Naturopathic Practitioner – The pH Miracle Ti Sana Detox Medical Spa
Could Sodium or Potassium Bicarbonate save your life? Low levels of bicarbonate ‘are linked to a 24 % higher risk of early death!
Jan 22, 2016
Compound in baking soda – bicarbonate – reduces early death, study found
Baking soda – or sodium bicarbonate – helps regulate pH levels
People with low bicarbonate levels have 24% higher risk of early death
People should ingest fruit and green vegetables to get more bicarbonate!
Many of us think of baking soda as little more than a vital part of the recipe to making a cake or baking cookies.
But two new studies have found that the ingredients of sodium and/or potassium bicarbonate plays a far more important role in human health: It can help save lives.
Older people with low levels of sodium and/or potassiumbicarbonate – which is found in baking soda – have a 24 per cent higher risk of dying an early death!
The findings suggest increasing bicarbonate levels can prolong a person’s life.
Study author Dr Kalani Raphael, of the University of Utah, said: ‘What we found was that generally healthy older people with low levels of bicarbonate had a higher risk of death.’
Sodium bicarbonate, a main component of baking powder, reduces the risk of premature death, scientists revealed. Older people with low bicarbonate levels are 24 per cent more likely to die young, a study found
The kidneys and lungs work together by varying the levels of sodium and/or potassium bicarbonate – a base or alkaline compound – and carbon dioxide – an acid – in the blood, interstitial fluids and intracellular fluids.
Sodium and potassium bicarbonate helps keep the body’s pH in a healthy range (7.365), which allows the body cells that make up our organs to work properly.
Critically ill patients with severe acid-base abnormalities have very low levels of sodium and/or potassium bicarbonate and are very unlikely of surviving their illness, according to the study.
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Using Sodium and Potassium Bicarbonate in the Prevention and Treatment of ALL Sickness and Disease – Dr. Robert O. Young – www.phoreveryoung.com
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Yet, it has been unclear whether more subtle changes to the body’s acid-base status affect the longevity of relatively healthy older people.
A team of scientists investigated how measurements of pH, carbon dioxide and bicarbonate are associated with long-term survival in healthy older people.
They analyzed data from 2,287 participants in the health, aging and body composition study.
That data focused on well-functioning black and white adults between the ages of 70 and 79.
Data started being collected in 1997 – with collection efforts extending through February 2014.
Each of the participants were followed for an average of 10.3 years.
Scientists recommend people with low levels of sodium and/or potassium bicarbonate should increase their intake of foods that produce it in the body, including fruit and green vegetables.
Because of the study’s results, blood bicarbonate concentrations – which are already commonly measured – may allow clinicians to better identify people with a higher risk of premature death.
Those with low sodium and potassium bicarbonate levels may benefit from increasing their intake of foods that produce bicarbonate in the body – including fruit and vegetables, according to the scientists.
The study was published in the Clinical Journal of American Nephrology. The study by Dr. Robert O. Young has been approved for publication in The International Journal of Complementary and Alternative Medicine.
“The cure for cancer is found in its prevention NOT in its treatment” Dr. Robert O. Young
Abstract
An anti-cancer lifestyle and diet is an important strategy you can use to reduce your risk for ANY cancerous condition. The American Cancer Society recommends, for example, that you eat at least five servings of fruit and vegetables daily and eat the right amount of (alkaline) food to stay at a healthy weight. In addition, researchers are finding that certain plant foods or herbs may be particularly useful in protecting and reversing many cancerous condition. The following article covers several of these medicinal herbs and their benefits in the prevention and treatment of brain cancer, lung cancer, breast cancer, blood cancers, prostate cancer, oral cancer, liver cancer thyroid cancer, kidney cancer, bowel cancer, stomach cancer, skin cancer and pancreatic cancer.
Make room in your diet for the following foods and drinks to prevent cancer. Why? Because an ounce of prevention is worth more than a pound of cure. The following are eleven herbs or spices that have been shown to be effective in the prevention, treatment and reversal of many cancerous conditions.
Garlic
[Figure 1: Cloves of garlic]
Several large studies have found that those who eat more garlic are less likely to develop various kinds of cancer, especially in digestive organs such as the esophagus, stomach, and colon. Ingredients in the pungent bulbs may keep cancer-causing substances in your body from working, or they may keep cancer cells from multiplying. I recommend a clove a day may be helpful.[1=22 ]
Cayenne Pepper
Most people know cayenne pepper for its spice. But it is actually an extraordinarily strong antioxidant and vaso-dialator. Studies have shown that by consuming cayenne pepper is highly alkaline and a powerful buffer of dietary and metabolic acids that cause cells to become cancerous. If you consume it regularly you can neutralize the acids that cause body cells to become cancerous.[23-29 ]
{Figure 2: Cyenne pepper]
Milk Thistle
Milk thistle is a crucial plant when it comes to liver health and cancer prevention. Milk thistle and the seeds from the plant can be used to eliminate acidic toxins that can bind to the liver, causing damage to the liver setting the stage for a cancerous condition. It protects the alkaline interstitial fluids that surround every body cell protecting them and indirectly preventing the formation of tumors, calcifications and/or cysts which make milk thistle a powerful antioxidant in the chelation of dietary and/or metabolic acids that cause cancer.[30-64 ]
[Figure 3: Milk Thistle]
Turmeric
[Figure 4: Turmeric root and spice]
This orange-colored spice, a staple in Indian curries, contains an ingredient called curcumin (not the same as cumin) that might be useful in reducing cancer risk. According to the American Cancer Society, curcumin can inhibit some kinds of cancer cells in laboratory studies and slow the spread of cancer or shrink tumors in some animals. Turmeric is easy to find in grocery stores, and you can use it in a variety of recipes.[65-130 ]
Bloodroot
Bloodroot is actually used in a medicine for treating cancer named Black Salve. You can use bloodroot on its own, because it has been shown in tests to be effective in shrinking of tumors.[131-159]
[Figure 5: Bloodroot plant and flower]
Feverfew
Feverfew was used in a study at a university in New York. The study found that it was great at killing off leukemia cells, even better than the actual cancer medication.[160-191]
[Figure 6: Feverfew plant and flower]
Wheatgrass
Consuming one tiny glass of wheatgrass a day either orally or even-better rectally has shown to dramatically increase the health of cancer patients and non-cancer patients alike. It is particularly useful for people who are suffering from the side effects of chemotherapy. It will help purify the blood from dietary and/or metabolic acids, improve blood and lymph circulation, increase the oxygen levels in the microenvironments, and help the body repair and continue to reduce acids loads in the extracellular fluids, interstitial fluids and intracellular fluids to prevent and/or reverse and spoiling of the body cells.[192-204]
[Figure 7: Wheatgrass}
Ruscus Aculeatus
This herb is always known as Butchers Broom and it is great at fighting cancer due to its active ingredient, ruscogenins. The active ingredient has been proven to shrink tumors and increase the cancer fighting cells in the body.[205-221 ]
[Figure 8: Ruscus Aculeatus or Butchers Broom]
Sheep’s Sorrell
Sheep’s Sorrell can be used in people who are suffering the harmful effects of cancer medications. It helps the tissues rebuild and get back to the condition that they were in before the cancer and medication to use it was introduced. Some have suggested that it can be used to ward off cancer cells and keep them from growing.[222-224]
[Figure 9: Sheep’s Sorrel}
Astragalus
This herb is grown in China and has been proven to help with cancer on a couple of different levels. First it boosts your body’s immune system, which in turn helps it identify cancer cells. A study showed that cancer patients who took this herb survived twice as long.[225-250 ]
[Figure 10: Astragalus}
Ginger
[Figure 11: Ginger root]
A new study reveals ginger contains a pungent compound that could be up to 10,000 times more effective than conventional chemotherapy in targeting the cancer stem cells at the root of cancer malignancy. [251]
[Figure 12: Research Shows The Efficacy of Ginger Root as a non-toxic form of chemotherapy]
The authors of the study further affirm these points:
“Cancer stem cells pose serious obstacle to cancer therapy as they can be responsible for poor prognosis and tumour relapse. To add into the misery, very few chemotherapeutic compounds show promise to kill these cells. Several researchers have shown that cancer stem cells are resistant to paclitaxel, doxorubicin, 5-fluorouracil, and platinum drugs [8, 16]. CSCs are thus an almost unreachable population in tumours for chemotherapy. Therefore any compound, that shows promise towards cancer stem cells, is a highly desirable step towards cancer treatment and should be followed up for further development.”
The researchers identified a variety of ways by which 6-shagoal targets breast cancer:
It reduces the expression of CD44/CD24 cancer stem cell surface markers in breast cancer spheroids (3-dimensional cultures of cells modeling stem cell like cancer)
It significantly affects the cell cycle, resulting in increased cancer cell death
It induces programmed cell death primarily through the induction of autophagy, with apoptosis a secondary inducer
It inhibits breast cancer spheroid formation by altering Notch signaling pathway through γ-secretase inhibition.
It exhibits cytotoxicity (cell killing properties) against monolayer (1-dimensional cancer model) and spheroid cells (3-dimensional cancer model)
It was in evaluating the last mode of 6-shagoal’s chemotherapeutic activity and comparing it to the activity of the conventional chemotherapeutic agent taxol that the researchers discovered an astounding difference. Whereas taxol exhibited clear cytotoxicity in the one-dimensional (flat) monolayer experimental model, it had virtually no effect on the spheroid model, which is a more “real world” model reflecting the 3-dimensionality of tumors and their stem cell subpopulations. Amazingly, this held true even when the concentration of taxol was increased by four orders of magnitude:
“In contrast [to 6-shagoal], taxol, even though was highly active in monolayer cells, did not show activity against the spheroids even at 10000 fold higher concentration compared to 6-shogoal.”
This is a highly significant finding, as it affirms a common theme in cancer research that acknowledges the primarily role of cancer stem cells: namely, while conventional techniques like surgery, radiation, and chemotherapy are effective at reducing a tumor’s size, sometimes to the point where it is “debulked,” burned,” or “poisoned” out of the body even below the threshold of re-detection, the appearance of “winning the battle” often comes at a steep price, as ultimately the cancer stem cell population regrows the tumors, now with increased vengeance and metastastic invasiveness, resulting in the cancer “winning the war.”
The monolayer model, which does not account for the complex immunity of actual cancer stem-cell based tumors against chemoagents like taxol, represents the old preclinical model of testing cancer treatments. The spheroid model, on the other hand, clearly shows that even 10,000 times higher concentrations of taxol are not capable of beating this ginger component at selectively targeting the root cause of the tumor malignancy.
In their concluding remarks, the authors point out a hugely important distinction between natural anti-cancer agents and conventional ones that have only been introduced in the past half century or so, namely, “Dietary compounds are welcome options for human diseases due to their time-tested acceptability by human bodies.”
Unlike modern synthetically produced and patented chemicals, ginger, curcumin, garlic, and hundreds of other compounds naturally found in the human diet, have been “time-tested” as acceptable to the human body in the largest and longest running “clinical trials” known: the tens of thousands of years of direct human experience, spanning thousands of different cultures from around the world, that constitute human prehistory. These experientially-based “trials” are validated not by RCTs, or a peer-reviewed publication process, but by the fact that we all made it through this incalculably vast span of time to be alive here today. Consider also that if our ancestors made the wrong dietary choice by simply mistaking an edible berry for a poisonous one, the consequences could be deadly. This places even greater emphasis on how the “time testing” of dietary compounds was not an academic but a life-death affair, and by implication, how the information contained within various cultural traditions as “recipes” passed down from generation to generation are “epigenetic inheritance systems” no less important to our health and optimal gene expression as the DNA in our own bodies.
Ultimately, this new study adds to a growing body of research indicating that cancer stem cell targeting approaches using natural substances present in the human diet for thousands of years are far superior than chemotherapy and radiation, both of which actually increase the relative populations of cancer stem cells versus non-tumorigenic ones.[251]
Cannabis
[Figure 11: Cannabis plant with buds]
Cannabis has been making a lot of noise lately. Multiple states across the United States and countries around the world have successfully legalized medical Marijuana, and the Uruguay parliament recently voted to create the world’s first legal marijuana market.[252-256] This is good news as the health benefits of Cannabis are vast, with multiple medical and scientific studies that confirm them. On the other hand, arguments against the use of marijuana is usually published in Psychiatric journals, which show no scientific evidence that Cannabis is harmful to human health. All psychological evaluations from the intake of cannabis are largely based on assumptions, suggestions and observations (257). When we look at the actual science behind Cannabis, the health benefits can be overwhelming. So what does one who opposes the use of cannabis base their belief on? Nothing, not scientific evidence anyways. The negative stigmatism attached to marijuana is due to it’s supposed psychotropic effects, yet again, there is no scientific evidence to show that marijuana has any psychotropic effects. Nonetheless, cannabis has recently been the focus of medical research and considered as a potential therapeutic treatment and cure for cancer.Cannabis is a great example of how the human mind is programmed and conditioned to believe something. Growing up, we are told drugs are bad, which is true, however not all substances that have been labelled as “drugs” by the government are harmful. Multiple substances are labelled as a “drug” in order to protect corporate interests. One example is the automobile and energy industry, a car made from hemp is stronger than steel, and can be fuelled from hemp alone. Henry Ford demonstrated this many years ago. Hemp actually has over 50,000 uses![258]Let’s take a look at the science behind Cannabis and Cancer. Although Cannabis has been proven to be effective for a large range of ailments, this article will focus mainly on it’s effectiveness in the treatment of cancer. Cannabinoids may very well be one of the best disease and cancer fighting treatments out there. Cannabinoids refer to any of a group of related compounds that include cannabinol and the active constituents of cannabis. They activate cannabinoid receptors in the body. The body itself produces compounds called endocannabinoids and they play a role in many processes within the body that help to create a healthy environment. Cannabinoids also play a role in immune system generation and re-generation. The body regenerates best when it’s saturated with Phyto-Cannabinoids. Cannabinoids can also be found in Cannabis. It is important to note that the cannabinoids are plentiful in both hemp and cannabis. One of the main differentiations between hemp and cannabis is simply that hemp only contains 0.3% THC while cannabis is 0.4% THC or higher. (Technically they are both strains of Cannabis Sativa.) Cannabinoids have been proven to reduce cancer cells as they have a great impact on the rebuilding of the immune system. While not every strain of cannabis has the same effect, more and more patients are seeing success in cancer reduction in a short period of time by using cannabis.While taking a look at these studies, keep in mind that cannabis can be much more effective for medicinal purposes when we eat it rather than smoking it. Below are 20 medical studies that prove cannabis can be an effective treatment and possible cure for cancer.[ [259-288] Please keep in mind that this is a very short list of studies that support the use of medicinal marijuana. Please feel free to further your research, hopefully this is a good starting point.
Brain Cancer
A study published in the British Journal of Cancer, conducted by the Department of Biochemistry and Molecular Biology at Complutense University in Madrid, this study determined that Tetrahydrocannabinol (THC) and other cannabinoids inhibit tumour growth. They were responsible for the first clinical study aimed at assessing cannabinoid antitumoral action. Cannabinoid delivery was safe and was achieved with zero psychoactive effects. THC was found to decrease tumour cells in two out of the nine patients.[289]A study published in The Journal of Neuroscienceexamined the biochemical events in both acute neuronal damage and in slowly progressive, neurodegenerative diseases. They conducted a magnetic resonance imaging study that looked at THC (the main active compound in marijuana) and found that it reduced neuronal injury in rats. The results of this study provide evidence that the cannabinoid system can serve to protect the brain against neurodegeneration.[290]A study published in The Journal of Pharmacology And Experimental Therapeuticsalready acknowledged the fact that cannabinoids have been shown to possess antitumor properties. This study examined the effect of cannabidiol (CBD, non psychoactive cannabinoid compound) on human glioma cell lines. The addition of cannabidiol led to a dramatic drop in the viability of glioma cells. Glioma is the word used to describe brain tumour. The study concluded that cannabidiol was able to produce a significant antitumor activity.[291]A study published in the journal Molecular Cancer Therapeuticsoutlines how brain tumours are highly resistant to current anticancer treatments, which makes it crucial to find new therapeutic strategies aimed at improving the poor prognosis of patients suffering from this disease. This study also demonstrated the reversal of tumour activity in Glioblastoma multiforme.[292]
A study published in the journal Oncogene, by Harvard Medical Schools Experimental Medicine Department determined that THC inhibits epithelial growth factor induced lung cancer cell migration and more. They go on to state that THC should be explored as novel therapeutic molecules in controlling the growth and metastasis of certain lung cancers.[297 ]A study published by the US National Library of Medicine by the Institute of Toxicology and Pharmacology, from the Department of General Surgery in Germany determined that cannabinoids inhibit cancer cell invasion. Effects were confirmed in primary tumour cells from a lung cancer patient. Overall, data indicated that cannabinoids decrease cancer cell invasiveness.[298 ]A study published by the US National Library of Medicine, conducted by Harvard Medical School investigated the role of cannabinoid receptors in lung cancer cells. They determined its effectiveness and suggested that it should be used for treatment against lung cancer cells.[299 ]
A study published in the journal Molecular Pharmacologyrecently showed that cannabinoids induce growth inhibition and apoptosis in matle cell lymphoma. The study was supported by grants from the Swedish Cancer Society, The Swedish Research Council and the Cancer Society in Stockholm.[303]A study published in the International Journal of Cancer also determined and illustrated that cannabinoids exert antiproliferative and proapoptotic effects in various types of cancer and in mantle cell lymphoma.[304]A study published in the US National Library of Medicine conducted by the Department of Pharmacology and Toxicology by Virginia Commonwealth University determined that cannabinoids induce apoptosis in leukemia cells.[305]
A study published in The American Journal of Cancer determined that cannabinoid receptors are expressed in human pancreatic tumor cell lines and tumour biopsies at much higher levels than in normal pancreatic tissue. Results showed that cannabinoid administration induced apoptosis. They also reduced the growth of tumour cells, and inhibited the spreading of pancreatic tumour cells.[308]
Conclusion
According to a 2004 report by Morgan, Ward, and Barton: “The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies. … survival in adults was estimated to be 2.3% in Australia and 2.1% in the USA.”Jun 16, 2014[309]
Medical oncologists are a long way from using medicinal herbs as an alternative or primary treatment for cancer. The research is significant and shows that the medicinal herbs discussed in this article are extraordinary plants and have shown excellent results in the prevention, treatment and reversal of many cancerous conditions.
References:
Milner JA. Garlic: Its anticarcinogenic and antitumorigenic properties. Nutrition Reviews1996; 54:S82–S86.
Ross SA, Finley JW, Milner JA. Allyl sulfur compounds from garlic modulate aberrant crypt formation. Journal of Nutrition 2006; 136(3 Suppl):852S–854S.
Amagase H, Petesch BL, Matsuura H, Kasuga S, Itakura Y. Intake of garlic and its bioactive components. Journal of Nutrition 2001; 131(3s):955S–962S.
Amagase H. Clarifying the real bioactive constituents of garlic. Journal of Nutrition 2006; 136(3 Suppl):716S–725S.
Fleischauer AT, Arab L. Garlic and cancer: A critical review of the epidemiologic literature. Journal of Nutrition 2001; 131(3s):1032S–1040S.
Gonzalez CA, Pera G, Agudo A, et al. Fruit and vegetable intake and the risk of stomach and oesophagus adenocarcinoma in the European Prospective Investigation into Cancer and Nutrition (EPIC-EURGAST). International Journal of Cancer 2006; 118(10): 2559–2566.
Steinmetz KA, Kushi LH, Bostick RM, Folsom AR, Potter JD. Vegetables, fruit, and colon cancer in the Iowa Women’s Health Study. American Journal of Epidemiology 1994; 139(1):1–15.
Gao CM, Takezaki T, Ding JH, Li MS, Tajima K. Protective effect of allium vegetables against both esophageal and stomach cancer: A simultaneous case-referent study of a high-epidemic area in Jiangsu Province, China. Japanese Journal of Cancer Research1999; 90(6):614–621.
Setiawan VW, Yu GP, Lu QY, et al. Allium vegetables and stomach cancer risk in China. Asian Pacific Journal of Cancer Prevention 2005; 6(3):387–395.
Hsing AW, Chokkalingam AP, Gao YT, et al. Allium vegetables and risk of prostate cancer: A population-based study. Journal of the National Cancer Institute 2002; 94(21):1648–1651.
Chan JM, Wang F, Holly EA. Vegetable and fruit intake and pancreatic cancer in a population-based case-control study in the San Francisco bay area. Cancer Epidemiology Biomarkers & Prevention 2005; 14(9):2093–2097.
Challier B, Perarnau JM, Viel JF. Garlic, onion and cereal fibre as protective factors for breast cancer: A French case-control study. European Journal of Epidemiology 1998; 14(8):737–747.
Li H, Li HQ, Wang Y, et al. An intervention study to prevent gastric cancer by micro-selenium and large dose of allitridum. Chinese Medical Journal (English) 2004; 117(8):1155–1160.
You WC, Brown LM, Zhang L, et al. Randomized double-blind factorial trial of three treatments to reduce the prevalence of precancerous gastric lesions. Journal of the National Cancer Institute 2006; 98(14):974–983.
Tanaka S, Haruma K, Kunihiro M, et al. Effects of aged garlic extract (AGE) on colorectal adenomas: A double-blinded study. Hiroshima Journal of Medical Sciences 2004; 53(3–4):39–45.
Tilli CM, Stavast-Kooy AJ, Vuerstaek JD, et al. The garlic-derived organosulfur component ajoene decreases basal cell carcinoma tumor size by inducing apoptosis. Archives of Dermatological Research 2003; 295(3):117–123.
Ruddock PS, Liao M, Foster BC, et al. Garlic natural health products exhibit variable constituent levels and antimicrobial activity against Neisseria gonorrhoeae, Staphylococcus aureus and Enterococcus faecalis. Phytotherapy Research 2005; 19(4):327–334.
Shenoy NR, Choughuley AS. Inhibitory effect of diet related sulphydryl compounds on the formation of carcinogenic nitrosamines. Cancer Letters 1992; 65(3):227–232.
Milner JA. Mechanisms by which garlic and allyl sulfur compounds suppress carcinogen bioactivation. Garlic and carcinogenesis. Advances in Experimental Medicine and Biology2001; 492:69–81.
L’vova GN, Zasukhina GD. Modification of repair DNA synthesis in mutagen-treated human fibroblasts during adaptive response and the antimutagenic effect of garlic extract. Genetika 2002; 38(3):306–309.
Boon H, Wong J. Botanical medicine and cancer: A review of the safety and efficacy. Expert Opinion on Pharmacotherapy 2004; 5(12):2485–2501.
Piscitelli SC, Burstein AH, Welden N, Gallicano KD, Falloon J. The effect of garlic supplements on the pharmacokinetics of saquinavir. Clinical Infectious Diseases 2002; 34(2):234–238.Athanasiou A., et al. Vanilloid receptor agonists and antagonists are mitochondrial inhibitors: how vanilloids cause non-vanilloid receptor mediated cell death. Biochem Biophys Res Commun 2007 Mar 2; 354(1): 50-55.
Athanasiou A., et al. Vanilloid receptor agonists and antagonists are mitochondrial inhibitors: how vanilloids cause non-vanilloid receptor mediated cell death. 2007 Mar 2; 354(1): 50-55.
Christensen, Jon. (December 12, 2005). Retrieved from The New York Times. Scientist at work: John Reed; running hot in pursuit of cancer treatment).
Mori, Akio, et al. (March 15, 2006). Capsaicin, A Component of Red Peppers, Inhibits The Growth of Androgen-Independent, p53 Mutant Prostate Cancer Cells. American Association of Cancer Research.
Samid, Dvorit. (1997). Compositions and methods for treating and preventing pathologies including cancer. The USA Dept. of HHS; pg. 56.
Sanchez AM et al. Apoptosis induced by capsaicin in prostate PC-3 cells involves ceramide accumulation, neutral sphingomyelinase, and JNK activation. Apoptosis 2007; 12(11): 2013-24.
Thoennissen NH, O’Kelly J, Lu D, et al. Capsaicin causes cell-cycle arrest and apoptosis in ER-positive and -negative breast cancer cells by modulating the EGFR/HER-2 pathway. Oncogene. 2010, Jan 14;29(2):285-96.
PDR® for Herbal Medicines™. 2nd ed. Montvale, NJ: Medical Economics, 2000.
Lee DY, Liu Y: Molecular structure and stereochemistry of silybin A, silybin B, isosilybin A, and isosilybin B, Isolated from Silybum marianum (milk thistle). J Nat Prod 66 (9): 1171-4, 2003. [PUBMED Abstract]
Napolitano JG, Lankin DC, Graf TN, et al.: HiFSA fingerprinting applied to isomers with near-identical NMR spectra: the silybin/isosilybin case. J Org Chem 78 (7): 2827-39, 2013. [PUBMED Abstract]
Hruby K, Csomos G, Fuhrmann M, et al.: Chemotherapy of Amanita phalloides poisoning with intravenous silibinin. Hum Toxicol 2 (2): 183-95, 1983. [PUBMED Abstract]
Wagner H, Hörhammer L, Münster R: [On the chemistry of silymarin (silybin), the active principle of the fruits from Silybum marianum (L.) Gaertn. (Carduus marianus L.)] Arzneimittelforschung 18 (6): 688-96, 1968. [PUBMED Abstract]
Campos R, Garrido A, Guerra R, et al.: Silybin dihemisuccinate protects against glutathione depletion and lipid peroxidation induced by acetaminophen on rat liver. Planta Med 55 (5): 417-9, 1989. [PUBMED Abstract]
Farghali H, Kameniková L, Hynie S, et al.: Silymarin effects on intracellular calcuim and cytotoxicity: a study in perfused rat hepatocytes after oxidative stress injury. Pharmacol Res 41 (2): 231-7, 2000. [PUBMED Abstract]
Lettéron P, Labbe G, Degott C, et al.: Mechanism for the protective effects of silymarin against carbon tetrachloride-induced lipid peroxidation and hepatotoxicity in mice. Evidence that silymarin acts both as an inhibitor of metabolic activation and as a chain-breaking antioxidant. Biochem Pharmacol 39 (12): 2027-34, 1990. [PUBMED Abstract]
Zhao J, Agarwal R: Tissue distribution of silibinin, the major active constituent of silymarin, in mice and its association with enhancement of phase II enzymes: implications in cancer chemoprevention. Carcinogenesis 20 (11): 2101-8, 1999. [PUBMED Abstract]
Valenzuela A, Guerra R, Videla LA: Antioxidant properties of the flavonoids silybin and (+)-cyanidanol-3: comparison with butylated hydroxyanisole and butylated hydroxytoluene. Planta Med (6): 438-40, 1986. [PUBMED Abstract]
Valenzuela A, Guerra R, Garrido A: Silybin dihemisuccinate protects rat erythrocytes against phenylhydrazine-induced lipid peroxidation and hemolysis. Planta Med 53 (5): 402-5, 1987. [PUBMED Abstract]
Valenzuela A, Aspillaga M, Vial S, et al.: Selectivity of silymarin on the increase of the glutathione content in different tissues of the rat. Planta Med 55 (5): 420-2, 1989. [PUBMED Abstract]
Mira ML, Azevedo MS, Manso C: The neutralization of hydroxyl radical by silibin, sorbinil and bendazac. Free Radic Res Commun 4 (2): 125-9, 1987. [PUBMED Abstract]
Mira L, Silva M, Manso CF: Scavenging of reactive oxygen species by silibinin dihemisuccinate. Biochem Pharmacol 48 (4): 753-9, 1994. [PUBMED Abstract]
Koch HP, Löffler E: Influence of silymarin and some flavonoids on lipid peroxidation in human platelets. Methods Find Exp Clin Pharmacol 7 (1): 13-8, 1985. [PUBMED Abstract]
Garrido A, Arancibia C, Campos R, et al.: Acetaminophen does not induce oxidative stress in isolated rat hepatocytes: its probable antioxidant effect is potentiated by the flavonoid silybin. Pharmacol Toxicol 69 (1): 9-12, 1991. [PUBMED Abstract]
Bosisio E, Benelli C, Pirola O: Effect of the flavanolignans of Silybum marianum L. on lipid peroxidation in rat liver microsomes and freshly isolated hepatocytes. Pharmacol Res 25 (2): 147-54, 1992 Feb-Mar. [PUBMED Abstract]
Altorjay I, Dalmi L, Sári B, et al.: The effect of silibinin (Legalon) on the the free radical scavenger mechanisms of human erythrocytes in vitro. Acta Physiol Hung 80 (1-4): 375-80, 1992. [PUBMED Abstract]
El-Shitany NA, Hegazy S, El-Desoky K: Evidences for antiosteoporotic and selective estrogen receptor modulator activity of silymarin compared with ethinylestradiol in ovariectomized rats. Phytomedicine 17 (2): 116-25, 2010. [PUBMED Abstract]
Scambia G, De Vincenzo R, Ranelletti FO, et al.: Antiproliferative effect of silybin on gynaecological malignancies: synergism with cisplatin and doxorubicin. Eur J Cancer 32A (5): 877-82, 1996. [PUBMED Abstract]
Bhatia N, Zhao J, Wolf DM, et al.: Inhibition of human carcinoma cell growth and DNA synthesis by silibinin, an active constituent of milk thistle: comparison with silymarin. Cancer Lett 147 (1-2): 77-84, 1999. [PUBMED Abstract]
Zi X, Agarwal R: Silibinin decreases prostate-specific antigen with cell growth inhibition via G1 arrest, leading to differentiation of prostate carcinoma cells: implications for prostate cancer intervention. Proc Natl Acad Sci U S A 96 (13): 7490-5, 1999. [PUBMED Abstract]
Duthie SJ, Johnson W, Dobson VL: The effect of dietary flavonoids on DNA damage (strand breaks and oxidised pyrimdines) and growth in human cells. Mutat Res 390 (1-2): 141-51, 1997. [PUBMED Abstract]
Vailati A, Aristia L, Sozzé E, et al.: Randomized open study of the dose-effect relationship of a short course of IdB 1016 in patients with viral or alcoholic hepatitis. Fitoterapia 64 (3), 219-28, 1993.
Salmi HA, Sarna S: Effect of silymarin on chemical, functional, and morphological alterations of the liver. A double-blind controlled study. Scand J Gastroenterol 17 (4): 517-21, 1982. [PUBMED Abstract]
Parés A, Planas R, Torres M, et al.: Effects of silymarin in alcoholic patients with cirrhosis of the liver: results of a controlled, double-blind, randomized and multicenter trial. J Hepatol 28 (4): 615-21, 1998. [PUBMED Abstract]
Moscarella S, Giusti A, Marra F, et al.: Therapeutic and antilipoperoxidant effects of silybin-phosphatidylcholine complex in chronic liver disease: preliminary results. Current Therapeutic Research 53 (1): 98-102.
Marena C, Lampertico M: Preliminary clinical development of silipide: a new complex of silybin in toxic liver disorders. Planta Med 57 (Suppl 2): A124-5, 1991.
Marcelli R, Bizzoni P, Conte D, et al.: Randomized controlled study of the efficacy and tolerability of a short course of IdB 1016 in the treatment of chronic persistent hepatitis. European Bulletin of Drug Research 1 (3): 131-5, 1992.
Flisiak R, Prokopowicz D: Effect of misoprostol on the course of viral hepatitis B. Hepatogastroenterology 44 (17): 1419-25, 1997 Sep-Oct. [PUBMED Abstract]
Ferenci P: [Therapy of chronic hepatitis C] Wien Med Wochenschr 150 (23-24): 481-5, 2000. [PUBMED Abstract]
Buzzelli G, Moscarella S, Giusti A, et al.: Therapeutic effects of a new silybin complex in chronic active hepatitis (CAH). [Abstract] Hellenic Journal of Gastroenterology 5 (Suppl): A-151, 38, 1992.
Albrecht M, Frerick H, Kuhn U, et al.: Therapy of toxic liver pathologies with Legalon®. Z Klin Med 47: 87-92, 1992.
Rambaldi A, Jacobs BP, Gluud C: Milk thistle for alcoholic and/or hepatitis B or C virus liver diseases. Cochrane Database Syst Rev (4): CD003620, 2007. [PUBMED Abstract]
Yang Z, Zhuang L, Lu Y, et al.: Effects and tolerance of silymarin (milk thistle) in chronic hepatitis C virus infection patients: a meta-analysis of randomized controlled trials. Biomed Res Int 2014: 941085, 2014. [PUBMED Abstract]
Anasuya Ray, Smreti Vasudevan, Suparna Sengupta, 6-Shogaol Inhibits Breast Cancer Cells and Stem Cell-Like Spheroids by Modulation of Notch Signaling Pathway and Induction of Autophagic Cell Death. Published: September 10, 2015. DOI: 10.1371/journal.pone.0137614
A. JOHNS, Psychiatric effects of cannabis. SUBSTANCE MISUSE PAPERS, The British Journal of Psychiatry Feb 2001, 178 (2) 116-122; DOI: 10.1192/bjp.178.2.116.
IBID.
Abrams DI, Jay CA, Shade SB, et al. Cannabis in painful HIV-associated sensory neuropathy: a randomized placebo-controlled trial. Neurology. 2007;68(7):515-521.
Ahmedzai S, Carlyle DL, Calder IT, Moran F. Anti-emetic efficacy and toxicity of nabilone, a synthetic cannabinoid, in lung cancer chemotherapy. Br J Cancer. 1983;48(5):657-663.
Bhattacharyya S, Crippa JA, Allen P, et al. Induction of psychosis by {delta}9-tetrahydrocannabinol reflects modulation of prefrontal and striatal function during attentional salience processing. Arch Gen Psychiatry. 2012;69(1):27-36.
Beal JE, Olson R, Laubenstein L, et al. Dronabinol as a treatment for anorexia associated with weight loss in patients with AIDS. J Pain Symptom Manage. 1995;10(2):89-97.
Beal JE, Olson R, Lefkowitz L, et al. Long-term efficacy and safety of dronabinol for acquired immunodeficiency syndrome-associated anorexia. J Pain Symptom Manage. 1997;14(1):7-14.
Cannabis-In-Cachexia-Study-Group, Strasser F, Luftner D, Possinger K, et al. Comparison of orally administered cannabis extract and delta-9-tetrahydrocannabinol in treating patients with cancer-related anorexia-cachexia syndrome: a multicenter, phase III, randomized, double-blind, placebo-controlled clinical trial from the Cannabis-In-Cachexia-Study-Group. J Clin Onc. 2006;24:3394-3400.
Ellis RJ, Toperoff W, Vaida F, et al. Smoked medicinal cannabis for neuropathic pain in HIV: a randomized, crossover clinical trial. Neuropsychopharmacology. 2009;34(3):672-680.
Guzmán M, Duarte MJ, Blázquez C, et al. A pilot clinical study of Delta9-tetrahydrocannabinol in patients with recurrent glioblastoma multiforme. Br J Cancer. 2006;95(2):197-203.
Haney M, Rabkin J, Gunderson E, Foltin RW. Dronabinol and marijuana in HIV(+) marijuana smokers: acute effects on caloric intake and mood. Psychopharmacology. (Berl). 2005;181:170-178.
Haney M, Gunderson EW, Rabkin J, et al. Dronabinol and marijuana in HIV-positive marijuana smokers. Caloric intake, mood,and sleep. J Acquir Immune Defic Syndr. 2007;45(5):545-554.
Herman TS, Einhorn LH, Jones SE, et al. Superiority of nabilone over prochlorperazine as an antiemetic in patients receiving cancer chemotherapy. N Engl J Med. 1979;300(23):1295-1297.
Jatoi A, Windschitl HE, Loprinzi CL, et al. Dronabinol versus megestrol acetate versus combination therapy for cancer-associated anorexia: a North Central Cancer Treatment Group study. J Clin Oncol. 2002;20(2):567-573.
Johnson JR, Burnell-Nugent M, Lossignol D, et al. Multicenter, double-blind, randomized, placebo-controlled, parallel-group study of the efficacy, safety, and tolerability of THC:CBD extract and THC extract in patients with intractable cancer-related pain. J Pain Symptom Manage. 2010;39(2):167-179.
Johnson JR, Lossignol D, Burnell-Nugent M, Fallon MT. An open-label extension study to investigate the long-term safety and tolerability of THC/CBD oromucosal spray and oromucosal THC spray in patients with terminal cancer-related pain refractory to strong opioid analgesics. J Pain Symptom Manage. 2013;46(2):207-218.
Karst M, Salim K, Burstein S, et al. Analgesic effect of the synthetic cannabinoid CT-3 on chronic neuropathic pain: a randomized controlled trial. JAMA. 2003;290:1757-1762.
Koppel BS, Brust JC, Fife T, et al. Systematic review: efficacy and safety of medical marijuana in selected neurologic disorders: report of the Guideline Development Subcommittee of the American Academy of Neurology. Neurology. 2014;82(17):1556-1563.
Kramer JL. Medical marijuana for cancer. CA Cancer J Clin. 2014 Dec 10.
Meiri E, Jhangiani H, Vredenburgh JJ, et al. Efficacy of dronabinol alone and in combination with ondansetron versus ondansetron alone for delayed chemotherapy-induced nausea and vomiting. Curr Med Res Opin. 2007;23(3):533-543.
Musty RE, Rossi R. Effects of Smoked Cannabis and Oral Δ9-Tetrahydrocannabinol on Nausea and Emesis After Cancer Chemotherapy: A Review of State Clinical Trials. Journal of Cannabis Therapeutics. 2001; 1(1): 29-56.
Portenoy RK, Ganae-Motan ED, Allende S, et al. Nabiximols for opioid-treated cancer patients with poorly-controlled chronic pain: a randomized, placebo-controlled, graded-dose trial. J Pain. 2012;13(5):438-449.
Radwan MM, Elsohly MA, Slade D, et al. Biologically active cannabinoids from high-potency Cannabis sativa. J Nat Prod. 2009;72(5):906-911.
Rog DJ, Nurmikko TJ, Young CA. Oromucosal delta9-tetrahydrocannabinol/cannabidiol for neuropathic pain associated with multiple sclerosis: an uncontrolled, open-label, 2-year extension trial. Clin Ther. 2007;29(9):2068-2079.
Ross SA, ElSohly MA, Sultana GN, et al. Flavonoid glycosides and cannabinoids from the pollen of Cannabis sativa L. Phytochem Anal. 2005;16(1):45-48.
Smith PF. New approaches in the management of spasticity in multiple sclerosis patients: role of cannabinoids. Ther Clin Risk Manag. 2010;6:59-63.
Tramér MR, Carroll D, Campbell FA, et al. Cannabinoids for control of chemotherapy induced nausea and vomiting: quantitative systematic review. BMJ. 2001;323:16-21.
Ware MA, Wang T, Shapiro S, et al. Smoked cannabis for chronic neuropathic pain: a randomized controlled trial. CMAJ. 2010;182(14):E694-701.
Woolridge E, Barton S, Samuel J, et al. Cannabis use in HIV for pain and other medical symptoms. J Pain Symptom Manage. 2005;29:358-367.
M Guzmán, M J Duarte, C Blázquez, J Ravina, M C Rosa, I Galve-Roperh, C Sánchez, G Velasco and L González-Feria, A pilot clinical study of Δ9-tetrahydrocannabinol in patients with recurrent glioblastoma multiforme, British Journal of Cancer (2006) 95, 197–203. doi:10.1038/sj.bjc.6603236 www.bjcancer.com Published online 27 June 2006.
M. van der Stelt, W. B. Veldhuis, P. R. Bär, G. A. Veldink, J. F. G. Vliegenthart, and K. Nicolay, Neuroprotection by Δ9-Tetrahydrocannabinol, the Main Active Compound in Marijuana, against Ouabain-Induced In Vivo Excitotoxicity. The Journal of Neuroscience, 1 September 2001, 21(17): 6475-6479.
Alessia Ligresti, Aniello Schiano Moriello, Katarzyna Starowicz, Isabel Matias, Simona Pisanti, Luciano De Petrocellis, Chiara Laezza, Giuseppe Portella, Maurizio Bifulco and Vincenzo Di Marzo, Anti-tumor activity of plant cannabinoids with emphasis on the effect of cannabidiol on human breast carcinoma. Endocannabinoid Research Group, Istituto di Chimica Biomolecolare, CNR Pozzuoli, Italy (AL, ASM, KS, IM, VDM); Istituto di Cibernetica, CNR Pozzuoli, Italy (ASM, LDP); Dipartimento di Biologia e Patologia Cellulare e Molecolare “L.Califano”, Università di Napoli “Federico II”(SP, CL, GP, MB) and Dipartimento di Scienze Farmaceutiche, Università degli Studi di Salerno, Fisciano, Italy (SP, MB).
María M Caffarel, Clara Andradas, Emilia Mira, Eduardo Pérez-Gómez, Camilla Cerutti, Gema Moreno-Bueno, Juana M Flores, Isabel García-Real, José Palacios, Santos Mañes, Manuel Guzmán and Cristina Sánchez, Cannabinoids reduce ErbB2-driven breast cancer progression through Akt inhibition, Molecular Cancer20109:196, 22 July 2010.
LUCIANO DE PETROCELLIS, DOMINIQUE MELCK, ANTONELLA PALMISANO§, TIZIANA BISOGNO, CHIARA LAEZZA, MAURIZIO BIFULCO, AND VINCENZO DI MARZO, The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation. Proc. Natl. Acad. Sci. USA, Vol. 95, pp. 8375–8380, July 1998 Pharmacology.
A Preet, R K Ganju, J E Groopman, Δ9-Tetrahydrocannabinol inhibits epithelial growth factor-induced lung cancer cell migration in vitro as well as its growth and metastasis in vivo. Oncogene (2008) 27, 339–346; doi:10.1038/sj.onc.1210641; published online 9 July 2007
Mimeault M, Pommery N, Wattez N, Bailly C, Hénichart JP. Anti-proliferative and apoptotic effects of anandamide in human prostatic cancer cell lines: implication of epidermal growth factor receptor down-regulation and ceramide production. Prostate. 2003 Jun 15;56(1):1-12.
Juan A. Ramos and Fernando J. Bianco, The role of cannabinoids in prostate cancer: Basic science perspective and potential clinical applications. Indian J Urol. 2012 Jan-Mar; 28(1): 9–14.
Kristin Gustafsson, Birger Christensson, Birgitta Sander and Jenny Flygare, Cannabinoid Receptor-Mediated Apoptosis Induced by R(+)-Methanandamide and Win55,212-2 Is Associated with Ceramide Accumulation and p38 Activation in Mantle Cell Lymphoma. Published online before print August 25, 2006, doi:10.1124/mol.106.025981Molecular PharmacologyNovember 2006 vol. 70 no. 5 1612-1620.
Kristin Gustafsson, Xiao Wang1, Denise Severa1, Maeve Eriksson1, Eva Kimby2, Mats Merup2, Birger Christensson1, Expression of cannabinoid receptors type 1 and type 2 in non-Hodgkin lymphoma: Growth inhibition by receptor activation. International Journal of Cancer, Volume 123, Issue 5, pages 1025–1033, 1 September 2008.
A. Carracedo, M. Gironella, M. Lorente, S. Garcia, M. Guzam, F. Velasco, J. Lovanna, Cannabinoids Induce Apoptosis of Pancreatic Tumor Cells via Endoplasmic Reticulum Stress–Related Genes. Cancer Research, The American Association of Cancer Research, 2006, May 5.
The Cure for Cancer? That’s an easy question to answer! The Cure for Cancer is Found in its Prevention NOT in its Treatment! – Dr. Robert O. Young
Do you know what rotten apples, grapefruit or bananas look like? If you do then you know what cancer cells look like. Cancer cells are nothing more that healthy cells that are spoiling because of a compromised environment! Look at the picture below and you will see colorized cancerous body cells rotting in their toxic acidic environment.
What compromises the internal environment of a human body that causes body cells to begin spoiling and rotting? The answer is simple! The body’s build-up of acidic metabolic and dietary waste that has not been properly eliminated through the four channels of elimination – urination, defecation, respiration and perspiration!
Cancer is not a noun but an adjective that describes what is happening to body cells in an acidic environment due to an acidic lifestyle and diet. www.phoreveryoung.com
To learn more about Dr. Robert O. Young go to: https://www.linkedin.com/in/drrobertoyoung
A former Los Angeles Times staff writer, Laurie Becklund, battled breast cancer since 1996. Earlier this year she knew her time was limited, and as she greeted her last few months, she wrote an opinion piece “As I Lay Dying” about her story. Becklund died Feb. 8 this year. This is what she wanted you to know about breast cancer.
Early detection does not cure cancer with mammograms DOES NOT SAVE LIVES!
Becklund: “I had more than 20 mammograms, and none of them caught my disease. In fact, we now have significant studies showing that routine mammogram screening, which may result in misdiagnoses, unnecessary treatment and radiation overexposure, can harm more people than it helps.”
To detect a cancer early in many cases means to catch it before it produces symptoms. That is a problem, because not every precancerous condition will actually become cancer or not the type of cancer that can affect a person’s life, but every case is treated as if it was the same type of cancer. Mammogram screening is responsible for about 25% of overdiagnosis in breast cancer, according to an article published in Oxford Journals. The overdiagnosis may harm patients and lead to “overuse of anticancer therapies” such as chemotherapy.
31% over diagnosed
Another article by The New England Journal of Medicine estimated that in 2008, 70,000 U.S. women were overdiagnosed with breast cancer, which is a shocking 31% of all breast cancer diagnoses.
The first time Becklund discovered a lump in her breast in 1996 during a self-exam, she was treated by a lumpectomy and radiation. She had the most “curable” type of breast cancer. Five years after the treatment her doctor told her she had minimal chance of it ever coming back.
Yet in 2009 she received a diagnosis of stage IV breast cancer that spread to her bones, liver, lungs and brain.
Metastatic breast cancer (MBC) is the only kind of breast cancer that kills
Metastatic cancer is “cancer that has spread from the place where it first started to another place in the body,” states Cancer.org. According to a non-profit patient advocacy group Metastatic Breast Cancer Network (MBCN) breast cancer itself does not kill, instead breast cancer patients die from cancer cells travelling to other vital organs.
Breast cancer most commonly spreads to bone, brain, liver and lung. And in Becklund’s case it spread to all four places. When she went to an MBCN conference other attendees were shocked that she was even alive. Almost everyone else had cancer spread to only one organ. When later a group of people she was in was asked to stand if they survived 2 years after diagnosis, most sat down. As far as she could see Becklund was the only one standing for 7 years of survival.
The medical establishment fails their patients
An estimated 40,000 MBC patients die annually. Another 250,000 are waiting for their death.
“I say ‘estimated because no one is required to report a metastatic diagnosis. Death certificates normally report symptoms such as “respiratory failure,” not the actual disease. We are literally uncounted,” Becklund wrote.
Lauire Becklund speaks to an audience. PHOTO: Stanford Medical X/Flickr
While the Surveillance, Epidemiology and End Results (SEER) Program is the main source for cancer statistics, it does not take into account metastatic breast cancer, according to MBCN. It is however estimated up to 30% of all cases are metastatic, and yet they are not counted. Moreover, only 2% of all breast cancer research has been estimated to go towards finding a solution for preventing or treating metastatic breast cancer, according to METAvivor, a non-profit MBC patients’ advocacy organization.
There is no one “cure”
“We are each, in effect, one-person clinical trials. Yet the knowledge generated from those trials will die with us because there is no comprehensive database of metastatic breast cancer patients…” Becklund wrote.
While there is a belief that if a person lives 5 years after the diagnosis they are a cancer survivor, for patients with MBC that means almost nothing. Though there is a treatment, MBC is incurable, according to Fred Hutchinson Cancer Research Center.
Early detection does not help MBC patients either. Another type of breast cancer that was once labeled “cured” by doctors, often comes back years later as stage IV metastatic. And one type of treatment does not work for all MBC patients.
Right now, while new therapies are just starting to emerge, there is still little hope for survival for MBC, and natural and holistic therapies that have shown promise are routinely ignored.
Susan G. Komen’s mission is not helping anyone
“Promise that you’ll never wear a pink ribbon in my name or drop a dollar into a bucket that goes to breast cancer ‘awareness’ for ‘early detection for a cure,’ the mantra of fund-raising juggernaut Susan G. Komen, which has propagated a distorted message about breast cancer and how to ‘cure’ it,” Becklund wrote.
I would be surprised if I could find one literate person who is not aware that breast cancer exists and that it is life-threatening for many patients. We are fully aware of that fact. Now what?
Susan G. Komen’s income was $287,409,269 in 2014 and allegedly 79% went into its programs for education, research and support, yet besides being aware, the money spent for over 30 years ($2.6 billion worth) did little for the survival rates of the breast cancer that actually kills – MBC.
“Pink is pretty, but it does not disguise the fact that metastatic breast cancer kills,” reads METAvivor take-action page.
For thousands of women and men who are dying from MBC right now, that is a more believable and honest public-awareness campaign. It is called The pH Miracle for Cancer – http://www.phoreveryoung.com
INVESTIGATION: Three days before Dr. Bradstreet was found dead in a river, U.S. govt. agents raided his research facility to seize a breakthrough cancer treatment called GcMAF
(NaturalNews) The history of the suppression of medical science in America is a long one, filled with true accounts of pioneering doctors and clinicians being threatened, intimidated and even assassinated in order to bury emerging cures and keep the “sick care” industry in control. (The American Medical Association, for example, has been found guilty by the U.S. federal courts of a conspiracy to destroy the chiropractic industry, by the way.)
Over the last few days, we’ve learned that before being found shot in the chest and floating in the river, pioneering medical researcher Dr. Bradstreet was working with a little-known molecule that occurs naturally in the human body. Called, “GcMAF”, this molecule has the potential to be a universal cancer cure for many people. It has also been shown to reverse signs of autism in the vast majority of patients receiving the treatment.
In this article, I summarize the videos, articles and documents covering GcMAF and the mysterious death of Dr. Bradstreet. An exhaustive investigation needs to be pursued on this matter, possibly involving private investigators. The timing and manner of Dr. Bradstreet’s death seems highly suspicious, especially in light of the many other holistic doctors who have recently been found dead under mysterious circumstances. (Dr. Nicholas Gonzalez died just days ago…)
Motive to murder medical researchers and suppress a promising cancer treatment breakthrough
Is there a motive for the murder of pioneering cancer researchers working on a possible universal cancer cure? Of course there is… it’s the most common motive in the world: MONEY.
A universal cancer cure would destroy the profitability of the highly lucrative cancer industry and collapse the American Cancer Society, hospitals, oncology clinics and pharmaceutical companies that depend on chemotherapy revenues to stay profitable. Key to their profitability is the inescapable fact that conventional cancer treatments simply don’t work most of the time (http://www.naturalnews.com/033847_chemotherapy_cancer_treatments.html), creating a reliable profit stream of repeat business from patients who are never cured (by design).
HealthNutNews story that covers the apparent series of murders of holistic doctors, many of whom are working on advanced treatment protocols that render high-profit sectors of conventional medicine OBSOLETE:
Yet another doctor was just found murdered inside his home here on the East Coast of Florida. This makes six doctors to be found dead in the last month just from this region of the country alone. Four out of the six were found dead here in Florida. We lost the holistic Dr. Teresa Sievers, MD, who was found murdered in her Florida home just weeks ago. We’ve also lost the alternative Dr. Jeff Bradstreet, MD, who was found in a river with a gunshot to his chest. He’d recently moved to Georgia from Florida. We’ve also lost the Osteopath. Dr. Riley, who was found in Georgia at her home; just a few hours from the Florida border. She was found with a gunshot wound to her head.
Now we’ve lost Dr. Schwartz MD, who was found murdered in his home, on Sunday, July 19th, 2015. This was four weeks to the day after the death of the first physician: (Dr. Bradstreet MD) who I broke the story on a month ago. His family is still seeking answers as to what happened to him and they’re some of the kindest people I know. The latest MD, Dr. Schwartz, in the picture above, lived just north of the fit, healthy, holistic Dr. Hedendal; who was the second doctor to be found dead this past Father’s Day, in Boca Raton. This was the same day that Dr. Holt died at the age of 33. Both were fathers; and again, both men died here in Florida on June 21st, 2015.
Stepwise incubation of purified Gc protein with immobilized beta-galactosidase and sialidase generated probably the most potent macrophage activating factor (termed GcMAF) ever discovered, which produces no adverse effect in humans…
After about 16-22 administrations (approximately 3.5-5 months) of GcMAF, these patients had insignificantly low serum enzyme levels equivalent to healthy control enzyme levels, ranging from 0.38 to 0.63 nmole/min/mg protein, indicating eradication of the tumors. This therapeutic procedure resulted in no recurrence for more than 4 years.
In other words, the administration of GcMAF eradicated tumors and left patients cancer-free for 4+ years with no additional treatment!
Both U.S. and UK governments desperately seizing all supply, shutting down clinics, even as millions die from cancer every decade…
GcMAF (Globulin component Macrophage Activating Factor), a blood product, claims to treat a range of conditions including cancer, HIV and autism…
More than 10,000 vials were seized at this site and production of this unlicensed medicine has now ceased. These products were sold through various European websites and UK patients may have bought it from one of these websites. We are working with colleagues in other countries to alert them to the potential risks. Our investigations are ongoing and we have received no reports to date of side effects caused by this product.
That same page lists some of the websites where GcMAF had been available for purchase:
GcMAF (Gc Protein derived Macrophage Activating Factor) – Gc MAF treatment is a highly effective macrophage activating therapy, used to stimulate the immune system and activate macrophages so that they can destroy cancer cells and other abnormal cells in the body.
What exactly is Second Generation GcMAF?
High Dose Second Generation Gc-MAF is produced using our new Patent Pending process which was developed here in Japan by Saisei Mirai in collaboration with Dr Hitoshi Hori and Dr Yoshihiro Uto at the University of Tokushima who have been studying GcMAF for over 20 years. Studies on GcMAF began at the University of Tokushima in 1992, after they were introduced to Dr Nobuto Yamamoto’s work and a collaboration began…
Second Generation GcMAF is made using a new and improved 2nd generation method of Gc-MAF production which is 10-20 times more concentrated and is more active and stable than other GcMAF that is currently available. Importantly, this much higher concentration GcMAF has been clinically demonstrated to be largely free of any side effects in the great majority of patients and is much more stable because it is resistant to oxidation.
That same site describes Oral GcMAF as follows: “Oral GcMAF is a form of GcMAF produced from bovine colostrum by Saisei Mirai which was developed in collaboration with Tokushima University.”
It also lists the following health conditions as being treatable with GcMAF, potentially a “universal cancer cure” substance:
Gc-MAF and/or oral Colostrum MAF macrophage activation therapy is indicated in the treatment of any diseases where there is immune dysfunction or where the immune system is compromised, such as:
Do you see yet why the medical establishment must SUPPRESS GcMAF and destroy all knowledge of its clinical applications? This one substance holds the potential to render numerous vaccines and pharmaceuticals utterly obsolete.
Researchers and practitioners have demonstrated that GcMAF can reverse diseases that attack the immune system such as: chronic inflammation, bacterial and viral infections, chronic herpes, chronic acne, Lyme disease, fibromyalgia osteoporosis, Hodgkin’s, Lupus, MS, Parkinson’s and remarkably – autism.
A clinical study out of Italy on 94 children with autism showed that 83 of them made considerable progress while on GcMAF. The most common reported improvements involve:
• Cognitive abilities including attention and focus, learning and understanding, receptiveness and awareness of the environment and both receptive and expressive language gains.
• Social Skills including willingness to interact and communicate with peers.
• Behavior including less hyperactivity, less stereotypical behaviors and improved cooperation and compliance.
In another study of 1500 children with autism, 85% had high levels of viruses and a compromised immune system. All 1500 received weekly GcMAF injections and 70% of the children responded to the treatment with reduced symptoms and another 15% made full recoveries. The other 15% did not respond.
It was stated that the reduction of autistic symptoms is permanent provided that GcMAF has been taken long enough for the body to produce its own GcMAF which typically takes 24 weeks.
The systematic suppression of medical science to protect the lucrative cancer treatment industry (chemotherapy, oncology, radiotherapy, etc.)
Back in 1993, Nobuto Yamamoto, then working at Temple University School of Medicine in Philadelphia, PA, USA, first described a remarkable molecule. His paper reported the conversion of vitamin D3 binding protein (DBP, known in humans as Gc) into a potent macrophage-activating factor (MAF), known as Gc-MAF. Macrophages are a key part of the human immune system with two roles: to engulf and destroy pathogens and cellular debris, and to recruit other immune cells to respond to the pathogen.
Gc-MAF hasn’t had the benefit of a single patent owner – as a natural molecule, it cannot be patented without being modified – with the will and resources to push it under the noses of the public and health authorities. Dr Yamamoto has run small human trials in breast, prostate and colorectal cancers, with promising results.
David Noakes might just be the person to bring Gc-MAF into the mainstream. He’s the CEO of Immuno Biotech Ltd. and spokesperson for First Immune Gc-MAF, a project he describes as, “PhD and BSc biochemists and biomedical scientists… with external doctors, oncologists and scientists who kindly provide advice, committed to bringing some of the increasing number of published but relatively unused medical cures to as many people as we can.” At the moment, Noakes and his colleagues are supplying Gc-MAF to 30 countries where it is legal, via a network of “around 300” doctors. Their Gc-MAF is made to extremely high standards, and is being used in ongoing clinical research by Noakes’ collaborators and others. Their ultimate goal is to, “Build the case that GcMAF is effective for various illnesses, which will help to make it available to the public”.
GcMAF suppliers fighting for survival against a global medical monopoly that profits from disease
The medical laws have been changed over the last 40 years so that all the brilliant breakthroughs in cancer are denied to the British public. Lord Maurice Saatchi had to watch his wife die, while his doctor told him the only thing he was allowed to prescribe her was chemotherapy, which would shorten her life. He hopes to bring the Medical Innovation Bill to Parliament, so instead of obeying a destructive government law, a doctor will be able to prescribe whatever treatment is best for the patient…
Bad law kills, and Britain has the worst medical laws in Europe. The 1939 Cancer Act makes it illegal to discuss the possibility cancer can be cured, which is partly why 160,000 people die unnecessarily of cancer in Britain every year. Science and treatments are decades ahead of where the medical industry is today. The MHRA’s job is to get life saving treatments like GcMAF out to people as quickly as possible. Instead they block them to protect billion dollar Big Pharma monopolies, who also fund the MHRA. Over a hundred thousand lives could be saved every year if the 1939 Cancer Act were repealed, and the MHRA were closed down.
There are 142 eminent scientists who have published GcMAF research papers on the US National Library of Medicine alone.
Your GcMAF empowers your body to cure itself. In a healthy person your own GcMAF has 11 actions discovered so far, including two on cells, three excellent effects on the brain, and 6 on cancer. Amongst these it acts as a “director” of your immune system. But viruses and malignant cells like cancer send out an enzyme called Nagalase that prevents production of your GcMAF: that stops its 11 beneficial effects, and neutralises your immune system. So diseases become chronic, and cancer cells grow unchecked.
Minutes after a receiving a dose, 10 of the body’s actions restart. In three weeks of two GcMAF 0.25ml doses a week, your immune system is rebuilt to above normal strength. You need two doses a week for typically 24 weeks for many diseases and early cancers, up to seven one ml doses a week and a year for stage 4 cancers. Your body then takes the disease down without side effects, and successfully in 80% of cases -depending upon how well you follow the protocol under “Treatment Protocol” on this website.
What is GcMAF?
It is a human protein. One week’s GcMAF looks like a small raindrop. If properly produced it is perfectly sterile, and a most ethical course for doctors.
GcMAF is therefore a replacement therapy for those who can’t make their own. Taking GcMAF replaces the missing part of the immune system, and also acts as the body’s own internal medicine.
GcMAF is extracted and isolated; its a 24 step process, and at the end it must have tests to prove its sterility and activity. (If it does not come with published tests, its probably not GcMAF.) One GcMAF has been tested in universities, laboratories and clinics, where, as a result of the testing, consistent activity and sterility have always been found, and been the subject of 40 scientific research papers.
What does GcMAF do?
The GcMAF Conference 2013 showed GcMAF is a far more powerful molecule than thought, both in terms of the science, and doctors’ results. In stage 4 cancer, some doctors who use the full protocol, listed on “Treatment Strategies,” are saving every patient (if they have not had chemotherapy.) Success can be achieved with all tumour cancers including breast, lung, prostate, pancreatic and melanoma.
GcMAF can eradicate chronic inflammation and viral infections. It is better than antibiotics in many areas, and 25% successful with Autism, 50% or more with Chronic Herpes, Chronic Acne, Chronic cirrhosis of the liver, Chronic kidney disease, Chronic depression, Colitis, Crohn’s, Fibromyalgia, Hepatitis, Herpes, LMBBS, ME/CFS, Osteoporosis, Periodontal disease, Psoriasis and various types of Immune dysfunction including allergies. Research shows GcMAF can halt deterioration in Parkinsons, multiple sclerosis (MS), dementia and ALS, and in its role of immune system regulator, can reverse diseases that attack the immune system like Lupus and Arthritis. And is effective with wound healing. Its successful with tumour cancers, and some others.
In addition to rebuilding a depressed immune system, GcMAF:
Inhibits angiogenesis – stops blood supply to tumours
Activates macrophages – phagocytosis and destruction of cancer cells
Apoptosis – suicide of cancer cells
Reverts the cancer cell phenotype to normal (Turns cancer cells into healthy cells)
Reduces the metastatic potential of human cancer cells in culture.
Increases energy production at the mitochondrial level – ME/CFS
Improves human neuronal metabolic activity through cAMP signaling – autism, ME/CFS, MS, ALS
Counters toxic effects including cadmium – ME/CFS
It abolishes neuropathic pain due to neuro-oxidative stress (stress due to the anti-cancer drug oxaliplatin) in the lab. (neurodegenerative diseases and autism that have oxidative stress as a pathogenetic mechanism)
It increases neuronal connectivity by promoting differentiation and the formation of dendrites and neuritis (autism and ME/CFS, where there is a lack of connectivity between neurons).
See the 31 research papers published, particularly Brescia, and the 60 published by others listed under “The science”.
80% of terminal stage four tumour cancers cases can be saved (40% if they’ve had chemo), but usually when they are closely monitored, which is why residential Treatment Centres are being run in Switzerland. If they have three months to live and have not had chemo, almost no one needs to be lost.
The 180 scientists who have published papers on trials of GcMAF selected those in the early stages of cancer and HIV, and reported nearly 100 percent success, with no recurrence after many years. They did not attempt trials on people with large tumours.
Our trials are quite different: many people are over 50, some over 80, with advanced or terminal cancers, with significant tumour mass. Most come to us when their doctors tell them they can do no more.
The life of GcMAF is only six days – you have to keep taking it until your disease has gone (ie your nagalase is under 0.65 nmol/min/mg) then a further 8 weeks, or the immune system gets shut down again.
How long should you take GcMAF for?
8 weeks for chronic herpes/acne, fibromyalgia, inflammation.
Allow 24 weeks plus of GcMAF for: Autism (85% improve, 25% eradication), CFS (70% eradication), HIV, Lyme (8% respond, most appear to have the VDR gene blocked and the viruses conceal themselves with biofilms) and stage 1 to 2 cancer, (80% respond).
Late stage cancer, if you follow “Treatment Protocol” again has 80% responders, but it takes a year to 18 months to become cancer free.
Cirrhosis of the liver: 16 months
Remember everyone responds differently. We can’t say how you will respond.
The more minor the disease, the easier it is for GcMAF to eradicate. GcMAF needs normal levels of vitamin D to function strongly (take 10,000iu a day). in low responders, larger doses are required.
We have probably proved GcMAF can work for people up to age 90, and can destroy large tumour mass. See “Participants experiences”.
If you have your blood taken for monocyte counts, relevant markers and vitamin D levels, and again for a nagalase test at the beginning, you should see on your next test after three weeks that your immune system is back to full strength, and after 8 weeks significantly falling nagalase will indicate the disease is losing its grip. Don’t stop the GcMAF until your nagalase gets below 0.65 nmol/min/mg, when it loses the ability to prevent your body producing your own GcMAF, and then you no longer need ours. Even better, get scans.
Autism children can improve at five weeks with substantial improvements at 8 weeks. See “Participants experiences.” But everyone is different.
The beauty of using your own immune system to attack disease or cancer is that it remembers how to defeat it for the rest of your life: it doesn’t come back. And unlike chemotherapy, the side effects are trivial.
The only way you can tell if GcMAF is genuine and active is to test with living cells in a laboratory. See “Quality and Tests of our GcMAF.” To recap:
We put live macrophages cells and MCF7 breast cancer cells together; nothing happens. Then we add GcMAF; in 72 hours the macrophages eat all the MCF7 cancer cells. We then put only GcMAF and MCF7 together, and the GcMAF turns the cancer cells back into healthy cells.
We have GcMAF available for preclinical trials. See “Buy GcMAF”.
You must read at least all of “Buy GcMAF” and “Treatment strategies” on the left if you want to take this further. And you must be prepared to give us feedback.
Cancerous cells and HIV-infected cells secrete -N-acetylgalactosaminidase into the blood stream, resulting in deglycosylation of serum Gc protein. This inactivates the MAF precursor activity of Gc protein, leading to immunosuppression… When peripheral blood monocytes/macrophages of 175 cancer patients bearing various types of cancer were treated in vitro with 100 pg GcMAF/ml, monocytes/macrophages (phagocytes) of all cancer patients were activated for phagocytic and superoxide generating capacity. This observation indicates that patient phagocytes are capable of being activated…
first heard about GcMAF almost a year ago. At the same time, I had first learned about “nagalase”, a blood test that is used to in part determine whether or not one might be a candidate for GcMAF therapy. Nagalase is an enzyme that prevents Vitamin D receptors (VDR) from being activated on the surface of the macrophage. As a result, macrophages are not “activated” and our immune systems are not able to properly respond to invaders.
Here are some points that I have learned thus far on GcMAF:
– GcMAF has reportedly been tested more for safety, purity, etc. than other human blood products.
– Macrophages are cultured, destroyed, and the GcMAF receptors are purified.
– Treatment is via injection 1x/week for 8-20 weeks. Dose is titrated initially to avoid exacerbation or Herx responses as much as possible.
– A commonly used dose is .25ml once weekly (a 2.2 ml vial should last 8 injections).
– The primary test used in looking at whether or not GcMAF may be a reasonable intervention is nagalase.
– Nagalase inactivates macrophages.
– I personally would NEVER consider this option without having a baseline nagalase test. Normal is < 0.95. Mine was 2.9.
The practitioner I worked with suggested that 2.9 was in the range of someone with HIV or cancer in terms of the impact on the immune system. I’d like to hear from others in the Lyme community as you get test results as well to see if there is a pattern of elevated nagalase in those with Lyme disease. Whether or not Lyme itself has anything to do with nagalase elevation is something I have not been able to find anything on. We certainly all have underlying viral co-factors that are likely in play as well, but I suspect that Borrelia may also play a role in nagalase elevation.
– In healthy college students, a nagalase 0.4 is not uncommon (the lower the better).
– At 2.9, my practitioner was surprised that I did not have more cognitive deficits such as memory loss and other cognitive issues.
– It has been suggested that ongoing antimicrobial therapy without a working immune system is like leaving the house with the door wide open inviting burglars in. By using GcMAF to activate macrophages, nagalase drops, and one may regain a functional immune system. The door is then closed to further invaders and we may no longer serve as a microbe hotel.
– Maintenance therapy should not be needed once the immune system is once again properly functioning.
– Activated macrophages only remain active for 7 days so any negative responses are generally short-lived. That said, some people do have strong inflammatory responses that are not believed to be typical die-off reactions.
– It has been indicated that in some cases, other medications may be needed in order to manage the inflammatory response. This is another reason that one needs to be working closely with a knowledgeable practitioner before considering GcMAF in my opinion. In the CFS and GcMAF world, this more severe form of a die-off reaction is called IRIS.
– VDR genetics do not seem to play a role in predicting response as earlier thought according to one practitioner that I have spoken with. That said, Vitamin D levels do correlate with the positive response rate of GcMAF. Thus, Vitamin D supplementation may be required in order to optimize outcome.
– Other than die-off reactions or activation of symptoms (inflammation), no other side effects are generally expected.
– Nagalase should be monitored every 1-2 months while on treatment to determine the required duration of the therapy. Target nagalase after treatment would be 0.4 to 0.6.
– Elevated nagalase has a profound detrimental effect on the immune system. Elevated nagalase is often presumed to be related to microbes of viral origin or cancer. Viruses that are nagalase producers open the door to chronic infections.
– Hemagglutinin contains nagalase and is also found in flagella of some bacteria so it could also be the case that some bacteria may produce nagalase.
– Parents with ASD children also often have elevated nagalase.
– A practitioner I spoke with likened Lyme disease to a “peat moss fire” burning below the surface. Activating macrophages should help to deal with the fire.
– GcMAF should be helpful in dealing with other infections that are not of viral origin; for example, Borrelia, Bartonella, and other infections commonly associated with Tick-Borne Infections (TBIs). GcMAF is active against many cancers and many different kinds of microbes.
– Neopterin is another test that is sometimes used as an indicator of immune suppression. As macrophages become activated, neopterin may rise and later fall. If one is in the normal range for neopterin and has an immune-related illness, this could be an indication that the immune system is suppressed and not responding appropriately.
– People with autoimmune conditions can generally use GcMAF. However, GcMAF may be contraindicated in people with Multiple Sclerosis.
– Reduction in nagalase is generally seen early in the course of treatment; within the first 3-6 weeks. In some studies, nagalase dropped by over 50% in less than six weeks.
– Cancer patients may initially feel as bad on GcMAF as they do on chemotherapy, but often feel much better after the first month.
– Anti-inflammatories may limited the effect of GcMAF.
– Enzymes and biofilm-reducing supplements may have a negative impact on GcMAF therapy and may be best avoided. It is still too early to know what the impact may be, but one practitioner I spoke with feels that it is best to avoid these.
– One should not be on any immune-suppressing agents while on GcMAF as the immune system must be partially functional in order to respond appropriately to the treatment.
– A common pattern is to see elevated lymphocytes, high nagalase, and low NK cells. Once nagalase drops, it may be the case that NK cell function could be positively impacted. CD57 is a type of NK cell. It is too early to know if this proves to be true, but it is one of the things I’m quite interested in.
Watch this video presentation on GcMAF therapy to learn more.
Researchers testing GcMAF stated it, “works 100% of the time to eradicate cancer completely, and cancer does not recur even years later.” (This was stated based on the tested group of patients -nothing works 100% for everyone) The weekly injection GcMAF, a harmless glyco-protein activates the human immune system which then can kill the growing cancer. Studies among breast cancer and colon cancer patients produced complete remissions lasting 4 and 7 years respectively. This glyco-protein ‘cure’ is totally without side effect but currently goes unused and completely ignored by cancer doctors. Why? Maybe it is because there is little money to be made in selling it. For less than $2000USD a cancer patient can obtain an adequate amount of GcMAC.
When human macrophages were treated in vitro with 100 pg GcMAF/ml for 3 hours and a prostate cancer cell line LNCaP was added with an effector/target ratio of 1.5, approximately 51% and 82% of LNCaP cells were killed by 4 and 18 hours of incubation, respectively [14,15]. This in vitro tumoricidal capacity of macrophages activated by GcMAF led us to investigate the therapeutic efficacy of GcMAF for prostate cancer. GcMAF therapy as a single remedy modality can eradicate metastatic breast and colorectal cancers most effectively…
Naturopathic Physician at the pH Miracle Ti Sana Medical Spa, Arlate, Italy
Is X-Ray Mammography Accelerating The Epidemic of Breast Cancer?
While a growing body of research now suggests that x-ray mammography is causing more harm than good in the millions of women who subject themselves to breast screenings, annually, without knowledge of their true health risks, the primary focus has been on the harms associated with over-diagnosis and over-treatment, and not the radiobiological dangers of the procedure itself.
In 2006, a paper published in the British Journal of Radiobiology, titled “Enhanced biological effectiveness of low energy X-rays and implications for the UK breast screening programme,” revealed the type of radiation used in x-ray-based breast screenings is much more carcinogenic than previously believed:
Recent radiobiological studies have provided compelling evidence that the low energy X-rays as used in mammography are approximately four times – butpossibly as much as six times – more effective in causing mutational damage than higher energy X-rays. Since current radiation risk estimates are based on the effects of high energy gamma radiation, this implies that the risks of radiation-induced breast cancers for mammography X-rays are underestimated by the same factor.[1]
In other words, the radiation risk model used to determine whether the benefit of breast screenings in asymptomatic women outweighs their harm, underestimates the risk of mammography-induced breast and related cancers by between 4-600%.
The authors continued:
Risk estimates for radiation-induced cancer – principally derived from the atomic bomb survivor study (ABSS) – are based on the effects of high energy gamma-rays and thus the implication is that the risks of radiation-induced breast cancer arising from mammography may be higher than that assumed based on standard risks estimates.
Sadly, even if one uses the outdated radiation risk model (which underestimates the harm done),* the weight of the scientific evidence (as determined by the work of The Cochrane Collaboration) actually shows that breast screenings are in all likelihood not doing any net good in those who undergo them.
In a 2009 Cochrane Database Systematic Review,** also known as the Gøtzsche and Nielsen’s Cochrane Review, titled “Screening for breast cancer with mammography,” the authors revealed the tenuous statistical justifications for mass breast screenings: http://www.greenmedinfo.com/article/x-ray-mammography-every-woman-whose-life-prolonged-10-womens-lives-will-be-shortened-ie
Screening led to 30% overdiagnosis and overtreatment, or an absolute risk increase of 0.5%. This means that for every 2000 women invited for screening throughout 10 years, one will have her life prolonged and 10 healthy women, who would not have been diagnosed if there had not been screening, will be treated unnecessarily. Furthermore, more than 200 women will experience important psychological distress for many months because of false positive findings. It is thus not clear whether screening does more good than harm.[2]
In this review, the basis for estimating unnecessary treatment was the 35% increased risk of surgery among women who underwent screenings. Many of the surgeries, in fact, were the result of women being diagnosed with ductal carcinoma in situ (DCIS), a “cancer” that would not exists as a clinically relevant entity were it not for the fact that it is detectable through x-ray mammography. DCIS, in the vast majority of cases, has no palpable lesion or symptoms, and some experts believe it should be completely reclassified as a non-cancerous condition.
A more recent study published in the British Medical Journal in 2011 titled, “Possible net harms of breast cancerscreening: updated modeling of Forrest report,” not only confirmed the Gøtzsche and Nielsen’s Cochrane Review findings, but found the situation likely worse:
This analysis supports the claim that the introduction of breast cancer screening might have caused net harm for up to 10 years after the start of screening.[3]
So, let’s assume that these reviews are correct, and at the very least, the screenings are not doing any good, and at worst, causing more harm than good. The salient question, however, is how much more harm than good? If we consider that, according to data from Journal of the National Cancer Institute (2011), a mammogram uses 4 mSv of radiation vs. the .02 mSv of your average chest x-ray (which is 200 times more radiation), and then, we factor in the 4-600% higher genotoxicity/carcinogenicity associated with the specific “low-energy” wavelengths used in mammography, it is highly possible that beyond the epidemic of over-diagnosis and over-treatment, mammograms are planting seeds of radiation-induced cancer within the breasts of millions of women.***
With the advent of non-ionizing radiation based medical diagnostic technologies, such as thermography and ultrasound, it has become vitally important that patients educate themselves about the alternatives to x-ray mammography that already exist. Until then, we must use our good sense – and research like this – to inform our decisions, and as far as the unintended adverse effects of radiation go, erring on the side of caution whenever possible.
Using 3-D Full Body Bio-Electro Scanning (FBBES), Full Body Thermography (FBT) and Full Body Ultrasound (FBU) to Determine the Best Possible Strategy for Preventing and/or Reversing a Cancerous Breast Condition [7]
In modern day oncology, surgeons biopsy the lymph nodes to determine how cancer is spreading or provide staging. Lymphocytes, a type of white blood cell that is found in these lymph nodes which are catch-basins for acidic waste and cancerous cells are responsible for breaking-down and removing cellular acidic waste and cancerous cells. Impaired lymphocytes and/or congested lymph nodes are at least one major factor in the many areas I test for functionality.
The lymphatic system, the lymph nodes and the lymphocytes themselves must be functional in preventing and reversing any cancerous condition.
Using electrodes attached to the head, hands and feet I am able to test the functionality of the lymphatic system, circulatory system, muscular system, skeletal system, endocrine system, neurological system, reproductive system, vascular system, digestive system, and respiratory system. interstitial chemistry, interstitial pH for metabolic acidosis and the electro-conductivity of the cells to determine the state of health of ALL organs, glands and tissues in the prevention and reversal of any cancerous condition.[7]
I also test clinically for nutritional deficiencies and metabolic alkalosis or acidosis by measuring the interstitial chemistry, interstitial pH and the electro-conductivity. Measuring the pH of the interstitial fluids is more revealing of a cancerous condition since the blood is always trying to maintain its delicate alkaline pH of 7.365 and will not vary much. Based upon my theory that cancer is a compromised acidic environment of the interstitial fluids which may negatively affect the state of health of ALL body cells which make up the organs, glands and tissues. It is significantly more important to measure interstitial and intracellular fluids than blood fluids in order to obtain a correct chemistry and pH when making nutritional recommendations in the prevention and treatment of a cancerous condition. [4, 5, 6,7,8,9]
The following are quantitative measurements in healthy patients, without cancer, comparing Blood fluids with Intracellular and Interstitial fluids of the body compartments as a benchmark which I use to determine deficiencies in alkalizing minerals, protein and whether or not the patient is in metabolic acidosis or a pre-cancerous or cancerous condition (Note: all cancer patients are in interstitial metabolic acidosis, low in interstitial sodium and high in interstitial calcium and potassium): [8,9]
As I correct the deficiencies in the intracellular and interstitial fluids targeted with key alkalizing nutritional treatments, patients see the difference through follow-up tests using quantitative non-invasive 3-D Full Body Bio-Electro scanning. They also feel the difference physiologically and functionally.[4,5,6,7]
This is how I know proper alkalizing nutritional support in any cancerous condition is important in the prevention and treatment of cancer, the metastasis of cancer and the shrinking of a cancerous cyst or mass without chemotherapy and/or radiation. The best part about these alkalizing nutritional treatments is they are helpful in most, if not in all cancerous conditions.[4, 5,6,7]
The following case study is with one of my patients who was diagnosed by biopsy with inflammatory ductal cell carcinoma who reversed her cancerous condition without chemotherapy, radiotherapy, and surgery.[7]
Using breast thermography and tumor location and size measured by breast ultrasound you can see the week by week thermography progress of a 14.2cm tumor in the left breast reduce to less than 2cm in 7 weeks of treatment using ANI protocol as outlined in this article and in Chapter 11 of the pH Miracle revised and updated book. (4,5,6,7)
The safest, painless, non-invasive, affordable full body screening tests are a combination of a Medical Diagnostic Ultrasound and Thermography, which may give the Physician about 95% accuracy in detecting breast cancer.[7]
Thermography is a physiological, non-invasive screening procedure that detects and records infrared heat emissions from the pre-cancerous or cancerous area, which can aid in the early detection of abnormal changes in body tissues, organs and glands. Thermography offers information that no other procedure can provide. The procedure is based on the principle that chemical and blood vessel activity in both pre-cancerous or cancerous tissue and the area surrounding a developing cancer is almost always higher in temperature than in the normal tissue.
Since pre-cancerous and cancerous masses are highly metabolic tissues, they need an abundant supply of nutrients to maintain their growth. The cells release substances that stimulate the formation of new blood vessels (neoangiogenesis). This process results in an increase in surface temperatures of the affected tissue, organ or gland.
The most promising aspect of medical diagnostic thermography is its ability to spot abnormalities years before the tumor is seen on any anatomical test. Since thermal imaging detects changes at the cellular level, this test can detect activity 8 to 10 years before any other test. This makes it unique in that it affords the physician the opportunity to view changes before the actual formation of the cancerous tumor.
Studies have shown that by the time a tumor has grown to sufficient size to be detectable by physical examination or mammography, it has in fact been growing for about seven years achieving more than 25 doublings of the malignant cell colony. At 90 days there are two cells, at one year there are 16 cells, and at five years there are 1,048,576 cells–an amount that is still undetectable by a mammogram. Thermography has the ability to provide the patient with future risk assessment. If discovered, certain thermographic risk markers can warn the patient that she/he needs to work closely with their physician with regular checkups to monitor her health. [7]
Summary
Full-body ultrasound is an anatomical non-invasive, painless screening test without ionized radiation. Ultrasound, also known as sonography, uses sound waves to outline a part of the body. For this test, a small instrument called a transducer is placed on the skin (which is often first lubricated with ultrasound gel) and emits sound waves off body tissues. The echoes are converted by a computer into an image that is displayed on a computer screen.
Ultrasound imaging is “real-time,” meaning that it can show exactly what’s happening in the tissue, organ or gland at that moment, help to distinguish between cysts (fluid-filled sacs) and solid masses, detect increased vascularity around or within the mass, see the shape, exact size and location of the mass, cyst, calcification or dilated mammary ducts.
These safe medical diagnostic tests can be done on early bases for a regular check up, or more often if the problem was detected, to monitor a noninvasive alkalizing nutritional treatment progress.
Early detection, which includes self examination and safe, painless, non-invasive medical diagnostic Full Body Bio-electro Scan(FBBES) Full Body Thermography (FBT) and Full Body Ultrasound (FBU) screenings with no ionizing radiation coupled with a supportive alkalizing nutritional diet and ANI whether or not the patient is receiving chemotherapy and/or radiation, I have found clinically that this approach in a precancerous or cancerous condition will saves lives![4,5,6,7]
If you have questions concerning any specific acidic cancerous condition or to learn more about ANI and a alkalizing nutritional dietary and lifestyle protocol in the prevention and reversal of any precancerous or cancerous condition, please read The pH Miracle revised and update, The pH Miracle for Cancer and Reverse Cancer NOW. [4,5,6] http://www.phoreveryoung.com You can also email: phmiraclelife@gmail.com
*This discrepancy in radiation risk models/estimates follows from two fundamental problems: 1) the older risk model was based on higher-energy radiation emissions, such as are given off from atomic bomb blasts 2) it was a crude model, developed before the discovery of DNA and a full understanding of radiotoxicity/genotoxicity.
** Keep in mind that the Cochrane Database Review is at the top of the “food chain” of truth, in the highly touted “evidence-based model” of conventional medicine. Cochrane Database Reviews are produced by The Cochrane Collaboration, which is internationally recognized as the benchmark for high quality, evidence-based information concerning the effectiveness (or lack thereof) of common health care interventions. The organization, comprised of over 28,000 dedicated people from over 100 countries, prides itself on being an “independent” source of information, and historically has not been afraid to point out the corrupting influence of industry, which increasingly co-opts the biomedical research and publishing fields.
***The low-energy wavelengths cause double strand breaks within the DNA of susceptible cells, which the cell can not repair. Through time these mutations result in “neoplastic transformation”; radiation has the ability to induce a cancerous phenotype within formerly healthy cells that has cancer stem cell-like (CSC) properties.
References:
[1] Enhanced biological effectiveness of low energy X-rays and implications for the UK breast screening programme. Br J Radiol. 2006 Mar ;79(939):195-200. PMID: 16498030
[2] Screening for breast cancer with mammography. Cochrane Database Syst Rev. 2009(4):CD001877. Epub 2009 Oct 7. PMID: 19821284
[3] Possible net harms of breast cancer screening: updated modelling of Forrest report. BMJ. 2011 ;343:d7627. Epub 2011 Dec 8. PMID: 22155336
[4] Robert O. Young, Ph.D., D.Sc., ND and Shelley Redford Young, LMT, “The pH Miracle revised and updated,”Hachett Publishing, Boston, USA, June, 2010.
[5] Robert O. Young, Ph.D., D.Sc., ND and Shelley Redford Young, LMT, “The pH Miracle for Cancer,” Hikari Omni Publishing, Valley Center, California, September, 2015.
[6] Robert O. Young, Ph.D., D.Sc., ND, Shelley Redford Young, LMT and Matt Traverso, “Reverse Cancer Now,” Hikari Omni Publishing, Valley Center, California, July, 2014.
[8] Reference Studies: Niels Fough-Anderson, Burton M, Attura, BElla T. Attura, Ole Siggard-Andersen, Clinical Chemistry, 41/10, 1522-1525, (1995)
[9] Gilariyi M., Bcriyi C., Fekete J, Ikreriyi K., Kovach AGB, “Ion Concentration in Subcutaneous Interstitial Fluid Measured versus Expected Values.” AMJ of Physiololgy 1988.
The Cure for Cancer? That’s an easy question to answer! The Cure for Cancer is Found in its Prevention NOT in its Treatment! – Dr. Robert O. Young
Do you know what rotten apples, grapefruit or bananas look like? If you do then you know what cancer cells look like. Cancer cells are nothing more that healthy cells that are spoiling because of a compromised environment! Look at the picture below and you will see colorized cancerous body cells rotting in their toxic acidic environment.
What compromises the internal environment of a human body that causes body cells to begin spoiling and rotting? The answer is simple! The body’s build-up of acidic metabolic and dietary waste that has not been properly eliminated through the four channels of elimination – urination, defecation, respiration and perspiration!
Naturopathic Physician at the pH Miracle Ti Sana Medical Spa, Arlate, Italy
Japanese women are the ones who are least susceptible to breast cancer. Allegedly, their slim line is the main reason behind this. But the real reason is their diet is rich in iodine!
According to the findings of a Norwegian study, individuals who consume three glass of milk daily, have a two times increased risk of breast cancer in comparison to the ones who consume half a cup, or less.
Unfortunately, the breast cancer is in expansion and we are all aware of it. If we take into consideration a whole range of factors, it turns out that milk could be responsible for the increased risk of this disease. Initially, it was thought that milk only accelerates the development of an already existent cancer, but the Norwegian scientists claim that apart from that, it is the main culprit for its occurrence in the first place.
United States, Sweden, Finland, Canada, and Britain are the countries in which most cases of breast cancer are identified. Interestingly, the consumption of milk takes top position particularly in these countries. On the other hand, breast cancer is an extremely rare occurrence or it doesn’t exist at all in countries where milk of animal origin is used. As mentioned in the very beginning, women who tend to drink three glasses of milk a day are more likely to develop a breast cancer in comparison to the ones drinking only half a cup.
Acidic waste products from glandular function called hormones and growth factors in milk are known to have carcinogenic content, and the same was proved for synthetic supplements of vitamin D, which milk is often enriched with. In general, patients consume two times more synthetic vitamin D than others.
However, these causes for the development of breast cancer are not the only ones. It was noted that the number of cases is growing with the increase of sugar, pastries, and some types of meat, such as pork. On the other hand, the consumption of fish and raw vegetables leads to significant decline.
Finally, there are the bras, especially the tight ones, which increase the risk as well. Women, who wear a bra at least 12 hours daily, increase the risk by more than 20% in comparison to the ones who use them moderately, rarely, or never. This especially refers to bras which contain plastic elements or metal.
Bitter melon appears to have therapeutic activity in the treatment of HIV infection. Click here to read the entire abstract
Pubmed Data : J Ethnopharmacol. 2009 May 23. PMID: 12346831 Article Published Date : May 23, 2009 Study Type : Human Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : HIV Infections : CK(609) : AC(186) Pharmacological Actions : Anti-HIV Agents : CK(108) : AC(60) Bitter melon contains compounds with anti-HIV activity. Click here to read the entire abstract
Pubmed Data : Thyroid. 2000 Aug;10(8):659-63. PMID: 7665070 Article Published Date : Aug 01, 2000 Study Type : Human Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : HIV Infections : CK(609) : AC(186) Pharmacological Actions : Antiviral Agents : CK(795) : AC(322) Bitter melon is superior to the drug rosiglitazone (Avandia) in type 2 diabetic patients. Click here to read the entire abstract
Pubmed Data : Phytomedicine. 2009 May;16(5):401-5. Epub 2009 Apr 10. PMID: 19362455 Article Published Date : May 01, 2009 Study Type : Human Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Diabetes Mellitus: Type 2 : CK(4292) : AC(359) Additional Keywords : Food as Medicine : CK(18) : AC(6), Hall of Fame : CK(35) : AC(5), Superiority of Natural Substances versus Drugs : CK(1197) : AC(214) Bitter gourd (Momordica charantia) stimulates beta cell regeneration in diabetic rats. Click here to read the entire abstract
Pubmed Data : Indian J Exp Biol. 2007 Dec;45(12):1055-62. PMID: 18254212 Article Published Date : Dec 01, 2007 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Diabetes Mellitus: Type 1 : CK(1217) : AC(235) Additional Keywords : Beta Cell Regeneration : CK(76) : AC(28) Bitter melon (M. charantia) may have a therapeutic role in conditions involving elevated ammonia levels. Click here to read the entire abstract
Pubmed Data : Evid Based Complement Alternat Med. 2010 Jan 4. Epub 2010 Jan 4. PMID: 20047891 Article Published Date : Jan 04, 2010 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Ammonia: Elevated : CK(89) : AC(17) Bitter melon (Momordica charantia) and Phyllanthus urinaria have hypoglyemic properties. Click here to read the entire abstract
Pubmed Data : Nippon Yakurigaku Zasshi. 1992 Nov;100(5):415-21. PMID: 1464400 Article Published Date : Nov 01, 1992 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18), Phyllanthus urinaria : CK(46) : AC(21) Diseases : Diabetes Mellitus: Type 1 : CK(1217) : AC(235) Pharmacological Actions : Hypoglycemic Agents : CK(870) : AC(162) Additional Keywords : Brazilian Traditional Medicine : CK(8) : AC(4) Bitter melon attenuates fructose-induced insulin resistance in fructose-fed rats. Click here to read the entire abstract
Pubmed Data : Antioxid Redox Signal. 2005 Nov-Dec;7(11-12):1612-20. PMID: 19429344 Article Published Date : Nov 01, 2005 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Adiponectin: Low Levels : CK(98) : AC(25), Fructose-Induced Toxicity : CK(140) : AC(52), Insulin Resistance : CK(1237) : AC(235) Additional Keywords : Plant Extracts : CK(5359) : AC(1705) Bitter melon demonstrates anti-tumor activity. Click here to read the entire abstract
Pubmed Data : Cancer Res. 1983 Nov;43(11):5151-5. PMID: 6616452 Article Published Date : Nov 01, 1983 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Tumors : CK(200) : AC(116) Bitter melon has a preventive efffect on abdominal and liver fat in high-fat-fed hamster. Click here to read the entire abstract
Pubmed Data : J Agric Food Chem. 2009 Jul 22;57(14):6461-7. PMID: 19601676 Article Published Date : Jul 22, 2009 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Abdominal Obesity (Midsection Fat) : CK(388) : AC(59), Fatty Liver : CK(540) : AC(127), Obesity : CK(2013) : AC(288) Additional Keywords : Plant Extracts : CK(5359) : AC(1705) Bitter melon has anti-ulcer properties. Click here to read the entire abstract
Pubmed Data : J Ethnopharmacol. 2009 Jun 25;123(3):464-9. Epub 2009 Mar 26. PMID: 19501279 Article Published Date : Jun 25, 2009 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Duodenal Ulcer : CK(89) : AC(26), Gastric Ulcer : CK(250) : AC(96) Pharmacological Actions : Anti-Ulcer Agents : CK(164) : AC(61) Additional Keywords : Plant Extracts : CK(5359) : AC(1705) Bitter melon has significant effect against insulin resistance and visceral obesity of mice on a high-fat diet. Click here to read the entire abstract
Pubmed Data : Diabetes Res Clin Pract. 2008 Aug;81(2):134-43. Epub 2008 Jun 11. PMID: 18550200 Article Published Date : Aug 01, 2008 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Abdominal Obesity (Midsection Fat) : CK(388) : AC(59), Diabetes Mellitus: Type 2 : CK(4292) : AC(359), Insulin Resistance : CK(1237) : AC(235) Bitter melon significantly reduces insulin resistance and suppresses visceral fat accumulation. Click here to read the entire abstract
Pubmed Data : Br J Nutr. 2008 Feb;99(2):230-9. Epub 2007 Jul 26. PMID: 17651527 Article Published Date : Feb 01, 2008 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Insulin Resistance : CK(1237) : AC(235), Metabolic Syndrome X : CK(675) : AC(124), Obesity : CK(2013) : AC(288), Overweight : CK(2347) : AC(362) Additional Keywords : Lipogenesis Inhibitor : CK(2) : AC(1) Extracts of jambul, bitter melon (M. charantia), gymnena and fenugreek (T. gracecum) have antidiabetic and drug sparing activities. Click here to read the entire abstract
Pubmed Data : Appl Biochem Biotechnol. 2009 Nov 11. Epub 2009 Nov 11. PMID: 19904502 Article Published Date : Nov 11, 2009 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18), Fenugreek : CK(102) : AC(31), Gymnema Sylvestre : CK(21) : AC(11), Jambul : CK(20) : AC(7) Diseases : Diabetes Mellitus: Type 2 : CK(4292) : AC(359) Additional Keywords : Drug: Glimepiride : CK(2) : AC(1), Drug Sparing : CK(451) : AC(50) Maitake and Bitter Melon (Momordica charantia) contain alpha-glucosidase inhibitors which may have therapeutic value in diabetes type 2. Click here to read the entire abstract
Pubmed Data : Biosci Biotechnol Biochem. 2002 Jul;66(7):1576-8. PMID: 12224646 Article Published Date : Jul 01, 2002 Study Type : Animal Study Additional Links Substances : Bitter Melon : CK(65) : AC(18), Maitake Mushroom : CK(51) : AC(33) Diseases : Diabetes Mellitus: Type 2 : CK(4292) : AC(359) Pharmacological Actions : Alpha-glucosidase inhibitor : CK(33) : AC(13), Enzyme Inhibitors : CK(417) : AC(225) Wild bitter melon leaf extract effectively inhibits P. acnes induced inflammatory responses and may be useful to relieve the inflammation of acne. Click here to read the entire abstract
Pubmed Data : Food Funct. 2015 Jun 22. Epub 2015 Jun 22. PMID: 26098998 Article Published Date : Jun 21, 2015 Study Type : Animal Study, In Vitro Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Acne : CK(240) : AC(39) Pharmacological Actions : Anti-Inflammatory Agents : CK(2488) : AC(682), Interleukin-8 downregulation : CK(111) : AC(29) Additional Keywords : Plant Extracts : CK(5359) : AC(1705) Bitter melon (M. charantia) compares favorably with insulin and rosiglitazone in potentiating glucose uptake. Click here to read the entire abstract
Pubmed Data : Ann N Y Acad Sci. 2006 Nov;1084:391-401. PMID: 19744549 Article Published Date : Nov 01, 2006 Study Type : In Vitro Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Diabetes Mellitus: Type 2 : CK(4292) : AC(359) Additional Keywords : Drug: Rosiglitazone : CK(17) : AC(5), Plant Extracts : CK(5359) : AC(1705), Superiority of Natural Substances versus Drugs : CK(1197) : AC(214) Bitter melon extract, NAC, grape seed extract and other natural antioxidants counteract drug-induced oxidative stress and resultant cell death. Click here to read the entire abstract
Pubmed Data : Biofactors. 2004;21(1-4):223-32. PMID: 15630201 Article Published Date : Jan 01, 2004 Study Type : Commentary Additional Links Substances : Antioxidant formulas : CK(487) : AC(73), Bitter Melon : CK(65) : AC(18), Grape Seed Extract : CK(171) : AC(58), NAC (N-acetyl-L-cysteine) : CK(275) : AC(66) Diseases : Oxidative Stress : CK(2855) : AC(750) Pharmacological Actions : Antioxidants : CK(5416) : AC(1618) Eleostearic acid, a fatty acid found within bitter melon seed oil, inhibits cancer cell proliferation and induce programmed cell death. Click here to read the entire abstract
Pubmed Data : Cancer Prev Res (Phila Pa). 2009 Oct;2(10):879-86. Epub 2009 Sep 29. PMID: 19789297 Article Published Date : Oct 01, 2009 Study Type : In Vitro Study Additional Links Substances : Bitter Melon : CK(65) : AC(18) Diseases : Breast Cancer : CK(2940) : AC(676) Pharmacological Actions : Antiproliferative : CK(1768) : AC(1208), Apoptotic : CK(2152) : AC(1476), Cell cycle arrest : CK(574) :
Naturopathic Physician at the pH Miracle Ti Sana Medical Spa, Arlate, Italy
Ginger is 10,000x More Effective Than Chemotherapy in the Treatment of Cancer!
A new study reveals ginger contains a pungent compound that could be up to 10,000 times more effective than conventional chemotherapy in targeting the cancer stem cells at the root of cancer malignancy.
The authors of the study further affirm these points:
“Cancer stem cells pose serious obstacle to cancer therapy as they can be responsible for poor prognosis and tumour relapse. To add into the misery, very few chemotherapeutic compounds show promise to kill these cells. Several researchers have shown that cancer stem cells are resistant to paclitaxel, doxorubicin, 5-fluorouracil, and platinum drugs [8, 16]. CSCs are thus an almost unreachable population in tumours for chemotherapy. Therefore any compound, that shows promise towards cancer stem cells, is a highly desirable step towards cancer treatment and should be followed up for further development.”
The researchers identified a variety of ways by which 6-shagoal targets breast cancer:
It reduces the expression of CD44/CD24 cancer stem cell surface markers in breast cancer spheroids (3-dimensional cultures of cells modeling stem cell like cancer)
It significantly affects the cell cycle, resulting in increased cancer cell death
It induces programmed cell death primarily through the induction of autophagy, with apoptosis a secondary inducer
It inhibits breast cancer spheroid formation by altering Notch signaling pathway through γ-secretase inhibition.
It exhibits cytotoxicity (cell killing properties) against monolayer (1-dimensional cancer model) and spheroid cells (3-dimensional cancer model)
It was in evaluating the last mode of 6-shagoal’s chemotherapeutic activity and comparing it to the activity of the conventional chemotherapeutic agent taxol that the researchers discovered an astounding difference. Whereas taxol exhibited clear cytotoxicity in the one-dimensional (flat) monolayer experimental model, it had virtually no effect on the spheroid model, which is a more “real world” model reflecting the 3-dimensionality of tumors and their stem cell subpopulations. Amazingly, this held true even when the concentration of taxol was increased by four orders of magnitude:
“In contrast [to 6-shagoal], taxol, even though was highly active in monolayer cells, did not show activity against the spheroids even at 10000 fold higher concentration compared to 6-shogoal.”
This is a highly significant finding, as it affirms a common theme in cancer research that acknowledges the primarily role of cancer stem cells: namely, while conventional techniques like surgery, radiation, and chemotherapy are effective at reducing a tumor’s size, sometimes to the point where it is “debulked,” burned,” or “poisoned” out of the body even below the threshold of re-detection, the appearance of “winning the battle” often comes at a steep price, as ultimately the cancer stem cell population regrows the tumors, now with increased vengeance and metastastic invasiveness, resulting in the cancer “winning the war.”
The monolayer model, which does not account for the complex immunity of actual cancer stem-cell based tumors against chemoagents like taxol, represents the old preclinical model of testing cancer treatments. The spheroid model, on the other hand, clearly shows that even 10,000 times higher concentrations of taxol are not capable of beating this ginger component at selectively targeting the root cause of the tumor malignancy.
In their concluding remarks, the authors point out a hugely important distinction between natural anti-cancer agents and conventional ones that have only been introduced in the past half century or so, namely, “Dietary compounds are welcome options for human diseases due to their time-tested acceptability by human bodies.”
Unlike modern synthetically produced and patented chemicals, ginger, curcumin, green tea, and hundreds of other compounds naturally found in the human diet, have been “time-tested” as acceptable to the human body in the largest and longest running “clinical trials” known: the tens of thousands of years of direct human experience, spanning thousands of different cultures from around the world, that constitute human prehistory. These experientially-based “trials” are validated not by RCTs, or a peer-reviewed publication process, but by the fact that we all made it through this incalculably vast span of time to be alive here today. Consider also that if our ancestors made the wrong dietary choice by simply mistaking an edible berry for a poisonous one, the consequences could be deadly. This places even greater emphasis on how the “time testing” of dietary compounds was not an academic but a life-death affair, and by implication, how the information contained within various cultural traditions as “recipes” passed down from generation to generation are “epigenetic inheritance systems” no less important to our health and optimal gene expression as the DNA in our own bodies.
Ultimately, this new study adds to a growing body of research indicating that cancer stem cell targeting approaches using natural substances present in the human diet for thousands of years are far superior than chemotherapy and radiation, both of which actually increase the relative populations of cancer stem cells versus non-tumorigenic ones.
Lets face it, Chemo does NOT work. According to a 2004 report by Morgan, Ward, and Barton: “The contribution of cytotoxic chemotherapy to 5-year survival in adult malignancies. … survival in adults was estimated to be 2.3% in Australia and 2.1% in the USA.”Jun 16, 2014
Consider a alkaline lifestyle and dietary change to treat this lifestyle and dietary dis-ease. Try an alkaline whole foods plant based lifestyle approach and diet to heal the core (the small and large intestines), rebuild the red blood cells and immune system, open the channels of elimination, and buffer and eliminate the acidic metabolic and dietary waste to prevent the primary cause of all cancerous conditions.
To learn more about a non-invasive and non-chemical alkaline approach to the prevention and reversal of a cancerous condition read: Reverse Cancer Now and The pH Miracle for Cancer – www.phoreveryoung.com
References and Evidence for the Efficacy of Ginger in Human Disease:
Naturopathic Physician at the pH Miracle Ti Sana Medical Spa in Arlate, Italy
In Latin, the word “acid” means poison or toxin and the word “inflammation” means “I ignite or set alight.” Like gasoline, when metabolic and/or dietary acids are collected and retained in the interstitial fluids or what I call the ‘third kidney’ and then collected in the connective tissues or the ‘acid catchers’ of the blood and interstitial fluids, the organs, tissues and glands will ‘light-up or ignite” with inflammation or pain.
The 7 Stages of Acidosis That Leads to Inflammation and Then Cancer
When metabolic acid, like lactic acid and/or a dietary acid, like uric acid are not properly eliminated through the four channel of elimination, namely, urination, defecation, perspiration or respiration, the body will store these acids in the connective tissues or what I call the ‘acid catchers,’ of the body, including the muscles, organs and glands. This is why an athlete will feel pain in the muscles, tendons or ligaments or someone who drinks alcohol excessively will feel pain on the right side of the body where the liver is located. I have described the retention of metabolic acids (from all body functions), dietary acids (from all food and drink), respiratory acids (from the oxygen process of the body) and environmental acids (from electro-magnetic fields, including cell-phones, air-dryers, or electric cars and chemical pollutions) and when not properly eliminated, these toxic liquids and gases will ‘ignite’ or ‘light-up’ the connective tissues causing the first stage of acidosis or the symptoms of enervation, fatigue or tiredness. If the elimination organs, including the lungs, bowels, kidney’s and skin become congested or blocked then acidic cellular waste cannot be properly eliminated and will build-up in the connective tissues causing the symptoms that conventional medicine calls sickness and disease. When the facts are known concerning the retention and build-up of acidic metabolic and/or dietary toxic waste, sickness and diseases are nothing more than the body’s inability to remove its own waste products leading to the one sickness and one disease – acidosis.
The one sickness and one disease or dis-ease is the over-acidification of the blood and then tissues due to an inverted way of living, eating and thinking. This one sickness and one disease or dis-ease has seven stages or seven expressions, which have been categorized by medical science as separate or different types of disease without any association or connection to the build-up of acidic waste in the body. But, there is NOT many diseases only one disease and one health! [1]
For example, inflammation is precursor stage to cancer and both are part of that one acidic disease. Cancer is an acidic condition that spoils healthy cells making them cancerous. Multiple sclerosis is part of that one disease as acid destroys the myelin sheath. Heart disease is the result of acid damage as is diabetes. Cystic fibrosis is also part of this one disease as healthy body cells are being protected from dietary and/or metabolic acids creating sticky mucous. Allergies, arthritis, osteopenia, osteoarthritis, osteoporosis, bowel restrictions and constipations, from diverticulitis to diverticulosis, IBS, ulcerated colitis, Crohn’s, all of these so-called diseases are the result of a compromised alkaline environment from individual acidic lifestyle and dietary choices.
The seven stages of disease or dis-ease or excess acidity begins in the bowels, then in the blood, pushed out into the tissues, organs and glands and expressed as follows:
1) The first stage of acidosis is enervation or the loss of energy. In this stage the body does not have the sufficient energy to completely remove dietary and/or metabolic acidic waste products which build up first in the blood and then in the connective and fatty tissues.
2) The second stage of acidosis are sensitivities and irritation. An example of stage two acidosis are sensitivities to food and/or air-born allergies.
3) The third stage of acidosis is catarrh or mucous buildup. An example of stage three acidosis would be the acidic condition of the lungs called cystic fibrosis. It is important to understand that mucous is created when the glands of the body release the alkaline compound sodium bicarbonate for the purpose of binding up dietary and/or metabolic acids. The combining of sodium bicarbonate to acid creates a sticky mucous. Since dietary, environmental and metabolic acids can breakdown and destroy healthy tissues and organs the glands of the body, such as the salivary glands, the pylorus glands, the pancreas and even the stomach release the alkalizing compound, sodium bicarbonate to protect and preserve healthy body cells that make up our tissues, organs and glands.
4) The fourth stage of acidosis is inflammation. There is only one cause of inflammation and that is acid. Acid equals pain and pain equals acid. There is no other cause. Any pain or inflammation in the body is the result of localized acid that has not been properly removed by the lymphatic system. That is why exercise is so important because the lymphatic circulation is activated by the contraction of muscle and especially the calf muscles. Therefore, inflammation is always caused by dietary, respiratory, environmental and/or metabolic acid.
5) The fifth stage of acidosis is induration or fibrotic tissue or the hardening of the tissues or organs. This is the classic symptomatology of cystic fibrosis. The tissues and organs are turning into leather. Another classic symptomatology of induration is atherosclerosis or the hardening of the vascular system.
6) The sixth stage of acidosis is the ulceration of tissues and/or organs such as in ulcerated colitis, or cirrhosis of the liver, or any lesion where ever it may appear.
7) And, the seventh and final stage of acidosis prior to death is the degeneration of tissues, organs and glands. All degenerative conditions are caused by dietary, respiratory, environmental and/or metabolic acids, such as in the symptomologies of osteoporosis, multiple sclerosis, ALL cancerous conditions, heart disease and all respiratory dis-eases are preceded by inflammation, induration and ulceration and all caused by the retention of acids..
It is important to keep in mind that whatever the disease or dis-ease condition there is only one cause. And, that one cause is the retention of excess acid first in the blood and then the tissues and organs. When excess acid is not eliminated through the four channels of elimination they are then deposited into the connective and fatty tissues. This is why I call the connective tissues the “acid catchers” of the blood.
You do not need a doctor to tell you your stage of acid imbalance. You can know this based upon your the symptom(s) you are experiencing or feeling. If you are overweight this is an acidic condition and the body protecting the organs that sustain life from excess dietary, environmental and/or metabolic acids. In other words, obesity is NOT a fat problem any more then cancer is a genetic problem. They are both an acid problem.
All stages of acidosis or sickness and disease are manifested in the tissues, organs or glands when cellular waste is NOT properly eliminated through the four channel of elimination – urination, defecation, respiration and perspiration. This is why I have stated that there is only one sickness and one disease and that one sickness and one disease is the over-acidification of the blood and THEN tissues due to an inverted way of living, eating and thinking. And if there is only one sickness, one disease and one cause THEN there must be only one treatment. The one treatment would be to prevent or reverse the retention and build-up of acidic waste in the interstitial fluids and the connective tissues or the ‘acid catchers by opening the channels of elimination, improving system circulation and micro-circulation, reducing acid loads on the body through an alkaline lifestyle and diet as outlined in The pH Miracle books, and finally hyper-perfusing the interstitial fluids with alkaline minerals such as sodium and potassium bicarbonate. [2]
The Microenvironment
A microenvironment of interstitial and/or intracellular metabolic acidosis can cause chronic stage four inflammation and increase the risk of a cancerous condition, bolstering chemotherapy resistance and turn on oncogenes, genes that can turn healthy functioning body cells into unhealthy, non-funcitonal cancerous cells. [3]
Most importantly, interstitial acidity causes inflammation throughout the entire body promoting the cellular breakdown and biological transformation or mutation of healthy cells into cancerous cells, systemically. What I am suggesting is the acids that cause inflammation and cancer run throughout the body and affect every body cell negatively in a lesser or greater degree.
Acidity that leads to inflammation causes the increase of blood flow in response to cellular degeneration in the buffering and/or elimination of acids and the repair, removal and/or replacement of inflamed and/or cancerous cells. If interstitial acidity continues to buildup causing uncontrolled degeneration of the affected cells the body will form tumors or encapsulation of these damaged cells and their associated acidic waste products in order to protect healthy surrounding cells that make up tissues, organs and glands. In addition, the formation of a tumor serves as the process for healing and regeneration of the acid injured tissue, much like clotted blood forms in the creation of the scab for regeneration after injury. The tumor inside the body is similar to the scab you see outside the body. Both serve to protect and to cause the regeneration of the specific tissue injured. Another example of this would be in the fracture of a bone. The blood flows to the injured bone at the specific area of the fracture. The blood because the clot and the clot differentiates into new bones. That is why when there is physical trauma to any tissue there is a risk of a tumor or clot forming such as in a head-bang injury or when someone is exposing their brain to radiation from a cell phone and a clot or tumor is formed in and around the affected area.
Powerful Insights to Cancer Treatment
Unfortunately, interstitial and intracellular acidity in the cause of inflammation and cancer is completely ignored for ALL inflammatory conditions and cancerous conditions in the oncology world. Basically, dietary and metabolic acids that cause inflammation and cancer are one of the leading factors that contributes to uncontrolled injury to healthy body cells creating more unhealthy cancerous body cells and spreading or what I call the ‘dominos effect’ (metastasis) where one cancerous cell spoils s healthy cell much like one rotten apple in a bushel of healthy apples will cause all apples to spoil, rot or become cancerous.
In this article I will explain helpful approaches to give acidic cancerous patients an edge in the prevention and treatment for overall cancer planning.
Uncovering, preventing or treating the cause of inflammation, rather than just treating the symptom, is an important key when reversing a cancerous or chronic dis-ease condition. To get to the root of the inflammation, we have to learn what causes inflammation and how to deal with it. Let’s get started.
What Causes Inflammation?
Inflammation is the result of damage to the tissues, organs and/or glands, caused by metabolic acids, respiratory acids, dietary acids, environmental acids, physical injury, ischemic injury (caused by an insufficient supply of blood to an organ), exposure to electrical/magnetic and air pollution or other types of trauma. The body’s response to the symptom of inflammation causes cellular transformations and immune responses that result in repair of the damaged tissue and cellular proliferation (biological transformations or spoiling) at the site of the injured tissue.
Inflammation can become chronic if the cause of the inflammation persists or certain control mechanisms in charge of shutting down the process fail. When these inflammatory responses become chronic, cell mutation and proliferation can result, often creating an environment that is conducive to the development of cancer. The so-called “perfect storm” is an extreme challenge that cancer patients face.
This is true for the onset of cancer, but also even more important for advancement of the disease. The cancer a patient begins with becomes very different in the later stages, becoming more mutated and complex to treat. Various signaling pathways are key contributors to creating epigenetic changes on the outside of the cell, switching on these internal mutations or biological transformations into bacteria, fungi and mold. Therefore, treating the inflammatory cause is always important. And what is that cause? It is a four letter word – ACID!
The Link Between Acids, Inflammation and Cancer
Despite popular belief, less than five percent of all cancerous conditions is solely genetic (in the sense of being directly inherited by family members). Most, if not ALL cancerous conditions have an acidic cause and those causes bring about chronic inflammation as part of the process. New research suggests an emerging link between acids, epigenetics and cancer. Changes catalyzed by pathogenic acid caused inflammation can biologically transform cells into cancerous cells and the formation of protective cancerous tumors. According to ScienceDirect.com, “Several types of inflammation—differing by cause, mechanism, outcome, and intensity—can promote cancer development and progression.” [4] A study by the Cancer Research Institute also agrees, saying, “Chronic inflammation plays a multifaceted role in carcinogenesis.” [5]
Researchers at the Ohio State University Comprehensive Cancer Center — Arthur G. James Cancer Hospital and Richard J. Solove Research Institute (OSUCCC — James) found that inflammation stimulates a rise in levels of a molecule called microRNA-155 (miR-155).
This, in turn, causes a drop in levels of proteins involved in DNA repair, resulting in a higher rate of spontaneous gene mutations, which can lead to cancer.
“Our study shows that miR-155 is upregulated by inflammatory stimuli and that overexpression of miR-155 increases the spontaneous mutation rate, which can contribute to tumorigenesis,” says first author and post-doctoral researcher Dr. Esmerina Tili. “People have suspected for some time that inflammation plays an important role in cancer, and our study presents a molecular mechanism that explains how it happens.”[3]
MicroRNAs form a large family of non-coding genes involved in many important cell processes. They carry out this function by suppressing the amount of particular proteins in cells, with each type of microRNA often affecting many different proteins.
MiR-155 is known to influence blood-cell maturation, immune responses and autoimmune disorders, and high levels of this molecule have been directly linked to the development of leukemias, and breast, lung and gastric cancers.
For this study, Tili and her colleagues examined the effects of inflammation-promoting substances such as tumor necrosis factor or lipopolysaccharide (found in the outer walls of bacteria) on miR-155 expression and on the frequency of spontaneous mutations in several breast-cancer cell lines.
When the researchers exposed breast-cancer cells to the two inflammatory factors the levels of miR-155 rose abnormally high, and the mutation rate increased two- to three-fold. To understand why, the investigators focused on the WEE1 gene, which stops the process of cell division to allow damaged DNA to be repaired.
The investigators learned that miR-155 also targets WEE1 and showed that high levels of miR-155 lead to low levels of WEE1. They reasoned that low levels of WEE1 allowed cell division to continue even when DNA damage is present, leading to a growing number of biological transformations or mutations.[6]
Many cancerous conditions are linked to once healthy cells that are damaged by dietary, metabolic, respiratory and/or environmental acid which causes these cells to biologically transform into bacteria, yeast or fungi and mold with their acidic waste products of exotoxins and mycotoxins causing reversible, epigenetic changes in other healthy body cells. At minimum, 20 percent or more of ALL cancerous conditions are linked to acid generated biological transformations of body cells and their associated extotoxins and mycotoxins, according to the Journal of American Medical Associates.
Some Examples of Acid Caused Cancers
Human Papillomavirus leads to cervical cancer.
Hepatitis C leads to liver cancer.
Epstein Barr leads to lymphoma.
Herpes Virus Six leads to brain cancer.
Helicobacter Pylori leads to stomach cancer.
HIV leads to Karposi’s sarcoma.
In my own research, I have found that the build-up of metabolic, dietary, respiratory and environmental acids in the connective tissues will lead to infection and the cause of ALL inflammatory and cancerous conditions. In other words, acid equals inflammation and inflammation equals acid. Another way to say the same thing is acid is pain and pain is acid. You cannot separate the acid from the pain or inflammation. They always co-exsist!
Today, conventional oncology recognizes only around 13% of all infections are reported to cause inflammation that leads to a cancerous condition. These acidic caused infections bring about cellular changes or breakdowns leading to even more acidity resulting in increased inflammation or acid that causes a cancerous condition.
In addition, chronic acidic inflammation held in the connective tissues produces increased body heat that can be seen and measured using full-body, non-invasive, non-radioactive medical thermography. A special infrared camera is used to view and measure the temperature of thehot spots of the body indicating localized tissue acidosis. The hottest areas of the body will show red to white.
The following thermography pictures show the heat coming off the left breast of a patient with medically diagnosed ductal cell carcinoma. You will notice in the left breast a large red to white area indicating a clot or tumor mass that initially measured in excess of 14cm. The patient followed a lifestyle and dietary protocol called the pH Miracle and within eight weeks the noted tumor mass and inflammation was eliminated and the tumor reduced to less than 2cm. The tumor was then surgically removed and tested and found to be non-cancerous.[7]
It is important to note that abnormally high body heat, caused by localized tissue acidity can lead to thermogenesis or hyperthermia showing metabolic acidosis or what I call ‘latent tissue acidosis’. Thermography is the only scientific technology that can visualize tissue inflammation and those specific acidic areas of the body that may be showing inflammation and at risk for becoming cancerous. When an area of tissue acidosis is viewed using thermography another non-invasive, non-radioactive medical diagnostic tool called ultrasound can be used to determine anatomically if there is a pathological clot or tumor mass, the size of the tumor mass and whether or not the tumor mass has angiogenic blood supply.
Other Changes in the Microenvironment
Acidity or inflammation is known to cause other such changes in the microenvironment of body cells. Body cells often undergo adaptive changes to survive stressful or acidic environments. These adaptive changes can include: an increased expression of sodium bicarbonate, increased antioxidant levels, increased anaerobic respiration and metabolism-; and development of angiogenic factors. This adaptation is usually transient, however, and allows normal body cells to survive only until the acidic condition that is causing the inflammation or cancerous condition is alleviated.
This means it’s not enough to have a strategy to eradicate cancerous body cells or to kill cancerous body cells with chemotherapy or radiation. Chronic acidic inflammation or ‘latent tissue decompensated acidosis’ needs to be buffered and eliminated through the four channels of elimination – urination, defecation, perspiration and respiratory in order to prevent any systemic acid build-up in the connective and/or fatty tissues leading to a cancerous condition.
Gene Pathways That Fuel Cancer Spread When Inflamed
According to a study by the Division of Hepatology and Gene Therapy at the CIMA-Universidad de Navarra in Spain, “Epidemiological studies have established that many tumors occur in association with persistent inflammation. One clear example of inflammation-related cancer is hepatocellular carcinoma (HCC). HCC slowly unfolds on a background of chronic inflammation triggered by exposure to infectious or agents (hepatotropic viruses), toxic compounds (ethanol/alcohol), or metabolic impairment.” [8]
Besides these acid causing inflammatory mediators, mounting evidence points to the deregulation of specific growth and survival-related pathways in HCC development. Among them is the pathway governed by the epidermal growth factor receptor (EGFR), which can be bound and activated by a broad family of ligands. Of special relevance is the fact that the EGFR engages in extensive crosstalk with other signaling pathways, serving as a “signaling hub” for an increasing list of growth factors, cytokines, and inflammatory mediators. In this review, I have summarize the most recent evidences supporting a role for the EGFR system in inflammation-related cell signaling, with special emphasis in liver inflammation and HCC. The molecular dissection of the pathways connecting the inflammatory reaction and neoplasia will facilitate the development of novel and more effective anti-tumor strategies.
Oncogenes can push for the greater expression of EGFR because tumors have genes just like healthy body cells. Why? Because tumors are the collection of unhealthy body cells which are now rotten or cancerous cells with their associated acidic waste products encapsulated in a web of cross-linked fibrin-monomers.
One of the most highly expressed receptors is called Epidermal Growth Factor Receptor (EGFR), which is normally used to tell cells to grow. It is found in all cancerous body cells. However, EGFR over-expression has been linked to numerous cancers, such as lung, prostate, anal and many others. The greater the expression of EGFR means faster growth and enhanced spreading via the ‘domino effect’ where one cell spoils or rots another cell just like one falling domino will topple another standing domino. The bottom line is, acid causing inflammation is the cause of it..
This increased expression is also associated with increased chemotherapy resistance, leading to tumors that are not well-formed and have blood supply. When you combine chronic acidic caused inflammation, these misused signaling pathways and overgrowth, you get a cancerous condition that is “immortalized.” Treating a metastatic cancerous condition is hard enough, but when it’s of this magnitude it can be extremely difficult, if not impossible, especially when ignoring the chronic acidic inflammation or ‘latent tissue acidosis’ at the unhealthy or spoiling cancerous cells’ metabolic core.
There is no singular legend drug that can currently treat any of these pathways. However, there are some integrative and complimentary approaches that, when used properly, can impact these inflamed targets from a multi-dimensional approach. This is how the integrative non-invasive, non-chemical, non-radioactive and non-surgical methods can be used to provide much needed support for buffering and eliminating metabolic, dietary, respiratory and environmental acids that are the cause of ALL cancerous conditions.
There are many different approaches to get at the root of a cancerous condition, rather than just a “one size fits all” protocol that offers little to no assistance to removing the acidic cause of inflammation that leads to all cancerous conditions.
How does inflammation occur?
There is only one cause. The one cause and only cause of inflammation that leads to a cancerous condition is the over-acidification of the blood and then tissues due to an inverted way of living, eating and thinking. It is that simple. Prevent and eliminate the acidic waste products of diet, metabolism, breathing and the environment through the four channels of elimination and you will prevent or reverse ALL potential and/or present cancerous conditions.
According to News-Medical.net, “[NF-Kappa-B] spends most of its life in the cell’s cytoplasm, quietly awaiting protective orders for healthy body cells.” But when extracellular and interstitial signals – of dietary and/or metabolic acids build-up in and around cells, for example – this sets off cellular protective alarms, the cell unchains its warhorse of antioxidants, such as glutathione, bicarbonates and hydroxyl ions, allowing it to protect itself from the acids that cause inflammation and cell death. [9]
These are only a few of the many signaling and inflammation pathways that can be targeted in an alternative and/or complimentary treatment. When the cause of inflammation is understood, it makes it that much easier to prevent and/or reverse a cancerous condition from building-up and negatively affecting the health of the tissues, glands and organs and outgrowing the window-of-opportuniy for a non-invasive, non-radioactive, non-chemical and non-surgical alkalizing lifestyle and dietary treatment. Helping enhance the effectiveness of any cancerous treatment with a holistic and/or integrated approach involves strong comprehensive anti-inflammatory or ant-acid and cancer-signaling treatments.
Summary
Buffering and eliminating metabolic, dietary, respiration and environmental acids that cause inflammation is only one part of a complete treatment plan – there are many other aspects to consider, including hyper-perfusing the tissues with alkalizing compounds such as sodium and potassium bicarbonate, alkalizing nutrition with vitamins and herbs, alkalizing colon hydrotherapy, alkalizing exercises, alkalizing food, healing the core and restoring its alkaline design, supporting the immune system, buliding healthy stem cells in the crypts of the small intestines with chlorophyl and hemp oil, targeting antioxidants, alkalizing exercise, infrared sauna and much more. However, if you can reduce interstitial acidosis or ‘latent tissue acidosis’ you will reduce inflammation and reduce the risk of a cancerous condition which makes it much easier to maintain and hopefully, overcome. Otherwise, if you keep building-up and holding onto the acids that cause inflammation, a cancerous condition can outgrow any holistic alkalizing treatment. It becomes a race-to-the-finish to slow down the build-up of dietary and metabolic acids in the interstitial and intracellular fluids that systemically flow through the body fluids touching every body cell and potentially causing more healthy cells to become unhealthy acidic cancerous cells. If this scenario is not stopped there is no-way to stop acids from causing all tissues, organs and gland to start spoiling and becoming cancerous. This is what conventional oncology calls metastasis. But it is NOT metastasis! It is systemic interstitial decompensated acidosis that leads to inflammation, tissue acidosis and an acidic condition conventional medicine calls ‘cancer.’
The best part about this alkalizing approach to the treatment of any inflammatory and cancerous condition is they are helpful for most, if not all cancers. If you have any questions about your inflammatory and/or cancerous condition, or would like to know more about how integrative and/or complimentary medicine might help YOU, please contact us today at: phmiracleliving@aol.com or universalmedicalimaging.com
[2] Young, R.O., Young, S.R., The pH Miracle, revised and updated, Hachett Publishing, June, 2010.
[3] E. Tili, J.-J. Michaille, D. Wernicke, H. Alder, S. Costinean, S. Volinia, C. M. Croce. Mutator activity induced by microRNA-155 (miR-155) links inflammation and cancer. Proceedings of the National Academy of Sciences, 2011; 108 (12): 4908 DOI: 10.1073/pnas.1101795108
The Cure for Cancer? That’s an easy question to answer! The Cure for Cancer is Found in its Prevention NOT in its Treatment! – Dr. Robert O. Young
Do you know what rotten apples, grapefruit or bananas look like? If you do then you know what cancer cells look like. Cancer cells are nothing more that healthy cells that are spoiling because of a compromised environment! Look at the picture below and you will see colorized cancerous body cells rotting in their toxic acidic environment.
What compromises the internal environment of a human body that causes body cells to begin spoiling and rotting? The answer is simple! The body’s build-up of acidic metabolic and dietary waste that has not been properly eliminated through the four channels of elimination – urination, defecation, respiration and perspiration!
Current research has determined that the key to breast cancer survival rests upon its earliest possible detection. If it’s discovered in its earliest stages, 95% cure rates are possible.
A breast self-examination involves checking the breasts to help detect breast problems or changes. Many breast problems are first discovered by women themselves, often by accident. A breast self-examination involves checking the breasts for lumps or changes while standing and lying in different positions and while looking at the breasts in a mirror to note any changes in their appearance. Once a woman knows what her breasts normally look and feel like, any new lump or change in appearance should be evaluated by a doctor. Breast lumps can be noncancerous (benign) or cancerous (malignant).
In its early stages, breast cancer usually has no symptoms. As a tumor develops, you may note the following signs:
• A lump in the breast or underarm that persists after the menstrual cycle. This is often the first apparent symptom of breast cancer. Lumps associated with breast cancer are usually painless, although some may cause a noticeable sensation. Lumps are usually visible on a diagnostic medical ultrasound long before they can be visually seen or felt.
• Swelling in the armpit.
• Redness, pain or tenderness in the breast. Although lumps are usually painless, pain or tenderness can be a sign of breast cancer.
• A noticeable flattening or indentation on the breast, which may indicate a tumor that cannot be seen or felt.
• Any change in the size, contour, texture, or temperature of the breast. A reddish, pitted surface like the skin of an orange could be a sign of advanced breast cancer.
• A change in the nipple, such as a nipple retraction, dimpling, itching, a burning sensation, or ulceration.
• Unusual discharge from the nipple that may be clear, bloody or another color. It’s usually caused by benign conditions but could be due to cancer in some cases.
• A marble-like area under the skin.
• An area that is distinctly different from any other area on either breast.
If you think you have any signs or symptoms that might mean breast cancer, be sure to see your doctor as soon as possible. Your doctor will ask you questions about your symptoms, any other health problems, and possible risk factors for benign breast conditions or breast cancer.
Your breasts will be thoroughly examined for any lumps or suspicious areas and tested for the feel of their texture, size, and relationship to the skin and chest muscles. Any changes in the nipples or clavicles may be palpated, because enlargement or firmness of these lymph nodes might indicate the spread of breast cancer. Your doctor will also do a complete physical exam to evaluate your general health and whether there is any evidence of cancer that may have spread.
If breast symptoms and/or the results of your physical exam suggest breast cancer might be present, more tests will probably be done. These might include different imaging tests. The safest, painless, non-invasive, affordable breast screening tests are a combination of a Medical Diagnostic Ultrasound and Thermography, which may give us about 95% accuracy in detecting breast cancer.
Breast Thermography is a physiological, non-invasive screening procedure that detects and records infrared heat emissions from the breast, which can aid in the early detection of abnormal changes in breast tissue. Breast Thermography offers women information that no other procedure can provide. The procedure is based on the principle that chemical and blood vessel activity in both pre-cancerous tissue and the area surrounding a developing breast cancer is almost always higher than in the normal breast.
Since pre-cancerous and cancerous masses are highly metabolic tissues, they need an abundant supply of nutrients to maintain their growth. The cells release substances that stimulate the formation of new blood vessels (neoangiogenesis). This process results in an increase in surface temperatures of the breast.
The most promising aspect of medical diagnostic thermography is its ability to spot abnormalities years before the tumor is seen on any anatomical test. Since thermal imaging detects changes at the cellular level, this test can detect activity 8 to 10 years before any other test. This makes it unique in that it affords us the opportunity to view changes before the actual formation of the tumor.
Studies have shown that by the time a tumor has grown to sufficient size to be detectable by physical examination or mammography, it has in fact been growing for about seven years achieving more than 25 doublings of the malignant cell colony. At 90 days there are two cells, at one year there are 16 cells, and at five years there are 1,048,576 cells–an amount that is still undetectable by a mammogram. Thermography has the ability to provide women with future risk assessment. If discovered, certain thermographic risk markers can warn a woman that she needs to work closely with her doctor with regular checkups to monitor her breast health.
Breast Ultrasound is an anatomical non-invasive, painless screening test without ionized radiation. Ultrasound, also known as sonography, uses sound waves to outline a part of the body. For this test, a small instrument called a transducer is placed on the skin (which is often first lubricated with ultrasound gel) and emits sound waves off body tissues. The echoes are converted by a computer into an image that is displayed on a computer screen. Ultrasound imaging is “real-time,” meaning that it can show exactly what’s happening in the breast at that moment, help to distinguish between cysts (fluid-filled sacs) and solid masses, detect increased vascularity around or within the mass, see the shape, exact size and location of the mass, cyst, calcification or dilated mammary ducts.
These safe diagnostic tests can be done on early bases for a regular check up, or more often if the problem was detected, to monitor a treatment progress.
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Please remember — early detection, which includes self examination and safe, painless, non-invasive medical diagnostic Ultrasound and Thermography screenings with NO radiation Saves Lives!
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“Health Education Instead of Medication and Radiation” ~ Dr. Galina Migalko
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If you would like to do a Full Body Thermography and Ultrasound at the same appointment time, please visit:
Los Angeles, CA Location http://www.universalmedicalimaging.com/
San Diego, CA Location http://www.phmiracleliving.com/t-MedicalImaging.aspx
Como, Italy Location http://www.phmiracleliving.com/p-593-ph-miracle-health-retreat-como-italy-event.aspx
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•Flexibility in patient scheduling, same day appointment available.
•No referral from a primary care physician is needed.
•Cost effective.
•Final 8 Doctors reports will be sent by email (Preliminary reports immediately).
•Dedicated and experienced licensed professionals.
•The highest quality of care.
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[Figure 1: Cloves of garlic]
{Figure 2: Cyenne pepper]
[Figure 3: Milk Thistle]
[Figure 4: Turmeric root and spice]
[Figure 5: Bloodroot plant and flower]
[Figure 6: Feverfew plant and flower]
[Figure 7: Wheatgrass}
[Figure 8: Ruscus Aculeatus or Butchers Broom]
[Figure 9: Sheep’s Sorrel}
[Figure 10: Astragalus}
[Figure 11: Ginger root]
[Figure 12: Research Shows The Efficacy of Ginger Root as a non-toxic form of chemotherapy]
[Figure 11: Cannabis plant with buds]
The Cure for Cancer? That’s an easy question to answer! The Cure for Cancer is Found in its Prevention NOT in its Treatment! – 
Monday, July 27, 2015
The following is a list of Scientific Research for Bitter Melon 
Breast Thermography is a physiological, non-invasive screening procedure that detects and records infrared heat emissions from the breast, which can aid in the early detection of abnormal changes in breast tissue. Breast Thermography offers women information that no other procedure can provide. The procedure is based on the principle that chemical and blood vessel activity in both pre-cancerous tissue and the area surrounding a developing breast cancer is almost always higher than in the normal breast.
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pHorever Young Blog 
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