Category Archives: Heart Disease

Dr. Robert O Young Lectures at Harvard

Towards the Ethics of Healing

Towards the Ethics of Healing begins the debate on human health and how we should treat any sickness and disease. Watch and learn the truth about Dr. Young’s work, research and findings and learn how to prevent, treat and reverse heart disease, diabetes, cancer, autism, just to name a few in this 3 Part lecture at Harvard University School of Medicine.

The Law of Change – Part 2

The New Biology – Part 3

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To learn more about research of Dr. Robert O Young go to: www.drrobertyoung.com and/or www.phmiracleproducts.com Education and pH Miracle retreats: www.phmiracleretreat.com
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Life Changing and Life Saving Knowledge!

Life transforming knowledge!

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Thank you Dr Young! Living the Alkaline Lifestyle since 2011! 😁🙏🏻❤️Every symptom of dis-ease is gone!

Does Cholesterol (LDL) Cause Heart Disease? What is the True Cause of Heart Disease?

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A physician’s word is often taken very seriously and with little skepticism. An opinion from one or two doctors, when made in a professional office or hospital, can persuade a worried patient to take drugs with complex side-effects, or even undergo traumatic treatments such as radiation and chemotherapy. Yet, when the same doctors, with years of experience and thousands of satisfied customers, give an opinion that questions a therapy established by mainstream medicine, the mainstream media calls them irresponsible, or quacks, or even criminals.

Which brings me to Dr. Dwight Lundell. He’s an experienced heart surgeon and retired Chief of Staff and Chief of Surgery at Banner Heart Hospital in Mesa, Arizona. Not so long ago, Dr. Lundell made the following statement of confession:

“We physicians with all our training, knowledge and authority often acquire a rather large ego that tends to make it difficult to admit we are wrong. So, here it is. I freely admit to being wrong. As a heart surgeon with 25 years experience, having performed over 5,000 open-heart surgeries, today is my day to right the wrong with medical and scientific fact.

I trained for many years with other prominent physicians labeled “opinion makers.” Bombarded with scientific literature, continually attending education seminars, we opinion makers insisted heart disease resulted from the simple fact of elevated blood cholesterol. The only accepted therapy was prescribing medications to lower cholesterol and a diet that severely restricted fat intake. The latter of course we insisted would lower cholesterol and heart disease. Deviations from these recommendations were considered heresy and could quite possibly result in malpractice. It Is Not Working!

These recommendations are no longer scientifically or morally defensible.”

Many doctors are highly admirable people, but they are still human beings. They all make mistakes, they all learn from them, but the really good ones are willing to admit to them.

Cholesterol does not cause heart disease and trying to reduce it with statin drugs is a waste of time, an international group of experts has claimed.

Not surprisingly, Lundell’s statement regarding the medical establishment’s approach to treating heart disease caused a ripple in the medical industry. It challenged the validity of statins – commonly known as cholesterol-lowering medications – such as Lipitor, Crestor, Zocor, and others.

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The reason Lundell’s statement created such a buzz is because statins are big business. In the United States alone, about 25% of the population takes statin medications. They cost from as little as $53 per month to more than $600. Pfizer’s Lipitor went on sale in 1997 and its lifetime sales have surpassed $125 billion. AstraZeneca’s Crestor was the top-selling statin in 2013, generating $5.2 billion in revenue that year alone. The statin industry is estimated at around $30 billion in sales per year. Nevertheless, in the United States, more die each year of heart disease than ever before.

Lundell went on to say:

“The discovery a few years ago that inflammation in the artery wall is the real cause of heart disease is slowly leading to a paradigm shift in how heart disease and other chronic ailments will be treated. The long-established dietary recommendations have created epidemics of obesity and diabetes, the consequences of which dwarf any historical plague in terms of mortality, human suffering and dire economic consequences.

I have peered inside thousands upon thousands of arteries. A diseased artery looks as if someone took a brush and scrubbed repeatedly against its wall. Several times a day, every day, the foods we eat create small injuries compounding into more injuries, causing the body to respond continuously and appropriately with inflammation. While we savor the tantalizing taste of a sweet roll, our bodies respond alarmingly as if a foreign invader arrived declaring war. Foods loaded with sugars and simple carbohydrates, or processed with omega-6 oils for long shelf life have been the mainstay of the American diet for six decades. These foods have been slowly poisoning everyone.”

Listen to Dr. Tom Sladic, MD has he explains his understanding of the true cause of Heart Disease:

So What is the True Cause of Heart Disease?

 

A review of research involving nearly 70,000 people found there was no link between what has traditionally been considered “bad” LDL cholesterol and the premature deaths of over 60-year-olds from cardiovascular disease.

Published in the BMJ Open journal, the new study found that 92 percent of people with a high cholesterol level lived longer. (BMJ Open. Published online June 12 2016)

The authors have called for a re-evaluation of the guidelines for the prevention of cardiovascular disease and atherosclerosis, a hardening and narrowing of the arteries, because “the benefits from statin treatment have been exaggerated”.

High cholesterol is commonly caused by an unhealthy acidic lifestyle and diet, and eating high levels of processed fat in particular, as well as smoking.

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It is carried in the blood attached to proteins called lipoproteins and has been traditionally linked to cardiovascular diseases such as coronary heart disease, stroke, peripheral arterial disease and aortic disease.

Co-author of the study Dr Malcolm Kendrick, an intermediate care GP, acknowledged the findings would cause controversy but defended them as “robust” and “thoroughly reviewed”. “What we found in our detailed systematic review was that older people with high LDL (low-density lipoprotein) levels, the so-called “bad” cholesterol, lived longer and had less heart disease.”

 

Vascular and endovascular surgery expert Professor Sherif Sultan from the University of Ireland, who also worked on the study, said cholesterol is one of the “most vital” molecules in the body and prevents infection, cancer, muscle pain and other conditions in elderly people. He also stated, “lowering cholesterol with medications is a total waste of time and money”.

“Lowering cholesterol with medications for primary cardiovascular prevention in those aged over 60 is a total waste of time and resources, whereas altering your lifestyle is the single most important way to achieve a good quality of life,” he said.

Lead author Dr Uffe Ravnskov, a former associate professor of renal medicine at Lund University in Sweden, said there was “no reason” to lower high-LDL-cholesterol.

Heart Disease and Cholesterol

The graph below shows the famous 10 year Framingham correlation study between cholesterol and coronary heart disease, published in the Lancet in 1986, that big Pharma relies on and sold to the American public at large.

 

The problem though, as you see in the next graph, after 20 years the correlation shows that high cholesterol saves lives and low cholesterol is a risk factor for heart disease!

Everyone in modern society has heard about cholesterol, and how bad it is. Most do not understand why it exists, and simply see it as a menace that must be eliminated as quickly as possible. This misunderstanding is exactly what the pharmaceutical complex promotes, because it allows them to perpetually treat high cholesterol with drugs like Lipitor. These drugs are prescribed for the remainder of a patient’s lifetime, and when he/she eventually dies of a “thought attack”, family and friends will believe that the disaster was inevitable from “high cholesterol”. The death will not be attributed to other health factors or to the drugs themselves, but to the “high cholesterol”; even though there are no known deaths from cholesterol in human history. It is all very convenient for the drug companies, so long as we do not examine what is up the other sleeve.

I am reminded of restless leg syndrome, whereby the dis-ease was ‘discovered’ immediately after the pharmaceutical for it was patented, as a reason to sell us this useless pharmaceutical drug. Now, restless leg syndrome has been upgraded to a new “disease”. The cause of restless leg syndrome is also the cause of heart disease – retained metabolic and/or dietary acids in the connective and fatty tissues leading to inflammation, induration, ulceration, degeneration and finally death

“Before 1920, coronary heart disease was rare in America — so rare that when a young internist named Paul Dudley White introduced the German Electrocardiograph to his colleagues at Harvard University, they advised him to concentrate on a more profitable branch of medicine. The new machine revealed the presence of arterial blockages, thus permitting early diagnosis of coronary heart disease. But in those days, clogged arteries were a medical rarity, and White had to search for patients who could benefit from his new technology. During the next forty years, however, the incidence of coronary heart disease rose dramatically, so much so that by the mid 1950’s, heart disease was the leading cause of death among Americans.”

— Mary Enig, Ph.D.

The amount of cholesterol that you eat actually has very little relationship with the amount that you have in your blood. When you eat more cholesterol, your body produces less, and when you eat less cholesterol, your body produces more. Another way to say this is like this – when you have more metabolic or dietary acid in your blood and interstitial fluids the body produces more LDL cholesterol, and when you have less metabolic or dietary acid in your blood and interstitial fluids the body produces less cholesterol. Why? Because LDL cholesterol is a buffer or chelator of metabolic and/or dietary waste. Understand? A body usually produces between three and four times the cholesterol that one eats. The amount produced is generally related to how much is needed. Cholesterol is indeed needed and critical for optimal health. The purpose of so-called “bad cholesterol” is not to give us heart attacks, but to buffer acidic metabolic and dietary waste and to repair the damage to arteries or veins from our acidic lifestyles and diets.

Whenever a poor acidic diet and lifestyle leads to damaged arteries, a thick and sticky substance is required to patch them. That substance is known as LDL or “bad cholesterol”. When this damaging behavior is continued, multiple patches are created, leading to what we know as “clogged arteries”. The problem is not the cholesterol, which is doing its wonderful job of preventing our death from internal bleeding. The problem is the fact that the arteries or veins are damaged enough from acidic lifestyle and dietary choices to risk internal bleeding. Blocking a body’s healthy countermeasure only leads to worse problems. It is the pharmaceutical standard of symptom suppression that is like hiding the timer of a time bomb, and then expecting it not to eventually go off. Thus, that so-called “BAD” cholesterol is not “BAD” at all. In fact LDL cholesterol is saving your acidic body from internal bleeding and inevitable death. LDL cholesterol ONLY increases in the presence of excess metabolic, dietary, respiratory and/or environmental acids which increase as a result of what you eat, what you drink and what you think. High LDL cholesterol is a warning sign of your poor acidic lifestyle and dietary choices and the body is in preservation mode. It is trying to protect itself from YOU!

Cholesterol is created to save your life! The following picture is what solidified metabolic acid bound cholesterol looks like in the blood.

 

Modern medicine spends a lot of time fighting this pitch, instead of the actual causes of arterial damage. Thus, it is not surprising that cholesterol-lowering drugs cause more heart dis-ease and more heart attacks and strokes. A massive portion of the elderly population is taking cholesterol-lowering drugs, even though research shows that the higher their cholesterol levels (especially LDL) the longer that they will live and the less risk for a heart attack or stroke. The graph below illustrates this point! Low cholesterol in the elderly is actually a sign that something is seriously wrong, and a heart attack or stroke may be imminent. Modern medicine has only recently come to accept that at least some cholesterol (LDL and HDL) is good and protective! But when you mention (LDL) cholesterol as “GOOD” you better take cover from current medical savants who will attack you with their ignorance!

 

Cholesterol is still suppressed with drugs, despite what science would make prudent from the long-term Framingham Study. It also has been proven that these drugs cause high suicide rates. The drugs can lead to personality changes, in a manner similar to (but not as intense as) S.S.R.I. antidepressants.

 

The anti-cholesterol hysteria began in the 1950’s, when researcher Ancel Keys proposed the Lipid Hypothesis. It stated that cholesterol and saturated fats lead to heart disease. His beliefs were promoted heavily by the new hydrogenated oils industry, which spent obscene amounts of money to convince every one of Keys’ indisputable findings. This successful marketing campaign was on par with similar marketing for fluoride at about the same time. Studies which had oppositional findings to Keys’ were ignored or maligned. As a result of his flawed scientific methodology (subjective cherry picking results to match what he wanted to find) saturated fats like butter and eggs were used less, in exchange for the poisonous trans-fats that are in hydrogenated oils. Heart disease rates have been rising exponentially ever-since.

The French eat more fats than any other group in the world, yet they have lower rates of heart disease. The Japanese eat more fats than Americans, yet have lower rates of heart disease. There are plenty of countries with similar patterns. The French lifestyle especially counters Keys’ hypothesis, and it also provides evidence that resveratrol (found in red or purple grapes) improves heart health. Resveratrol has been shown to reverse atherosclerosis (hardening of the arteries). Maybe, just maybe its being American that causes higher rates in heart attacks.   The bottom-line medical research is subjective NOT objective!

Just recently the Food and Drug Administration issued new safety warnings about a popular class of drugs used to control and lower cholesterol levels. The FDA says the drugs, known as statins, can cause several side effects, including cognitive problems such as memory lapses and confusion. But the agency is stressing that the side effects appear to be rare and not serious. I have suggested that taking any drug, like statin drugs that lowers LDL cholesterol without removing acidic lifestyle and dietary choices is a risk for heart attack, stroke and other dis-eases like diabetes. I have lowered cholesterol successfully in all cases of hyper-chlolesterolemia without drugs by just changing the diet and lifestyle to an alkaline pH Miracle lifestyle and diet that restores the alkaline design of the body.

One of my research clients Maren Hale was diagnosed with familial hypercholesterolemia and hyper-triglycerides with LDL’s over 400 mg/dl and triglycerides over 200 mg/dl. She was also overweight. Over a period of four years Maren lost over 70 pounds and lowered her cholesterol and triglycerides to healthy normal ranges on the pH Miracle Lifestyle and Diet. Maren and her family and extended family have been a research study of the University of Utah for familial hypercholesterolemia for over 60 years. Maren was the first of all family members to lower her cholesterol and triglycerides to normal ranges due to her commitment to living a pH Miracle Lifestyle and Diet.

 

High cholesterol levels should be a warning to most people who inflammation caused by metabolic and dietary acid is present. It is a risk marker, and a symptom that can save your life! Eliminating the LDL cholesterol through drugs is the equivalent to eliminating the thermometer in a room that is too hot. It is illogical, and it does nothing to eliminate the dangerous cause of the symptom being expressed.

LDL cholesterol levels naturally drop whenever the body’s becomes less acidic and more alkaline in the interstitial fluids where acids are stored! And LDL cholesterol should never be forced lower with drugs because they WILL cause a heart attack or stroke! The pH Miracle alkaline lifestyle and diet can reduce LDL cholesterol, but it is never because of a lowered cholesterol intake.

The natural drop in cholesterol and triglycerides happens only when a person stops eating toxic acidic foods, drinking toxic acidic drinks and stops toxic acidic thoughts that produce toxic acidic waste products that destroy the arteries and veins!

Do YOU Understand?

Because healthy arteries and veins do not need patching. Remember that a body typically produces 3-4 times the amount of LDL cholesterol than consumed. The fats that a person eats are therefore comparatively insignificant. Cholesterol will rise whenever the body’s need for cholesterol rises and in direct relationship to the level of acidic thoughts, words and deeds. So acidic trans-fats and inflammatory acidic substances are what need to be avoided. These toxic acidic wastes are what damage the arteries and veins, and a body will be required to do a great deal of patching as a consequence. I will reference to alkalizing or chelating herbs and minerals that lower cholesterol levels naturally later, but alkalizing and chelating herbs and minerals do it by lowering the body’s need for LDL cholesterol, not by forcefully lowering it like pharmaceuticals do.

 

Studies on the link between cholesterol and heart health have been manipulated for decades. The first studies on eggs showed elevated cholesterol levels because they had used dehydrated eggs, and studies of coconut oil yielded similar results because they had used partially hydrogenated coconut oil to get the results that they wanted. That is why I state that ALL scientific research is subjective NOT objective!!!!!!!!!!!!!!!!!!!!!! Read about it here: http://wp.me/p5ggLY-a5

It is Simple – Cholesterol DOES NOT CAUSE Heart Disease!

Simply stated, without acid caused inflammation being present in the body, there is no way that cholesterol would accumulate on and in the wall of the blood vessel and cause heart disease and strokes. Without acid caused inflammation, cholesterol would move freely throughout the body as nature intended. It is acid caused inflammation from acidic lifestyle and dietary choices that causes cholesterol to become trapped.

Acid caused inflammation is not complicated. The cycle of metabolic and dietary acid inflammation is perfect in how the body releases cholesterol to bind acids that cause inflammation in the first place. However, if we chronically expose the body to injury to acidic poisonous toxins from acidic foods and drinks the human body was never designed to process, a condition occurs called systemic latent tissue acidosis that is the cause of ALL inflammation. Chronic acidic inflammation is just as harmful as acute acidic inflammation and are both caused by an increase of dietary and metabolic acids.

What thoughtful person would willfully expose himself or herself repeatedly to acidic foods, drinks, drugs or other substances that are known to cause injury to the body? Well, smokers, alcohol, coffee black tea, soda pop, energy and sport beverage drinkers perhaps, but at least they made that choice willfully.

The rest of us have simply followed the recommended mainstream acidic diet that is low in polyunsaturated fats, high in acidic carbohydrates and highly acidic animal flesh, not knowing we were causing repeated acidic injury to our blood vessels. This repeated injury creates chronic acidic inflammation leading to heart disease, stroke, diabetes and obesity.

 

Let me repeat: The injury and inflammation caused from acidic foods, drinks and metabolism in our blood vessels is the cause of stokes, heart attacks, diabetes and obesity and NOT the increase of cholesterol. A low healthy fat and salt diet recommended for years by mainstream medicine will cause strokes, heart attacks, diabetes and obesity.

What are the biggest culprits of chronic acidic inflammation? Quite simply, they are the overload of simple, highly processed carbohydrates (sugar, dairy products, animal flesh, chocolate, coffee, tea, including green tea, alcohol, soda pops, vinegar, peanuts, mushrooms, flour and corn and all the products made from them) and the excess consumption of saturated vegetable oils like soybean, corn and sunflower that are found in many processed foods.

Take a moment to visualize rubbing a stiff brush repeatedly over soft skin until it becomes quite red and nearly bleeding if you kept this up several times a day, every day for five years. If you could tolerate this painful brushing, you would have a bleeding, swollen infected area that became worse with each repeated acid causing injury. This is a good way to visualize dietary and metabolic acids as the brush leading to the inflammatory process that could be going on in your body right now.

 

Regardless of where the acidic inflammatory process occurs, externally or internally, it is the same. Using Ultrasound I have peered inside thousands upon thousands of arteries. A diseased artery looks as if someone took a brush and scrubbed repeatedly against its wall. Several times a day, every day, the acidic foods we eat create small injuries compounding into more injuries, causing the body to respond continuously and appropriately with increased acid caused inflammation.

While we savor the tantalizing taste of a sweet roll, chocolate or a carbonated drink our body responds alarmingly as if a foreign invader arrived declaring war. ACIDIC foods loaded with sugars and simple carbohydrates, or processed with saturated oils for long shelf life have been the mainstay of the American diet for six decades. These acidic foods have been slowly poisoning everyone.

How does eating a simple sweet roll or a piece a chocolate create a cascade of acid causing inflammation to make you sick?

Imagine spilling acidic sugary syrup on your keyboard and you have a visual of what occurs inside the cell. When we consume simple carbohydrates such as sugar, blood sugar rises rapidly. In response, your pancreas secretes insulin and sodium bicarbonate whose primary purpose is to bind and solidify acids so they do NOT destroy healthy body and blood cells and cause internal bleeding. In addition, the body releases cholesterol to help solidify excess dietary and/or metabolic acids that have NOT been properly eliminated through the four channels of elimination – urination, perspiration, respiration and defecation.

 

The body solidifies acids to protect healthy tissues, glands and organs from ulceration and then degeneration. After years of an acidic lifestyle and diet solidified acids will build-up on the wall of the arteries and veins leading to atherosclerosis, stroke and heart attack.

What does all this have to do with inflammation? Blood sugar which is a metabolic acid is controlled in a very narrow range. Extra acidic sugar molecules that are not solidified and eliminated through the four channels of elimination will injure the blood vessel wall. This repeated acidic injury to the blood vessel wall causes irritation, inflammation, ulceration and eventual degeneration or heart disease and/or cancer. When you spike your blood sugar levels or acid levels several times a day, every day, with acidic foods or thoughts it is exactly like taking sandpaper to the inside of your delicate blood vessels.

While you may not be able to see it, rest assured, tissue, gland and organ acidosis is present. I have seen it in over 40,000 client/patients spanning over 30 years who all shared one common denominator — dietary and metabolic acid caused inflammation in their veins, arteries, glands, tissues and organs. This is what retained physiological acid looks like in the tissues using full-body thermography to show the acidic red and white hot spots.

 

Let’s get back to the sweet roll and chocolate. These innocent looking goodies not only contain the acid sugar, they are also fermented and processed in one of many saturated oils. Chips and fries are soaked in soybean oil; processed foods are manufactured with saturated oils for longer shelf life.

If the balance shifts by consuming excessive sugar, animal protein, vinegar, coffee, tea, alcohol, corn, peanuts and saturated oil, the cell membranes will be damaged and the body and blood cells will begin to degenerate causing even more acids leading to greater risk of inflammation and dis-ease.

Today’s mainstream American ACIDIC diet has produced an extreme imbalance in the alkaline design of the body and an increase in dietary and metabolic acids that cause ALL sickness and dis-ease. You read this correctly – ALL sickness and dis-ease is caused by metabolic, dietary, respiratory and/or environmental ADIDS! There are no other causes. Germs and viruses are the symptoms of cellular breakdown and NOT the cause of ANY disease. Simply said, germs do NOT cause dis-ease!

 

To make matters worse, eating these acidic foods and drinks causes the body to hold on to more fat as a depository for these excess acids that are NOT being properly eliminated through the four channels of elimination. That is why people get fat. The increase in fat is in direct relationship to the increase of acidic foods, drinks and lifestyle choices. The process that began with a sweet roll or a cup of coffee, or a piece of chocolate or a glass of wine turns into a vicious cycle over time that creates heart disease, stroke, high blood pressure, diabetes, obesity and finally, Alzheimer’s disease, as the acid caused inflammatory process continues unabated.

 

There is no escaping the fact that the more we consume prepared and processed acidic foods, the more we increase the inflammation switch little by little each day. The human body cannot process, nor was it designed to consume, foods packed with sugars, animal flesh, dairy products, vinegar, alcohol, coffee, tea, chocolate, soda pop, mushrooms, peanuts, corn, flour and saturated processed oils.

There is but one answer to quieting acid caused inflammation, and that is returning to foods closer to their natural alkaline state. To build muscle, eat more chlorophyll concentrated alkaline foods.

 

Choose carbohydrates that are very complex such as colorful fruit and vegetables. Cut out of your diet saturated oils from corn or soybean.

One tablespoon of corn oil contains 7,280 mg of saturated oil; soybean contains 6,940 mg. Instead, use olive oil, avocado oil, hemp oil or fax oil.

Forget the “science” that has been drummed into your head for decades. The science that saturated fat alone causes heart disease is non-existent. The science that saturated fat raises blood cholesterol is also very weak. Since we now know that cholesterol is not the cause of heart disease, the concern about saturated fat having no place on its hydrogen chain to buffer metabolic and dietary acid is real science. It is acid that causes disease and ALL polyunsaturated oils help to buffer excess acids by the carbon chain picking up the hydrogen ion or acid on its unsaturation. In other words, all polyunsaturated fats whether Omega 1, 3, 6 or 9 buffer or neutralize all dietary and/or metabolic acids on their unsaturated carbon.

The cholesterol theory led to the no-fat, low-fat recommendations that in turn created the very acidic foods now causing an epidemic of acid caused inflammation,induration, ulceration and degeneration. Mainstream medicine made a terrible mistake when it advised people to avoid foods high in cholesterol. We now have an epidemic of arterial acidic caused inflammation leading to heart disease and other silent killers.

Government nutrition guidelines recommend a diet high in carbohydrate regardless of the ample evidence of the health risks it promotes. Yet, heart disease and obesity rates have risen in correlation with a reduced intake of dietary fat. The Food Standards Agency states all individuals’ diets should contain “plenty of starchy foods such as rice, bread, pasta and potatoes”. In addition to this, “just a little saturated fat”. This recommendation is a recipe for heart disease and stroke because of its high level of dietary acid.

While science has moved on, nutritional advice lags behind. And in a study published in Open Heart, a group of researchers conclude that national dietary advice on fat consumption issued to millions in the 1970s to reduce the risk of heart disease which suggested that fat should form no more than 30% of daily food intake lacked any solid trial evidence and shouldn’t have been introduced.

While more circumspect, cardiologist Rahul Bahl wrote in a linked editorial:

“There is certainly a strong argument that an over-reliance in public health on saturated fat as the main dietary villain for cardiovascular disease has distracted from the risks posed by other nutrients, such as carbohydrates.”

Fat and High-Carbohydrate Foods

Some fats aren’t good – trans fats, for example, which are mostly man-made – while others, such as monounsaturated fats found in olive oil are seen as having beneficial qualities.

Today, government guidelines recommend that fats should compose no more than 35% of an individual’s daily calorie intake – and that saturated fat, in particular, ought to supply less than 11%.

Fat intake decreased from 36.6% to 33.7% from 1971 to 2006, while the intake of carbohydrates rose from 44.0% to 48.7%. Yet obesity levels have escalated.

There is evidence to also show that carbohydrates can lead to feelings of increased hunger. A recent study in The American Journal of Clinical Nutrition found that eating carbohydrate foods with a high glycemic index (bread, rice, pasta) caused effects on the brain that led to feelings of increased hunger, which could in turn lead to eating more.

Another study in 2013 found high-carb meals could leave you feeling hungrier hours later compared to a low-carb meal with more fibre, protein and fat. The team behind the research attributed this to the plummeting levels of blood sugar that regularly follows high-carb meals.

The Diet-Heart Hypothesis

At the University of Hull they have been also looking at the effects of saturated fats on triglyceride levels – a type of fat (lipid) found in the blood. Using coconut oil because of its high (90%) saturated fat content, we found that when coupled with exercise, it significantly reduced triglyceride levels. A recent Brazilian rat study also found that coconut oil and exercise could lower blood pressure.

So where does our unshakable idea that fat leads to heart disease come from? The diet-heart hypothesis, that low density lipoproteins (LDL) cholesterol is raised in the blood by eating saturated fat, which then leads to clogged arteries and eventual heart disease, is not a credible claim.

 

This theory linking saturated fat and heart disease has been around since 1955 when Ansel Keys introduced his lipid hypothesis. Despite it being the foundation of dietary recommendations, it has never been proven and we have been advised to avoid certain foods including meat, dairy products and coconuts. And these myths are so deeply embedded in our minds, that recent science advocates have seen how hard it is to challenge established thinking.

 Saturated Fat and Cholesterol

When we talk about high-density lipoprotein (HDL) or LDL – often referred to as good and bad cholesterol – we aren’t actually referring to cholesterol itself. These lipoproteins actually carry cholesterol, fat and fat soluble vitamins in the bloodstream. It appears that elevated levels of cholesterol (or more accurately, cholesterol which is transported around the blood by lipioproteins) is correlated with an increase in the risk of heart disease.

However, correlation does not mean causation. Very low cholesterol is linked with an increased risk of death (though not from heart disease). And in the very old, research suggests cholesterol can be protective. So it’s fair to say the relationship between cardiovascular disease and total cholesterol is complex.

Type of cholesterol is important. The “good” (HDL) cholesterol is strongly linked with a reduced risk of heart disease. However, LDL, the “bad” cholesterol, is associated with an increased risk of heart disease. But it turns out that there are in fact subtypes of LDL which make this black and white picture more complicated. The actual size of the LDL particle is significant. Individuals are at a heightened risk of heart disease if they have most small, dense LDL particles, that may more easily lodge in the arteries, as opposed to those who have large LDL particles.

Your blood lipid profile is frequently used as a medical screening tool for abnormalities in lipids (including triglycerides and cholesterol). These blood lipid profile tests can identify approximate risks for cardiovascular disease and specific genetic diseases. Studies have also shown that saturated fats do not harm your blood lipid profile – and can actually improve it. Saturated fats could lower the risk of heart disease by shifting LDL cholesterol from dense small LDL to large LDL.

Numerous short-term feeding trials have shown that an increase in saturated fat consumption leads to a rise in overall LDL. Nevertheless, the result is inconsistent and weak. The methods used in a number of these research studies have been criticised – and plenty of studies support the contrary, that no association exists between total LDL and saturated fat consumption.

Cause and Correlation

If it was true that saturated fat did cause heart disease, then it follows that people who consume more would be at higher risk. But observational studies – again only illustrative of correlation not cause – haven’t shown this. One study looked at a population of 347,747 subjects from a total of 21 studies and concluded that there was “no significant evidence for concluding that dietary saturated fat is associated with an increased risk of coronary heart or cardiovascular disease”. This has also been the conclusion of other reviews.

So What About Randomized Controlled Trials?

One such study divided 12,866 male subjects at a high risk of heart disease into a low-fat or Western diet group. After six years, no difference was found between them. The Women’s Health Imitative, the biggest randomized controlled trial in diet history, comprised of 48,835 postmenopausal women who were also divided into two similar groups and came up with similar findings.

The Cold-Pressed Organic Coconut Oil Connection

If you don’t care for the science, then take an everyday example. Look at the large populations of the Masai in Africa who consume large amounts of saturated fat but have low levels of coronary heart disease. Or the Tokelauans of New Zealand who consume a massive amount of saturated fat through coconuts: more than 60% of their daily calories come from coconuts. These populations have no history of heart disease. And the health benefits of coconut oil are now becoming known more widely.

 

We are learning so much more about fats and that there is no evidence that saturated fat causes heart disease. Leading nutrition experts have been calling for an amendment to dietary recommendations for more than ten years. But despite these calls and the high-quality evidence assembled throughout the past decade, doctors, governments – and by extension the public – still take extraordinarily little notice. But a decade of research to the contrary would suggest it’s time we moved away from entrenched thinking, towards a more enlightened attitude to saturated fat.

 

What you can do is choose whole, organic, raw, NON-GMO, alkaline foods your grandmother served and not those your mom turned to as grocery store aisles filled with manufactured acidic foods and drinks. By eliminating acidic causing inflammatory foods and adding essential nutrients from fresh, raw, organic, alkaline unprocessed food, you will reverse years of damage in your arteries and throughout your body from consuming the typical American ACIDIC diet.

To learn more read the following article, THE PH MIRACLE FOR HEART DISEASE – DISCOVER THE TRUTH ABOUT HEART DISEASE, CONGESTIVE HEART FAILURE, ATHEROSCLEROSIS, CHOLESTEROL, HYPERTENSION, STROKE AND MORE! –

https://phoreveryoung.wordpress.com/2015/08/06/a-self-care-to-a-self-cure-for-heart-disease-a-number-1-killer/

 

To learn more read the following article: https://www.amazon.com/gp/product/B01KBMFRA4/ref=dbs_a_def_rwt_hsch_vapi_taft_p2_i8

 

 

 

 

To learn more about the work, research, findings of publications of Robert O Young CPT, MSc, DSc, PhD and Naturopathic Practitioner go to: http://www.drrobertyoung.com

References

  1. https://www.ahajournals.org/doi/abs/10.1161/circ.130.suppl_2.1898\
  2. Ravnskov U, Diamond DM, Hama R, et al. Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly: a systematic review
  3. Office Of Dietary Supplements Fact Sheet: Folate
  4. Doshi SN, McDowell IF, Moat SJ, Payne N, Durrant HJ, Lewis MJ, Goodfellos J. Folic acid improves endothelial function in coronary artery disease via mechanisms largely independent of homocysteine. Circulation. 2002;105:22-6.
  5. Doshi SN, McDowell IFW, Moat SJ, Lang D, Newcombe RG, Kredean MB, Lewis MJ, Goodfellow J. Folate improves endothelial function in coronary artery disease. Arterioscler Thromb Vasc Biol 2001;21:1196-1202.
  6. Wald DS, Bishop L, Wald NJ, Law M, Hennessy E, Weir D, McPartlin J, Scott J. Randomized trial of folic acid supplementation and serum homocysteine levels. Arch Intern Med 2001;161:695-700.
  7. Jennings E. Folic acid as a cancer preventing agent. Med Hypothesis 1995;45:297-303.
  8. Freudenheim JL, Grahm S, Marshall JR, Haughey BP, Cholewinski S, Wilkinson G. Folate intake and carcinogenesis of the colon and rectum. Int J Epidemiol 1991;20:368-74.
  9. Giovannucci E, Stampfer MJ, Colditz GA, Hunter DJ, Fuchs C, Rosner BA, Speizer FE, Willett WC. Multivitamin use, folate, and colon cancer in women in the Nurses’ Health Study. Ann Intern Med 1998;129:517-24.
  10. A Paoloni-Giacobino, R Grimble, C Pichard. Genetics and nutrition. Clinical Nutrition Volume 22, Issue 5, Pages 429-435 (October 2003)
  11. Corradaa MM, Kawasab CH, Hallfrischc J, Mullerd D, Brookmeyere R. Reduced risk of Alzheimer?s disease with high folate intake: The Baltimore Longitudinal Study of Aging. Alzheimer’s and Dementia Volume 1, Issue 1, Pages 11-18 (July 2005).
  12. Wang HX, Wahlin Å, Basun H, Fastbom J, Winblad B, Fratiglioni L. Vitamin B12 and folate in relation to the development of Alzheimer?s disease. Neurology May 8, 2001 vol. 56 no. 9 1188-1194.
  13. Office Of Dietary Supplements Fact Sheet
  14. Appel LJ. Nonpharmacologic therapies that reduce blood pressure: A fresh perspective. Clin Cardiol 1999;22:1111-5.
  15. Simopoulos AP. The nutritional aspects of hypertension. Compr Ther 1999;25:95-100.
  16. Appel LJ, Moore TJ, Obarzanek E, Vollmer WM, Svetkey LP, Sacks FM, Bray GA, Vogt TM, Cutler JA, Windhauser MM, Lin PH, Karanja N. A clinical trial of the effects of dietary patterns on blood pressure. N Engl J Med 1997;336:1117-24.
  17. Saris NE, Mervaala E, Karppanen H, Khawaja JA, Lewenstam A. Magnesium: an update on physiological, clinical, and analytical aspects. Clinica Chimica Acta 2000;294:1-26.
  18. Institute of Medicine. Food and Nutrition Board. Dietary Reference Intakes: Calcium, Phosphorus, Magnesium, Vitamin D and Fluoride. National Academy Press. Washington, DC, 1999.
  19. Paolisso G, Sgambato S, Gambardella A, Pizza G, Tesauro P, Varricchio H, D’Onofrio F. Daily magnesium supplements improve glucose handling in elderly subjects. Am J Clin Nutr 1992;55:1161-7.
  20. Altura BM and Altura BT. Magnesium and cardiovascular biology: An important link between cardiovascular risk factors and atherogenesis. Cell Mol Biol Res 1995;41:347-59.
  21. Ford ES. Serum magnesium and ischaemic heart disease: Findings from a national sample of US adults. Intl J of Epidem 1999;28:645-51.
  22. Liao F, Folsom A, Brancati F. Is low magnesium concentration a risk factor for coronary heart disease? The Atherosclerosis Risk in Communities (ARIC) Study. Am Heart J 1998;136:480-90.
  23. Ascherio A, Rimm EB, Hernan MA, Giovannucci EL, Kawachi I, Stampfer MJ, Willett WC. Intake of potassium, magnesium, calcium, and fiber and risk of stroke among US men. Circulation 1998;98:1198-204.
  24. Elisaf M, Milionis H, Siamopoulos K. Hypomagnesemic hypokalemia and hypocalcemia: Clinical and laboratory characteristics. Mineral Electrolyte Metab 1997;23:105-12.
  25. Xing JH and Soffer EE. Adverse effects of laxatives. Dis Colon Rectum 2001;44:1201-9.
  26. Mauskop A, Altura BM. Role of magnesium in the pathogenesis and treatment of migraines. Clin Neurosci. 1998;5(1):24-27.
  27. Peikert A, Wilimzig C, Kohne-Volland R. Prophylaxis of migraine with oral magnesium: results from a prospective, multi-center, placebo-controlled and double-blind randomized study. Cephalalgia. 1996;16(4):257-263.
  28. Pfaffenrath V, Wessely P, Meyer C, et al. Magnesium in the prophylaxis of migraine–a double-blind placebo-controlled study. Cephalalgia. 1996;16(6):436-440.
  29. Wang F, Van Den Eeden SK, Ackerson LM, Salk SE, Reince RH, Elin RJ. Oral magnesium oxide prophylaxis of frequent migrainous headache in children: a randomized, double-blind, placebo-controlled trial. Headache. 2003;43(6):601-610.
  30. Bendich A. The potential for dietary supplements to reduce premenstrual syndrome (PMS) symptoms. J Am Coll Nutr. 2000;19(1):3-12.
  31. Rude RK. Magnesium deficiency: A cause of heterogeneous disease in humans. J Bone Miner Res 1998;13:749-58.
  32. Rude KR. Magnesium metabolism and deficiency. Endocrinol Metab Clin North Am 1993;22:377-95.
  33. Kelepouris E and Agus ZS. Hypomagnesemia: Renal magnesium handling. Semin Nephrol 1998;18:58-73.
  34. Ramsay LE, Yeo WW, Jackson PR. Metabolic effects of diuretics. Cardiology 1994;84 Suppl 2:48-56.
  35. Kobrin SM and Goldfarb S. Magnesium Deficiency. Semin Nephrol 1990;10:525-35.
  36. Lajer H and Daugaard G. Cisplatin and hypomagnesemia. Ca Treat Rev 1999;25:47-58.
  37. Tosiello L. Hypomagnesemia and diabetes mellitus. A review of clinical implications. Arch Intern Med 1996;156:1143-8.
  38. Paolisso G, Scheen A, D’Onofrio F, Lefebvre P. Magnesium and glucose homeostasis. Diabetologia 1990;33:511-4.
  39. Elisaf M, Bairaktari E, Kalaitzidis R, Siamopoulos K. Hypomagnesemia in alcoholic patients. Alcohol Clin Exp Res 1998;22:244-6.
  40. Abbott L, Nadler J, Rude RK. Magnesium deficiency in alcoholism: Possible contribution to osteoporosis and cardiovascular disease in alcoholics. Alcohol Clin Exp Res 1994;18:1076-82.
  41. Rude RK, Shils ME. Magnesium. In: Shils ME, Shike M, Ross AC, Caballero B, Cousins RJ, eds. Modern Nutrition in Health and Disease. 10th ed. Baltimore: Lippincott Williams & Wilkins; 2006:223-247.
  42. Food and Nutrition Board, Institute of Medicine. Magnesium. Dietary Reference Intakes: Calcium, Phosphorus, Magnesium, Vitamin D, and Fluoride. Washington D.C.: National Academy Press; 1997:190-249.
  43. Schwartz R, Walker G, Linz MD, MacKellar I. Metabolic responses of adolescent boys to two levels of dietary magnesium and protein. I. Magnesium and nitrogen retention. Am J Clin Nutr. 1973;26(5):510-518.
  44. Shils ME. Magnesium. In Modern Nutrition in Health and Disease, 9th Edition. (edited by Shils, ME, Olson, JA, Shike, M, and Ross, AC.) New York: Lippincott Williams and Wilkins, 1999, p. 169-92.
  45. Spencer H, Norris C, Williams D. Inhibitory effects of zinc on magnesium balance and magnesium absorption in man. J Am Coll Nutr. 1994;13(5):479-484.
  46. Torsten Bohn, Lena Davidsson*, Thomas Walczyk and Richard F. Hurrel Fractional magnesium absorption is signi?cantly lower in human subjects from a meal served with an oxalate-rich vegetable, spinach, as compared with a meal served with kale, a vegetable with a low oxalate content. Laboratory for Human Nutrition, Institute of Food Science and Nutrition, Swiss Federal Institute of Technology, Zurich, Switzerland (Received 27 May 2003 – Revised 7 November 2003 – Accepted 28 November 2003
  47. FDA Drug Safety Communication: Low magnesium levels can be associated with long-term use of Proton Pump Inhibitor drugs (PPIs)
  48. Leach RM, Harris ED. Manganese. In: O’Dell BL, Sunde RA, eds. Handbook of nutritionally essential minerals. New York: Marcel Dekker, Inc; 1997:335-355.
  49. Freeland-Graves J, Llanes C. Models to study manganese deficiency. In: Klimis-Tavantzis DL, ed. Manganese in health and disease. Boca Raton: CRC Press, Inc; 1994.
  50. Reginster JY, Strause LG, Saltman P, Franchimont P. Trace elements and postmenopausal osteoporosis: a preliminary study of decreased serum manganese. Med Sci Res. 1988;16:337-338.
  51. Odabasi E, Turan M, Aydin A, Akay C, Kutlu M. Magnesium, zinc, copper, manganese, and selenium levels in postmenopausal women with osteoporosis. Can magnesium play a key role in osteoporosis? Ann Acad Med Singapore. 2008;37(7):564-567.
  52. Keen CL, Zidenberg-Cherr S. Manganese. In: Ziegler EE, Filer LJ, eds. Present Knowledge in Nutrition. 7th ed. Washington D.C.: ILSI Press; 1996:334-343.
  53. Carl GF, Gallagher BB. Manganese and epilepsy. In: Klimis-Tavantzis DL, ed. Manganese in health and disease. Boca Raton: CRC Press, Inc; 1994:133-157.
  54. Blaurock-Busch, E. Wichtige Nahrstoffe fur Gesunde Haut und Haare, Kosmetik Internat. 3/87.
  55. Collipp, P.J., et al. Manganese in infant formulas and learning disability. Ann. Nutr. Metab. 27(6):488-494, 1983.
  56. “Guidelines for Niacin Therapy For the Treatment of Elevated Lipoprotein a (Lpa)”. Rush Hemophilia & Thrombophilia Center. August 15, 2002, Revised July 27, 2005. Retrieved 20 November 2009. “facial flushing is a common side effect of niacin therapy that usually subsides after several weeks of consistent niacin use”
  57. Katzung, Bertram G. (2006). Basic and clinical pharmacology. New York: McGraw-Hill Medical Publishing Division. ISBN 0071451536.
  58. Greenbaum CJ, Kahn SE, Palmer JP. Nicotinamide’s effects on glucose metabolism in subjects at risk for IDDM. Diabetes. 1996;45(11):1631-1634.
  59. Lampeter EF, Klinghammer A, Scherbaum WA, et al. The Deutsche Nicotinamide Intervention Study: an attempt to prevent type 1 diabetes. DENIS Group. Diabetes. 1998;47(6):980-984.
  60. Hageman GJ, Stierum RH. Niacin, poly(ADP-ribose) polymerase-1 and genomic stability. Mutat Res. 2001;475(1-2):45-56.
  61. Jacobson EL, Shieh WM, Huang AC. Mapping the role of NAD metabolism in prevention and treatment of carcinogenesis. Mol Cell Biochem. 1999;193(1-2):69-74.
  62. Weitberg AB. Effect of nicotinic acid supplementation in vivo on oxygen radical-induced genetic damage in human lymphocytes. Mutat Res. 1989;216(4):197-201.
  63. Tang AM, Graham NM, Saah AJ. Effects of micronutrient intake on survival in human immunodeficiency virus type 1 infection. Am J Epidemiol. 1996;143(12):1244-1256.
  64. Brown RR, Ozaki Y, Datta SP, Borden EC, Sondel PM, Malone DG. Implications of interferon-induced tryptophan catabolism in cancer, auto-immune diseases and AIDS. Adv Exp Med Biol. 1991;294:425-435.
  65. Murray MF, Langan M, MacGregor RR. Increased plasma tryptophan in HIV-infected patients treated with pharmacologic doses of nicotinamide. Nutrition. 2001;17(7-8):654-656.
  66. Office of Dietary Supplements Fact Sheet: Vitamin B6
  67. New SA, Bolton-Smith C, Grubb DA, Reid DM. Nutritional influences on bone mineral density: a cross-sectional study in premenopausal women. Am J Clin Nutr. 1997;65(6):1831-1839.
  68. New SA, Robins SP, Campbell MK, et al. Dietary influences on bone mass and bone metabolism: further evidence of a positive link between fruit and vegetable consumption and bone health? Am J Clin Nutr. 2000;71(1):142-151.
  69. Tucker KL, Hannan MT, Chen H, Cupples LA, Wilson PW, Kiel DP. Potassium, magnesium, and fruit and vegetable intakes are associated with greater bone mineral density in elderly men and women. Am J Clin Nutr. 1999;69(4):727-736.
  70. Ascherio A, Rimm EB, Hernan MA, et al. Intake of potassium, magnesium, calcium, and fiber and risk of stroke among US men. Circulation. 1998;98(12):1198-1204.
  71. Iso H, Stampfer MJ, Manson JE, et al. Prospective study of calcium, potassium, and magnesium intake and risk of stroke in women. Stroke. 1999;30(9):1772-1779.
  72. Fang J, Madhavan S, Alderman MH. Dietary potassium intake and stroke mortality. Stroke. 2000;31(7):1532-1537.
  73. Bazzano LA, He J, Ogden LG, et al. Dietary potassium intake and risk of stroke in US men and women: National Health and Nutrition Examination Survey I epidemiologic follow-up study. Stroke. 2001;32(7):1473-1480.
  74. Green DM, Ropper AH, Kronmal RA, Psaty BM, Burke GL. Serum potassium level and dietary potassium intake as risk factors for stroke. Neurology. 2002;59(3):314-320.
  75. Barri YM, Wingo CS. The effects of potassium depletion and supplementation on blood pressure: a clinical review. Am J Med Sci. 1997;314(1):37-40.
  76. Hajjar IM, Grim CE, George V, Kotchen TA. Impact of diet on blood pressure and age-related changes in blood pressure in the US population: analysis of NHANES III. Arch Intern Med. 2001;161(4):589-593.
  77. Appel LJ, Moore TJ, Obarzanek E, et al. A clinical trial of the effects of dietary patterns on blood pressure. DASH Collaborative Research Group. N Engl J Med. 1997;336(16):1117-1124.
  78. Gennari FJ. Hypokalemia. N Engl J Med. 1998;339(7):451-458.
  79. http://lpi.oregonstate.edu/infocenter/minerals/potassium/potassiumrefs.html
  80. Shearer MJ. The roles of vitamins D and K in bone health and osteoporosis prevention. Proc Nutr Soc. 1997;56(3):915-937.
  81. Booth SL. Skeletal functions of vitamin K-dependent proteins: not just for clotting anymore. Nutr Rev. 1997;55(7):282-284.
  82. Suttie JW. Vitamin K. In: Shils ME, Shike M, Ross AC, Caballero B, Cousins RJ, eds. Modern Nutrition in Health and Disease. 10th ed. Baltimore: Lippincott Williams & Wilkins; 2006:412-425.
  83. Allison (2001). The possible role of vitamin K deficiency in the pathogenesis of Alzheimer’s disease and in augmenting brain damage associated with cardiovascular disease. Medical hypotheses 57 (2): 151?5. doi:10.1054/mehy.2001.1307. PMID 11461163.
  84. ODS Fact Sheet on Coumadin – http://ods.od.nih.gov/pubs/factsheets/coumadin1.pdf
  85. Office of Dietary Suppliments Face Sheet: Vitamin C
  86. Gokce N, Keaney JF, Jr., Frei B, et al. Long-term ascorbic acid administration reverses endothelial vasomotor dysfunction in patients with coronary artery disease. Circulation. 1999;99(25):3234-3240.
  87. Audera, C (2001). “Mega-dose vitamin C in treatment of the common cold: a randomised controlled trial”. Medical Journal of Australia 389: 175.
  88. Hemilä, Harri; Chalker, Elizabeth; Douglas, Bob; Hemilä, Harri (2007). “Vitamin C for preventing and treating the common cold”. Cochrane database of systematic reviews (Online) (3): CD000980.
  89. Fleming DJ, Tucker KL, Jacques PF, Dallal GE, Wilson PW, Wood RJ (December 2002). “Dietary factors associated with the risk of high iron stores in the elderly Framingham Heart Study cohort”.
  90. The American Journal of Clinical Nutrition 76 (6): 1375?84.Institute of Medicine. Food and Nutrition Board. Dietary Reference Intakes for Vitamin C, Vitamin E, Selenium, and Carotenoids. Washington, DC: National Academy Press, 2000.
  91. Weinstein M, Babyn P, Zlotkin S. An orange a day keeps the doctor away: scurvy in the year 2000. Pediatrics 2001;108:E55.
  92. Hoffman FA. Micronutrient requirements of cancer patients. Cancer. 1985;55 (1 Suppl):295-300.
  93. Deicher R, HĂśrl WH. Vitamin C in chronic kidney disease and hemodialysis patients. Kidney Blood Press Res 2003;26:100-6.
  94. Aishah Al-Jarallah, Fatima Igdoura, Yi Zhang, Christine B Tenedero, Elizabeth J White, Melissa E Macdonald, Suleiman A Igdoura, Bernardo L Trigatti. The effect of pomegranate extract on coronary artery atherosclerosis in SR-BI/APOE double knockout mice. Atherosclerosis. 2013 May ;228(1):80-9. Epub 2013 Mar 7. PMID: 23528829
  95. Michael Aviram, Mira Rosenblat, Diana Gaitini, Samy Nitecki, Aaron Hoffman, Leslie Dornfeld, Nina Volkova, Dita Presser, Judith Attias, Harley Liker, Tony Hayek. Pomegranate juice consumption for 3 years by patients with carotid artery stenosis reduces common carotid intima-media thickness, blood pressure and LDL oxidation. Clin Nutr. 2004 Jun;23(3):423-33. PMID: 15158307
  96. GreenMedInfo.com, Pomegranate’s Anti-Inflammatory Properties
  97. Mahalaxmi Mohan, Harshal Waghulde, Sanjay Kasture. Effect of pomegranate juice on Angiotensin II-induced hypertension in diabetic Wistar rats. Phytother Res. 2009 Dec 17. PMID: 20020514
  98. Filomena de Nigris, Maria Luisa Balestrieri, Sharon Williams-Ignarro, Francesco P D’Armiento, Carmela Fiorito, Louis J Ignarro, Claudio Napoli. The influence of pomegranate fruit extract in comparison to regular pomegranate juice and seed oil on nitric oxide and arterial function in obese Zucker rats. Nitric Oxide. 2007 Aug ;17(1):50-4. Epub 2007 May 5. PMID: 17553710
  99. Filomena de Nigris, Sharon Williams-Ignarro, Vincenzo Sica, Lilach O Lerman, Francesco P D’Armiento, Russell E Byrns, Amelia Casamassimi, Daniela Carpentiero, Concetta Schiano, Daigo Sumi, Carmela Fiorito, Louis J Ignarro, Claudio Napoli. Effects of a pomegranate fruit extract rich in punicalagin on oxidation-sensitive genes and eNOS activity at sites of perturbed shear stress and atherogenesis. Cardiovasc Res. 2007 Jan 15;73(2):414-23. Epub 2006 Sep 1. PMID: 17014835
  100. Yasunori Sawayama, Kyoko Okada, Shinji Maeda, Hachiro Ohnishi, Norihiro Furusyo, Jun Hayashi. Both hepatitis C virus and Chlamydia pneumoniae infection are related to the progression of carotid atherosclerosis in patients undergoing lipid lowering therapy. Fukuoka Igaku Zasshi. 2006 Aug;97(8):245-55. PMID: 17087362
  101. M Aviram, L Dornfeld, M Rosenblat, N Volkova, M Kaplan, R Coleman, T Hayek, D Presser, B Fuhrman. Pomegranate juice consumption reduces oxidative stress, atherogenic modifications to LDL, and platelet aggregation: studies in humans and in atherosclerotic apolipoprotein E-deficient mice. Am J Clin Nutr. 2000 May ;71(5):1062-76. PMID: 10799367
  102. Adde FV, Rodrizues JC, Cardoso AL. Nutritional follow-up of cystic fibrosis patients: the role of nutrition education. J Pediatr (Rio J). 2004;80(6):475-82.
  103. Beckles Willson N, Elliot TM, Everard ML. Omega-3 fatty acids (from fish oils) for cystic fibrosis. Cochrane Database Syst Rev. 2002;(3):CD002201.
  104. Bope. Conn’s Current Therapy 2010. 1st ed. Philadelphia, PA: Saunders, An Imprint of Elsevier; 2009.
  105. Bruzzese E, Raia V, Gaudiello G, et al. Intestinal inflammation is a frequent feature of cystic fibrosis and is reduced by probiotic administration. Aliment Pharmacol Ther. 2004;20(7):813-9.
  106. Cabrera C, Artacho R, Gimenez R. Beneficial effects of green tea — a review. J Am Coll Nutr. 2006;25(2):79-99.
  107. Campbell, T.M. The China Study, BenBella Books; First Paperback Edition edition (May 11, 2006).
  108. Caramia G, Cocchi M, Garliardini R, et al. Fatty acids composition of plasma phospholipids and triglycerides in children with cystic fibrosis. The effect of dietary supplementation with an olive and soybean oils mixture. Pediatr Med Chir. 2003;25(1):42-9.
  109. Chin J. Intestinal microflora: negotiating health outcomes with the warring community within us. Asia Pac J Clin Nutr. 2004;13(Suppl):S24-5.
  110. Cvetnic Z, Vladimir-Knezevic S. Antimicrobial activity of grapefruit seed and pulp ethanolic extract. Acta Pharm. 2004;54(3):243-50.
  111. D’Agostino, Russell MW, Huse DM et al. ‘Primary and subsequent coronary risk appraisal: new results from the Framingham Study’, American Heart Journal 2000.
  112. D’Agostino, Russell MW, Huse DM et al. ‘Primary and subsequent coronary risk appraisal: new results from the Framingham Study’, American Heart Journal 2000.
  113. D’Agostino, Vasan, Pencina, Wolf, Cobain, Massaro, Kannel. ‘A General Cardiovascular Risk Profile for Use in Primary Care: The Framingham Heart Study’.
  114. D’Agostino, Wolf, Belanger, Kannel ‘Stroke Risk Profile: Adjustment for Antihypertensive Medication’, Stroke 1994.
  115. Dolinoy D.C., Weidman J.R., Waterland R.A., Jirtle R.L. (2006). Maternal Genistein Alters Coat Color and Protects Avy Mouse Offspring from Obesity by Modifying the Fetal Epigenome. Environmental Health Perspectives, 114:567-572.
  116. Dolinoy D.C., Huang D., Jirtle R.L. (2007). Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development. PNAS, 104: 13056-13061.
  117. Doron S, Gorbach SL. Probiotics: their role in the treatment and prevention of disease. Expert Rev Anti Infect Ther. 2006;4(2):261-75.
  118. Farrell P, Rosenstein B, White T, et al. Guidelines for Diagnosis of Cystic Fibrosis in Newborns through Older Adults: Cystic Fibrosis Foundation Consensus Report. Journal of Pediatrics. 2008;153(2)
  119. Ferri. Ferri’s Clinical Advisior 2010. 1st ed. Philadelphia, PA: Mosby, An Imprint of Elsevier; 2009.
  120. Gonclaves C, Dinis T, Batista MT. Antioxidant properties of proanthocyanidins of Uncaria tomentosa bark decoction: a mechanism for anti-inflammatory activity. Phytochemistry. 2005;66(1):89-98.
  121. Grey V, Mohammed SR, Smountas AA, et al. Improved glutathione status in young adult patients with cystic fibrosis supplemented with whey protein. J Cyst Fibros. 2003;2(4):195-8.
  122. Guo R, Pittler MH, Ernst E. Herbal medicines for the treatment of COPD: a systematic review. Eur Respir J. 2006;28(2):330-8.
  123. Hale LP, Greer PK, Trinh CT, James CL. Proteinase activity and stability of natural bromelain preparations. Int Immunopharmacol. 2005;5(4):783-93.
  124. Harlan M. Krumholz, MD; Teresa E. Seeman, PhD; Susan S. Merrill, PhD; Carlos F. Mendes de Leon, PhD; Viola Vaccarino, MD; David I. Silverman, MD; Reiko Tsukahara, MD; Adrian M. Ostfeld, MD; Lisa F. Berkman, PhD. Lack of Association Between Cholesterol and Coronary Heart Disease Mortality and Morbidity and All- Cause Mortality in Persons Older Than 70 Years. JAMA. 1994;272(17):1335-1340. doi:10.1001/jama.1994.03520170045034.
  125. Heggers JP, Cottingham J, Gussman J, et al. The effectiveness of processed grapefruit-seed extract as an antibacterial agent: II
  126. Mechanism of action and in vitro toxicity. J Altern Complement Med. 2002;8(3):333-40.
  127. Huang SH, Schall JI, Zemel BS, Stallings VA. Vitamin E status in children with cystic fibrosis and pancreatic insufficiency.J Pediatr. 2006;148(4):556-559.
  128. Infante P, Redecillas F, Torrent V, et al. Improvement of intestinal function in cystic fibrosis patients using probiotics. An Pediatr. 2008;69(6):501-5.
  129. Jonsdottir B, Bergsteinsson H, Baldursson O. Cystic Fibrosis–Review. Laeknabladid. 2008;94(12):831-7.
  130. Kannel, D’Agostino, Silbershatz, Belanger, Wilson, Levy. ‘Profile for Estimating Risk of Heart Failure’ – Arch Intern. Med. 1999.
  131. Kaati G., Bygren L.O., Pembrey M., Sjostrom M. (2007). Transgenerational response to nutrition, early life circumstances and longevity. European Journal of Human Genetics, 15: 784-790.
  132. Kormosh N, Laktionov K, Antoshechkina M. Effect of a combination of extract from several plants on cell-mediated and humoral immunity of patients with advanced ovarian cancer. Phytother Res. 2006;20(5):424-5.
  133. Kucharski R., Maleszka J., Foret S., Maleszka R. Nutritional Control of Reproductive Status in Honeybees via DNA Methylation (2008). Science, 319: 1827-1830 (registration required)
  134. McCabe H. Riboflavin deficiency in cystic fibrosis: three case reports. J Hum Nutr Diet. 2001;14(5):365-70.
  135. McGowan P.O., Meaney M.J., Szyf M. (2008).  Diet and the epigenetic (re)programming of phenotypic differences in behavior. Brain Research, 1237: 12-24 (subscription required).
  136. Mizejewski GJ, Pass KA. Fatty acids, alpha-fetoprotein, and cystic fibrosis. Pediatrics. 2001;108(6):1370-3.
  137. Murray KL, Lee CK, Mogayzel PJ Jr, Zeitlin PL, Rosenstein BJ. Dietary supplement use in pediatric patients with cystic fibrosis. Am J Health Syst Pharm. 2008;65(6):562-5.
  138. National Institutes of Health (U.S. Department of Health and Human Services) www.nih.gov.
  139. Nutrition Reviews 54: 1-30, 1996. Raised glutathione levels fight the oxdiation of fats circulating in the bloodstream including cholesterol, retarding the process of plaque formation in the arteries leading to most heart attacks and strokes.
  140. Olveira G, Olveira C. Nutrition, cystic fibrosis and the digestive tract. Nutr Hosp. 2008;23(2):71-86.
  141. Paterson PG, Juurlink BH. Nutritional Regulation of Glutathione in Stroke. Neurtox Res. 1999 Dec; 1(2): 99-112.
  142. Parikh, Pencina, Wang, Benjamin, Lanier, Levy, D’Agostino, Kannel, Vasan. ‘A Risk Score for Predicting Near-Term Incidence of Hypertension: The Framingham Heart Study’, Annals of Internal Medicine 2008.
  143. Pencina, D’Agostino, Larson, Massaro, Vasan. ‘Predicting the 30-Year Risk of Cardiovascular Disease: The Framingham Heart Study’, Circulation 2009.
  144. Proesmans M, Vermeulen F, De Boeck K. What’s new in cystic fibrosis? From trating symptoms to correction of the basic defect. Eur J Pediatr. 2008;167(8):839-49.
  145. RahmanI, MacNee W. Oxidative Stress and Regulation of Glutathione in Lung Inflammation. Eur Respir J. 2000 Sep; 16(3):534-54.
  146. Roum JH, Buhl R, McElvaney NG, et al. Systemic Deficiency of Glutathione in Systic Fibrosis. J Appl Physiol 1993; 75:19-24.
  147. Rubin BK. The pharmacologic approach to airway clearance: Mucoactive agents. Paediatr Respir Rev. 2006;7 Suppl 1:S215-9.
  148. Schnabel RB, Sullivan LM, Levy D, Pencina MJ, Massaro JM, D’Agostino RB, Sr., Newton-Cheh C, Yamamoto JF, Magnani JW, Tadros TM, Kannel WB, Wang TJ, Ellinor PT, Wolf PA, Vasan RS, Benjamin EJ. Development of a risk score for atrial fibrillation (Framingham Heart Study): a community-based cohort Lancet 2009;373:739-745.
  149. Simopoulos AP. Omega-3 fatty acids in inflammation and autoimmune diseases. J Am Coll Nutr. 2002;21(6):495-505.
  150. The Adult Treatment Panel III, JAMA. 2001.
  151. Wilson, Meigs, Sullivan, Fox, Nathan, D’Agostino. ‘Prediction of Incident Diabetes Mellitus in Middle-aged Adults: The Framingham Offspring Study,’ Archives of Internal Medicine 2007.
  152. Young, RO, Sick and Tired, Woodland Publishing, Orem, Utah, 2001.
  153. Yoon JH, Baek SJ. Molecular targets of dietary polyphenols with anti-inflammatory properties. Yonsei Med J. 2005;46(5):585-96.
  154. Young, RO, Young, SR, Young, The pH Miracle Revised and Updated, Grand Central Publishing, New York, NY, 2010Pollak O J. 1952, An Etiologic Concept of Atherosclerosis Based on Study of Intimal Alterations after Shock. Circulation;5;539-550. Full free paper at:http://circ.ahajournals.org/cgi/reprint/5/4/539.pdf
  155. 148. Ross R, Glomset J, Harker L. 1977. Response to injury and atherogenesis. Am J Pathol. Mar;86(3):675-8
  156. Press release. 2006. New Explanation For The Cause Of Atherosclerosis: The Acidity Theory, Medical News Today, Aug 10 at http://www.medicalnewstoday.com/articles/49244.php
  157. Press release. 2006. Beyond Lipids: Understanding the Mechanics of Atherosclerosis (press release). UCSD News, July 12. at: http://www.jacobsschool.ucsd.edu/news/news_releases/release.sfe?id=554
  158. Kaunas R, Usami S, Chien S. 2006 Regulation of stretch-induced JNK activation by stress fiber orientation. Cellular Signalling, Nov;18(11):1924-31 at http://www.ncbi.nlm.nih.gov/pubmed/16581230
  159. Haga JH, Li Yi-Shuan J. and Chien S. 2007. Molecular basis of the effects of mechanical stretch on vascular smooth muscle cells, Journal of Biomechanics, 40(5):947-60.
  160. Mesquita QHde. 1979. Myogenic Theory of Myocardial Infarction (Teoria Miogênica do Enfarte do Miocárdio, Gemini, Sao Paulo, SP – Brazil Book in Portuguese language with a summary in English at: http://www.infarctcombat.org/LivroTM/parte8.htm
  161. Mesquita QHde, Baptista CAS. 1994. Why Myogenic Theory not Thrombogenic Theory. Arq Bras Cardiol, V. 62 (4) – (Official Journal of Brazilian Cardiology Society). Full translated paper at http://www.infarctcombat.org/MTxTT-ABC.pdf
  162. Fernandes VS et al. 2006, Subclinical atherosclerosis and incipient regional myocardial dysfunction in asymptomatic individuals. The Multi-Ethnic Study of Atherosclerosis (MESA), J Am Coll Cardiol 47: 2420-8 Full free paper at http://content.onlinejacc.org/cgi/content/full/j.jacc.2005.12.075v1
  163. Marwah R, Doux J, Lee P and Yun A. 2007. Is atherosclerosis a neurogenic phenomenon? Medical Hypotheses, V 69, I 4: 884-887
  164. Selye H. 1950. The physiology and pathology of exposure to stress: A treatise based on the concepts of the general-adaptation-syndrome and the diseases of adaptation”, Montreal, Acta, Inc. / Selye H et al. 1970 Experimental Cardiovascular Diseases, Volume 1 (History, Cardiovascular Disease, Factors Influencing Cardiovascular Disease); Volume 2 (Histology and Histochemistry, Chemical and Functional Changes, References), Springer-Verlag, Berlin New York
  165. Cannon WJ. 1914. The emergency function of the adrenal medulla in pain and the major emotions. Am J Physiol. 33:356-372
  166. Benson JC, Eckert SP, McCleskey EW. 1999. Acid-Evoked Currents in Cardiac Sensory Neurons – A possible mediator of myocardial ischemic sensation, Circulation Research, 84:921-928. Full free paper at http://circres.ahajournals.org/cgi/content/full/84/8/921
  167. Gianni M et al. 2006. Apical ballooning syndrome or takotsubo cardiomyopathy: a systematic review, European Heart Journal, V27,N13: 1523-1529
  168. Akashi YJ et al. 2002. Reversible left ventricular dysfunction “takotsubo” cardiomyopathy related to catecholamine cardiotoxicity, J. Electrocardiol 2002; 35:351-356
  169. Arora S et al. 2006. Transient left ventricular apical ballooning after cocaine use; is catecholamine cardiotoxicity the pathologic link? Mayo Clin Proc. 2006; 81:820-832. Full free paper at http://www.mayoclinicproceedings.com/pdf/8106/8106cr2.pdf
  170. Wittstein IS et al. 2005. Neurohumoral features of myocardial stunning due to sudden emotional stress, New Engl J Med, Feb 10, V352: 539-548
  171. Graham LN, Smith PA et al. 2004. Sympathetic neural hyperactivity and its normalization following unstable angina and acute myocardial infarction, Clin Sci (Lond), Jun;106(6):605-11
  172. Gazes PC, Richardson JA et al. 1959. Plasma catecholamine concentrations in myocardial infarction and angina pectoris, Circulation 19:657-661
  173. Waldenstrom AP et al. 1978. A possible role of noradrenaline in the development of myocardial infarction, Am Heart J. 95:43-51
  174. Nadeau RA, de Champlain J. 1979. Plasma catecholamine in acute myocardial infarction, Am Heart J, 98: 548-554
  175. McCance AJ, Thompson PA, Forfar JC. 1993. Increased cardiac sympathetic nervous activity in patients with unstable coronary heart disease, Eur Heart J, Jun;14(6):751-7
  176. Makikalio A. 2005. Cardiovascular autonomic and hormonal dysregulation in ischemic stroke with an emphasis on survival, International Journal of Circumpolar Health 64:5
  177. Korner P. 2007. Essential Hypertension and Its Causes: Neural and Non-Neural Mechanisms. New York, Oxford University Press
  178. Rainforth MV, Schneider RH, Nidich SI, Gaylord-King C, Salerno JW, Anderson JW. 2007. Stress Reduction Programs in Patients with Elevated Blood Pressure: A Systematic Review and Metaanalysis. Curr Hypertens Rep Dec;9(6):520-8. Full free paper at: http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=18350109
  179. Barnett PA, Spence JD, Manuck SB, et al. 1997. Psychological stress and the progression of carotid artery disease, J Hypertens 15:49–55
  180. Kamarck TW, Everson SA, Kaplan GA, et al. 1997. Exaggerated blood pressure responses during mental stress are associated with enhanced carotid atherosclerosis in middle-aged Finnish men: findings from the Kuopio ischemic heart disease study. Circulation,96:3842–8 Full free paper at: http://circ.ahajournals.org/cgi/content/full/96/11/384210
  181. Jennings JR, Kamarck TW et al. 2004. Exaggerated blood pressure responses during mental stress are associated with enhanced carotid atherosclerosis in middle-aged Finnish men: findings from the Kuopio ischemic heart disease study, Circulation;110:2198-2203. Full free paper at: http://circ.ahajournals.org/cgi/content/full/110/15/2198
  182. Hauss WH et al. 1990. Adrenaline and noradrenaline as possible chemical mediators in the pathogenesis of arteriosclerosis. Ann N Y Acad Sci 598:91-101
  183. Matthews KA et al. 1998. Stress-Induced Pulse Pressure Change predicts women’s carotid atherosclerosis, Stroke 29:1525-1530
  184. Matthews KA, Zhu S, Tucker DC, Whooley MA. 2006. Blood pressure reactivity to psychological stress and coronary calcification in the Coronary Artery Risk Development in Young Adults Study, Hypertension, Mar; 47(3):391-5. Full free paper at http://hyper.ahajournals.org/cgi/content/full/47/3/391
  185. Ghiadone L et al. 2000. Mental stress induces transient endothelial dysfunction in humans, Circulation102:2473. Full free paper at http://circ.ahajounals.org/cgi/content/full/102/20/2473
  186. Steptoe A. et al. 2006. Delayed blood pressure recovery after psychological stress is associated with carotid intima-media thickness. Arterioscler. Thromb. Vasc. Biol. Nov, 26(11):2547-51
  187. Eller NH, Netterstrom. 2007. Psychosocial factors at home and at work and four-years progression in intima-media thickness. In J Behav Med 2007; 14 (1):21-29
  188. Faramawi et al. 2007. Relation between depressive symptoms and common carotid artery atherosclerosis in American persons > 65 years of age, Am J Cardiol; 99:1610-1613
  189. Schoner W. 2002. Endogenous cardiac glycosides, a new class of steroid hormones. Eur J Biochem. 268, 2440-2448, Full free paper at http://www.ejbiochem.org/cgi/content/full/269/10/2440
  190. Nesher M, Shpolansky U, Rosen H, Lichtstein D. 2007.The digitalis-like steroid hormones: New mechanisms of action and biological significance. Life Sci. May 15;80(23):2093-107
  191. Sophocleus A et al. 2003. Circulating endogenous digitalis-like factors (EDLF) in man is derived from the adrenals and its secretion is ACTH-dependent. J Endocrinol Invest Jul;26(7):668-74
  192. Weidemann H et al. 2004. Diverse effects of stress and additional adrenocorticotropic hormone on digitalislike compounds in normal and nude mice, Journal of Neuroendocrinology, Vol 16, 458-463. Full free paper at http://physiology.huji.ac.il/pdf/lichtstein/weiden-et-al04.pdf
  193. Hassan M. AM Qazzaz et al. 2004. De Novo Biosynthesis and Radiolabeling of Mammalian Digitalis-Like Factors. Clin Chem. Mar;50(3):612-20. Full free paper at http://www.clinchem.org/cgi/content/full/50/3/612
  194. Rose AM, Valdes RJ. 1994. Understanding the sodium potassium pump and its relevance to disease, Clin. Chem. 40/9: 1674-1685 Full free paper at: http://www.clinchem.org/cgi/reprint/40/9/1674
  195. Vasilyev A, Khater K, and Rakowski RF. 2004. Effect of Extracellular pH on Presteady-State and SteadyState Current Mediated by the Na+/K+ Pump,. J Membr Biol. March 15; 198(2):65–76. Full free paper at: http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1357233
  196. Li C, Geering K, Horisberger JD. 2006. The Third Sodium Binding Site of Na,K-ATPase Is Functionally Linked to Acidic pH-Activated Inward Current. Membr Biol. 213(1):1-9.
  197. Mesquita QHde, Baptista CAS. 2002. Cardiotônico: insuperåvel na preservação da estabilidade miocårdica como preventivo das síndromes coronårias agudas e responsåvel pela prolongada sobrevida, Ars Cvrandi, maio 35:3. Full free paper at http://www.infarctcombat.org/28anos/digitalicos.html . Summary in English at: http://www.infarctcombat.org/heartnews-16.html
  198. Mesquita QHde, Baptista CAS et al. 2002. Efeitos do cardiotônico + dilatador coronårio na coronåriomiocardiopatia crônica eståvel, com e sem enfarte prÊvio, a longo prazo. Ars Cvrandi, setembro;35:7. Full free paper at http://www.infarctcombat.org/qhm/cme.pdf Summary in English athttp://www.infarctcombat.org/heartnews-16.html
  199. Kern B. 1970. Der Myokard-Infarkt. Haug-Verlag, Heidelberg.
  200. Gao JRS et al. 2002. Isoform specific stimulation of cardiac Na/K pumps by nM concentrations of glycosides, J Gen Physiol 119:297-312. Full free paper at http://www.jgp.org/cgi/content/full/119/4/297
  201. Schobel HP et al. 1991.Contrasting effects of digitalis and dobutamine on baroreflex sympathetic control in normal humans, Circulation V84, 1118-1129. Full free paper at: http://circ.ahajournals.org/cgi/reprint/84/3/1118
  202. Gutman Y, Boonyaviroj P. Naunyn Schmiedebergs. 1977. Mechanism of inhibition of catecholamine release from adrenal medulla by diphenylhydantoin and by low concentration of ouabain (10 (-10) M). Arch Pharmacol Feb;296(3):293-6
  203. von Ardenne M. 1978. Die Hemmung der mikrozirculation beim myokardinfarkt und das perlingual applizierte g-strophanthin, Arzneimittel-Forsch. 28; 202:
  204. Pierre SV et al. 2007. Ouabain triggers preconditioning through activation of the NA+, K+-ATPase signalling cascade in rat hearts, Cardiovasc Res, Feb 1;73(3): 488-96
  205. Pugin J, Dunn-Siegrist I, Dufour J, Tissieres P, Charles PE, Comte R. 2007. Cyclic Stretch of Human Lung Cells Induces an Acidification and Promotes Bacterial Growth, Am J Respir Cell Mol Biol. Oct 5 doi:10.1165/rcmb.2007-0114OC
  206. Levy B, Gibot S, Franck P, Cravoisy A, Bollaert PE. 2005. Relation between muscle Na+K+ ATPase activity and raised lactate concentrations in septic shock: a prospective study. Lancet. Mar 5-11;365(9462):871-5.
  207. Schade DS.1982. The role of catecholamines in metabolic acidosis. Ciba Found Symp;87:235-53
  208. Abarquez RF Jr. 1967. Digitalis in the treatment of hypertension. A preliminary report. Acta Med Philipp. Jan-Mar;3(3):161-70
  209. Yuan CM, Manunta P, Hamlyn JM et al. 1993. Long-term ouabain administration produces hypertension in rats. Hypertension, 3;22;178-187 Full free paper at: http://hyper.ahajournals.org/cgi/reprint/22/2/178
  210. Manunta, P., Hamilton, J., Rogowski, A.C., Hamilton, B.P., Hamlyn, J.M. 2000. Chronic hypertension induced by ouabain but not digoxin in the rat:antihypertensive effect of digoxin and digitoxin. Hypertension Research 23 (Suppl), S77–S85.
  211. Yang Q, Huang W, Jozwik C, Lin Y, Glasman M et al. 2005. Cardiac glycosides inhibit TNF-alpha/NF-kappaB signaling by blocking recruitment of TNF receptor-associated death domain to the TNF receptor. Proc Natl Acad Sci USA Jul 5;102(27):9631-6. Full free paper at http://www.pnas.org/cgi/content/full/102/27/963111
  212. Sternberg EM. 2001. Neuroendocrine regulation of autoimmune/inflammatory disease, J Endocrinol Jun; 169(3):429-35. Full free paper at http://joe.endocrinology-journals.org/cgi/reprint/169/3/429
  213. Brum PC, Kosek J, Patterson A et al. 2002. Abnormal cardiac function associated with sympathetic nervous system hyperactivity in mice. Am J Physiol Heart Circ Physiol 283: H1838-H1845. Full free paper at http://ajpheart.physiology.org/cgi/content/full/283/5/H1838#B4
  214. F. E. Demartini, P. J. Cannon, W. B. Stason, and J. H. Laragh. 1965. Lactic Acid Metabolism in Hypertensive Patients. Science 11 June, Vol. 148. no. 3676, pp. 1482 – 1484
  215. Sharda S, Gupta SN and Khuteta KP. 1975. Effect on mental stress on intermediate carbohydrate-and lipidmetabolism. Indian J Physiol Pharmacol. Apr-Jun;19(2):86-9.
  216. Hall JB, Brown DA. 1979. Plasma glucose and lactic acid alterations in response to a stressful exam. Biol Psychol. May;8(3):179-88.
  217. von Ardenne M, Reitnauer PG. 1989. Increase of perfusion pressure at constant perfusion rate caused by low pH values, Biomed Biochim Acta, 48(4):317-23
  218. Yasushi Horai et al. 2005. Changes in pH increase perfusion pressure of coronary arteries in the rat. J Pharmacol Sci 97; 400: 407
  219. Austin C, Wray S. 2000. Interactions Between Ca2+ and H+ and Functional Consequences in Vascular Smooth Muscle, Mini Review, Circulation Research 86:355. Full free paper at: http://circres.ahajournals.org/cgi/content/full/86/3/355
  220. Kim YM et al. 2005. Contribution of Na_-K_ pump and KIR currents to extracellular pH-dependent changes of contractility in rat superior mesenteric artery, Am J Physiol Heart Circ Physiol 289:792-800 Full free paper at http://ajpheart.physiology.org/cgi/reprint/289/2/H792
  221. Carter G, Gavin JB. 1989. Endocardial damage induced by lactate, lowered pH and lactic acid in nonischemic beating hearts. Pathology Apr;21(2):125-30
  222. Sharma AM, Kribben A et al. 1990. Salt sensitivity in humans is associated with abnormal acid-base balance. Hypertension; 16, 407-413. Full free paper at http://hyper.ahajournals.org/cgi/reprint/16/4/407
  223. Harold T. Edwards, Edward H. Bensley, David B. Dill and Thorne M. Carpenter. 1944. Human Respiratory Quotients in Relation to Alveolar Carbon Dioxide and Blood Lactic Acid After Ingestion of Glucose, Fructose, or Galactose. Journal of Nutrition Vol. 27 No. 3 March, pp. 241-251. Full free paper at http://jn.nutrition.org/cgi/reprint/27/3/241
  224. Hallfrisch J. 1990. Metabolic effects of dietary fructose. FASEB J, Vol 4; Jun: 2652-2660. Full free paper at http://www.fasebj.org/cgi/reprint/4/9/2652.pdf
  225. Mesquita QHde. 1982. Aspectos angiogrĂĄficos coronĂĄrios e ventriculograficos do primeiro enfarte do miocĂĄrdio em coronariopatia crĂ´nica silenciosa. Rev. Bras. Med., V 39: N7
  226. LA Naves and McCleskey EW. 2005. An acid-sensing ion channel that detects ischemic pain. Braz J Med Biol Res, 38 (11) 1561-69 http://www.scielo.br/pdf/bjmbr/v38n11/v38n11a01.pdf
  227. Vogt AM, Ackermann C, Yildiz M, Schoels W, KĂźbler W. 2002. Lactate accumulation rather than ATP depletion predicts ischemic myocardial necrosis: implications for the development of lethal myocardial injury, Biochim Biophys Acta Mar 16;1586(2):219- 26.
  228. Todd GL, Baroldi G, Pieper GM, Clayton FC, Eliot RS. 1985. Experimental catecholamine- induced myocardial necrosis. I. Morphology, quantification and regional distribution of acute contraction band lesions. J Mol Cell Cardiol. Apr 17(4):317- 38.
  229. Henning RJ, Well MH, Weiner F. 1982. Blood lactate as prognostic indicator of survival in patients with acute myocardial infarction. Circ Shock, 9(3):307-15
  230. Vikhert AM, Cherpachenko NM. 1985. Histoenzymological characteristics of the myocardium in sudden cardiac death. Arkh Patol 47(7):29-34
  231. Huang Y, McNamara JO. 2004. “Ischemic Stroke: “Acidotoxicity” Is a Perpetrator”, Cell, Volume 118, Issue 6, 17 September, Pages 665-666Tennant R. 1935. Factors concerned in the arrest of contraction in an ischemic myocardial area. Am J Physiol: 133; 677-682
  232. Katz AM, Hecht H. H. 1969. The early pump failure of the ischaemic heart. Am J Med: 47; 497-502
  233. Elharrer V, Zipes D.P. 1977. Cardiac electrophysiologic alterations during myocardial ischaemia. Am J Physiol: 233: H329-345
  234. Pan HL et al. 1999. Role of protons in activation of cardiac sympathetic C-fibre afferents during ischaemia in cats. J Physiol. Aug 1;518 ( Pt 3):857-66. Full free paper at http://jp.physoc.org/cgi/content/full/518/3/857
  235. Leake DS. 1997. Does an acidic pH explain why low density lipoprotein is oxidised in atherosclerotic lesions? Atherosclerosis. Mar 21;129(2):149- 57
  236. Gown MA, Benditt PE. 1982. Lactate dehydrogenase (LDH) isozymes of human atherosclerotic plaques. Am J Pathol 1982, 107:316-321
  237. Morgan J, Leake DS. 1995. Oxidation of low density lipoprotein by iron or copper at acidic pH. J Lipid Res. Dec;36(12):2504- 12. Full free paper at: http://www.jlr.org/cgi/reprint/36/12/2504
  238. Patterson RA, Leake DS. 1998. Human serum, cysteine and histidine inhibit the oxidation of low density lipoprotein less at acidic pH. FEBS Lett. Sep 4;434(3):317- 21.
  239. Naghavi M et al. 2002. pH Heterogeneity of human and rabbit atherosclerotic plaques; a new insight into detection of vulnerable plaque. Atherosclerosis Sep, V 164; 1:27-35
  240. Khan T, Soller B, Naghavi M, Casscells W. 2005. Tissue pH determination for the detection of metabolically active inflamed vulnerable plaques using near-infrared spectroscopy: an in-vitro feasibility study. Cardiology.; 103(1): 10-6.
  241. Sneck M, Kovanen PT, Oorni K. 2005. Decrease in pH strongly enhances binding of native, proteolysed, lipolysed, and oxidized low density lipoprotein particles to human aortic proteoglycans, Journal of Biological Chemistry, 280;45: Nov. Full free paper at http://www.jbc.org/cgi/reprint/280/45/37449
  242. Oorni K and Kovanen PT. 2006. Enhanced extracellular lipid accumulation in acidic environments. Curr Opin Lipidol 17(5);534-40: Oct
  243. Patterson RA, Horsley ETM, Leake DS. 2003. Prooxidant and antioxidant properties of human serum ultrafiltrates toward LDL: important role of uric acid. Journal of Lipid Research, Vol. 44, 512-521, March Full free paper at http://www.jlr.org/cgi/reprint/44/3/51212
  244. Hayden MR, Tyagi SC. 2004. Uric acid: A new look at an old risk marker for cardiovascular disease, metabolic syndrome, and type 2 diabetes mellitus: The urate redox shuttle. Nutr Metab (Lond). 1: 10, October 19. Full paper at http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=529248
  245. McCully KS. 1969. Vascular pathology of homocysteinemia: implications for the pathogenesis of atherosclerosis. Am J Pathology 56:111:28
  246. Stoney CM. 1999. Plasma homocysteine levels increase in women during psychological stress, Life Sci 64(25):2359-65
  247. Stoney CM and Engebretson TO. 2000. Plasma homocysteine concentrations are positively associated with hostility and anger, Life Sci 66(23):2267-75
  248. Hapuarachchi JR, Chalmers AH et al. 2003. Changes in clinically relevant metabolites with psychological stress parameters. Behav Med. Summer;29(2):52-9
  249. Jerlich A et al. 1999. Correlation of low-density lipoprotein modification by myeloperoxidase with hypocholorous acid formation, Int. J. Clin, Lab, Res 29(4):155-61
  250. Podrez EA, Abu-Soud HM, Hasen SL. 2000. Myeloperoxidase-generated oxidants and atherosclerosis. Free Radic Biol Med 28:1717–172
  251. Yang J, Cheng Y, Ji R, Zhang C. 2006. Novel model of inflammatory neointima formation reveals a potential role of myeloperoxidase in neointimal hyperplasia. Am J Physiol Heart Circ Physiol. Dec;291(6):H3087- 93.
  252. Meuwese MC, Stroes ESG, Hazen SL, et al. 2007. Serum myeloperoxidase levels are associated with the future risk of coronary artery disease in apparently healthy individuals: The EPIC-Norfolk Prospective Population Study..J Am Coll Cardiol 50:159-165
  253. Wong ML et al. 2000. Acute systemic inflammation up-regulates secretory sphingomyelinase in vivo: A possible link between inflammatory cytokines and atherogenesis, PNAS 97;8681-8686 Full free paper at http://www.pnas.org/cgi/content/full/97/15/8681
  254. Abela GS. 2006. Plaque Rupture by Cholesterol Crystallization – A Novel Concept for Acute Coronary Syndrome, American College of Cardiology Annual Scientific Session, March 13, Full free paper at http://www.cardiosource.com/rapidnewssummaries/summary.asp?SumID=164
  255. Malek AM, Alper SL, Izumo S. 1999. Hemodynamic shear stress and its role in atherosclerosis JAMA 282: 2035-2042
  256. Cheng C et al. 2006. Atherosclerotic lesion size and vulnerability are determined by patterns of fluid shear stress. Circulation 113:2744-2753. Full free paper at at: http://circ.ahajournals.org/cgi/content/abstract/113/23/2744
  257. Cunningham KS and Gotlieb AI. 2005. The role of shear stress in the pathogenesis of atherosclerosis (Mini review), Laboratory Investigation 85, 9-23, Full free paper at: http://www.nature.com/labinvest/journal/v85/n1/full/3700215a.html
  258. Texon M. 1957. A hemodynamic concept of atherosclerosis, with particular reference to coronary occlusion. Arch Intern Med 99:418–42
  259. Imparato AM, Lord JW Jr, Texon M, Helpern M. 1961. Experimental atherosclerosis produced by alteration of blood vessel configuration. Surg Forum 12:245–247.
  260. Rittersma SZH, van der Wal AC, Koch KT, et al. 2005. Plaque instability frequently occurs days or weeks before occlusive coronary thrombosis. A pathological thrombectomy study in primary percutaneous coronary intervention. Circulation; 111:1160-1165. Full free paper at: http://circ.ahajournals.org/cgi/content/full/111/9/1160
  261. Ojio S, Takatsy H, et al. 2000. Considerable time from the onset of plaque rupture and/or thrombi until the onset of acute myocardial infarction in humans coronary angiographic findings within 1 week before the onset of infarction. Circulation;102:2063. Full free paper at:http://www.circ.ahajournals.org/cgi/reprint/102/17/206
  262. Ulrich E. Heidlan, Bodo E. Strauer. 2001. Left ventricular muscle mass and elevated heart rate are associated with coronary plaque disruption, Circulation 104:1477. Full free paper at: http://circ.ahajournals.org/cgi/content/full/104/13/1477
  263. Baroldi G, Bigi R, Cortigiani L. 2004. Ultrasound imaging versus morphopathology in cardiovascular diseases. Coronary collateral circulation and atherosclerotic plaque. Cardiovascular ultrasound; 3: 6. Full free paper at: http://www.cardiovascularultrasound.com/content/3/1/6
  264. Roberts W. C. 1974. Coronary Thrombosis and Fatal Myocardial Ischemia. Circulation;49;1-3 Full free paper at http://circ.ahajournals.org/cgi/reprint/49/1/1.pdf
  265. Rioufol G, Finet G, Andre-Fouet X et al. 2002. Multiple atherosclerotic plaque rupture in acute coronary syndrome: a three-vessel intravascular ultrasound study. Circulation; 106:804-808. Full free paper at: http://www.circ.ahajournals.org/cgi/reprint/01.CIR.0000025609.13806.31v1
  266. Yasunori Ueda, Masanori Asakura, et al. 2001. The healing process of infarct-related plaque: Insights from 18 months of serial angioscopic follow-up. Am Coll Cardiol, 38:1916-1922.Full free paper at: http://content.onlinejacc.org/cgi/reprint/38/7/1916
  267. Giorgio Baroldi, Riccardo Bigi and Lauro Cortigiani.2005. Ultrasound imaging versusmorphopathology in cardiovascular diseases. Myocardial cell damage. Cardiovascular Ultrasound 3:32. Full free paper at http://www.cardiovascularultrasound.com/content/3/1/32
  268. Murakami T, Mizuno S, Takahashi Y, Ohsato K et al. 1998. Intracoronary aspiration thrombectomy for acute myocardial infarction, Am. J Cardiology Oct 1;82 (7):839-44
  269. Wang HX, Leineweber C, et al. 2007. Psychosocial stress and atherosclerosis: family and work stress accelerate progression of coronary disease in women. The Stockholm Female Coronary Angiography Study. Journal of Internal Medicine 261;245-254
  270. Richmond AC et al. 2000. Effects of stress reduction on carotid atherosclerosis in hypertensive African Americans, Stroke 31:568-573. Full free paper at: http://stroke.ahajournals.org/cgi/reprint/31/3/568
  271. Fields JZ et al. 2002. Effect of a multimodality natural medicine program on carotid atherosclerosis in older subjects: a pilot trial of Maharishi Vedic Medicine, American Journal of Cardiology, 89; 8:952-958
  272. Manchanda SC, Narang R, Reddy KS, Sachdeva U, Prabhakaran D, Dharmanand S, Rajani M and Bijlani R. 2002. Retardation of coronary atherosclerosis with yoga lifestyle prevention, J Assoc Physicians India Jul; 48(7): 687-94 13.
  273. Rainer Rauramaa et al. 2004. Effects of aerobical physical exercise on inflammation and atherosclerosis in men: The DNASCO Study. Annals of Internal Medicine, 15 June, 140:12:1007-1014,
  274. Lichtor T et al. 1987.The sympathetic nervous system and atherosclerosis. J Neurosurg Dec;67(6):906-1,Pauletto P et al. 1991. Sympathetic drive and vascular damage in hypertension and atherosclerosis, Hypertension Apr;17(4 Suppl):III75-81.
  275. Wikstrand J, Berglund G, Hedblad B, Hulthe, Wikstrand J. 2003.Anti-atherosclerotic effects of beta-blockers. Am J Cardiol. Jun 19;91(12A):25H-29H.
  276. Sipahi I et al. 2007. B-Blockers and progression of coronary atherosclerosis; Pooled analysis of 4 intravascular trials. Annals of Internal Medicine, 3 July, V147; Issue 1: 10-18
  277. Mesquita QHde, Kerbrie SV, Mari SM, Baptista CA, Monteiro J, Maciel MC. 1978. Preservação funcional do miocårdio isquêmico pelo cardiotonico a longo prazo: recateterização de 29 casos. Medicina de Hoje, março 1978
  278. Hansson GK. 2005. Inflammation, atherosclerosis and coronary artery disease, NEJM V 352; N16 April 21.
  279. Malcolm Kendrick. 2007. Are statins overused? Future Lipidol, 2 (5)
  280. Player MS, King DE, et al. 2007. Psychosocial Factors and Progression From Prehypertension to Hypertension or Coronary Heart Disease, Ann Fam Med ;5(5):403-411. Full free paper at http://www.medscape.com/viewarticle/565806?src=mp.
  281. Palatini P, Longo D, Zaetta V, Perkovic D, Garbelotto R, Pessina AC. 2006. Evolution of blood pressure and cholesterol in stage 1 hypertension: role of autonomic nervous system activity, J Hypertens.Jul;24(7):1375-81.
  282. Grassi G, Quarti-Trevano F, Seravalle G, Dell’Oro R. 2007. Cardiovascular risk and adrenergic overdrive in the metabolic syndrome. Nutr Metab Cardiovasc Dis Jul; 17(6): 473-81.
  283. Choi CS, Kiim YB, Lee FN, et al. 2002. Lactate induces insulin resistance in skeletal muscle by suppressing glycolysis and impairing insulin signaling. Am J Physiol Endocrinol Metab 283: E233–E240, 2002. Full free paper at: http://ajpendo.physiology.org/cgi/content/full/283/2/E233,
  284. Tentolouris N, Tsigos C, Perea D et al. 2003. Differential effects of high-fat and high-carbohydrate isoenergetic meals on cardiac autonomic nervous system activity in lean and obese women. Metabolism. Nov;52(11):1426- 32.
  285. Calynn Davis Bunol, 2005. Thesis, Autonomic nervous system modulation of the heart following a high carbohydrate liquid meal, December. Full free paper at http://etd.lsu.edu/docs/available/etd-09082005-165133/unrestricted/Bunol_thesis.pdf
  286. Erkilla AT, Matthan NR, et al. 2006. Higher plasma docosahexaenoic acid is associated with reduced progression of coronary atherosclerosis in women with CAD. J Lipid Res; 47: 2814-19 Full free paper at http://www.jlr.org/cgi/reprint/47/12/2814.
  287. Ogilve GK, Fettman MJ et al. 2000. Effect of fish oil, arginine, and doxorubicin chemotherapy on remission and survival time for dogs with lymphoma: A double-blind, randomized placebo-controlled study, Cancer; 88: 1016-28. Full free paper at: http://www3.interscience.wiley.com/cgibin/fulltext/75504731/PDFSTART
  288. Graziani Y. 1977. Regulation of cyclic AMP level and lactic acid production in Ehrlich ascites tumor cells. Biochim Biophys Acta April 27;497(2):499-506.
  289. Nazam Ansari N, Bhandari U, Pillai KK. 2007. Protective role of curcumin in myocardial oxidative damage induced by isoproterenol in rats. Hum Exp Toxicol, Dec;26(12):933-8.
  290. Dernek S et al. 2004. Cardioprotection with resveratrol pretreatment: improved beneficial effects over standard treatment in heart rats after global ischemia. Scand Cardiovasc J Aug;38(4):245-54.
  291. Al Makdessi S, Sweidan H, MĂźllner S, Jacob R. 1996. Myocardial protection by pretreatment with Crataegus oxyacantha: an assessment by means of the release of lactate dehydrogenase by the ischemic and reperfused Langendorff heart. Arzneimittelforschung Jan;46(1):25-7.
  292. Leor J, Goldbourt U et al. 1995. Digoxin and increased mortality among patients recovering from acute myocardial infarction: importance of digoxin dose, Cardiovasc Drugs Ther. Oct;9(5):723-
  293. Adams KF Jr, Patterson JH et al. 2005. Relationship of serum digoxin concentration to mortality and morbidity in women in the digitalis investigation group trial: a retrospective analysis. J Am Coll Cardiol. Aug 2;46(3):497-504.
  294. Wycoff C.C. 1969. New Concepts of Digitalis, Calif Med. 1969 December; 111(6): 423–432. Full free paper at http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1503737.
  295. T Bjornheden, M Levin, M Evaldsson, O Wiklund. 1999. Evidence of hypoxic areas within the arterial wall in vivo, Arteriosclerosis, Thrombosis and Vascular Biology; 19:870-87.
  296. Quick AJ. 1935. The effect of exercise on the excretion of uric acid. The Journal of Biological Chemistry. Full free paper at: http://www.jbc.org/cgi/reprint/110/1/107.pdf
  297. Flierl MA, Rittirsch D, Nadeau BA et al. 2007. Phagocyte-derived catecholamines enhance acute inflammatory injury. Nature Oct 11;449 (7163):721-5
  298. Lee KS and Klaus W. 1971. The subcellular basis for the mechanisms of inotropic action of cardiac glycosides. Pharmacol Rev 23:193-261
  299. Wen Y, Leake DS. 2007. Low density Lipoprotein oxidation undergoes within lysosome in cells. Circ.Res. 100;1337-1343. Full free paper at http://circres.ahajournals.org/cgi/content/full/100/9/1337
  300. Marshall MW and Iacono JM (1976). Changes in lactate dehydrogenase, LDH isoenzymes, lactate, and pyruvate as a result of feeding low fat diets to healthy men and women. Metabolism. 1976 Feb;25(2):169-78.
  301. Yoshimura T, Miyoshi T, et al. (1986). Effect of high carbohydrate diet on serum lactate
  302. dehydrogenase isozyme pattern in Japanese young men. Acta Biol Hung. 1986;37(3-4):243-8.

The pH Miracle for Heart Disease – Discover the Truth About Heart Disease, Congestive Heart Failure, Atherosclerosis, Cholesterol, Hypertension, Stroke and More!

Introduction

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Heart Disease has many symptomologies including, Atherosclerosis, Coronary Artery Disease (CAD), Carotid Artery Disease (CAD), Peripheral Arterial Disease (PAD), Hypertension, Hypercholesterolemia, Congestive Heart Failure (CHF) and Death
According to my theory ALL of the symptoms or conditions of Heart Disease are ALL the result of an excess of dietary, environmental, respiratory and/or metabolic acid that have not been properly eliminated through the four channels of elimination.  The four channels of elimination include urination, defecation, respiration and perspiration.
When metabolic and/or dietary acidic waste is not eliminated through the four channels of elimination they are either solidified by the body with alkaline compounds or pushed out into the connective and fatty tissues away from the organs and glands that sustain life.
Acidic metabolic and dietary waste when not eliminated from the blood or lymph fluid  is solidified and buffered with alkaline compounds such as sodium, potassium, calcium and/or LDL cholesterol forming plaque or acid crystals that build up on the inside of the arteries, veins and lymphatic vessels.  Plaque is made up of alkalizing fats, cholesterol, calcium, sodium bicarbonate, potassium and other alkaline substances found in the blood and interstitial fluids.  Over time, acid bound plaque hardens or crystallizes forming a solid mass and narrows the arteries, veins and lymphatic vessels as seen in this picture.  7f871-scan0183The flow of oxygen-rich blood and alkalizing compounds to the tissues, organs and glands that sustain life  is reduced due to the narrowing of the blood and lymph vessels.  The restriction of blood and lymph circulation and the build-up of metabolic and/or dietary acids can then lead to tissue, organ and gland ulceration and/or degeneration causing a stroke or heart attack leading to heart failure and sudden death.
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Heart Disease is  Epidemic – Something Has Really Gone Wrong!

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Today over 25% of the US population takes expensive and highly acidic statin medications and despite the fact we have reduced the fat content of our diets, more Americans will die this year of heart disease than ever before caused by acidic drugs and acidic lifestyles.
Statistics from the American Heart Association show that 75 million Americans currently suffer from heart disease, 20 million have diabetes and 57 million have pre-diabetes. These disorders are affecting younger and younger people in greater numbers every year.

No health topic is more important, more full of misinformation, and more complex than understanding the cause and effect relationship between inflammation and cardiovascular disease. This article tackles the four most common conditions associated with inflammation and  “heart disease”, including all of its symptomologies: hypertension, stroke, coronary heart disease, and congestive heart failure.  I believe the information herein will be life changing and life saving for all heart dis-ease problems.

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Cardiovascular or heart disease is the number one killer in the United States, and yet it does not induce terror or fear as cancer does. The reason for this is because people are horrified of the acidic cancer treatments or chemotherapy, radiation and surgery and NOT the disease itself.  Alas, heart health is frequently ignored in lieu of cancer concerns, with breast cancer being an excellent example of this short sightedness. Case in point: For every woman who dies of breast cancer or from the cancer treatments (70 percent or higher) 11 more will die from coronary heart disease. About 60% of heart disease deaths happen suddenly in people who had no previous symptoms and normal LDL cholesterol levels. These people simply collapse unexpectedly. The real lesson here is to be educated, as you will see.  Everything that we have been taught about cardiovascular/heart disease is WRONG!  That’s right EVERYTHING!  If the so-called experts whom many of us have been listening to were right, than cardiovascular/heart disease would not be the #1 failure of modern-day medicine.

Adhering to a critical holistic principle of non-invasive alternative medicine or true health care, it is important that we never adopt the foolishness of a typical physician, who will obsessively focus on only the organ displaying obvious symptoms. It is wiser to treat the whole patient instead of an organ, which means correcting problems with both lifestyle and diet. As is the case for practically all other chronic dis-eases! A heart patient usually brings the dis-ease upon himself or herself with his or irresponsible acidic lifestyle behavior for a period of many years, and only he/she can get himself/herself out of this acidic mess. Acidic toxic drugs can suppress symptoms for a while, like they do, but real change comes only from real changes in what you eat, what you drink and what you think.  Merely masking the symptoms with toxic acidic drugs is the health policy of fools. The time bomb for a heat attack or stroke is still ticking, even though the timer has been hidden from view with useless modern-day big Pharma drugs.

Continue reading The pH Miracle for Heart Disease – Discover the Truth About Heart Disease, Congestive Heart Failure, Atherosclerosis, Cholesterol, Hypertension, Stroke and More!

Drinking Coffee Causes Heart Disease, Cancer and Many Other Serious Dis-Ease Conditions!

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The United States buys nearly one-half of the world’s supply of coffee beans. It is said that a food service operation can stand or fall on its reputation of the coffee it serves. Every mid-morning and mid-afternoon working day millions of office and factory workers abandon jobs for an employer-paid “coffee break.”

Over 15 million Americans are coffee addicts, and most of them don’t even know it, so insidious is its addictive onslaught. The child or adult may unknowingly ingest several hundred milligrams of caffeine daily.

Like narcotics, alcohol, or cigarettes, coffee and caffeinated beverages are addictive, destructive drugs that each year predispose millions of Americans to crippling illnesses and sometimes fatal diseases.

Coffee and black tea are the two most popular beverages in America. Coffee is America’s number one addictive drug problem leading to hypertension, hypercholesterolemia, and other heart dis-ease problems.

Caffeine Withdrawal

Caffeine withdrawal can occur from missing just one cup of coffee in the morning. Symptoms of caffeine withdrawal are headaches, irritability, inability to work effectively, nervousness, restlessness, and lethargy. A steady user of caffeine may, at times, experience tight headaches in the back of the neck area and be quick to anger or irritation.

Caffeine Acts as a Stimulant

Caffeine is a toxic acidic stimulant. This is not natural for the body. It activates the “fight or flight” response.

It’s important to remember that the caffeine in coffee is a powerful substance. It can stimulate the central nervous system, increase heartbeat and metabolic rate, increase the secretion of stomach sodium bicarbonate, and step up kidney and bladder action. It’s also well known for its annoying ability to affect sleep.i

At higher doses, caffeine can cause “coffee nerves”—a wide assortment of symptoms including anxiety, irritability, headaches, light-headedness, nausea, and diarrhea.

Coffee can cause a temporary increase in blood sugar, but it is quickly followed by a decrease, and stimulates the release of adrenaline, which causes body tissues to be broken down into sugar giving rise to high blood sugar associated with diabetes. Too much insulin is produced, and the blood sugar falls to a low level.

Caffeine is an Acidic Poison

Caffeine, which is the main chemical in coffee, is a powerful poison! A drop of caffeine injected into the skin of an animal will produce death within a few minutes. An infinitely small amount injected into the brain will cause convulsions. The amount of caffeine in a cup of coffee is quite small, yet people drink coffee because of the effect of the caffeine, just as people  smoke because of the effect of the nicotine. Both are drugs, and both are habit-forming.  Both are poisons.  And both will eventually kill you by causing heart failure or a cancerous conditions.

Coffee Drinking and Stomach Ulcers

The general public usually associates ulcers and heart trouble with coffee drinking. J.A. Roth and A.C. Ivy, whose experiments on coffee are famous, state this:

“Caffeine produces gastro-duodenal ulcers in animals to whom the drug is given in a beeswax container so that their stomachs are absorbing caffeine continually. Also, caffeine produces very definite changes in the blood vessels of animals, which are similar to changes produced by prolonged resentment hostility and anxiety.”

Coffee Linked to Hip Fractures

“People who drink more than two cups of coffee or four cups of black or green tea a day could be increasing their risk of hip fracture in old age, according to a new study.”  The study, published in the October issue of the American Journal of Epidemiology, is the first to link caffeine consumption with hip fractures that occur in older people whose bones have weakened. A hip fracture often marks an elderly person’s final decline into dependency or death.

Brown University’s Dr. Douglas P. Kiel and his colleagues looked at how much coffee or tea 3170 people reported drinking over 14 years. They then looked to see which ones fractured their hips, a sign that bones had become brittle. They found that heavy caffeine drinkers were 53% more likely to suffer hip fractures.

Infertility

Trying to become pregnant? Stop drinking caffeinated drinks. Among 104 women, those who drank just one cup of caffeinated coffee a day were half as likely to become pregnant during any given menstrual cycle as those who drank less, according to a 1988 study by Allen Wilcox of the National Institute of Environmental Health Sciences.

Most of the studies conducted since then have also found that caffeine impairs fertility, but usually only at three or more cups of regular coffee a day.

But the research is only as good or bad as the women’s memories. For example, scientists at John Hopkins University found that among 2500 women who were trying to become pregnant, consuming more than 300 mg of caffeine a day reduced their chances of succeeding in any given month by 17%. But those results were based on the amount of coffee and soft drinks the women could remember having consumed as many as ten years earlier.

Even so, “it’s probably prudent for women who are trying to become pregnant, and especially for those having trouble, to cut back on caffeine,” says Mark Klebanoff of the National Institute of Child Health and Human Development in Bethesda, Maryland.

Does Coffee Cause Heart Disease and  Cancer?

There is mounting evidence suggesting that if you want to avoid certain heart and cancerous conditions, you are well-advised to kick the coffee habit. Consider these examples:

One study revealed that not only was coffee drinking associated with increased risk of bladder cancer, but the drinking of non-diet cola drinks also was linked to this problem.

Coffee drinking increases the risk of birth defects.

Coffee drinking increases blood pressure, increasing the risk of heart disease.

It is commonly thought that the drinking of coffee, soft drinks, and other caffeinated drinks is a minor matter as far as our health is concerned. But is it?  We cannot estimate its effect on mind and emotions, discrimination, and judgment.  And then there are the harmful effects of the stimulation on the heart and other vital organs.

Coffee and other caffeinated beverages are poor substitutes for water. The body needs alkaline fluids, but not stimulating drugs. Giving up the coffee and tea habit is relatively easy to do for most folks—once a commitment has been made. Since caffeine is a less toxic drug than alcohol or street drugs, the majority of coffee and tea drinkers can give up the habit without the sort of difficulties that alcoholics and drug addicts typically experience.

When giving up caffeine, eat and drink only what contributes in some way to good alkaline nutrition for the body. Any food or drink that does not contain alkalizing chlorophyll, oil, water, salts, vitamins, or minerals, should automatically be crossed off the list. While breaking the coffee, tea, cola, energy drink habit, be sure to drink plenty of fresh alkaline green juices and alkaline water at a pH of at least 9.5.

Just how widespread is coffee, tea, soda drinking? The average American drinks over twenty-six gallons of coffee, tea and cola drinks per year, but perhaps more germane to the discussion is caffeine itself.  Coffee has over three hundred chemicals; caffeine is only one of them.

23  Reasons to NEVER Drink Coffee Again!

1. Methyl parathion

This is the most toxic pesticide of all. It is  is highly toxic to humans, birds, fish, and mammals. It’s used to fight leaf miner infestations. Leaf miners are insects that eat at leaves of plants.  Despite how dangerous it is, it’s still (mis)used in some countries.

2. Endosulfan

This pesticide is used against coffee cherry borer, a common coffee consuming bug. It’s doesn’t dissolve easily and takes ages to break down in soil and is toxic to most animals. It affects the central nervous system, reproductive organs, kidneys, and liver, and is considered to be worse than the pest itself; it’s even been responsible for human death!

3. Chlorpyrifos

This is also used against common coffee pests and has been banned in the US for household use because it has caused human death and birth defects. Needless to say, it’s quite detrimental to delicate ecosystems.

4. Triadimefon

Copper-based fungicide used to against coffee rust. Only slightly toxic to birds, little is known about its effect on humans, but it is suspected that there is potential for reproductive problems with chronic exposure.  (Like people who drink coffee every day.) It has been found to induce hyperactivity in rats. The major concern is that long-term use of this and other copper-based fungicides is copper accumulation in soils, such as that found in coffee farms in Kenya and in Costa Rica.  Copper toxicity has been found in other crops grown in these soils, and copper impacts other biochemical and biological processes in soil which will poison people eating these crops not to mention the people who drink coffee.

5. Caffeine

One 8-ounce cup of coffee has 95 milligrams of caffeine and a 1-ounce single shot of espresso has 64 milligrams. Consuming too much caffeine can make you restless, anxious, irratable and then dead.  Caffeine is an acid or oxidant poison with a pH of 5.5 and an oxidative reduction pH of over +250 mV. When you drink a cup of coffee the body reacts to the poisons in the coffee and you feel it as adrenalin rush as the body starts releasing alkaline buffers to neutralize the poison or the caffeine acid.  It only takes 3 cups of coffee or acid to go into potential caffeine intoxication or 300mg which can cause a cardiac arrest.  One drop of pue caffeine or 2 grams of caffeine powder will kill you instantly.

6. pH

Coffee has a perfect pH for cancerous cells 5.5. Research at the Brigham Young University showed that you can keep cancer cells alive indefinitely in a cup of coffee.

7. Oxidative Reduction Potential or ORP

Coffee is NOT an antioxicant but an oxidant which acitivates the alkaline buffering system and depletes your body of alkalizing minerals such as sodium bicaronatea and potassium bicaronate.

8.  Stomach

Because coffee an acid beverage it causes the stomach to produce sodium bicarbonate which increases the hydrochloric acid in the stomach leading to acid reflux, GERD, ulcers and stomach cancer.

9.  Damages Cover Cells of the Stomach

Coffee is a hot beverage and any hot beverage will damage the cover cells of the stomach which cells produce sodium bicarbonate for maintaining the alkaline design of all the body fluids.

10.  Stimulate

Coffee is saturated with hydrogen ions or protons and thus steals energy from your body making you more tired after the stimulating effects wear-off.  This creates the addiction for more coffee in order to achieve an energy increase or buzz.  Continued use of coffee leads to enervation then irritation, then inflammation, then ulceration and finally degeneration or cancer.

11.  Cafestol

Coffee increases the level of cholesterol. Why? Because coffee contains an acid substance called cafestol which triggers the rise of cholesterol levels. The cafestol blocks a receptor in an intestinal pathway crucial for cholesterol regulation, and is the most potent food chemical to do this.   Increased amounts of this acid will increase cholesterol for the purpose of buffering and maintaining the alkaline design of the body.

12.  Intestinal Villi Damage

Coffee compromises the alkalinity of the small intestine at a pH of 8.4.  This causes the intestinal villi to lie down preventing the biological transformation of food or chyme into stem cells which takes place in the crypts of the intestinal villi.   This leads to lowered blood counts, improper blood cell formation and symptoms anemia, pernicious anemia, and hemolytic anemia.  Coffee also damages the intestinal villi leading to more serious conditions such as proper bone, muscle and organ regeneration.

13. Cancer

Coffee contains over 1000 chemicals of which only 22 have been studied leaving 978 left to study.  All of the chemicals studied to date have been found to be carcinogentic.  So next time to pick up that cup of coffee think of it as your cup of cancer.

14.  Sugar and Cream

Add the sugar and cream and you just created one toxic and addictive acidic/poisonous beverage.

15. Heart Disease –

There is controversial scientific evidence linking coffee consumption to heart diseases. Some studies even state that “consumption is associated with significantly increased risk of cardiovascular disease.” These same studies have shown a cholesterol-raising effect in some of the chemical compounds of coffee, such as determines, cafestol, kahweol and plasma homocysteine. This may be of-set by some of the antioxidants, but the overall agreement is that coffee may adversely effect the heart.

16. Blood Vessels

Coffee disturbs the functioning of blood vessels, both in turgidity and tone.

17. Cardiovascular System

Coffee affects our nervous system, heart rhythms and has been consistently linked to irregular heartbeats. It may also adversely affect blood pressure.

18. Osteoporosis

Coffee drinking should be heavily avoided by people at risk, or who have Osteoporosis. Studies show a link between drinking coffee and urinary calcium excretion.

19.  Heartburn

Many people report that coffee increases heartburn.

20.  Sleep Disturbance

Coffee, particularly in the evening or at night, can lead to sleep disturbance.

21. Dehydration 

Drinking coffee depletes water reserves in the body.

22. Addiction 

While the FDA recognizes caffeine as “safe,” it is still a drug, as it significantly alters the nervous system, leading to addiction

over time.

23. Extreme Withdrawal Symptoms

You may experience withdrawal symptoms when you try to give up coffee. This can lead to headaches, irritability, body aches, and other more extreme symptoms.

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Coffee beans are green and alkaline on the tree until they are fermented and spoiled rotten to a brownish black acidic,  heart dis-ease causing color.

For more information, check out these references:

Charles F. Wetherall, Kicking The Coffee Habit, Wetherall Publ. Co. MN.

Andrew Weil, MD & Winifred Rosen, Chocolate To Morphine, Houghton Mifflin Co., Boston, Mass.

Mervyn G. Hardinge, MD, A Philosophy of Health, Loma Linda University, CA.


1. Executive Fitness Newsletter (October 13, 1984).

2. Gastroenterology (November, 1948).

3. Providence Journal (October 1, 1990).

4. Carol Simontacchi, The Crazy Makers (New York: Tarcher, 2001).

Does Taking Aspirin Daily Protect You Against Cardiovascular Disease?

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A once-daily, low-dose aspirin did not significantly reduce the risk of the composite outcome of cardiovascular death, non-fatal stroke, and non-fatal MI among Japanese patients aged 60 years or older with atherosclerotic risk factors. http://ja.ma/1tAxh3k   = http://jama.jamanetwork.com/article.aspx?articleid=1936801&utm_campaign=social_111714&utm_medium=facebook&utm_source=jama_fb 

Reversing Atrial Fibrillation, CHF, Hypertension, Hypercholesterolemia, Diabetes, and even Alzheimer’s with Dr. Robert O. Young’s pH Miracle Self-Care to a Self-Cure for Heart Disease!

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The following beautiful story comes from a loving and caring daughter, Jeri, who shares her 82 yr old Mother’s reversal of Hypertension, Hypercholesterolemia, Congestive Heart Failure, Atrial Fibrillation, and even Alzheimer’s!  And, she no longer wears diapers!

“One Lucky Gal! My mother Beverly Harris (82) has had an amazing transformation.  I have been intensely cradling and nurturing mom since May 2013 as I have her following Dr. Robert O. Young’s pH Miracle alkaline protocol.   Two years later she is once again able to read books, do crossword puzzles, prepare some food dishes, participate in Silver Sneakers and often walk up to a mile a day.

Mom and Dad had lived with my family in an in-law arrangement from 2000-2010 when I decided to move them into an independent community living situation.  Dad age 80 passed within 3 months. Several times a week I drove 90 minutes to see Mom for 1-1/2 years, she was failing fast with uncontrolled diabetes, congestive heart failure, and severe dementia. While at a doctor’s visit in May 2013, her heart was in atrial fibrillation and they called an ambulance immediately. After drug therapy and a roller coaster of trial medications, Mom came home to live with me again, she was despondent and unable to care for herself.  Wearing diapers, she rocked in a chair and tapped her fingers often dazed in a fog, unable to spit her words out, unable to even watch TV, her eyes were empty… all the while I tried to regulate her 11 medications, which induced severe acidic side effects, from bleeding profusely, sleeping all the time and worsening mental issues.  I decided I would try to put her on Dr. Young’s pH Miracle healthy supplementation program and change her acidic diet.

I had to stop work completely, sell my office condo to stay afloat to relieve me of the burdening costs. The other day mom was teary-eyed with joy as we were setting up a kitchen area so she could make her own food.  Mom went off all her 11 medications except insulin, but she reduced the insulin from 60 units a day to 16.  Her blood pressure is perfect; she slimmed down and looks wonderful for her age.  She can even fold clothes!!!  Sounds silly but that takes a lot of skill and training.  Here is a picture of her now after following Dr. Young’s pH Miracle for (reversing) Heart Disease lifestyle and Diet!  She is bright, shining, happy and aware!

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Even though having to live on a shoestring budget, we manage to pamper her along with the pH MIracle diet changes and exercise.  She gets monthly massages, facials, manicure and pedicures, too. Touch is so important!

Although I know that I will never be able to leave her alone, this journey is a short one for both of us and the experience in reversing all her dis-eases with the pH Miracle protocol and her being virtually drug-free is a beautiful blessing!”    If you are suffering from any symptom of heart disease you NOW have the blessing to change your life and save your life with a self-care to a self-cure following Dr. Young’s pH Miracle for reversing Heart Disease!

To learn more about reversing the symptoms of heart disease read, share and like the following article by Dr. Robert O. Young located at: http://wp.me/p5ggLY-1kt

 NOW Is The Time To Change Your Life and Save Your Life!

The longer that a person has had heart disease and hardening of the arteries, the more challenging it is to reverse. In some cases, the damage will be too severe to completely eliminate, but stopping the progression and reversing it partially is possible in even the worst cases.

Hawthorn has been proven to help those with congestive heart failure, pulse irregularities, and angina. It is the most recommended supplement for heart health, in both alternative and mainstream medicine. Hawthorn is considered to be the swiss army knife of heart supplements. According to the University of Maryland Medical Center, hawthorn has been used since the 1st century as a remedy for heart and respiratory ailments. In Germany, it is regularly prescribed by mainstream doctors to those with heart failure or hypertension. Hawthorn also reduces the likelihood of blood clots, and has a strong anti-inflammatory/anti-acid effect. The research study, Alternative Therapies: Part II. Congestive Heart Failure and Hypercholesterolemia, aggregated the results of 50 clinical trials. It was performed by the Louisiana State University School of Medicine. It concluded that a “clear improvement in the subjects receiving hawthorn was observed”. The standard dosage in the studies was 500 mg. per 100 lbs. of body weight.

Perform a heavy acid and metal cleanse. Those with heart disease generally have a history of heavy acids and metal toxicity, and the difference that a heavy acid and metal cleanse makes can be dramatic. As noted earlier, always begin with a thorough liver cleanse. When heavy acids and metals are removed from the connective and fatty tissues, the body can properly create nitric oxide. Nitric oxide allows vessels to expand and contract efficiently, and it allows greater oxygen into cells.

Toxic elemental iron accumulation inside the blood is a problem specific to men. Therefore, adult males should never use supplements containing elemental iron. Men develop an excess of elemental iron in their bodies because of male acidic hormones like testosterone, and because the male body will not remove excess elemental  iron without proper alkalizing nutritional augmentation. The typical resultant iron excess in males is believed to be one of the main reasons why men die younger than women. The bodies of females of child-bearing age flush excess elemental iron through their menstrual process, which acts as a natural blood-letting. A woman’s remaining iron reserves are then used to produce replacement blood. Donating blood is the quickest way for a man to reduce his iron levels, and it may therefore save two lives. The study, A Historical Cohort Study of the Effect of Lowering Body Iron Through Blood Donation on Incident Cardiac Events, has shown that men who give blood have a 30% reduced risk of heart disease. An unusual but potentially effective option is to occasionally supplement with colloidal silver, for silver is able to neutralize iron in the body.

Dr. Linus Pauling discovered that resveratrol, found in grape skins, aids in the removal of iron. Resveratrol can also directly reverse plaque deposits. A study by the University of Wisconsin found that when people with coronary artery disease drank 20 fluid ounces of purple grape juice for just two weeks, their blood vessels became noticeably more elastic, and both platelet aggregation and LDL oxidation decreased. Due to its resveratrol content, wine consumption has been identified as one of the primary reasons for France’s low rate of heart disease.

In perhaps the greatest irony herein, unprocessed whole mineral salt is needed to reverse heart disease. Natural salts are needed because of the trace minerals that they contain, which are not attainable by any other means. I recommend that readers replace all table salt with mineral-rich unprocessed sea salt. Do not buy the fake sea salts from regular grocery stores, because they are just as refined as the table salts, and are thereby counterproductive. True sea salt should never be a bright white color. Always avoid these salts. Use unrefined, non-adulterated, mineral-rich sea salt that can be purchased online or from a health food store. Liberal amounts of natural sea salt up to 14 grams a day based on a body weight of 70k or 154 lbs. should be added to alkaline meals as a healthy and flavor-enhancing supplement.

The human body can create new arteries when its old arteries become clogged, but it needs new healthy blood and copper to accomplish this. Oral supplementation with copper is dangerous, because it quickly becomes toxic with an overdose, and it takes only a tiny amount to overdose. Excessive amounts are known to cause severe organ damage; especially to the kidneys and liver. Therefore, I discourage people from orally consuming any man-made copper solutions, such as colloidal copper or copper hydroxide.  So get your copper from a good unprocessed mineral salt.

For the sake of safety, copper should only be supplemented by way of the following nutritional methods. Natural sea salts contain safe amounts of natural copper and important trace minerals. Another option is non-glycerin chlorophyll extract, which will have the additional benefits of increasing the number of red blood cells, increasing blood oxygenation, protecting against radiation, and neutralizing benzene compounds. It contains much more copper than sea salt. 32901-1476284_781639965184671_646394062_nChlorophyll is the dark green pigment of plants and vegetables, but it can be purchased in a concentrated extract for supplementation. It is a recommended supplement for all heart disease, cancer and diabetic patients. In liberal amounts, chlorophyll safely gets large amounts of copper into the human body.  You cannot drink too much.

Avoid all processed foods. They contain excessive amounts of sodium from dangerous sources, such as monosodium glutamate. MSG is especially dangerous to the heart, because it neutralizes L=taurine, which is necessary for pulse regulation. Monosodium glutamate intake in combination with deficiencies of both magnesium and L-taurine can make MSG deadly. This combination is the cause of many unexplained heart attacks in otherwise healthy people; including young athletes in their prime years, whose only mistake was that last meal at K.F.C. (Kentucky Fried Chicken).

Dr. Matthias Rath, M.D., is a leading researcher of heart disease, who worked with the late Linus Pauling. He has continued much of Dr. Pauling’s work. Dr. Rath explained that the first step in reversing coronary heart disease is nutrition, and people with coronary heart disease are deficient in trace minerals. In particular, vitamin C supplementation is essential. In his book, Why Animals Don’t Get Heart Disease, But People Do, Rath explained that the only other species to get heart disease is the guinea pig, and it is also the only other species that does not internally produce its own vitamin C. Hardening of the arteries is a pre-scurvy condition, and it will occur if not enough vitamin C is available. The two doctors proved that vitamin C also bonds with iron, allowing excess iron to be removed, it suppresses cholesterol oxidation, and it shields the body against metabolic and dietary acids.

Niacin (vitamin B-3) and pantothenate (vitamin B-5) work in conjunction with vitamin C. Niacin protects the arteries, and is metabolized in the body from L-tryptophan. L-tryptophan naturally occurs in all green vegetables, but can also be obtained through supplementation. It is recommended that people avoid time-released niacin, because too many people have had bad experiences, including blackouts and hallucinations from the chemical impurities. Both niacin and L-tryptophan are effective for treating depression, and this again reinforces the connection between emotional and heart health. Vitamin B-5 (sold as “pantothenic acid”) works with vitamin C to protect against arterial damage, thus reducing a body’s overall need to produce cholesterol. Vitamin B-5 can be found naturally in vegetables, grasses, sprouts, and legumes.

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In the 1950’s, Dr. Johanna Budwig was reversing heart disease with what is now known as the Budwig Protocol. However, this alternative protocol is much more famous for reversing cancers, and there is a strong connection between the two inflammatory disease states.

At the core of the anti-inflammatory/anti-acid pH Miracle protocol is a combination of flax and hemp seed oil and a food item that is rich in chelating antioxidants. I have discovered that maximum absorption of omega-3 into cells is required to buffer intracellular acids.  Whenever properly combined, this solution is best able to penetrate into the deep tissues and through cellular membranes.  bigstock-Spoon-Of-Olive-Oil-974537Flax and hemp seed oil supplies the building blocks for cellular repair, helps dissolve excess acid-bound cholesterol inside the arteries, balances the high omega-6 concentrations that are found in trans-fats and processed foods, increases cellular energy, increases the efficiency of respiration, and it is an overall tonic to general cellular functions. The heart benefits of flax and hemp seed oil (omega-3 supplementation) are now well-recognized, even by orthodox medicine. Whenever flax and hemp seed oil is heated or exposed to air, it rapidly becomes rancid, and thereby harmful. People should avoid products which contain heated or exposed flax and hemp seed. This often includes cereals, breads, and flour. The companies that sell these products deceptively promote them as being healthier, but ironically, most of the omega oils have been destroyed by the processing, and any remaining oils have become inflammatory and carcinogenic.

The medical establishment has sabotaged alternative medicine’s otherwise beneficial pH Miracle Protocol into being something less than beneficial. Instead of following the original protocol that uses flax and hemp seed oil as the source for omega-3 supplementation, the medical establishment prescribes fish oil to heart disease patients, which has been industrially processed by the pharmaceutical industry with high-heat and poisonous chemical solvents to extract the oil from fish livers. The livers of fish are where the PCB’s, heavy metals, and pesticides are stored; because such substances are too toxic for their digestive and immune systems to excrete normally. A liver is a body’s toxic waste dump, in other words. The high heat and extreme processing that is used to chemically extract the omega oil from fish livers means that the derived oil is always rancid by the time that it reaches the patients; and therefore, this doctor-prescribed ‘medicine’ actually causes heart disease and cancers. Most of the fish that are used to pharmaceutically produce omega oil have high amounts of the inflammatory omega-6 and very low amounts of omega-3, because the pharmaceutical industry’s fish are farm-raised in unhealthy conditions in Chinese fish farms. Adding an even greater insult to the injury, American patients are charged hundreds of dollars for this impure and carcinogenic version of omega-3 by the medical industry, while the safe and more effective equivalent (flax and hemp seed oil) can be found cheaply at any health or natural food store. Health or natural food stores also sell fish oil, but it too should be avoided in lieu of the superior flax and hemp option.

Co-enzyme Q10 is absolutely essential for recovery from heart disease and heart failure. It can be found in some foods (particularly avocado) and it is produced by the body during exercise. While studies have shown great benefits through intravenous supplementation, oral supplements are unfortunately not absorbed well. Thus, it must be obtained through dietary sources and exercise. CoQ10 is essential for the emulsification of fats. It also lowers the blood pressure and provides an increased chance of survival following cardiac arrest. Research has also found that those with gingivitis have a deficiency of CoQ10. Vegetarians may attempt supplementation, even though absorption will be very low. Nevertheless, even a small amount will be helpful.

Japan currently has the lowest rate of heart disease in the world, so it is logical to examine their dietary and medicinal practices, in order to adopt them into our own society. The Japanese eat lots of fish, and thereby get large amounts of vitamin D, omega-3, and safe iodine. In addition to that, whenever heart disease is diagnosed, L-taurine is a common recommendation by Japanese doctors. Taurine is a natural nutrient, which makes it unpatentable (unprofitable), so it is “not approved” for medical use in the United States. It can stabilize the heart rate, neutralize MSG, and it helps the body to better regulate potassium, calcium, and sodium in the body. It is known for its ability to protect the kidneys from damage.

L-Taurine supplementation has also been shown to reverse some of the damage caused by smoking, even amongst smokers who continued smoking during the clinical trials. A 2003 study by the Royal College of Surgeons found that damage caused by chronic smoking can be reversed in as little as five days with a dosage of 1.5 grams of L-taurine. The inner lining of the blood vessels and the diameter of blood vessels returned to normal in the study.

It is wise to take anti-inflammatories or anti-acids when attempting to reverse heart disease. As already stated, inflammation is the first step in arterial plaque. Effective natural anti-inflammatories or anti-acids include devil’s claw, vitamin C, MSM, cherry concentrate, and resveratrol. Unknown-1Curcumin, a component of the turmeric spice, is also an anti-inflammatory and it has the added benefit of reversing some arterial plaque. Never underestimate the benefits of culinary spices, which are sometimes known as herbal medicine.

I have already written an entire article about the benefits of cayenne pepper, and it is absolutely vital for people with heart problems. It aids circulation, stabilizes blood pressure, and it can actually stop a heart attack in its tracks. Seriously. Remember this, because cayenne pepper can save a life. This is especially true whenever it is mixed with L-taurine. The suffering patient may need to hold them in his mouth for fastest results. During a heart attack, nobody ever complains that something tastes too spicy. Cayenne pepper is even good for painless weight loss. It can be conveniently supplemented in a pill form that is available from health food stores. We recommend that everyone with heart problems supplement with cayenne, or put it into a tea daily. Dr. David Christopher has been labeled “Dr. Cayenne” for his endless promotion of this spice.

Another reason for solidification of acids inside the arterial walls is an excess of blood-borne calcium, which is typical in Western nations. Vitamin D and magnesium chloride are critically needed for a body to make proper use of its calcium, so that it does not simply get dumped onto the artery inner walls. A large portion of people are not able to properly assimilate vitamin D supplements, so it is best obtained through sunlight.

It is important that you reduce or eliminate nicotine in cases of severe heart problems. additives-1Nicotine, along with alcohol, caffeine, and a host of pharmaceuticals, interferes with the absorption of key nutrients, which are vital for recovery. If you must smoke, ensure that you are making your own cigarettes with organic tobaccos and a rolling machine, or else you will be inhaling a host of chemicals including (but not limited to) carpet glue. Modern cigarettes have intentionally been made much more harmful than they need to be by the chemical industry, and they are fertilized with radioactive fertilizers. Indian tobacco can help eliminate a large number of addictions, including nicotine, and it can also help to clean the lungs.

Almonds and hazel nuts lower the heart disease risk because they contain alpha-linolenic acid (LNA). It is an omega-3 fatty acid. Countries with the most LNA in their diets also have the lowest risk of heart disease. Walnuts and cashews are the most nutritious nuts available.  Peanuts contain the least amount of nutrients, are likely to have pesticide residues and contain molds and mycotoxins.

Ginkgo biloba improves circulation, and it reduces cholesterol concentrations in the arterial walls. It also enables the arteries and veins to more fully relax between pumping cycles. The National Institutes of Health reported that ginkgo is very useful for alleviating leg pain that is attributed to blocked arteries.

bf3aa-photo-101Exercise is essential for preventing heart disease or recovering from it. Not only does exercise allow the body to create its own CoQ10, but it also creates nitric oxide and L-carnitine. As already stated, nitric oxide from an amino acid called L-arginine aids the vessels to expand and contract more efficiently, and L-carnitine can improve the heart’s function. We recommend finding an exercise that you can enjoy, and if possible, do it with others. Try to remember how enjoyable exercise was when you were a child, and attempt to make it that way again. Let us not forget that positive emotions are vital to heart health. Jogging on a treadmill is a chore, and it can even be a humiliating, demeaning experience.  I personally enjoy rebounding, walking, jogging and static contraction weight-lifting.

5dbaa-grapefruitGrapefruit and especially grapefruit pectin is available in supplement form. It reverses arterial plaque, reduces cholesterol, and widens the arteries. While it is not a standard supplement, we would nevertheless recommend it to anyone with chronic heart problems.

Due to the importance of nutrition, I recommend that people follow my pH Miracle green juice feast for about two weeks when they are strong enough. Juicing is the process of mixing alkaline fruit and vegetables together in a blender or juicer, and then using the extracted juices as a food source. A juice fast is a means of flooding a body with alkalizing nutrients that it is normally lacking in due to our modern diets. The first few days may be difficult, but the long-term benefits can be dramatic.

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I recognize that many people perceive they require a source of pure protein each day (such as organic hemp seed or hemp protein which is one of the best sources) to be able to continue with their normal lives uninterrupted.  The need for protein in the diet is less than 5 grams a day.  The body is a great recycler and obtains all of its protein needs from within.  Also, all body cells are made from red blood cells and red blood cells are made from stem cells developed in the crypts of the small intestines.  2e491-intestinalvillus

Stem cells are made from the liquid alkaline food called chyme.  The quality of these stem cells is determined by what you eat, what you drink and what you think. The fasting or feasting diet incorporating juiced and purred green vegetables, fruit, alkaline water, mineral salts and polyunsaturated oils is the key to producing healthy stem cells to red blood cells in mass.  I call this approach to the restoration of energy, vitality, health and fitness to the body COWS.  10888626_10152974722002008_541873310624558933_nCOWS is an acronym that stands for the four food groups required to increase stem cell production and to improve the quality of these stem cells produced.  The ‘C’ stands for chlorophyll which can be found in all organic green vegetables and fruit which becomes the foundational light or life force of intelligence in the stem cells.  The ‘O’ stands for oil and more specific polyunsaturated oils for building the lipid membrane of stem cells that holds the liquid light from the chlorophyll. The ‘W’ stands for water and alkaline water which provides the environment for stem cell for movement, intelligence and life.  And finally ‘S’ which stands for salt and specifically sodium which is the basis for conductivity and the transport of electrical energy without and within the stem cell.   The production of healthy stem cells requires a healthy alkaline pH at 8.4 in the crypts of the small intestines.  As you increase the quality and quantity of stem cells in the gut you will increase the quality and quantity of your red blood cells throughout circulation.  As you increase healthy red blood cells traveling throughout the vascular system you begin to replace unhealthy body cells with healthy biologically transformed red blood cells to brain cells, nerve cells, muscle cells, bone cells, liver cells, artery cells and new healthy heart cells.  You replace and restore an unhealthy heart with a new healthy and youthful heart.  This is how YOU reverse ANY health challenge including the many symptoms of heart disease.  Understand?

Without Salt There is NO Alkalinity and There is NO Life!

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“Salt is as important as oxygen to the body.  It is salt that transports energy from cell to cell and protects the body from dietary and metabolic acid.  In all chronic dis-ease, including heart dis-ease there is always a deficiency of salt.  Salt is the glue that holds the spirit body inside the physical body.  Salt is the Sol of life.  Salt we misjudged you!  Salt is GOOD for you! Eating more salt could even lower the chances of heart disease.”Dr. Robert O. Young

For years, doctors have been telling us that too much salt is bad for us. Until now!

A recent study claims that cutting down on salt can actually increase the risk of dying from a heart attack or a stroke.

Its findings indicate that those who eat the least sodium – about one teaspoon a day – don’t show any health advantage over those who eat the most.

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In the eight-year study, people with the lowest salt intake had the highest rate of death from heart disease!

In fact, those with less salty diets actually had slightly higher death rates from heart disease.

The study, which followed 3,681 healthy European men and women aged 60 or younger, for about eight years, also found that above-average salt intake did not appear to increase the danger of developing high blood pressure.

The report, in the latest issue of the Journal of the American Medical Association, was released just three months after the U.S. government launched a public health campaign urging restaurants and food manufacturers to cut down on their use of salt.

Sodium was measured in the urine of those taking part, at the beginning and end of the study.

A little more than six per cent of the participants suffered a heart attack, a stroke or some other cardiovascular emergency during the eight years. About a third of these were fatal.

Those who consumed the least salt had a 56 per cent higher risk of death from a heart attack or stroke compared with those who consumed the most.

This was even after obesity, cholesterol levels, smoking, diabetes and other risk factors were taken into account.

There were 50 deaths in the third of participants with the lowest salt consumption, 24 in the third with medium intake and just ten deaths in those with the highest salt levels.

Lead researcher Jan Staessen, head of the hypertension laboratory at the University of Leuven, in Belgium, said: ‘Our findings do not support a generalized reduction of salt intake in the population.

The scientists did not have a firm explanation for their results, but

they reportedly speculated that low levels of salt in the body could cause more stress in the nervous system, decrease sensitivity to insulin and affect hormones that control blood pressure and sodium absorption.

Bottom-line if you want to be healthy and prevent or reverse heart disease then you must increase your salt intake as you are decreasing your acid intake.  As we said in the 60’s you have to stop “dropping acid!” if you want to be healthy.  Back then and even now it was LSD!  A toxic acid from mold called Ergot!

If You want to get high?  Get high on a life of feeling incredible!

The Prognosis

Throughout this article, I resisted the temptation to rant about the sorry state of cardiac medicine, if it can indeed be reasonably called medicine. I began by citing that the medical industry’s own statistics prove that standard medicine for cardiac issues is the single biggest failure of orthodox medicine. Now I end this report with an honest commentary about our well-earned biases against the status quo.

Sometimes emergency medical personnel save lives with emergency medicine, but this is the best that orthodox medicine can do. They fail at everything else, and this is partly because effective therapies are illegal.  All approved therapies must be drug-based, which means that all of the successful nutritional therapies (to what is a very nutritional problem) are forbidden to all doctors who wish to remain licensed to practice medicine. The consequence is that it is actually illegal for any licensed doctor to cure or to successfully mitigate heart disease, cancer or diabetes since successful and natural therapies are not approved by medical regulators. If you receive the standard treatments (chemical symptom suppression) for heart conditions, or the establishment’s supposedly preventative treatments, which usually do the opposite of what they are presented to do, then you will die of either heart disease, or the treatments themselves. There is a 70% risk that you will die of complications caused by the medications, such as the development of a cancer, or liver failure, or diabetes. They call this  “progress”.

Heart disease is not truly normal or natural, and it is NOT genetic. The only thing that is genetic about our cardiac catastrophe is that lifestyles, family doctors, and eating habits are inherited. At the core of the problems are ignorance and irresponsibility that span generations. In this sense, there are genetic components to heart disease, but the problem was never God’s engineering.

Heart disease takes lots of work to reverse, but it is more-or-less self-curable in most cases, regardless of what a doctor tells you.

Remember that it took years to get yourself into this state of ill-health, and it may take a long time to get yourself out of it. Do not disregard the importance of emotional and spiritual health, because these things have a major impact upon the heart, and a full recovery may not be possible otherwise. Whenever I use the term “spiritual health”, I are referring to real spirituality, such as forming a personal relationship with our Creator.

Acidic Symptoms of a Heart Attack

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  • Discomfort, pressure, heaviness, or pain in the left arm or chest
  • Discomfort radiating to the back, jaw, throat, or arm
  • Fullness, indigestion, or choking feeling (may feel like heartburn)
  • Sweating
  • Nausea
  • Dizziness
  • Extreme weakness
  • Anxiety
  • Shortness of breath
  • Rapid or irregular heartbeat

Emergency Alkaline Protocol for Heart Attacks

In the event of a heart attack, do not forgo calling 911 for an ambulance. The establishment’s forté is emergency medicine, and the risk is too great to delay calling for help. While waiting for the ambulance to arrive, small amounts of these alkalizing supplements should be placed into the mouth of the victim, and allowed to absorb through the walls of his/her cheeks:

  • L-taurine
  • N-acteyl-cysteine
  • Cayenne
  • Magnesium chloride

Americans need not be concerned with legalities when trying to employ these methods. Every State in America has a Good Samaritan law, which provides special legal protection to those who make a good faith effort to save a life!

My hope after you read this article is that you will do your own research and find out for yourself as I have that there is a Self-Care natural health and wellness protocol for a Self-Cure for Heart Disease!

To learn more about how to reverse heart disease, cancer, diabetes, weight loss and weight gain or if you are dealing with fatigue and tiredness read the following books by Dr. Robert O. Young – http://www.phoreveryoung.com, http://www.amazon.com, http://www.phmiracle.com, www,phoreveryoung.wordpress.com, http://www.phmiracle.com

1) The pH Miracle for Heart Disease

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2) The pH Miracle for Cancer

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3) Reverse Cancer NOW!

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4) The pH Miracle for Weight Loss

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5) The pH Miracle for Diabetes

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6) The pH Miracle Book 1

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7) The pH Miracle Revised and Updated Book 2

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8) Sick and Tired

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9) Back to the House of Health I Recipe Books

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10) Back to the House of Health II Recipe Books

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Part 3 – The Key to Prevention for a Self-Care to a Self-Cure for ALL Sickness and Disease!

Health and Healing Crusade welcomed Robert O. Young M.Sc., D.Sc., Ph.D. ND who gave three incredible talks on promoting an alkaline Lifestyle and diet. He claims that health depends primarily on proper balance between an alkaline and acid environment in the human body, and that an acid environment causes cancer, obesity, osteoporosis, yeast overgrowth, flu, skin disorders, and other diseases.

Please listen carefully as he teaches some important truths about our wonderful universe, our bodies, and the importance of having an alkaline environment to keep us free from ALL sickness and disease caused by and acidic diet and our neglect to care for our body!

Part 2 – The Prevention and Self- Care to a Self-Cure for ALL Sickness and Disease!

Health and Healing Crusade welcomed Robert O. Young M.Sc., D.Sc., Ph.D. ND who gave three incredible talks on promoting an alkaline Lifestyle and diet. He claims that health depends primarily on proper balance between an alkaline and acid environment in the human body, and that an acid environment causes cancer, obesity, osteoporosis, yeast overgrowth, flu, skin disorders, and other diseases.

Please listen carefully as he teaches some important truths about our wonderful universe, our bodies, and the importance of having an alkaline environment to keep us free from ALL sickness and disease caused by and acidic diet and our neglect to care for our body!

The Prevention and Self-Care to a Self-Cure for ALL Sickness and Disease

Health and Healing Crusade welcomed Robert O. Young M.Sc., D.Sc., Ph.D. ND who gave three incredible talks on promoting an alkaline Lifestyle and diet. He claims that health depends primarily on proper balance between an alkaline and acid environment in the human body, and that an acid environment causes cancer, obesity, osteoporosis, yeast overgrowth, flu, skin disorders, and other diseases.

Please listen carefully as he teaches some important truths about our wonderful universe, our bodies, and the importance of having an alkaline environment to keep us free from ALL sickness and disease caused by and acidic diet and our neglect to care for our body!

The Game of Life and How it Ends!

The Game of Life and Death!

Odds-of-Dying

Let’s see if the odds are in your favor.

Lifetime odds of death by cause:

Most Americans won’t die of any of the below causes.

But about 56 million will die of…
Heart disease and cancer: 1 in 7
Chronic lower respiratory: 1 in 29

And motor vehicle incidents (1 in 112) will claim about as many as intentional self harm (1 in 103)

While around 4.7 million present day Americans will due of Unintentional poisoning by and exposure to noxious substances (1 in 119)
And falls (1 in 152)

Assault by firearm will claim about a 890,000 (1 in 356).
Much more than accidental firearm discharges, which clocks in at 48,000 (1 in 6,509).

There’s a chance you’ll be on the street.
Motor vehicle incidents: 1 in 112
Car occupant: 1 in 492
Pedestrian: 1 in 723
Motorcycle rider: 1 in 922

Though your safest bets are bikes, planes and spacecraft:
Pedalcyclist: 1 in 4,974
Air and space travel incidents: 1 in 8,321

Around 609,000 people can expect to die in one of these unsavory ways:
Unintentional drowning and submersion: 1 in 1,043
Exposure to fire, flames, or smoke: 1 in 1,418
Choking from inhalation and ingestion of food: 1 in 3,649

At least mother nature won’t kill many:
Exposure to excessive natural heat: 1 in 8,321
Exposure to electric current, radiation, temperature and pressure: 1 in 12,174
Contact with sharp objects: 1 in 37,566
Contact with heat and hot substances: 1 in 62,608
Contact with hornets, wasps and bees: 1 in 75,852
Cataclysmic storm: 1 in 83,922

And your much likelier to be killed by a dog than a venomous snake:
Bitten or struck by dog: 1 in 103,798
Venomous snake: 1 in 1,224,099[4]

Similar numbers of people will be legally executed as well be killed by lightning:
Legal execution: 1 in 96,203
Lightning: 1 in 136,011

Or you could die in a terrorist attack (1 in 256,410)[3]
A number comparable to your chances of being crushed to death by your own television(1 in 253,205)

Though the chances of just furniture (1 in 1,346,153) or appliances crushing you (1 in 2,025,641) are much more slim.

But it really all depends on your age

Top ten causes of death by age:

<1: Congenital anomalies, short gestation, SIDS, Maternal pregnancy complication, placenta cord membrane, unintentional injury, respiratory distress, bacterial sepsis, neonatal hemorrhage, circulatory system disease
1-4: Unintentional injury, congenital anomalies, malignant noplasms, homicide, heart disease, influenza/pneumonia, septicema, perinatal period, chronic low respiratory disease, benign neoplasms
5-9: Unintentional injury, malignant neoplasms, congenital anomalies, homicide, heart disease, influenza/pneumonia, septicemia, benign neoplasms, chronic low respiratory disease, cerebro-vascular
10-14: Unintentional injury, malignant neoplasms, suicide, congenital anomalies, homicide, heart disease, chronic low respiratory disease, influenza/pneumonia, benign neoplasms, cerebro-vascular
15-24: Unintentional injury, homicide, suicide, malignant neoplasms, heart disease, congenital anomalies, influenza/pneumonia, cerebro-vascular, chronic low respiratory disease, HIV
25-34: Unintentional injury, suicide, homicide, malignant neoplasms, heart disease, HIV, diabetes melitus, cerebro-vascular, congenital anomalies, influenza/pneumonia
35-44: Unintentional injury, malignant neoplasms, heart disease, suicide, HIV, Homicide, liver disease, cerebro-vascular, diabetes melitits, influenza/pneumonia
45-54: Malignant neoplasms, heart disease, unintentional injury, liver disease, suicide, cerebro-vasclar, diabetes melititus, HIV, chronic low respiratory, viral hepatitis
55-64: Malignant neoplasms, heart disease, chronic low respiratory disease, diabetes melititus, cerebro-vascular, unintentional injury, liver disease, suicide, nephritis, septicemia
65+: Heart disease, malignant neoplasms, cerebro-vascular, chronic low respiratory disease, alzheimer’s disease, influenza/pneumonia, diabetes melititus, nephritis, unintentional injury, septicemia.

Total deaths by age bracket:

<1: 19,229
1-4: 3,645
5-9: 2,358
10-14: 3,128
15-24: 28,677
25-34: 32,740
35-44: 69,448
45-54: 139,380
55-64: 220,404
65+: 1,471,011

Total: 1,990,020 per year
(From 2003)
That’s 1 in 145 whose time is up, every year.

Such is the game of life!

Odds-of-Dying

Citations: