYPICAL SOURCES OF FERMENTATION INSULT (MAT) IN BIOLOGICAL SYSTEMS INITIATING DIC

Table12

Table 13

POSITIVE CHARGE OF CANCEROUS CELLS AND TUMORS AND THE FORMATION OF FIBRIN NETS AND TREES IN RESPONSE TO MAT

Table13

What Happens to ALL the Disappearing Can

12311128_1639530499647197_8412997691509003664_nWhat Happens to ALL the Disappearing Cancer Patients?

It’s been almost 20 years since I met my first disappearing patient — a nurse in her early 40s, let’s call her Kate. Kate was diagnosed with breast cancer. As a nurse, she had seen the results of breast cancer treatments. She was terrified, and determined. She was not heading for surgery, nor chemotherapy, nor radiation.

But Kate worked in a hospital. She worked with the doctors who diagnosed her cancer, and she worked with the surgeon, who wanted to schedule her into surgery “as soon as possible.”

The first thing Kate did was slow down. She did some research. It didn’t take her long to remind herself that in Canada, and in the USA, the treatments for cancer are akin to law. No hospital would dare deviate from the deadly three (cut, poison, burn).

Kate’s cancer was not large. She had been tested for cancer last year and no cancer was found. She knew it took many years for cancers to develop. At first, she was furious, “If it is here today, it must have been here last year. Why didn’t you find it last year?” It had not metastasized. It was not growing rapidly and was not affecting her health in any way. In theory, she had lots of time. So, she took some time.

But Kate didn’t look for magic cures. She didn’t search for the latest “cancer medicine.” She wasn’t interested in curing herself. She knew she was a nurse, not a doctor. She searched instead for the “cured” – patients who were diagnosed with cancer, and no longer had cancer. She knew from her work in the hospital, from conversations with patients, and with some staff, that these people existed — but from the perspective of the medical establishment, they seemed to disappear.

It didn’t take her long to find some patients who claimed they were cured. They hadn’t disappeared from life. They were eating, drinking, loving, and living full healthy and prosperous lives. But according to the medical records, they didn’t exist. They were “never cured.”

The medical system treated their cures as “anecdotal.” It ignored them. There was no attempt by any doctors to understand what happened to these cancer patients. They were no longer sick. The medical system looks after sick patients, treats sick patients. These patients were not sick.

Kate looked and listened. Her interest was not clinical science vs. anecdotal evidence. Her interest was personal. She talked, listened, compared stories. From several, she learned about a clinic that did not claim to cure cancer. It did not use medicines to treat cancers. But patients were cured, somehow. This clinic was not in Canada. It was not in the USA. She would have to go to Mexico to learn more.

There are lots of alternative treatment clinics in Mexico. Are some of them valid, using important techniques to cure cancers? Are some of them scams, wanting to take money from desperate clients? Do some of them have a cure that works sometimes, but might not work for her? Kate didn’t know. She did more research. She called the clinic.

The staff did not claim to cure cancer. Claiming to cure cancer is dangerous, even for a clinic outside of North America. They suggested Kate visit the clinic and see what happens there, no charge for a visit, but she would need to pay for her travel to Mexico. Kate had done her research. She had met and talked to patients whose cancers had disappeared.

Kate made her decision. She was familiar with cancer diagnosis techniques in Canada. She had undergone a physical examination, a mammogram, that detected a lump in her breast. Then she had a biopsy, where tissue was taken from the lump and was sent to a lab for analysis. The lab technician tested and examined the sample and issued a diagnosis “cancer” or “not cancer.” Once the diagnosis is issued, everybody swings into action. Kate knew that the mammogram had a high false positive rate and a false negative rate. Many people who are diagnosed with a “possible cancer” by a mammogram do not actually have cancer. She was also aware that cancer biopsies have a false positive rate and a false negative rate, as well. Her work in the hospital, with real patients, had made this very clear.

She didn’t really know for certain if she had cancer. Her surgeon, on the other hand, was still pressing her to schedule treatment.

Kate knew one thing. She had time. She cashed out some savings and booked a “holiday” in Mexico.

At the clinic, Kate was surprised that there was no “cancer diagnosis.” They did check the presence and size of the lump on her breast. But they didn’t repeat the biopsy. The clinic read her diagnostic reports, but did not investigate them further. There was instead a very thorough analysis completed by a suite of doctors. It took two full days of tests and interviews, if I remember correctly.

Kate was asked about her family’s medical histories. She gave blood samples. She was questioned extensively about her diet, about what she eats on a regular basis. What foods does she like and eat often. What foods does she not like and never eat. Doctors examined her lungs, her heart, liver, and other bodily organs with various tests. Her immune system was tested. Extensive interviews about her life, her work, her relationships, and more.

At the time I talked to Kate, I didn’t realize that she was not getting a “medical analysis,” she was actually getting a “healthicine analysis.” Her tests and questions fit perfectly to the hierarchy of healthicine: genetics, nutrition, cells, tissues, organs, bodily systems, body, mind, spirit, and community.

Kate’s genetics were analyzed through family history. There may have been further genetic analysis, I don’t remember all of the details. Her nutritional status was analyzed, not just by analyzing what she ate, and what she preferred to eat, but also by studying what she didn’t like to eat, what she deliberately never ate, what foods she believed she was allergic to. Her cells and tissues were analyzed directly, through blood samples and physical examination, and indirectly through medical history and other tests. Many of her organs were tested for healthiness. Her bodily systems, immune system, circulatory system, respiratory system, hormonal systems and more were analyzed and assessed. Her physical body was measured, weighed, and examined. Her mental health was assessed, as well as her spiritual healthiness. She was in good spirits, even in light of a potentially life threatening illness. Her community health was analyzed as well. Her family, her relationships with her children, her spouse, her parents, her work community, and more.

After a few days, Kate met with a group of doctors to discuss her health, not her illness, her healthiness. Diagnosing illness is difficult. Analyzing healthiness is more complex. It took several doctors and several hours for Kate to learn and understand what they had learned about her healthinesses and her unhealthinesses.

They then “prescribed” two weeks, if I remember correctly, of healthiness training, tailored to Kate’s specific situation. She spent the next two weeks at the clinic, learning to be healthier, not learning how to be “healthier in principle,” rather – learning what Kate needed to do to make her diet, her body, her mind, her spirits, and even her relationships with her communities healthier. She could not change her work community. But she could change how she reacted to and interacted with it – to improve her own health. After two weeks of learning at the clinic, her breast lump had started to shrink.

Kate went back to Canada, to put her learning into action. The lump disappeared. Her diagnosis was still there on paper. But her “cancer” had disappeared. She was retested at her hospital and no cancer was found.

Then Kate began to disappear.

When the surgeon asked again, she explained that she was not going to surgery. The surgeon looked away. He refused to look her in the eye after that.

But Kate didn’t disappear from her family. She went back to her family. She didn’t disappear from her job. She went back to her job. She disappeared from the cancer system. Her cancer disappeared, so, as a cancer patient, she disappeared.

Was she cured? We don’t know. There is no useful definition of a cancer cure. No medical or scientific test that can prove a patient has been cured of cancer. Our cancer treatment statistics have no count for people who are cured of cancer. Patients that are cured, whether they are cured with medicines or not, are not counted. No breast cancer patients are officially cured by medicine. If their cancer goes away without treatment, they disappear from statistics. If their cancer is killed by radiation, chemotherapy or surgery, they are not cured, they are a “survivor.” Everyone knows that cancer survivors are always waiting for the cancer to reappear. Their symptoms are in remission, but their cancer is not cured. They are not cured. With no proof of a cure, it might just be hidden.

Kate no longer has cancer. She paid, from her own pocket, for her trip to a clinic in Mexico. After the trip, her cancer disappeared. She had medical insurance. But her insurance wouldn’t pay for her trip. Insurance pays for treatments, not for cures. It pays for treatments, even if they fail. But it does not pay for success. Success disappears.

There are two ways for a cancer patient to disappear. You might be cured by health. Or you might be cured by a medicine that is not approved. In both cases, the medical system will ignore the cure, and ignore the patient.

In healthicine, there are no incurable diseases. If it is not curable – it is not a disease, it is a handicap, a disability, a deficiency, or simply an attribute of the person. All diseases can be cured by definition.

I have since met several cancer patients who have disappeared, and not just cancer patients. Maybe you have too? I’ve met more by internet, email, etc. There is no way for me to determine if a disappeared patient actually had cancer, if their treatment cured their cancer, if their body cured their cancer or if they still have cancer. We can only tell if there is another cancer diagnosis. Nothing can be told from the absence of a diagnosis.

There is no way for any doctor to tell either. There are no tests for a cancer cure. There is no way to recognize, much less document a cancer cure. There are no statistics for cancers cured.

Many cured patients don’t disappear quietly. They speak out. They write books and newspaper articles. They blog. But it doesn’t matter. They still don’t count. Once cured, they disappear. The medical system does not study their cases, does not study their diagnosis, does not study their cures. For chronic diseases, like cancer, arthritis, diabetes, heart disease, even obesity, and many more, there are no techniques to document “cured patients.” As a result, there are no statistics for “cured patients” of any chronic illness.

Once they are cured, they disappear. Health doesn’t cure illness, it disappears illness. And medicine doesn’t count people who have disappeared.

To your health

Ten Acidic Signs That Your Liver is Toxic and Sick!

liver-disease-s1a-did-you-knowLiver disease is any disturbance of liver function that causes illness. The liver is responsible for many critical functions within the body and should it become dis-eased or injured, the loss of those functions can cause significant damage to the body.  Liver dis-ease is also referred to as hepatic dis-ease.

Liver dis-ease is a broad term that covers all the potential problems that cause the liver to fail to perform its designated functions. Usually, more than 75% or three-quarters of liver tissue needs to be affected before a decrease in function occurs.

The liver is the largest solid organ in the body; and is also considered a gland because among its many functions, it makes and secretes an alkaline substance called bile. The liver is located in the upper right portion of the abdomen protected by the rib cage. It has two main lobes that are made up of tiny lobules. The liver cells have two different sources of blood supply. The hepatic artery supplies oxygen rich blood that is pumped from the heart, while the portal vein supplies alkalizing minerals from the large intestine and the spleen.

Normally, veins return blood from the body to the heart, but the portal vein allows alkaline minerals from the large intestines to enter the liver for “detoxification” and filtering prior to entering the general circulation. The portal vein also efficiently delivers minerals and fats that liver cells need to produce the proteins, cholesterol, and electrons required for normal body activities.

There are several early signs of  an acidic liver to understand in order to protect the liver and its many functions from sickness and dis-ease.
Without a fully functioning liver,  your health and wellbeing will be compromised.  Fortunately your liver is capable of repairing and renewing itself every six weeks.  Understanding the following acidic liver conditions and spotting them early,  will help to prevent and/or reverse a serious life-threatening degenerative live dis-ease.

livertoxicity

Warning Sign # 1 – Skin discoloration – Jaundice

One of the early signs of excess liver acidity and the beginning of liver dis-ease is the liver’s inability to filter out all of the dietary and/or metabolic toxins from the blood.  With a build-up of toxins this may also lead to a build-up of Bilirubin which is a breakdown product of the blood.  The breakdown of the blood which increases bilirubin is caused by an acidic lifestyle, diet, congested liver and gallbladder and constipation of the elimination organs,  The body and specifically the gallbladder uses bile  to help alkalize the food ingested coming out of the stomach.  When the body cannot evacuate Bilirubin from the liver/gallbladder and blood via the bowels, it will accumulate in the bloodstream and results in the skin taking on a yellowish hue.  This yellowing can also affect the fingernails, the tips of the fingers, and especially the eyes. This acidic condition caused by an acidic lifestyle and diet is known as Jaundice.  Read, share and like more:

Continue reading Ten Acidic Signs That Your Liver is Toxic and Sick!

A Self-Care to a Self-Cure for Terminal Metastatic Breast, Lung, Liver, Lymphatic, Lymph and Bone Cancer!

Watch the following YOUTUBE Video of Catherine Livingston sharing her incredible life-changing and life-saving Metastatic Cancer Reversal story!

Catherine Livingstone was diagnosed with breast cancer in 2011 that metastasized to her lungs, liver, lymphatic system. lymph nodes and bone. She was given only 2 months to live in 2012. She started the pH Miracle Lifestyle and Diet!  It is now June, 2015 and she is in complete remission with NO CANCER in the lungs (over 25 tumors), NO CANCER in the liver (the liver was full of tumors with to many to count), NO CANCER in the lymph nodes, NO CANCER in the bones and in fact NO CANCER anywhere in her body. Catherine is healthy, happy and NOW CANCER FREE living the pH Miracle Lifestyle and Diet!

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In her own words, “I intend to live a long and healthy life free from cancer thanks to the pH Miracle lifestyle.”

To learn more read The pH Miracle book 1, The pH Miracle revised and updated book 2, Reverse Cancer NOW!, The pH Miracle for Cancer, and Sick and Tired by Dr. Robert O. Young – www.phoreveryoung.com, www.phmiracle.com, www.amazon.com and www.barnesandnoble.com

Support Dr. Robert O. Young and his fight for Health Freedom to choose your health care, your own doctor and your own treatment whether it be conventional, traditional or integrated!

http://www.phmiracleliving.com/t-Innerlight-Foundation.aspx

Share Dr. Robert O. Young and his revolutionary life-changing and life-saving research with all those who you love and care about at:

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www.phoreveryoung.com and www.phoreveryoung.wordpress.com

The Cause and Self-Care to a Self-Cure for Breast, Ovarian, Uterine, and Prostate Cancer

A rise in the alkalinity of the blood above pH 7.365 (alkaline phosphate)—any rise—is a result or a compensatory reaction due to over-acidity in body tissues as the blood attempts to maintain pH balance. There is no exception for the rule of alkalinity. The body will ALWAYS overcompensate for the excess acidity in the tissues by over-alkalizing the blood to maintain homeostasis. I call this the “teeter-totter” effect. Along comes the traditional medical attendant and perceives that there is too much alkalinity, when really there is not.

This is an important concept to grasp, so let’s oversimplify a bit. The tissues have become acidic. The blood “knows” that. So, it pours out extra alkalinity or alkaline phosphate into the blood and the blood pH spikes up to a higher than normal pH. It’s like when we get the bejeebers scared out of us by something innocent, we over-react. When suddenly alarmed, a person might scream, holler, faint, get mad, strike out, drop the vase, kick the dog, or even have a heart attack. The blood does the same thing. A knee-jerk reaction…well, actually, a blood-jerk reaction.

Alternatively, how many times have you heard of a car going off the shoulder of the road and the driver over-reacts, jerks the wheel back, and flies into the other lane of oncoming traffic. It happens all the time. Incidentally, if that does happen to you, you’re better off not to interfere. Stay on the shoulder. Let the wheel stay there for a moment. Slow the car down. But don’t overreact.

Mainstream medicine, not understanding the cause of the excessive alkalinity pouring into the body, may try and stop the rushing over-alkalization. But that’s the wrong move. We’re better off not to interfere.

Once more. When your little boy falls down, sees mama going out the door, or is scared of the boogey man, what happens? He not only cries, but how often do we see a child go into a big, fat over-reaction? Sometimes, they really get worked up. It’s a natural over-reaction to a typical situation.

Now it’s Dad’s turn to over-react. Along comes Dad and says to keep quiet, shut-up, don’t be such a little sissy, put a lid on it, grow up, stop that crying, OR ELSE…

Since I have digressed to make a point, I may as well digress all the way. Wrong move, Dad. If you do that often enough, the message you send to your child is don’t have feelings, don’t express your feelings, you are not acceptable, don’t act like a child even though you are a child, and don’t be who you are. So don’t over-react Dad. Better to let the child get it out, stay in the room, validate their feelings, and use a little Active Listening (www.gordontraining.com). Strong feelings can come and go…or come and stay. If you’re really klutzy, you could be orchestrating chronic emotional issues for a lifetime. Gee, thanks Dad.

Now, back to your blood. Tissues are acid. Here comes a flood of alkalinity—even so much that the pH rises and concerns the western medical establishment. But whatever it was that caused the pH to over-react must be understood. Acidic tissues mean problems ahead, correct? Not only do we need alkalinity but lots of it. The acid tissues will soon even out the rise in blood pH, and we will need additional alkalinity to wipe out the acidic tissue problem.

Liver cancer is not a disease of alkalinity but a disease of acidity. The body uses the calcium of the bones as well as other buffers (bicarbonate, hemoglobin, sodium, etc.) to chelate acidity! That is why there are always microcalcifications in the liver before the liver cancer tumor shows up. Why prior to the tumor? Because the body will always try to protect and preserve itself by buffering acids with the alkalinity of calcium. The bones are always affected in any cancer because the bones are an excellent source for calcium.So is liver cancer the disease? No.

Then is the loss of bone mass the disease or the calcium deposits in the liver the disease. NO?

Is the increase in the alkaline phosphates the disease? NO! NO!

These are all symptoms, not diseases!

Then the disease must be the over-acidity? Well yes, and well no.

Then what is the disease? The “yes” part I call acidosis or hyperacidity. That is an acceptable term for the condition. But it is really much more. The “no” part is that it’s more than acidity. It’s a psychological disorder. It’s a sociological malaise. It’s a cultural-anthropological phenomenon. And once people understand the truth and the scientific foundation of New Biology, and once people understand the science of what I have been writing about for two the better part of two decades, it may than become to be understood as a “moral disease” as well.

And why is that, you ask? Is committing suicide a moral issue? Well, yes. Is drinking yourself to death a moral issue? Well, yes. Is allowing your children to become obese flying in the face of natural law? Well, yes, assuming you are aware of what’s happening and have other options.

If you say “yes” to these last few questions, then we are looking at a very, complex psychological, sociological, cultural, biological and moral phenomenon. Once you know and believe that over-acidity causes every disease and most dis-ease, then to ignore that fact is a form of suicide. When you eat poorly, you pull the trigger every day of your life, and eventually, the gun fires. The bullet might hit you square in the head like a massive heart attack, or it may kill you more slowly like a cancer, or it may simply put you in a fog for the next 15 years like Alzheimer’s or dementia.

This “disease-phenomenon” is an inverted way of living, eating and thinking!!! Yes, this is the cause of ALL disease—ALL that disturbs the central balance of organized matter that leads to excess acidity. It is ALL that leads to increases in alkaline phosphates. It is ALL microcalcifications in the liver, ALL liver tumors, ALL liver cancer and ALL potential bone cancer!!!!

First, we must understand that ALL of the above sicknesses and diseases are NOT sicknesses or diseases but a symptom of Acidosis and catarrh that has built up in the blood and tissues that has significantly effected the white blood cells’ ability (the janitorial and garbage collectors for the blood and tissues) to remove metabolic acids and morbid matter. When we are dealing with any symptom or any effect, we need to look to the cause. To understand the cause is not difficult nor is the understanding of the treatment. “New Biology” explains the cause and effect of all sickness and disease in addition to explaining how to improve the quality and quantity of life.For example, enervation (the deprivation of force or strength) and muscle weakness per se is not a disease. Weakness, or lost power, is not a disease; but, by causing a flagging of the elimination of tissue-waste which is toxic, the blood becomes charged with acids.

I call this Acidosis—poison in the blood and tissues. This is disease and when the toxin accumulates beyond the toleration point, a crisis takes place. This means that the poison or acid is being eliminated—often through the skin, the third kidney. We can call this disease, but it is not. The only disease is systemic Acidosis which localizes in the weakest parts of our body.

And what we call disease is symptoms produced by the forced vicarious elimination of acids through the mucous membrane. When the elimination takes place through the mucous membrane of the nose, it is called a cold—catarrh of the nose. And where these crises are repeated for years, the mucous membrane thickens and ulcerates, and the bones enlarge, closing the passages. At this stage, hay fever or asthma develops. When the throat and tonsils, or any of the respiratory passages, become the seat of the crises of acidity, we have croup, tonsillitis, pharyngitis, laryngitis, bronchitis, asthma, pneumonia, etc.

When the acids locate in the cranial cavity we have dementia, Parkinson’s, Alzheimer’s, muddle thinking, forgetfulness, and even depression. When the acids locate in the gastrointestinal tract we have IBS, gastrointestinal dysmotility, autonomic dysfunction, carotid stenosis and ischemic colitis. When the acids are expressed through the skin we have psoriasis. When the acids locate in the liver tissue we have microcalcifications of these acids that lead to tumors and liver cancer.
What’s in the name? All are symptoms of the expulsion of acids from the blood and tissues at the different points named. They are of the same character essentially and evolve from the one cause, namely, systemic Acidosis, a crisis of toxemia.

The description can be extended to every organ of the body, including the largest organ, the skin. For any organ that is enervated below the average standard from stress of habit, from work, or worry, from injury, or any other cause, that organ may become the location of the crises of systemic Acidosis. The symptoms presented differ with each organ affected. That fact gives color to the erroneous belief that every symptom-complex is a separate and distinct disease. But, thanks to the new light being shed by “New Biology” upon nomenclature involved in the naming of a disease, every symptom-complex goes back to the one and only cause of all diseases, namely, systemic Acidosis.

To find the cause of all symptomologies including liver and bone cancer, start with colds and catarrh, and watch the pathology as it travels through the seven stages of acidity, from sensitivity, irritation (IBS), catarrh, inflammation, induration (lupus), ulceration and then to degeneration—cancer. How well could you try to find the cause of man by ignoring his conception, embryonic life, childhood, manhood, etc.Nature’s order is interfered with by enervation habits until Acidosis is established. Then a vaccination (seen in Gulf War Syndrome and Spanish Flu Epidemic) or an infection (an outfection) from any source will act as a firebrand. Sooner or later cause the most vulnerable organ (the bowels) will undergo organic change. The organ, however, has nothing to do with cause, and directing treatment toward the organ compounds the problem and is nonsense. Examples of this wrong thinking yield blood transfusions for pernicious anemia, gland treatment for gland impotency, the cutting out of stones, ulcers and tumors.

There is no question that one of the most pernicious practices in vogue today is treating so-called disease with disease and immunizing with the products of disease. Current medical science calls this form of pathological thinking a “vaccination.”When the cause is not known, how is prevention or cure possible except by luck? Producing a mild form of smallpox using vaccine is the same as introducing a poison into a healthy person. It makes no sense. Certainly only pathological thinking can arrive at such conclusions. Vaccine or autogenous remedies (metabolic acids) are made from the products of disease. The idea that disease can be made to cure itself is an end-product of pathological thinking! If prevention and cure mean producing disease, surely prevention and cure are not desirable. If prevention can be accomplished, then cures will not be needed!It is not disease, it is cause “in all its aspects” that we need to know before we can take steps to prevent or cure “disease.”Cause is constant, ever present, and always the same. Only effects, and the object on which a cause acts, change. And the change is most inconstant.

To illustrate: a catarrh of the stomach presents first irritation, then inflammation, then ulceration, and finally induration and cancer. Not all cases run true to form. Only a small percentage evolve to ulcer and fewer reach the cancer stage. More toxins exit via acute food poisoning or acute indigestion then by chronic diseases. Most Americans are challenged with the symptomology of indigestion, which can include acid reflux, diarrhea and/or constipation.The proper way to study disease is to study health and every influence favorable or not favorable to its continuance. Our western system of medicine has been preoccupied with the study of disease, not health. Disease is perverted health. Any influence that lowers energy becomes disease producing. Disease cannot be its own cause, neither can it be its own cure and certainly not is own prevention!

My personal discovery of the truth of ALL sickness and disease—that Acidosis is the cause of all so-called diseases—came about slowly, step by step, line upon line, precept upon precept, here a little and there a little.At first, I postulated that yeast and molds must be the general cause of disease. Then I decided that it was not yeast and molds but that the body becoming enervated. But wait a minute, enervation is not a disease; disease must be due to metabolic acids. I reasoned that localized or systemic Acidosis is the true general cause of all disease and must be autogenerated. And if disease is due to autogenerated acids, what is the cause of that autogeneration?

The answer is found in understanding the nature of matter and how it organizes and disorganizes. I realized that there must be a physical or emotional disturbance to organized matter before it can begin its disorganization. And when matter begins to disorganize, it gives rise to autogenerated acids. This is true for all matter!

To illustrate, take a physical injury to a joint which is often complicated with the prior symptom of rheumatism. The rheumatism previous to the injury was potentially in the blood and/or tissues. Just what change had taken place in the matter which, under stress of injury or shock of any kind, would cause a reaction with fever? I could not understand until the “Acid Theory” suggested itself to my mind. After that, the cause of disease unfolded before me in an easy and natural manner. I called this new paradigm for ALL sickness and disease “The Cycle of Imbalance.” You can read about “The Cycle of Imbalance” in my book, “Sick and Tired, Reclaim You Inner Terrain.” You can order this book at: www.phmiracleliving.com.

In a few words, without Acidosis, there can be no sickness or disease and there can be NO CANCER! It is also true that without Acidosis there can be NO PAIN! Therefore, pain equals acid and acid equals pain. I knew that the waste products of cellular disorganization and metabolism were toxic and that the only reason why we were not poisoned by it was because it was removed from the organism as fast as it was produced.

Then I discovered that the acid was retained in the blood and tissues when there was a checking of elimination. Then, the cause of the checking had to be determined. In time, I thought out the cause of all sickness and disease. I knew that when we had normal energy, organic functioning was normal. Then came the discovery that enervation caused a checking of elimination.

Eureka! The cause of ALL sickness and disease is NOW found! Enervation checks elimination of the waste-products — ACIDS — of cellular disorganization and metabolism. Retention of metabolic ACIDS is the first and the only cause of sickness and disease!

One of the first things to do to get rid of any so-called disease is to get rid of all the acid, for it is this state of the blood and tissues that makes disease possible. Infection, drugs and food poisoning may kill, but if they do not, they will be short-lived in a subject that is free from enervation and acid. Conversely, the poisoning will linger in the system until the acid is overcome. Then and only then will elimination remove all traces of outfection.

Syphilitic outfection is pronouncedly an acidic subject thrown into great virulence by poor nutrition, lifestyle and conventional treatment. The same is true with HIV/AIDS. The so-called infection is the least offender of the trio. Add fear (false evidence appearing real) and wrong eating and we have a formidable symptom complex that serves to justify all that professional syphilomaniacs say and write about the disease. Remove Acidosis, drugging, fear, and vile eating, and there is little left. What is left can be easily thrown out of the body by Nature!Scientific research is being carried on vigorously in an attempt to find the cause of disease. The conception of disease is that the cause is individual. Here is where investigators meet their Waterloo. All of the so-called diseases are increasing symptom complexes due to repeated crises of Acidosis. They have no independent existence! As soon as acidity is controlled, the symptoms disappear unless an organ has been forced by innumerable crises to degenerate. Even organic change, when the organ is not destroyed, will come back by correcting the life and getting rid of the true cause—the crisis of Acidosis!

All symptoms of all so-called diseases have one origin. All diseases are ONE! Unity in all things is Nature’s plan. Polytheism is gone, and everything pertaining to it and coming out of it must go.So there is only one sickness, one disease, and NOW one treatment. The one sickness and disease is the over-acidification of the tissues and then blood due to an inverted way of living, eating, and thinking. The one treatment is to alkalize and energize with Dr. Robert and Shelley Young’s pH Miracle Living Plan. You can learn more about this program on our website or in our books, The pH Miracle, The pH Miracle for Diabetes, The pH Miracle for Weight Loss, Back to the House of Health I and II and Sick and Tired which you can also purchase through Amazon.com, Barnes and Noble or Borders Books.

The complete program is a 12 week program that includes the foods outlined in the foundational section of Shelley’s book, “Back to the House of Health.” You start off the program with a 10 to 14 day liquid feast. You can eat as much and as often as you like as long as the food is green and purred. The soups found in Shelley’s book such as the Broccoli Soup, Aspar/Zinc Soup, The Healing Soup and the Popeye Soup with lots of avocados are excellent to eat during the liquid feast. You also need to begin taking the nutritional supplements while drinking at least 4 to 6 liters of Greens a day. Start out gradually drinking 1 liter of Greens per day and then work up to 2, then 3, then 4, until you are drinking 6 liters a day.

When you take the nutritional supplements, take 5 drops 6 times a day of the liquid colloids under the tongue, (except the pH drops which are taken in purified water and NEVER taken under the tongue) away from meals, or taking 1 capsule 6 times a day of the capsule products with meals. I would suggest taking 4 capsules every 4 waking hours of the bowel cleansing formula. The bowel cleansing product helps to keep things moving through normal elimination.

After you complete the 10 to 14 day liquid feast, you can then begin introducing some solid food but it still needs to be as green as possible. I would suggest not only the vegetable soups, but steam fry vegetables and lots of salads. Make sure you use only lemon or lime and good oils on your salads for the dressing. Another tip is to include liberal amounts of flax and olive oil in or with your soups and salads. I suggest a minimum of 5 to 6 tablespoons of good oils each day.

In conclusion, the medical world has been looking for a remedy to cure disease, notwithstanding the obvious fact that nature needs no remedy. She needs only an opportunity to exercise her own prerogative of self-healing.Cures! There are NO cures! The subconscious builds health or disease according to OUR ORDER. If we send impulses of irritation, discontent, unhappiness, complaining, hate, envy, selfishness, greed, lust, and the biggest one of all pride, the subconscious builds us in the image of OUR ORDER!

The truth is that we need no doctor. We need to empower ourselves to effect a reconciliation between our subconscious creator and ourselves. What we need is to learn self-control, respect, poise, and relaxation! And when these impulses are sent over the sympathetic nerves to our subconscious creator, we will begin to receive images of a more ideal man or woman, until an approach to Perfection is Attained.

Sickness and disease, including the symptoms of cancer, tumors, AIDS, diabetes, MS, lupus, HIV/AIDS, depression, hyperthyroidism, Wilson’s Syndrome, fybromyalgia, pain in every joint and muscle, chronic fatigue syndrome, muscle cramps, allergies (food), asthma, bronchitis, frequent colds, candida, hypoglycemia, allergic reaction to any chemical, chronic fatiguing, food cravings, indigestion, inflamed joints, insomnia, mood swings, gas, bloating, diverticulitis, irritable bowel, pneumonia, ulcers, stomach and bowel cramps and even memory loss is the culmination of years of abuse of nutrition and years of acids from faulty elimination by forcing the bowels to move. We don’t GET sick and tired we DO sick and tired! The most powerful way to eliminate acids in the blood and tissues is the pH Miracle Living Lifestyle and Dietary Plan.

You are the builder of tomorrow, and you need not pay a fortune-teller, doctor, lawyer, preacher, or banker to tell you what will happen to you tomorrow. Nothing will happen. The inevitable will come. You will inherit the fruits of today’s sowing.I hope you find these thoughts and suggestions helpful when dealing with any symptomatology, whether physical, emotional or spiritual.

In love and healing light,

Dr. Robert O. Young

To join are pH Miracle Living community go to:

http://www.phmiracleliving.com

23 Good Reasons Why You Should Never Drink a Single Cup of Coffee

1. Methyl parathion

This is the most toxic pesticide of all. It is  is highly toxic to humans, birds, fish, and mammals. It’s used to fight leaf miner infestations. Leaf miners are insects that eat at leaves of plants.  Despite how dangerous it is, it’s still (mis)used in some countries.                                                                          

2. Endosulfan

This pesticide is used against coffee cherry borer, a common coffee consuming bug. It’s doesn’t dissolve easily and takes ages to break down in soil and is toxic to most animals. It affects the central nervous system, reproductive organs, kidneys, and liver, and is considered to be worse than the pest itself; it’s even been responsible for human death!

3. Chlorpyrifos

This is also used against common coffee pests and has been banned in the US for household use because it has caused human death and birth defects. Needless to say, it’s quite detrimental to delicate ecosystems.

4. Triadimefon

Copper-based fungicide used to against coffee rust. Only slightly toxic to birds, little is known about its effect on humans, but it is suspected that there is potential for reproductive problems with chronic exposure.  (Like people who drink coffee every day.) It has been found to induce hyperactivity in rats. The major concern is that long-term use of this and other copper-based fungicides is copper accumulation in soils, such as that found in coffee farms in Kenya and in Costa Rica.  Copper toxicity has been found in other crops grown in these soils, and copper impacts other biochemical and biological processes in soil which will poison people eating these crops not to mention the people who drink coffee.                                                                                      

5. Caffeine

One 8-ounce cup of coffee has 95 milligrams of caffeine and a 1-ounce single shot of espresso has 64 milligrams. Consuming too much caffeine can make you restless, anxious, irratable and then dead.  Caffeine is an acid or oxidant poison with a pH of 5.5 and an oxidative reduction pH of over +250 mV. When you drink a cup of coffee the body reacts to the poisons in the coffee and you feel it as adrenalin rush as the body starts releasing alkaline buffers to neutralize the poison or the caffeine acid.  It only takes 3 cups of coffee or acid to go into potential caffeine intoxication or 300mg which can cause a cardiac arrest.  One drop of pue caffeine or 2 grams of caffeine powder will kill you instantly.                      

6. pH

Coffee has a perfect pH for cancerous cells 5.5. Research at the Brigham Young University showed that you can keep cancer cells alive indefinitely in a cup of coffee.

7. ORP

Coffee is NOT an antioxicant but an oxidant which acitivates the alkaline buffering system and depletes your body of alkalizing minerals such as sodium bicaronatea and potassium bicaronate.

8.  Stomach

Because coffee an acid beverage it causes the stomach to produce sodium bicarbonate which increases the hydrochloric acid in the stomach leading to acid reflux, GERD, ulcers and stomach cancer.                    

9.  Damages Cover Cells of the Stomach

Coffee is a hot beverage and any hot beverage will damage the cover cells of the stomach which cells produce sodium bicarbonate for maintaining the alkaline design of all the body fluids.                                                                            

10.  Stimulate

Coffee is saturated with hydrogen ions or protons and thus steals energy from your body making you more tired after the stimulating effects wear-off.  This creates the addiction for more coffee in order to achieve an energy increase or buzz.  Continued use of coffee leads to enervation then irritation, then inflammation, then ulceration and finally degeneration or cancer.                                                                                      

11.  Cafestol

Coffee increases the level of cholesterol. Why? Because coffee contains an acid substance called cafestol which triggers the rise of cholesterol levels. The cafestol blocks a receptor in an intestinal pathway crucial for cholesterol regulation, and is the most potent food chemical to do this.   Increased amounts of this acid will increase cholesterol for the purpose of buffering and maintaining the alkaline design of the body.                                                                                    

12.  Intestinal Villi Damage

Coffee compromises the alkalinity of the small intestine at a pH of 8.4.  This causes the intestinal villi to lie down preventing the biological transformation of food or chyme into stem cells which takes place in the crypts of the intestinal villi.   This leads to lowered blood counts, improper blood cell formation and symptoms anemia, pernicious anemia, and hemolytic anemia.  Coffee also damages the intestinal villi leading to more serious conditions such as proper bone, muscle and organ regeneration.                                                  

13. Cancer

Coffee contains over 1000 chemicals of which only 22 have been studied leaving 978 left to study.  All of the chemicals studied to date have been found to be carcinogentic.  So next time to pick up that cup of coffee think of it as your cup of cancer.

14.  Sugar and Cream

Add the sugar and cream and you just created one toxic and addictive acidic/poisonous beverage.

15. Heart Disease – 

There is controversial scientific evidence linking coffee consumption to heart diseases. Some studies even state that “consumption is associated with significantly increased risk of cardiovascular disease.” These same studies have shown a cholesterol-raising effect in some of the chemical compounds of coffee, such as determines, cafestol, kahweol and plasma homocysteine. This may be of-set by some of the antioxidants, but the overall agreement is that coffee may adversely effect the heart.

16. Blood Vessels


Coffee disturbs the functioning of blood vessels, both in turgidity and tone.

17. Cardiovascular System

Coffee affects our nervous system, heart rhythms and has been consistently linked to irregular heartbeats. It may also adversely affect blood pressure.

18. Osteoporosis 

Coffee drinking should be heavily avoided by people at risk, or who have Osteoporosis. Studies show a link between drinking coffee and urinary calcium excretion. 

19.  Heartburn  

Many people report that coffee increases heartburn.

20.  Sleep Disturbance 

 Coffee, particularly in the evening or at night, can lead to sleep disturbance.

21. Dehydration 

Drinking coffee depletes water reserves in the body.

22. Addiction 

While the FDA recognizes caffeine as “safe,” it is still a drug, as it significantly alters the nervous system, leading to addiction over time. 

23. Extreme Withdrawal Symptoms 


You may experience withdrawal symptoms when you try to give up coffee. This can lead to headaches, irritability, body aches, and other more extreme symptoms.

Coffee beans are green and alkaline on the tree until they are fermented and spoiled rotten to a brown acidic, dis-ease causing color.

 

Have We Lost Our Medical Freedom?

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One of the signers of the DECLARATION OF INDEPENDENCE IN 1776, wrote the following statement regarding medical freedom!

“Unless we put medical freedom in the constitution, the time will come when medicine will organize into an undercover dictatorship…The constitution of this Republic should make special provision for medical freedom as well as religious freedom.”

-Benjamin Rush M.D signer of the Declaration of Independence

Did he have insight?? What about those of us who have insight today? Will our words also be ignored? Has medicine organized into an undercover dictatorship? Is this prophetic or what???? Now millions are fighting for just that, medical freedom to choose safer and effective alternatives to conventional medicine.

Some people have been arrested for refusing to give their children chemotherapy, yet, those who have the means to fight, are not arrested. Why? One was a single mother, arrested. The others, a married couple, NOT ARRESTED. Hmmmm?

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My vision of the relative purpose of medicine is to include prevention of illness and promotion of health and fitness rather than focusing all our attention on the diagnosis and treatment of disease. I believe the ultimate purpose of medicine is to help people discover something fundamental within themselves. And that is the awareness that the true source of well-being, joy, and contentment that we all seek lies within one’s mind and heart – the emotions and the spirit – not in the physical world.

This is important, so we can all begin to be freed from the process of grasping for happiness in this physical world. To support this approach, I believe we must begin to embrace a more spiritual vision of ourselves and of humanity as a whole. While providing great love, care and attention to the physical body, medicine can then and only then can we help people discover the nonphysical, spiritual dimensions of themselves.

When this happens, we can all live and work with less fear, stress, grasping to preserve the physical body at all costs – we can truly be free.

In love and healing light,

Dr. Robert O. Young

100 Scientific Reasons to NOT Eat Eggs, Beef, Chicken, Turkey, Pork, or Fish Flesh!

1.) Neu5Gc is found only in animal meat. Neu5Gc appears to have a strong link to cancer and heart disease.

Notes: Neu5Gc is not produced by the human body or our great ape ancestors (probably due to a common mutation). But Neu5Gc is usually always found in human tumors. The inflammation it causes seems to feed tumors and harden arteries.
SourceDiversity in specificity, abundance, and composition of anti-Neu5Gc antibodies in normal humans: potential implications for disease. Glycobiology. 2008 Oct;18(10):818-30.
2.) Top 15 foods for advanced glycation end products (AGEs) are all meat sourced with roasted BBQ chicken skin and fried bacon being the top.
Notes: AGEs are gerontotoxins (aka aging toxins). AGEs cause proteins to cross together causing stiffness, oxidation stress, and inflammation in muscles, brain tissue, eyes, heart, bone, red blood cells, and kidneys. Thought to contribute to muscle loss as we age.
SourceAdvanced glycation end products in foods and a practical guide to their reduction in the diet. J Am Diet Assoc. 2010 Jun;110(6):911-16.e12. 
SourceDoes accumulation of advanced glycation end products contribute to the aging phenotype? J Gerontol A Biol Sci Med Sci. 2010 Sep;65(9):963-75. Epub 2010 May 17.
3.) Arachidonic acid (naturally found in animal foods) is linked to brain inflammation, depression, anxiety, and stress.
Notes: Arachidonic acid is used by our bodies to create inflammation. Our bodies produce all the arachidonic acid we need unlike other animals (e.g. cats) who produce little to none because their bodies expect to get theirs from their diet (meat). Excess arachidonic acid means excess inflammation.
SourcePreliminary evidence that vegetarian diet improves mood. American Public Health Association annual conference, November 7-11, 2009. Philadelphia, PA.
4.) Chicken and eggs are the top sources of arachidonic acid.
Notes: Humans already produce the natural levels needed for our bodily functions.
SourceNational Cancer Institute. 2010. Sources of Selected Fatty Acids among the US Population, 2005–06.
5.) A single meal of high-fat animal products has been shown to spike inflammation within hours that can stiffen one’s arteries.
Notes: Sausage and egg McMuffins were used as the high fat meal in the study. Possible explanations could be the saturated fat or endotoxemia (see points 7, 8, and 9 below).
SourceEffect of a single high-fat meal on endothelial function in healthy subjects. Am J Cardiol. 1997 Feb 1; 79(3):350-4.
6.) High-fat animal products consumed will cause inflammation within the lungs.
Notes: Same study as above.
SourceEffect of a single high-fat meal on endothelial function in healthy subjects. Am J Cardiol. 1997 Feb 1; 79(3):350-4.
7.) Significant levels of bacterial toxins are found in animal products that cause endotoxemia (bacterial toxins in the bloodstream) within hours of eating.
Notes: 40 different types of common foods were tested.
SourceThe capacity of foodstuffs to induce innate immune activation of human monocytes in vitro is dependent on food content of stimulants of Toll-like receptors 2 and 4. Br J Nutr. 2011 Jan; 105(1):15-23.
8.) Bacteria endotoxins from animal products have been shown to survive high heat cooking for long periods, acid (like our stomachs), and digestive enzymes.
SourceThe capacity of foodstuffs to induce innate immune activation of human monocytes in vitro is dependent on food content of stimulants of Toll-like receptors 2 and 4. Br J Nutr. 2011 Jan; 105(1):15-23.
9.) Endotoxins have a strong affinity for the fat transport system in our digestive tract.
Notes: Since our body is using our fat transport system to let in all the saturated fat (which our body loves to absorb) from the animal products the endotoxins can slip right in.
SourceThe capacity of foodstuffs to induce innate immune activation of human monocytes in vitro is dependent on food content of stimulants of Toll-like receptors 2 and 4. Br J Nutr. 2011 Jan; 105(1):15-23.
10.) Even wild, grass consuming animals cause inflammation in our bodies.
Notes: Inflammation was less than domestic animals. It is believe that the lower fat percentage for wild animals explains this.
SourceDifferences in postprandial inflammatory responses to a ‘modern’ v. traditional meat meal: a preliminary study. Br J Nutr. 2010 Sep;104(5):724-8.
12.) The consumption of chicken has been shown to cause urinary tract infections.
SourceFood reservoir for Escherichia coli causing urinary tract infections. Emerg Infect Dis, 16(1):88-95, 2010.
13.) Feeding of cow brains to fish is still legal in the United States (brain cows being fed to cows and other livestock used to be legal and practiced a few years ago) and german scientists have shown fish can acquire mad cow disease.
Notes: Please provide any information if the feeding of cow brains to fish is no longer practiced or legal in the United States.
SourceEvaluation of the possible transmission of BSE and scrapie to gilthead sea bream (Sparus aurata). PLoS One, 4(7):e6175, 2009. 
Source:Bovine spongiform encephalopathy and aquaculture. J Alzheimers Dis, 17(2):277-279, 2009. 
Source:Food and Drug Administration, HHS § 589.2001
14.) Cholesterol has been shown to feed and promote the growth of cancer.
SourceCholesterol and breast cancer development. Current Opinion in Pharmacology. 2012 12 (6):677–682.
15.) Half an egg a day or more is shown to double the odds of mouth, throat, esophageal, prostate, and bladder cancer; triple the odds of colon and breast cancer.
Notes: May be explained by the dixons present. While banned, levels are still present in our food and seem to be worst in animal products.
SourceEgg consumption and the risk of cancer: a multisite case-control study in Uruguay.
16.) The obesogen (chemicals that signal cells to turn into fat cells) organotin has been found in large amounts in fish.
SourceEnvironmental obesogens: Organotins and endocrine disruption via nuclear receptor signaling. Endocrinology, 147(6 – Suppl):-50, 2006.
SourceDietary intake of organotin compounds in Finland: a market-basket study.
17.) Meat has little to no antioxidants. Most can’t beat iceberg lettuce.
Notes: One animal source of food did have an extremely high amount of antioxidants: human breast milk.
SourceThe total antioxidant content of more than 3100 foods, beverages, spices, herbs and supplements used worldwide. Nutr J. 2010 Jan 22;9:3.
19.) Harvard studies of 37,698 men and 83,644 women, over 22 and 30 years, respectively, found red meat to increase total mortality rates and cancer mortality rates.
Notes: Results were after controlling for age, weight, alcohol, exercise, smoking, family history, calorie intake, and intake of whole plant foods. Nuts were found to be protective when taken as an alternative protein source.
SourceRed Meat Consumption and Mortality: Results From 2 Prospective Cohort Studies. Arch Intern Med. 2012;0(2012):201122871-9.
20.) Nitrites in processed meat form nitrosamines (carcinogens also found in cigarette smoke) and are associated with the two leading pediatric cancers, brain tumors and childhood leukemia.
Notes: Hot dogs have some of the highest levels. Pregnant women should probably avoid hot dogs.
SourceA meta-analysis of maternal cured meat consumption during pregnancy and the risk of childhood brain tumors. Neuroepidemiology. 2004 Jan-Apr;23(1-2):78-84. 
Source:Nitrites, nitrosamines, and cancer. Lancet. 1968 May 18;1(7551):1071-2.
21.) 47% of U.S. retail meat tested is contaminated with staph (Staphylococcus) bacteria. Multidrug resistant strains were common.
Notes: Turkey was the most common with 77% and chicken and pork with 41% and 42%, respectively. A superbug version (methicillin resistant) was also found of MRSA that can jump from pig to human.
SourceMultidrug-Resistant Staphylococcus aureus in US Meat and Poultry. Clin Infect Dis. 2011 May;52(10):1227-30. 
SourceInfectious disease. From pigs to people: the emergence of a new superbug.
22.) Eating meat just a few times a month greatly increases the chances of abdominal aortic aneurysm.
Notes: Once an abdominal aortic aneurysm begins to tear you have less than a 15% survival rate.
SourceAnalysis of risk factors for abdominal aortic aneurysm in a cohort of more than 3 million individuals. J Vasc Surg. 2010 Sep;52(3):539-48.
23.) Our liver can only detox about 50% of the heterocyclic amines (carcinogens) formed from cooked chicken. Not the originally thought 99% which other animals can.
Notes: The animal that can detox 99% is the lab rat. Thus, the prior incorrect conclusion.
SourceBiomonitoring of urinary metabolites of 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (phip) following human consumption of cooked chicken. Food Chem. Toxicol., 46(9):3200-3205, 2008.
24.) One of the longest running studies showed meat consumption to increase allergies. This included asthma, bee stings, drug allergies, and hayfever.
Notes: Meat (including fish) consumed by pregnant woman can cause their children to have allergies.
SourceKnutsen SF. Lifestyle and the use of health services. Am J Clin Nutr. 1994 May;59(5 Suppl):1171S-1175S. 
SourceMaternal meat and fat consumption during pregnancy and suspected atopic eczema in Japanese infants aged 3-4 months: the Osaka Maternal and Child Health Study. Pediatr Allergy Immunol. 2010 Feb;21(1 Pt 1):38-46. Epub 2009 Jun 23.
26.) 100% of human Yersinia enterocolitica outbreak over the last decade was caused by pork.
NotesY. enterocolitica usually causes bloody diarrhea and can have very harmful long-term effects if left untreated. Half of American pig herds were found to be infected.
SourceRanking the disease burden of 14 pathogens in food sources in the United States using attribution data from outbreak investigations and expert elicitation. J. Food Prot. 75, 1278 – 1291 (2012). 
Source:Prevalence of pathogenic Yersinia enterocolitica strains in pigs in the United States. Appl. Environ. Microbiol. 71, 7117 – 7121 (2005).
27.) Processed meat is greatly associated with stomach, colon, rectum, pancreatic, lung, prostate, testicular, kidney, and bladder cancer.
SourceCanadian Cancer Registries Epidemiology Research Group. Salt, processed meat and the risk of cancer. Eur J Cancer Prev. 2011 Mar;20(2):132-9.
29.) After breast cancer diagnosis, increase in saturated fat consumption increased mortality from breast cancer by 41%.
Notes: Top 5 saturated fat sources include cheese, pizza, pastries, ice cream, and chicken (notice beef is not in the top 5).
SourcePost-diagnosis dietary factors and survival after invasive breast cancer. Breast Cancer Res Treat. 2011 Jul;128(1):229-36. 
SourceNational Cancer Institute. 2010. Top Food Sources of Saturated Fat among US Population.
31.) Chicken handling significantly increased risk of dying from penile (penis) cancer, thought to be due to exposure to cancer causing viruses in poultry.
SourceCancer mortality in poultry slaughtering/processing plant workers belonging to a union pension fund. Environ Res. 2010 Aug;110(6):588-94.
32.) 14% of retail eggs contain viruses of the leukosis/sarcoma group.
Notes: These viruses are one of the most potent cancer causing viruses in chicken. Virus exposure to humans seems to increased the risk of dying from several different cancers.
SourceDetection of exogenous and endogenous avian leukosis virus in commercial chicken eggs using reverse transcription and polymerase chain reaction assay. Avian Pathology (1999) 28, 385±392 
SourceCancer mortality in poultry slaughtering/processing plant workers belonging to a union pension fund. Environ Res. 2010 Aug;110(6):588-94.
33.) Besides cancer, poultry workers suffer more from a range of diseases (e.g. thyroid conditions, schizophrenia, autoimmune neurological disorders, peritonitis, and disease of the kidneys).
SourceMortality in the Baltimore union poultry cohort: non-malignant diseases. Int Arch Occup Environ Health. 2010 Jun;83(5):543-52.
34.) Cured meat seems to increase the chance of getting chronic obstructive pulmonary disease (COPD).
Notes: COPD is generally defined as lung diseases (e.g. emphysema). As of 2012, COPD is the third most common killer in the United States.
SourceConsumption of cured meats and prospective risk of chronic obstructive pulmonary disease in women. Am J Clin Nutr. 2008 Apr;87(4):1002-8.
35.) Increased meat consumption increases the risk of developing cataracts.
SourceDiet, vegetarianism, and cataract risk. Am J Clin Nutr. 2011 May;93(5):1128-35.
36.) Bacteria-eating viruses (bacteriophages) have been approved as meat additives.
SourceBacteria-eating virus approved as food additive. FDA Consum. 2007 Jan-Feb; 41(1):20-2.
37.) Meat contaminated with fecal food-poisoning bacteria (e.g. salmonella) can legally be sold.
SourcePublic knowledge and beliefs about diarrheal disease. Foodborne Pathog Dis. 2011 Jan; 8(1):165-7.
38.) Meat, fish, cheese, and general animal protein intake have been associated with an increased risk of inflammatory bowel disease (IBD).
SourceAnimal protein intake and risk of inflammatory bowel disease: The E3N prospective study. Am J Gastroenterol. 2010 Oct; 105(10):2195-201.
39.) One of the largest studies ever links meat consumption with increased overall death, death by cancer, and death by cardiovascular disease.
Notes: Study followed 500,000 people over 10 years.
SourceMeat intake and mortality: a prospective study of over half a million people. Arch Intern Med. 2009 March 23; 169(6): 562–571.
40.) High intake of meat, dairy, and butter have been shown to promote skin wrinkling.
Notes: For those curious, prunes, apples, and tea (especially green) appeared to be the most protective and reduced wrinkling and scaling the most.
SourceSkin wrinkling: can food make a difference? J Am Coll Nutr. 2001 Feb;20(1):71-80.
41.) Abdominal fat has been linked to meat, egg, and milk consumption. Poultry seems to be the worst offender.
SourceWill all Americans become overweight or obese? Estimating the progression and cost of the US obesity epidemic. Obesity (Silver Spring), 16(10):2323-2330, 2008.
42.) Heterocyclic Amines (normally only found in cooked meat) have also been found in cheese and eggs.
SourceFormation and biochemistry of carcinogenic heterocyclic aromatic amines in cooked meats. Toxicol Lett. 2007 Feb 5;168(3):219-27. Epub 2006 Nov 16.
43.) Kidney failure is linked to meat.
Notes: Meat consumption was shown to cause human proteins to be urinated out (microalbuminuria). Something that should never happen.
SourceAssociations of diet with albuminuria and kidney function decline. Clin J Am Soc Nephrol. 2010 May; 5(5):836-43.
45.) Iron found in meat passes through the digestive system without regulation.
Notes: Iron is a pro-oxidant, which can cause oxidative stress and DNA damage. Too much iron can cause colon cancer, cardiovascular disease, infection, and inflammatory conditions. Body has no means to get rid of excess iron through a regulatory system.
SourceMechanisms of heme iron absorption: current questions and controversies. World J Gastroenterol. 2008 Jul 14; 14(26):4101-10.
46.) Animal foods (including turkey) shown to decrease tryptophan in the brain.
Notes: Tryptophan rich animal foods like turkey will increase the tryptophan levels in the blood, but this in turns decreases it in the brain. Plant based foods high in tryptophan, when compared to other amino acids, and carbohydrates (like seeds) work best to elevate mood and tryptophan in the brain.
SourceProtein-source tryptophan as an efficacious treatment for social anxiety disorder: a pilot study. Can J Physiol Pharmacol. 2007 Sep;85(9):928-32.
47.) Propionate. Meat has no fiber to support healthy bacteria in our guts. That means our bacteria cannot produce propionate which is used to regulate cholesterol and either help us feel satisfied or possibly regulate generation of new fat cells.
SourcePropionate. Anti-obesity and satiety enhancing factor? Appetite. 2011 Apr;56(2):511-5.
48.) Refined grains, eggs, and poultry shown to cause prostate enlargement the most.
SourceFood groups and risk of benign prostatic hyperplasia. Urology. 2006 Jan;67(1):73-9.
49.) Even when meat consumption is reduced to only fish and eggs, insulin-like growth factor (IGF-1) remained relatively the same.
Notes: IGF-1 has been shown to promote cancer growth.
SourceThe associations of diet with serum insulin-like growth factor I and its main binding proteins in 292 women meat-eaters, vegetarians, and vegans. Cancer Epidemiol Biomarkers Prev. 2002 Nov;11(11):1441-8.
50.) Arsenic, lead, mercury, lead, cadmium, polycyclic aromatic hydrocarbons (PAH), and veterinary drugs have been found contaminating animal products.
SourceChemical safety of meat and meat products. Meat Sci. 2010 Sep;86(1):38-48.
51.) Fire retardant chemicals (PBDE) and polychlorinated naphthalenes (PCNs) found heavily in meats.
Notes: For PBDEs, fish was the worst offender, followed by turkey, and the third worst being chicken. PCNs have a dixion-like effect on the body. The animal with the highest levels was fish. Second was chicken.
SourcePolybrominated diphenyl ether (PBDE) levels in an expanded market basket survey of U.S. food and estimated PBDE dietary intake by age and sex. Environ Health Perspect. 2006 Oct;114(10):1515-20. 
Source:Polybrominated diphenyl ethers in U.S. Meat and poultry from two statistically designed surveys showing trends and levels from 2002 to 2008. Agric Food Chem. 2011 May 25;59(10):5428-34.
52.) Consumption of meat, fish, and dairy products associated with mothers passing on DDT (dichlorodiphenyltrichloroethane) to their unborn child even decades after the pesticide being banned.
SourceOrganochlorine pesticides in umbilical cord blood serum of women from Southern Spain and adherence to the Mediterranean diet. Food Chem Toxicol. 2010 May;48(5):1311-5.
54.) 30 year follow up of multiple sclerosis patients showed 95% of those who stopped consuming saturated fat had no progression of the disease.
Notes: A 50 year follow up showed those 95% who started to consume animal fat again instantly had the disease return. The conclusion of the study said MS is mostly likely caused from saturated animal fat.
SourceEffect of low saturated fat diet in early and late cases of multiple sclerosis. Lancet 1990 336(8706):37 – 39. 
SourceReview of MS patient survival on a Swank low saturated fat diet. Nutrition 2003 19(2):161 – 162.
56.) Almost 80% of all antimicrobials (antibiotics) produced are used on and feed to livestock.
Source2009 Summary Report on Antimicrobials Sold or Distributed for Use in Food-Producing Animals. U.S. Food and Drug Administration. 2010.
57.) Elderly people given milk as children have triple the risk of colorectal cancer.
SourceChildhood dairy intake and adult cancer risk: 65-y follow-up of the boyd orr cohort. American Journal of Clinical Nutrition, 86(6):1722, 2007.
58.) All types of meat (no matter how it is cooked) increases cancer of the uterus.
Notes: Poultry and fish increased the risk the most.
SourceAnimal food intake and cooking methods in relation to endometrial cancer risk in shanghai. Br. J. Cancer, 95(11):15861592, 2006.
59.) Only purines (meats) and fructose increases uric acid levels in our bodies.
Notes: Uric acid increases gout, high blood pressure, obesity, diabetes, kidney disease, and cardiovascular disease.
SourceThe role of uric acid as an endogenous danger signal in immunity and inflammation. Curr Rheumatol Rep. 2011 Apr;13(2):160-6.
60.) PhIP (a type of heterocyclic amines carcinogen in cooked meats) not only damages DNA, but also activates estrogen receptors (almost as strong as the hormone estrogen itself) on breast cancer cells and promotes its growth.
Notes: PhIP has been found in mother’s breast milk. Meaning PhIP from cooked meat does make its way to the breast tissues.
SourceThe cooked food derived carcinogen 2-amino-1-methyl-6-phenylimidazo[4,5-b] pyridine is a potent oestrogen: A mechanistic basis for its tissue-specific carcinogenicity. Carcinogenesis 2004 25(12):2509 – 2517
61.) PhIP stimulates breast cancer cells to invade healthy cells more so than the hormone estrogen itself. Even when PhIP is at low concentrations.
Notes: PhIP is most common in chicken, beef, and fish.
SourceThe cooked meat-derived mammary carcinogen 2-amino-1-methyl-6-phenylimidazo[4,5-b]pyridine promotes invasive behaviour of breast cancer cells. Toxicology 2011 279(1 – 3):139 – 145
62.) Meat fumes from cooked meat may be hazardous for fetal development and increase the risk of cancer.
Notes: Simply being around the vapors (polycyclic aromatic hydrocarbons or PAHs for short) was associated with a birth weight decrease and head shrinkage.
SourceImpact of barbecued meat consumed in pregnancy on birth outcomes accounting for personal prenatal exposure to airborne polycyclic aromatic hydrocarbons: Birth cohort study in Poland. Nutrition. 2012 Apr;28(4):372-7.
63.) A diet high in protein, particularly animal protein, has been associated with relapse of inflammatory bowel disease and a higher risk of inflammatory bowel disease.
SourceDiet and risk of inflammatory bowel disease. Dig Liver Dis 2012 44(3):185 – 194
64.) Microparticles (titanium dioxide and aluminum silicate), common additive in pastries and processed food, consumed with endotoxins cause an inflammatory response in the gut wall 6x greater compared to endotoxins alone.
Notes: Most people are digesting a trillion particles of titanium dioxide a day. Researchers found these microparticles in all 18 diseased colons (colon cancer or inflammatory bowel) used for a study. No microparticles were found in the healthy colons studied. Titanium dioxide is used to make things white. Thus white powdered donuts tend to have the most titanium dioxide out of all foods.
SourceImmune potentiation of ultrafine dietary particles in normal subjects and patients with inflammatory bowel disease. J. Autoimmun. 2000 14(1):99 – 105
65.) Meat consumption linked to premature puberty in boys and girls.
Notes: Premature purberty has been linked to increased risk of developing breast cancer in girls and abdominal fat and heart disease in boys.
SourceInternal exposure to pollutants and sexual maturation in Flemish adolescents. J Expo Sci Environ Epidemiol 2011 21(3):224 – 233
67.) Animal consumption linked to breast pain.
Notes: Prolactin has been shown to cause breast pain. When vegans and vegetarians in South Africa (who have lower levels of prolactin and breast pain compared to women in western civilization) were fed meat, their prolactin levels went up to match western women. Two separate studies showed significant reduction in cyclical breast pain when meat was removed from the diet.
SourceDiet, lifestyle, and menstrual activity. Am J Clin Nutr. 1980 Jun;33(6):1192-8. 
SourceDiet and prolactin release. Lancet. 1976 Oct 9;2(7989):806-7. 
SourceSerum prolactin and oestradiol levels in women with cyclical mastalgia. Horm Metab Res. 1981 Dec;13(12):700-2.
68.) Amino acid L-carnitine (found heavily in red meat and popular energy drinks) has been found to cause heart disease.
Notes: While our bodies produce L-carnitine, the problem comes when our gut bacteria comes in contact with it and produces a toxic substance called trimethylamine oxide (TMAO). This substance has been found circulating in our blood after L-carnitine consumption. TMAO also appears to be linked to cancer.
SourceIntestinal microbiota metabolism of l-carnitine, a nutrient in red meat, promotes atherosclerosis. Nat Med. 2013 Apr 7.
69.) Chickens consumption and handling linked to bladder infections.
Notes: When handling frozen chicken the UTI bacteria causing strains end up in that person’s rectum. Even when the chicken is well cooked before consumption. This is because the jump happens before it is cooked. The strains are usually antibacterial resistant.
SourceChicken as reservoir for extraintestinal pathogenic Escherichia coli in humans, Canada. Emerging Infect. Dis. 2012 18(3):415 – 421
70.) Thorough cleaning with bleach right after every use has been found to be the only method to significantly reduce chicken pathogens in kitchens. However, pathogens were still detectable on some kitchen items.
Notes: All items used were washed in beach and surfaces were sprayed and wiped. Washcloth was soaked in bleach. Bleach was allowed to sit on surfaces for 5 minutes before test results were taken. Pathogens were still found on utensils, counters, and washcloth.
SourceThe effectiveness of hygiene procedures for prevention of cross-contamination from chicken carcases in the domestic kitchen. Lett. Appl. Microbiol. 1999 29(5):354 – 358
71.) A 5% increase of calories from saturated fat (at the expense of calories from carbohydrates) can result in a 38% lower sperm count.
SourceDietary fat and semen quality among men attending a fertility clinic. Hum. Reprod. 2012 27(5):1466 – 1474
72.) Xenoestrogens (human made chemicals with estrogenic effects) have been found the most in fish.
SourceRole of environmental estrogens in the deterioration of male factor fertility. Fertil Steril. 2002 Dec;78(6):1187-94.
73.) Fish eaters have been shown to have only a fraction of the sperm count of vegans.
SourceRole of environmental estrogens in the deterioration of male factor fertility. Fertil Steril. 2002 Dec;78(6):1187-94.
74.) The American Heart Association took legal action through the FDA (which was upheld by the Supreme Court) to have the egg industry cease and desist promoting eggs as having no harmful effects on your health.
Notes: Not a scientific fact, but interesting nonetheless. The notable statement by the egg industry was an advertising campaign that stated there is no scientific evidence that eggs cause heart disease. After the courts reviewed the evidence, they found the statement to be clearly false and misleading.
SourceDietary cholesterol, serum cholesterol, and risks of cardiovascular and noncardiovascular diseases. Am. J. Clin. Nutr. 1998 67(3):488 – 492
75.) One egg contains almost 2/3 of the cholesterol limit suggested by the American Heart Association for healthy people.
SourceAHA Website
76.) Meat handlers have a much high mortality rate of cancer.
Notes: Most of the highest rates were found with the workers handling the final product.
SourceCancer mortality in workers employed in cattle, pigs, and sheep slaughtering and processing plants. Environ Int 2011 37(5):950 – 959.
77.) Growing up on an animal farm increases the chance of blood cancers (poultry farms were the worst).
Notes: Growing up on farms only growing crops showed no increased chance of blood cancers.
SourceFarming, growing up on a farm, and haematological cancer mortality. Occup Environ Med 2012 69(2):126 – 132.
78.) Eating meat may cause cellular cannibalism (auto-immune polyradiculoneuropathy) and thus explaining autoimmune attack (rheumatoid arthritis).
Notes: By consuming organisms in our own kingdom, our immune system may be identifying our own flesh as foreign objects. Auto-immune polyradiculoneuropathy has never been found to be caused by plant consumption.
SourceMeat-induced joint attacks, or meat attacks the joint: rheumatism versus allergy. Nutr Clin Pract. 2010 Feb;25(1):90-1. 
SourceAuto-immune polyradiculoneuropathy and a novel IgG biomarker in workers exposed to aerosolized porcine brain. J. Peripher. Nerv. Syst. 2011 16 (Suppl 1):34 – 37
79.) Methionine (an amino acid) is required for many cancers and tumors to stay alive and grow. Methionine is found virtually only in animal products (with eggs, fish, and chicken being the worst).
SourceThe effect of replacement of methionine by homocystine on survival of malignant and normal adult mammalian cells in culture. Proc. Natl. Acad. Sci. USA 1974 71(4):1133 – 1136.
80.) Meat is acidic which causes higher risk of kidney stones and lower urine acid clearance.
SourceDiet-induced metabolic acidosis. Clin Nutr 2011 30(4):416 – 421.
81.) Choline, a substance very high in eggs, can cause trimethylamine (the smell of rotten fish) to your breath, urine, sweat, and vagina.
SourceSmelling like dead fish: A case of trimethylaminuria in an adolescent. Clin Pediatr (Phila) 2006 45(9):864 – 866.
82.) Choline is converted to TMAO which, as already stated, is linked to heart disease and cancer.
SourceIntestinal microbiota metabolism of l-carnitine, a nutrient in red meat, promotes atherosclerosis. Nat Med. 2013 Apr 7.
83.) Choline is associated with prostate cancer progression and death.
SourceCholine intake and risk of lethal prostate cancer: Incidence and survival. Am. J. Clin. Nutr. 2012 96(4):855 – 863.
84.) Pork tapeworm brain infection is the most common parasitic disease in the brain for people and is on the rise for humans in the United States.
Notes: Referred to as neurocysticercosis in the study.
Source:Clinical manifestations, diagnosis, and treatment of neurocysticercosis. Curr Neurol Neurosci Rep 2011 11(6):529 – 535.
85.) Meat eaters have a lower resting metabolism compared to vegans and vegetarians.
SourceSympathetic nervous system activity and resting metabolic rate in vegetarians. Metab. Clin. Exp. 1994 43(5):621 – 625.
86.) Phosphorus preservatives are being injected into meat. These phosphorus preservatives may damage blood vessels, accelerate the aging process, and contribute to osteoporosis.
Notes: Higher phosphate levels are associated with significantly lower life span.
SourcePhosphate additives in food–a health risk. Dtsch Arztebl Int. 2012 109(4):49 – 55. 
SourceThe prevalence of phosphorus-containing food additives in top-selling foods in grocery stores. J Ren Nutr. 2013 23(4):265-270.
87.) Phosphorus preservatives injected into poultry dramatically increase the growth of food poisoning Campylobacter bacteria.
NotesCampylobacter is recognized as the main cause of bacterial foodborne disease in many developed countries.
Source:Effects of polyphosphate additives on Campylobacter survival in processed chicken exudates. Appl. Environ. Microbiol. 2010 76(8):2419 – 2424
88.) Chicken nuggets from 2 national food chains found actual chicken meat was not the predominant ingredient as fat was found in greater quantities along with epithelium, bone, nerve (brain and spine), and connective tissue.
SourceThe autopsy of chicken nuggets reads chicken little. Am J Med. 2013 126(11):1018-1019.
90.) Mercury and PCB exposure, due to fish consumption, shown to harm fetus brain development.
Source:Functional MRI approach to developmental methylmercury and polychlorinated biphenyl neurotoxicity. Neurotoxicology 2011 32(6):975 – 980.
91.) Mercury in fish shown to outweigh benefits of omega-3s when it comes to brain development (specifically IQ).
SourceFish consumption during child bearing age: a quantitative risk-benefit analysis on neurodevelopment. Food Chem Toxicol. 2013 54:30-34.
92.) Due to mercury “sticking” to our bodies, most women planning on getting pregnant need to avoid mercury containing foods 1 year before.
Notes: Half-life of mercury is about 2 months. That means after 2 months the mercury amount present is cut in half.
Source:Fish consumption during child bearing age: A quantitative risk-benefit analysis on neurodevelopment. Food Chem. Toxicol. 2013 54(NA):30 – 34.
93.) Pregnant women eating fish once a week give their infants more mercury than if they were injected with six mercury containing vaccines.
SourceSpeciation of methyl- and ethyl-mercury in hair of breastfed infants acutely exposed to thimerosal-containing vaccines. Clin. Chim. Acta. 2011 412(17 – 18):1563 – 1566.
94.) Some chemicals (dioxins, PCB, and DDE) found in fish have half lives as high as 10 years.
Notes: A 10 year half life means after 10 years only half of those chemicals are gone from your body. So it would take a lifetime to get even close to 1 percent of your present levels.
SourceElimination half-lives of polychlorinated biphenyl congeners in children. Environ. Sci. Technol. 2008 42(18):6991 – 6996.
95.) Gestational diabetes (diabetes during pregnancy which causes abnormal fetal growth, birth defects, and infant mortality) risk is increased when meat is consumed before pregnancy.
Notes: Bacon, processed meats, and eggs seem to do the worst damage.
SourceA prospective study of dietary patterns, meat intake and the risk of gestational diabetes mellitus. Diabetologia. 2006 49(11):2604 – 2613. 
Source:Risk of gestational diabetes mellitus in relation to maternal egg and cholesterol intake. Am. J. Epidemiol. 2011 173(6):649 – 658.
96.) Cow’s milk contains estrogen and other hormones (naturally) which promote the conversion of precancerous cell to invasive cancer and enhance the progression of cancer cells.
Notes: Organic milk was used in the study.
SourceMilk stimulates growth of prostate cancer cells in culture. Nutr Cancer. 2011 63(8):1361 – 1366.
97.) Just how smoking is a risk factor for lung cancer, milk consumption is a risk factor for prostate cancer
Notes: This is from a meta-analysis (a summation of all studies to date found on milk consumption and prostate cancer) on case-control studies (looking at what people with and without prostate cancer had consumed in their past) and another meta-analysis on cohort type studies (following people throughout the years, what they consumed, and who got prostate cancer).
SourceMilk consumption is a risk factor for prostate cancer: Meta-analysis of case-control studies. Nutr Cancer. 2004 48(1):22 – 27. 
SourceMilk consumption is a risk factor for prostate cancer in Western countries: Evidence from cohort studies. Asia Pac J Clin Nutr. 2007 16(3):467 – 476.
98.) “Meat glue” enzyme, transglutaminase, has potential food safety and allergy implications.
Notes: The enzyme functions as an auto-antigen and will give problems to those who are gluten intolerant. Bacteria from other parts of the animal (like E. coli O157:H7) can be found along the glue line.
SourceTransglutaminase, gluten and celiac disease: Food for thought. Nat. Med. 1997 3(7):725 – 726. 
Source:Escherichia coli O157: H7 risk assessment for production and cooking of restructured beef steaks. Report of progress (Kansas State University. Agricultural Experiment Station and Cooperative Extension Service); 873 2010.
99.) Study found 70% of purchased chicken breasts for the study contained cancer causing form of arsenic beyond the safety thresholds of the FDA.
SourceRoxarsone, inorganic arsenic, and other arsenic species in chicken: A U.S.-Based market basket sample. Environ Health Perspect. 2013 121(7):818 – 824.
100.) Amino acid leucine has the greatest effect on increasing mTORC1 (believed to accelerate the aging process). Meat products have the most leucine.
Notes: Calorie restriction is known to down-regulate mTORC1. However, protein restriction, especially the amino acid leucine, has been found to be just as effective.
SourceAmino acid sensing and regulation of mTORC1. Semin Cell Dev Biol. 2012 23(6):621 – 625. 
Source:Nutrient control of TORC1, a cell-cycle regulator. Trends Cell Biol. 2009 19(6):260 – 267.For more information on the hazards of eating meat go to: http://badassu.net/100-scientific-reasons-to-not-eat-meat/

Glenn Stone’s pH Miracle Story – Reversing Cancer NOW!

GlennImage_2-2-1

Glenn Stone’s pH Miracle Story
I was born August 22nd 1956 in Brooklyn, New York as a completely healthy baby. According to my mom at 11 months I spoke a few words and was crawling without any challenges. At 15 months old I had a complete vocabulary and was already walking normally. At 18 months I developed (hosted) baby Roseola and ran a high fever of 104 degrees F for three days straight. During that time the doctors told my parents to place me into cold baths and keep cold cloths on my joints and body several times a day.
After the baby Roseola, for the next 2 years western medicine tried to figure out what was wrong with me. They labeled me with the possibility of MS (Multiple Sclerosis), Cerebal Palsey, and other neuromuscular diseases. At approximately 24 months old I was walking up a flight of stairs and my mom noticed that I was limping and asked me if I had any pain. I replied: What is pain? My mom explained to me that it was a burning, aching, pulling, stabbing or sharp feeling and I said it was all of those things.
At the age of 3 my mom took me to the Hospital for Special Surgery in Manhattan. She reported to the Dr. that I had pain, inflammation and swelling in all of my major joints.  They then recommended a specialist in rheumatology. Dr. Roguff ordered a Sed Rate and RH-Factor test of my blood. Both were high off the charts positive. I was then diagnosed me with JRA (Juvenile Rheumatoid Arthritis). The treatment for JRA in 1959 was unfortunately the same as it is now. High doses of anti-inflammatory medication, auto-immune builders, and cortisone.
Also at 3 years of age, the Doctors immediately put me on highly acidic St. Joseph’s chewable aspirin for children: 8 at a time, 5 times a day for a total of 40 pills a day. One year later they increased the dose from 8 to 12 pills at a time for a total of 60 pills a day. They continued this dosage until I was finally able to swallow Bayer aspirins and it was 2 of them every 3-4 hours. This later on all proved to wreak havoc on my entire digestive system. At age 5-8 years old they were giving me gold injections, yes that’s real gold, one CC of it in the hip once a month. Luckily for me the nurse was really cute but the injection really hurt and took almost a half hour to administer. At about 8 ½ years old we were up visiting my grandparents in the Catskill mountains in Mountaindale, New York after finishing a passover dinner I complained to my parents that I had a slight belly ache. The slight belly ache turned out to be an appendix that was about to burst inside of me.
After I complained about the belly ache, my mom proceeded to put a heating pad on my belly and giving me milk of magnesia. Unknowingly, both of these remedies made the situation a lot worse. When the pain got worse for me my mom knew that if I complained about it, something was really seriously wrong, due to my high pain tolerance. She called the local doctor and this countryman came in wearing his pajamas and slippers. He proceeded to do a rectal appendix test with his finger and said that the appendix was extremely swollen and ready to burst. My mom paged the doctors at HSS and asked them if we could drive back to the city from the Catskills, which was about a 2 ½ hour drive at night, safely enough for them to treat me. The doctor in the Catskills told my primary doctors that he felt that I would not make the trip back. My parents agreed to take me to the local hospital where they are really equipped for skiing, hiking and outdoor accidents. My parents were concerned about their ability to deal with an erupting appendix. By the time I got to the hospital the pain had quadrupled and my blood test showing that it was appendicitis. They immediately prepped me for surgery. During the surgery the appendix blew up and I had a drain in the side of my chest for almost 4 weeks. The best thing about this was my Aunt and Uncle drove up that night with Rockem Sockem robots for me to play with at the hospital. Later on western medicine figured out that the gold shots blew up my appendix. That put an end to the gold shots. They then started to put me on cortisone for the inflammation and pain. The cortisone made my body, face, hands and arms look like somebody hooked me up to an airpump and blew me up.
For the next few years western medicine had been increasing my anti-inflammatory “acidic” medication and cortisols. When I was 10 years old my mom and dad took me off all the cortisones and for the next few years I continued to take the other acidic medications. Later that year we moved out of Brooklyn to New Jersey so that I could attend school without having to walk up and down stairs with crutches. All of the public schools either had an elevator or were single floor. At age 13 my body went through puberty and the Juvenile Rheumatoid Arthritis symptoms seemed to dissipate but I was left with all of the deformities.
For most of my high school years I had bleeding ulcers, continual heartburn and was diagnosed with reflux esophagitis and I had difficulties swallowing. As a byproduct I became anemic. The doctors gave me iron pills which caused chronic constipation and rectal bleeding. It took three hiatal hernia repair surgeries to alleviate many of my symptoms. I was in intensive care for most of my seven week stay. The third operation was done by a specialist in Boston who invented the Nissen Wrap. During this time I also had the Gastroenterologist continually dilate my esophagus by inserting mercury filled rubber tubes down my esophagus to keep it open enough for me to swallow food. My doctor also advised that I drink chocolate and vanilla milkshakes with raw egg for energy and to neutralize the acid in my stomach and esophagus. All this did was create more mucus, more congestion and more health challenges. They kept trying to treat the symptoms instead of getting to the root cause.
Throughout my high school and college years, the Orthopedic Doctors put me on pain narcotics such as Tylenol-3 and Percocet to alleviate my functional pain and inflammation due to the deformities in my body. No one told me at this point that all I needed to do was to take different kinds of healthy oils to lubricate my joints.
At 17 years of age I drove a regular car with no special equipment and in 1974 I attended Rutgers University studying pre-medicine. I dropped out of college my third year because I did not want to be associated with the doctors of the future. I then went on to become an entrepreneur and established my own sound, communications and DJ company. In March of 1982 I met my significant other and soul partner Lori Gilmore and by September 10th 1984 I was engaged to be married.
I brought Lori to my yearly checkup to meet my Orthopedic Surgeon from 1962 to 1986 whose Father pioneered the first total hip replacement in the United States. He recommended two total hip replacements because my hips were locking every time I went to the bathroom or bent over, and to be able to easily perform sexually. I had two total hip replacements and was up walking with crutches in two weeks. I was featured in the New York Times magazine May 5th 1985 in an article titled “Redesigning Human Bones”. Both surgeries were performed in record time. Just when I thought I was ready to start my new life with Lori, on the day of discharge a resident Orthopedist working four days straight without sleep dropped me off of an X-Ray table during my final X-Ray and broke the right thigh completely in half. It took five major procedures over the course of one year for me to recover. During one of the procedures they put me into skeletal traction and a full body cast which caused me to eat and drink laying down which re-aggravated my esophagus. In order to take me home for the summer in a body cast, Lori had to learn how to give me high doses of Morphine Sulfate injections seven to nine times a day. In 1986 Lori and I made a dream come true by committing our lives to each other and I was able to stand for the entire ceremony.

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This is when I met Dr. Young and started making major shifts in my life.
In 1998 I went to an Anthony Robbins seminar and little did I know my life would never be the same again. In addition to doing a 10 foot hot coal firewalk, transformation was about to begin for Lori and I. We were taught a paradigm shift about our lifestyle and diet. We went from eating the Standard American (SAD) diet to starting a 10 day challenge where Lori and I moved away from all animal products, dairy, sugars and processed foods. We continued to live on a fruit and vegetable way of living and we never felt better. I was now able to cut my pain medication from 480mg a day to less than 200 mg a day. I also resigned from going to a pain psychologist and neurologist and told them it was time to take charge of my own health. Later on that year in September at another Anthony Robbins Event called Life Mastery, Tony introduced us to Shelley and Dr. Robert O. Young. Tony asked Dr. Young to demonstrate microscopy personally on Lori and I. Dr. Young did my live and dry blood and told me I was loaded with yeast, fungus and mold. My colon was a mess and that my esophagus had a lot of inflammation. From looking at my blood, he suggested that I eliminate the sweet fruits and carbohydrates that were feeding the yeast and go on an alkaline lifestyle and diet. I was feeling so good that I did not listen. Lori on the other hand, embraced the program, started drinking the greens and other supplements that Dr. Young had created and ran the New York City marathon with only two months training from Stu Mittleman. Six months later I had blood in my stool and food got stuck in my esophagus. I was rapidly losing weight and it was apparent that anything I was eating was not being assimilated or eliminated properly. Anthony Robbins found out about my situation through a mutual friend and called us. Using the power of questions Tony convinced me to go into the hospital to use western medicine for diagnostics and to get my essential body fluids back to normal. He also suggested that we contact Dr. Young to offer us support and guidance. In the hospital I was diagnosed with Digestive Failure/Stage 4 Colon Cancer and Stage 3 Esophogeal Cancer. I went from a 34 inch waste down to a 28 inch waste and from 134 lbs down to 65 lbs. I literally looked like a skeleton. Dr. Young suggested that we get Glenn strong enough so he can leave the hospital and start a 100% alkalizing program. The treatment suggested by my gastroenterologist and nutritionist was chemical therapy (chemo), radiation and a colostomy bag. When we declined this treatment and were ready to check ourselves out of the hospital they would only allow us to do so after we agreed to be seen by a hospital psychiatrist and take full legal responsibility for signing ourselves out. Lori had to borrow another patients wheelchair in order to transport me from the hospital bed to the car. Upon leaving, the Gastroenterologist admitted that Dr. Young’s colloidal liquid lightning vitamin and mineral drops as well as alkalizing supplements kept me alive and sustained me during this hospital stay. Once we left I immediately started drinking and eating 100% alkaline liquids: soups, shakes, smoothies and a gallon of water with pH drops per day. I went on what would be a one year liquid feast. Dr. Young told Lori and I that in order to get rid of all the acid that was in my body for so many years that I was going to possibly lose additional weight because fat binds to acid. We had blind faith in Dr. Young and Shelly and in 12 weeks both cancers were gone. I am proud to say in October 2013 I have been cancer free for 16 years, lowered my pain medication from 400mg down to 78mg and living an awesome alkalarian with the love of my life. I am a pH Miracle.
 
Two other times Dr. Young has saved my life
On one of my gasping for air trips to the hospital, Dr. Young and Tony Robbins called to wish me a happy birthday. Tony told me that the definition of insanity is doing the same thing over and over again expecting a different result.  He suggested that an endocrinologist and a kidney doctor take me completely off of the hydrocortisone and fludrocortizone and monitor my condition while I was in the hospital. Within 24 hours my blood pressure went down to 120/70. I eliminated the fludrocortizone and lowered the hydrocortizone from 50mg to 5mg in the morning and 2 ½ at night.
My last microscopy showed my adrenal glands were 90% better.
In 2009, 4 years ago, my sodium levels due to all the metal in my body, went down to 107 and I went in a coma for almost 5 days. Western medicine told us that the blood sodium should be 130-139. Dr. Young suggested that my blood sodium should be 142 or higher. When I got out of the coma, Dr. Young created the 4 salts, the pHlavor Salts and the pHlush supplements to help everyone by providing the body with alkalizing sodium. He also created the COWS program which stands for Cholorophyll, Oxygen, Oil, Water and Salts. This program saved my lfie once again. In the last 16 years since I have known the Youngs, they have helped save and change my life 7 times, all due to western medicine screw ups. In 2007 the doctors put me on higher doses of hydrocortizone and fludocortizone for what I was labeled with having addisons disease and adrenal insufficiency, besides the JRA which arrested itself at age 13. Both uncurable according to western medicine. These doses of medication caused congested heart failure and a very high blood pressure of 180/120.
Dr. Young taught me that all of our organs and cells can regenerate within 120 days. He was right.  There has been a total of 7 times that Dr. Young has saved my life due to mistakes that western medicine has made. I strongly recommend this program and lifestyle to anyone at any age. The biggest life lesson I have learned is to ask lots of questions and don’t assume that just because they wear a white coat that they are god and can determine whether you can live or die. You must be your own self advocate. Our bodies are alkaline by design, acidic by function and will heal itself when it’s properly supported with alkalizing foods and drinks.

In 2008, Lori and I moved from New Jersey to San Diego in order to spread this message around the world, and Lori and I worked at the Ranch for 3 years. We continue to do cleanses for Dr. Young every three months with our friend Brian Claypool teaching other people how to become their own pH Miracle.

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