Stanford School of Medicine Validates Cellular Transformation!

Stanford University School of Medicine validates Dr. Robert O. Young’s cellular transformation research.

Pleomorphism is a scientific doctrine originally suggested by French medical doctor and researcher Dr. Antione BeChamp in the late 1800’s. He suggested that plant, animal and human cells will biologically transform into different cells depending on environmental factors. Such as a red blood cell would morph or transform into white blood cell or a white blood cell could morph or transform into a body cell such as a connective tissue cell. Dr. BeChamp also suggested that a body cell would transform into a bacterial cell or a yeast cell when there was a declining alkaline pH of the environment. In other words, the germ is born in us and from us out of a transforming blood or body cell.

The foundation of my theory is that the red blood cell is the foundational stem cell that can become all other body cells, including white blood cells, brain cells, bones cells, liver cells or muscles cells, etc. All cell transformations are pH or environment sensitive. For example, when the pH begins to decline from 7.365 to 7.2, the red blood cell will morph into bacteria, then yeast and finally mold. In other words healthy blood and body cells will transform into bacteria and than mold in a declining pH just like the food in the refrigerator as it ages or spoils from a compromised alkaline environment.

The cell transformations of healthy cells to cancerous cells and then back to healthy cells is also triggered by the acidity or alkalinity or the environment in which these cells are found. I have explained this natural phenomenon in a simple metaphor – when the fish is sick what would you do treat the fish or change the water? The correct answer is change the water! Why? Because the fish is only as healthy as the water in which it resides and swims. This is also true for plant, animal and human blood and body cells.
I have suggested for over 3 decades that the human body is alkaline by design although acidic by function. When acidic metabolic waste products are NOT properly eliminated through the four channels of elimination than these toxins or acids build-up in the connective and fatty tissues setting the stage for cell transformations that lead to enervation, irritation, inflammation, induration, ulceration and finally degeneration.
The scientists at Stanford University have just recently validated my research and confirmed the pleomorphic research of Dr. Antione BeChamp. Dr. BeChamp was right and Pasteur was wrong. The germ is NOTHING and the TERRAIN is everything!

I also documented these biological cell transformations over 20 years ago in 1994 before the investigators from Stanford University. It feels good to know you are not alone and other scientists are finally viewing cell transformation which was first discovered by Dr. Antione BeChamp over 100 years ago and rediscovered by Dr. Robert O. Young over 20 years ago. It’s about time!

To learn more about the science of Dr. Robert O. Young and cell transformations read Sick and Tired, Reclaim Your Inner Terrain – www.phmiracle.com, www.phoreveryoung.com,www.phoreveryoung.wordpress.com

Watch the following video of a rod bacterial form transform into a red blood cell as recorded by Dr. Robert O. Young in 1994.

Watch the following video of a rod bacteria transform into a red blood cell as recorded by Dr. Robert O. Young in 1994.

To learn more about the science of Dr. Robert O. Young and cell transformations read Sick and Tired, Reclaim Your Inner Terrain – www.phmiracle.com, www.phoreveryoung.com,www.phoreveryoung.wordpress.com

Click here to read the article on cell transformation or pleomorphism: https://m.facebook.com/story.php…

The Cure for Pancreatic Cancer – Pancreatic Cancer Cells Can Be Biologically Transformed into Normal Healthy Pancreatic Cells Validating the Research of Dr. Robert O. Young

Pancreatic Cancer Breakthrough: Scientists Turn Cancer Cells into Normal Cells

Scientists find a novel avenue for therapeutic intervention of the “silent cancer”

A new research study has shown that pancreatic cancer cells can be coaxed  to revert back toward normal cells by changing the environment by introducing a protein called E47. E47 binds to specific DNA sequences and controls genes involved in growth and differentiation. The research provides hope for a new treatment approach for the more than 40,000 people who die from the disease each year in the United States.

“For the first time, we have shown that overexpression of a single gene can reduce the tumor-promoting potential of pancreatic adenocarcinoma cells and reprogram them toward their original cell type. Thus, pancreatic cancer cells retain a genetic memory which we hope to exploit,” said Pamela Itkin-Ansari, Ph.D., adjunct professor in the Development, Aging, and Regeneration Program at Sanford-Burnham and lead author of the study published today in the journal Pancreas.

E47 turns the clock back
The study, a collaborative effort between Sanford-Burnham, UC San Diego, where Itkin-Ansari holds a joint appointment, and Purdue University, generated human pancreatic ductal adenocarcinoma cell lines to make higher than normal levels of E47. The increased amount of E47 caused cells to stall in the G0/G1 growth phase, and differentiate back toward an acinar cell phenotype.

In vivo studies showed that when the reprogrammed cancer cells were introduced into mice, their ability to form tumors was greatly diminished compared to untreated adenocarcinoma cells.

“Presently, pancreatic adenocarcinoma is treated with cytotoxic agents, yet the average survival for patients post-diagnosis is merely six months, and the improvements in therapies are measured in days,” said Andrew M. Lowy, M.D., professor of surgery at the UC San Diego Moores Cancer Center and co-chair of the National Cancer Institute’s Pancreatic Cancer Task Force. “The finding that we can differentiate these cancer cells back to a non-threatening phenotype is encouraging. Indeed, there is a precedent for cell differentiation therapy in that the approach has been used to treat acute promyelocytic leukemia (APL) and some neuroblastomas successfully.”

“Our next step is to test primary patient-derived tumor tissue to determine whether E47 can produce similar results, potentially providing a novel therapeutic approach to combat this highly lethal disease,” said Itkin-Ansari. “Additionally, we are screening for molecules—potential drugs—that can induce overexpression of E47.”

Pancreatic adenocarcinoma
Pancreatic adenocarcinoma is the most common form of pancreatic cancer. It’s primarily caused by a mutation in the oncogene called Kras that causes the digestive enzyme-secreting cells (acinar cells) to differentiate into a destabilized duct-like cell type, which is cancerous. The disease is often called a “silent” cancer because it rarely shows early symptoms—it tends to be diagnosed at advanced stages when it causes weight loss, abdominal pain, and jaundice.

The study was funded by support from the Hartwell Foundation, The Hirshberg Foundation, by the NCI under award number 5P30CA030199, by the NIH under award numbers DK55489 and CA124586, by the National Research Foundation of Korea award number 2011-0013127, and Yonsei University College of Medicine for 2012 (6-2012-0078)

ABOUT SANFORD-BURNHAM MEDICAL RESEARCH INSTITUTE
Sanford-Burnham Medical Research Institute is dedicated to discovering the fundamental molecular causes of disease and devising the innovative therapies of tomorrow. Sanford-Burnham takes a collaborative approach to medical research with major programs in cancer, neurodegeneration and stem cells, diabetes, and infectious, inflammatory, and childhood diseases. The Institute is recognized for its National Cancer Institute-designated Cancer Center, its NIH-designated Neuroscience Center Cores, and expertise in drug discovery technologies. Sanford-Burnham is a nonprofit, independent institute that employs more than 1,000 scientists and staff in San Diego (La Jolla), Calif., and Orlando (Lake Nona), Fla. For more information, visit us at sanfordburnham.org.