Category Archives: Diabetes

Dr. Robert O Young Lectures at Harvard

Towards the Ethics of Healing

Towards the Ethics of Healing begins the debate on human health and how we should treat any sickness and disease. Watch and learn the truth about Dr. Young’s work, research and findings and learn how to prevent, treat and reverse heart disease, diabetes, cancer, autism, just to name a few in this 3 Part lecture at Harvard University School of Medicine.

The Law of Change – Part 2

The New Biology – Part 3

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To learn more about research of Dr. Robert O Young go to: www.drrobertyoung.com and/or www.phmiracleproducts.com Education and pH Miracle retreats: www.phmiracleretreat.com
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Life Changing and Life Saving Knowledge!

Life transforming knowledge!

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Thank you Dr Young! Living the Alkaline Lifestyle since 2011! đŸ˜đŸ™đŸ»â€ïžEvery symptom of dis-ease is gone!

The Most Important Article I Have Ever Written!

What Causes Cancer, Heart Attacks, Strokes, Diabetes, MS, Lupus, Dementia, Sepsis, Multiple Sclerosis and the list goes on and on and on?

A rise in the alkalinity of the blood plasma above pH 7.365 (alkaline phosphate)–any rise–is a result or a compensatory reaction due to over-acidity in the interstitial fluids of the Interstitium (I call this dis-ease latent tissue acidosis) as the blood attempts to maintain its delicate pH at 7.365.

There is no exception for the rule of alkalinity.

The body will ALWAYS overcompensate for the excess acidity in the Interstitium fluids (the largest organ of the human body) by over-alkalizing the blood to maintain homeostasis.
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I call this the “teeter-totter” effect.
Along comes the traditional medical attendant and perceives that there is too much alkalinity, when really there is not.
This is an important concept to grasp, so let’s oversimplify a bit. The Interstitium fluids have become acidic.

The blood “knows” that!

So, it pours out extra alkalinity or alkaline phosphate into the blood and the blood pH spikes up to a higher than normal pH.
It’s like when we get the bejeebers scared out of us by something innocent, we over-react.
When suddenly alarmed, a person might scream, holler, faint, get mad, strike out, drop the vase, kick the dog, or even have a heart attack. The blood does the same thing. A knee-jerk reaction
well, actually, a blood-jerk reaction.
Alternatively, how many times have you heard of a car going off the shoulder of the road and the driver over-reacts, jerks the wheel back, and flies into the other lane of oncoming traffic. It happens all the time. Incidentally, if that does happen to you, you’re better off not to interfere. Stay on the shoulder. Let the wheel stay there for a moment. Slow the car down. But don’t overreact.
Mainstream medicine, not understanding the cause of the excessive alkalinity pouring into the blood, may try and stop the rushing over-alkalization. But that’s the wrong move.

We’re better off not to interfere.

Once more. When your little boy falls down, sees mama going out the door, or is scared of the boogey man, what happens? He not only cries, but how often do we see a child go into a big, fat over-reaction?
Sometimes, they really get worked up. It’s a natural over-reaction to a typical situation.
Now it’s Dad’s turn to over-react. Along comes Dad and says to keep quiet, don’t be such a little sissy, put a lid on it, grow up, stop that crying, OR ELSE

Since I have digressed to make a point, I may as well digress all the way. Wrong move, Dad. If you do that often enough, the message you send to your child is don’t have feelings, don’t express your feelings, you are not acceptable, don’t act like a child even though you are a child, and don’t be who you are. So don’t over-react Dad. Better to let the child get it out, stay in the room, validate their feelings, and use a little Active Listening (www.gordontraining.com).
Strong feelings can come and go
or come and stay. If you’re really klutzy, you could be orchestrating chronic emotional issues for a lifetime.
Gee, thanks Dad.

Now, back to the blood and Interstitium fluids.

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Interstitial fluids of the Interstiitum are acidic. Here comes a flood of alkalinity–even so much that the pH rises in the blood plasma and concerns the western medical establishment.
But whatever it was that caused the pH to over-react must be understood. Acidic Interstitium fluids mean problems ahead, correct? (We are the only research group in the World measuring the pH of the Interstitium)
Not only do we need alkalinity but lots of it!
The acidic Interstium fluids will soon even out the rise in
blood pH, and we will need additional alkalinity to wipe out the acidic Interstium fluid problem.
Cancer, heart disease, stroke, diabetes, MS, sepsis, etc. are not diseases of alkalinity but are diseases of acidity!
The body uses the calcium of the bones as well as other buffers (bicarbonate, hemoglobin, sodium, magnesium from the muscles, etc.) to chelate or buffer acidity! That is why there are always microcalcifications found in the liver, pancreas, breast, bowels, bladder, bones, prostate, muscles, joints, blood vessels and brain before the liver, pancreas, breast, bowels, bladder, bones, prostate, muscles, joints, blood vessels and brain cancer tumor shows up.

Why prior to the tumor?

Because the body will always try and protect and preserve the blood and Interstitium by buffering acids with the alkalinity of calcium. The bones are always affected in any cancer, heart condition, stroke, diabetes, lupus, arthritis, MS, sepsis, etc. because the bones are an excellent source of calcium for buffering dietary and
metabolic acids.
So is Cancer, Heart Attack, Stroke or Diabetes, MS, dementia, ALS, sepsis, HIV, etc., the disease?
No!
Then is the loss of bone mass the disease or the calcium deposits in the liver, pancreas, breast, brain, etc. the disease?
NO! NO!
Is the increase in the alkaline phosphates the disease?
NO! NO! NO!
These are all symptoms, not diseases!
Then the disease must be the over-acidity?
Well yes, and no.

Then what is the disease?

The “yes” part I call acidosis or hyperacidity. That is an acceptable term for the condition.
But it is really much more.
The “no” part is that it’s more than acidity. It’s a psychological disorder. It’s a sociological malaise. It’s a cultural-anthropological phenomenon. And once people understand the truth and the scientific foundation of “New Biology”TM, and once people understand the science of what I have been writing about for three, the better part of 4 decades, it may then become to be understood as a “moral disease” as well.
And why is that, you ask?
Is committing suicide a moral issue?
Well, yes.
Is drinking yourself to death a moral issue?
Well, yes.
Is allowing your children to become obese flying in the face of natural law?
Well, yes, assuming you are aware of what’s happening and have other options.
If you say “yes” to these last few questions, then we are looking at a very, complex psychological, sociological, cultural, biological and moral phenomenon.
Once you know and believe that over-acidity causes every disease and ALL dis-ease, then to ignore that fact is a form of suicide!
Just like Samson who killed a 1000 Philistines with the jawbone of an ass, every day people use same instrument with their daily lifestyle and dietary choices – the jawbone.
When you eat poorly, you pull the trigger every day of your life, and eventually, the gun fires. The bullet might hit you square in the head like a massive heart attack or stroke, or it may kill you more slowly like a cancer, or it may simply put you in a fog for the next 15 years like Alzheimer’s or dementia.
This “disease-phenomenon” is an inverted way of living, eating, drinking, breathing, thinking and believing!!!
Yes, this is the cause of ALL disease–ALL that disturbs the central balance of organized matter that leads to excess acidity in the blood and then the Interstium fluids that then leads to genetic mutations and the death of the cell, the tissues, the organs and the glands. In other words the death of the whole body, physically, emotionally and spiritually.
It is ALL that leads to increases in alkaline phosphates.
It is ALL microcalcifications in the liver, pancreas, breast, brain, prostate, and so on, and so on.
ALL liver, pancreas, breast, bladder, bowel, prostate, brain, etc., tumors. ALL liver, pancreas, breast, bladder, bowel, prostate, brain, etc. cancer and ALL potential bone cancer!!!!
First, we must understand that ALL of the above sicknesses and diseases are NOT sicknesses or diseases but a symptom of acidosis and catarrh that has built up in the blood and intestitial fluids that has significantly effected the white blood cells’ ability (the janitorial and garbage collectors for the blood and tissues) to remove metabolic, dietary acids and degenerative matter.
When we are dealing with any symptom or any biological effect, we need to look to the cause. To understand the cause is not difficult nor is the understanding of the treatment.
The “New Biology”TM explains the cause and effect of all sickness and disease in addition to explaining how to improve the quality and quantity of life.
For example, enervation (the deprivation of force or strength) and muscle weakness per se is not a disease.
Weakness, or lost power, is not a disease; but, by causing a flagging of the elimination of tissue-waste which is toxic, the blood becomes charged with acids.
I call this Acidosis–poison in the blood and then the Interstitium fluids. This is disease and when the acidic toxins accumulate beyond the toleration point, a crisis takes place. This means that the poison or acid (the so-called virus) is being eliminated–often through the skin, the third kidney.
We can call this disease, but it is not!
The only disease is systemic Acidosis which localizes in the weakest parts of our body.
And what we call disease, are symptoms produced by the forced vicarious elimination of acids from the interstitial fluids of the Interstitium through the mucous membrane.
When the elimination takes place through the mucous membrane of the nose, it is called a cold–catarrh of the nose. And where these crises are repeated for years, the mucous membrane thickens and ulcerates, and the bones enlarge, closing the passages.
At this stage, hay fever or asthma develops.
When the throat and tonsils, or any of the respiratory passages, become the seat of the crises of acidity, we have croup, tonsillitis, pharyngitis, laryngitis, bronchitis, asthma, pneumonia, etc.
When the acids locate in the cranial cavity we have dementia, Parkinson’s, Alzheimer’s, muddle thinking, forgetfulness, and even depression.
When the acids locate in the gastrointestinal tract we have IBS, gastrointestinal dysmotility, autonomic dysfunction, carotid stenosis and ischemic colitis.
When the acids are expressed through the skin we have psoriasis.
When the acids locate in the liver, pancreas, breast, bowels, bladder, prostate, uterus or brain tissue we have microcalcifications of these acids that then leads to tumors and liver, pancreas, breast, bowels, bladder, prostate, uterus or brain cancer.

What is in the name?

All are symptoms of the expulsion of acids from the blood, to the Interstitum fluids at the different points named.
They are of the same character essentially and evolve from the one cause, namely, systemic acidosis, as a crisis of toxemia.
The description can be extended to every organ of the body, including the largest organ, the skin, causing melanoma cancer.
For any organ that is enervated below the average standard from stress of habit, from work, or worry, from injury, or any other cause, that organ may become the location of the crises of systemic acidosis.
The symptoms presented differ with each organ affected. That fact gives color to the erroneous belief that every symptom-complex is a separate and distinct disease.
But, thanks to the new light and knowledge of being shed by the “New Biology”TM upon nomenclature involved in the naming of a disease, every symptom-complex goes back to the one and only cause of all diseases, namely, systemic acidosis of the Interstitium fluids and then the blood plasma and finally the intracellular fluids causing genetic mutation to the death of the cell.
To find the cause of all symptomologies including cancer, heart attack, stroke, diabetes, sepsis, muscular dystrophy, AIDS, sepsis, etc., start with colds and catarrh, and watch the pathology as it travels through “The Seven Stages of Acidity(TM), beginning with:
1) enervation,
2) sensitivity and irritation (IBS),
3) catarrh, (the common cold)
4) inflammation, (arthritis)
5) induration (lupus, lymes, fibromyalgia),
6) ulceration and then to (colitis)
7) degeneration – cancer, heart attack, stroke, diabetes, multiple sclerosis, polio, multiple dystrophy, ALS, etc.
How well could you try to find the cause of man by ignoring his conception, embryonic life, childhood, manhood, etc.
Nature’s order is interfered with by enervation habits until systemic acidosis of the Interstitium fluids and then the blood is established.
Then a vaccination (seen in Gulf War Syndrome and Spanish Flu Epidemic) or an infection (really an outfection) from any source will act as a firebrand. Sooner or later cause the most vulnerable organ (the bowels) will undergo organic change. The organ, however, has nothing to do with cause, and directing treatment toward the organ compounds the problem and is nonsense!
Examples of this wrong thinking yield blood transfusions for pernicious anemia, gland treatment for gland impotency, the cutting out of stones, ulcers and tumors.
There is no question that one of the most pernicious practices in vogue today is treating so-called disease with disease and immunizing with the products of disease.
Current medical science calls this form of pathological thinking a “vaccination.” This my friends is the real psuedo-science!
When the cause is not known, how is prevention or cure possible except by luck?
Producing a mild form of smallpox using vaccine is the same as introducing a poison or acidity into a healthy person.
It makes no sense!
Certainly only pathological thinking can arrive at such conclusions. Vaccine or autogenous remedies (metabolic acids) are made from the products of disease.
This also includes ALL antibiotics which are products of disease!
The idea that disease can be made to cure itself is an end-product of pathological thinking!
If prevention and cure mean producing disease, surely prevention and cure are not desirable. If prevention can be accomplished, then cures will not be needed!
It is not disease, it is cause “in all its aspects” that we need to know before we can take steps to prevent or cure “disease.”
Cause is constant, ever present, and always the same. Only effects, and the object on which a cause acts, change.
And the change is most inconstant.
To illustrate: a catarrh of the stomach presents first irritation, then inflammation, then ulceration, and finally indulation and cancer.
Not all cases run true to form. Only a small percentage evolve to ulcer and fewer reach the cancer stage. More acidic toxins exit via acute food poisoning or acute indigestion then by chronic diseases.
Most Americans are challenged with the symptomology of indigestion, which can include acid reflux, diarrhea and/or constipation.
The proper way to study disease is to study health and every influence favorable or not favorable to its continuance.
Our western system of medicine has been preoccupied with the study of disease, not health.

Disease is Perverted health!

Any influence that lowers energy becomes disease producing. Disease cannot be its own cause, neither can it be its own cure and certainly not is own prevention!
My personal discovery of the truth of ALL sickness and disease – that acidosis is the cause of all so-called diseases–came about slowly, step by step, line upon line, precept upon precept, here a little and there a little.
At first, I postulated that yeast and molds must be the general cause of disease. Then I decided that it was not yeast and molds but that the body becoming enervated. But wait a minute, enervation is not a disease; disease must be due to dietary, metabolic, respiratory and/or environmental acids.
I reasoned that localized or systemic acidosis is the true general cause of all disease and must be autogenerated. And if disease is due to autogenerated acids, what is the cause of that autogeneration?
The answer is found in understanding the nature of matter and how it organizes and disorganizes. I realized that there must be a physical or emotional disturbance to organized matter before it can begin its disorganization.
And when matter begins to disorganize, it gives rise to autogenerated acids. This is true for all matter!
To illustrate, take a physical injury to a joint which is often complicated with the prior symptom of rheumatism. The rheumatism previous to the injury was potentially in the blood and/or tissues. Just what change had taken place in the matter which, under stress of injury or shock of any kind, would cause a reaction with fever?
I could not understand until the “Acidosis Theory” suggested itself to my mind. After that, the cause of disease unfolded before me in an easy and natural manner.
I called this new paradigm for ALL sickness and disease “The Cycle of Imbalance.” You can read about “The Cycle of Imbalance” in my book, “Sick and Tired, Reclaim You Inner Terrain.” You can order this book at:
In a few words, without systemic acidosis, there can be no sickness or disease and there can be NO CANCER! NO HEART ATTACK! NO STROKE! NO DIABETES! NO DEMENTIA! NO SEPSIS, NO ALS, NO ALZHEIMERS, in other words, NO DIS-EASE!
It is also true that without systemic acidosis there can be NO PAIN!
Therefore, pain equals acid and acid equals pain.
I knew that the waste products of cellular disorganization and metabolism were toxic and that the only reason why we were not poisoned by it was because it was removed from the organism as fast as it was produced.
Then I discovered that the acid was retained in the blood and then interstitial fluids of the Insterstitium when there was a checking of elimination.
Then, the cause of the checking had to be determined. In time, I thought out the cause of all sickness and disease. I knew that when we had normal energy, organic functioning was normal.
Then came the discovery that enervation caused a checking of elimination.
Eureka!
The cause of ALL sickness and disease is NOW found!
Enervation checks elimination of the dietary, metabolic, respiratory and environmental acidic waste products!

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of cellular transformation or disorganization.
Retention of metabolic, respiratory, environmental and dietary ACIDS is the first and the only cause of ALL sickness and disease!
One of the first things to do to get rid of any so-called disease is to get rid of all the acid, for it is this state of the blood and then Interstitium fluid that makes disease possible.
Infection, drugs and food poisoning may kill, but if they do not, they will be short-lived in a subject that is free from enervation and acids that are being held in the compartments of the Interstitium fluids.
Conversely, the poisoning will linger in the system until the acid is overcome. Then and only then will elimination remove all traces of outfection – ACID.
Syphilitic outfection (herpes, warts, moles of any color) is pronouncedly an acidic subject thrown into great virulency by poor nutrition, lifestyle and conventional treatment.
The same is true with HIV/AIDS. The so-called infection which in reality is an outfection of acid, is the least offender of the trio. Add fear (false evidence appearing real) wrong eating, poor sanitation and wrong lifestyle choices and we have a formidable symptom complex that serves to justify all that professional syphilomaniacs HIV/AID somaniacs say and write about these dis-eases.
Remove systemic acidosis of the Interstitium fluids, drugging, FEAR, poor sanitation, wrong lifestyle choices, and vile eating, and there is little left. What is left can be easily thrown out of the body by Nature!
Scientific research is being carried on vigorously in an attempt to find the cause of disease. The conception of disease is that the cause is individual. Here is where investigators meet their Waterloo. All of the so-called diseases are increasing symptom complexes due to repeated crises of systemic acidosis of the Interstitium and then the blood plasma.

Diseases have no independent existence!

As soon as acidity is controlled, the symptoms disappear unless an organ has been forced by innumerable crises to degenerate.
Even organic change, when the organ is not destroyed, will come back by correcting the life and getting rid of the true cause–the crisis of acidosis!
All symptoms of all so-called diseases have one origin.
All diseases are ONE!
Unity in all things is Nature’s plan.
Polytheism is gone, and everything pertaining to it and coming out of it must go.
So you NOW have the truth! No more LIES!
There is only one sickness, one disease, and NOW only one treatment.
The one sickness and disease is the over-acidification of the Interstitium and then the blood and then finally the tissues, organs and glands due to an inverted way of living, eating, drinking, breathing, thinking and believing.
The one treatment is to alkalize and energize with Dr. Robert Young’s pH Miracle Lifestyle and Diet Plan.
We all need to go back to the Garden of Eden and start living and eating GREEN. (Genesis Chapter 1 verses 28 – 30)

I call this eating like “COWS” which stands for the following:

C = Chlorophyll from green plants
O = Oxygen – nascent oxygen or singlet oxygen
W = Water – alkaline water at a 9.5 pH
S = Salt – liquid mineral ionized salts
You can learn more about this program on our website or in my books, The pH Miracle, The pH Miracle for Diabetes, The pH Miracle for Weight Loss, The pH Miracle revised and updated, The pH Miracle for Cancer, Back to the House of Health I and II and Sick and Tired, which you can also purchase through Amazon.com or http://www.phmiracleproducts.com
The complete program is a 12 to 16 week program that includes the alkaline foods outlined in Chapter 11 of the pH Miracle revised and updated book.
You start off the program with a 14 to 21 day liquid feast.
You can eat as much and as often as you like as long as the food is green and pureed and/or juiced.
The soups such as the Broccoli Soup, Aspar/Zinc Soup, The Healing Soup and the Popeye Soup with lots of avocados are excellent to eat during the liquid feast.
You also need to begin taking the nutritional supplements while drinking at least 4 to 6 liters of Greens a day.
Start out gradually drinking 1 liter of Greens per day and then work up to 2, then 3, then 4, until you are drinking 6 liters a day.
When you take the nutritional supplements, take 5 drops 6 times a day of the liquid colloids under the tongue, (except the pH drops which are taken in purified water and NEVER taken under the tongue) away from meals, or taking 1 capsule 3 to 6 times a day of the encapsulated products with meals.
I would suggest taking 4 capsules every 4 waking hours of the bowel cleansing formula.
The bowel cleansing product helps to keep acids moving through normal elimination.
After you complete the 14 to 21 day liquid feast, you can then begin introducing some solid food but it still needs to be as green as possible. Remember, when you are green you are clean. I would suggest not only the vegetable soups, but steam fry vegetables and lots of salads.
Make sure you use only lemon or lime and good oils on your salads for the dressing.
NO VINEGAR, NO ENZYMES, NO FERMENTED FOODS and NO PROBIOTICS!
NO ACID FOODS or DRINKS!
Another tip is to include liberal amounts of flax, hemp, avocado and olive oil in or with your soups and salads. I suggest a minimum of 5 to 6 tablespoons of good oils every day. And you should be supplementing pomegranate seed oil everyday.
For information on pomegranate and avocado seed oil go to:
In conclusion, the medical world has been looking for a remedy to cure ALL disease, notwithstanding the obvious fact that nature needs NO REMEDY and NO CURE. She needs only an opportunity to exercise her own prerogative of self-healing.

Cures! There are NO CURES! And there will be NO CURES Found!

The subconscious builds health or disease according to OUR ORDER! And we do it with our thoughts, our words and our deeds.
If we send impulses of irritation, discontent, unhappiness, complaining, hate, envy, selfishness, greed, lust, and the biggest one of all pride, the subconscious builds us in the image of OUR ORDER!
The truth is that WE NEED NO DOCTOR and WE NEED NO NATIONAL HEALTH INSURANCE PLAN!
The movie “Sicko” suggests we need a health program for every man, woman and child in the US. This health program must start with the education on how to prevent disease with what we eat, drink, breathe, think and believe.
To insure more people in the US under the current health system does not bring more health. We do not need more medicine! We need more education on how to maintain the alkaline design of our body!
We need to empower ourselves to effect a reconciliation between our subconscious creator and ourselves.
What we need is to learn self-control, respect, poise, relaxation, deep breathing, alkaline eating and drinking, and especially alkaline exercising!
And when these impulses are sent over the sympathetic nerves to our subconscious creator, we will begin to receive images of a more ideal man or woman, until an approach to “Perfection is Attained”.
Sickness and disease, including the symptoms of cancer, heart attacks, strokes, diabetes, sepsis, tumors, HIV/AIDS, MS, lupus, depression, hyperthyroidism, Wilson’s Syndrome, fibromyalgia, pain in every joint and muscle, chronic fatigue syndrome, muscle cramps, allergies (food), asthma, bronchitis, frequent colds, candida, hypoglycemia, allergic reaction to any chemical, chronic fatiguing, food cravings, indigestion, inflamed joints, insomnia, mood swings, gas, bloating, diverticulitis, irritable bowel, pneumonia, ulcers, stomach and bowel cramps and even memory loss is the culmination of years of abuse of nutrition and years of acids from faulty elimination by forcing the bowels to move.

We don’t GET sick and tired we DO sick and tired!

The most powerful way to eliminate metabolic and dietary acids in the blood, the Interstitium fluids and finally the intracellular fluids is following the pH Miracle Lifestyle and Dietary Plan.
You are the builder of tomorrow, and you need not pay a fortuneteller, doctor, lawyer, preacher, or banker to tell you what will happen to you tomorrow.
Nothing will happen.
The inevitable will come.
You will inherit the fruits of today’s sowing.
I hope you find these thoughts and suggestions helpful when dealing with ANY symptomology, whether physical, emotional or spiritual.
Your life, Your Health, Your Fitness, Your Energy is the consequence of your daily choices and you are free to choose!
I pray that you choose wisely! The quality and the quantity of your life depends upon it.
In love and Gods healing light,
Robert O. Young DSc, Phd, Naturopathic Practitioner, Preventologist
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PS My hope is that you will share this articles with everyone that you know, love and care about.
In the last days Paul prophesied, “Men ever learning, and never able to come to the knowledge of the truth” 2 Timonthy 3:7
Paul’s prophesy has been fulfilled in our day!
Not part of our pH Miracle Living Community?
For other scientific articles of Dr. Robert O Young go to: http://www.drrobertyoung.com
For non-invasive blood and interstitial fluid of the Interstitium testing for the chemistry and functionality of all organs and organ systems call: 818-987-6886 or go to: http://www.universalmedicalimaging.com or email: universalmedicalimaging@yahoo.com
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Are YOU Following a LIVE-IT or a DIE-IT?

An Acidic Diet and Lifestyle Is More Deadly Than Smoking and the Cause of Heart Attacks, Diabetes and Cancer!
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A new landmark study published in the Lancet found that, poor or acidic diet is responsible for more than 1 in 5 deaths globally, making it more deadly than tobacco.
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Consuming both low amounts of healthy alkaline foods and high amounts of unhealthy acidic foods are key to these findings.
 
These unhealthy highly acidic foods include, beef, chicken, pork, duck, turkey, fish, dairy, eggs, vinegar, sugar, alcohol, vinegar, carbonated drinks, energy drinks, coffee, black tea, just to name a few.
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The healthy alkaline foods include, low sugar fruit like tomato, avocado, grapefruit, cucumber, peppers of all colors, lemon and lime and vegetables, such as broccoli, spinach, arugula, celery, cabbage, cauliflower, brussels sprouts, just to name a few.
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What you put on your plate can play a serious role in how likely you are to die before your time: According, to the study, a poor or acidic diet is actually the leading cause of death worldwide, contributing to more of them than conventional risk factors like tobacco use.
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In the study, researchers analyzed food consumption habits of adults ages 25 and older from 1990 to 2017 in 195 countries and compared how their diet affected their chances of premature death.
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They found that in 2017, 11 million deaths—or 22 percent—worldwide were caused by a poor acidic diet. More specifically? Of these deaths, 9.5 million were due to cardiovascular disease, over 900,000 to diet-related cancers, over 330,000 to diabetes, and over 136,000 to kidney diseases.
 
On the other hand, more commonly known risk factors like high blood pressure and tobacco use was linked to 10.4 million and 8 million deaths, respectively. Researchers also found that a poor acidic diet is linked to more years lived with disability, too.
 
According to Dr. Robert O Young at the pH Miracle Research Center, USA, “an acidic diet is an equal opportunity killer and the number 1 cause of ALL sickness and disease!”
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The following are the references to this very important 27 year Lancet published study validating the work of Dr. Robert O Young, who suggested in 1984 that there is only one sickness, one disease and one treatment.
 
This one sickness and one disease was described by Dr. Young as the over-acidification of the blood and interstitial fluids of the Interstitium due to an inverted way of living, eating, drinking, breathing, thinking and believing. He then suggested that there was only one treatment in order to restore the alkaline design of the body fluids by using an alkaline lifestyle and diet as described in his research, published papers and books, such as, A Nutritional Approach to the Prevention and Treatment of Any Cancerous Condition, The pH Miracle, The pH Miracle revised and update, The pH Miracle for Weight Loss, The pH Miracle For Diabetes, The pH Miracle for the Heart and The pH Miracle for Cancer.
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You can find Dr. Young’s published research and books at: https://www.amazon.com/Robert-O.-Young/e/B001ILKCSU?ref=dbs_p_ebk_r00_abau_000000 or on his website at: drrobertyoung dotcom
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References

 
1. Health effects of dietary risks in 195 countries, 1990–2017: a systematic analysis for the Global Burden of Disease Study 2017
GBD 2017 Diet Collaborators
Open AccessPublished:April 03, 2019DOI:https://doi.org/10.1016/S0140-6736(19)30041-8
 
2. Willett WC Stampfer MJ
Current evidence on healthy eating.
Annu Rev Public Health. 2013; 34: 77-95
 
3. Norat T Chan D Lau R Aune D Vieira R Corpet D
The associations between food, nutrition and physical activity and the risk of colorectal cancer.
Date: Oct, 2010
Date accessed: December 12, 2016
 
4. World Cancer Research Fund/American Institute for Cancer Research
Diet, nutrition, physical activity and cancer: a global perspective. Continuous Update Project Expert Report.
Date: 2018
Date accessed: February 19, 2019
 
5. Micha R Shulkin ML Peñalvo JL et al.
Etiologic effects and optimal intakes of foods and nutrients for risk of cardiovascular diseases and diabetes: systematic reviews and meta-analyses from the Nutrition and Chronic Diseases Expert Group (NutriCoDE).
PLoS One. 2017; 12: e0175149
 
6. Micha R Kalantarian S Wirojratana P et al.
Estimating the global and regional burden of suboptimal nutrition on chronic disease: methods and inputs to the analysis.
Eur J Clin Nutr. 2012; 66: 119-129
 
7. Colditz GA
Overview of the epidemiology methods and applications: strengths and limitations of observational study designs.
Crit Rev Food Sci Nutr. 2010; 50: 10-12
 
8. Nishida C Uauy R Kumanyika S Shetty P
The joint WHO/FAO expert consultation on diet, nutrition and the prevention of chronic diseases: process, product and policy implications.
Public Health Nutr. 2004; 7: 245-250
 
9. Lloyd-Jones DM Hong Y Labarthe D et al.
Defining and setting national goals for cardiovascular health promotion and disease reduction: the American Heart Association’s strategic Impact Goal through 2020 and beyond.
Circulation. 2010; 121: 586-613
 
10. McGuire S
U.S. Department of Agriculture and U.S. Department of Health and Human Services, Dietary Guidelines for Americans, 2010.
7th Edition. U.S. Government Printing Office, Washington, DC; January 2011Adv Nutr. 2011; 2: 293-294
 
11. Lim SS Vos T Flaxman AD et al.
A comparative risk assessment of burden of disease and injury attributable to 67 risk factors and risk factor clusters in 21 regions, 1990–2010: a systematic analysis for the Global Burden of Disease Study 2010.
Lancet. 2012; 380: 2224-2260
 
12. Forouzanfar MH Alexander L et al.GBD 2013 Risk Factors Collaborators
Global, regional, and national comparative risk assessment of 79 behavioural, environmental and occupational, and metabolic risks or clusters of risks in 188 countries, 1990–2013: a systematic analysis for the Global Burden of Disease Study 2013.
Lancet. 2015; 386: 2287-2323
 
13. GBD 2015 Risk Factors Collaborators
Global, regional, and national comparative risk assessment of 79 behavioural, environmental and occupational, and metabolic risks or clusters of risks, 1990–2015: a systematic analysis for the Global Burden of Disease Study 2015.
Lancet. 2016; 388: 1659-1724
 
14. GBD 2016 Risk Factors Collaborators
Global, regional, and national comparative risk assessment of 84 behavioural, environmental and occupational, and metabolic risks or clusters of risks, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016.
Lancet. 2017; 390: 1345-1422
 
15. Mozaffarian D Fahimi S Singh GM et al.
Global sodium consumption and death from cardiovascular causes.
N Engl J Med. 2014; 371: 624-634
 
16. Singh GM Micha R Khatibzadeh S et al.
Estimated global, regional, and national disease burdens related to sugar-sweetened beverage consumption in 2010.
Circulation. 2015; 132: 639-666
 
17. Singh GM Micha R Khatibzadeh S et al.
Global, regional, and national consumption of sugar-sweetened beverages, fruit juices, and milk: a systematic assessment of beverage intake in 187 countries.
PLoS One. 2015; 10: e0124845
 
18. Micha R Khatibzadeh S Shi P et al.
Global, regional and national consumption of major food groups in 1990 and 2010: a systematic analysis including 266 country-specific nutrition surveys worldwide.
BMJ Open. 2015; 5: e008705
 
19. Micha R Khatibzadeh S Shi P et al.
Global, regional, and national consumption levels of dietary fats and oils in 1990 and 2010: a systematic analysis including 266 country-specific nutrition surveys.
BMJ. 2014; 348: g2272
 
20. Wang Q Afshin A Yakoob MY et al.
Impact of nonoptimal intakes of saturated, polyunsaturated, and trans fat on global burdens of coronary heart disease.
J Am Heart Assoc. 2016; (published online Jan 20.)
DOI:10.1161/JAHA.115.002891
 
21. Schmidhuber J Sur P Fay K et al.
The Global Nutrient Database: availability of macronutrients and micronutrients in 195 countries from 1980 to 2013.
Lancet Planet Health. 2018; 2: e353-e368
 
22. Gakidou E Afshin A Abajobir AA et al.
Global, regional, and national comparative risk assessment of 84 behavioural, environmental and occupational, and metabolic risks or clusters of risks, 1990–2016: a systematic analysis for the Global Burden of Disease Study 2016.
Lancet. 2017; 390: 1345-1422
 
23. Singh GM Danaei G Farzadfar F et al.
The age-specific quantitative effects of metabolic risk factors on cardiovascular diseases and diabetes: a pooled analysis.
PLoS One. 2013; 8: e65174
 
24. Mente A O’Donnell M Rangarajan S et al.
Associations of urinary sodium excretion with cardiovascular events in individuals with and without hypertension: a pooled analysis of data from four studies.
Lancet. 2016; 388: 465-475
 
25. Trinquart L Johns DM Galea S
Why do we think we know what we know? A metaknowledge analysis of the salt controversy.
Int J Epidemiol. 2016; 45: 251-260
 
26. GBD 2016 Causes of Death Collaborators
Global, regional, and national age-sex specific mortality for 264 causes of death, 1980–2016: a systematic analysis for the Global Burden of Disease Study 2016.
Lancet. 2017; 390: 1151-1210
 
27. Djalalinia S Saeedi Moghaddam S Moradi-Lakeh M et al.
Prevalence and years lived with disability of 310 diseases and injuries in Iran and its neighboring countries, 1990–2015: findings from Global Burden of Disease Study 2015.
Arch Iran Med. 2017; 20: 392-402
 
28. WHO
Global action plan for the prevention and control of noncommunicable diseases: 2013–2020.
Date: 2013
Date accessed: December 12, 2016
 
29. WHO
2008–2013 action plan for the global strategy for the prevention and control of noncommunicable diseases: prevent and control cardiovascular diseases, cancers, chronic respiratory diseases and diabetes.
World Health Organization, Geneva; 2009
 
30. Afshin A Penalvo J Del Gobbo L et al.
CVD prevention through policy: a review of mass media, food/menu labeling, taxation/subsidies, built environment, school procurement, worksite wellness, and marketing standards to improve diet.
Curr Cardiol Rep. 2015; 17: 98
 
31. Mozaffarian D Afshin A Benowitz NL et al.
Population approaches to improve diet, physical activity, and smoking habits: a scientific statement from the American Heart Association.
Circulation. 2012; 126: 1514-1563
 
32. WHO
Interventions on diet and physical activity: what works. Summary report.
World Health Organization, Geneva; 2009
 
33. Cobiac LJ Veerman L Vos T
The role of cost-effectiveness analysis in developing nutrition policy.
Annu Rev Nutr. 2013; 33: 373-393
 
34. Bibbins-Domingo K Chertow GM Coxson PG et al.
Projected effect of dietary salt reductions on future cardiovascular disease.
N Engl J Med. 2010; 362: 590-599
 
35. Smith-Spangler CM Juusola JL Enns EA Owens DK Garber AM
Population strategies to decrease sodium intake and the burden of cardiovascular disease: a cost-effectiveness analysis.
Ann Intern Med. 2010; 152: 481-487
 
36. Owen L Morgan A Fischer A Ellis S Hoy A Kelly MP
The cost-effectiveness of public health interventions.
J Public Health. 2012; 34: 37-45
 
37. Lachat C Otchere S Roberfroid D et al.
Diet and physical activity for the prevention of noncommunicable diseases in low- and middle-income countries: a systematic policy review.
PLoS Med. 2013; 10: e1001465
 
38. Downs SM Thow AM Leeder SR
The effectiveness of policies for reducing dietary trans fat: a systematic review of the evidence.
Bull World Health Organ. 2013; 91 (69H): 262
 
39. Anand SS Hawkes C de Souza RJ et al.
Food consumption and its impact on cardiovascular disease: importance of solutions focused on the globalized food system: a report from the workshop convened by the World Heart Federation.
J Am Coll Cardiol. 2015; 66: 1590-1614
 
40. Brown GW Yamey G Wamala S The handbook of global health policy. Wiley, Hoboken; 2014
 
41. Tilman D Clark M
Global diets link environmental sustainability and human health.
Nature. 2014; 515: 518-522
 
42. Auestad N Fulgoni VL
What current literature tells us about sustainable diets: emerging research linking dietary patterns, environmental sustainability, and economics.
Adv Nutr. 2015; 6: 19-36
 
43. Heller MC Keoleian GA Willett WC
Toward a life cycle-based, diet-level framework for food environmental impact and nutritional quality assessment: a critical review.
Environ Sci Technol. 2013; 47: 12632-12647
 
44. Sabaté J Soret S
Sustainability of plant-based diets: back to the future.
Am J Clin Nutr. 2014; 100: 476S-482S
 
45. Food and Agriculture Organization of the United Nations
Food consumption database.
Date accessed: December 12, 2016
 
46. Hill RJ Davies PS
The validity of self-reported energy intake as determined using the doubly labelled water technique.
Br J Nutr. 2001; 85: 415-430
 
47. McLean RM
Measuring population sodium intake: a review of methods.
Nutrients. 2014; 6: 4651-4662
 
48. Illner A-K Freisling H Boeing H Huybrechts I Crispim SP Slimani N
Review and evaluation of innovative technologies for measuring diet in nutritional epidemiology.
Int J Epidemiol. 2012; 41: 1187-1203

Has the Existence of Polio, Measles, HIV, CMV, EBV, Hep C, Ebola, the Flu, and Now Zika Viruses Been Demonstrated and Scientifically Proven?

Dismantling The Viral Theory

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[Electron micrograph of the so-called Polio virus that has never been demonstrated scientifically to cause the symptoms of paralysis.  Illustration has been colorized for effect]

The first isolation of a virus was achieved in 1892 by Russian bacteria hunter Dimitri Iwanowski, who gathered fluid from diseased tobacco plants. He passed this liquid through a filter fine enough to retain bacteria; yet to Iwanowski’s surprise, the bacteria-free filtrate easily made healthy plants sick. In 1898 a Dutch botanist, Martinus Willem Beijerinck, repeating the experiment, also recognized that there was an invisible cause and named the infectious agent “tobacco mosaic virus.” In the same year as Beijerinck’s report, two German scientists purified a liquid containing filterable viruses that caused foot-and-mouth disease in cattle (viruses were at one time called “filterable viruses,” but eventually the term “filterable” came to apply only to viruses, and was dropped). Walter Reed followed in 1901 with a filtrate responsible for yellow fever, and soon dozens of other disease-causing viruses were found.

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In 1935 another American, Wendell M. Stanley, went back to the beginning and created pure crystals of tobacco mosaic virus from a filtered liquid solution. He affirmed that these crystals could easily infect plants, and concluded that a virus was not a living organism, since it could be crystallized like salt and yet remained infectious. Subsequently, bacteriologists all over the world began filtering for viruses, and a new area of biology was born-virology.

Historically, medical science has vacillated on the question of whether a virus is alive. Originally it was described as nonliving, but is currently said to be an extremely complex molecule or an extremely simple microorganism, and is usually referred to as a parasite having a cycle of life. (The term “killed” is applied to certain viral vaccines, thus implying an official conviction that viruses live.) Commonly composed of either DNA or RNA cores with protein coverings, and having no inherent reproductive ability, viruses depend upon the host for replication. They must utilize the nucleic acids of living cells they infect to reproduce their proteins (i.e., trick the host into producing them), which are then assembled into new viruses like cars on an assembly line. Theoretically, this is their only means of surviving and infecting new cells or hosts.

The Replicating Virus Theory

Then it was discovered that, when bacteria slowly begin to die, bacteria create tiny, apparently lifeless forms of survival, the so-called spores. It was then suspected that these spores were toxic and that they were the so-called pathogenic poisons. This was then refuted, since the spores are rapidly developing into bacteria when their vital resources are being restored. When scientists in the laboratory observed that the weak, highly inbred bacteria perished very quickly while turning into much smaller structures than the spores, it was first believed that the bacteria were being killed by the alleged pathogenic poisons, called viruses, and that the viruses were thereby replicating.

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[The micrograph above was done using Dark Field Microscopy showing red blood cells and the evolution of bacterial pHages and bacterial spores (the white spots0 from red blood cell biological transformation]

The Replicating Virus Theory

Then it was discovered that, when bacteria slowly begin to die, bacteria create tiny, apparently lifeless forms of survival, the so-called spores. It was then suspected that these spores were toxic and that they were the so-called pathogenic poisons. This was then refuted, since the spores are rapidly developing into bacteria when their vital resources are being restored. When scientists in the laboratory observed that the weak, highly inbred bacteria perished very quickly while turning into much smaller structures than the spores, it was first believed that the bacteria were being killed by the alleged pathogenic poisons, called viruses, and that the viruses were thereby replicating.

The Invention of Bacterial Viruses

Due to the belief that these – at the time of their discovery still invisible- structures were killing the bacteria, they were called phages/bacteriophages, “eaters of bacteria”. Only later it was determined that merely highly inbred and therefore almost non-viable bacteria can be made to turn into phages, or bacteria which are being destroyed so fast that they do not have time to form spores.

The introduction of the electron microscopy led to the discovery of the structures resulting from the biological transformation or pleomorphism of bacteria when these were suddenly dying or when the metabolism of the highly inbred germs was overwhelmed by processes triggered by the adding of “phages”. It was also discovered that there are hundreds of types of different-looking “phages”. The discovery of phages, the so-called bacterial “viruses”, reinforced the wrong assumption and the belief that there were human and animal viruses that looked the same and had the same structure. This is not and cannot be the case, for several different reasons.

After introducing chemical examination techniques in biology, it was discovered that there are thousands of types of phages and that phages of one type always have the same structure. They consist of a particular molecule, made of nucleic acid, which is covered in a shell of proteins of a given number and composition. It was only later discovered that merely the bacteria which had been highly inbred in the test tube could turn into phages themselves, by contact with phages, but this never applied to natural bacteria or bacteria which had just been isolated from their natural environment. In this process, it was discovered that these “bacterial viruses” actually serve to provide other bacteria with important molecules and proteins, and that the bacteria themselves emerged from such structures.

Before it could be established that the “bacterial viruses” cannot kill natural bacteria, but they are instead helping them to live and that bacteria themselves emerge from such structures, these “phages” were already used as models for the alleged human and animal viruses. It was assumed that the human and animal viruses looked like the “phages”, were allegedly killing cells and thereby causing diseases, while at the same time producing new disease poisons and in this way transmitting the diseases. To date, many new or apparently new diseases have been attributed to viruses if their origin is unknown or not acknowledged. This reflex found an apparent confirmation in the discovery of the “bacterial viruses”.

It is important to note that the theories of fight and infection were accepted and highly praised by a majority of the specialists only if and when the countries or regions where they lived were also suffering from war and adversity. In times of peace, other concepts dominated the world of science.[272]

It is very important to note that the theory of infection – starting from Germany – has only been globalized through the third Reich, when the Jewish researchers, most of which had opposed and refuted the politically exploited theories of infection, were removed from their positions.[273]

The Detection of Phages and Biological Transformation

The existence of phages can be proved rapidly

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[Bacterial pHage being born out of a blood and/or body cell.  A biological process known as pleomorphism]

First step: their presence is confirmed through an effect, namely the transformation of bacteria into phages, and also through an electron micrograph of those phages. The control experiments show that phages do not appear if bacteria do not change or if bacteria randomly start decomposing due to extrinsic sudden annihilation, without forming phages.

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[The micrographs (micrographs #1 through #6] above show the cellular transformation of red blood cells, using pHase contrast microscopy, into rod bacteria, cell-wall deficient bacteria, Y-form yeast and then bacteria pHages]

Second step: the liquid containing the phages is concentrated and applied on another liquid, which has a high concentration at the bottom of the test tube and a low concentration at the top of the test tube. The test tube with the phages is then powerfully spun (centrifuged) and all the particles gather according to their mass and weight to the place of their own density. The density is the ratio of weight (mass) per unit of volume, expressed as Kg/l or g/mg, respectively. That is why this concentration and purification step for particles with the same density is called density gradient centrifugation.

The layer where many particles of the same density gather becomes “cloudy”, which is called a “band.” This step is being documented, then the particles concentrated, purified and sedimented in a “band” are removed with a syringe needle. The extracted concentrated amount of particles is called an isolate. A fast and simple electron micrograph will confirm the presence of phages in the isolate, which at the same time is an indication for the purity of the isolate, if the micrograph shows no other particles but the phages. The appearance and the diameter of the phages will also be established with the help of this micrograph.

The control experiment performed for this step consists in treating and centrifuging the liquid from bacteria which did not form any phages, where no phages appear at the end of the procedure.

After the step of successfully isolating the phages, the decisive biochemical characterization of the phages follows. The biochemical characterization of their composition is essential for identifying the specific type of phage, since different types of phages often appear to be similar. The isolate obtained through the density gradient centrifugation is now divided in two parts. One part is used to determine the size, type and composition of the nucleic acid; in a separate procedure, the other part is used to determine the amount, size and morphology of the proteins of the phages. Since the 1970s, these tests have been simple standard techniques that are learned by every biology student in their first semesters.

These tests represent the biochemical characterization of the phages. In almost every case, these results have been and are being published in only one publication, since a phage has a very simple structure which is very easy to analyze. The control experiments for these tests use liquid from bacteria which do not form phages and thus cannot present any biochemical proof. The existence of approximately two thousand different types of phages have been scientifically demonstrated this way

The So-Called Pathogenic Viruses

The “bacteriophages,” correctly defined as incomplete mini spores and building blocks of the bacteria, have been scientifically isolated, while the so-called pathogenic viruses have never been observed in humans or animals or in their body fluids and have never been isolated and subsequently biochemically analyzed. To date, none of the researchers involved in virology research seems to have realized this very important point.

The use of electron microscopy and the biochemistry were very slowly returning to normal after 1945 and no one had realized that not one pathogenic virus had ever been isolated in humans or animals; thus, as of 1949 researchers started applying the same idea used for the (bacterio) phages, in order to replicate the human and animal “viruses.” John Franklin Enders, born in 1897 in the family of a rich financier, was active in various fraternities after having finished his studies, then he worked as a real estate agent and studied foreign languages for four years before turning to bacterial virology, which fascinated him. He then simply transferred the ideas and concepts that he learned in this area of research to the supposed pathogenic viruses in humans.

UnScientific Experiments and Interpretations Gave Birth to Virology

With his unscientific experiments and interpretations that he had never confirmed through negative controls, Enders brought the entire “viral” infectious medicine to a dead end. It is important to note at this point that Enders, like many infectious diseases specialists, worked for the U.S. military, which had always been and remains to date a huge victim of the fear of contagions. It was mainly the U.S. military which spread its erroneous belief that besides chemical weapons there were also biological weapons in the form of bacteria and viruses.

In 1949, Enders announced that he had managed to cultivate and grow the alleged polio virus in vitro on various tissues. The American expert opinion believed everything immediately. What Enders did was to add fluids from patients with poliomyelitis to tissue cultures which he claimed to have had sterilized, then he alleged that the cells were dying because of the virus, that the virus was replicating in this way and that a vaccine could be harvested from the respective culture. At that time, summer polio epidemics (polio = flaccid paralysis) were very frequent during summer and they were believed to be caused by the polio virus. A vaccine was to help eradicate the alleged virus. After the polio vaccine was introduced, the symptoms were then re-diagnosed among other things as multiple sclerosis, flaccid acute paralysis, aseptic meningitis etc. and later polio was claimed to have been eradicated. During his experiments, Enders et al. sterilized the tissue cultures in order to exclude the possibility of bacteria killing the cells. What he didn’t take into consideration was that the sterilization and the treatment of the cell culture when preparing it for the alleged infection was exactly what was destroying and killing the cells. Instead, he interpreted the cytopathic effects as the existence and the action of a so-called polio virus, without ever having isolated a single virus and describing its biochemistry. The necessary negative control experiments, which would have shown that the sterilization and the treatment of the cells prior to the “infection” in the test tube was killing the cells, have never been performed. However, for this “performance” Enders received the Nobel prize in 1954.

The Invention of the Polio Virus and ‘YES” the Measles Virus Too!

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[Measles virus or a bacterial pHage?]

1954 is also the year in which Enders applied and introduced the same technique in order to allegedly replicate the measles virus. As he had been awarded the Nobel prize for the alleged polio virus the same year, all researchers believed his technique to be scientifically valid. Thus, to date, the entire concept of polio and measles has been based upon this unscientific technique and fraud.

Thus, the polio and measles vaccines do not contain viruses, but particles of dead monkey kidney tissue or human cancerous body cells. To date, no negative control experiments have been done with respect to the so-called polio and measles viruses either, which would have shown that it was the laboratory procedures that lead to the cytopathic effects on the cells.

Additionally, all claims and experiments made by Enders et al. and subsequent researchers lead to the only objective conclusion, that in fact they were observing and analyzing the cellular particles or fragments and the activity thereof in the test tube, misinterpreting these as particles and characteristics of the alleged polio and/or measles viruses.

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ALL Viruses from HIV, EBV, CMV, Hepatitis C, West Nile Virus, Ebola, Measles, Zika, etc., are ALL Phantom Viruses

Viral Existence Has NEVER Been Scientifically Demonstrated and Never Proven!

The following explanations applies to all the so-called (human or animal) “pathogenic viruses”. The six papers provided by Dr. Bardens in the course of the “measles trial” as proof for the existence of the measles virus described in a didactically ideal way the various steps of the chain of misinterpretations up to the belief in the existence of a measles virus.

The first paper was published in 1954 by Enders et al.: “Propagation in tissue cultures of cytopathogenic agents from patients with measles” (Proc Soc Exp Biol Med. 1954 Jun; 86 (2): 277–286).

This publication can be found on the internet, like all the other publications presented at the measles trial. In that experiment, Enders et al. cut down dramatically on the nutrient solution and added cell-destroying antibiotics to the cell culture before introducing the allegedly infected fluid. The subsequent dying of the cells was then misinterpreted as presence and also isolation of the measles virus. No control experiments were performed to exclude the possibility that it was the deprivation of nutrients as well as the antibiotics which led to the cytopathic effects.

Enders’ and his colleagues’ blindness can be explained by the fact that he truly wanted to help people, while the ‘virus hysteria’ was intensifying after the war and during the cold war. It can also be explained by the fact that Enders and many of his colleagues had no idea about medicine or biochemistry and they were competing with the Soviet Union for the development of the first measles vaccine. Such a pressure for success can also explain why Enders and his colleagues ignored their own reservations and cautions expressed in 1954, when they had observed and noted that many cells also died after being treated normally (i.e. without being “infected”), which they thought to have been caused by unknown viruses and other factors.  All these facts and cautions were subsequently disregarded.

The second paper presented by the claimant in the ‘measles trial’ was published in 1959[274] and, for the reasons presented above, the authors concluded that the technique introduced by Enders was not appropriate for the isolation of ANY virus. This rebuttal is not only NOT being discussed by ALL the other researchers, but it is being ignored completely!

The ‘Viral Dogma’ of Pathogenic Viruses is Still Being Promoted Today!

In a third paper[275], the authors photographed typical cellular particles inside the cells and misinterpreted these as measles virus. They did not isolate any virus. For unexplained reasons, they failed to determine and describe the biochemical structure of what they were presenting as a virus in a separate experiment. In the short description of the methods used, one can read that the authors did not apply the standard isolation technique for viruses, i.e. the density gradient centrifugation. They simply centrifuged fragments of dead cells at the bottom of a test tube and then, without describing their biochemical structure, they misinterpreted the cellular debris as viruses.

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From the way the experiments were performed, one can only conclude that cellular particles were misinterpreted as viruses. We find the same situation in the fourth[276] and the sixth[277] publication put forward by the claimant as proof of the existence of a measles virus. The fifth publication[278] is a review describing the consensus process as to which nucleic acid molecules from the dead cells would represent the so-called genome of the polio or measles virus. The result is that dozens of research teams work with short pieces of cell-specific molecules, after which -following a given model – they put all the pieces together on paper. However, this jigsaw puzzle made of so many pieces was never scientifically proven to exist as a whole and was never isolated from a virus, for a polio, measles, HIV or Hepatitis C, Ebola or Zika viruses have never been seen, neither in humans nor in a test tube. Referring to this publication, the court-appointed expert stated that it described the gold standard, i.e. the entire virus genome. It is obvious that the expert did not read this paper, whose authors stated that the exact molecular composition and functions of the measles virus genome will have to be the object of further research, which is why they had to rely on other virus models in order to achieve a consensus on the structure and functions of ANY virus genome. The easiest thing for anyone to notice is that in all of these publications, as well as in all other publications on the “measles virus” and other pathogenic viruses, including HIV, EBV, CMV, Ebola and Zika, no control experiments have ever been performed. No researchers used the density gradient centrifugation technique; instead, they only centrifuged cellular debris at the bottom of a test tube. This technique, used to collect all the particles from a fluid, is called pelletizing. From a logical and scientific perspective, it can be said that in all publications on the so-called “pathogenic viruses”, the researchers demonstrated in fact only particles and characteristics of cells. I would also like to point out that the so-called giant viruses[279] , i.e. an enwrapped nucleic acid can be found everywhere in the sea and in basic organisms. Like all bacterial phages, not only are they harmless, but they have beneficial functions. They can be also isolated by using the density gradient centrifugation, which proves their existence (see the graphic above).

I also recommend Prof. LĂŒdtke’s relevant review (1999).[280] He noted that at the early beginnings of virology, the majority of virologists always concluded that the structures they had mistaken for viruses turned out to be components of the cells and thus, they were only the result of the experiment and not the cause of the changes observed.

After the discovery and characterization of the phages and after introducing the dogma that the nucleic acid was the genome of all cells and viruses, the consensus was born, according to which such viruses must exist in humans and animals as well. In 1992, the dogma stating that the nucleic acid is the genotype of all cells was retracted in the scientific community. The ‘viral dogma’ of pathogenic viruses, however, is still being promoted today to the harm of billions of people. – for what?

The Bottom Line Concerning Phantom Viruses and the Polio and Measles Virus

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[An Electron micrograph of the so-called Polio virus that has never been demonstrated scientifically to cause the symptoms of paralysis.  Illustration has been colorized for effect]

My bottom line still holds the truth that the terrain or internal environment is everything and the germ or so-called virus is NOTHING! The germ or so-called virus can only be a symptom of cellular breakdown due to an imbalance of the delicate alkaline pH balance of the body fluids and NOT the cause of that breakdown. That is why years ago I offered any scientist in the World a finders fee of 5 million US dollars if they could prove the existence of the HIV virus using Koch’s postulates. It has now been over 20 years and I am still waiting even though currently I no longer have the funds to pay the prize due to political assassination! It is unfortunate that a former 5 million US dollar prize offered 20 years ago was not enough money to change the current medical viral dogma that is currently paying out trillions of dollars to guess who?[281]

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Click here to read more: http://medcraveonline.com/IJVV/IJVV-02-00032.php

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​​Second Thoughts About Cover copy

Lecture in Dubai – The 2nd Annual Conference on Bacterial, Viral and Infectious Diseases

http://www.drrobertyoung.com/events.html

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Join Robert O Young PhD and Galina Migalko MD in Dubai on June 18th and 19th, 2019 for the Annual Conference on Bacterial, Viral and Infectious Diseases. They will be Key Note Speakers and doing a workshop on the New Biology.

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For more information and to register go to: https://bacterialdiseases.infectiousconferences.com/organiz


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The following is the abstract for Dr. Young’s lecture:

The Dismantling of the Viral Theory

Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner

Abstract

There is now over 100 years of documented history and research on the Polio virus and whether or not its treatment by inoculation has been successful in eradicating Polio. I am suggesting in this article and in my lecture that there are significant findings based on historical and past and current research, including my own that the viral theory of Polio and possibly other modern-day diseases, such as Post-Polio Syndrome, Polio Vaccine-Induced Paralysis, Legionnaires, CNS disease, Cancer, HIV/AIDS and now Zika may be caused by acidic chemical poisoning from DDT (dichloro-diphenyl-trichloroethane) and other related DDT pesticides, acidic vaccinations, and other factors including lifestyle and dietary factors rather than from a lone infectious virus. I will present ten historical graphs outlining the history of Polio, the production of DDT, BHC, Lead, Arsenic, Polio vaccinations and the author’s theory that chemical poisoning, vaccination, and lifestyle and dietary choices are a more likely causes for the symptoms of Polio, neurological diseases, Cancer, HIV/AIDS and now Zika.

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References:

[1] Morton S. Biskind, MD. “Public Health Aspects of the New Insecticides”. American Journal of Digestive Diseases, New York, 1953, v 20, p331.

[2] Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991, p769

[3] Toxicological Profile: for DDT, DDE, and DDE. Agency for Toxic Substances and Disease Registry, September 2002.

[4] U. Beck, E. Löser “Chlorinated Benzenes and other Nucleus-Chlorinated Aromatic Hydrocarbons” Ullmann’s Encyclopedia of Industrial Chemistry, 2012, Wiley-VCH, Weinheim.

[5] Chlorobenzene”. Immediately Dangerous to Life and Health. National Institute for Occupational Safety and Health (NIOSH)

[6] U.S. Vital Statistics, U.S. Government Printing Office, Washington, D.C.

[7] Historical Statistics of the U.S., The U.S. Government Printing Office, Washington, D.C.

[8] Van Nostrand’s Encyclopedia of Science and Engineering (1995), vol. 5, p1725. The phrase “Pesticides As A Panacea: 1942-1962” is a subtitle found in Encyclopedia Britannica, Macropaedia (1986).

[9] Thomas, Robert E. (1955), Salt & Water, Power & People: A Short History of Hooker Electrochemical Co. Niagara Falls, NY: Hooker Chemical Co.

[10] Booth, Gerald (2000), “Ullmann’s Encyclopedia of Industrial Chemistry – Nitro Compounds, Aromatic”. doi:10.1002/14356007.a17_411. ISBN 3527306730

[11] Weber, Manfred; Weber, Markus; Kleine-Boymann, Michael (2004). “Ullmann’s Encyclopedia of Industrial Chemistry – Phenol”. doi:10.1002/14356007.a19_299.pub2. ISBN 3527306730.

[12] Haller, H. L., Bartlett, P. D., Drake, N. L., and others: The Chemical Composition of Technical DDT, American Chemical Society, Journal, volume 67, pages 1591- 1602, 1945.

[13] Jo-Yu Chin, Christopher Godwin, Chunrong Jia, Thomas Robins, Toby Lewis, Edith Parker, Paul Max, and Stuart Batterman, “Concentrations and Risks of p-Dichlorobenzene in Indoor and Outdoor Air,” Indoor Air, 2013 Feb; 23(1): 40–49, Published online 2012 Jul 18. doi: 10.1111/j.1600-0668.2012.00796.x.

[14] Duesberg, PH, “Inventing the AIDS Virus,” Regnery, (1996). ISBN 0-89526-399-8. [15] Icon Group International (Author), Chlorobenzene: Webster’s Timeline History, 1851 – 2007 May 17, 2010

[16] Ibid [17] Ibid

[18] Risse, GB (1988). Fee E, Fox DM, eds. Epidemics and History: Ecological Perspectives. in AIDS: The Burden of History. University of California Press, Berkeley. ISBN 0-520-06396-1.

[19] A Disease of Cleanliness: Polio in New York City, 1900-1990, in David Rosner, ed., Hives of Sickness: Public Health and Epidemics in New York City Rutgers University Press, 1995, pp. 115-130.

[20] McDonough, F., The Origins of the First and Second World Wars (Cambridge Perspectives in History), Cambridge University Press, August 28, 1997.

[21] Goel, A, Aggarwal, P, “Pesticide Poisoning,” Natl Med J India. 2007 Jul-Aug; 20(4):182-91.

[22] Ibid.

[23] Biskind, MS (1953) “Public Health Aspects of the New Insecticides,” American Journal of Digestive Diseases 20: 331-341.

[24] TIME Magazine, U.S. Edition, March 14, 1994 Vol. 143 No. 11. [25] Baily, J. W.: J. Am. Vet. M. A. 113: 251, Sept. 1948.

[26] Biden-Steele, K. and Stuckey, R. E.: “Poisoning by DDT Emulsion: Report of a Fatal Case”, Lancet, 2: 235-236, Aug. 17, 1946.

[27] Biskind, M. S.: “DDT Poisoning and X Disease in Cattle”, J. Am. Vet. M. A. 114: 20, Jan. 1949.

[28] Biskind, M. S.: “DDT Poisoning a Serious Public Health Hazard”, Am. J. Dig. Dis. 16: 73, Feb. 1949.

[29] Biskind, M. S.: “DDT Poisoning and the Elusive ‘Virus X’: A New Cause for Gastro- Enteritis”, Am. J. Dig. Dis. 16: 79, March 1949.

[30] Boyd, C. L.: “A Report on “XX Disease in Texas”, J. Am. Vet. M. A. 113: 463, Nov. 1948.

[31] Cameron, C. R., and Burgess, F.: “The Toxicity of DDT”, Brit. M. J. 1: 865-871, June 23, 1945.

[32] Carte; R. H., Hubanks, P. E., et al: “Effect of Cooking on the DDT Content of Beef”, Science, 107: 347, April 2, 1948.

[33] Case, R. A. M.: Toxic Effects of DDT in Man”, Brit. M. J., 2: 842-845, Dec. 15, 1945.

[34] Council on Pharmacy and Chemistry, A. M. A.: “Health Hazards of Pesticides”, J. A. M. A. 137: 1603, Aug. 28, 1948.

[35] Crescitelli, F., and Gillman, A.: “Electrical Manifestations of Cerebellum and Cerebral Cortex Following DDT Administration to Cats and Monkeys”, Am. J. Physiol., 147: 127- 137, Sept. 1946.

[36] Deederer, C.: “DDT Toxicity”, M.Rec. 161: 216-220, April 1948

[37] Domenici, T. J.: “Hepatitis without Jaundice and without Hepatomegaly”, N. Eng. J. Med. 240: 88, Jan. 20, 1949

[38] Dunn, J. E., Dunn, J. C., and Smith, R. S.: “Skin Sensitising Properties of DDT for 31

Guinea Pig”, Pub. Health Rep. 61: 1614-1620, 1949.

[39] Editorial: Pesticides: “Chemical Contaminants of Foods”, J.A.M.A. 137: 1604, Aug. 28, 1948.

[40] Fitzhugh, O. G., and Nelson, A. A.: “The Chronic Oral Toxicity of DDT”, J. Pharm.acol. and Exper. Therap. 89: 18-30, Jan. 1947.

[41] Gamier, G.: “Treatment of Scabies with DDT”, .Presse Med. 56: 458, June 23, 1948. [42] Garett, ii. M., “Toxicity of DDT for Man”, Alabama St. M. A. J., 17: 74, Aug. 1947.

[43] Globus, J. H.: “DDT Poisoning; Histopathologic Observations on the Central Nervous System in So-Treated Monkeys, Dogs, Cats and Rats”, J. Neuropath. 7: 418-431, Oct. 1948.

[44] Haymaker, W., Ginzler, A. M., and Ferguson, J. L.: “Toxic Effects of Prolonged Ingestion of DDT on Dogs, with Special Reference to Lesions in Brain”, Am. J. M. Sc. 212: 423, Oct. 1946.

[45] Hill, K. R., and Daniiani, C. R.: “Death Following Exposure to DDT, Report of a Case”, New Eng. J. Med., 235: 897-899, Dec. 19, 1946.

[46] Hill, K. 3. and Robinson, G.: “A Fatal Case of DDT Poisoning in a Child, with an Account of Two Accidental Deaths in Dogs”. Brit. M. J. 2: 845-847, Dee. 15, 1945.

[47] Ingle, L.: “Toxicity of Chlordane to White Rats”, J. Econ. Entomol. 40: 264-268, 1947.

[48] Jandorf, B. J;. Sanett, H. P., and Bodansky, Oscar: “Effect of Oral Administration of DDT on Metabolism of Glucose and Pyruvie Acid in Rat Tissues”, J. Pharmaeol. and Exper. Therap. 88: 333-337, Dec. 1946.

[49] Jenkins, D. W.: “A Review of the Insecticide Hexachloro-cyclohexane (‘666’)”, Office of Technical Services, U. S. Dept of Commerce, Washington, D ‱ C., No. PB 4034, Med. Div. Rept. No. 56, Sept. 26, 1945.

[50] Kempe, H. E.: “Progress Report on Benzene Hexachloride for the Destruction of Sheep Scab Mites”, Vet. Med., Feb. 1948, pp. 76-79.

[51] Kirk, H.: Vet. Red. 58: 43, 1946.

[52] Kirk, H.: “DDT in Canine Practice”, Vet. Med. Feb. 1947, PP. 76-78.

[53] Lawhon, G. J., Jr.: “X Disease in South Carolina”, N. Am. Vet. 29: 643, Oct. 1948.

[54] Leider, M.: “Allergenic Eczematous Contact-Type Dermatitis Caused by DDT”, J. Invest. Dermatol. 8: 125-126., March 1947.

[55] Lillie, R. D., Smith, M. I., and Stohlman, E. F.: Pathologic Action of DDT and Certain of its Analogs and Derivatives”, Arch. Path. 43: 127-142, Feb. 1947.

[56] Mackerras, I. M., and West, R. F. K.: “DDT Poisoning in Man”, M. J. Australia, 1: 400-401, March 23, 1946.

[57] Mobbs, J. F.:” Toxicity of Hexaehloroeyclohexane in Scabies, J.A.M.A. 138: 1253, Dec. 25, 1948. Personal Communication.

[58] Morrill, C. C.: “Hyperkeratosi.s or X Disease”, N. Am. Vet. 29: 642, Oct. 1948.

[59] Neal, P. A., Sweeney, T. B., Spicer, S. S., and von Oettingen, W. F.: “The Excretion of DDT in Man, Together with Clinical Observations”, Pub. Health Rep., 61: 403, March 22, 1946.

[60] Neal, P. A., von Oettingen, W. F., Smith, W. W., et al: Toxicology and Potential Dangers of Aerosols, Mists and Dusting Powders Containing DDT”, Pub. Health Rep. Suppl. 177, 1944.

[61] Neal, P. A., von Oettingeu, W. F., Dunn, R. C., and Sharpless, N. E.: “Toxicology and Potential Dangers of Aerosols and Residues from Aerosols Containing 3 Percent of DDT. Second Report, ibid., Suppl. 183, 1945.

[62] Nelson, A. A., Draize, 3. H., Woodard, G., et al: “Histopathological Changes Following Administration of DDT to Several Species of Animals”, U. S. Pub. Health Rep. 59: 1009, Aug. 4, 1944.

[63] Neve, Helen: “Toxic Effects of DDT on a Cat”, Vet. Rec. 58: 43, 1946. Vet. Med., Feb. 1947, p. 78.

[64] Niedelman, M. L.: “Contact Dermatitis Due to DDT”, Occup. Med. 1: 391-395, April 1946.

[65] Radeleff, R. D.: “DDT Spray Outmodes Dipping Vat”, Vet. Med. Oct. 1947, pp. 372- 373.

[66] Radeleff, R. D.: “Chlordane Poisoning: Symptomatology and Pathology, Vet. Med. Aug. 1948, pp. 342-347.

[67] Robinson, J. H.: “Harvest Analysis of DDT Residues”, Food Packer, 29: 50-53, 1948.

[68] Riker, W. F., Jr., Huebner, Virginia, R., Raska, S. B., and Cattell, McKeen: “Studies on DDT, Effects on Oxidative Metabolism”, J. Pharmacol. and, Exper. Therap., 88: 327- 332, Dec. 1946.

[69] Sarrett, H. P., and Jandorf, B. J.: “Effects of Chronic DDT Intoxication in Rats on Lipids and Other Constituents of Liver”, ibid., 91: 340-344, Dec. 1947.

[70] Smith, M. I.: “Accidental Ingestion of DDT, with a Note on its Metabolism in Man”, J.A.M.A., 131: 519-520, Juno 8, 1946.

[71] Smith, M. I., and Stohlnian, E. F.: “Pharmacologic Action of 2, 2 his (p-Chlorophenyl) 1,1,1-Trichloroethane and its Estimation in the Tissues and Body Fluid”, Pub. Health Rep., 59: 984, July 28, 1944.

[72] SmIth, M. I., and Stohlman, E. F.: “Further Studies on the Pharmacologic Action of DDT”, ibid., 60: 289, March 16, 1945.

[73] Smith, N. 3.: “Death Following Accidental Ingestion of DDT”, J.A.M.A., 136: 469- 471, Feb. 14, 1948.

[74] Smith, R. F., Fullmes, O. H., and Messenger, P. S.: “DDT Residues on Alfalfa Hay and Seed Chaff”, J. Econ. Entomol. 41: 755-8, 1948.

[75] Strycker, G. V., and Godfroy, B.: “Dermatitis Resulting from Exposure to DDT”, J. Missouri St. M. A., 43: 384-386, June 1948.

[76] Taylor, E. L.: “Danger of Ununction with DDT”, Lancet, 2: 320, Sept. 8, 1945.

[77] Telford, H. S., and Guthrie, J. E.: “Transmission of the Toxicity of DDT Through the Milk of White Rats and Goats”, Science, 102: 647, Dec. 21, 1945.

[78] Thoungh, TI. C.: “Poisonous Effects of DDT on Humans”, Indian M. Ga:. 81: 432, Oct. 1946.

[79] U. S. Dept. Agriculture, “Bureau of Entomology and Plant Quarantine: Now Insecticides in Grasshopper Control”, Bull. E-722, May 1947. Bull. EC.1, March 1948.

[80] U. S. Dept. Agriculture, Bureau of Entomology and Plant Quarantine: “New Insecticides for Controlling External Parasites of Livestock”, Bull. E. 762, Dec. 1948.

[81] Westerfteld, C.: “The Use of DDT in Medicine-A Review”, Vet. Med., Oct. 1946, pp. 355-360.

[82] Wigglesworth, V. D.: “A Case of DDT Poisoning in Man”, Brit M. J. 1: 517, April 14, 1945.

[83] Wilson, J. B.: Are Pesticides Making Your Food Unsafer? Hygiea, Jan. 1949. p. 44.

[84] Woodard, G., Ofner, Ruth B., and Montgomery, C. M.: “Accumulation of DDT in the Body Fat and its Appearance in the Milk of Dogs”, Science, 102: 177-178, Aug. 17, 1945.

[85] Wright, C. S., Doan, C. A., and Haynie, H. C.: “Agranulocytosis Occurring after Exposure to DDT Pyrethrum Aerosol Bomb”, Am. J. Med., 1: 562-567, Nov. 1946.

[86] The Pesticide Residues Amendment of 1954, Pub. L. No. 83-518, ch. 559, 68 Stat. 511 [codified at 21 USC § 346a (1981)]; and the Food Additives Amendments of 1958, Pub. L. No. 85-529, Ch. 4.72 Stat. 1785 [codified at 21 USC § 348 (1981)], respectively.

[87] 20 Fed. Reg. 750 (1955) [codified until repealed at 21 CFR § 120. 1(f) (1956). [88] DDT Regulatory History: A Brief Survey (to 1975). United States Environmental

Protection Agency (EPA).

[89] Ibid.

[90] TIME Magazine, U.S. Edition, March 14, 1994 Vol. 143 No. 11.

(91] Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991.

[92] Peter Duesberg and Brian J. Ellison, Inventing the AIDS Virus, Regnery Pub.,1996. [93] Ibid.

[94] Biskind, MS (1953) “Public Health Aspects of the New Insecticides,” American Journal of Digestive Diseases 20: 331-341.

[95] Peter Duesberg and Brian J. Ellison, Inventing the AIDS Virus, Regnery Pub.,1996. [96] DDT Regulatory History: A Brief Survey (to 1975). United States Environmental

Protection Agency (EPA).

[97] Poliomyelitis: Fact sheet N°114″. World Health Organization. Sep 2016. Retrieved 14 Sep 2016.

[98] Ibid.

[99] DDT Regulatory History: A Brief Survey (to 1975). United States Environmental

Protection Agency (EPA).

[100] Ibid.

[101] Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R.

Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991.

[102] Rea WJ, Johnson AR, Fenyves E, Butler J. Related Articles: The environmental aspects of the post-polio syndrome. Birth Defects Orig Artic Ser. 1987;23(4):173-81. No abstract available. Pub Med ID: 3620615; UI: 87299998.

[103] Ibid.

[104] Casarett and Doull’s Toxicology (1996).

[105) Rea WJ, Johnson AR, Fenyves E, Butler J. Related Articles: The environmental aspects of the post-polio syndrome. Birth Defects Orig Artic Ser. 1987;23(4):173-81. No abstract available. Pub Med ID: 3620615; UI: 87299998.

[106] PubMed ID: 7611631, UI: 95336052 (London, May, 1995)

[107] Pub Med ID: 7611630, UI: 95336051 (Bethesda, MA, May, 1995)

[108] Pub Med ID: 8818905, UI: 96415998 (Lyon, France, Aug., 1996)

[109] Alfredo Morabia (1 January 2004). A History of Epidemiologic Methods and Concepts. Springer. pp. 133–4. ISBN 978-3-7643-6818-0. Retrieved 22 June 2013.

[110] Ibid.

[111] Morton S. Biskind, MD. “Public Health Aspects of the New Insecticides”. American

Journal of Digestive Diseases, New York, 1953, v 20, p331. [112] Ibid.

[113] Young RO (2016) Second Thoughts Concerning Viruses, Vaccines and the HIV/AIDS Hypothesis – Part 2. Int J Vaccines Vaccin 2(3): 00034. DOI: 10.15406/ijvv.2016.02.00034

[114] Dirt and Disease: Polio before FDR Rutgers University Press, 1992. [115] Ibid.

[116] Menkes, John H., Child Neurology, pg. 420, (1995).

[117] A Paralyzing Fear: The Story of Polio in America. Produced by Paul Wagner, Nina Gilden Seavey. Directed, written by Nina Gilden Seavey. Narration written by Stephen Chodorov. With: Narrator: Olympia Dukakis. Camera (Colorlab color), Allen Moore, Reuben Aaronson; editor, Catherine Shields; music, Paul Christianson; associate producers, Tom Wentworth, Malvina Anderson Martin. Reviewed on videocassette, N.Y., March 3, 1998. Running time: 90 min.

[118] FILM REVIEW; Once a Fear Beyond Fear Itself, by STEPHEN HOLDEN, Published: March 4, 1998, New York Times.

[119] Ibid.

[120] Duesberg, Peter and Ellison, Brian J., Inventing the AIDS Virus, Regnery Pub.,1996.

[121] Ibid.

[122] Ibid.

[123] Ibid.

[124] Ibid.

[125] Rose DR (2004). “Fact Sheet—Polio Vaccine Field Trial of 1954.” March of Dimes Archives. (2004).

[126] Ibid.

[127] American Journal of Digestive Diseases, 1953 20:330 [128] Ibid.

[129] Ibid.

[130] Jenkins, D. W.: “A Review of the Insecticide Hexachloro-cyclohexane (‘666’)”, Office of Technical Services, U. S. Department of Commerce, Washington, D.C., No. PB 4034, Med. Div. Rept. No. 56, Sept. 26, 1945.

[131] Biskind, M., “DDT Poisoning and the Elusive ‘Virus X’.” A New Cause for Gastroenteritis.” Am. J. Dig., Vol. 16, Num 3, pg. 79-84, (1949).

[132] Biskind, MS, Bieber, I, “DDT Poisoning A New Syndrome With Neuropsychiatric Manifestations,” American Journal of Psychotherapy, p261, (1949).

[133] Presented before the Select Committee to Investigate the Use of Chemicals in Food Products, United States House of Representatives, U.S. December 12, 1950 Westport, Conn.

[134] “Salk and Sabin: poliomyelitis immunisation”. J Neurol Neurosurg Psychiatry. 75 (11): 1552. doi:10.1136/jnnp.2003.028530. PMC 1738787. PMID 15489385.

[135] H. Rept. No. 2356, 82d Cong., 2d sess. 1 (1952), reprinted in A Legislative History of the Federal Food, Drug and Cosmetic Act and Its Amendments 499 (hereinafter Legislative History)

[136] Scobey, RR, “Is The Public Health Law Responsible For The Poliomyelitis Mystery?” Syracuse, N.Y., Archive of Pediatrics (May, 1951).

[137] White, Mark; Sharon M. McDonnell; Denise H.Werker; Victor M. Cardenas; Stephen B. Thacker (2001). “Partnerships in International Applied Epidemiology Training and Service,”. American Journal of Epidemiology 154 (11): 993–999. doi:10.1093/aje/154.11.993.

[138] Van Nostrand’s Encyclopedia of Science and Engineering, Van Nostrand Reinhold 1995, v 5, p1775

[139] “Salk and Sabin: poliomyelitis immunisation”. J Neurol Neurosurg Psychiatry. 75 (11): 1552. doi:10.1136/jnnp.2003.028530. PMC 1738787. PMID 15489385.

[140] Ralph R. Scobey, MD. “The Poison Cause of Poliomyelitis and Obstructions to Its Investigation.” Archive of Pediatrics, April 1952.

[141] The National Adipose Tissue Survey, reported in Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991, pg. 303.

[142] The National Adipose Tissue Survey, reported in Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991, pg. 303.

[143] Van Nostrand’s Encyclopedia of Science and Engineering (1995), vol. 5, pg.1725. [144] Offit, Paul A. (2007). The Cutter Incident: How America’s First Polio Vaccine Led to

the Growing Vaccine Crisis. Yale University Press. p. 38. ISBN 0-300-12605-0. [145] Albert Sabin to Henry Kumm, Sabin Papers, UC, Pittsburgh Press, 1954. [146] American Journal of Digestive Diseases, 1953 20:330.

[147] Trevelyan, B., Smallman-Raynor, M. and Cliff, A.D., The Spatial Dynamics of Poliomyelitis in the United States: From Epidemic Emergence to Vaccine-Induced Retreat, 1910–1971, Ann Assoc Am Geogr. 2005 Jun; 95(2): 269–293.

[148] Baicus, A., History of Polio Vaccination, World J Virol. 2012 Aug 12; 1(4): 108–114. Published online 2012 Aug 12. doi: 10.5501/wjv.v1.i4.108.

[149] Ibid.

[150] Women’s History Month: “Oveta Culp Hobby” by Senator Kay Bailey Hutchison

Humanities Texas, March 2012.

[151] Harry M. Marks, “The 1954 Salk Poliomyelitis Vaccine Field Trial,” Institute of the History of Medicine, Johns Hopkins University, Baltimore, MD: 2008.

152[ National Museum of American History, “Whatever Happened to Polio?” Time line, http://americanhistory.si.edu/polio/timeline/index.htm (accessed March 28,, 2012).

[153] Abid.

[154] Norrby E., Prusiner S.B., Polio and Nobel Prizes: looking vack 50 years. Ann Neurol.

2007 May;61(5):385-95.

[155] Eloise Batic, You Are There 1955: Ending Polio exhibit text (2012).

[156] Boston Herald newspaper, April 18, 1955, “Drug Companies Expecting Big Profit on

Salk Vaccine”,

[157] Washington Bureau of the Detroit Free Press reports, June 3, 1955.

[158] Michigan University. Poliomyelitis Evaluation Center (1955), An evaluation mof the 1954 poliomyelitis vaccine trials; summary report. Ann Arbor: n.p. , pp. 17-18 as quoted in Marks, Harry M. “The 1954 Salk Poliomyelitis Vaccine Field Trial.” Institute of the History of Medicine, Johns Hopkins University. Baltimore: 2008, p. 20.

[160] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1

[161] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research, 1957:119.

[162] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1

[163] Offit, Paul A. The Cutter Incident: How America’s First Polio Vaccine Led to the Growing Vaccine Crisis, Yale University Press, 2005, pp. 100, 116–19, 133. ISBN 0-300- 10864-8

[164] Ibid.

[165] Smith, JS, “Patenting the Sun: Polio and the Salk Vaccine,” 1st Edition, William

Morrow & Co; 1st edition (April 1990).

[166] Offit PA (2005), “The Cutter incident, 50 years later” (PDF). N. Engl. J. Med. 352 (14): 1411–1412. doi:10.1056/NEJMp048180. PMID 15814877

[167] McBean E., The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1.

[168] Harris RJ et al Contaminant viruses in two live vaccines produced in chick cells. J Hyg (London) 1966 Mar:64(1) : 1-7

[169] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1

[170] Ibid.

[171] Ibid.

[172] Ibid.

[173] Ii. Results. American journal of public health and the nation’s health. 1955;45:15–48. [PMC free article] [PubMed]

[174] Harper’s Magazine. “’Who is responsible, and why, for the chaotic confusion over the polio inoculations?’ A noted medical journalist disentangles the essential facts.” August, 1955.

[175] Ibid.

[176] Ibid.

[177] American Cancer Society, Volume 8, Issue 1, Pages 1–218, (1955).

[178] Paul JR. A history of poliomyelitis. New Haven, CT: Yale University Press; 1971.

[179] Ibid.

[180] Ibid.

[181] Ibid.

[182] Rogers N. Dirt and disease: Polio before fdr. New Brunswick, NJ: Rutgers University Press; 1992.

[183] Ibid.

[184] Smith, Derek R; Leggat Peter A (2005). “Pioneering figures in medicine: Albert Bruce Sabin–inventor of the oral polio vaccine”. The Kurume medical journal. 52 (3): 111–6. doi:10.2739/kurumemedj.52.111. PMID 16422178

[185] Rose, David, March of Dimes Archives, August 26, 2010. http://www.marchofdimes.org/mission/a-history-of-the-march-of-dimes.aspx

[186] American Journal of Public Health and the Nations Health: May 1956, Vol. 46, No. 5: 547–562. Citation | PDF (2177 KB) | PDF Plus (744 KB)

[187]

[188] Sweet BH, Hilleman MR. The Vacuolating Virus: SV-40. As cited in The polio vaccine and simian virus 40 by Moriarty, T.J. http://www.chronicillnet.org/online/bensweet.html

[189] O’Hern M. Profiles: Pioneer Women Scientists. Bethesda, MD: National Institutes of Health.

[190] Curtis T, Manson P. Scientist’s Polio Fear Unheeded: How U.S. Researcher’s Warning Was Silenced. The Houston Post 1992:A1 and A12.

[191] Sweet BH, Hilleman MR. The Vacuolating Virus: SV-40. As cited in The polio vaccine and simian virus 40 by Moriarty, T.J. http://www.chronicillnet.org/online/bensweet.html

[192] Moriarty T.J. The polio vaccine and simian virus 40. Online News Index.

http://www.chronicillnet.org/online/bensweet.html

[193] Shah K, Nathanson N. Human exposure to SV40. American Journal of Epidemiology, 1976;103:1-12.

[194] Curtis T. The origin of AIDS: A startling new theory attempts to answer the question, “Was it an act of God or an act of man”, Rolling Stone, March 19,1992:57.

[195] Bookchin D, Schumaker J. Tainted Polio Vaccine Still Carries Its Threat 40 Years Later. The Boston Globe, January 26, 1997.

[196] Innis MD. Oncogenesis and poliomyelitis vaccine. Nature, 1968;219:972–3. [197] Soriano F, et al. Simian virus 40 in a human cancer. Nature, 1974; 249:421–4.

[198] Weiss AF, et al. Simian virus 40-related antigens in three human meningiomas with defined chromosome loss. Proceedings of the National Academy of Science, 1975;72(2):609–13.

[199] Scherneck S, et al. Isolation of a SV-40-like papovavirus from a human glioblastoma. International Journal of Cancer, 1979;24:523–31.

[200] Stoian M, et al. Possible relation between viruses and oromaxillofacial tumors. II. Research on the presence of SV40 antigen and specific antibodies in patients with oromaxillofacial tumors. Virologie, 1987;38:35–40.

[201] Stoian M, et al. Possible relation between viruses and oromaxillofacial tumors. II. Detection of SV40 antigen and of anti-SV40 antibodies in patients with parotid gland tumors. Virologie, 1987;38:41–6.

[202] Bravo MP, et al. Association between the occurrence of antibodies to simian vacuolating virus 40 and bladder cancer in male smokers. Neoplasma, 1988;35:285–8.

[203] O’Connell K, et al. Endothelial cells transformed by SV40 T-antigen causeKaposi’s sarcoma-like tumors in nude mice. American Journal of Pathology, 1991;139(4):743–9.

[204] Weiner LP, et al. Isolation of virus related to SV40 from patients with progressive multifocal leukoencephalopathy. New England Journal of Medicine, 1972;286:385–90.

[205] Tabuchi K. Screening of human brain tumors for SV-40-related T-antigen. International Journal of Cancer 1978;21:12–7.

[206] Meinke W, et al. Simian virus 40-related DNA sequences in a human brain tumor. Neurology 1979;29:1590–4.

[207] Krieg P, et al. Episomal simian virus 40 genomes in human brain tumors. Proceedings of the National Academy of Science 1981; 78:6446-50.

[208] Krieg P, et al. Cloning of SV40 genomes from human brain tumors. Virology 1984;138:336–40.

[209] Geissler E. SV40 in human intracranial tumors: passenger virus or oncogenic >hit- and-run= agent? Z Klin Med, 1986;41:493–5.

[210] Geissler E. SV40 and human brain tumors. Progress in Medical Virology, 1990;37:211–22.

[211] Bergsagel DJ, et al. DNA sequences similar to those of simian virus 40 in ependymomas and choroid plexus tumors of childhood. New England Journal of Medicine, 1992;326:988–93.

[212] Martini, M., et al. Human brain tumors and simian virus 40. Journal of the National Cancer Institute, 1995;87(17):1331.

[213] Lednicky JA, et al. Natural Simian Virus 40 Strains are Present in Human Choroid Plexus and Ependymoma Tumors. Virology, 1995;212(2):710–7.

[214] Tognon M, et al. Large T Antigen Coding Sequence of Two DNA Tumor Viruses, BK and SV-40, and Nonrandom Chromosome Changes in Two Gioblastoma Cell Lines. Cancer Genetics and Cytogenics, 1996;90(1): 17–23.

[215] Vilchez RA, et al. Association between simian virus 40 and non-hodgkin lymphoma. Lancet, (March 9, 2002), 359: 817–23.

[216] Carbone, M., et al. SV-40 Like Sequences in Human Bone Tumors. Oncogene, 1996;13(3):527–35.

[217] Pass, HI, Carbone, M., et al. Evidence For and Implications of SV-40 Like Sequences in Human Mesotheliomas. Important Advances in Oncology, 1996:89-108.

[218] Rock, Andrea. The Lethal Dangers of the Billion Dollar Vaccine Business, Money, December 1996:161.

[219] Carlsen, W. Rogue virus in the vaccine: Early polio vaccine harbored virus now feared to cause cancer in humans. San Francisco Chronicle, July 15,2001:7. Research by Susan Fisher, epidemiologist, Loyola UniversityMedical Center.

[220] National Institutes of Health. Zones of Contamination: Globe Staff Graphic.

[221] Bookchin D, Schumacher J. Tainted polio vaccine still carries its threat 40 years later. The Boston Globe, January 26, 1997.

[222] SV-40 Contamination of Polio Vaccine. Well Within Online, (February 3,2001, updated). http://www.nccn.net/~wwithin/polio.htm

[223] Rosa FW, et al. Absence of antibody response to simian virus 40 afterinoculation with killed-poliovirus vaccine of mothers offspring with neurological tumors. New England Journal of Medicine, 1988;318:1469.

[224] Rosa FW, et al. Response to: Neurological tumors in offspring after inoculation of mothers with killed poliovirus vaccine. New England Journal of Medicine, 1988, 319:1226.

[225] Martini F, et al. SV-40 Early Region and Large T Antigen in Human Brain Tumors, Peripheral Blood Cells, and Sperm Fluids from Healthy Individuals. Cancer Research, 1996;56(20):4820–5.

[226] Fisher, Barbara. Vaccine safety consumer group cites conflict of interest in government report on cancer and contaminated polio vaccine link. National Vaccine Information Center (NVIC); Press Release, January 27, 1998.

[227] National Cancer Institute (June 2001).

[228] The Landsteiner and Popper study, first published in Germany, was reported in Robert W Lovett, MD. The Occurrence of Infantile Paralysis in Massachusetts in 1908. Boston Medical and Surgical Journal, pg. 112, July 22, 1909.

[229] Young, RO (2016) Second Thoughts about Viruses, Vaccines, and the HIV/AIDS Hypothesis – Part 1. Int J Vaccines Vaccin 2(3): 00032. DOI: 10.15406/ijvv.2016.02.00032

[230] Young, RO (2016) Second Thoughts Concerning Viruses, Vaccines and the HIV/AIDS Hypothesis – Part 2. Int J Vaccines Vaccin 2(3): 00034. DOI: 10.15406/ijvv.2016.02.00034

[231] Young RO (2016) Second Thoughts Concerning Viruses, Vaccines and the HIV/AIDS Hypothesis – Part 3 HIV/AIDS and the Monomorphic Disease Model. Int J Vaccines Vaccin 2(3): 00035. DOI: 10.15406/ijvv.2016.02.00035

[232] Young RO (2016) Who Had Their Finger on the Magic of Life – Antoine Bechamp or Louis Pasteur?. Int J Vaccines Vaccin 2(5): 00047. DOI: 10.15406/ijvv.2016.02.00047

[233] Peter Duesberg and Brian J. Ellison, Inventing the AIDS Virus, Regnery Pub., 1996. [234] Gerald L. Geison, The Private Science Of Louis Pasteur, Princeton University Press, 1995.

[235] The Landsteiner and Popper study, first published in Germany, was reported in Robert W Lovett, MD. The Occurrence of Infantile Paralysis in Massachusetts in 1908. Boston Medical and Surgical Journal, pg. 112, July 22, 1909.

[236] Shaw D. Unintended casualties in war on polio. Philadelphia Inquirer June 6, 1993:A1.

[237] Moriarty T.J. The polio vaccine and simian virus 40. Online News Index. http://www.chronicillnet.org/online/bensweet.html

[238] Koprowksi H. Tin anniversary of the development of live virus vaccine. Journal of the American Medical Association 1960;174:972–6.

[239] Hayflick L, Koprowski H, et al. Preparation of poliovirus vaccines in a human fetal diploid cell strain. American J Hyg 1962;75:240–58.

[240] Hayflick L, Koprowski H, et al. Preparation of poliovirus vaccines in a human fetal diploid cell strain. American J Hyg 1962;75:240–58.

[241] Koprowski H. In a letter sent to the Congressional Health and Safety Subcommittee, April 14, 1961.

[242] Rock, Andrea. The Lethal Dangers of the Billion Dollar Vaccine Business, Money, December 1996:161.

[243] Scheibner V. Vaccination: 100 Years of Orthodox Research Shows that Vaccines represent a Medical Assault on the Immune System. Blackheath, NSW, Australia: Scheibner Publications, 1993153.

[244] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[245] Carlsen, W. Rogue virus in the vaccine: Early polio vaccine harbored virus now feared to cause cancer in humans. San Francisco Chronicle, July 15,2001:7. Research by Susan Fisher, epidemiologist, Loyola UniversityMedical Center.

[246] Neustaedter R. The Vaccine Guide. Berkeley, California: North Atlantic Books, 1996:107–8

[247] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[248] Essex M, et al. The origin of the AIDS virus. Scientific American, 1988; 259:64–71. [249] Karpas A. Origin and Spread of AIDS. Nature, 1990; 348:578.

[250] Kyle WS. Simian retroviruses, poliovaccine, and origin of AIDS. Lancet, 1992; 339:600–1.

[251] Elswood BF, Stricker RB. Polio vaccines and the origin of AIDS. Medical Hypothesis, 1994:42:347–54.

[252] Myers G, et al. The emergence of simian/human immunodeficiency viruses. AIDS Res Human Retro 1992:8:373–86.

[253] Curtis T. The origin of AIDS: A startling new theory attempts to answer the question “Was it an act of God or an act of man”, Rolling Stone, March 19,1992:57.

[254] O’Hern M. Profiles: Pioneer Women Scientists. Bethesda, MD: National Institutes of Health.

[255] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[256] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[257] Essex M, et al. The origin of the AIDS virus. Scientific American, 1988; 259:64–71. [258] Karpas A. Origin and Spread of AIDS. Nature, 1990; 348:578.

[259] Kyle WS. Simian retroviruses, poliovaccine, and origin of AIDS. Lancet, 1992; 339:600–1.

[260] Elswood BF, Stricker RB. Polio vaccines and the origin of AIDS. Medical Hypothesis, 1994:42:347–54.

[261] Workshop on Simian Virus-40 (SV-40): A Possible Human Polyomavirus. National Vaccine Information Center, January 27-28, 1997. http://www.909shot.com/polio197.htm (Includes a summary of evidence presented at the Eighth Annual Houston Conference on AIDS.)

[262] Martin B. Polio vaccines and the origin of AIDS: The career of a threatening idea. Townsend Letter for Doctors, January 1994:97–100.

[263] Curtis T. Did a polio vaccine experiment unleash AIDS in Africa? The Washington Post, April 5, 1992:C3+.

[264] Myers G, et al. The emergence of simian/human immunodeficiency viruses. AIDS Res Human Retro 1992:8:373–86.

[265] World Health Organization. T-lymphotropic retroviruses of nonhuman primates. WHO informal meeting. Weekly Epidemiology Records, 1985; 30:269–70.

[266] Ibid.

[267] Elswood BF, Stricker RB. Polio vaccines and the origin of AIDS. Medical

Hypothesis, 1994:42:347–54.

[268] Ohta Y, et al. No evidence for the contamination of live oral poliomyelitis vaccines with simian immunodeficiency virus. AIDS, 1989; 3:183–5.

[269] Huet T, et al. Genetic organization of a chimpanzee lentivirus related to HIV-1. Nature, 1990; 345:356–9.

[270] Desrosiers RC. HIV-1 origins: A finger on the missing link. Nature, 1990;345:288– 9.

[271] Sabin AB. Properties and behavior of orally administered attenuated poliovirus vaccine. Journal of the American Medical Association, 1957; 164:1216–23.

[272] Siehe AusfĂŒhrungen zu Virchows Leben und Wirkung in WissenschafftPlus Nr. 5/2015 und Nr. 6/2015. 2 Anticontagionism between 1821 and 1867.

[273] Aufsatz von Erwin H. Ackerknecht in der Zeitschrift Bulletin of the History of Medicine, Volume XXII, The Johns Hopkins Press, 1948.

[274] Bech V, Magnus Pv. Studies on measles virus in monkey kidney tissue cultures. Acta Pathol Microbiol Scand. 1959; 42 (1): 75–85.

[275] Nakai M, Imagawa DT. Electron microscopy of measels virus replication. J. Virol. 1969 Feb; 3v (2): 187–97.

[276] Lund GA, Tyrell, DL, Bradley RD, Scraba DG. The molecular length of measles virus RNA and the structural organization of measles nucleocapsids. J. Gen. Virol. 1984 Sep;65 (Pt 9): 1535–42.

[277] Daikoku E, Morita C, Kohno T, Sano K. Analysis of Morphology and Infectivity of Measles Virus Particles. Bulletin of the Osaka Medical College. 2007; 53 (2): 107–14.

[278] Horikami SM, Moyer SA. Structure, Transcription, and Replication of Measles Virus. Curr Top Microbiol Immunol. 1995; 191: 35–50.

[279] Siehe WissenschafftPlus Nr. 1/2014.

[280] Zur Geschichte der frĂŒhen Virusforschung. Übersichtsarbeit von Prof. Karlheinz LĂŒdtke. Reprint 125 des MAX-PLANCK-INSTITUT FÜR WISSENSCHAFTSGESCHICHTE, 89 Seiten, 1999.

[281) The government of the United States of America holds patents on the following viruses: Ebola, Patent number #CA2741523A1, Swine Flu, Patent number 8124101, HIV, Patent number #5676977, the cure for cancer, Patent number #6630507.

In Memory of Martin Luther King, Jr. – January 21st, 2019

Martin Luther King, Jr. One of the Greatest Freedom Fighters of our time!

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Free at Last, Free at Last! Thank God I am Free at Last!

 
Several years ago I had the beautiful experience to speak freely at the Martin Luther King Jr Memorial Chapel in Atlanta, Georgia on the campus of Morehouse College by Professor Dean Lawrence Carter, Jr. (below I am pictured with Professor Lawrence Carter, Dean of the Martin Luther King Chapel at More House College, Atlanta, Georgia.
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When introducing me to speak at the Martin Luther King, Jr. Memorial Chapel, Dr. Carter stated, “Dr. Robert O. Young is the Martin Luther King Jr. of the 21st Century.”) It was one of my greatest memories to stand at the same pulpit where Martin Luther King, Jr., Ikada, Ghandi and Mandela delivered powerful messages of freedom, love and light. There in front of thousands, I was blessed to have the opportunity to share my message of freedom, love and light. A message that has now blessed the lives of millions around the World. Thank you God for the blessing of service in my life. Thank you all for the opportunities you have given me to serve you, my brothers and sisters – my friends.
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One of my favorite quotes of Martin Luther King, Jr. is one that has impacted my life in so many ways, when he said, “Never, never be afraid to do what’s right, especially if the well-being of a person or animal is at stake. Society’s punishments are small compared to the wounds we inflict on our soul when we look the other way.” Yes, it was hard to be incarcerated for 5 months at the East Mesa Re-entry Facility, but I want you all to know that I have no regrets! Today, I am so grateful to be home with my family and friends.
 
After almost 35 years of studying and learning about acid – base chemistry in vertebrates, and the same number of years attempting to share what I have observed and learned, I am enormously gratified to see the larger scientific community beginning to recognize and validate my work, research and discoveries. It has been a long journey out of darkness. Almost every day now some new scientific paper is published that validates my work, such as the discovery of a new and the largest organ of the body called the Interstitium, which I have been studying for over 25 years.
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Recently at a scientific conference I heard a noted scientist state, “In certain conditions, we believe it is better to have the tissues properly alkalized.” He did not give me credit, but his knowledge came from my work. You have no idea how far the journey has been from where I started to hearing those words from a distinguished member of the scientific community.  To learn more about the interstitial fluids of the Interstitium read, “Alkalizing Nutritional Therapy in the Prevention and Treatment of Any Cancerous Condition.”
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I have lived with doubt and criticism for so long that I have come to understand it as actually encouraging and exciting. No one takes the time to write to a newspaper about something that does not interest them. When people take the time to read, investigate and try to understand and then to sit down and write to an editor to complain, what they are really doing is asking questions; asking the author to explain his or her self; to defend their work. It is wonderfully energetic and encouraging to see people interested and asking questions. Asking questions is the first step towards knowledge. It is a sign of courage and intellectual bravery to ask questions and seek knowledge.
 
We, as humans, live in such profound darkness. Not knowing what is in the dark is a very scary thing. We, like children, need to know there are no boogey men under the bed. The truth is adults are afraid too. We tell our children there are no boogey men, but we still look under the bed ourselves just to be sure. The truth is we don’t know any more than we do know.
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We live in a Universe of what Donald Rumsfeld, the former American Secretary of Defense, called “Unknown Unknowns”. The more we learn, the more we realize how much we still don’t know. Albert Einstein once quipped, “Intelligence is a very humbling thing. It makes us realize that what the greatest of us knows, pales in comparison to that which none of us knows.” Knowledge is always being accumulated. Much of it disturbs our serenity. We want to believe we know at least most of what is to be known. But, alas, we know so very little.
 
We are much better off today than most humans were when they died in their 30â€Čs, of things like infected teeth, which dentists today deal with so easily, and from minor wounds, that surgeons today routinely stitch up in minor medical clinics. Our knowledge is greater than it was for even our parents. We continue to learn, in spite of our very human desire to believe we already know most of what we need to know. Today it is said that all of human knowledge is compounding about every 3 years. In other words, we will learn more in the next 3 years than we have learned in our entire previous recorded history. My work is in that record. Today the medical professions, and healers around the world are just beginning to understand what I have been teaching for over 3 decades.
 
The very thing that people complain about, is actually a result of the broader acceptance of my work in the scientific community. When someone writes, “I
was shocked to discover a number of UK companies promoting practices and diets based on his theories.” It both excites and encourages me that people are finally beginning to “get it.” I can understand why “getting it” is so unnerving. It recognizes that all along there has been a boogey man under the bed that we did not know was there! The good news is, now that we know that living an acidic lifestyle will make us sick, and accelerate aging and hastens death, we can do something about it! Just like now we can treat infected teeth and stitch up wounds, that once killed us at a very early age.
I still laugh every now and then about a joke I heard on the old American Hee Haw TV show years ago, “Junior” said, “A man told me he broke his leg in two places. I told him the thing to do was to stay out of those places!” It’s the same with an acidic lifestyle. If the things you are doing are making you sick, then stop doing them! As it has been said, “The definition of insanity is doing the same thing over and over again and expecting a different result.”
 
I have had people object to my saying that an HIV, Ebola or Zika Virus does not cause AIDS or disease and that vaccination can protect you. A great percentage of the larger scientific community does not believe that either. Are you familiar with the name Luc Antoine Montagnier? He received the Nobel Prize in medicine for discovering the HIV virus. Where is he NOW? He was a World Famous Professor and Scientist at the University of Paris. In 2011, Dr, Luc Montagnier lost his position at the University of Paris and was exiled to China. Why? Because he reversed his position on the so-called HIV virus, its existence and cause of AIDS. How would I know this? Because we lectured together as the Key Note Speakers in October, 2011 in Milan, Italy and he shared his horrific story with me.
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I am in good company. The way to shut us all up is not to exile us or to through us in jail, or even kill us like many of my colleges, but for someone to prove that HIV, Ebola or Zika actually does cause AIDS or disease, using the scientific method called, Koch’s postulates. That hasn’t been done, because it can’t be done. HIV, Hep C, Ebola, Polio, Measles and Zika are all phantom viruses. Scientists have never isolated these viruses or proven that they cause any disease. In fact, everyone in Brazil knows that Zika is not a virus and does not cause birth defects. They know that these birth defects are being caused by eating fruit and vegetables that are laced with an acidic toxic chemical called Glyphosate (N-(phosphonomethyl)glycine), a broad-spectrum systemic herbicide and crop desiccant.
 
Some people object to my theory of multi-forms or pleomorphism and the origins of what are called bacteria, yeast, mold and viruses. But, you don’t have to know or understand the origins of these biological forms to understand that if your body is properly alkalized none of them can reproduce and none of them can cause any of the ill effects thought to be associated with them. How these biological forms arise is, and has been for centuries, a great debate. The proof of my work is in the results. For at least a century, it has been known that cancers form and thrive only in overly acidic tissue.” I did not develop that knowledge, I only explained it. Don’t blame the messenger for the message.
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Diabetes is another condition that has been largely misunderstood. For decades the way the medical community dealt with diabetes was only to treat the symptoms. The symptoms were targeted, because it was not known what causes diabetes. I like to say, we have always known what caused diabetes, we just did not like the answer. The answer has always been, change your lifestyle, and change your diet!
 
But, we humans like our cures to fit our lifestyles not to adjust our lifestyles to prevent the conditions. You want to turn diabetes around over night? Get all of the animal proteins out of your diet, along with all of the simple carbohydrates and sugars, stop drinking acidic beverages, and eating highly acid foods, add back in the alkaline green plants and simply watch what happens. Learn the cause and the self-cure for Type 1 and Type 2 diabetes by reading The pH Miracle for Diabetes!
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Here is something fun for your family members to do. Go on the Internet and Google “Eggs cause Diabetes.” I have been saying this for years to howls of criticism. Now the larger scientific community is beginning to understand what I have been saying, and my critics are stunned
 What!?
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ALL animal proteins are acidic and cause degenerative conditions we like to call diseases. Sorry.. it’s the message that is not liked. I’m just the messenger.
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One last thing, I get criticized frequently because I did not receive my DSc. Ph.D. and ND from Harvard or Johns Hopkins, or some other favored institution. I wish I could have afforded those institutions, but the schools I attended were and are fine institutions. Snob appeal does not make a good institution. We just love to establish ranks of exclusions. In the U. S. to have attended a fine engineering school you need to have attended MIT, or Stanford
 most recently California Institute of Technology has taken the lead, but the truth is the Indian Institute of Technology in India is widely recognized as the finest engineering school in the World.
 
Institutions do not make the quality of their students. The students make the quality of the Institutions. In fact, institutions do not “teach” creativity or innovation. All institutions do is teach what is presently known, not what is yet to be discovered. More often than not, throughout time, our greatest discoveries have come from individuals with very little formal education, Steve Jobs from Apple, Mark Zuckerberg, the guy that started Facebook and Bill Gates, the founder of Microsoft. Unfettered by dogma and entrenched lore, visionaries look at the world with new eyes and see things others could not or cannot see.
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I am very proud of the knowledge I was given by the institutions I attended, but I am most proud of the work I have done that has expanded that knowledge and built on what was known when I was at the University
 work, that after 35 plus years is finally being recognized and validated
 work that is finally reaching the victims of ignorance, and making a difference in their lives.  You can review a copy of my CV at: https://www.drrobertyoung.com/curriculim-vitae
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Read the following article of Inger Hartelius and how she reversed her terminal lung cancer condition –
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Click on this link to read Inger’s story:
 
 
 
What I encourage everyone to do, especially my critics, is to continue to read, study, ponder, listen and learn; take charge of your own health and do what works! This is how I have come to all of my conclusions
 watching and studying what works, and building on that evidence. Scholars can argue about WHY an apple falls from the tree, but the important thing is to note is that it does!
 
God bless you all and God bless America with the capacity to love one another and NEVER turn away from a soul who is in need of a helping hand.  I promise you it will do your soul good.
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In the words of the late Charles Krauthammer, in his book, “The Point of it All,” said something that I believe with all my heart, “You’re betraying your whole life if you don’t say what you think – and you don’t say it honestly and bluntly.”
 
So to all my loved ones, family, friends and even my critics and enemies, thank you for being in my life! You have all, in your own way, taught me how to care more, love more, serve more, live more and to be grateful for this beautiful opportunity to learn and live together on Earth!
 
In Love and God’s Healing Light,
 
Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner
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PS What Does It Mean to be Truly Free?

 
What is freedom if it is not to be free in every way, from our most minute cell to our most expansive dreams? He is free who can afford to let the interactions between the cell and spirit take place in a most harmonious and loving way. There is no freedom in the philosophies of men. Freedom of that sort lasts for only a duration of a thought, of an act. To be truly free is to be able to establish peace between all opposition within us! To realize that the circumstances of our lives are not important as compared to the kindness, thoughtfulness, acceptance, understanding, and love we show to others. The picture below is a micrograph of healthy live red blood cells seen under pHase Contrast microscopy.
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To read and learn more about the work, research and findings of Robert O Young go to: http://www.drrobertyoung.com
 
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To attend a pH Miracle Retreat go to: http://www.phmiracleretreat.com

 

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Come listen and learn from Key Note Speakers, Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner and Galina Migalko MSc, MD, NMD, in four different countries around the World as they lecture on non-invasive medical diagnostics, the interstitium, pH, nutrition and their break-through research on prevention and non-invasive treatments for cancer, diabetes, heart disease, arthritis, osteoporosis, lupus, multiple sclerosis, infections, and many more acidic-caused diseases.
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To Attend a Medical Conference

 
To pre-register for one or more World Conferences please email phmiraclelife@gmail.com and receive an additional 10 to 20 percent discount on the listed early-bird pricing. You can also register by phone by calling me direct at: 760 484 1075.
 
Please check out the Countries, Cities, Dates and Pricing on my personal website at: http://www.drrobertyoung.com!
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How Long Do YOU Want to LIVE?

Life and Death is all about managing and maintaining the delicate alkaline pH environment of the blood plasma, the interstitial fluids of the Interstitium and the intracellular fluids of the body cells, at 7.365.
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[The first micrograph shows a lactic acid crystal in the live blood..  The second mircrograph shows a citric acid crystal in the live blood]
You can manage and support all of the body fluids with what you eat, what you drink, what you breathe, what you think and what you believe.  Yes, even your thoughts and beliefs produce metabolic acidic waste products that can make you sick and tired.  Your thoughts and beliefs do become biology and do affect the pH of all body fluids.
Today, medical science teaches that the body regulates the pH of the body fluids automatically and factors such as what you eat, drink, breathe, think or believe have no bearing or influence on the biochemistry of the body fluids.  In other words, your lifestyle has NO influence on the chemistry, including the pH of the blood, interstitial fluids or the fluids inside the cells or intracellular fluids!
This thinking should and does not make common sense since we do know scientifically that uric acid from eating animal protein causes gout, lactic acid from metabolism and from dairy products causes inflammation and cancer, acetaldehyde and alcohol causes breast cancer, diabetes, and pancreatic and liver diseases, including cancer and the acidic pesticide DDT and glyphosate (the main chemical in the pesticide Round-up) causes birth defects, dementia, polio, measles, MS, Zika and YES cancer.
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The good news is a medical research group in America just announced last year that they had discovered a new organ in the human body called the Interstitium. Researchers are claiming that this organ is not only the largest organ of the human body but it contains compartments that hold metabolic, dietary, respiratory and environment acidic waste.  This acidic waste in the interstitial fluids of the Interstitium is held in these compartments until they can be eliminated via urination, defecation, perspiration, menstruation and respiration.  This new anatomical, functional and physiological discovery changes the false belief by the current medical establishment.

Click here to watch the video on the Interstitial fluids of the Interstitium: https://youtu.be/flnE1-XHE8Y

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With new medical technology we can now test the chemistry, including the pH of ALL the body fluids which proves that the Interstitium, I call the third kidney, is holding predominately the majority of all acidic waste in these holding compartments.  This is done by the body in order to maintain  and protect the delicate pH balance of the blood plasma  and keep you alive.
Just testing the chemistry or parameters of the blood plasma can and many times will give medical doctors a false positive or false negative.  Why?  Because all the the normal or negative factors happening in the body can only be revealed by testing the fluids of the Interstitium where all the acidic toxins are being stored, until eliminated through the channels of elimination.
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When medical doctors make false statements that what you eat, drink, breathe or think does not effect the chemistry of the body fluids, they do not realize that the pH of the interstitial fluids of the Interstitium are being compromised every day by ALL of these factors.  Testing the chemistry of the interstitial fluids of the Interstitium and comparing this information to the chemistry of the blood plasma is critical in correctly determining a real or accurate medical diagnosis!  I have determined from my own research over the years that the testing of the Interstitial fluids of the Interstitium is the gold standard in medical diagnostics and the holy grail to prevention, diagnostic, and effective treatments for ALL sickness and disease!
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Currently, we are the only research and diagnostic group, in the World, testing ALL of the body fluids for chemistry, including pH!
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Medical Testing and Diagnostics Needs to Change!  

It is so critical that YOU share this new medical science with everyone you love and care about.  Testing the interstitial fluids of the Interstitium will save lives and prevent serious health challenges down the road.  It will provide YOU the certainty that your heath practitioner needs to advise YOU correctly on your current health and fitness condition.  Measuring and understanding the chemistry of the Interstitium will reveal whether or not your current health and fitness plan is effective and healthful.  This would include your nutritional protocol, exercise, or even the legend drugs you maybe taking!  No more guessing or diagnosing a health condition based upon symptoms or beliefs or even your doctors beliefs.
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Quantifying the interstitial fluid chemistry is the KEY to understanding YOUR REAL current health and fitness condition!

The picture below is a diagram of the breakdown of body fluids (Figure 1) and a cross-section of the largest organ of the human body – The Interstitium (Figure 2). Keep in mind if you ask your doctor about the Interstitium his/her eyes will glaze over because most medical doctors know absolutely nothing about this organ! In fact, medical science just recognized this organ in 2018 – a organ I have been studying for over 30 years!
Figure 1
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I Want To Know More!

To learn more about the Interstitium and the chemistry of the blood, interstitial fluids and the Intracellular fluids read my peer-reviewed research, “Alkalizing Nutritional Therapy in the Prevention and Treatment of Any Cancerous Condition.”
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On February 9th, 2019, ACTA Scientific Cancer Biology, a peer-reviewed journal, will be publishing my article on the cause, treatment and reversal of Cystic Fibrosis and Pulmonary Adenocarcinoma Lung Cancer! The title of my article is: “Cystic Fibrosis and Pulmonary Adenocarcinoma Lung Cancer both Metabolic and Dietary Acidic Conditions.”
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The following is the abstract for this article:

Cystic fibrosis (CF) [1,2] and Pulmonary Adenocarcinoma (PAC) [3] have similar symptomologies and are chronic, progressive, and frequently fatal acidic conditions of the respiratory system (lungs), lymphatic system (lymph nodes), intestines, pancreas, urinary tract system, reproductive organs and the skin as the alkaloid glands (the salivary glands, stomach, and small and large intestines) produce and secrete alkaline compounds, such as sodium bicarbonate to buffer and preserve the alkaline design of the body and the specific organs and glands affected. These metabolic and dietary acidic conditions resulting in the build-up of mucous [3] can affect any organ or organ system but primarily affects the respiratory, lymphatic system, digestive, and reproductive tracts in children and young adults with CF and the lungs and surrounding lymph nodes in PAC. I have suggested from my own clinical research that both of these conditions are the result of latent tissue acidosis (LTA) in the interstitial fluids of the Interstitium or the fluids that surround every cell, created from metabolism, diet, thoughts and environment and may be successfully treated and reversed with an alkaline lifestyle and diet (ALD) [4].
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The Answer to My Question to YOU!

Now, in answer to my question to YOU, “How long do YOU want to LIVE,” I have included a picture of a 400 year old Bonsai Tree to give you a glimpse of the possibilities for increasing your longevity by actively managing and measuring the chemistry of YOUR body fluids, especially the interstitial fluids of the Interstitium.
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Dr. Alexis Carrel received the Nobel prize in medicine many years after his death for discovering the longevity of the human cell. What did he find? That he could keep a body cell alive forever if he would simply change-out the acidic fluids that surround the cell every 24 to 48 hours. His experiment was with a chicken heart that he kept alive for over 20 years by changing-out the interstitial fluids each day. The chicken heart only died after he stopped managing the interstitial fluids of the heart.
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You must protect, manage and maintain the alkaline design of the body fluids if YOU want to prevent sickness or disease and extend the quality and quantity of your life!
You must be protected just like Dr. Alex Carrel’s chicken heart or the 400 year old Bonsai tree above that was protected by a concrete walled nursery from the atomic bomb dropped on Hiroshima, Japan.
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I am suggesting in my work, research and findings that the KEY to a healthy and long life, can be found in the interstitial fluids of the Interstiitum – the environment that bathes every cell of the human body determines how long a cell will live to even the DNA expression and ultimately determines how long YOU will live!  Genes DO NOT Determine YOUR Destiny!  The interstitial fluids of the Interstitium determines YOUR Destiny!
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[The blue and dark blue areas represent the interstitial fluids of the Intersititium that determine the expression of the DNA]
Life is a choice just as death is a choice. You choose it every day with what you eat, what you drink, what you breathe, what you think and what you believe. So Choose Wisely!
Finally, remember that ALL cells of the human body are only as healthy as their environment or the interstitial fluids of the Interstitium, in which they reside and live!
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To learn more about Dr. Young’s 35 plus years of published peer-reviewed research you can go to his personal website at: http://www.drrobertyoung.com or you can email Dr. Young at:
phmiraclelife@gmail.com
To schedule a non-invasive blood plasma, interstitial fluids and intracellular fluids test for chemistry, including pH please contact us at: phmiraclelife@gmail.com
Dr. Robert O. Young and Dr. Galina Migalko will be speaking at the Global Summit on Oncology and Cancer in London, England on March 18th and 19th.  If you would like to register for these events please make your request at: phmiraclelife@gmail.com
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Using Sodium and Potassium Bicarbonates in the Prevention and Treatment of ALL Sickness and Disease

Using Sodium and Potassium Bicarbonates in the Prevention and Treatment of ALL Sickness and Disease
Robert Young PhD

Naturopathic Practitioner – The pH Miracle Ti Sana Detox Medical Spa

Using Sodium and Potassium Bicarbonates in the Prevention and Treatment of ALL Sickness and Disease

Abstract

This article suggests that the use sodium and potassium bicarbonates are non-toxic primary alkalizing agents in the prevention and  treatment of all cancers, kidney disease, liver disease, Type I & Type II diabetes, Lupus, heart disease, Pharmacological toxicosis, vascular surgery operation, tonsillar herniation due to cerebral edema, lactic acid toxicosis, and hyponatremia or low salt or loss of salts due to excessive or over-exercise!

[Key words: cancer, diabetes, lupus, heart disease, vascular surgery, herniation, cerebral edema, lactic acid toxicosis, liver disease, kidney disease, hyponatremia, Pharmacological toxicosis]

Introduction

Sodium and potassium bicarbonate are excellent agents for a natural alkaline approach in the treatment for all sickness and disease, including cancer. Sodium bicarbonate is the universal mainstream treatment of acidosis. It is used every day by oncologists to neutralize the heavy acidic nature of their chemical and chemotherapeutic agents which are often quite toxic. Sodium bicarbonate is also used routinely in many clinical situations as herein noted including many peer–reviewed journals:

1) Severe diabetic ketoacidosis (1)

2) Cardiopulmonary resuscitation (2)

3) Pregnancy (3)

4) Hemodialysis (4)

5) Peritoneal dialysis (5)6) Pharmacological toxicosis (6)

7) Hepatopathy (7)

8) Vascular surgery operations (8)

Medics and emergency room medical doctors are accustomed to participating in a flurry of activity when trying to save a persons live after a cardiac arrest–inserting IVs and breathing tubes, performing defibrillation to restart the heart, etc. Sodium bicarbonate is a constant performer under such conditions and is more commonly used than magnesium injections, which is traditionally at the top of every doctor’s protocol for cardiac arrest.

Mainstream oncologists recognize the routine involvement of late stage infections which I refer to as outfections in all cancerous conditions. Medical savants also recognize that bacteria, yeast and mold is present in over forty percent of all cancerous conditions. (9) The most recent research in this area demonstrates how even viruses, which I describe as crystallized acid, is present in fifty percent of certain types of cancerous conditions. (10)

Sodium and potassium bicarbonate increases the hydroxyl ions or electron levels through increased alkalinity to the cells buffering the metabolic acids that can cause cancer.(20)  It is also one of the most basic medicines in allopathic and alternative medicine we have for the treatment of kidney disease.  Research by British scientists at the Royal London Hospital shows that sodium bicarbonate can dramatically slow the progress of chronic kidney disease.(11) We don’t need a thousand years of scientific tests to understand something as simple and essential as alkaline water and it is quite the same with sodium and potassium bicarbonate. Sodium and potassium bicarbonate are always present in the best alkaline drinking waters and organic raw green foods and is constantly being produced by the cover cells of the stomach to alkalize the acidic foods and liquids we ingest, including buffering metabolic and respiratory acids in order to maintain the alkaline design of the blood and tissues at a delicate pH of 7.365.(20)

What is Latent Tissue Acidosis?
Medical doctors are not taught in medical school and therefore do not understand or recognize latent tissue acidosis. They understand and recognize compensated acidosis and decompensated acidosis. In compensated acidosis, breathing increases in order to blow off more carbonic acid which decreases PCO2 because of the lowered carbonate or HCO3. When the breathing rate can no longer get any faster and when the kidneys can no longer increase its’ function to keep up with the acid load, then the blood pH starts to change from a pH of 7.365 to 7.3 then to 7.2. At a blood pH of 6.95 the heart relaxes and the client goes into a coma or dies.

Latent “acidosis” is a condition that exists when there are not enough bases in the alkalophile glands because they have been used up in the process of neutralizing the acids adsorbed to the collagen fibers. This leads to compensated “acidosis.” This means the blood pH has not changed but other body systems have changed. This can then lead to decompensated “acidosis” where the alkaline reserves of the blood are used up and the pH of the blood is altered. Decompensated “acidosis” can be determined by testing the blood pH, urine pH and the saliva pH. The decrease in the alkaline reserves in the body  can occur because of hyper-proteinization, (eating meat and cheese!) or too much protein, and hyper-carbonization, or too much sugar or from excessive or over-excercise. This is why young athletes fall over dead or why 80 to 90 year old folks are all shrunk up and look like prunes. They have very little or no alkaline reserves in their alkalophile glands. When all the alkaline minerals are gone, so are you and your battery runs out of charge. The charge of your cellular battery can be measured by testing the ORP or the oxidative reduction potential of the blood, urine or saliva using an ORP meter. As you become more acidic this energy potential or ORP increases.

How Is Sodium Bicarbonate Created In The Body?

The parietal or cover cells of the stomach split the sodium chloride of the blood. The sodium ion is used to bind with water and carbon dioxide to form the alkaline salt, sodium bicarbonate or NaHCO3. The biochemistry is: H20 + CO2 + NaCl = NaHCO3 + HCL. This is why I call the stomach an alkalizing organ NOT an organ of digestion. The stomach DOES NOT digest the food or liquids we ingest but it alkalizes the foods and liquids we ingest.  We have one instrument in the human body to digest food and it is NOT the stomach it is your teeth.  Once we swallow our food or drink the stomach begins to prepare the food by alkalizing it in a bath of sodium bicarbonate.

For each molecule of sodium bicarbonate (NaHCO3) made, a molecule of hydrochloric acid (HCL) is made and secreted into the so-called digestive system – specifically, the stomach (the gastric pits in the stomach) – to be eliminated via the blood. Therefore HCL is an acidic waste product of sodium bicarbonate created by the stomach to alkalize the food and liquids ingested.

Exercise Creates Metabolic Acidic Waste Products Which Are Harmful To The Blood and Tissues

When one exercises or over-exercises the body needs additional alkaline bicarbonate salts to buffer lactic acids.  The additional bicarbonate is created in the stomach lining to buffer the increased amounts of lactic acids produced as a waste product of metabolism.  The production of sodium bicarbonate will always leave an acidic waste product of hydrochloric acid in the gastric pits of the stomach leading to nausea, light headedness, dizziness, muddle thingking, and poor circulation.  If the excessive exercise continues this can then lead to a dificiency of mineral and bicarbonate salts (electrolytes lost through perspiration or urination) which may lead to latent tissue acidosis, pain, edema, hyponatrenia and death.

But how does something like sodium and/or potassium bicarbonate, so seemingly innocuous have such a dramatic effect? During prolonged or intense exercise muscles produce large amounts of acidic waste products, such as lactic acid, that lead to soreness, stiffness, fatigue and possible edema if these acids are not buffered and eliminated through urination or perspiration. Because sodium and potassium bicarbonate naturally reduces metabolic acids, it acts as a buffer against these performance-limiting by-products.

Current research suggests that supplemental sodium bicarbonate, like the pH Miracle pHour Salts (contains sodium and potassium bicarbonate) is particularly helpful in speed-based events, including sprints, football and other fast-moving games, and middle-distance (up to 10km) running, swimming and cycling. “Essentially, sodium bicarbonate is an alkaline substance that increases the pH of the blood,” Dr Folland says. “This seems to reduce and offset the acidity produced in the muscles during intense, anaerobic exercise that produces lactic acid most quickly, such as fast running or swimming.”

In Dr Folland’s study, swimmers who took the sodium bicarbonate knocked 1.5 seconds off their time for 200m, a difference that may seem insignificant to recreational swimmers but which is substantial at elite level.

“At the last Olympics, the top four swimmers in the men’s 200m freestyle were separated by just 1.4 seconds,” Dr Folland says. “So, in theory, it could be the difference between winning a medal and not.”

Anyone can try it, he says, but only those who are serious enough to monitor their times and progress in sports such as running, swimming or cycling may notice the few seconds advantage it might provide. “The increments of improvement are relatively small to the average person, although significant to someone who competes,” Dr Folland says.

Athletes for years have sworn that taking a spoonful of bicarbonate of soda (baking soda) helps them to keep going for longer. For years, experts doubted that there was anything other than a placebo effect to these claims until they subjected the substance to rigorous examination. Most exercise scientists investigating the trend for “soda-doping” among athletes and gym-goers have shown that it offers significant benefits for endurance and speed.”

At Loughborough University, for instance, physiologists reporting in the June issue of the International Journal of Sports Medicine showed that swimmers who took baking soda about one hour before a 200m event were able to shave a significant time off their usual performances. Dr Jonathan Folland, who led the study, says that it is not uncommon for top swimmers to take sodium bicarbonate (another name for the substance) before a competition to give them an edge. Indeed, he showed that of nine swimmers tested, eight recorded their fastest times after ingesting a supplement of the common baking ingredient – sodium bicarbonate.

Where are Bicarbonates Created In The Human Body and Why?

The chloride ion from the sodium chloride (salt) binds to an acid or proton forming HCL as a waste product of sodium bicarbonate production. HCL has a pH of 1 and is highly toxic to the blood and tissues and the cause of indigestion, acid reflux, ulcers, diabetes, cancer, hyponatremia, edema, tonsilar herniation and death.  When large amounts of acids, including HCL, enter the stomach from a rich animal protein or dairy product meal, such as meat and cheese, or from starchy foods from root vegetables like potatoes or during extreme exercise, acid is withdrawn from the acid-base household. The organism would die if the resulting alkalosis – or NaHCO3 (base flood) or base surplus – created by the stomach was not taken up by the alkalophile glands (salivary glands, pancreas, kidney, pylorus glands, Brunner’s glands, Lieberkuhn glands and liver) that need these quick bases in order to build up their strong sodium bicarbonate secretions. These alkalizing glands and organs are the stomach, pancreas, Brunner’s glands (between the pylorus and the junctions of the bile and pancreatic ducts), Lieberkuhn’s glands in the liver and its bile with its strong acid binding capabilities which it has to release on the highly acidic meat, cheese, potato, acid water or metabolic and/or respiratory acids from over-exercise to buffer its strong acids of nitric, sulphuric, phosphoric, uric and lactic acids in daily metabolism, respiration and excessive or over-exercise.

Bicarbonate acts to stimulate the ATPase by acting directly on it.(12)

The simple household product used for baking, cleaning, bee stings, treating asthma, cancer and acid indigestion is so effective in treating disease that it prevents patients from having to be put on kidney dialysis. The findings have been published in the Journal of the American Society of Nephrology. Bicarbonate is a truly strong universal concentrated nutritional medicine that works effectively in many clinical situations that we would not normally think of. Bicarbonates of sodium and potassium are a prime emergency room and intensive care medicine that can save a person’s life in a heartbeat and it is also a supermarket item that you can take right off the shelf and use for more things than one can imagine – including diaper rash.

Dr. SK Hariachar, a nephrologist who oversees the Renal Hypertension Unit in Tampa, Florida stated, upon seeing the research on sodium bicarbonate and kidney disease, “I am glad to see confirmation of what we have known for so long.  I have been treating my patients with bicarbonate for many years in attempts to delay the need for dialysis, and now we finally have a legitimate study to back us up. Not only that, we have the added information that some people already on dialysis can reverse their condition with the use of sodium bicarbonate”.

A dialysis technician at the same center as Dr. Hariachar, who used to be on dialysis himself for 2 years as a result of kidney failure, had his kidneys miraculously start functioning to the point where dialysis was no longer needed. He states that he was prescribed oral doses of sodium bicarbonate throughout his treatment, and still takes it daily to prevent recurrences of kidney failure. Dr. Hariachar maintains though, that not everyone will be helped by taking bicarbonate. He says that those patients who have difficulty excreting acids, even with dialysis using a bicarbonate dialysate bath, that, “oral bicarbonate makes all the difference.”

The Stomach, Pancreas and Kidneys Naturally Produce Sodium Bicarbonate Every Day

The exocrine section of sodium bicarbonate from the stomach and the pancreas have been greatly ignored in the treatment of diabetes and cancer even though its impairment is a well documented condition. The stomach and the pancreas is primarily responsible for the production of sodium bicarbonate necessary for normal alkalization of food and liquids ingested. Sodium bicarbonate is so important for protecting the kidney’s that even the kidneys get into the act of producing sodium bicarbonate.  We now know the common denominator between hyponatremia, inflammation, edema, diabetes, kidney disease, and cancer is the lack of sodium and potassium bicarbonate or the body’s inability to produce sodium and potassium bicarbonate because of a lack of mineral salts in the diet. When the body is hit with reductions in sodium bicarbonate output by these three organs,’ acid conditions build up and then the entire body physiology begins to change from a state of oxygenation to fermentation. Likewise when acid build-up outstrips these organs normal sodium bicarbonate capacity, cellular, tissue, glandular and organ deterioration begins.

The stomach, pancreas and the kidneys alone produce about five hundred
grams (about one pound) of sodium and/or potassium bicarbonate per day in an attempt to neutralize dietary and/or metabolic acid in the blood and interstitial fluids that surround the body cells.

The stomach, pancreas and the kidneys monitor and control the acidity or “acid-base” (pH) balance of the blood and tissues. If the blood and tissues are too acidic, the stomach and/or the kidney’s make sodium bicarbonate to restore the blood and tissue pH back to a delicate pH balance of 7.365. If the blood or tissues are too alkaline, then the kidney excretes sodium bicarbonate into the urine to restore the 7.365 alkaline balance. Acid-base balance is the net result of two processes, first, the removal of sodium bicarbonate subsequent to hydrogen ion production from the metabolism or dietary constituents; second, the synthesis of “new” sodium bicarbonate by the stomach and/or the  kidney’s.(13)  The stomach and kidneys pull salt, water and carbon dioxide from the blood to make sodium bicarbonate to maintain the alkaline design of the body during all functions of the body from the ingestion of food or drink to exercise.  The chemical formula is as follows:  NaCl + H2O + CO2 = NaHCO3 + HCL.  The waste product of sodium bicarbonate is hydrochloric acid which is eliminated by kidneys as an acidic excretion of the urine.
It is considered that normal adults eating ordinary Western diets have chronic, low-grade acidosis which increases with age. This excess acid, or acidosis, is considered to contribute to many diseases and to contribute to the aging or rotting process. Acidosis occurs often when the body cannot produce enough sodium bicarbonate ions (or other alkaline compounds) to neutralize the acids in the body formed from metabolism and eating and drinking highly acid foods and drinks like chicken, pork, beef, dairy products, coffee, tea, alcohol, chocolate, soft drinks, just to name a few.  We are also testing bottled mineral water and finding that these waters are acidic and may contribute to overall tissue acidosis.
Acid-buffering by means of base supplementation (The pH Miracle pHour Salts) of sodium bicarbonate is one of the major roles of dialysis. Sodium bicarbonate concentration in the dialysate (solution containing water and chemicals (electrolytes) that passes through the artificial kidney to remove excess fluids and wastes from the blood, also called “bath.”) should be personalized in order to reach a midweek pre-dialysis serum sodium bicarbonate concentration of 22 mmol/l.(14)  Use of sodium bicarbonate in dialysate has been shown in studies to better control some metabolic aspects and to improve both treatment tolerance and patients’ life quality.  Sodium bicarbonate dialysis, unlike acetate-free biofiltration, triggers mediators of inflammation and apoptosis.(15)

One of the main reasons we become over-acid is from over-consumption of animal protein, dairy products, high sugar fruit, grains, alcohol, coffee, tea, chocolate, soft drinks and over-exercise or under-exercise. Eating meat and dairy products may increase the risk of prostate cancer, research suggests.(16) We would find the same for breast and other cancers as well metastatic cancers.(17) Conversely mineral deficiencies are another reason and when you combine high protein intake with decreasing intake of alkaline minerals you have a dis-ease in the making through lowering of pH into highly acidic conditions. When protein breaks down in our bodies they break into strong acids, such as, nitric, uric, sulphuric and phosphoric acid.

Unless a treatment actually removes acidic toxins  from the body and increases oxygen, water, and nutrients most medical interventions come to naught.

These metabolic and dietary acids must be excreted by the kidney’s because they contain sulfur, phosphorus, and/or nitrogen which cannot break down into water and carbon dioxide to be eliminated as weak acids. In their passage through the kidney’s these strong acids of ntric, sulphuric, phosphoric and uric acid must take a basic mineral with them because in this way they are converted into their neutral salts and don’t burn or destroy the kidney’s on their way out. This would happen if these strong acids were excreted in their free acidic form.

Substituting a sodium bicarbonate solution for saline
infusion prior to administration of radiocontrast
material seems to 
reduce the incidence of nephropathy.(18)
Dr. Thomas P. Kennedy
American Medical Association

Sodium and potassoum bicarbonate ions neutralize the acids that cause chronic inflammatory reactions. Hence, sodium and potassium bicarbonate are of benefit in the treatment of a range of chronic inflammatory and autoimmune diseases. Sodium and potassium bicarbonate are well-studied and used salts with known effects. Sodium and potassium bicarbonate are effective in treating poisonings or overdoses from many chemicals and pharmaceutical drugs by negating their cardiotoxic and neurotoxic effects.(19)  It is the main reason it is used by orthodox oncology – to mitigate the highly toxic effects of chemotherapy.

Sodium and potassium bicarbonates possess the property of absorbing heavy metals, dioxins and furans. Comparison of cancer tissue with
healthy tissue from the same person shows that the cancer tissue
has a much higher concentration of toxic chemicals, pesticides, etc.

Sodium and potassium bicarbonate intravenous infusions are indicated in the treatment of metabolic acidosis, which may occur in severe renal disease, uncontrolled diabetes, and circulatory insufficiency due to shock or severe dehydration, extracorporeal circulation of blood, cardiac arrest, tonsillar herniation due to cerebral edema, severe primary lactic acidosis and hyponatremia due to excessive or over-exercise.  During heavy exercise, if the the resulting lactic acid is not adsorbed by the collagen fibers, the specific acid catchers of the body, the blood pH will drop and the body will go into a coma and the person will die.

The total collection of these fibers is the largest organ of the body called SCHADE, the colloidal connective tissue organ. NO liquid exchange occurs between the blood and the parenchyma cells, or in reverse, unless it passes through this connective tissue organ. This organ connects and holds everything in our bodies in place. This organ is composed of ligaments, tendons, sinew, and the finer fibers that become the scaffolding that holds every single cell in our bodies in place. When acids are stored in this organ, which includes the muscles, inflammation or edema and pain develop. The production of lactic acid is increased with excessive exercise and the ingestion of milk, cheese, yogurt, butter, ice cream, high sugar fruit and starchy root vegetables like potatoes.

That is why I have stated, “acid is pain and pain is acid or acid is edema and edema is pain”.  You cannot have one without the other. This is the beginning of latent tissue acidosis leading to irritation, inflammation, edema and degeneration of the cells, tissues and organs and eventual or sudden death.  It is why we are seeing so many amateur and professional atheletes pass out and die on the playing fields.  Metabolic, respiratory and gastrointestinal acids can and do kill and death can be overted by simply maintaining the alkaline design of the body fluids with protective hydration of alkaine sodium bicarbonate fluids.

The acid/alkaline balance is one of the most overlooked aspects of diagnostic medicine. In general, the world population is heavily acidic, excepting alkalarian vegans (those who ingest raw, organic green fruit, vegetables, mineral salts, alkaline water and unsaturated seed and nut oils), and even their bodies have to face increasing levels of environmental toxic exposure, which may contribute to an acidic pH condition of the blood and then tissues.

With over 30 years of research and testing over 100,000 individual samples of blood and over 100,000 samples of urine and saliva, I have come to the conclusion that the human body is an acidic producing organism by function – yet, it is an alkaline organism by design. Eating animal protein, especially meat and cheese, sugar, fermented foods, starchy foods like potatoes, acidic water, alcohol, coffee, tea, chocolate,  and excessive exercise or under-exercise, obsessive behaviors, lack of rest, lack of sunshine, and emotional stress are deadly acidic lifestyle choices.

All enervation, under-performance, sensitivity, irritation, inflammation, edema, catarrh, induration, ulcerations, degeneration, aging and cancerous conditions are caused by a four letter word – ACID, which is an acronym which stands for:

A = acidic food and drink, attitudes and activities,
C = compromised internal acidic environment,
I = illness and dis-ease, and,
D = desire for more acidic foods, drinks, attitudes and activities, and the cycle repeats itself.[20]

We ingest acidic medicines to lessen the symptoms of our illness. We stimulate the body with unhealthy forms of energy providing quick, often temporary relief from our symptoms which begins the cycle all over again creating a very powerful pattern of poor health and dis-ease.

Conclusion

The pH Alkalizing Lifestyle and Diet is a low acid producing diet and lifestyle that focuses on the foundational principal that the body is alkaline by design and yet acidic by function. This makes this program the ultimate program for preventing and reversing aging and the onset of sickness and disease. I would say that the pH Alkalizing Lifestyle and Diet is the perfect diet and lifestyle for a longer healthier life.(20)

References

1. Gamba, G., “Bicarbonate therapy in severe diabetic ketoacidosis. A double blind, randomized, placebo controlled trial.” (Rev Invest Clin 1991 Jul-Sep;43(3):234-8). Miyares Gom ez A. in “Diabetic ketoacidosis in childhood: the first day of treatment.” (An Esp Pediatr 1989 Apr;30(4):279-83)

2. Levy, M.M., “An evidence-based evaluation of the use of sodium bicarbonate during cardiopulmonary resuscitation.” (Crit Care Clin 1998 Jul;14(3):457-83). Vukmir, R.B., Sodium bicarbonate in cardiac arrest: a reappraisal (Am J Emerg Med 1996 Mar;14(2):192-206). Bar-Joseph, G., “Clinical use of sodium bicarbonate during cardiopulmonary resuscitation–is it used sensibly?” (Resuscitation 2002 Jul;54(1):47-55).

3. Zhang. L., “Perhydrit and bicarbonate improve maternal gases and acid-base status during the second stage of labor.” Department of Obstetrics and Gynecology, Xiangya Hospital, Hunan Medical University, Changsha 410008. Maeda, Y., “Perioperative administration of bicarbonated solution to a patient with mitochondrial encephalomyopathy.” (Masui 2001 Mar;50(3):299-303).

4. Avdic. E., “Bicarbonate versus acetate hemodialysis: effects on the acid-base status.” (Med Arh 2001;55(4):231-3).

5. Feriani, M., “Randomized long-term evaluation of bicarbonate-buffered CAPD solution.” (Kidney Int 1998 Nov;54(5):1731-8).

6. Vrijlandt, P.J., odium bicarbonate infusion for intoxication with tricyclic antidepressives: recommended inspite of lack of scientific evidence. Ned Tijdschr Geneeskd 2001 Sep 1;145(35):1686-9). Knudsen, K., ñ€ƓEpinephrine and sodium bicarbonate independently and additively increase survival in experimental amitriptyline poisoning.” (Crit Car e Med 1997 Apr;25(4):669-74).

7. Silomon, M., “Effect of sodium bicarbonate infusion on hepatocyte Ca2+ overload during resuscitation from hemorrhagic shock.” (Resuscitation 1998 Apr;37(1):27-32). Mariano, F., “Insufficient correction of blood bicarbonate levels in biguanide lactic acidosis treated with CVVH and bicarbonate replacement fluids.” (Minerva Urol Nefrol 1997 Sep;49(3):133-6).

8. Dement’eva, I.I., “Calculation of the dose of sodium bicarbonate in the treatment of metabolic acidosis in surgery with and deep hypothermic circulatory arresta.” (Anesteziol Reanimatol 1997 Sep-Oct;(5):42-4).

9. “I believe that, conservatively, 15 to 20 percent of all cancer is caused by infections; however, the number could be larger — maybe double,” (Dr. Andrew Dannenberg, Director of the Cancer Center at New York-Presbyterian Hospital/Weill Cornell Medical Center.”) Dr. Dannennberg made the remarks in a speech in December 2007 at the annual international conference of the American Association for Cancer Research.

10. A sexually transmitted virus that causes cervical cancer is also to blame for half of all cases of cancer of the penis.

11.  www.nelm.nhs.uk/en/NeLM-Area/News/2009—July/20/
Bicarbonate-supplementation-may-slow-renal-decline-in-chronic-kidney-disease/

12. Origin of the Bicarbonate Stimulation of Torpedo Electric Organ Synaptic Vesicle ATPase. Joan E. Rothlein  1 Stanley M. Parsons. Department of Chemistry and the Marine Science Institute, University of California, Santa Barbara, Santa Barbara, California, U.S.A.

13. Levine DZ, Jacobson HR: The regulation of renal acid secretion: New observations from studies of distal nephron segments. Kidney Int 29:1099–1109, 1986

14.  www.uptodate.com/patients/content/abstract.do?topicKey=~G/p55S8w8sQDwqG&refNum=28

15.  www.ncbi.nlm.nih.gov/pubmed/16523427

16.  news.bbc.co.uk/2/hi/health/7655405.stm

17.  Cancer Res. 2009 Mar 15;69(6):2260-8. Epub 2009 Mar 10.
Bicarbonate increases tumor pH and inhibits spontaneous metastases.
Robey IF, Baggett BK, Kirkpatrick ND, Roe DJ, Dosescu J, Sloane BF, Hashim AI, Morse DL, Raghunand N, Gatenby RA, Gillies RJ. Source: Arizona Cancer Center, University of Arizona, Tucson, Arizona, USA

18.  JAMA 2004;291:2328-2334,2376-2377.www.urotoday.com/56/browse_categories/renal_transplantation_vascular_disease/
sodium_bicarbonate_may_prevent_radiocontrastinduced_renal_injury.html

19. These include, Benzotropines (valium) cyclic antidepressants (amytriptayine), organophosphates, methanol (Methyl alcohol is a cheap and potent adulterant of illicit liquors) Diphenhydramine (Benedryl), Beta blockers (propanalol) Barbiturates, and Salicylates (Aspirin).   Poisoning by drugs that block voltage-gated sodium channels produces intraventricular conduction defects, myocardial depression, bradycardia, and ventricular arrhythmias. Human and animal reports suggest that hypertonic sodium bicarbonate may be effective therapy for numerous agents possessing sodium channel blocking properties, including cocaine, quinidine, procainamide, flecainide, mexiletine, bupivacaine, and others.

20. www.phmiracle.com. Young.R.O., Young, S.R., The pH Miracle Revised and Updated, Hachett, 2010.

Pathological Blood Coagulation and the Mycotoxic Oxidative Stress Test

 Robert Young PhD

Naturopathic Practitioner – The pH Miracle Ti Sana Detox Medical Spa and Universal Medical Imaging Group

Abstract

Historical analysis suggests that conventional understandings of Disseminated Intravascular Coagulation (DIC) may be misguided; further examination may be necessary.  Here, a theoretical analysis provides an alternative explanation for DIC pathology; it is suggested that the cause and mechanics of DIC are largely due to the proliferation of several intravascular microforms and their associated metabolic toxic acidic waste products — Mycrozymian Acidic Toxins (MAT) and Exotoxic-Mycotoxic-Producing Microorganisms (EMPO).  The Mycotoxic Oxidative Stress Test (MOST) is presented here as an easy, inexpensive and non-invasive alternative to conventional measurements for the detection of intravascular  acidic toxins, DIC  and oxidative stress.

Introduction and Historical Perspective

More than 150 years ago, British physician T. W. Jones asked the question, “Why does the blood circulating in the vessels not coagulate?”[1]  though a general answer to this question is now obvious, the biochemical mechanisms involved in how the blood coagulates (clots) are complex and varied, and all the intricacies have not yet been explained. A. Trousseau, recognized that the blood of cancer patients is in a hyper-coagulable state in the process of coagulation, even while confined in the blood vessels.[2]  The name given to this discovery is still in use today, as “Trousseau’s Syndrome.”[2]  Early in his career, Rudolph Virchow, the Father of Pathology, was interested in thrombosis and embolism.  He speculated that intravascular blood could be altered so it would clot as a result of a stimulus too weak to clot normal blood.[3]  In 1856 Virchow delivered a lecture setting forth this concept.

Although the concept of partial clotting within vessels reaches back to the beginnings of modern medicine, much of the discovery of its biochemical mechanisms – the activation of clotting factors – has been left to chance.  The admission of a patient to the hospital with an unceplained bleeding disorder challenged researchers to discover the cause of hemorrhaging.  Analysis of blood from normal persons helped in the study of the patient with the blood disorder. A new clotting factor was hereby discovered which was missing from the  patient’s blood.  For this reason, several clotting factors have been named after the individuals in which they were missing: e.g., Christmas factor (factor IX)[4], Hageman factor (factor XII)[4].

In this article, the causes of pathological (intravascular) clotting will be described, as will various methods of detecting this condition, especially a blood test I call the Mycotoxin Oxidative Stress Test (MOST).

The Mechanics of Blood Coagulation

Blood clotting is a highly detailed chemical-mechanism involving many distinct components.  The problem for the hematologist hs been to understand it at the biochemical level.  Undoubtedly, efforts to fully understand blood clotting will continue for many more years.

Recalling Antione Bechamp’s[8] and Gunther Enderlein’s[9] research into the sub cellular living elements and combining this with what is known of colloidal flocculation[6], it is suggested that the clotting of blood begins with the end-linking (polymerizing) of the fundamental protein unit called by Bechamp the microzyma[8].  A chain of these living units constitutes fibrinogen, which is still dispersed 9micro-hetergenous0 in the blood, and it may or may not be further processed.  If processing continues, it will be either by continued end-linking or by cross-linking.  End-linked fibrinogen is referred to here as fibrin monomer, which I have suggested is a repair protein also dispersed in the blood. Due to a number of blood clotting factors, the process may continue until the excess fibrin monomer and/or until fibrin becomes excessively end-linked.

Cross-linking the polymerized strands to form a three-dimensional network results in what is called the hard clot (fibrin – the major protein of clotting blood).  Factor XIII, which instigates the forming of these blood networks. is always present but latent in the blood, and must be activated before the formation can occur.  Persons who are producing fibrin monomer or excessively linked fibrinogen are said to be in a hyper-coagulable state, while those having diminished  ability to form clots are in a hypo-coagulated state.  It is the activation of the colloidal clotting factors which is so complex.  Blood clotting may occur through many pathways and be initiated by many different stimuli.  Regardless of initiation factors, the process is a sequence of events in which the activation of one factor triggers another, until, after a series of discrete steps, fibrin is formed.

When blood is clotted prematurely, and the factors involved are consumed (incorporated into) the body recognizes a deficiency of clotting agents and generates more.  Thus, people with a tendency to clot excessively will alternate between a hyper coagulable state and a hypo-coagulatable state.  When in the hypo coagulated state, such people hemorrhage until the deficient clotting factors are replaced.[4]  When only fibrin monomer or excessively linked fibrinogen is formed (no cross-linking), it is quite subtle and may go undetected.  It may be detected by a change in blood viscosity (sedimentation rate), by the Mycotoxic Oxidative Stress Test (described later), or by other more subtle means.  If strands of fibrinogen are cross-linked, however, a suggicient amount of insoluble precipitate of fires may result, and these can be detected microscopically using a phase contrast and dark-field microscopy in prepared slides of fresh tissue or blood.  An excessive formation of fibrin leads to  an impairment in circulation, and eventual organ failure usually results.[5]

With this background, we are in a position to consider a standard medical term: disseminated intravascular coagultion (DIC).[6]  This term encompasses the hyper coagulable state, i refer to as pathological blood coagulation which consists of both insoluble and excess dispersed polymers of colloidal proteins.

Key Ingredients of Pathological Blood Coagulation

Before discussing DIC in more detail, it si necessary to introduce its fur important ingredients according to this view – mycotoxins, endotoxins, exotoxins, and tissue factor.  Any of these elements, or any combination of them, can play a major role in initiating unwanted DIC.[6]  However, mycotoxins or the acids from yeast have been found to be the underlying element which instigates and intensifies the participation of the other three.[6]  Each will now be described in turn and brought into the clotting picture.

(Micrograph 1: left, shows normal hyper-coagulated blood in a healthy blood clot sample and right, hypo coagulated blood in an unhealthy blood clot sample)

Mycotoxins and Metabolism by Fermentation

As discussed in the main text of my published book, Sick and Tired book[7 ]. acidification of blood and body tissues and organs and the accompanying lack of oxygen lead to pathological metabolic fermentation, which is carried out primarily by yeast and mold.  Such pathological microorganisms, or their precursors, ar inherent to the human body and to all higher organisms.  Their precursors according to Bechamp, the microzymas, carry on a nominal and homeostatic fermentation themselves. under healthy conditions.[8]  The primary function of yeast and mold is to decompose the body upon the death of the animal or human organism.  Their premature overgrowth indicates a biochemical environment akin to death.  During pathological metabolic fermentation, high concentrations of several acidic substances called mycotoxins are created.  They are highly damaging, always acidic, metabolic products.  If not immediately buffered by specific antioxidants, such as hydrogen peroxide and the hydroxyl free-radical, mycotoxins can seriously disrupt the physiology by disrupting normal metabolism and by penetrating blood and body cells and poisoning them.  As will be seen, they interact with many of the mechanisms for DIC in various pathological symptomologies.

In my published article called The Finger on the Magic of Life: Antoine Bechamp, 19th Century Genius (1816-1908),  I discuss pleomorphism in some detail.[7] Understanding this phenomenon – the rapid evolution of microorganisms across traditional taxonomic  lines is helpful in getting a complete picture of DIC.  Briefly stated, collodial living microzymas evolve intracellularly into more complex forms (microorganisms), beginning with a healthy primitive stage comprising of repair proteins.  As the disease condition worsens, morbid intermediate forms (filterable bacteria or viruses, cell-wall deficient forms and full bacteria) develop from repair proteins, or directly from microzymas.  A third macrostage comprises the commonly recognized culminate microorganisms which are yeast, fungus to mold.  In terms of pleomorphism, all of these microorganisms represent a single family of variously functioning forms.[8]  The culminate forms produce the lions share of acids, which are mycotoxins and the primary focus of my research.[7][8][9]  For convenience, bacteria, yeast, fungus and mold that produce acidic metabolic wastes and protein cellular fragments called exotoins, endotoxins and mycotoxins will here be referred to collectively ash EMPO, or exotoxic, mycotoxic-producing microorganisms.

What follows is a shortened description or the description and origin of several exotoxins and mycotoxins, referred to collectively microzymian acidic toxins of MAT, which are involved in the processes leading to DIC.  The bio-effects, or the pathology of cellular fermentation, of these toxic metabolites are know as mycotic illness, mycotoxicosis, or mycotoxic stress as seen in the MOST and described and published by Dr. Bolin in the 1940’s.[10]

One such metabolic product is acetyl aldehyde, which is formed by  cellular breakdown of food, especially carbohydrate and the birth of  EMPO.  Acetyl aldehyde can also break down into a secondary substance know as ethyl alcohol.  Although acetyl aldehyde presents an immediate hazard to health and well-being, nature has provided a means of buffering of neutralizing this acidic by-product of cellular digestion and fermentation almost as soon as it is created.[11] The controls of acetyl aldehyde (and ethyl alcohol) are the sulfur amino acids, cysteine, taurine, methionine and the peptide glutathione which is found in red blood cells and almost all cells utilizing oxygen.[12]  In an attempt to buffer or neutralize MAT, the body will also bind or chelate both fats and minerals to them.[12]

Another member of the MAT family is uric acid, which is formed by the digestion of protein and the creation of EMPO.[13]  Uric acid can also break down into secondary substance, on of which is alloxan.[14] This has been shown to damage the insulin-producing pancreatic beta cells leading to diabetes [Refer to Tables 1 and 2]

A shortage of alkalizing nutrients or an excess of MAT initi­ates an immune response in which a special class of free radicals which I call microzymian oxidative buffering species (MOBS) are released.[15] These oxygen metabolites carry unpaired electrons and are intended to disrupt bacteria, yeast, fungus and mold, and buffer exotoxins, endotoxins, and mycotoxins. Current medical savants believe that they can disrupt just about any­thing they contact, including healthy cells and tissue: this is not accurate. The fact is that MOBS carriers a nega­tive surface-charge and repel healthy cells, which also have a negative surface-charge. [16] It is the positively surface-charged bacteria, yeast/fungus, mold, exotoxins, endotoxins, and myco­toxins that MOBS bind too.[17]  This aspect gives some insight into autoimmune phenomena, which are not, as is often maintained, the result of an overburdened immune system. They result either as a side-effect of the immune system’s attempt to remove foreign or toxic ele­ments, or as a direct attempt by the immune system to remove cells or tissue rendered useless or disturb­ing to the body by MAT.

In every degenerative symptomatology I have studied, I have found excessive MAT and MOBS (see Tables 1-3). Some of these degenerative symptoms and their underlying disease conditions, including cancer are described in my recently published paper on a deficiency on alkaline nutrition and cancer. [15] But the fact that myco­toxins cause harm to humans and other animals is purely a secondary effect, since, as noted, the prima­ry function of the microorganism is not to cause illness. We know from the fossil record that pleomorphic microforms existed long before animals.[19] In fact, humans and animals developed in terms of micro­organisms.[20] The reverse, however, is not true. Since micro­organisms appeared first in the developmental sequence, they are not physiologically aware of humans and animals. There is much evidence that human and animal physiologies are highly aware of, and respond to MAT – these acidic compounds signaling the presence of bacteria, yeast, fungi and/or mold or  EMPO.[21].

Endotoxins

Also involved in the process leading to DIC are endotoxins, substances endogenous to symptogenic (i.e., “pathogenic” in orthodox terms) bacteria. Endotoxins are a family of related substances having certain common characteristics, but differing from one bacterial form (or strain) to another. Endotoxins are lipopolysaccharides (LPS). LPS form a widely diversified group because of (1) the number of long- chain fatty acids composing lipids; (2) the number of individual sugars as well as their modes of linkage to one another; (3) the branching of sugar chains; and (4) the number of possible arrangements of these units. Endotoxins also contain proteins, further com­pounding the structural diversity.[22]

One theory on endotoxin states that its purpose is to act as a semi-permeable membrane for the bac­terium, limiting and regulating substances entering the organism.[22] Endotoxin resides solely on or near the interior surface of the cell membrane and is shed into the surrounding medium only upon the death of the bacterium. Thus, as these microforms die off, or are lysed by bodily activity, endotoxin is released. (This fact may well be an explanation for the Herxheimer reaction, in which a patient becomes worse following the administration of toxic drugs or other forms of treatment that drastically alter the associated organ­ism.[23]) Another endotoxin theory states that LPS are a constituent of the membrane, and as the organism grows, endotoxin fragments are repeatedly sloughed off into the medium. This phenomenon has been observed in the digestive tract.[24] Since bacterial translocation into the blood is not only possible but common where epithelial hyperpermeability exists, one can assume that the process will continue there. Both theories may be correct if we think of the first one as true of “adult” forms, and the second as true of newly developed and expanding ones.

Basic to the structure of an endotoxin is the lipid common to all forms, designated lipid A, to which is attached a “core” polysaccharide, identical for large groups of bacteria. To the core polysaccharide is attached the O-antigen, consisting of various lengths of polysaccharide chains which are chemically unique for each type of organism and LPS. These chains pro­vide endotoxin specificity.[25] Experiments conducted over many years indicate that most, if not all, of the toxic effects of an endotoxin may be attributed to the lipid portion, and it is sometimes used per se in experiments rather than the entire molecule.[26] An important additional feature of lipid A is its phos­phate content. Each phosphate group carries a nega­tive charge, and since lipid A is a rather large mole­cule, it provides, essentially, a negatively charged sur­face. The importance of this will be seen shortly.

Exotoxins

These are the metabolic excretions of bacteria. While endotoxin’s ongoing effect is, in a manner of speaking, in the background, exotoxins, like myco­toxins, present a double-edged sword. Not only do they initiate DIC, but they produce, or influence the body to produce, the various and numerous infec­tious symptomatologies, such as typhoid fever, diph­theria, etc. (See “Vaccination Reconsidered” in Section 4 of the Appendix of Sick and Tired for details on the action of diphtheria toxin.)[7] By comparison, mycotoxins not only initiate DIC, but there is much evidence to sug­gest that they produce, or influence the body to pro­duce, degenerative symptomatologies, such as arthri­tis, diabetes, etc., and cancer and AIDS as well.

Tissue Factor

Crucial to the understanding of DIC is recogni­tion of the role of tissue factor (TF), formerly known as thromboplastin. This transmembrane lipoprotein exists on the surface of platelets, vas­cular endothelial cells, leukocytes, monocytes, and most cells producing EMPO.[27] It plays a major role in several biochemical mechanisms leading to DIC.

TF is the primary cell-bound initiator of the blood coagulation cascade. Its gene is activated in wound healing and other conditions. By itself it is capable of initiating clotting, but also becomes active when complexed with factor VII or activated factor VII (Vila).[28] TF has been described as the receptor for factor VII because of the close association between the two proteins and because it causes a shape change (conformational) in factor VII, allowing it to attain activity. Both factor Vila and the TF/VII com­plex activate factors IX and X, which initiate the clotting cascade and the formation of thrombin.[29]

Development of Disseminated
Intravascular Coagulation
(DIC)

DIC Induced by MAT and Tissue Factor

An infusion of toxins into the blood has a direct effect on TF gene expression in leukocytes. Contact of MAT, endotoxins (lipid A), or exotoxins with leukocytes, activates proteins that bind to DNA nucleotide sequences, thereby activating the TF gene.[30] (See Tables 4-6.)

Endothelial cells damaged in culture by exotoxins, endotoxins, or mycotoxins attract polymorphonuclear leukocytes (PMNs), which adhere to the damaged cells. Once the leukocytes are bound, they can still have their TF gene activated if it hasn’t yet occurred, and they may release MOBS in response to toxins and to organisms of disease, possibly creating further dis­turbances. (Cellular disorganization then releases acti­vating proteins into the blood, which is discussed in more detail later.) Research shows that exotoxic and mycotoxic stress resulting in bound PMNs can be blocked by “antioxidants.”[31] These might better be called anti-exotoxins or antimycotoxins. Both observa­tion and study have led the author to conclude that cellular disorganization is initiated and primarily caused by fermentation pathology, not, as is the cur­rent belief, by the MOBS, or free radicals, generated to destroy toxins and microorganisms. MOBS or free radicals, because of their negative charge, are released to chelate or bind EMPO and MAT. It is suggested by current savants that free radical tissue damage is the secondary, “shotgun” effect of intense immune response to EMPO toxification and MAT-damaged cells. This could not be the case since healthy cells or their membranes carry a negative charge and would resist any electromagnetic attraction because of simi­lar charge. The concentration and instability of MAT generated in a compromised terrain, as opposed to the fleeting existence of free radicals, especially exoge­nous ones, also lead to this conclusion.

Endothelial cells grown in culture can be induced to express tissue factor. In one experiment, no procoagulant activity could be detected in the absence of toxins. However, the addition of mycotoxins from Aspergillus niger or Micrococcus neoformas (Mucor racemosus Fresen) resulted in procoagulant activity which reached a maximum in four to six hours and was dose-dependent. The same experiment was applied using E. coli and Salmonella enteritidis endo­toxin with a similar result.[32] A single intravenous injection of a mycotoxin from Aspergillus niger into experimental animals resulted in circulating endothelial cells within five minutes. In other exper­iments with the mycotoxin, detachment of endothe­lial cells from the basement membrane was noted.[33] (See Table 8.)

Removal of endothelial cells has dire conse­quences from two standpoints: First, the surface of these cells is covered with a specific prostaglandin (PGI2) known as prostacyclin. If blood contacts a surface not covered with PGI2, it will clot. For example, surfaces devoid of this prostaglandin are formed whenever a vessel is cut or punctured. An abrasion or other injury may also expose a surface on which PGI2 is lacking. The removal of endothelial cells by exotoxins or mycotoxins creates a surface devoid of PGI2, leading to blood clotting (see Table 7). Secondly, disorganization of endothelial cells cre­ates increased levels of EMPO and MAT which are attracted to an exposed surface (basement mem­brane) which expresses a negative charge. This also leads to clotting.

DIC Induced by Electrostatic Attraction

It was discovered in 1964 that blood will clot sim­ply from contacting a negatively charged surface.[34] Previously it was believed that the clotting process comprised a cascade of enzyme activity in which one activated the next, etc. The discovery that blood could be clotted simply by contacting a negatively charged surface ruled out the purely enzyme hypoth­esis. Only some of the known clotting factors have been shown to be enzymes.[35] As a result of this sur­prising discovery, detailed research was conducted in an attempt to describe the process. In some experi­ments, the negatively charged surfaces of selected, finely divided, inorganic crystals, including alu­minum oxide, barium sulfate, jeweler’s rouge, quartz, and titanium oxide, were considered.[36]

The clotting factor eventually shown to be activat­ed when whole blood contacted negatively charged surfaces was factor XII, also known as the Hageman factor. This is a positively charged protein migrating in an electric field (electrophoresis) toward the anode.[37] It is believed that factor XII is normally in the shape of a hairpin which binds to the negatively charged sur­face at the bend. Electrostatic attraction forces the two arms to lie flat on the surface, thereby exposing the inner faces and activating the molecule.

It was discovered that if the negatively charged particles were smaller than the clotting factor itself, activation was minimal. Or, if the concentration of clotting factor was too great, there was little or no activation.[38] Both of these observations indicated that the process was one of electrostatic attraction between the negatively charged surface and the clot­ting factor, which is a “basic” protein, that is, posi­tively charged.[39]

Activation of factor XII allows the activation of factor XI, which then activates factor IX. Thus, the blood clotting cascade continues to the formation of fibrin in the normal manner.[40] However, due to a series of activations begun by contact of factor XII with a negatively charged surface, trace amounts of factor Xa also show up in the blood. Factor VII is activated to Vila by factor Xa. Factor Vila then acti­vates factors IX and X, leading to the formation of thrombin. Factor Xa, with co-factor Va, continues the clotting cascade until fibrinogen is activated, leading to fibrin formation.[41] (See Table 5.)

As discussed earlier in terms of prostacyclin, beneath endothelial cells is another surface—the basement membrane. Called the extracellular matrix, it is a thin, continuous net of specialized tis­sue between endothelial cells and the underlying connective tissue. It has four or more main con­stituents, including proteoglycans (protein/polysac- charide).[42] The removal of endothelial cells by’MAT exposes this membrane, which is negatively charged by virtue of its sulfonated polysaccharides in the pro­teoglycans. This brings a reduced negatively charged surface into direct contact with the blood, which activates factor XII and the clotting cascade.[43]The positively charged toxic components of MAT also activate factor XII, as do disturbed disorganized cells, yeast/fungus cells, moldy cells, and the phos­phate groups in the lipid A component of endotoxin. (See Tables 2-5.)

To summarize this section, exotoxic, mycotoxic, and oxidative stress resulting from the overgrowth of bacteria, yeast/fungus, and then mold, has multiple actions, all leading to disseminated intravascular coagulation:

MAT activation of tissue factor gene in leukocytes; subsequent activation of factors VII, IX, and X, resulting in the blood clotting cascade.

MAT activation of tissue factor gene in endothelial cells, again leading to the clotting cascade.

MAT damage to endothelial cells, resulting in neu­trophil attraction, with TF gene activation and generation of MOBS, which, in turn, neutralize MAT, protecting healthy endothelial cells or the basement membrane and supporting the janitorial services of the leukocytes.

Removal of negatively charged endothelial cells by positively charged exotoxins, endotoxins, and mycotoxins, creating a surface devoid of PGI2, also exposes the negatively charged basement membrane, leading to the activation of factor XII and initiation of the clotting cascade. Positively charged components of EMPO, exotoxins and mycotoxins, and several other elements, including the lipid A component of bacterial endotoxin, also activate factor XII and the clotting cascade.

Endothelial Cells as Antithrombotics or Procoagulants

Normal, resting (unstimulated) endothelial cells show antithrombotic activity in several ways: (1) by the inhibition of prostacyclin (platelet adhesion and aggregation); (2) the inhibition of thrombin genera­tion; and (3) the activation of the fibrinolytic system, leading to clot lysis.[45] We will take a brief look at the thrombin aspect.

On the surface of endothelial cells is a protein called thrombomodulin, which acts as a receptor for thrombin. When bound to thrombomodulin, throm­bin can activate protein C. Activated protein C then catalyzes the proteolytic cleavage of factors Va and Vila, thereby destroying their participation in blood clotting. Thus thrombin, which normally activates fib­rinogen, plays an opposite role in this case and inhibits the clotting process.[46,47] (See Table 7.)

On the other side of the coin, the endothelial cell becomes a procoagulant agent when acted on by cer­tain lymphokines, such as interleukin-1. Not only can interleukin-1 induce TF gene expression, but it also suppresses transcription of the thrombomodulin gene in endothelial cells. As in other situations, the lymphokine-activated endothelial cell expresses TF on its surface as a result of TF gene activation. This leads to the production of thrombin and the trigger­ing of the blood clotting cascade.[48] (See Table 5.) Many lymphokines also stimulate adhesion of leuko­cytes to endothelial cells damaged by MAT, resulting in recycling of the cells by MOBS, as described later.

DIC Induced by Intracellular Exotoxic, Mycotoxic, Oxidative Stress by Bacteria, Yeast/Fungus and/or Mold

Any cell which has gone from an oxidative to a fer­mentative state can biochemically cause macrophage production of the lymphokine tumor necrosis factor (TNF). This protein has been shown to activate the gene for TF in fermenting cells, which are so behaved due to morbid evolution of bacteria, yeast/fungus, and then mold.[49,50] In the author’s view, a cell having been switched entirely to fermentation metabolism as a result of a physical or emotional disturbance of that cell, is what constitutes cancer (see Tables 5 and 13). (One might argue that this definition does not fit all “forms” of cancer, such as leukemia, for example. This is because leukemia is not cancer, but an immune response to the rise in EMPO and MAT in the body, and a relatively easy compensation to correct.)

The surface of many disorganizing or fermented cells (cancer cells) is characterized by small projec­tions in the plasma membrane which pinch off, becoming free vesicles containing toxins as well as TF complexed with factor VII. These vesicles can aggre­gate and/or lodge anywhere, ultimately releasing their contents. Also, the presence of excessive amounts of TF/factor VII complexes on the surface of fermented cells allows the formation of a fibrin net around the cell and around the entire mass of cells (tumor). This seems to be an attempt by the body to encapsulate and contain the mass. However, fermented cells do escape from the primary fibrin net, perhaps due to some electromagnetic effect, and become free-float­ing in the circulation. They may thus lodge elsewhere and instigate the fermentation of other cells by fungal penetration or by poisoning them and provoking a morbid evolution of their inherent microzymas.

Because of the surrounding fibrin net, these mobi­lized fermenting cells are protected from collection by the immune system while in transit.[51,52] (See Table 4.) The blockage or dissolution of fibrin net forma­tion by an anticoagulant such as heparin allows freed, fermenting (metastasizing) cells to be dismantled by natural killer cells and other immune cells (see Tables 5, 12 and 13).

DIC Induced by MAT/EMPO and Immune System Response (Release of MOBS)

Unsaturated fatty acids are highly susceptible to EMPO as well as MAT. Linoleic acid, a long-chain fatty acid present in white cells, has 18 carbons and 2 unsaturations. Subjected to MAT, linoleic acid binds the exotoxin, endotoxin, or mycotoxin, there­by forming an epoxide at the first unsaturation.[53] Research has revealed that this compound, named leukotoxin, is highly disturbing to other cells. It caus­es platelet lysis, thereby releasing TF and initiating DIC.[54] (See Table 10.) The fact that MAT result in fermented fats lends further credence to the sugges­tion that the initial and primary degenerative damage to structures and substances in the body is caused by exotoxins and/or mycotoxins, and that damage by MOBS, or by other free radicals, is not possible.

Another mechanism leading to DIC is the release of a special glycoprotein, sialic acid, from the terminal ends of cell-membrane polysaccharides, where it is always found. Polysaccharides play a highly significant role in biochemical processes, with both enzymes and membrane receptors recognizing various groupings of specific sugars linked in highly specific ways.

Immediately preceding the release of sialic acid in the polysaccharide chain is the sugar galactose. The sialic acid/galactose arrangement is utilized as a biolog­ical indicator of cellular and molecular aging. As cells age, sialic acid is naturally expressed from the terminal ends of polysaccharides, thereby exposing galactose. A membrane-bound enzyme from the liver, galactose oxi­dase, recognizes galactose and eventually disorganizes it, disrupting cell function integrity and hastening demise. Aged red blood cells, which have expressed a significant amount of sialic acid, are removed from the blood by this process. (I theorize that the biological ter­rain may be at work in normal cell aging. That is, the rate at which sialic acid is expressed is determined by the levels of corrosive acids in the system and the body’s ability to remove them, although there are no doubt intracellular factors at work as well.)

I suggest from my years of  clinical research  that cellular breakdown is compounded by the fermentation of the galactose by the microzyma. This is a process that begins from within and not necessarily from without. Not only does this action create more sialic acid, it creates other toxic waste products such as acetic aldehyde, alcohol, uric acid, oxalic acid, etc. The increase in cellular disturbances and fermenta­tion of the galactose creates biochemical signals for more galactose oxidase. This leads to greater cellular disorganization and developmental morbidity, espe­cially in the red blood cells, and a rise in the level of detrital serum proteins, which encourages clotting. From this perspective, diabetes, arthritis, atheroscle­rosis and other symptomatologies become more clearly “degenerative” (see Tables 2-5, 12 and 13).

Fibrinogen is a rather elaborate protein having the structure of three beads on a string. Expressed on the end beads is sialic acid, which indicates the beginning of disorganization of the fibrinogen and a declining negative charge to the positive. Prior to the declining charge and the expression of sialic acid on the end beads, fibrinogen, which is negatively charged, will not polymerize the healthy blood due to mutual repulsion. However, fibrinogen will poly­merize to damaged cells, EMPO, MAT and other positively charged areas of the body for repair pur­poses. Thus, as more and more sialic acid is expressed, there will be a significant reduction in the charge of the fibrinogen, acting as the primary requirement for the polymerization of fibrinogen (hypercoagulable state). The resulting polymer, fib­rin monomer, is the protein chain used in the repair of cells and clotting of blood.[55] End-linking will take place after the release of sialic acid (positive charge) by whatever means.

With this background, it is interesting to note that blood taken from persons suffering from anxiety is expressing sialic acid from fibrinogen, and is halfway toward clotting. Hormones released during anxiety states are easily fermented, giving more momentum to MAT and thereby resulting in this important change in fibrinogen. It leads to a clotting pattern characteristic of anxiety stress, and is readily identi­fied in the MOST. As can be seen in this picture, the pattern is a “snowstorm” of protein polymeriza­tions measuring from 2 to 10 microns.

allergiesbefore

 

 

 

 

 

 

 

[Micrograph 2: An Anxiety Profile showing a ‘snowstorm’ of 2 to 10 micron protein polymerizations starting from the center of the clot and moving out towards the edge]

As mentioned earlier, despite the attempt by the body to neutralize EMPO and MAT, an excess will initiate the release of MOBS by immune cells. A major MOBS is superoxide, designated chemically as O 2. It may exist alone or be attached to another ele­ment, such as potassium (KO’2) or sulfur (SO). Again, however, nature has provided a means of pro­tecting healthy cells—their negative charge[1]. Another protection against superoxide is the enzyme superox­ide dismutase (SOD), also found in all healthy cells.

A second member of the MOBS family is hydro­gen peroxide (H202). This molecule is very unstable and tends to react rapidly with other biological mol­ecules, damaging them. The release of hydrogen per­oxide in the body is a response to the overgrowth of decompositional organisms in a declining pH (com­promised biological terrain). The control for healthy cells against hydrogen peroxide is their negative charge and the protective enzyme catalase, one of the most efficient enzymes known.

When leukocytes and other white blood cells are stimulated by the presence of bacteria, yeast/fungus and mold, they treat these organisms as foreign par­ticles to be eliminated. During and prior to phagocy­tosis, the foregoing oxidative cytotoxins, along with the hydroxyl radical (OH’), are generated and released specifically for neutralizing microforms or harmful substances. This release is referred to as an “oxidative burst.” As a result of fermentation and the production of exotoxins and mycotoxins that fer­ment galactose from cells, the immune system is activated. An oxidative burst is released to neutralize the morbid microforms and mycotoxicity.[56] Like other biological processes faced with constantly alarming situations, the continued release of MOBS can get out of control. This may damage endothelial cells, the basement membrane, or other body ele­ments, and this activates fibrinogen to fibrin monomer (repair protein), leading to DIC [see Table 9]. Interestingly, the white blood cells capable of neutralizing MAT through MOBS production are the same ones capable of phagocytosis, the process by which foreign matter, waste products and microor­ganisms are collected and dumped in the liver.[57]

To summarize this section, pathological microforms and their acids create DIC by a number of pathways:

Leukotoxin (linoleic acid bound to mycotoxin) is highly toxic to cells. It causes platelet lysis, there­by releasing TF and initiating DIC.

The expression or release of sialic acid residues from healthy cells that have been disturbed allows for the fermentation of galactose, creating exotox­ins and mycotoxins, biochemically activating galactose oxidase, which further disturbs and dis­organizes healthy cells. This cycle loads the blood with debris.

EMPO and MAT disturb fibrinogen, which releas­es sialic acid and reduces the charge, allowing it to polymerize into fibrin monomer and fibrin nets.

The presence of exotoxins, endotoxins, and myco­toxins and their poisoning of cells activates the immune system. White blood cells generate MOBS (e.g., superoxide [0′2] or hydrogen perox­ide [H202]). These substances bind to and neu­tralize EMPO and MAT. MOBS are repelled by healthy endothelial cells and the basement mem­brane because of their negative charge. Cellular disturbances and disorganization stimulate the generation of fibrin monomer for repair purposes, leading to DIC.

Detection of Disseminated Intravascular Coagulation

The Sonodot Analyzer

The Sonoclot Coagulation Analyzer provides a reaction-rate record of fibrin and clot formation with platelet interaction. An axially vibrating probe is immersed to a controlled depth in a 0.4 ml sample of blood. The viscous drag imposed upon the probe by the fluid is sensed by the transducer. The electronic circuitry quantifies the drag as a change in electrical output. The signal is transmitted to a chart recorder which provides a representation of the entire clot for­mation, clot contraction and clot lysis processes. The analyzer is extremely sensitive to minute changes in visco-elasticity and records fibrin formation at a very early stage. The Sonoclot has been evaluated scientif­ically and shown to provide an accurate measurement of the clotting process.[58,59]

One application of the Analyzer has been the development of a test to distinguish non-advanced breast cancer from tumors that are benign. The ratio­nale for the test is the hypercoagulable state seen in cancer patients (Trousseau’s Syndrome), resulting from the generation of TF by leukocytes (mono­cytes).[60] (See Table 4.)

Fibrin Degradation
Products and Fibrin Monomer

DIC can be seen as a two-step process. First, fib­rinogen, which is always present in the blood, is acti­vated by any of several mechanisms. This activation leads to an automatic polymerization (chain forma­tion) resulting in fibrin monomer. This is not apparent in a microscope unless the blood is allowed to clot, as in the MOST.[61,62] The second step is the precipitation or deposition of fibrin (hard clot) by several other mechanisms. One of these is the formation of cross­links through the action of factor XIII. Another such mechanism may be poor circulation in an organ already blocked by deposited fibrin. The deposition of precipitated fibrin may be detected microscopically in tissue sections and diagnosed as DIC.[62]

Because fibrin monomer is not readily detected, a chemical test for it is of immense value in diagnosing DIC. Research has indicated that its detection may be very useful in the early diagnosis of DIC and MAT.[63] There are three fundamental physiologic areas related to blood clotting: (1) the prevention of blood clotting, (2) the clotting of blood, and (3) the removal of clotted blood once it has formed.

Enzymes are present that are capable of removing (lysing) clotted blood, one of which is plasmin. Another enzyme, plasminogen, is always present in the blood, but is inactive as a proteolytic agent. Plasminogen acti­vator converts plasminogen to plasmin, which can degrade deposited fibrin. This process is not specific for fibrin, however, and other proteins may be affected. When fibrin is degraded (fibrinolysis), fibrin monomer, as well as several other products, are formed. Commercial kits are available for the analysis of fibrin degradation. This test is an indirect measure of the pres­ence of DIC and MAT.[64]

Other tests include:

Protamine Sulfate: Protamine sulfate is a heparin binder sometimes used in surgery for excessive bleed­ing. The test, which indicates fibrin strands and fibrin degradation products, is conducted in a test tube, with fibrin monomer and fibrin forming early and polymer­ization of fibrin degradation products occurring later.[65] Ethanol Gelation: A white precipitate is formed by the addition of ethanol to a solution in a test tube containing fibrin monomer as a degradation product of fibrin, indicating DIC and MAT.[66]

The Mycotoxic Oxidative Stress Test (MOST)

Up to now, blood chemistries have been the prima­ry mode of diagnosis or analysis for the presence of pathology. In the view presented here, the bright-field microscope, is used to easily and inexpensively reveal a disease state as reflected by changes in certain aspects of blood composition and clotting ability. DIC is char­acterized by the abnormal presence in the blood of fib­rin monomer. When allowed to clot, blood containing such an abnormal artifact will exhibit distortions of normal patterns. The presence in the blood of soluble fragments of the extracellular matrix and soluble fibronectin, as well as other factors, will also create abnormal blood clotting patterns as described below.

A small amount of blood from a fingertip is con­tacted with a microscope slide. A series of drops is allowed to dry and clot in a normal manner. Under the compound microscope, the pattern seen in healthy subjects is essentially the same—a dense mat of red areas interconnected by dark, irregular lines, completely filling the area of the drop. The blood of people under mycotoxic/oxidative stress exhibits a variety of characteristic patterns which deviate from nor­mal, but with one striking, common abnormality: “clear” or white areas, in which the fibrin net/red blood cell conglomerate is missing.

BowelCancerLive Blood Dried Blood_0166

 

 

 

 

 

 

 

 

[Micrograph 3; An abnormal clot with striking ‘clear’ or white areas or protein polymerization as seen in the hyper coagulated blood of a patient with lower bowel imbalances]

Why the fibrin net is missing may be understood from the following: Two peptides—A and B—in the central protein bead of the fibrinogen structure become bound in the cross-linking process. There are two ways this can be configured: (1) Thrombin is capable of activating peptides A and B, resulting in the formation of a polymer loosely held together only by hydrogen bonds; (2) With peptides A and B acti­vated normally, the resulting hard clot is insoluble, indicating that the peptides are linked by covalent bonds. The difference in bonds results from factor XIII, an enzyme which links the two fibrin strands with a glutamine-lysine peptide bond.

Additional research has shown that the release of sialic acid from fibrinogen inhibits the action of factor XIII, resulting in a soft, white clot. In addition, acetic aldehyde has been shown to inactivate factor XIII directly. The soft clotting, compounded by other polymeric aggregations (described below), results in clear areas in the dry specimens. In the opposite extreme, high serum levels of calcium, for the pur­pose of neutralizing MAT, activates factor XIII, lead­ing to excessive cross-linking of fibrin to form a clot harder than normal. This is reflected in the MOST pattern characteristic of definite hypercalcemia— that of a series of cracks in the clot radiating outward from the center, resembling the spokes of a wheel. High serum calcium is the body’s attempt to com­pensate for the acidity of mycotoxic stress by pulling this alkalizing mineral from bone into the blood. This demand creates endocrine stress in turn, because reabsorption of bone is mediated by parathormone (PTH). Therefore, this clotting pattern indicates cal­cium deficiency and thyroid/parathyroid imbalance.

calciumpattern

 

 

 

 

 

 

 

[Micrograph 4: A mineral deficiency or more specifically a calcium deficiency pattern associated with an imbalance of they thyroid and/or parathyroid}

Advanced research has shown that there are seven carbohydrate chains in fibrinogen (each terminated by sialic acid). A second action of factor XIII is to ferment a large amount of carbohydrate during clot­ting. Because carbohydrate is most often water solu­ble, the loss of this material undoubtedly adds to the insolubility of a clot, while pathological retention contributes to the softness of the abnormal clot.

Clinical experience demonstrates that the MOST is a reliable indicator of exotoxic and mycotoxic stress and, concurrently, of various disorganizing symptoma­tologies associated with fermentative and oxidative processes. As various cellular degradation occurs, the blood-borne phenomena which accompany such symptoms as diabetes, arthritis, heart attack, stroke, atherosclerosis and cancer show up in the MOST, often with sialic acid beads in the clear areas of poly­merized proteins. (Determination of the liberation of sialic acid from carbohydrate has been approved by the U.S. Food and Drug Administration as an accept­ed indicator for cancer, and is clinically available.)

sialicacid

[Micrograph 5: Sialic acid beads are seen inside the protein
polymerization of the hypocoagulated blood as black dots]

The extent and shape of the clear areas are reflec­tive of particular symptomatologies which have arisen from the way in which the disease condition manifests in a given individual. This observation is borne out by having the patient undergo appropriate alkalizing therapy. With success of treatment based on the patient’s freedom from symptoms, sense of well-being, and live blood exams discussed in the main text of Sick and Tired, Reclaim Your Inner Terrain, Appendix C,[7] repeated analysis with the MOST reveals a progressively improving clotting pattern.

[Micrographs 6 and 7: Medically diagnosed cancer patient with large polymerized protein pools (PPP) in the hypo-coagulated blood above. In the picture below PPP’s have significantly reduced in size and the blood is moving to a more hyper-coagulated state as a result of reducing acid loads with an alkaline lifestyle and diet (7, 70)]

Because of its very nature, the MOST is emi­nently suited to reveal and measure the presence in the blood of abnormal substances, clotting factors, and disorganization of cells due to an inverted way of living, eating, and thinking, which gives rise to MAT. The MOST indicates both the direct and indirect activity of MAT on blood clotting, endothelium, and the extracellular matrix (described next), as well as on biochemical pathways, including hormonal ones. The generation of excessive MOBS in response to EMPO and MAT, the inability that accompanies all degenerative symptoms to neutralize or eradicate EMPO and MAT, and the recognized hyper- and hypocoagulable states seen in various symptomatolo­gies, will beyond doubt be revealed in the MOST.

Aspergillusnigercrystal

 

 

 

 

 

[Micrograph 8 and 9: Medically Diagnosed HIV/AIDS micrograph showing above an Aspergullus niger mold crystal using dark field microscopy and below a hypocoagulated blood clot with systemic protein polymerizations measuring in excess of 40 microns using bright field microscopy}

HIV

 

 

 

 

 

 

As mentioned, hormones are easily fermented, and this will show up as a hypocoagulated blood pattern in the MOST. It is my opinion, this hypocoagulated blood appears in the MOST as misty clouds of protein polymerizations throughout the clot, as seen in the accompanying picture.

poorfibrin

[Micrograph 10: Poor fibrin interconnection in the clot associated with endocrine or hormonal imbalance]

The MOST from Solubilized Extracellular Matrix

There is now a clearer picture of the biochemical rationale for correlating abnormal blood clotting patterns with the presence of degenerative symptoms.  A link between symptoms and the distorted clotted blood patterns has been delineated in the MOST.
Another reason for the abnormal clotting patterns accompanying pathological states, in addition to insufficient bonding of fibrinogen peptides as seen in the MOST, is presence in the blood of water-soluble fragments of the extracellular matrix.

Extracellular Matrix Degradation by MAT

The extracellular matrix (EM) is a three-dimen­sional gel, binding cells together and composed of five or more major constituents: collagen (protein), hyaluronic acid (polysaccharide), proteoglycans (pro- tein/polysaccharide), fibronectin and laminin. Also included are glycosaminoglycans and elastin.[67] In every degenerative disease studied by this author, evidence has been found for MAT activity destruc­tive of EM.

One of the proteolytic enzymes activated in response to EMPO and MAT is alpha-1 antitrypsin (capable of neutralizing MAT), normally not active in the presence of the enzyme trypsin. The active por­tion of this anti-exotoxin and antimycotoxin contains the amino acid methionine, which includes a C-S-C linkage. When chelated by the hydroxyl radical (one of the MOBS oxidants), methionine’s central sulfur atom acquires one or two oxygen atoms (forming the sulfone or sulfoxide respectively). The fermentation of methionine is a secondary effect of immune response to an alarming situation, intended to neutral­ize MAT and prevent degradation of the EM. Once alpha-1 antitrypsin is exhausted, MAT will have more access to the EM. If the EM is damaged beyond repair, then the enzyme trypsin is released to disorganize and recycle the cells involved.[68]

A similar scenario holds for the enzymes collage- nase and elastase. Thus, the absence of alpha-1 antitrypsin in the presence of EMPO and MAT activates three enzymes which degrade the extracellular matrix. Degradation of the EM by enzymes and MAT puts into the blood the water-soluble fragments (proteins and glycoproteins) of normally insoluble EM components (see Table 11). The presence of these fragments modifies the normal clotting pattern (described below), as seen in the M/OST, and is therefore an indication of EM degradation, which is always found with degenerative symptoms. (Also present is fibrin monomer, which has been found in the blood of patients suffering from collagen dis­ease.[69] See Table 11.)

Fibronectin is a molecule in EM having several binding sites for various long-chain molecules— heparin (a sulfonated polysaccharide) and collagen, for example. As such, it functions as a cellular glue, bind­ing cells together as well as various components of the EM. A soluble form of fibronectin is normally found free in the blood, and enters into the formation of a blood clot through the action of factor XIII. This form of fibronectin binds to fibrin. Elevated, bound-serum fibronectin results from EM fragmentation by MAT, and accompanies degenerative symptoms such as arthritis and emphysema (collagen diseases).

Water-soluble fragments of the EM bound by fibronectin form a three-dimensional network or gel in the pathologically clotted blood (fibrin and com­ponents of the blood clotting cascade). Since fibronectin binds to both fibrin and collagen, the two polymeric networks are superimposed and intermin­gled, resulting in a modification of the normal clot­ting pattern. Exactly how the pattern is modified depends upon the nature of the collagen abnormally present, the nature and extent of hyaluronate pre­sent, and the degree to which EM fibronectin has been released by MAT.

Conclusion

Thus, it is easily seen that there are many forms which the pattern of clotted blood may take, depending on the individual and the internal terrain that produced the modifying substances. The MOST reveals not only the presence of exotoxic and mycotoxic stress, but indicates as well the nature of the symptom(s) resulting from the stress (see Table 12). Since MAT underlie the entire complex of events which degrade the extracellular matrix, I must conclude that the absence of these exotoxins, endotoxins and mycotoxins would provide substantial improvements in tissue integrity and the overall physiology and functionality of the organism or animal and human.

­

­

References

[1]  Jones, T.W., “Observations on some points in the anatomy, physiology and pathology of the blood.”  British Foreign Medical Review, 1842. 14 : 585.

[2] Trousseau, A., Phlegmasis alba delens. “Clinque Medicale de L’Hotel Dieu de Paris.”, 1865, 3:94

[3]  Virchow, R., “Hypercoagulability: A review of its development, clinical application, and recent progress.”  Gesammelte Abhandlungen our Wussenschaftlichen Medizin, 1856, 26:477.

[4]  Rapaport, S.I., “Blood Coagulation and its Alterations in Hemorrhagic, and Thrombotic Disorders.”  The Western Journal of Medicine, 1993; 158: 153.

[5]  Hamilton, P.J. et al., “Disseminatied Intravascular Coagulation: A Review.”  Journal of Clinical Pathology, 1978, 31: 609

[6] The Harper Collins Illustrated Medical Dictionary, 1994, p.13.

[7] Young, RO, “Sick and Tired, Reclaim Your Inner Terraine,” Woodland Publishing, 1999.

[8] BeChamp, A., “The Blood and Its Third Anatomical Element,”  Hikari Omni Publishing, 1999.

[9]  Schwerdtle, C, Arnoul, F, Enerlein, G, “Introduction to Darkfield Diagnostics”, Semmelweis-Verlag (2006).

[10]  Hawk, BO, Thoma, GE, Inkley, JJ, The Evaluation of the Bolen Test as a Screening Test for Malignancy*, cancerres.aacrjournals.org on December 5, 2015. © 1951 American Association for Cancer Research.

[11]  Uchida, K., “Role of Reactive Aldehyde in Cardiovascular Diseases”,  Labortory of Food and Biodynamics, Nagoya University Graduate School of Bioagricultural Sciences, Nagoya, Japan , Free Radical Biology and Medicine, Volume 28, Issue 12, 15 June 2000, Pages 1685–1696

 [12] Chang JC, van der Hoeven LH, Haddox CH, “Glutathione reductase in the red blood cells”,  Ann Clin Lab Sci. 1978 Jan-Feb;8(1):23-9.

[13] Kutzing, MK, Firestein, BL, “Altered Uric Acid Levels and Disease States”, Department of Cell Biology and Neuroscience (M.K.K., B.L.F.), Graduate Program in Biomedical Engineering (M.K.K.), Rutgers University, Piscataway, New Jersey. Address correspondence to: Dr. Bonnie L. Firestein, Department of Cell Biology and Neuroscience, Rutgers University, Piscataway, NJ 08854-8082. E-mail: firestein@biology.rutgers.edu

[14] Claudino, M,. Ceolin,,DS, Alberti, S.,  Cestari, TM,  Spadella, CT, Fischer Rubira-Bullen, IR, Gustavo Pompermaier Garlet, Gerson Francisco de Assis, ” Alloxan-Induced Diabetes Triggers the Development of Periodontal Disease in Rats”,  Published: December 19, 2007. DOI: 10.1371/journal.pone.0001320

[15] Young RO (2015), “Alkalizing Nutritional Therapy in the Prevention and Reversal of any Cancerous Condition. Int J Complement Alt Med 2(1): 00046. DOI: 10.15406/ijcam.2015.02.00046

[16] Heloise Pöckel Fernandes, Carlos Lenz Cesar, and  Maria de Lourdes Barjas-Castro, “Electrical properties of the red blood cell membrane and immunohematological investigation”, Rev Bras Hematol Hemoter. 2011; 33(4): 297–301. doi:  10.5581/1516-8484.20110080 PMCID: PMC3415751

[17] Harris, JO, “The Relationship Between the Surface Charge and the Absorption of Acid Dyes by Bacterial Cells”, Department of Bacteriology, Kansas Agricultural Experiment Station, Manhattan,Kansas, Received for publication March 3, 195.

[18] Young, RO, “Metabolic and Dietary Acids are the Fuel That Lights the Fuse that Ignites Inflammation that Leads to Cancer”. https://www.linkedin.com/pulse/metabolic-dietary-acids-fuse-ignites-inflammation-causes-young. 2015.

[19] Snaders, R, “Did Bacteria Spark Evolution of Multicellular Life?” Berkeley News, Research, Science and Environment,  October 24, 2012.

[20] Wenner, M, “Humans Carry More Bacterial Cells than Human Ones”. Scientific American, November 30th, 2007.

[21} Animals and humans respond to MAT as a poison.

[22]  Morrison, D.C. et al. The effects of bacterial endotox­ins on host mediation systems. American Journal of Pathology, 1978; 93: 526.

[23]  Ibid.

[24]  Ibid.

[25]  Van Deventer, S.J.H. et al. Intestinal Endotoxemia. Gastroenterology, 1988; 94(3): 825-831.

[26]  Morrison, D.C. et al., op. cit.

[27]  Ibid.

[28]  Hu, T. et al. Synthesis of tissue factor messenger RNA and procoagulant activity in breast cancer cells in response to serum stimulation. Thrombosis Research, 1993; 72: 155.

[29]  Rapaport, op. cit. (Ref. 4).

[30]  Ibid.

[31]  Mackman et al. Lipopolysaccharides—mediated tran­scriptional activation of the human tissue factor gene in THP-1 monocytic cells requires both activator protein 1 and nuclear factor kappa B binding sites. Journal of Experimental Medicine, 1991; 174: 1517.

[32]  Yamada, O. et al. Deleterious effects of endotoxins on cultured endothelial cells: An in vitro model of vascular injury. Inflammation, 1981; 5: 115.

[33]  Colucci, M. et al. Cultured human endothelial cells: An in vitro model of vascular injury. Journal of Clinical Investigation, 1983; 71: 1893.

[34]  Cho, T.H. et al. Effects of Escherichia coli toxin on structure and permeability of myocardial capillaries.

[35]  Acta Pathologica Japonica, 1991; 41: 12.

[36]  Rapaport, op. cit. (Ref. 4).

[37]  Ibid.

[38]  Margolis, J. The interrelationship of coagulation of plasma and release of peptides. Annals of the New York Academy of Sciences, 1963; 104: 133.

[39]  23-25. Ibid.

[40]  Morrison, D.C. et al., op. cit.

[41]  Rapaport, op. cit. (Ref. 4).

[42]  Alberts, B. et al, eds. Molecular Biology of the Cell. New York: Garland Publishing, Inc., 1989 (2nd ed.), p. 818.

[43]  Rapaport, op. cit. (Ref. 4).

[44] Bertz, A., et al. Modulation by cytokines of leukocyte endothelial cell interactions. Implications for thrombo­sis. Biorheology, 1990; 27: 455.

[45]  Rapaport, op. cit. (Ref. 4).

[46]  Nachman, R.L. et al. Hypercoagulable states. Annab of Internal Medicine, 1993; 119: 819.

[47]  Ibid.

[48]  Tallman, M.S., et al. New insights into the pathogene­sis of coagulation dysfunction in acute promyelocytic leukemia. Leukemia and Lymphoma, 1993; IT. 27.

[49]  Silberberg, J.M., et al. Identification of tissue factor in two human pancreatic cancer cell lines. Cancer Research, 1989; 49: 5443.

[50]  Grimstad, I.A. et al. Thromboplastin release, but not content, correlates with spontaneous metastasis of can­cer cells. International Journal of Cancer, 1988; 41: 427.

[51]  Gunji, Y. et al. Role of fibrin coagulation in protection of murine tumor cells from destruction by cytotoxic cells. Cancer Research, 1988; 48: 5216.

[52]  Sugiyama, S. et al. The role of leukotoxin (9, 10- epoxy-12-octadecenoate) in the genesis of coagulation abnormalities. Life Sciences, 1988; 43: 221.

[53]  Ibid.

[54]  White, A. et al, eds. Principles of Biochemistry. McGraw-Hill Book Co., New York, 1964, p. 648.

[55]  Mueller, H.E. et al. Increase of microbial neu­raminidase activity by the hydrogen peroxide concen­tration. Experientia, 1972; 23: 397.

[56]  Young, Robert O. Fermentology and oxidology. The study of fungus-produced mycotoxic species and the activation of the immune system and release of microzymian oxidative buffering species (MOBS). Self- published: InnerLight Biological Research Foundation, Alpine, Utah, 1994.

[57]Chandler, WL. et al. Evaluation of a new dynamic vis­cometer for measuring the viscosity of whole blood and plasma. Clinical Chemistry, 1986; 32: 505.

[58]  Saleem, A. et al. Viscoelastic measurement of clot for­mation: A new test of platelet function. Annals of Clinical and Laboratory Science, 1983; 13: 115.

[59]  Spillert, C.R. et al. Altered coagulability: An aid toselective breast biopsy. Journal of the National Medical Association, 1993; 85: 273.

[60]  Bowie, E.J. et al. The clinical pathology of intravascular coagulation. Bibliotheca Haematologica, 1983; 49: 217.

[61]  Muller-Berghaus, G. et al. The role of granulocytes in the activation of intravascular coagulation and the pre­cipitation of soluble fibrin by endotoxin. Blood, 1975; 45: 631.

[62]  Bick, R.L. Disseminated intravascular coagulation. Hematology/Oncology Clinics of North America, 1993; 6: 1259.

[63]  Bredbacka, S. et al. Laboratory methods for detecting disseminated intravascular coagulation (DIC): New aspects. Acta Anaesthesiologica Scandinavica, 1993; 37: 125.

[64]  Sigma Diagnostics, St. Louis, MO 63178; tel: 314- 771-5765.

[65]  Nachman, R.L. et al. Detection of intravascular coag­ulation by a serial-dilution protamine sulfate test. Annals of Internal Medicine, 1971; 75: 895.

[66]  Breen, F.A. et al. Ethanol gelation: A rapid screening test for intravascular coagulation. Annals of Internal Medicine, 1970; 69: 1197.

[67] Hay, E.D., ed. Cell Biology of Extracellular Matrix. New York: Plenum Press, 1981, p. 653.

[68]  Carp, H. et al. In vitro suppression of serum elastase- inhibitory capacity by ROTS generated by phagocytos- ing polymorphonuclear leukocytes. Journal of Clinical Investigation, 1979; 63: 793.

[69]  Wilson, C.L. The alternatively spliced V region con­tributes to the differential incorporation of plasma and cellular fibronectins into fibrin clots. Journal of Cell Biology, 1992; 119: 923.

[70] Young, RO, Young, SR, “The pH Miracle Revised and Updated”, Hachette Publishing, 2010.

Tables

Table 1

Expression of Sialic Acid/Galactose [MAT] from Cell and Protein Degeneration (From All Serum Proteins, RBC/WBC and Other Cell Surfaces)

  1.  Carbohydrate, Proteins, and Fats From Diet, Body Cells or Reserves
  2. As cells breakdown or ferment they give birth to bacteria, yeast, fungus and mold [EMPO] and their associated metabolic acidic waste [MAT]
  3. Exotoxins, Endotoxins, and Mycotoxins [MAT]
  4. Acetyl Aldehyde, Ethyl Alcohol, Uric Acid, Alloxan, Lactic Acid are examples of MAT
  5. MAT  Ferments Other Body Cells and their Extracellular Membranes and Proteins
  6. MAT Modifies Glycoprotein
  7. Binds to liver Galactosidase
  8. Creating an Increase in Cell and Protein Fermentation and Degeneration and Increased Amounts of Exotoxins, Endotoxins and Mycotoxins [MAT]

Table1a

Table 2

Expression of Sialic Acid [MAT] From the Fermentation of Degeneration of Insulin Producing Pancreatic Beta-Cells in Type I, Type II and Type III Diabetes

  1. Pancreatic Insulin producing Beta-Cells with no or minimal Surface Sialic Acid [MAT]A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Diet
  2. Normal regulation of Insulin Production
  3. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choicesdd
  4. Leads to cellular fermentation and degeneration and the birth of EMPO
  5. This lead to increased abnormal amounts of MAT that the immune system, the alkaline buffering system and the elimination organs has to deal with
  6. Fermenting and degenerating Insulin Producing Beta Cells
  7. Giving Rise to Surface Cell Sialic Acid [MAT}
  8. Increased Amounts of Sialic Acid Activates the Immune Response [MOBS] and Sialidase [AB]
  9. Leads to Lowered or No Insulin Production
  10. Symptoms of Type I, Type II or Type III Expressed
  11. The insulin producing beta cells of the Islets of Langerhans express silica acid on their surface as a break down metabolite.  I have suggested that when insulin producing beta cells are physically disturbed by MAT they begin to disorganize and express sialic acid on the surface of the cell.  This indicates the death of the cell and insulin production will stop.

Table2a

Table 3

HIGH BLOOD PRESSURE, ATHEROSCLEROSIS, HEART ATTACKS, STROKES, and CONGESTIVE HEART FAILURE

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This lead to increased abnormal amounts of MAT that activates the immune system to chelate the MAT.
  4. Increased amounts of MAT will cause endothelial breakdown and the expression of Sialic acid.
  5. Increased Amounts of Sialic Acid and damage to the endothelial will cause a reduction in the negative surface-charge leading to the release of Glycoproteins.
  6. The release of Glycoproteins will cause the activation of Factor XII and the blood clotting cascade.
  7. This cause the creation and formation of fibrin monomers and the increase of Platelet Deposition out of the red blood cells for clotting purposes
  8. The immune system will activate and MOBS will be released as well as sodium bicarbonate, calcium, lipids and other alkaline buffers to reduce metabolic acidity.
  9. The build-up of fibrin monomers in the clotting cascade will lead to fibrin nets and clots causing an increase in blood pressure and the risk of blockages potentially causing a Stroke or Heart Attack.

Table3a

Table 4

DISSEMINATED INTRAVASCULAR COAGULATION RESULTING
FROM INTRACELLULAR DISORGANIZATION OR FERMENTATION WHICH GIVES RISE TO MAT
 AND EMPO

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This lead to increased abnormal amounts of MAT that activates the Tumor Necrosis Factor (TNF).
  4. Increased amounts of TNF activates the Tissue Factor Gene (TF)
  5. Increased Amounts of TF causes the release of Thromboplastin.
  6. The release of Thromboplastin activates the release of clotting Factors VII (VIIa) and trace amounts of Factor Xa into the blood.
  7. This activates the release of Factors IX and X to IXa and the increase of Factor Xa.
  8. The activation of the blood clotting cascade leads to Disseminated Intravascular coagulation and the clotting or thickening of the blood inside the blood vessels.
  9. The DIC or hyper-coagulation will mask the fermentation of healthy cells to unhealthy cells or cancerous cells.
  10. As the unhealthy cells or cancerous cells increase the body will go into preservation mode and begin forming fibrin nets to encapsulated these unhealthy cells to protect healthy body cells.
  11. As body and blood cells breakdown from MAT this causes an increase of MAT and EMPO leading to systemic latent tissue acidosis and a potential metastatic cancerous condition.

Table4a

 Table 5

DISSEMINATED INTRAVASCULAR COAGULATION RESULTING
IN CELLULAR DISORGANIZATION OR FERMENTATION/OXIDATON AND THE INCREASE OF MAT AND EMPO

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices.
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This lead to increased abnormal amounts of MAT that activates the Tumor Necrosis Factor (TNF).
  4. Increased amounts of TNF activates the Tissue Factor Gene (TF)
  5. Increased Amounts of TF causes the release of Thromboplastin.
  6. The release of Thromboplastin activates the release of clotting Factors VII and Factor Xa in the blood.
  7. This activates the release of Factors IX and X to IXa and the increase of Factor Xa.
  8. The activated blood clotting cascade leads to Disseminated Intravascular coagulation and the clotting or thickening of the blood inside the blood vessels.
  9. The DIC or hyper-coagulation will mask the fermentation of healthy cells to unhealthy cells or cancerous cells.
  10. As the unhealthy cells or cancerous cells increase the body will go into preservation mode and begin forming fibrin nets to encapsulated the unhealthy cells.
  11. This leads to tumor formation of the unhealthy or cancerous cells.
  12. As the body and blood cells breakdown this causes an increase of MAT and EMPO leading to an increased risk of  systemic metastatic cancer.

Table5aTable 6

ENDOTHEIAl CELL CONVERSION FROM AN
ANTITHROMBOTIC STATE TO A PROCOAGULANT STATE
CELLULAR DISORGANIZING PATHWAY

  1. A Physical and/or Emotional Disturbance Occurs from Lifestyle and/or Dietary choices
  2. Leads to cellular fermentation and degeneration and the birth of EMPO
  3. This leads to increased abnormal amounts of MAT that damages the protective endothelial cover cells leading to a reduction of PGI2
  4. The absence of PGI2 causes the release of Interleukin-1 and/or Tumor Necrosis Factor (TNF).
  5. In addition the loss of protective endothelial cover cells leads to Tissue Factor Gene Activation and the release of Thrombin causing a pro-coagulate state leading to DIC
  6. Another pathway to DIC would be the loss of protective endothelial cover cells and the absence of PGI2 causes the suppression of Thromomodulin, Protein C leading to procogradulation and DIC.

Talble6

 Table 7

ENDOTHELIAL CELL CONVERSION
FROM AN ANTITHROMBOTIC STATE
(NORMAL PATHWAY)

Table7

Table 8

MECHANISM OF DISSEMINATED INTRAVASCULAR COAGULATION GENERATED BY MAT

Table8Table 9

ACTIVATION OF SIALIDASE AND MICROZYMIAN OXIDATIVE BUFFERING SPECIES (MOBS) BY EMPO AND MAT

Table9

Table 10

DISSEMINATED INTRAVASCULAR COAGULATION RESULTING FROM PHAGOCYTIC OXIDATIVE BURST

Table10

Table 11

MOST BLOOD TEST and DISSEMINATED INTRAVASCULAR COAGULATION WITH SOLUBILIZED EXTRACELLULAR MATRIX

Table11

Table 12

TYPICAL SOURCES OF FERMENTATION INSULT (MAT) IN BIOLOGICAL SYSTEMS INITIATING DIC

Table12

Table 13

POSITIVE CHARGE OF CANCEROUS CELLS AND TUMORS AND THE FORMATION OF FIBRIN NETS AND TREES IN RESPONSE TO MAT

Table13

A Self-Care to a Self-Cure for Type I Diabetes!

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Today is Jakob’s 8th birthday. Jakob was diagnosed with Type I diabetes and was taking daily insulin shots to regulate his blood sugar levels. Now he is following Dr. Robert O. Young’s pH Miracle for Diabetes Protocol as outlined in The pH Miracle for Diabetes. It has NOW been 241 days since Jakob needed a single daily insulin shot. His blood sugar levels have been normal for over 8 months. He is a very happy and healthy boy now. And his parents are even more happy that they have a happy and healthy boy free from high blood sugars and the daily insulin shots! I am also happy for Jakob and his family. You know who is not happy about Jakob’s self-cure from Type I Diabetes and wants Dr Young’s protocol suppressed?

In the words of Martin Luther King, Jr., “Our lives begin to end the day we become silent about things that matter.” The Self-Care to a Self-Cure for diabetes matters.

As long as I am alive i will continue to proclaim to all the World to all who will listen to the truth that there is a natural non-invasive, drug-free, self-cure for Type I, Type II and Type III diabetes, for heart disease and for cancer.

There is a natural non-invasive, drug-free, self-care to a self-cure for ALL sickness and disease. No child needs to be diabetic if the child and his or her parents are willing to follow The pH Miracle for Diabetes Protocol. UNDERSTAND?

If you have Type I, II or III diabetes or know someone who has diabetes and is insulin dependent, the pH Miracle for Diabetes Protocol will begin the process of your/their Self-Care to a Self-Cure regardless of how long you’ve/they’ve been doing diabetes! Understand?

Stand-up for health freedom! Education NOT Medication! Health Care NOT Sick Care! Read and share the truth with your family and friends. Juvenile diabetes is a world-wide epidemic and we NOW know the cause and we have the self-cure. Every child with diabetes can be healed.

Remember it is YOUR Body, YOUR LIFE and YOUR CHOICE. YOU MUST BE THE CHANGE AND THE CURE YOU WANT TO SEE!

For more information of reversing Type I, II and III diabetes go to: http://www.phoreveryoung.wordpress.com, http://www.phmiracle.com

The Sugar Fructose from Fruit Causes Diabetes!

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Credit: © flaviuz / Fotolia

Recent studies have shown that added sugars, particularly those containing fructose, are a principal driver of diabetes and pre-diabetes, even more so than other carbohydrates. Clinical experts writing in Mayo Clinic Proceedings challenge current dietary guidelines that allow up to 25% of total daily calories as added sugars, and propose drastic reductions in the amount of added sugar, and especially added fructose, people consume.

Worldwide, approximately one in ten adults has type 2 diabetes, with the number of individuals afflicted by the disease across the globe more than doubling from 153 million in 1980 to 347 million in 2008. In the United States, 29 million adults (one in eleven) have type 2 diabetes and another 86 million (more than one in three) have pre-diabetes.

“At current levels, added-sugar consumption, and added-fructose consumption in particular, are fueling a worsening epidemic of type 2 diabetes,” said lead author James J. DiNicolantonio, PharmD, a cardiovascular research scientist at Saint Luke’s Mid America Heart Institute, Kansas City, MO. “Approximately 40% of U.S. adults already have some degree of insulin resistance with projections that nearly the same percentage will eventually develop frank diabetes.”

The net result of excess consumption of added fructose is derangement of both overall metabolism and global insulin resistance say the authors. Other dietary sugars not containing fructose seem to be less detrimental in these respects. Indeed, several clinical trials have shown that compared to glucose or starch, isocaloric exchange with fructose or sucrose leads to increases in fasting insulin, fasting glucose, and the insulin/glucose responses to a sucrose load. “This suggests that sucrose (in particular the fructose component) is more harmful compared to other carbohydrates,” added Dr. DiNicolantonio. Dr. DiNicolantonio and his co-authors, James H O’Keefe, MD, Saint Luke’s Mid America Heart Institute, Kansas City, MO, and Sean C. Lucan, MD, MPH, MS, a family physician at Montefiore Medical Center, Albert Einstein College of Medicine, Bronx, NY, examined animal experiments and human studies to come to their conclusions.

Data from recent trials suggest that replacing glucose-only starch with fructose-containing table sugar (sucrose) results in significant adverse metabolic effects. Adverse effects are broader with increasing baseline insulin resistance and more profound with greater proportions of added fructose in the diet.

The totality of the evidence is compelling to suggest that added sugar, and especially added fructose (usually in the form of high-fructose corn syrup and table sugar), are a serious and growing public health problem, according to the authors.

The 2010 Dietary Guidelines for Americans say it is acceptable for some people to consume up to 19% of calories from added sugars, and the Institute of Medicine permits up to 25% of total calories from added sugars. In contrast, the World Health Organization recommends that added sugars should make up no more than 10% of an entire day’s caloric intake, with a proposal to lower this level to 5% or less for optimal health. Such levels would be more in line with what the authors would recommend and similarly restrictive to existing American Heart Association (AHA) recommendations–to consume no more than six teaspoons (24 grams) of sugar per day for women and no more than nine teaspoons (36 grams) of sugar per day for men.

While fructose is found naturally in some whole foods like fruits and vegetables, consuming these foods poses no problem for human health. Indeed, consuming fruits and vegetables is likely protective against diabetes and broader cardiometabolic dysfunction, explained DiNicolantonio and colleagues. The authors propose that dietary guidelines should be modified to encourage individuals to replace processed foods, laden with added sugars and fructose, with whole foods like fruits and vegetables. “Most existing guidelines fall short of this mark at the potential cost of worsening rates of diabetes and related cardiovascular and other consequences,” they wrote.

The authors also think there should be incentives for industry to add less sugars, especially fructose-containing varieties, to food-and-beverage products. And they conclude that at “an individual level, limiting consumption of foods and beverages that contain added sugars, particularly added fructose, may be one of the single most effective strategies for ensuring one’s robust future health.”


Story Source:

The above story is based on materials provided by Elsevier Health Sciences. Note: Materials may be edited for content and length.


Journal Reference:

  1. James J. DiNicolantonio, James H. O’Keefe, Sean C. Lucan. Added Fructose: A Principal Driver of Type 2 Diabetes Mellitus and Its Consequences. Mayo Clinic Proceedings, 2015; DOI: 10.1016/j.mayocp.2014.12.019
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