Martin Luther King, Jr. One of the Greatest Freedom Fighters of our time!
Free at Last, Free at Last! Thank God I am Free at Last!
PS What Does It Mean to be Truly Free?
To attend a pH Miracle Retreat go to: http://www.phmiracleretreat.com
1. Consume mainly alkaline foods – At least 80%!
2. Pay attention to portion sizes. The plate should include two palm sizes of vegetables and one-palm size of protein
3. Stop eating two to three hours before bed
4. You cannot combine protein and complex carbs for a meal
5. Cannot eat fruit alone
6. Little to no carbs for breakfast
7. Eat foods based on seasons
8. Consume 20oz of alkaline water at a pH of 9.5 right when you wake up
9. If you cannot give up coffee then limit caffeine intake to 200mg (two cups of coffee)
10. No soda, carbonation, milk, dairy, fruit juices, sweetened drinks, and alcohol!
Giving it a try! Toronto-based fitness trainer Keltie O’Connor shared with her followers how she attempted the alkaline diet!
Before the pH Alkaline diet Keltie admitted she was bloated!
After 30 days on the pH Alkaline Diet no more digestive problems and bloating!
Victoria Beckham keeps her slender physique in shape by following the pH Alkaline Lifestyle and Diet, recommended by Robert O. Young PhD. This plan means you ingest ONLY alkaline foods and liquids to keep your acidic levels in your blood, interstitial fluids, intracellular fluids at an alkaline pH between 7.35 and 7.45. All of these fluids can be tested with the Full-Body 3-D Bio-Electro Scan and the non-invasive blood testing of the chemistry, including pH of the blood, stomach, intestines and interstitial fluids.
To learn more about these non-invasive medical tests click here: (http://www.universalmedicalimaging.com/index.html
The following chart lists some of the acidic foods on the pH Miracle Alkaline Diet to eliminate completely or eat sparingly:
The following chart lists some of the foods you can eat freely on the pH Miracle alkaline diet:
The supermodel Elle Macpherson stated that following a plant-based pH Miracle alkaline lifestyle and diet and taking supplements have helped her look younger than her 54 years. (wwwijuicenow.com and http://www.phoreveryoung,com)
“When I turned 50 I realized things I did in my 20s weren’t working anymore,” Macpherson said. “I follow a plant-based alkaline diet, focusing on healthy, whole food. I take green powder and protein powder every day, and I drink three liters of water a day.” (www.ijuicenow.com, http://www.phoreveryoung.com)
So where does a 6’7″ man who weighs over 270 pounds get his protein from? Tony Robbins eats broccoli! Over 50 percent of the calories from steamed organic broccoli comes from protein. It is important to note that the body does not build muscle from protein – it builds it from red blood cells. Muscle, bone, and all organs and glands are made from red blood cells NOT protein! Tony is a strong advocate and walking testimony of Dr. Young’s alkaline lifestyle and diet which he teaches from the stage at ALL his events.
Prince Harry and Meghan follow the pH Miracle Alkaline Lifestyle and Diet. In fact it was Meghan who introduced Harry to the alkaline lifestyle.
So what keeps Tom Brady so healthy, fit and strong at the age of 41 and still playing NFL Football? The answer is the pH Miracle alkaline lifestyle and diet!
Beyonce is a big fan of Marcos Borges 22 days of Vegan program which is a vegan meal service. Although not a full vegan, the singer ordered in the service to get in shape for Coachella. “The benefits of a plant-based diet need to be known,” Beyoncé said. “We should spend more time loving ourselves, which means taking better care of ourselves with good nutrition and making healthier food choices.”
The following are just a few of the foods I recommend that you can eat freely when following an Vegan Diet as outlined in The pH Miracle revised and updated book and The pH Miracle for Weight Loss – http://www.phoreveryoung.com
To keep her energy levels at an all-time high, Angelina Jolie snacks on ancient grains such as quinoa, chia seeds, millet, buckwheat and spelt. “She’s into eating products made from ancient grains and raves about their health benefits,” a source told Marie Claire. “She claims they provide her with nutrients she can’t find anywhere else, plus shinier skin.”
The wonderful benefits of ancients grains like quinoa, millet, buckwheat and spelt, they are low in carbohydrate (sugar) and higher in protein. Keep in mind though I only recommend these grains sparingly and no more than 10 grams of protein daily.
Remember, all body cells, including bone and muscle are made from blood NOT protein. And blood is made from chlorophyll (green foods), unsaturated oil, alkaline water and mineral salts or sodium, potassium, magnesium and calcium.
Jessica Biel credits her super svelte physique down to the Paleo diet. Heavily endorsed by Pete Evans, the diet works on the ethos you go back to eating like a caveman and eradicate dairy, grains and legumes from your diet. “Eating Paleo just leans you down and slims you up and takes that little layer of fat and water-weight right off your body,” says Jessica. “I do a lot of cooking at home using fresh fish or lean meat like chicken and vegetables,” she adds.
The Paleo diet will provide short term benefits but long term damage from ALL the acidic foods from dairy, legumes and grains such as wheat. You are better off with both short and long term benefits by sticking with a diet that does not cause eventual gland, organ and tissue damage. That diet would be low carbohydrate, low protein and liberal amounts of healthy unsaturated oil, like hemp seed, flax seed, broccoli seed, carrot seed, cabbage seed, just to name a few.
Kim Kardashian lost 25 kilos in 11 months on the Atkins diet, which is a high acidic protein, low carb diet. “ Anyone who has had kids knows your body changes, and it’s hard to get your body back in shape,” she said. “It takes so much determination, and mental and physical power and energy.”
Unfortunately this diet also has short term benefits with long term damage, especially to the intestinal villi if you are ingesting animal protein which does not digest (unless you juice the animal flesh). Maybe that is why Kim looks bloated in the lower abdominal area. It is important to stay away from this diet unless your protein sources are from green plants such as avocado, broccoli and buckwheat.
Check out the above list of foods to avoid and especially avoid animal sources for safe and healthy weight loss.
For safe and effective weight loss or weight gain read The pH Miracle for Weight Loss by Robert O Young PhD – http://www.phoreveryoung.com or on amazon.com at: https://www.amazon.com/gp/product/0446694703/ref=dbs_a_def_rwt_hsch_vapi_taft_p1_i1
After sequencing his own genome, pioneer genomic researcher Craig Venter remarked at a leadership for the twenty-first century conference, “Human biology is actually far more complicated than we imagine. Everybody talks about the genes that they received from their mother and father, for this trait or the other. But in reality, those genes have very little impact on life outcomes. Our biology is way too complicated for that and deals with hundreds of thousands of independent factors. Genes are absolutely not our fate. They can give us useful information about the increased risk of a disease, but in most cases they will not determine the actual cause of the disease, or the actual incidence of somebody getting it. Most biology will come from the complex interaction of all the proteins and cells working with environmental factors, not driven directly by the genetic code” (http://indiatoday.digitaltoday.in/index.php?
This statement is very important because looking to the human genome for solutions to most chronic illnesses, including the diagnosis, prevention, and treatment of cancer, is overemphasized in today’s world. Observational studies, however, have indicated that as we migrate from one country to another, our chances of being diagnosed with most chronic illnesses are determined not by the country we come from but by the country we migrate to (1–4). In addition, studies with identical twins have suggested that genes are not the source of most chronic illnesses. For instance, the concordance between identical twins for breast cancer was found to be only 20% (5). Instead of our genes, our lifestyle and environment account for 90–95% of our most chronic illnesses.
Cancer continues to be a worldwide killer, despite the enormous amount of research and rapid developments seen during the past decade. According to recent statistics, cancer accounts for about 23% of the total deaths in the USA and is the second most common cause of death after heart disease (6). Death rates for heart disease, however, have been steeply decreasing in both older and younger populations in the USA from 1975 through 2002. In contrast, no appreciable differences in death rates for cancer have been observed in the United States (6).
By 2020, the world population is expected to have increased to 7.5 billion; of this number, approximately 15 million new cancer cases will be diagnosed, and 12 million cancer patients will die (7). These trends of cancer incidence and death rates again remind us of Dr. John Bailer’s May 1985 judgment of the US national cancer program as a “qualified failure,” a judgment made 14 years after President Nixon’s official declaration of the “War on Cancer.” Even after an additional quarter century of extensive research, researchers are still trying to determine whether cancer is preventable and are asking “If it is preventable, why are we losing the war on cancer?” In this review, we attempt to answer this question by analyzing the potential risk factors of cancer and explore our options for modulating these risk factors.
Cancer is caused by both internal factors (such as inherited mutations, hormones, and immune conditions) and environmental/acquired factors (such as tobacco, diet, radiation, and infectious organisms; Fig. 1). The link between diet and cancer is revealed by the large variation in rates of specific cancers in various countries and by the observed changes in the incidence of cancer in migrating. For example, Asians have been shown to have a 25 times lower incidence of prostate cancer and a ten times lower incidence of breast cancer than do residents of Western countries, and the rates for these cancers increase substantially after Asians migrate to the West (http://www.dietandcancerreportorg/?p=ER).
The role of genes and environment in the development of cancer. A The percentage contribution of genetic and environmental factors to cancer. The contribution of genetic factors and environmental factors towards cancer risk is 5–10% and 90–95% respectively. B Family risk ratios for selected cancers. The numbers represent familial risk ratios, defined as the risk to a given type of relative of an affected individual divided by the population prevalence. The data shown here is taken from a study conducted in Utah to determine the frequency of cancer in the first-degree relatives (parents + siblings + offspring). The familial risk ratios were assessed as the ratio of the observed number of cancer cases among the first degree relatives divided by the expected number derived from the control relatives, based on the years of birth (cohort) of the case relatives. In essence, this provides an age-adjusted risk ratio to first-degree relatives of cases compared with the general population.
C Percentage contribution of each environmental factor. The percentages represented here indicate the attributable-fraction of cancer deaths due to the specified environmental risk factor.
The importance of lifestyle factors in the development of cancer was also shown in studies of monozygotic twins (8). Only 5–10% of all cancers are due to an inherited gene defect. Various cancers that have been linked to genetic defects are shown in Fig. 2. Although all cancers are a result of multiple mutations (9, 10), these mutations are due to interaction with the environment (11, 12).
These observations indicate that most cancers are not of hereditary origin and that lifestyle factors, such as dietary habits, smoking, alcohol consumption, and infections, have a profound influence on their development (13). Although the hereditary factors cannot be modified, the lifestyle and environmental factors are potentially modifiable. The lesser hereditary influence of cancer and the modifiable nature of the environmental factors point to the preventability of cancer. The important lifestyle factors that affect the incidence and mortality of cancer include tobacco, alcohol, diet, obesity, infectious agents, environmental pollutants, and radiation.
Smoking was identified in 1964 as the primary cause of lung cancer in the US Surgeon General’s Advisory Commission Report (http://profiles.nlm.nih.gov/NN/Views/AlphaChron/date/10006/05/01/2008), and ever since, efforts have been ongoing to reduce tobacco use. Tobacco use increases the risk of developing at least 14 types of cancer (Fig. 3). In addition, it accounts for about 25–30% of all deaths from cancer and 87% of deaths from lung cancer. Compared with nonsmokers, male smokers are 23 times and female smokers 17 times more likely to develop lung cancer.
(http://www.cancer.org/docroot/STT/content/STT_1x_Cancer_Facts_and_Figures_2008.asp accessed on 05/01/2008).
The carcinogenic effects of active smoking are well documented; the U. S. Environmental Protection Agency, for example, in 1993 classified environmental tobacco smoke (from passive smoking) as a known (Group A) human lung carcinogen.
(http://cfpub2.epa.gov/ncea/cfm/recordisplay.cfm?deid=2835 accessed on 05/01/2008).
Tobacco contains at least 50 carcinogens. For example, one tobacco metabolite, benzopyrenediol epoxide, has a direct etiologic association with lung cancer (14). Among all developed countries considered in total, the prevalence of smoking has been slowly declining; however, in the developing countries where 85% of the world’s population resides, the prevalence of smoking is increasing. According to studies of recent trends in tobacco usage, developing countries will consume 71% of the world’s tobacco by 2010, with 80% increased usage projected for East Asia.
(http://www.fao.org/DOCREP/006/Y4956E/Y4956E00.HTM accessed on 01/11/08)
The use of accelerated tobacco-control programs, with an emphasis in areas where usage is increasing, will be the only way to reduce the rates of tobacco-related cancer mortality.
Percentages represent the cancer mortality attributable to alcohol and smoking in men and women as reported by Irigaray et al. (see 13).
How smoking contributes to cancer is not fully understood. We do know that smoking can alter a large number of cell-signaling pathways. Results from studies in our group have established a link between cigarette smoke and inflammation. Specifically, we showed that tobacco smoke can induce activation of NF-κB, an inflammatory marker (15,16). Thus, anti-inflammatory agents that can suppress NF-κB activation may have potential applications against cigarette smoke.
We also showed that curcumin, derived from the dietary spice turmeric, can block the NF-κB induced by cigarette smoke (15). In addition to curcumin, we discovered that several natural phytochemicals also inhibit the NF-κB induced by various carcinogens (17). Thus, the carcinogenic effects of tobacco appear to be reduced by these dietary agents. A more detailed discussion of dietary agents that can block inflammation and thereby provide chemopreventive effects is presented in the following section.
The first report of the association between alcohol and an increased risk of esophageal cancer was published in 1910 (18). Since then, a number of studies have revealed that chronic alcohol consumption is a risk factor for cancers of the upper aerodigestive tract, including cancers of the oral cavity, pharynx, hypopharynx, larynx, and esophagus (18–21), as well as for cancers of the liver, pancreas, mouth, and breast (Fig. 3). Williams and Horn (22), for example, reported an increased risk of breast cancer due to alcohol. In addition, a collaborative group who studied hormonal factors in breast cancer published their findings from a reanalysis of more than 80% of individual epidemiological studies that had been conducted worldwide on the association between alcohol and breast cancer risk in women. Their analysis showed a 7.1% increase in relative risk of breast cancer for each additional 10 g/day intake of alcohol (23). In another study, Longnecker et al., (24) showed that 4% of all newly diagnosed cases of breast cancer in the USA are due to alcohol use. In addition to it being a risk factor for breast cancer, heavy intake of alcohol (more than 50–70 g/day) is a well-established risk factor for liver (25) and colorectal (26,27) cancers.
There is also evidence of a synergistic effect between heavy alcohol ingestion and hepatitis C virus (HCV) or hepatitis B virus (HBV), which presumably increases the risk of hepatocellular carcinoma (HCC) by more actively promoting cirrhosis. For example, Donato et al. (28) reported that among alcohol drinkers, HCC risk increased linearly with a daily intake of more than 60 g. However, with the concomitant presence of HCV infection, the risk of HCC was two times greater than that observed with alcohol use alone (i.e., a positive synergistic effect). The relationship between alcohol and inflammation has also been well established, especially in terms of alcohol-induced inflammation of the liver.
How alcohol contributes to carcinogenesis is not fully understood but ethanol may play a role. Study findings suggest that ethanol is not a carcinogen but is a cocarcinogen (29). Specifically, when ethanol is metabolized, acetaldehyde and free radicals are generated; free radicals are believed to be predominantly responsible for alcohol-associated carcinogenesis through their binding to DNA and proteins, which destroys folate and results in secondary hyperproliferation. Other mechanisms by which alcohol stimulates carcinogenesis include the induction of cytochrome P-4502E1, which is associated with enhanced production of free radicals and enhanced activation of various procarcinogens present in alcoholic beverages; a change in metabolism and in the distribution of carcinogens, in association with tobacco smoke and diet; alterations in cell-cycle behavior such as cell-cycle duration leading to hyperproliferation; nutritional deficiencies, for example, of methyl, vitamin E, folate, pyridoxal phosphate, zinc, and selenium; and alterations of the immune system. Tissue injury, such as that occurring with cirrhosis of the liver, is a major prerequisite to HCC. In addition, alcohol can activate the NF-κB proinflammatory pathway (30), which can also contribute to tumorigenesis (31). Furthermore, it has been shown that benzopyrene, a cigarette smoke carcinogen, can penetrate the esophagus when combined with ethanol (32). Thus anti-inflammatory agents may be effective for the treatment of alcohol-induced toxicity.
In the upper aerodigestive tract, 25–68% of cancers are attributable to alcohol, and up to 80% of these tumors can be prevented by abstaining from alcohol and smoking (33). Globally, the attributable fraction of cancer deaths due to alcohol drinking is reported to be 3.5% (34). The number of deaths from cancers known to be related to alcohol consumption in the USA could be as low as 6% (as in Utah) or as high as 28% (as in Puerto Rico). These numbers vary from country to country, and in France have approached 20% in males (18).
In 1981, Doll and Peto (21) estimated that approximately 30–35% of cancer deaths in the USA were linked to diet (Fig. 4). The extent to which diet contributes to cancer deaths varies a great deal, according to the type of cancer (35). For example, diet is linked to cancer deaths in as many as 70% of colorectal cancer cases. How diet contributes to cancer is not fully understood. Most carcinogens that are ingested, such as nitrates, nitrosamines, pesticides, and dioxins, come from food or food additives or from cooking.
Heavy consumption of red meat is a risk factor for several cancers, especially for those of the gastrointestinal tract, but also for colorectal (36–38), prostate (39), bladder (40), breast (41), gastric (42), pancreatic, and oral (43) cancers. Although a study by Dosil-Diaz et al., (44) showed that meat consumption reduced the risk of lung cancer, such consumption is commonly regarded as a risk for cancer for the following reasons. The heterocyclic amines produced during the cooking of meat are carcinogens. Charcoal cooking and/or smoke curing of meat produces harmful carbon compounds such as pyrolysates and amino acids, which have a strong cancerous effect. For instance, PhIP (2-amino-1-methyl-6-phenyl-imidazo[4,5-b]pyridine) is the most abundant mutagen by mass in cooked beef and is responsible for ~20% of the total mutagenicity found in fried beef. Daily intake of PhIP among Americans is estimated to be 280–460 ng/day per person (45).
Nitrites and nitrates are used in meat because they bind to myoglobin, inhibiting botulinum exotoxin production; however, they are powerful carcinogens (46). Long-term exposure to food additives such as nitrite preservatives and azo dyes has been associated with the induction of carcinogenesis (47). Furthermore, bisphenol from plastic food containers can migrate into food and may increase the risk of breast (48) and prostate (49) cancers. Ingestion of arsenic may increase the risk of bladder, kidney, liver, and lung cancers (50). Saturated fatty acids, trans fatty acids, and refined sugars and flour present in most foods have also been associated with various cancers. Several food carcinogens have been shown to activate inflammatory pathways.
According to an American Cancer Society study (51), obesity has been associated with increased mortality from cancers of the colon, breast (in postmenopausal women), endometrium, kidneys (renal cell), esophagus (adenocarcinoma), gastric cardia, pancreas, prostate, gallbladder, and liver (Fig. 5). Findings from this study suggest that of all deaths from cancer in the United States, 14% in men and 20% in women are attributable to excess weight or obesity. Increased modernization and a Westernized diet and lifestyle have been associated with an increased prevalence of overweight people in many developing countries (52).
Various cancers that have been linked to obesity. In the USA overweight and obesity could account for 14% of all deaths from cancer in men and 20% of those in women (see 51).
Studies have shown that the common denominators between obesity and cancer include neurochemicals; hormones such as insulinlike growth factor 1 (IGF-1), insulin, leptin; sex steroids; adiposity; insulin resistance; and inflammation (53).
Involvement of signaling pathways such as the IGF/insulin/Akt signaling pathway, the leptin/JAK/STAT pathway, and other inflammatory cascades have also been linked with both obesity and cancer (53). For instance, hyperglycemia, has been shown to activate NF-κB (54), which could link obesity with cancer. Also known to activate NF-κB are several cytokines produced by adipocytes, such as leptin, tumor necrosis factor (TNF), and interleukin-1 (IL-1) (55). Energy balance and carcinogenesis has been closely linked (53). However, whether inhibitors of these signaling cascades can reduce obesity-related cancer risk remains unanswered. Because of the involvement of multiple signaling pathways, a potential multi-targeting agent will likely be needed to reduce obesity-related cancer risk.
Worldwide, an estimated 17.8% of neoplasms are associated with infections; this percentage ranges from less than 10% in high-income countries to 25% in African countries (56, 57). Viruses account for most infection-caused cancers (Fig. 6). Human papillomavirus, Epstein Barr virus, Kaposi’s sarcoma-associated herpes virus, human T-lymphotropic virus 1, HIV, HBV, and HCV are associated with risks for cervical cancer, anogenital cancer, skin cancer, nasopharyngeal cancer, Burkitt’s lymphoma, Hodgkin’s lymphoma, Kaposi’s sarcoma, adult T-cell leukemia, B-cell lymphoma, and liver cancer.
Various cancers that have been linked to infection. The estimated total of infection attributable cancer in the year 2002 is 17.8% of the global cancer burden. The infectious agents associated with each type of cancer is shown in the bracket. HPV Human papilloma virus, HTLV human T-cell leukemia virus, HIV human immunodeficiency virus, EBV Epstein–Barr virus (see 57).
In Western developed countries, human papillomavirus and HBV are the most frequently encountered oncogenic DNA viruses. Human papillomavirus is directly mutagenic by inducing the viral genes E6 and E7 (58), whereas HBV is believed to be indirectly mutagenic by generating reactive oxygen species through chronic inflammation (59–61). Human T-lymphotropic virus is directly mutagenic, whereas HCV (like HBV) is believed to produce oxidative stress in infected cells and thus to act indirectly through chronic inflammation (62, 63). However, other microorganisms, including selected parasites such as Opisthorchis viverrini or Schistosoma haematobium and bacteria such as Helicobacter pylori, may also be involved, acting as cofactors and/or carcinogens (64).
The mechanisms by which infectious agents promote cancer are becoming increasingly evident. Infection-related inflammation is the major risk factor for cancer, and almost all viruses linked to cancer have been shown to activate the inflammatory marker, NF-κB (65). Similarly, components of Helicobacter pylorihave been shown to activate NF-κB (66). Thus, agents that can block chronic inflammation should be effective in treating these conditions.
Environmental pollution has been linked to various cancers (Fig. 7). It includes outdoor air pollution by carbon particles associated with polycyclic aromatic hydrocarbons (PAHs); indoor air pollution by environmental tobacco smoke, formaldehyde, and volatile organic compounds such as benzene and 1,3-butadiene (which may particularly affect children); food pollution by food additives and by carcinogenic contaminants such as nitrates, pesticides, dioxins, and other organochlorines; carcinogenic metals and metalloids; pharmaceutical medicines; and cosmetics (64).
Various cancers that have been linked to environmental carcinogens. The carcinogens linked to each cancer is shown inside bracket. (see 64).
Numerous outdoor air pollutants such as PAHs increase the risk of cancers, especially lung cancer. PAHs can adhere to fine carbon particles in the atmosphere and thus penetrate our bodies primarily through breathing. Long-term exposure to PAH-containing air in polluted cities was found to increase the risk of lung cancer deaths. Aside from PAHs and other fine carbon particles, another environmental pollutant, nitric oxide, was found to increase the risk of lung cancer in a European population of nonsmokers. Other studies have shown that nitric oxide can induce lung cancer and promote metastasis. The increased risk of childhood leukemia associated with exposure to motor vehicle exhaust was also reported (64).
Indoor air pollutants such as volatile organic compounds and pesticides increase the risk of childhood leukemia and lymphoma, and children as well as adults exposed to pesticides have increased risk of brain tumors, Wilm’s tumors, Ewing’s sarcoma, and germ cell tumors. In utero exposure to environmental organic pollutants was found to increase the risk for testicular cancer. In addition, dioxan, an environmental pollutant from incinerators, was found to increase the risk of sarcoma and lymphoma.
Long-term exposure to chlorinated drinking water has been associated with increased risk of cancer. Nitrates, in drinking water, can transform to mutagenic N-nitroso compounds, which increase the risk of lymphoma, leukemia, colorectal cancer, and bladder cancer (64).
Up to 10% of total cancer cases may be induced by radiation (64), both ionizing and non-ionizing, typically from radioactive substances and ultraviolet (UV), pulsed electromagnetic fields. Cancers induced by radiation include some types of leukemia, lymphoma, thyroid cancers, skin cancers, sarcomas, lung and breast carcinomas. One of the best examples of increased risk of cancer after exposure to radiation is the increased incidence of total malignancies observed in Sweden after exposure to radioactive fallout from the Chernobyl nuclear power plant. Radon and radon decay products in the home and/or at workplaces (such as mines) are the most common sources of exposure to ionizing radiation. The presence of radioactive nuclei from radon, radium, and uranium was found to increase the risk of gastric cancer in rats. Another source of radiation exposure is x-rays used in medical settings for diagnostic or therapeutic purposes. In fact, the risk of breast cancer from x-rays is highest among girls exposed to chest irradiation at puberty, a time of intense breast development. Other factors associated with radiation-induced cancers in humans are patient age and physiological state, synergistic interactions between radiation and carcinogens, and genetic susceptibility toward radiation.
Non-ionizing radiation derived primarily from sunlight includes UV rays, which are carcinogenic to humans. Exposure to UV radiation is a major risk for various types of skin cancers including basal cell carcinoma, squamous cell carcinoma, and melanoma. Along with UV exposure from sunlight, UV exposure from sun beds for cosmetic tanning may account for the growing incidence of melanoma. Depletion of the ozone layer in the stratosphere can augment the dose-intensity of UVB and UVC, which can further increase the incidence of skin cancer.
Low-frequency electromagnetic fields can cause clastogenic DNA damage. The sources of electromagnetic field exposure are high-voltage power lines, transformers, electric train engines, and more generally, all types of electrical equipments. An increased risk of cancers such as childhood leukemia, brain tumors and breast cancer has been attributed to electromagnetic field exposure. For instance, children living within 200 m of high-voltage power lines have a relative risk of leukemia of 69%, whereas those living between 200 and 600 m from these power lines have a relative risk of 23%. In addition, a recent meta-analysis of all available epidemiologic data showed that daily prolonged use of mobile phones for 10 years or more showed a consistent pattern of an increased risk of brain tumors (64).
Fruits, vegetables, spices, condiments and cereals with potential to prevent cancer. Fruits include 1 apple, 2apricot, 3 banana, 4 blackberry, 5 cherry, 6 citrus fruits, 7 dessert date, 8 durian, 9 grapes, 10 guava, 11 Indian gooseberry, 12 mango, 13 malay apple, 14 mangosteen, 15 pineapple, 16 pomegranate. Vegetables include 1artichok, 2 avocado, 3 brussels sprout, 4 broccoli, 5 cabbage, 6 cauliflower, 7 carrot, 8 daikon 9 kohlrabi, 10onion, 11 tomato, 12 turnip, 13 ulluco, 14 water cress, 15 okra, 16 potato, 17 fiddle head, 18 radicchio, 19komatsuna, 20 salt bush, 21 winter squash, 22 zucchini, 23 lettuce, 24 spinach. Spices and condiments include 1 turmeric, 2 cardamom, 3 coriander, 4 black pepper, 5 clove, 6 fennel, 7 rosemary, 8 sesame seed, 9 mustard, 10 licorice, 11 garlic, 12 ginger, 13 parsley, 14 cinnamon, 15 curry leaves, 16 kalonji, 17 fenugreek, 18camphor, 19 pecan, 20 star anise, 21 flax seed, 22 black mustard, 23 pistachio, 24 walnut, 25 peanut, 26 cashew nut. Cereals include 1 rice, 2 wheat, 3 oats, 4 rye, 5 barley, 6 maize, 7 jowar, 8 pearl millet, 9 proso millet, 10 foxtail millet, 11 little millet, 12 barnyard millet, 13 kidney bean, 14 soybean, 15 mung bean, 16 black bean, 17 pigeon pea, 18 green pea, 19 scarlet runner bean, 20 black beluga, 21 brown spanish pardina, 22green, 23 green (eston), 24 ivory white, 25 multicolored blend, 26 petite crimson, 27 petite golden, 28 red chief.
Join Robert O Young PhD and Galina Migalko MD in Dubai on December 5th and 6th, 2018 for the Annual Conference on Bacterial, Viral and Infectious Diseases. They will be Key Note Speakers and doing a workshop on the New Biology.
For more information and to register go to: https://bacterialdiseases.infectiousconferences.com/organizing-committee.php
The following is the abstract for Dr. Young’s lecture:
Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner
There is now over 100 years of documented history and research on the Polio virus and whether or not its treatment by inoculation has been successful in eradicating Polio. I am suggesting in this article and in my lecture that there are significant findings based on historical and past and current research, including my own that the viral theory of Polio and possibly other modern-day diseases, such as Post-Polio Syndrome, Polio Vaccine-Induced Paralysis, Legionnaires, CNS disease, Cancer, HIV/AIDS and now Zika may be caused by acidic chemical poisoning from DDT (dichloro-diphenyl-trichloroethane) and other related DDT pesticides, acidic vaccinations, and other factors including lifestyle and dietary factors rather than from a lone infectious virus. I will present ten historical graphs outlining the history of Polio, the production of DDT, BHC, Lead, Arsenic, Polio vaccinations and the author’s theory that chemical poisoning, vaccination, and lifestyle and dietary choices are a more likely causes for the symptoms of Polio, neurological diseases, Cancer, HIV/AIDS and now Zika.
1. L. N. Kolonel, D. Altshuler, and B. E. Henderson. The multiethnic cohort study: exploring genes, lifestyle and cancer risk. Nat. Rev. Cancer. 4:519–27 (2004) doi:10.1038/nrc1389. [PubMed]
2. J. K. Wiencke. Impact of race/ethnicity on molecular pathways in human cancer. Nat. Rev. Cancer. 4:79–84 (2004) doi:10.1038/nrc1257. [PubMed]
3. R. G. Ziegler, R. N. Hoover, M. C. Pike, A. Hildesheim, A. M. Nomura, D. W. West, A. H. Wu-Williams, L. N. Kolonel, P. L. Horn-Ross, J. F. Rosenthal, and M. B. Hyer. Migration patterns and breast cancer risk in Asian-American women. J. Natl. Cancer Inst.85:1819–27 (1993) doi:10.1093/jnci/85.22.1819. [PubMed]
4. W. Haenszel and M. Kurihara. Studies of Japanese migrants. I. Mortality from cancer and other diseases among Japanese in the United States. J. Natl. Cancer Inst.40:43–68 (1968). [PubMed]
5. A. S. Hamilton and T. M. Mack. Puberty and genetic susceptibility to breast cancer in a case-control study in twins. N. Engl. J. Med.348:2313–22 (2003) doi:10.1056/NEJMoa021293. [PubMed]
6. A. Jemal, R. Siegel, E. Ward, T. Murray, J. Xu, and M. J. Thun. Cancer statistics, 2007. CA Cancer J. Clin.57:43–66 (2007). [PubMed]
7. F. Brayand, and B. Moller. Predicting the future burden of cancer. Nat. Rev. Cancer. 6:63–74 (2006) doi:10.1038/nrc1781. [PubMed]
8. P. Lichtenstein, N. V. Holm, P. K. Verkasalo, A. Iliadou, J. Kaprio, M. Koskenvuo, E. Pukkala, A. Skytthe, and K. Hemminki. Environmental and heritable factors in the causation of cancer—analyses of cohorts of twins from Sweden, Denmark, and Finland. N. Engl. J. Med.343:78–85 (2000) doi:10.1056/NEJM200007133430201. [PubMed]
9. K. R. Loeb, and L. A. Loeb. Significance of multiple mutations in cancer. Carcinogenesis. 21:379–85 (2000) doi:10.1093/carcin/21.3.379. [PubMed]
10. W. C. Hahn, and R. A. Weinberg. Modelling the molecular circuitry of cancer. Nat. Rev. Cancer. 2:331–41 (2002) doi: 10.1038/nrc795. [PubMed]
11. L. A. Mucci, S. Wedren, R. M. Tamimi, D. Trichopoulos, and H. O. Adami. The role of gene-environment interaction in the aetiology of human cancer: examples from cancers of the large bowel, lung and breast. J. Intern. Med.249:477–93 (2001) doi:10.1046/j.1365-2796.2001.00839.x. [PubMed]
12. K. Czene, and K. Hemminki. Kidney cancer in the Swedish Family Cancer Database: familial risks and second primary malignancies. Kidney Int.61:1806–13 (2002) doi:10.1046/j.1523-1755.2002.00304.x.[PubMed]
13. P. Irigaray, J. A. Newby, R. Clapp, L. Hardell, V. Howard, L. Montagnier, S. Epstein, and D. Belpomme. Lifestyle-related factors and environmental agents causing cancer: an overview. Biomed. Pharmacother.61:640–58 (2007) doi:10.1016/j.biopha.2007.10.006. [PubMed]
14. M. F. Denissenko, A. Pao, M. Tang, and G. P. Pfeifer. Preferential formation of benzo[a]pyrene adducts at lung cancer mutational hotspots in P53. Science. 274:430–2 (1996) doi:10.1126/science.274.5286.430.[PubMed]
15. R. J. Anto, A. Mukhopadhyay, S. Shishodia, C. G. Gairola, and B. B. Aggarwal. Cigarette smoke condensate activates nuclear transcription factor-kappaB through phosphorylation and degradation of IkappaB(alpha): correlation with induction of cyclooxygenase-2. Carcinogenesis. 23:1511–8 (2002) doi: 10.1093/carcin/23.9.1511. [PubMed]
16. S. Shishodiaand, and B. B. Aggarwal. Cyclooxygenase (COX)-2 inhibitor celecoxib abrogates activation of cigarette smoke-induced nuclear factor (NF)-kappaB by suppressing activation of IkappaBalpha kinase in human non-small cell lung carcinoma: correlation with suppression of cyclin D1, COX-2, and matrix metalloproteinase-9. Cancer Res. 64:5004–12 (2004) doi:10.1158/0008-5472.CAN-04-0206. [PubMed]
17. H. Ichikawa, Y. Nakamura, Y. Kashiwada, and B. B. Aggarwal. Anticancer drugs designed by mother nature: ancient drugs but modern targets. Curr Pharm Des. 13:3400–16 (2007) doi:10.2174/138161207782360500. [PubMed]
18. A. J. Tuyns. Epidemiology of alcohol and cancer. Cancer Res. 39:2840–3 (1979). [PubMed]
19. H. Maier, E. Sennewald, G. F. Heller, and H. Weidauer. Chronic alcohol consumption—the key risk factor for pharyngeal cancer. Otolaryngol. Head Neck Surg.110:168–73 (1994). [PubMed]
20. H. K. Seitz, F. Stickel, and N. Homann. Pathogenetic mechanisms of upper aerodigestive tract cancer in alcoholics. Int. J. Cancer. 108:483–7 (2004) doi:10.1002/ijc.11600. [PubMed]
21. R. Doll, and R. Peto. The causes of cancer: quantitative estimates of avoidable risks of cancer in the United States today. J. Natl. Cancer Inst. 66:1191–308 (1981). [PubMed]
22. R. R. Williams, and J. W. Horm. Association of cancer sites with tobacco and alcohol consumption and socioeconomic status of patients: interview study from the Third National Cancer Survey. J. Natl. Cancer Inst.58:525–47 (1977). [PubMed]
23. N. Hamajima et al. Alcohol, tobacco and breast cancer—collaborative reanalysis of individual data from 53 epidemiological studies, including 58,515 women with breast cancer and 95,067 women without the disease. Br. J. Cancer. 87:1234–45 (2002) doi:10.1038/sj.bjc.6600596. [PMC free article] [PubMed]
24. M. P. Longnecker, P. A. Newcomb, R. Mittendorf, E. R. Greenberg, R. W. Clapp, G. F. Bogdan, J. Baron, B. MacMahon, and W. C. Willett. Risk of breast cancer in relation to lifetime alcohol consumption. J. Natl. Cancer Inst.87:923–9 (1995) doi:10.1093/jnci/87.12.923. [PubMed]
25. F. Stickel, D. Schuppan, E. G. Hahn, and H. K. Seitz. Cocarcinogenic effects of alcohol in hepatocarcinogenesis. Gut. 51:132–9 (2002) doi:10.1136/gut.51.1.132. [PMC free article] [PubMed]
26. H. K. Seitz, G. Poschl, and U. A. Simanowski. Alcohol and cancer. Recent Dev Alcohol. 14:67–95 (1998) doi:10.1007/0-306-47148-5_4. [PubMed]
27. H. K. Seitz, S. Matsuzaki, A. Yokoyama, N. Homann, S. Vakevainen, and X. D. Wang. Alcohol and cancer. Alcohol Clin. Exp. Res.25:137S–143S (2001). [PubMed]
28. F. Donato, U. Gelatti, R. M. Limina, and G. Fattovich. Southern Europe as an example of interaction between various environmental factors: a systematic review of the epidemiologic evidence. Oncogene. 25:3756–70 (2006) doi:10.1038/sj.onc.1209557. [PubMed]
29. G. Poschl, and H. K. Seitz. Alcohol and cancer. Alcohol Alcohol. 39:155–65 (2004) doi:10.1093/alcalc/agh057. [PubMed]
30. G. Szabo, P. Mandrekar, S. Oak, and J. Mayerle. Effect of ethanol on inflammatory responses. Implications for pancreatitis. Pancreatology. 7:115–23 (2007) doi:10.1159/000104236. [PMC free article][PubMed]
31. B. B. Aggarwal. Nuclear factor-kappaB: the enemy within. Cancer Cell. 6:203–208 (2004) doi:10.1016/j.ccr.2004.09.003. [PubMed]
32. M. Kuratsune, S. Kohchi, and A. Horie. Carcinogenesis in the esophagus. I. Penetration of benzo(a) pyrene and other hydrocarbons into the esophageal mucosa. Gann. 56:177–87 (1965). [PubMed]
33. C. La Vecchia, A. Tavani, S. Franceschi, F. Levi, G. Corrao, and E. Negri. Epidemiology and prevention of oral cancer. Oral Oncol.33:302–312 (1997). [PubMed]
34. P. Boffetta, M. Hashibe, C. La Vecchia, W. Zatonski, and J. Rehm. The burden of cancer attributable to alcohol drinking. Int. J. Cancer. 119:884–887 (2006) doi:10.1002/ijc.21903. [PubMed]
35. W. C. Willett. Diet and cancer. Oncologist. 5:393–404 (2000) doi:10.1634/theoncologist.5-5-393.[PubMed]
36. S. A. Bingham, R. Hughes, and A. J. Cross. Effect of white versus red meat on endogenous N-nitrosation in the human colon and further evidence of a dose response. J. Nutr.132:3522S–3525S (2002).[PubMed]
37. A. Chao, M. J. Thun, C. J. Connell, M. L. McCullough, E. J. Jacobs, W. D. Flanders, C. Rodriguez, R. Sinha, and E. E. Calle. Meat consumption and risk of colorectal cancer. JAMA. 293:172–182 (2005) doi:10.1001/jama.293.2.172. [PubMed]
38. N. Hogg. Red meat and colon cancer: heme proteins and nitrite in the gut. A commentary on diet-induced endogenous formation of nitroso compounds in the GI tract. Free Radic. Biol. Med.43:1037–1039 (2007) doi:10.1016/j.freeradbiomed.2007.07.006. [PubMed]
39. C. Rodriguez, M. L. McCullough, A. M. Mondul, E. J. Jacobs, A. Chao, A. V. Patel, M. J. Thun, and E. E. Calle. Meat consumption among Black and White men and risk of prostate cancer in the Cancer Prevention Study II Nutrition Cohort. Cancer Epidemiol. Biomarkers Prev. 15:211–216 (2006) doi:10.1158/1055-9965.EPI-05-0614. [PubMed]
40. R. Garcia-Closas, M. Garcia-Closas, M. Kogevinas, N. Malats, D. Silverman, C. Serra, A. Tardon, A. Carrato, G. Castano-Vinyals, M. Dosemeci, L. Moore, N. Rothman, and R. Sinha. Food, nutrient and heterocyclic amine intake and the risk of bladder cancer. Eur. J. Cancer. 43:1731–1740 (2007) doi:10.1016/j.ejca.2007.05.007. [PubMed]
41. A. Tappel. Heme of consumed red meat can act as a catalyst of oxidative damage and could initiate colon, breast and prostate cancers, heart disease and other diseases. Med. Hypotheses. 68:562–4 (2007) doi:10.1016/j.mehy.2006.08.025. [PubMed]
42. L. H. O’Hanlon. High meat consumption linked to gastric-cancer risk. Lancet Oncol. 7:287 (2006) doi:10.1016/S1470-2045(06)70638-6. [PubMed]
43. T. N. Toporcov, J. L. Antunes, and M. R. Tavares. Fat food habitual intake and risk of oral cancer. Oral Oncol. 40:925–931 (2004) doi:10.1016/j.oraloncology.2004.04.007. [PubMed]
44. O. Dosil-Diaz, A. Ruano-Ravina, J. J. Gestal-Otero, and J. M. Barros-Dios. Meat and fish consumption and risk of lung cancer: A case-control study in Galicia, Spain. Cancer Lett.252:115–122 (2007) doi:10.1016/j.canlet.2006.12.008. [PubMed]
45. S. N. Lauber, and N. J. Gooderham. The cooked meat derived genotoxic carcinogen 2-amino-3-methylimidazo[4,5-b]pyridine has potent hormone-like activity: mechanistic support for a role in breast cancer. Cancer Res.67:9597–0602 (2007) doi:10.1158/0008–5472.CAN-07-1661. [PubMed]
46. D. Divisi, S. Di Tommaso, S. Salvemini, M. Garramone, and R. Crisci. Diet and cancer. Acta Biomed. 77:118–123 (2006). [PubMed]
47. Y. F. Sasaki, S. Kawaguchi, A. Kamaya, M. Ohshita, K. Kabasawa, K. Iwama, K. Taniguchi, and S. Tsuda. The comet assay with 8 mouse organs: results with 39 currently used food additives. Mutat. Res.519:103–119 (2002). [PubMed]
48. M. Durando, L. Kass, J. Piva, C. Sonnenschein, A. M. Soto, E. H. Luque, and M. Munoz-de-Toro. Prenatal bisphenol A exposure induces preneoplastic lesions in the mammary gland in Wistar rats. Environ. Health Perspect.115:80–6 (2007). [PMC free article] [PubMed]
49. S. M. Ho, W. Y. Tang, J. Belmonte de Frausto, and G. S. Prins. Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4. Cancer Res.66:5624–32 (2006) doi:10.1158/0008-5472.CAN-06-0516.[PMC free article] [PubMed]
50. A. Szymanska-Chabowska, J. Antonowicz-Juchniewicz, and R. Andrzejak. Some aspects of arsenic toxicity and carcinogenicity in living organism with special regard to its influence on cardiovascular system, blood and bone marrow. Int. J. Occup. Med. Environ. Health. 15:101–116 (2002). [PubMed]
51. E. E. Calle, C. Rodriguez, K. Walker-Thurmond, and M. J. Thun. Overweight, obesity, and mortality from cancer in a prospectively studied cohort of U.S. adults. N Engl J Med. 348:1625–1638 (2003) doi:10.1056/NEJMoa021423. [PubMed]
52. A. Drewnowski, and B. M. Popkin. The nutrition transition: new trends in the global diet. Nutr. Rev.55:31–43 (1997). [PubMed]
53. S. D. Hursting, L. M. Lashinger, L. H. Colbert, C. J. Rogers, K. W. Wheatley, N. P. Nunez, S. Mahabir, J. C. Barrett, M. R. Forman, and S. N. Perkins. Energy balance and carcinogenesis: underlying pathways and targets for intervention. Curr. Cancer Drug Targets. 7:484–491 (2007) doi:10.2174/156800907781386623. [PubMed]
54. A. Nareika, Y. B. Im, B. A. Game, E. H. Slate, J. J. Sanders, S. D. London, M. F. Lopes-Virella, and Y. Huang. High glucose enhances lipopolysaccharide-stimulated CD14 expression in U937 mononuclear cells by increasing nuclear factor kappaB and AP-1 activities. J. Endocrinol.196:45–55 (2008) doi:10.1677/JOE-07-0145. [PubMed]
55. C. H. Tang, Y. C. Chiu, T. W. Tan, R. S. Yang, and W. M. Fu. Adiponectin enhances IL-6 production in human synovial fibroblast via an AdipoR1 receptor, AMPK, p38, and NF-kappa B pathway. J. Immunol.179:5483–5492 (2007). [PubMed]
56. P. Pisani, D. M. Parkin, N. Munoz, and J. Ferlay. Cancer and infection: estimates of the attributable fraction in 1990. Cancer Epidemiol. Biomarkers Prev.6:387–400 (1997). [PubMed]
57. D. M. Parkin. The global health burden of infection-associated cancers in the year 2002. Int. J. Cancer. 118:3030–3044 (2006) doi:10.1002/ijc.21731. [PubMed]
58. S. Song, H. C. Pitot, and P. F. Lambert. The human papillomavirus type 16 E6 gene alone is sufficient to induce carcinomas in transgenic animals. J. Virol.73:5887–5893 (1999). [PMC free article] [PubMed]
59. B. S. Blumberg, B. Larouze, W. T. London, B. Werner, J. E. Hesser, I. Millman, G. Saimot, and M. Payet. The relation of infection with the hepatitis B agent to primary hepatic carcinoma. Am. J. Pathol.81:669–682 (1975). [PMC free article] [PubMed]
60. T. M. Hagen, S. Huang, J. Curnutte, P. Fowler, V. Martinez, C. M. Wehr, B. N. Ames, and F. V. Chisari. Extensive oxidative DNA damage in hepatocytes of transgenic mice with chronic active hepatitis destined to develop hepatocellular carcinoma. Proc. Natl. Acad. Sci. U S A. 91:12808–12812 (1994) doi:10.1073/pnas.91.26.12808. [PMC free article] [PubMed]
61. A. L. Jackson, and L. A. Loeb. The contribution of endogenous sources of DNA damage to the multiple mutations in cancer. Mutat. Res.477:7–21 (2001) doi:10.1016/S0027-5107(01)00091-4. [PubMed]
62. N. De Maria, A. Colantoni, S. Fagiuoli, G. J. Liu, B. K. Rogers, F. Farinati, D. H. Van Thiel, and R. A. Floyd. Association between reactive oxygen species and disease activity in chronic hepatitis C. Free Radic. Biol. Med.21:291–5 (1996) doi:10.1016/0891–5849(96)00044-5. [PubMed]
63. K. Koike, T. Tsutsumi, H. Fujie, Y. Shintani, and M. Kyoji. Molecular mechanism of viral hepatocarcinogenesis. Oncology. 62(Suppl 1):29–37 (2002) doi:10.1159/000048273. [PubMed]
64. D. Belpomme, P. Irigaray, L. Hardell, R. Clapp, L. Montagnier, S. Epstein, and A. J. Sasco. The multitude and diversity of environmental carcinogens. Environ. Res.105:414–429 (2007) doi:10.1016/j.envres.2007.07.002. [PubMed]
65. Y. S. Guan, Q. He, M. Q. Wang, and P. Li. Nuclear factor kappa B and hepatitis viruses. Expert Opin. Ther. Targets. 12:265–280 (2008) doi:10.1517/1472818.104.22.1685. [PubMed]
66. S. Takayama, H. Takahashi, Y. Matsuo, Y. Okada, and T. Manabe. Effects of Helicobacter pylori infection on human pancreatic cancer cell line. Hepatogastroenterology. 54:2387–2391 (2007). [PubMed]
67. K. A. Steinmetz, and J. D. Potter. Vegetables, fruit, and cancer prevention: a review. J. Am. Diet Assoc.96:1027–1039 (1996) doi:10.1016/S0002–8223(96)00273-8. [PubMed]
68. P. Greenwald. Lifestyle and medical approaches to cancer prevention. Recent Results Cancer Res.166:1–15 (2005). [PubMed]
69. H. Vainio, and E. Weiderpass. Fruit and vegetables in cancer prevention. Nutr. Cancer. 54:111–42 (2006) doi:10.1207/s15327914nc5401_13. [PubMed]
70. L. W. Wattenberg. Chemoprophylaxis of carcinogenesis: a review. Cancer Res. 26:1520–1526 (1966).[PubMed]
71. B. B. Aggarwal, and S. Shishodia. Molecular targets of dietary agents for prevention and therapy of cancer. Biochem. Pharmacol.71:1397–1421 (2006) doi:10.1016/j.bcp.2006.02.009. [PubMed]
72. H. Nishino, M. Murakosh, T. Ii, M. Takemura, M. Kuchide, M. Kanazawa, X. Y. Mou, S. Wada, M. Masuda, Y. Ohsaka, S. Yogosawa, Y. Satomi, and K. Jinno. Carotenoids in cancer chemoprevention. Cancer Metastasis Rev.21:257–264 (2002) doi:10.1023/A:1021206826750. [PubMed]
73. K. B. Harikumar, and B. B. Aggarwal. Resveratrol: A multitargeted agent for age-associated chronic diseases. Cell Cycle. 7:1020–1037 (2008). [PubMed]
74. G. L. Russo. Ins and outs of dietary phytochemicals in cancer chemoprevention. Biochem. Pharmacol. 74:533–544 (2007) doi:10.1016/j.bcp.2007.02.014. [PubMed]
75. R. Agarwal, C. Agarwal, H. Ichikawa, R. P. Singh, and B. B. Aggarwal. Anticancer potential of silymarin: from bench to bed side. Anticancer Res. 26:4457–98 (2006). [PubMed]
76. E. G. Rogan. The natural chemopreventive compound indole-3-carbinol: state of the science. In Vivo. 20:221–228 (2006). [PubMed]
77. N. Juge, R. F. Mithen, and M. Traka. Molecular basis for chemoprevention by sulforaphane: a comprehensive review. Cell Mol Life Sci. 64:1105–27 (2007) doi:10.1007/s00018-007-6484-5. [PubMed]
78. L. Chen, and H. Y. Zhang. Cancer preventive mechanisms of the green tea polyphenol (−)-epigallocatechin-3-gallate. Molecules. 12:946–957 (2007). [PMC free article] [PubMed]
79. P. Anand, C. Sundaram, S. Jhurani, A. B. Kunnumakkara, and B. B. Aggarwal. Curcumin and cancer: An “old-age” disease with an “age-old” solution. Cancer Lett. in press (2008). [PubMed]
80. F. Khanum, K. R. Anilakumar, and K. R. Viswanathan. Anticarcinogenic properties of garlic: a review. Crit. Rev. Food Sci. Nutr.44:479–488 (2004) doi:10.1080/10408690490886700. [PubMed]
81. G. Sethi, K. S. Ahn and B. B. Aggarwal. Targeting NF-kB activation pathway by thymoquinone: Role in suppression of antiapoptotic gene products and enhancement of apoptosis. Mole Cancer Res. in press (2008). [PubMed]
82. Y. J. Surh. Anti-tumor promoting potential of selected spice ingredients with antioxidative and anti-inflammatory activities: a short review. Food Chem. Toxicol.40:1091–1097 (2002) doi:10.1016/S0278-6915(02)00037-6. [PubMed]
83. Y. Shukla, and M. Singh. Cancer preventive properties of ginger: a brief review. Food Chem. Toxicol.45:683–690 (2007) doi:10.1016/j.fct.2006.11.002. [PubMed]
84. M. M. al-Harbi, S. Qureshi, M. Raza, M. M. Ahmed, A. B. Giangreco, and A. H. Shah. Influence of anethole treatment on the tumour induced by Ehrlich ascites carcinoma cells in paw of Swiss albino mice. Eur. J. Cancer Prev.4:307–318 (1995) doi:10.1097/00008469-199508000-00006. [PubMed]
85. C. K. Sen, K. E. Traber, and L. Packer. Inhibition of NF-kappa B activation in human T-cell lines by anetholdithiolthione. Biochem. Biophys. Res. Commun.218:148–53 (1996) doi:10.1006/bbrc.1996.0026.[PubMed]
86. R. A. Lubet, V. E. Steele, I. Eto, M. M. Juliana, G. J. Kelloff, and C. J. Grubbs. Chemopreventive efficacy of anethole trithione, N-acetyl-L-cysteine, miconazole and phenethylisothiocyanate in the DMBA-induced rat mammary cancer model. Int. J. Cancer. 72:95–101 (1997) doi:10.1002/(SICI)1097-0215(19970703)72:1<95::AID-IJC14>3.0.CO;2-9. [PubMed]
87. Y. Nakagawa, and T. Suzuki. Cytotoxic and xenoestrogenic effects via biotransformation of trans-anethole on isolated rat hepatocytes and cultured MCF-7 human breast cancer cells. Biochem. Pharmacol.66:63–73 (2003) doi:10.1016/S0006-2952(03)00208-9. [PubMed]
88. S. Lam, C. MacAulay, J. C. Le Riche, Y. Dyachkova, A. Coldman, M. Guillaud, E. Hawk, M. O. Christen, and A. F. Gazdar. A randomized phase IIb trial of anethole dithiolethione in smokers with bronchial dysplasia. J. Natl. Cancer Inst.94:1001–1009 (2002). [PubMed]
89. S. Shishodia, and B. B. Aggarwal. Diosgenin inhibits osteoclastogenesis, invasion, and proliferation through the downregulation of Akt, I kappa B kinase activation and NF-kappa B-regulated gene expression. Oncogene. 25:1463–1473 (2006) doi:10.1038/sj.onc.1209194. [PubMed]
90. R. Ghosh, N. Nadiminty, J. E. Fitzpatrick, W. L. Alworth, T. J. Slaga, and A. P. Kumar. Eugenol causes melanoma growth suppression through inhibition of E2F1 transcriptional activity. J. Biol. Chem.280:5812–5819 (2005) doi:10.1074/jbc.M411429200. [PubMed]
91. K. Sukumaran, M. C. Unnikrishnan, and R. Kuttan. Inhibition of tumour promotion in mice by eugenol. Indian J. Physiol. Pharmacol.38:306–308 (1994). [PubMed]
92. K. Imaida, M. Hirose, S. Yamaguchi, S. Takahashi, and N. Ito. Effects of naturally occurring antioxidants on combined 1,2-dimethylhydrazine- and 1-methyl-1-nitrosourea-initiated carcinogenesis in F344 male rats. Cancer Lett.55:53–59 (1990) doi:10.1016/0304-3835(90)90065-6. [PubMed]
93. M. Pisano, G. Pagnan, M. Loi, M. E. Mura, M. G. Tilocca, G. Palmieri, D. Fabbri, M. A. Dettori, G. Delogu, M. Ponzoni, and C. Rozzo. Antiproliferative and pro-apoptotic activity of eugenol-related biphenyls on malignant melanoma cells. Mol Cancer. 6:8 (2007) doi:10.1186/1476-4598-6-8.[PMC free article] [PubMed]
94. S. S. Kim, O. J. Oh, H. Y. Min, E. J. Park, Y. Kim, H. J. Park, Y. Nam Han, and S. K. Lee. Eugenol suppresses cyclooxygenase-2 expression in lipopolysaccharide-stimulated mouse macrophage RAW264.7 cells. Life Sci. 73:337–348 (2003) doi:10.1016/S0024–3205(03)00288-1. [PubMed]
95. H. P. Deigner, G. Wolf, U. Ohlenmacher, and J. Reichling. 1¢-Hydroxyeugenol- and coniferyl alcohol derivatives as effective inhibitors of 5-lipoxygenase and Cu(2+)-mediated low density lipoprotein oxidation. Evidence for a dual mechanism. Arzneimittelforschung. 44:956–961 (1994). [PubMed]
96. C. J. Rompelberg, M. J. Steenwinkel, J. G. van Asten, J. H. van Delft, R. A. Baan, and H. Verhagen. Effect of eugenol on the mutagenicity of benzo[a]pyrene and the formation of benzo[a]pyrene-DNA adducts in the lambda-lacZ-transgenic mouse. Mutat. Res.369:87–96 (1996) doi:10.1016/S0165-1218(96)90052-X. [PubMed]
97. D. P. Richardson. The grain, the wholegrain and nothing but the grain: the science behind wholegrain and the reduced risk of heart disease and cancer. Nutr. Bull.25:353–360 (2000) doi:10.1046/j.1467-3010.2000.00083.x.
98. H. E. Miller, F. Rigelhof, L. Marquart, A. Prakash, and M. Kanter. Antioxidant content of whole grain breakfast cereals, fruits and vegetables. J. Am. Coll. Nutr.19:312S–319S (2000). [PubMed]
99. J. L. Slavin, D. Jacobs, and L. Marquart. Grain processing and nutrition. Crit. Rev. Food Sci. Nutr.40:309–326 (2000) doi:10.1080/10408690091189176. [PubMed]
100. L. Chatenoud, A. Tavani, C. La Vecchia, D. R. Jacobs, Jr, E. Negri, F. Levi, and S. Franceschi. Whole grain food intake and cancer risk. Int. J. Cancer. 77:24–8 (1998) doi:10.1002/(SICI)1097-0215(19980703)77:1<24::AID-IJC5>3.0.CO;2-1. [PubMed]
101. D. R. Jacobs, Jr, L. Marquart, J. Slavin, and L. H. Kushi. Whole-grain intake and cancer: an expanded review and meta-analysis. Nutr. Cancer. 30:85–96 (1998). [PubMed]
102. L. Marquart, K. L. Wiemer, J. M. Jones, and B. Jacob. Whole grains health claims in the USA and other efforts to increase whole-grain consumption. Proc. Nutr. Soc.62:151–160 (2003) doi:10.1079/PNS2003242. [PubMed]
103. M. Eastwood, and D. Kritchevsky. Dietary fiber: how did we get where we are? Annu. Rev. Nutr.25:1–8 (2005) doi:10.1146/annurev.nutr.25.121304.131658. [PubMed]
104. A. McIntyre, P. R. Gibson, and G. P. Young. Butyrate production from dietary fibre and protection against large bowel cancer in a rat model. Gut. 34:386–391 (1993) doi:10.1136/gut.34.3.386.[PMC free article] [PubMed]
105. J. L. Slavin, D. Jacobs, L. Marquart, and K. Wiemer. The role of whole grains in disease prevention. J. Am. Diet Assoc.101:780–5 (2001) doi:10.1016/S0002-8223(01)00194-8. [PubMed]
106. K. S. Ahn, G. Sethi, K. Krishnan, and B. B. Aggarwal. Gamma-tocotrienol inhibits nuclear factor-kappaB signaling pathway through inhibition of receptor-interacting protein and TAK1 leading to suppression of antiapoptotic gene products and potentiation of apoptosis. J. Biol. Chem.282:809–820 (2007) doi:10.1074/jbc.M610028200. [PubMed]
107. F. H. Sarkar, S. Adsule, S. Padhye, S. Kulkarni, and Y. Li. The role of genistein and synthetic derivatives of isoflavone in cancer prevention and therapy. Mini Rev. Med. Chem.6:401–407 (2006) doi:10.2174/138955706776361439. [PubMed]
108. K. W. Lee, H. J. Lee, Y. J. Surh, and C. Y. Lee. Vitamin C and cancer chemoprevention: reappraisal. Am. J. Clin. Nutr.78:1074–1078 (2003). [PubMed]
109. B. A. Ingraham, B. Bragdon, and A. Nohe. Molecular basis of the potential of vitamin D to prevent cancer. Curr. Med. Res. Opin.24:139–149 (2008) doi:10.1185/030079907X253519. [PubMed]
110. F. W. Booth, M. V. Chakravarthy, S. E. Gordon, and E. E. Spangenburg. Waging war on physical inactivity: using modern molecular ammunition against an ancient enemy. J. Appl. Physiol.93:3–30 (2002).[PubMed]
111. G. A. Colditz, C. C. Cannuscio, and A. L. Frazier. Physical activity and reduced risk of colon cancer: implications for prevention. Cancer Causes Control. 8:649–67 (1997) doi:10.1023/A:1018458700185.[PubMed]
112. A. R. Shors, C. Solomon, A. McTiernan, and E. White. Melanoma risk in relation to height, weight, and exercise (United States). Cancer Causes Control. 12:599–606 (2001) doi:10.1023/A:1011211615524.[PubMed]
113. A. Tannenbaum, and H. Silverstone. The initiation and growth of tumors. Introduction. I. Effects of underfeeding. Am. J. Cancer. 38:335–350 (1940).
114. S. D. Hursting, J. A. Lavigne, D. Berrigan, S. N. Perkins, and J. C. Barrett. Calorie restriction, aging, and cancer prevention: mechanisms of action and applicability to humans. Annu. Rev. Med.54:131–152 (2003) doi:10.1146/annurev.med.54.101601.152156. [PubMed]
115. M. H. Ross, and G. Bras. Lasting influence of early caloric restriction on prevalence of neoplasms in the rat. J. Natl. Cancer Inst.47:1095–1113 (1971). [PubMed]
116. D. Albanes. Total calories, body weight, and tumor incidence in mice. Cancer Res.47:1987–92 (1987).[PubMed]
117. L. Gross, and Y. Dreyfuss. Reduction in the incidence of radiation-induced tumors in rats after restriction of food intake. Proc. Natl. Acad. Sci. U S A. 81:7596–7598 (1984) doi:10.1073/pnas.81.23.7596. [PMC free article] [PubMed]
118. L. Gross, and Y. Dreyfuss. Prevention of spontaneous and radiation-induced tumors in rats by reduction of food intake. Proc. Natl. Acad. Sci. U S A. 87:6795–6797 (1990) doi:10.1073/pnas.87.17.6795.[PMC free article] [PubMed]
119. K. Yoshida, T. Inoue, K. Nojima, Y. Hirabayashi, and T. Sado. Calorie restriction reduces the incidence of myeloid leukemia induced by a single whole-body radiation in C3H/He mice. Proc. Natl. Acad. Sci. U S A. 94:2615–2619 (1997) doi:10.1073/pnas.94.6.2615. [PMC free article] [PubMed]
120. V. D. Longo, and C. E. Finch. Evolutionary medicine: From dwarf model systems to healthy centenarians? Science. 299:1342–1346 (2003) doi:10.1126/science.1077991. [PubMed]
In September 2017, Brady released his book, The TB12 Method: How to Achieve a Lifetime of Sustained Peak Performance. In this book, Brady detailed exactly what he eats every day. One main feature of his diet is liberal amounts of alkaline foods and liquids.
In the mornings, Brady doesn’t eat a full meal. When he wakes up at 6:00 am, he drinks 20 ounces of alkaline water infused with electrolytes, including sodium, potassium, magnesium and calcium. He then drinks a smoothie and/or juices containing alkalizing grasses, vegetables, fruit, nuts and seeds. Two hours later, he has another glass of alkaline electrolyte-infused water, and a post-workout protein shake. Brady claims to drink somewhere between 12 and 25 glasses of alkaline water per day.
He also heavily encourages snacking. He usually snacks at around 11:00 am, just before lunch. For lunch, Brady will usually have a piece of fatty fish like salmon and a lot of green vegetables. In the afternoon, he may have another protein shake or protein bar, and around 6:00 pm, Brady eats dinner, which, again, consists of mostly green vegetables.
His book provides recipes for green juices, green soups, green salads, and a few carbohydrate recipes such as his pasta dish — which is odd, considering that he supposedly rarely eats carbs. But even Brady treats himself sometimes. He doesn’t often eat dessert, but he does give a recipe for his famous alkaline avocado ice cream.
His book also contains several alkalizing rules for eating. Brady won’t eat carbohydrates and protein together. He recommends eating carbs or protein with green vegetables instead, as he knows that this is better for assimilation and elimination.
Brady’s chef Allen Campbell says that 80 per cent of his diet is green vegetables and the rest of his diet is grass-fed organic steak and wild salmon.
Brady follows what he refers to as an alkaline lifestyle and diet created by Robert O. Young PhD, in order to minimize muscle inflammation caused by the buildup of lactic acid in the interstitial fluids of the Interstitium (see illustration below). This entails limiting ‘acidifying foods,’ which mostly includes starchy foods like potato, pasta, bread and ALL dairy products.
What is even more interesting is the list of acidic foods that Brady doesn’t eat. For Brady, caffeine, white sugar, white flour, dairy, and some nightshade vegetables — eggplant and mushrooms — are completely off the table. He also won’t consume olive oil if it’s used in cooking — but he’ll have it raw. And he won’t eat high sugar fruit, unless it’s in a smoothie.
Since there are profound benefits with Brady’s pH alkaline diet, and it is clearly sustaining his play on the field, there a 100’s of specific health and fitness benefits of the pH alkaline lifestyle and diet which are backed by published scientific evidence.
He claims that limiting acidic foods helps control the body’s pH balance. What one eats, drinks, breaths and thinks has a huge effect on the body fluids, including the blood plasma, interstitial and intracellular fluid pH which is ideal at 7.365.
Brady also knows that the alkaline lifestyle and diet can decrease the lactic acids that causes inflammation in the body, leading to ALL sickness and disease, including connective tissue disorders that can end an athlete’s career.
At 41 years young, which is considered ancient in football years, Brady says he wants to play at least another five years. While he is certainly capable, his pH Miracle lifestyle and diet will be a major reason he WILL achieve HIS goal.
Can positive or negative thoughts and emotions affect your body’s delicate biochemistry or the acid/alkaline pH balance?
Love, fear, joy, anger, sadness, happiness, resentment. Can positive or negative emotions affect your body’s physical, mental and spiritual health?
Is a woman more likely to become pregnant if she eats a lot of vegetables or if she were to go on a long, relaxing vacation?
Are you more likely to do cancer if you have a hot temper?
Do people who laugh a lot live longer?
Does your anxiety or fear of crowds, elevators, blood, heights, spiders, hospitals, or airplanes somehow affect your health?
My theory of one sickness, one disease and one health, set forth in what I call “The New Biology,” not only considers how our diet affects our physiology, but also how our psychology affects our physiology and how our psychology affects our spirituality.
Not only does the health of your body affect the emotions of your mind, but your thoughts and feelings can affect the health of your entire body.
Bottom line, your mental state is ever so critical. In so many ways, your mental state, if it’s negative, can create more metabolic acids than the acidic food that you’re eating
In fact, you can create two or three times more metabolic acids from your thoughts or your mental and emotional state than from ingesting highly acidic dairy, animal protein, sugar and alcohol.
So your thoughts are critical. Your thoughts or words do become matter, and can affect your physiology in a negative or positive way. Your thoughts do become biology. And the way that thoughts become biology is as follows:
When you have a thought or say a word, it requires electrical or electron energy for the brain cell(s) to produce those actions. And as you carry on with that thought, you are burning or consuming energy. And when you are consuming electrical energy in your thoughts, you are producing a biological waste product called acid which is an energetic waste product which can be measured in pH, oxidative reduction potential, rH2 or redox, hertz and decibels.
Next, if the metabolic acids from your thoughts are not properly eliminated through the four channels of elimination which are urination, perspiration, respiration or defecation (form women menstruation), then the acids from your thoughts are moved out into your connective and fatty tissues because it must not be allowed to affect the pH of the blood and the interstitial fluids of the interstitium or the fluids that surround the cells. The delicate pH balance of the blood and the interstitium must remain quite constant 7.365 to remain healthy.
What happens next is this. As the excess and overload of acid are thrown out into the body tissues or intestitium, this can easily lead to all sorts of symptomologies: lupus, fibromyalgia, Lyme’s, arthritis, muscle pain, fatigue, tiredness, obesity, cancerous breasts, cancerous prostate, cancerous stomach and/or bowels, indigestion, acid reflux, heart burn, heart attacks, multiple sclerosis, Parkinson’s, dementia, autism, and the list goes on and on.
For example let’s say you’ve been doing sadness or depression. This downer feeling is coming from a negative experience that you keep looping and re-looping in your head. It’s like a mind movie. It’s a mini-drama that you keep playing over and over. And because you are constantly thinking about it, eventually you even start to be concerned or worried about the fact that you are so preoccupied with the whole affair. So now in addition to the sad drama, you are experiencing upset about the fact that you’re having the drama itself. All of this thinking requires electrical energy and when you’re consuming electrical energy in the form of electrons you are also producing metabolic acids.
Do you know any angry people? You may not know it, but many people who become angry easily not only get angry at various people, events, and situations, but eventually they are irritated with themselves for being so angry at everything else. Anger, for instance, requires a tremendous amount of energy and emits a great deal of electrical energy. You have undoubtedly felt the vibrational energy of someone who is angry. Or maybe you have felt your own anger and how it can upset your physiology, i.e., especially upset your stomach and bowels with excess acid leading to indigestion, stomach pain, acid reflux or ulcers.
Even worse, many of these negative emotions are chronic and can be traced all the way back to early childhood experiences. So, at one level or another, it’s been going on for a long time and creating excessive acid all along.
For many people, early childhood represents some of the most fearful and vulnerable years. Have you ever wondered why you can’t remember much before age five or six? Many of those years are filled with fears and tears, mads and sads and how about the “bads”? Do you remember what happened when you were “bad?” Imagine the acid from those experiences. In addition to the punitive experience itself, imagine the acidity a child deals with by simply a) remembering such a “bad” experience or b) anticipating the possibility of another such “bad” experience…or c) both! Some “children” remember these events forever!
“O dear white children casual as birds, Playing among the ruined languages,
So small beside their large confusing words,
So gay against the greater silences, Of dreadful things you did…”
It is during these vulnerable and unprotected years that we often plant eternal seeds of emotion that will yield an unwelcome harvest of acidic internal results, perhaps throughout one’s entire life.
The turmoil between parents and children, not to mention the conflicts between children and children, have been documented by many thousands of social science books and articles.
“Children begin by loving their parents; after a time they judge them;
Rarely, if ever, do they forgive them.”
So, let’s take a look at all of that emotion. Perhaps you are feeling a strong emotion. It could be any emotion.
First of all, emotions are energy in motion. When you are (e)motional, you are energetic, either in a positive or negative way. And if you are energetic, you are literally energy in (e)motion. You are now producing metabolic acids at a very high rate which is a waste product of such (e)motions.
The rate of acid production in an (e)motional state can be even greater than that of someone who is jogging or working out. So, your thoughts do become biological or metabolic acids that can make you sick, tired, depressed, angry and even too fat or underweight.
When you start producing acids with your thoughts, words and actions, what happens inside? First, you activate the alkaline-buffering systems of the body in order to neutralize these (e)motional acids. The body begins making a primary alkaline buffer known as sodium bicarbonate. It’s actually made from the blood in corporation with the cover cells of the stomach and during its production, it creates a waste product known as hydrochloric acid.
Hydrochloric acid is a poisonous acidic toxin and cannot remain in the blood. So it is dropped down into the gastric pits of the stomach. This is why people get upset stomachs or become constipated when they are (e)motional. This increase of sodium bicarbonate is critical in maintaining the alkaline design of the body, the pH of 7.365 for the blood, and for maintaining alkalinity of the interstitial fluids. If these acids, including hydrochloric acid, are not buffered and/or eliminated through the four channels of elimination, they can create serious health challenges in your body, mind, and spirit.
On the other hand, positive (e)motions, such as love, peace, hope, faith, joy, forgiveness and charity can be alkalizing to the blood and tissues. These (e)motions require far less energy and can cause you to be relaxed in your mind and stop the playing of some acidic toxic movie in your head. Students of higher consciousness know that you can even enter into a state of bliss wherein you have no thoughts and wherein you are producing no metabolic acid.
For myself, I have decided to call this wonderful place “Young Love.” That’s because I exercise and meditate every day. And I Love it! And it raises my level of consciousness and positive connection with the world. The connections between “Young” and “Love” are numerous. My name is Young, of course, but more importantly, being young is a term we normally associate with being youthful, energetic, open, optimistic, and filled with excitement. And the ultimate purpose of life is Love. And Love is the sweetest expression of life. So Young and Love go together.
To be sure, I Love my exercising and it Loves me back in terms of its gifts to me. I find myself Loving this state of bliss daily which I know is helping to alkalize my body. That is why I am addicted to why I Love this type of alkalizing exercise that I do every day. It’s called a “Positive Addiction”. I Love to have my friends and guests work out with me as I lead them through the steps. I teach them the Young version of Yoga. I tell them that it is known as “Younga Yoga”. They Love that. (Well, at least they laugh.) It incorporates proper breathing, stretching, toning, mediation, relaxation, and of course some sweating to remove yesterday’s dietary and metabolic acid and to help bring me into a state of happiness and bliss.
Through my personal and clinical research, I have found that maintaining the alkaline design of my body with an alkaline lifestyle and diet is the most important thing anyone can do to live a happier and more blissful life. Having an alkaline day is a way of life that I call “Young Living”. I guarantee you that what I call “Young Love” will go hand-in-hand with the goal of “Young Living”.
Now this next thought is very important! The negative (e)motions of anger, resentment, and fear being the most powerful and acidifying of all (e)motions are all highly acidic to the blood and tissues and in many ways are paralyzing to all bodily functions. Over time, the fear of the unknown is probably the most powerful and acidic of them all. Fear is so devastating to the body that even if you’re on an alkaline diet, overcoming a serious health challenge is practically impossible.
In such a dire case, with what may seem to be little or no improvement, you might be wondering if the pH Miracle Lifestyle and Diet may not be working. You may be asking, “What else am I not doing that I should be? How come I feel the way that I’m feeling? I’m eating the right way, I’m drinking the right alkaline electron rich water, but I can’t seem to achieve the type of extraordinary health and energy that I’m seeking.”
In most cases like this, when you are eating and drinking correctly, it will come down to your negative acidic (e)motions or thoughts that are holding you back from achieving extraordinary health, fitness, mental clarity, happiness, and bliss. However, keep this in mind:
When you’re eating an alkaline diet and you are doing everything you know how to do, and yet you are overwhelmed with worry, doubt and negative (e)motions, thank God you’re eating an alkaline diet! If your body were not seriously in the alkaline direction, you might very well be experiencing a struggle for your life. Your acidic (e)motions can literally kill you. So the alkaline diet is the saving grace. Knowing that should give you the positive hope that you can hang on, get through the emotional stress, and still come out physically and mentally able.
Hope and positive expectations are always the key, and knowing that you are on an alkaline diet should aid significantly in boosting your hope and confidence. You can live without food for forty days. You can live without water for about four days. You can live without air for maybe four minutes. But you cannot live without hope and love at all. Hope, love, positive expectations, confidence in what you are doing, and trust in your own good intentions this is the key, and that’s what the pH Miracle Lifestyle and Diet will do for you. It will give you hope!
The leading cause of death in the world today is said to be heart attacks. But people are really having “thought attacks,” NOT “heart attacks.” There are studies showing that over 80% of all heart attacks are (e)motionally triggered. I have said that people don’t die of a heart attack. They die of a thought attack that medical science simply refers to as a heart attack because that’s the end result.
And if you have wondered if you can die from a broken heart, the answer is absolutely! And the cause? Acids from energy in motion or (e)motion. The loss of a cherished love one can increase your metabolic acids from the (e)motion to the point that it can stop your heart from beating and pumping life-giving blood throughout your blood vessels. And we all know or should know that life and death is in the blood, the most important “organ” of the body.
So let’s take a moment to talk about what I do when I have a client who’s in a highly negative acid-forming (e)motional situation and all the body fluids, including the blood, will show a decline in the pH even when this person has been eating an alkaline diet.
In order to buffer the acid forming (e)motions, the client will have to hyper-alkalize the blood and then the tissues in order to bring the body back into alkaline balance. When the client is hyper-alkalizing, the pH of the urine will increase into the high 8’s and even into the 9’s. Hyper-alkalization is necessary in order to overcompensate for the negative acidic producing (e)motions and to bring the body back to health, energy, vitality, hope, peace, harmony and love.
So, does a person have a fair chance of healing themselves from a degenerative disease or dis-ease like heart disease or cancer? Can you ever achieve a state of blissful happiness? Can you recover from the devastating shock of a loss or from having been diagnosed with a scary-sounding health challenge? I say “absolutely, YES!” And I just told you how.
Given the importance of (e)motions in cancer or acidic causation, etc., I have been particularly interested in the unique biochemistry of the “reptilian brain” which includes the Amygdala, a part of the brain associated with the senses and emotions and their storage or memory. Acid or sugar specifically activates the areas of the Amygdala. I have often wished that our traditional medical industry would spend some of their billions of research dollars checking out and verifying for the world what I have demonstrated for years that the pH Miracle electron-rich alkaline Lifestyle and Diet would be much more calming to the lower (e)motions of grief, shame, guilt, anger, fear, etc., responses of the reptilian brain as compared to a toxic acidic chemical drug.
A chemical acidic drug may temporarily calm a person down, but it will also inhibit the entire spectrum of normal and healthy functioning of the Amygdala. I am assuming here that most of us still value and are interested in the healthy functions of socialization, sexual attraction, and the enjoyment of the myriad of feelings associated with home and hearth. All of these wonderful human experiences and memories are also functions of the Amygdala every bit as much as the feisty adrenal functions responding to “fight and flight.”
In our attempts to find a chemical drug to treat almost everything, we (more often than not) create more problems than we eliminate one step forward and two steps backward. I know that attention deficit problems (ADHD) respond to an alkaline regimen….and hyperactivity is an Amygdala function. So it follows that an alkaline lifestyle and diet would produce less overall adrenal and most important Amygdala “stress” as well (really just the fight or flight mechanism by another name).
The pH Miracle electron-rich alkaline lifestyle and diet is calming to the mind and thus calms the negative (e)motions or energy in motion. This appropriate calming of the Amygdala function produces less “stress.” And, with less “stress” you have less “acid.” And, with less “acid” you have less sickness, dis-ease, so-called disease, depression and unhappiness. Understand NOW?
I have said that cancer is a four letter word ACID. When you are doing negative acidic (e)motions, such as anger, revenge, hate, sadness or depression, you are creating metabolic acids that can cause ANY and ALL cancerous conditions across all body tissues. If metabolic acids are not removed via urination, perspiration, defecation or respiration (menstruation -why women live longer), then they are delivered to body tissues. When constant excess acid from negative (e)motions are poured into the body tissues, the body tissues will degenerate causing a cancerous condition. Pharmaceutical companies are creating drugs addressing symptoms that may give you the illusion of feeling better, but they DO NOT deal with the causative metabolic acids from eating, drinking and negative acidic (e)motions. This can only lead to more physical and (e)motional pain and unnecessary suffering.
When you are in a negative (e)motional state, it can become impossible for you to heal your serious degenerative or acidic challenge. But, I will say this: if you are willing to commit to change and begin the alkalizing process, even if you are not completely out of your state of fear, anger, depression or anger, you will begin to put more “Young Life,” “Young Energy,” and “Young Love” into your mind, body and spirit.
I have found over the years that when you start feeling better, you start thinking better. And when you start thinking better, you start doing better. So, you don’t have to have your (e)motions completely under control in order to start losing weight, feeling better, reversing a serious illness, having more sustainable energy and to start being happy and more spiritually connected.
When you start the pH Miracle Lifestyle and Diet program, you are then making a conscious decision to try to do a little better. And, when you get on this healing path that leads to Young Living, Young Energy, and Young Love this gradual alkalizing process you start having those little and then those big pH miracles. You start feeling better and you start thinking better. And, when you start feeling and thinking better, you realize at some point that you have forgotten your depression and your sadness. Feelings of anger have disappeared and even what you were upset about. You soon forget what you were fearful about in the first place.
Why? These changes come about because you feel so good. You are rewriting your genetic expressions with your positive (e)motions. You are taking your alkalizing eraser and erasing all your past life’s negative emotions. On the pH Miracle Lifestyle and Diet your (e)motions or energy in motion will finally be under your control. You will become the master of your mind, body and spirit. You will be living an alkaline lifestyle and diet full of energy, happiness, bliss and love. You will be living and breathing “Young Love.”
To learn more about the affect of negative and positive (e)motions on the brain and body and to learn more about “Young Living” “Young Energy” and “Young Love” read, The pH Miracle, The pH Miracle revised and update, The pH Miracle for Diabetes, The pH Miracle for Weight Loss and The pH Miracle for Cancer – http://www.phoreveryoung.com
Come listen and learn from Key Note Speakers, Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner and Galina Migalko MSc, MD, NMD, in four different countries around the World as they lecture on non-invasive medical diagnostics, the interstitium, pH, nutrition and their break-through research on prevention and non-invasive treatments for cancer, diabetes, heart disease, arthritis, osteoporosis, lupus, multiple sclerosis, infections, and many more acidic-caused diseases.
To pre-register for one or more World Conferences please email email@example.com and receive an additional 10 to 20 percent discount on the listed early-bird pricing. You can also register by phone by calling 760 484 1075.
When you enroll in one of our Conferences you will receive a credit for a live and dried blood cell analysis, valued at 1200 euros.
Please check out the Countries, Cities, Dates and Pricing below!
By Inger Hartelius,
This article was initially published in the magazine ”Tidslerne”, (Danish Cancer Association Tidslerne) in January 2018.
I was diagnosed with pulmonary adenocarcinoma lung cancer in one of my lungs and lymph nodes near the esophagus in July, 2011. I chose to say NO to chemo and NO to radiation and today – six and a half years later after a life threatening terminal diagnosis. Today, I have no evidence of cancer in anywhere in my body.
In a small dark office, without windows, at the Pulmonary Department in Roskilde Hospital, my husband and I were informed that on the basis of tests from a PET-CT scanning, they had found lung adenocarcinoma, stage 2, R7 og 4L, T1bN3MO, a diagnosis so severe that the doctors in an interdisciplinary conference had booked me for chemotherapy and radiation at Herlev Hospital already the following week.
As written in my medical record, I was “appropriately in tears”, while saying no thank you to the offer and later also to an orientation on the treatment possibilities, side effects and potential consequences of the hospitals offer. An offer which, according to the doctor, could prolong life – not cure. And, it was a matter of a short extension of lifespan, which was also confirmed by the statistical evidence I asked for. Potentially it was a matter of just a few months.
Even before I got the final diagnosis, I wasn’t considering chemotherapy or radiation. Between the scan and the results I researched into alternative treatments.
Today I have no evidence and no symptoms of metastatic lung cancer. A CAT scanning in April, 2016 confirmed my belief of being cured of terminal metastatic pulmonary adenocarcinoma lung cancer. (No one has ever been cured of metastatic pulmonary adenocarcinoma lung cancer) In many ways I feel better than before I was diagnosed. I am 64 years old – and I believe that I have many more healthy years ahead of me.
Did they give the correct diagnosis? The doctor who gave me the results of the scan in April, 2016 asked himself this out loud while reading my medical journal. Am I just one of the lucky ones who indescribably doesn’t follow the statistics (approx. 1 year lifespan post diagnosis and with treatment), or is what I chose to do instead of chemo and radiation the reason why I am still alive, health and cancer free? Who knows?
Though it is difficult to know for sure why I have survived cancer it is important for me to tell the world that some of us actually survive cancer without the conventional treatments and also therefore avoid the medical side effects, one of which is death – and gaining many positive results, which we choose instead.
Many have asked me: How did you dare? This question actually surprises me because this wasn’t how I was thinking. Many tell me they think I am brave.
Before the diagnosis I thought that the people who chose the conventional treatments were extremely brave. How can they let their bodies be filled with chemo with all its horrible side effects, which often result in injuries both inside and outside the body, including death? To entirely trust the doctor’s hasty decisions on standardized cancer treatment programes, without being able to see what is happening and take control over one’s own life.
If I only had a few months to live I definitely didn’t want to spend it in a hospital. On top of that I had first hand experience seeing how chemotherapy didn’t only treat, but resulted in days and weeks of deathly side effects – potentially lasting the rest of life – sometimes with death as a consequence; maybe the treatments would also shorten my lifespan.
I couldn’t do it, as a calming nurse suggested after a consultation with the doctor: “Put your life in the hands of your doctor”. I would rather not!
I am very thankful for the nurse saying this to me. It was at a moment where I was consumed by the confusion of the diagnosis and thoughts of never getting to experience having grandkids, that something inside me became connected. I got myself together, dried my eyes, stood up straight and took my final decision. Either I would die from cancer or I would find another way to be cured!
A long, conventional treatment program wasn’t something I, nor my family, would let myself go through, instead I would look for other possibilities. I left the hospital in shock, but with a decision to go to an alternative way of treating my cancer.
Already, when I was told I needed to have a biopsy taken from the area in my lungs and the swollen lymph nodes, I got in touch with a volunteer at the Cancer Association ”Tidslerne”. I had Googled the risks of taking the biopsy, and was aware that there was a 25% risk that the cancer would spread afterwards.
No-one at the hospital had informed me of this. That is why I needed to talk to others. Simultaneously, the conversations strengthened me in my belief of following my gut feeling and pursuing alternative treatment methods for my cancer. Many others had done this before me with great results.
I read the book: Andreas Moritz: “Cancer is not a disease. It is a survival mechanism”. Some other possibilities were META-medicine, healing and Dr. Robert Young [i], who is known for having a highly effective approach to treating cancer. (over 80% success with terminal metastatic cancer and over 90% success with Stage 1, 2 and 3 cancers)
In Denmark I found advice and guidance by Dr. Claus Hancke, MD in Lyngby, who suggested high dose of Vitamin C intravenously as well as supplements of vitamins and minerals. I also consulted Frede Damgaard’s clinic of complementary treatment in Aarhus. Their key focus is on nutritional guidance supplemented with natural medicine/herbs, vitamins and minerals. His recommendations were built on extensive analysis of my body’s resources and weaknesses.
Descriptions of Dr. Robert Young’s live and dry blood tests combined with focus on the body’s resources and regulation of the body’s pH-levels is what spoke to me. I wrote an email to him and was later encouraged to call him. In the following conversation with one of Dr. Young’s assistants, I was encouraged to bring my husband and kids with me and come to California. I was lucky. There was a house available for us if we could come within a couple of days. They believed that with the serious diagnosis I had, I would have a greater chance of survival if i invested in a retreat at Dr. Young’s pH Miracle Center, in Valley Center, California.
It was a miracle: Being with my husband, kids and my son’s girlfriend was fantastic. Being in an avocado and grapefruit plantation in California and living in a house feeling like I was in the middle of a great dream during my life’s biggest nightmare. While we were there I asked myself many times: Am I dreaming?
Because a couple of days ago I was getting my head around the concept that I was going to die. Instead I was now in paradise, being inspired to change my mindset of why people get cancer. At the same time we were informed daily on how to live according to Dr. Young’s recommendations, to prevent cancer and get rid of it by building up the body’s resources, so that it will not accumulate cancer cells.
Dr. Young’s blood tests showed that I should not fear dying from that cancer which the doctors had discovered in my body. I had many resources I could activate and through a whole body cleanse I could rid my body of this cancerous condition.
The blood test took place in a large teaching room where there was plenty of space for all five of us and one of Dr. Young’s assistants. We were surrounded by posters and other interesting teaching materials. A small prick in the finger was enough to make a live blood test, and the seven drops of blood dried on a glass plate. I sat by Dr. Young and his computer and followed along. The others saw the tests on the wall. He placed the blood from my finger on the glass plates and placed them under a microscope connected to a computer and a projector.
It was fantastic getting to see the tests instantly with my own eyes. There was no waiting time and Dr. Young let me in on how he interpreted the tests. It was personal and caring; “Try to see the many regular round blood cells floating freely around each other surrounded by clear liquid. The more of these there are and the clearer the liquid, the better the blood’s ability is to clean and transport oxygen to your body. The liquid between the cells shows no sign that the current cancer is a serious threat to your body. Here some of the cells are aggregating, which is a sign of dehydration. And the shape of the cells here shows that you need more nutritional oils.”
In the dries blood tests Dr. Young was focused on the patterns in which the blood coagulated. Experience shows that patterns can tell a lot about a person’s health and current challenges and resources. In my tests it was clear that I had to focus on my immune system and my digestion. On top of that there was a sign that I had had a lot of heavy metals in my blood – maybe because of the long period in my life where I ate a lot of fish.
Along with the blood tests I tested the pH levels of my saliva and my urine every morning and night. There was space for improvement. The pH levels of my saliva and urine were between 5 and 6. It should in both cases be a minimum of 7.4, a little higher than the pH levels of the blood.
From the blood tests and the pH levels Dr. Young made a protocol, which I followed, telling me which special supplements I should take with my alkaline meals[ii] as well as which activities I should carry out.
First and foremost I had to drink approximately 4 liters of liquid every day as well as a glass of salt water every morning and night. The liquid should consist of juice from vegetables and water with high pH levels, preferably with freeze dried vegetable powder and liquid chlorophyll. [iii]. I also had to stay physically active on a daily basis and partake in various therapeutic treatments.
It was very in depth and I have to admit it was a little hard to grasp it all. Luckily my son was good at helping me stay on top of it all so I could go in depth with it all one step at a time.
After the blood test we moved our focus from the cancer in my body to building up healthier and a more well functioning body. An exciting journey into the pH Miracle lifestyle. We focused on how we could keep our blood alive and healthy while strengthening the body’s ability to maintain a high pH level. It was all about what we eat. what we drink, what we breath, what we think, as well as how we challenged ourselves both physically and mentally.
The days were full of exciting activities: Younga Yoga in the morning followed by Dr. Young’s workshop, breakfast with delicious avocado-smoothies, juice from vegetables and almond milk, food demonstrations, time in an infrared sauna, salt baths and activities on the center’s many training machines as well as hiking and running trips in the area.
In Dr. Young’s plan there was a therapeutic colonic hydrotherapy with 20 liters of liquid consisting of water with high pH-levels, powder of freeze dried alkaline vegetables, salt and chlorophyll. I got a minimum of one hour’s massage focused on activating my lymphatic system.
At home we started preparing alkaline food and I started training to run 5 kilometers. In the beginning it was just a small run where I live. I was exhausted. Later it was longer trips along the beach.
To make it easier to prepare the food we invested in an effective blender and a juicer. We also got an infrared sauna, a bathtub (for salt baths), a rebounder (to jump on), a colonic board (to frequently clean my colon with 20 liters of water) as well as a pH Miracle water ionizing machine. The cleansing ionized water played an especially big role in the change I could see in the pH levels of my saliva and urine – both in the morning and the evening. The pH levels rose steadily and landed somewhere between 9 and 10 in the urine and 7 and 8 in my saliva. The values are still at this level.
I had consultations with Dr. Pernille Knudtzon, MD, a psychologist and reflexologist. Dr. John Arnved, MD at the Lung- and Allergy clinic in Copenhagen followed me and tested my lungs frequently as well as my allergy reaction to mold. My own doctor followed my progress with blood tests to keep an eye on the mineral and vitamin levels in my body.
I was busy and sometimes completely overwhelmed with all the changes in my body and the doubt: Was it the right thing, I had started? Why was I still losing weight? Would I be cured? Just think… I didn’t trust my body completely; maybe the cancer was growing despite my hard work to get rid of it. The support of family, friends and the people whom I contacted for help was very important to me.
After three months I had two medical thermography scans with a month’s time between each. The results were quite shocking. The American doctors analysed the pictures and recommended that I start conventional treatment as the pictures showed that the cancer may have spread.
I decided to go to Spain to see Dr. Pernille Knudtzon, MD, who would supplement what I could do myself to be cured, with a week of intensive cleansing and building up of the body and soul. The experiences of the week with Pernille Knudtzon gave me new tools to tackle my thoughts and feelings so they weren’t in the way of my work on getting healthy. After a week in Spain with my sister I returned home with renewed courage. [ii]
In April, 2012 Dr. Young had a retreat in Como, Italy (pH Miracle protocol is now available at Forte Village, Sardegna, Italy) where I had the chance to regain inspiration and support to intensify my healing process. My husband and daughter went with me and we had a fantastic week. My blood tests again showed a big improvement, so Dr. Young recommended that I continue my process to take care of myself and my health.
In September of the same year Dr. Young invited me to another retreat in Como, Italy to give me another chance to be thermographically scanned and get an ultrasound by his partner, Dr. Galina Migalko (MD, NMD, RDMS).[iii] Neither test methods are harmful to the body. The tests showed, to everyone’s pleasure, that I had built up my immune system. It was now a year since I received the diagnosis and none of the tests showed any trace of cancer in my body. I had no symptoms either and had more energy and was starting to gain weight again.
When I came home, I decided that I wanted to share my journey. I needed to share my experiences with others to confirm to myself that it was a success. It could motivate me to continue living an alkaline lifestyle as taught by Dr. Young.
To stand in front of a large group of people and talk about how the lifestyle I had chosen had played a role in me being healthy, compelled me to continue. I knew now that ensuring the daily maintenance of my health was the best way for me to prevent the cancer from returning to my body. See the YouTube video: ”10 Steps to Perfect Health 2012”, a film about the workshop I had at the National Museum in Copenhagen with Paulo Fernandes, one of Dr. Young’s students.
In the summer of 2012 my son and sister took part in a course in California where Dr. Young was teaching his experiences and theories behind his way of analyzing living and dried blood tests. They both brought a microscope with them home so that they could connect to their laptops. I could now sit with them and see my own blood. They got very good at analyzing it, which gave us all the possibility to frequently keep an eye on how our bodies reacted to different challenges and changes in our lives.
When I saw my blood tests after an appendicitis which ended in a burst appendix, it was clear that I now had to invest in my cleansing activities. In this period I started coughing, losing weight and sweating again. The fear of the intensive operation meant that again there would be cancer in my lungs. Cancer with renewed power. I felt weak and powerless.
The family was again there to help me get back on track. My blood tests showed progression. An ultrasound scan at the Scanning clinic in Herlev showed that my inner organs were healthy and in good shape. At the same time the test that I had done at the Allergy and Lung clinic in Copenhagen showed that my lungs were not seriously affected by the cough. Dr. John Arnved, MD, dared to say that such positive results wouldn’t be there if the cancer was growing in my lungs again. So he encouraged me to start up my runs by the beach again so I could cough up what was irritating my lungs. Fantastic advice – I ran again for my life and coughed a lot by the beach for a couple of days. After a week’s time I discovered that I wasn’t coughing anymore! Wow! All this fear for no reason.
As previously said I renounced contact with the hospital. I knew from what I had read that it was very hard for the body to be scanned. I was also very aware of the psychological challenges. Both the experience of being in the scanner, the waiting time between the scan and the results as well as the thick atmosphere I experienced with the results coming in. It is not easy to have hope for life in such a universe. In the big picture though I managed with help from all those who believed in my decision. The time periods in the beginning where I had mistrust and ideas about how it would be to die from lung cancer died out, so in 2016 I built up the courage to be CT scanned. I wanted to know if such a test also confirmed that I was cured from metastatic pulmonary adenocarcinoma lung cancer..
The CT scan in 2016 showed that the area which was compressed in my lung was still the same size, and there were also no more swollen lymph nodes. According to the doctors there were scars from the original cancer in the lung.
There was also a little compression of 8 millimeters further down the lungs. They wanted to follow the little spot, so I had some more tests done a couple of months later. The next test showed that there was still no change, not even in the small 8mm compression.
After this I again said no thank you to the hospital’s offer for further investigations. When the compression hadn’t changed in over five years and there were no signs of enlarged lymph nodes or signs of cancer in any other parts of my body, I didn’t wish to provoke my body with more physically and psychologically stressful investigations.
I still want to continue living an alkaline lifestyle, not because I need to, but because I experience that it is life affirming on many levels. It gives me a special energy and courage, which I in no way wish to lose.
It is fantastic and strengthens my belief that I still have many more healthy years ahead of me. I get a lot of time to be there for those whom I love and those I can share an active work life with. I also have the belief that there will be many years, where I can be the grandmother of my grandchildren when they come one day.
 Robert Oldham Young CPT, MSc, DSc, PhD, ND, is a naturopathic practitioner and not a medical doctor. The titles after his name represent different doctoral graduations he has obtained in the USA where he has, among other things, studied nutrition, hematology, microbiology and chemistry. As a practitioner he has worked as an American Naturopath. He is also the author of 75 books published in 29 different languages, 20 peer-reviewed published articles, over 3000 blog published articles and hundreds of youtube videos concerning alkaline nutrition, lifestyle, detoxification, human pH research and chemistry of the blood and interstitium. www.drrobertyoung.com, https://www.youtube.com/user/pHMiracleCenter, https://www.amazon.com/Robert-O.-Young/e/B001ILKCSU/ref=sr_tc_2_0?qid=1526157267&sr=8-2-ent
He is now practicing in Marbella, Spain and Sardegna, Italy, and produces delicious, organic, alkaline products in Italy and the USA: www.iJuicenow.com, www.phoreveryoung.com, www.phmiraclestore.com, www.alkalinecare.com, and www.phmlife.com.
You can contact Dr. Young at the following email addresses: firstname.lastname@example.org and email@example.com
Meals containing food which produce as little acid as possible and as much alkaline as possible in the body when they are digested.
Chlorophyll is the green pigment found in plants. It can be extracted from green plants and algae. It contains magnesium and antioxidants. The material in its basic structure is similar to the molecules of our blood. It can help increase the production of red blood cells, cleanse the body from poison and waste products hence raising our energy levels. www,ijuicenow.com
[ii] Pernille Knudtzon is one of Europe’s most groundbreaking doctors. She is a traveller in the field of health and says: “Health is a choice – you can make a difference”. Residing in Spain, she hosts consultations, lectures, workshops and retreats – helping thousands of people overcome serious illnesses – also in Denmark. Read more on http://www.vitafakta.es. At Pernille Knudtzon’s clinic you can, among other things get support to cleanse and rebuild your body on several levels. You can receive live and dried blood tests, medical thermographic scans and deep insight into yourself and your healing potentials.
[iii] Galina Migalko MD, MND, RDMS, is a medical doctor with a speciality in non-invasive medical imaging, diagnostics and naturopathic medicine. http://www.universalmedicalimaging.com and firstname.lastname@example.org
Taking YOUR reservations NOW – Email us at: http://email@example.com
Watch the video below as YOU see what YOU will experience during your stay at one of the most incredible resorts and medical spas in the World!
1) A beautiful deluxe Bungalow within walking distance of the Spa, Pools, and beaches!
2) Alkalizing green juices daily
3) Alkaline breakfast, lunch, dinner and snacks
4) Seven alkalizing detoxing and energizing pools
5) Non-invasive blood testing (150 blood parameters tested), thermography and live and dried blood cell analysis
6) Daily Yoga and mediation
7) Beautiful white sand beaches
8) Tennis (Clay, Grass and Hard Courts)
9) Salt water, aloe vera and magnesium pools for detoxification and pain relief
14) Fitness Training Center
15) Daily Lymphatic Massage
16) Sports Center
17) Infrared Sauna
18) Turkish Bath
19) Steam Bath
21) Night Life and Summer Concerts including the group Sting in July
22) Eighteen Restaruants
Check out the map of the Forte Village Resort!
Over 40 acres of beauty!
To learn more or for pricing contact us at: firstname.lastname@example.org or call 760-484-1075