Tag Archives: Measles

Has the Existence of Polio, Measles, HIV, CMV, EBV, Hep C, Ebola, the Flu, and Now Zika Viruses Been Demonstrated and Scientifically Proven?

Dismantling The Viral Theory

[Electron micrograph of the so-called Polio virus that has never been demonstrated scientifically to cause the symptoms of paralysis.  Illustration has been colorized for effect]

The first isolation of a virus was achieved in 1892 by Russian bacteria hunter Dimitri Iwanowski, who gathered fluid from diseased tobacco plants. He passed this liquid through a filter fine enough to retain bacteria; yet to Iwanowski’s surprise, the bacteria-free filtrate easily made healthy plants sick. In 1898 a Dutch botanist, Martinus Willem Beijerinck, repeating the experiment, also recognized that there was an invisible cause and named the infectious agent “tobacco mosaic virus.” In the same year as Beijerinck’s report, two German scientists purified a liquid containing filterable viruses that caused foot-and-mouth disease in cattle (viruses were at one time called “filterable viruses,” but eventually the term “filterable” came to apply only to viruses, and was dropped). Walter Reed followed in 1901 with a filtrate responsible for yellow fever, and soon dozens of other disease-causing viruses were found.

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In 1935 another American, Wendell M. Stanley, went back to the beginning and created pure crystals of tobacco mosaic virus from a filtered liquid solution. He affirmed that these crystals could easily infect plants, and concluded that a virus was not a living organism, since it could be crystallized like salt and yet remained infectious. Subsequently, bacteriologists all over the world began filtering for viruses, and a new area of biology was born-virology.

Historically, medical science has vacillated on the question of whether a virus is alive. Originally it was described as nonliving, but is currently said to be an extremely complex molecule or an extremely simple microorganism, and is usually referred to as a parasite having a cycle of life. (The term “killed” is applied to certain viral vaccines, thus implying an official conviction that viruses live.) Commonly composed of either DNA or RNA cores with protein coverings, and having no inherent reproductive ability, viruses depend upon the host for replication. They must utilize the nucleic acids of living cells they infect to reproduce their proteins (i.e., trick the host into producing them), which are then assembled into new viruses like cars on an assembly line. Theoretically, this is their only means of surviving and infecting new cells or hosts.

The Replicating Virus Theory

Then it was discovered that, when bacteria slowly begin to die, bacteria create tiny, apparently lifeless forms of survival, the so-called spores. It was then suspected that these spores were toxic and that they were the so-called pathogenic poisons. This was then refuted, since the spores are rapidly developing into bacteria when their vital resources are being restored. When scientists in the laboratory observed that the weak, highly inbred bacteria perished very quickly while turning into much smaller structures than the spores, it was first believed that the bacteria were being killed by the alleged pathogenic poisons, called viruses, and that the viruses were thereby replicating.

[The micrograph above was done using Dark Field Microscopy showing red blood cells and the evolution of bacterial pHages and bacterial spores (the white spots0 from red blood cell biological transformation]

The Replicating Virus Theory

Then it was discovered that, when bacteria slowly begin to die, bacteria create tiny, apparently lifeless forms of survival, the so-called spores. It was then suspected that these spores were toxic and that they were the so-called pathogenic poisons. This was then refuted, since the spores are rapidly developing into bacteria when their vital resources are being restored. When scientists in the laboratory observed that the weak, highly inbred bacteria perished very quickly while turning into much smaller structures than the spores, it was first believed that the bacteria were being killed by the alleged pathogenic poisons, called viruses, and that the viruses were thereby replicating.

The Invention of Bacterial Viruses

Due to the belief that these – at the time of their discovery still invisible- structures were killing the bacteria, they were called phages/bacteriophages, “eaters of bacteria”. Only later it was determined that merely highly inbred and therefore almost non-viable bacteria can be made to turn into phages, or bacteria which are being destroyed so fast that they do not have time to form spores.

The introduction of the electron microscopy led to the discovery of the structures resulting from the biological transformation or pleomorphism of bacteria when these were suddenly dying or when the metabolism of the highly inbred germs was overwhelmed by processes triggered by the adding of “phages”. It was also discovered that there are hundreds of types of different-looking “phages”. The discovery of phages, the so-called bacterial “viruses”, reinforced the wrong assumption and the belief that there were human and animal viruses that looked the same and had the same structure. This is not and cannot be the case, for several different reasons.

After introducing chemical examination techniques in biology, it was discovered that there are thousands of types of phages and that phages of one type always have the same structure. They consist of a particular molecule, made of nucleic acid, which is covered in a shell of proteins of a given number and composition. It was only later discovered that merely the bacteria which had been highly inbred in the test tube could turn into phages themselves, by contact with phages, but this never applied to natural bacteria or bacteria which had just been isolated from their natural environment. In this process, it was discovered that these “bacterial viruses” actually serve to provide other bacteria with important molecules and proteins, and that the bacteria themselves emerged from such structures.

Before it could be established that the “bacterial viruses” cannot kill natural bacteria, but they are instead helping them to live and that bacteria themselves emerge from such structures, these “phages” were already used as models for the alleged human and animal viruses. It was assumed that the human and animal viruses looked like the “phages”, were allegedly killing cells and thereby causing diseases, while at the same time producing new disease poisons and in this way transmitting the diseases. To date, many new or apparently new diseases have been attributed to viruses if their origin is unknown or not acknowledged. This reflex found an apparent confirmation in the discovery of the “bacterial viruses”.

It is important to note that the theories of fight and infection were accepted and highly praised by a majority of the specialists only if and when the countries or regions where they lived were also suffering from war and adversity. In times of peace, other concepts dominated the world of science.[272]

It is very important to note that the theory of infection – starting from Germany – has only been globalized through the third Reich, when the Jewish researchers, most of which had opposed and refuted the politically exploited theories of infection, were removed from their positions.[273]

The Detection of Phages and Biological Transformation

The existence of phages can be proved rapidly

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[Bacterial pHage being born out of a blood and/or body cell.  A biological process known as pleomorphism]

First step: their presence is confirmed through an effect, namely the transformation of bacteria into phages, and also through an electron micrograph of those phages. The control experiments show that phages do not appear if bacteria do not change or if bacteria randomly start decomposing due to extrinsic sudden annihilation, without forming phages.

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[The micrographs (micrographs #1 through #6] above show the cellular transformation of red blood cells, using pHase contrast microscopy, into rod bacteria, cell-wall deficient bacteria, Y-form yeast and then bacteria pHages]

Second step: the liquid containing the phages is concentrated and applied on another liquid, which has a high concentration at the bottom of the test tube and a low concentration at the top of the test tube. The test tube with the phages is then powerfully spun (centrifuged) and all the particles gather according to their mass and weight to the place of their own density. The density is the ratio of weight (mass) per unit of volume, expressed as Kg/l or g/mg, respectively. That is why this concentration and purification step for particles with the same density is called density gradient centrifugation.

The layer where many particles of the same density gather becomes “cloudy”, which is called a “band.” This step is being documented, then the particles concentrated, purified and sedimented in a “band” are removed with a syringe needle. The extracted concentrated amount of particles is called an isolate. A fast and simple electron micrograph will confirm the presence of phages in the isolate, which at the same time is an indication for the purity of the isolate, if the micrograph shows no other particles but the phages. The appearance and the diameter of the phages will also be established with the help of this micrograph.

The control experiment performed for this step consists in treating and centrifuging the liquid from bacteria which did not form any phages, where no phages appear at the end of the procedure.

After the step of successfully isolating the phages, the decisive biochemical characterization of the phages follows. The biochemical characterization of their composition is essential for identifying the specific type of phage, since different types of phages often appear to be similar. The isolate obtained through the density gradient centrifugation is now divided in two parts. One part is used to determine the size, type and composition of the nucleic acid; in a separate procedure, the other part is used to determine the amount, size and morphology of the proteins of the phages. Since the 1970s, these tests have been simple standard techniques that are learned by every biology student in their first semesters.

These tests represent the biochemical characterization of the phages. In almost every case, these results have been and are being published in only one publication, since a phage has a very simple structure which is very easy to analyze. The control experiments for these tests use liquid from bacteria which do not form phages and thus cannot present any biochemical proof. The existence of approximately two thousand different types of phages have been scientifically demonstrated this way

The So-Called Pathogenic Viruses

The “bacteriophages,” correctly defined as incomplete mini spores and building blocks of the bacteria, have been scientifically isolated, while the so-called pathogenic viruses have never been observed in humans or animals or in their body fluids and have never been isolated and subsequently biochemically analyzed. To date, none of the researchers involved in virology research seems to have realized this very important point.

The use of electron microscopy and the biochemistry were very slowly returning to normal after 1945 and no one had realized that not one pathogenic virus had ever been isolated in humans or animals; thus, as of 1949 researchers started applying the same idea used for the (bacterio) phages, in order to replicate the human and animal “viruses.” John Franklin Enders, born in 1897 in the family of a rich financier, was active in various fraternities after having finished his studies, then he worked as a real estate agent and studied foreign languages for four years before turning to bacterial virology, which fascinated him. He then simply transferred the ideas and concepts that he learned in this area of research to the supposed pathogenic viruses in humans.

UnScientific Experiments and Interpretations Gave Birth to Virology

With his unscientific experiments and interpretations that he had never confirmed through negative controls, Enders brought the entire “viral” infectious medicine to a dead end. It is important to note at this point that Enders, like many infectious diseases specialists, worked for the U.S. military, which had always been and remains to date a huge victim of the fear of contagions. It was mainly the U.S. military which spread its erroneous belief that besides chemical weapons there were also biological weapons in the form of bacteria and viruses.

In 1949, Enders announced that he had managed to cultivate and grow the alleged polio virus in vitro on various tissues. The American expert opinion believed everything immediately. What Enders did was to add fluids from patients with poliomyelitis to tissue cultures which he claimed to have had sterilized, then he alleged that the cells were dying because of the virus, that the virus was replicating in this way and that a vaccine could be harvested from the respective culture. At that time, summer polio epidemics (polio = flaccid paralysis) were very frequent during summer and they were believed to be caused by the polio virus. A vaccine was to help eradicate the alleged virus. After the polio vaccine was introduced, the symptoms were then re-diagnosed among other things as multiple sclerosis, flaccid acute paralysis, aseptic meningitis etc. and later polio was claimed to have been eradicated. During his experiments, Enders et al. sterilized the tissue cultures in order to exclude the possibility of bacteria killing the cells. What he didn’t take into consideration was that the sterilization and the treatment of the cell culture when preparing it for the alleged infection was exactly what was destroying and killing the cells. Instead, he interpreted the cytopathic effects as the existence and the action of a so-called polio virus, without ever having isolated a single virus and describing its biochemistry. The necessary negative control experiments, which would have shown that the sterilization and the treatment of the cells prior to the “infection” in the test tube was killing the cells, have never been performed. However, for this “performance” Enders received the Nobel prize in 1954.

The Invention of the Polio Virus and ‘YES” the Measles Virus Too!

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[Measles virus or a bacterial pHage?]

1954 is also the year in which Enders applied and introduced the same technique in order to allegedly replicate the measles virus. As he had been awarded the Nobel prize for the alleged polio virus the same year, all researchers believed his technique to be scientifically valid. Thus, to date, the entire concept of polio and measles has been based upon this unscientific technique and fraud.

Thus, the polio and measles vaccines do not contain viruses, but particles of dead monkey kidney tissue or human cancerous body cells. To date, no negative control experiments have been done with respect to the so-called polio and measles viruses either, which would have shown that it was the laboratory procedures that lead to the cytopathic effects on the cells.

Additionally, all claims and experiments made by Enders et al. and subsequent researchers lead to the only objective conclusion, that in fact they were observing and analyzing the cellular particles or fragments and the activity thereof in the test tube, misinterpreting these as particles and characteristics of the alleged polio and/or measles viruses.

ALL Viruses from HIV, EBV, CMV, Hepatitis C, West Nile Virus, Ebola, Measles, Zika, etc., are ALL Phantom Viruses

Viral Existence Has NEVER Been Scientifically Demonstrated and Never Proven!

The following explanations applies to all the so-called (human or animal) “pathogenic viruses”. The six papers provided by Dr. Bardens in the course of the “measles trial” as proof for the existence of the measles virus described in a didactically ideal way the various steps of the chain of misinterpretations up to the belief in the existence of a measles virus.

The first paper was published in 1954 by Enders et al.: “Propagation in tissue cultures of cytopathogenic agents from patients with measles” (Proc Soc Exp Biol Med. 1954 Jun; 86 (2): 277–286).

This publication can be found on the internet, like all the other publications presented at the measles trial. In that experiment, Enders et al. cut down dramatically on the nutrient solution and added cell-destroying antibiotics to the cell culture before introducing the allegedly infected fluid. The subsequent dying of the cells was then misinterpreted as presence and also isolation of the measles virus. No control experiments were performed to exclude the possibility that it was the deprivation of nutrients as well as the antibiotics which led to the cytopathic effects.

Enders’ and his colleagues’ blindness can be explained by the fact that he truly wanted to help people, while the ‘virus hysteria’ was intensifying after the war and during the cold war. It can also be explained by the fact that Enders and many of his colleagues had no idea about medicine or biochemistry and they were competing with the Soviet Union for the development of the first measles vaccine. Such a pressure for success can also explain why Enders and his colleagues ignored their own reservations and cautions expressed in 1954, when they had observed and noted that many cells also died after being treated normally (i.e. without being “infected”), which they thought to have been caused by unknown viruses and other factors.  All these facts and cautions were subsequently disregarded.

The second paper presented by the claimant in the ‘measles trial’ was published in 1959[274] and, for the reasons presented above, the authors concluded that the technique introduced by Enders was not appropriate for the isolation of ANY virus. This rebuttal is not only NOT being discussed by ALL the other researchers, but it is being ignored completely!

The ‘Viral Dogma’ of Pathogenic Viruses is Still Being Promoted Today!

In a third paper[275], the authors photographed typical cellular particles inside the cells and misinterpreted these as measles virus. They did not isolate any virus. For unexplained reasons, they failed to determine and describe the biochemical structure of what they were presenting as a virus in a separate experiment. In the short description of the methods used, one can read that the authors did not apply the standard isolation technique for viruses, i.e. the density gradient centrifugation. They simply centrifuged fragments of dead cells at the bottom of a test tube and then, without describing their biochemical structure, they misinterpreted the cellular debris as viruses.

From the way the experiments were performed, one can only conclude that cellular particles were misinterpreted as viruses. We find the same situation in the fourth[276] and the sixth[277] publication put forward by the claimant as proof of the existence of a measles virus. The fifth publication[278] is a review describing the consensus process as to which nucleic acid molecules from the dead cells would represent the so-called genome of the polio or measles virus. The result is that dozens of research teams work with short pieces of cell-specific molecules, after which -following a given model – they put all the pieces together on paper. However, this jigsaw puzzle made of so many pieces was never scientifically proven to exist as a whole and was never isolated from a virus, for a polio, measles, HIV or Hepatitis C, Ebola or Zika viruses have never been seen, neither in humans nor in a test tube. Referring to this publication, the court-appointed expert stated that it described the gold standard, i.e. the entire virus genome. It is obvious that the expert did not read this paper, whose authors stated that the exact molecular composition and functions of the measles virus genome will have to be the object of further research, which is why they had to rely on other virus models in order to achieve a consensus on the structure and functions of ANY virus genome. The easiest thing for anyone to notice is that in all of these publications, as well as in all other publications on the “measles virus” and other pathogenic viruses, including HIV, EBV, CMV, Ebola and Zika, no control experiments have ever been performed. No researchers used the density gradient centrifugation technique; instead, they only centrifuged cellular debris at the bottom of a test tube. This technique, used to collect all the particles from a fluid, is called pelletizing. From a logical and scientific perspective, it can be said that in all publications on the so-called “pathogenic viruses”, the researchers demonstrated in fact only particles and characteristics of cells. I would also like to point out that the so-called giant viruses[279] , i.e. an enwrapped nucleic acid can be found everywhere in the sea and in basic organisms. Like all bacterial phages, not only are they harmless, but they have beneficial functions. They can be also isolated by using the density gradient centrifugation, which proves their existence (see the graphic above).

I also recommend Prof. Lüdtke’s relevant review (1999).[280] He noted that at the early beginnings of virology, the majority of virologists always concluded that the structures they had mistaken for viruses turned out to be components of the cells and thus, they were only the result of the experiment and not the cause of the changes observed.

After the discovery and characterization of the phages and after introducing the dogma that the nucleic acid was the genome of all cells and viruses, the consensus was born, according to which such viruses must exist in humans and animals as well. In 1992, the dogma stating that the nucleic acid is the genotype of all cells was retracted in the scientific community. The ‘viral dogma’ of pathogenic viruses, however, is still being promoted today to the harm of billions of people. – for what?

The Bottom Line Concerning Phantom Viruses and the Polio and Measles Virus

[An Electron micrograph of the so-called Polio virus that has never been demonstrated scientifically to cause the symptoms of paralysis.  Illustration has been colorized for effect]

My bottom line still holds the truth that the terrain or internal environment is everything and the germ or so-called virus is NOTHING! The germ or so-called virus can only be a symptom of cellular breakdown due to an imbalance of the delicate alkaline pH balance of the body fluids and NOT the cause of that breakdown. That is why years ago I offered any scientist in the World a finders fee of 5 million US dollars if they could prove the existence of the HIV virus using Koch’s postulates. It has now been over 20 years and I am still waiting even though currently I no longer have the funds to pay the prize due to political assassination! It is unfortunate that a former 5 million US dollar prize offered 20 years ago was not enough money to change the current medical viral dogma that is currently paying out trillions of dollars to guess who?[281]

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Click here to read more: http://medcraveonline.com/IJVV/IJVV-02-00032.php

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Lecture in Dubai – The 2nd Annual Conference on Bacterial, Viral and Infectious Diseases

http://www.drrobertyoung.com/events.html

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Join Robert O Young PhD and Galina Migalko MD in Dubai on June 18th and 19th, 2019 for the Annual Conference on Bacterial, Viral and Infectious Diseases. They will be Key Note Speakers and doing a workshop on the New Biology.

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For more information and to register go to: https://bacterialdiseases.infectiousconferences.com/organiz…

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The following is the abstract for Dr. Young’s lecture:

The Dismantling of the Viral Theory

Robert O Young CPT, MSc, DSc, PhD, Naturopathic Practitioner

Abstract

There is now over 100 years of documented history and research on the Polio virus and whether or not its treatment by inoculation has been successful in eradicating Polio. I am suggesting in this article and in my lecture that there are significant findings based on historical and past and current research, including my own that the viral theory of Polio and possibly other modern-day diseases, such as Post-Polio Syndrome, Polio Vaccine-Induced Paralysis, Legionnaires, CNS disease, Cancer, HIV/AIDS and now Zika may be caused by acidic chemical poisoning from DDT (dichloro-diphenyl-trichloroethane) and other related DDT pesticides, acidic vaccinations, and other factors including lifestyle and dietary factors rather than from a lone infectious virus. I will present ten historical graphs outlining the history of Polio, the production of DDT, BHC, Lead, Arsenic, Polio vaccinations and the author’s theory that chemical poisoning, vaccination, and lifestyle and dietary choices are a more likely causes for the symptoms of Polio, neurological diseases, Cancer, HIV/AIDS and now Zika.

https://www.linkedin.com/…/lecture-dubai-annual-conference…/
https://bacterialdiseases.infectiousconferences.com/organiz…

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[2] Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991, p769

[3] Toxicological Profile: for DDT, DDE, and DDE. Agency for Toxic Substances and Disease Registry, September 2002.

[4] U. Beck, E. Löser “Chlorinated Benzenes and other Nucleus-Chlorinated Aromatic Hydrocarbons” Ullmann’s Encyclopedia of Industrial Chemistry, 2012, Wiley-VCH, Weinheim.

[5] Chlorobenzene”. Immediately Dangerous to Life and Health. National Institute for Occupational Safety and Health (NIOSH)

[6] U.S. Vital Statistics, U.S. Government Printing Office, Washington, D.C.

[7] Historical Statistics of the U.S., The U.S. Government Printing Office, Washington, D.C.

[8] Van Nostrand’s Encyclopedia of Science and Engineering (1995), vol. 5, p1725. The phrase “Pesticides As A Panacea: 1942-1962” is a subtitle found in Encyclopedia Britannica, Macropaedia (1986).

[9] Thomas, Robert E. (1955), Salt & Water, Power & People: A Short History of Hooker Electrochemical Co. Niagara Falls, NY: Hooker Chemical Co.

[10] Booth, Gerald (2000), “Ullmann’s Encyclopedia of Industrial Chemistry – Nitro Compounds, Aromatic”. doi:10.1002/14356007.a17_411. ISBN 3527306730

[11] Weber, Manfred; Weber, Markus; Kleine-Boymann, Michael (2004). “Ullmann’s Encyclopedia of Industrial Chemistry – Phenol”. doi:10.1002/14356007.a19_299.pub2. ISBN 3527306730.

[12] Haller, H. L., Bartlett, P. D., Drake, N. L., and others: The Chemical Composition of Technical DDT, American Chemical Society, Journal, volume 67, pages 1591- 1602, 1945.

[13] Jo-Yu Chin, Christopher Godwin, Chunrong Jia, Thomas Robins, Toby Lewis, Edith Parker, Paul Max, and Stuart Batterman, “Concentrations and Risks of p-Dichlorobenzene in Indoor and Outdoor Air,” Indoor Air, 2013 Feb; 23(1): 40–49, Published online 2012 Jul 18. doi: 10.1111/j.1600-0668.2012.00796.x.

[14] Duesberg, PH, “Inventing the AIDS Virus,” Regnery, (1996). ISBN 0-89526-399-8. [15] Icon Group International (Author), Chlorobenzene: Webster’s Timeline History, 1851 – 2007 May 17, 2010

[16] Ibid [17] Ibid

[18] Risse, GB (1988). Fee E, Fox DM, eds. Epidemics and History: Ecological Perspectives. in AIDS: The Burden of History. University of California Press, Berkeley. ISBN 0-520-06396-1.

[19] A Disease of Cleanliness: Polio in New York City, 1900-1990, in David Rosner, ed., Hives of Sickness: Public Health and Epidemics in New York City Rutgers University Press, 1995, pp. 115-130.

[20] McDonough, F., The Origins of the First and Second World Wars (Cambridge Perspectives in History), Cambridge University Press, August 28, 1997.

[21] Goel, A, Aggarwal, P, “Pesticide Poisoning,” Natl Med J India. 2007 Jul-Aug; 20(4):182-91.

[22] Ibid.

[23] Biskind, MS (1953) “Public Health Aspects of the New Insecticides,” American Journal of Digestive Diseases 20: 331-341.

[24] TIME Magazine, U.S. Edition, March 14, 1994 Vol. 143 No. 11. [25] Baily, J. W.: J. Am. Vet. M. A. 113: 251, Sept. 1948.

[26] Biden-Steele, K. and Stuckey, R. E.: “Poisoning by DDT Emulsion: Report of a Fatal Case”, Lancet, 2: 235-236, Aug. 17, 1946.

[27] Biskind, M. S.: “DDT Poisoning and X Disease in Cattle”, J. Am. Vet. M. A. 114: 20, Jan. 1949.

[28] Biskind, M. S.: “DDT Poisoning a Serious Public Health Hazard”, Am. J. Dig. Dis. 16: 73, Feb. 1949.

[29] Biskind, M. S.: “DDT Poisoning and the Elusive ‘Virus X’: A New Cause for Gastro- Enteritis”, Am. J. Dig. Dis. 16: 79, March 1949.

[30] Boyd, C. L.: “A Report on “XX Disease in Texas”, J. Am. Vet. M. A. 113: 463, Nov. 1948.

[31] Cameron, C. R., and Burgess, F.: “The Toxicity of DDT”, Brit. M. J. 1: 865-871, June 23, 1945.

[32] Carte; R. H., Hubanks, P. E., et al: “Effect of Cooking on the DDT Content of Beef”, Science, 107: 347, April 2, 1948.

[33] Case, R. A. M.: Toxic Effects of DDT in Man”, Brit. M. J., 2: 842-845, Dec. 15, 1945.

[34] Council on Pharmacy and Chemistry, A. M. A.: “Health Hazards of Pesticides”, J. A. M. A. 137: 1603, Aug. 28, 1948.

[35] Crescitelli, F., and Gillman, A.: “Electrical Manifestations of Cerebellum and Cerebral Cortex Following DDT Administration to Cats and Monkeys”, Am. J. Physiol., 147: 127- 137, Sept. 1946.

[36] Deederer, C.: “DDT Toxicity”, M.Rec. 161: 216-220, April 1948

[37] Domenici, T. J.: “Hepatitis without Jaundice and without Hepatomegaly”, N. Eng. J. Med. 240: 88, Jan. 20, 1949

[38] Dunn, J. E., Dunn, J. C., and Smith, R. S.: “Skin Sensitising Properties of DDT for 31

Guinea Pig”, Pub. Health Rep. 61: 1614-1620, 1949.

[39] Editorial: Pesticides: “Chemical Contaminants of Foods”, J.A.M.A. 137: 1604, Aug. 28, 1948.

[40] Fitzhugh, O. G., and Nelson, A. A.: “The Chronic Oral Toxicity of DDT”, J. Pharm.acol. and Exper. Therap. 89: 18-30, Jan. 1947.

[41] Gamier, G.: “Treatment of Scabies with DDT”, .Presse Med. 56: 458, June 23, 1948. [42] Garett, ii. M., “Toxicity of DDT for Man”, Alabama St. M. A. J., 17: 74, Aug. 1947.

[43] Globus, J. H.: “DDT Poisoning; Histopathologic Observations on the Central Nervous System in So-Treated Monkeys, Dogs, Cats and Rats”, J. Neuropath. 7: 418-431, Oct. 1948.

[44] Haymaker, W., Ginzler, A. M., and Ferguson, J. L.: “Toxic Effects of Prolonged Ingestion of DDT on Dogs, with Special Reference to Lesions in Brain”, Am. J. M. Sc. 212: 423, Oct. 1946.

[45] Hill, K. R., and Daniiani, C. R.: “Death Following Exposure to DDT, Report of a Case”, New Eng. J. Med., 235: 897-899, Dec. 19, 1946.

[46] Hill, K. 3. and Robinson, G.: “A Fatal Case of DDT Poisoning in a Child, with an Account of Two Accidental Deaths in Dogs”. Brit. M. J. 2: 845-847, Dee. 15, 1945.

[47] Ingle, L.: “Toxicity of Chlordane to White Rats”, J. Econ. Entomol. 40: 264-268, 1947.

[48] Jandorf, B. J;. Sanett, H. P., and Bodansky, Oscar: “Effect of Oral Administration of DDT on Metabolism of Glucose and Pyruvie Acid in Rat Tissues”, J. Pharmaeol. and Exper. Therap. 88: 333-337, Dec. 1946.

[49] Jenkins, D. W.: “A Review of the Insecticide Hexachloro-cyclohexane (‘666’)”, Office of Technical Services, U. S. Dept of Commerce, Washington, D • C., No. PB 4034, Med. Div. Rept. No. 56, Sept. 26, 1945.

[50] Kempe, H. E.: “Progress Report on Benzene Hexachloride for the Destruction of Sheep Scab Mites”, Vet. Med., Feb. 1948, pp. 76-79.

[51] Kirk, H.: Vet. Red. 58: 43, 1946.

[52] Kirk, H.: “DDT in Canine Practice”, Vet. Med. Feb. 1947, PP. 76-78.

[53] Lawhon, G. J., Jr.: “X Disease in South Carolina”, N. Am. Vet. 29: 643, Oct. 1948.

[54] Leider, M.: “Allergenic Eczematous Contact-Type Dermatitis Caused by DDT”, J. Invest. Dermatol. 8: 125-126., March 1947.

[55] Lillie, R. D., Smith, M. I., and Stohlman, E. F.: Pathologic Action of DDT and Certain of its Analogs and Derivatives”, Arch. Path. 43: 127-142, Feb. 1947.

[56] Mackerras, I. M., and West, R. F. K.: “DDT Poisoning in Man”, M. J. Australia, 1: 400-401, March 23, 1946.

[57] Mobbs, J. F.:” Toxicity of Hexaehloroeyclohexane in Scabies, J.A.M.A. 138: 1253, Dec. 25, 1948. Personal Communication.

[58] Morrill, C. C.: “Hyperkeratosi.s or X Disease”, N. Am. Vet. 29: 642, Oct. 1948.

[59] Neal, P. A., Sweeney, T. B., Spicer, S. S., and von Oettingen, W. F.: “The Excretion of DDT in Man, Together with Clinical Observations”, Pub. Health Rep., 61: 403, March 22, 1946.

[60] Neal, P. A., von Oettingen, W. F., Smith, W. W., et al: Toxicology and Potential Dangers of Aerosols, Mists and Dusting Powders Containing DDT”, Pub. Health Rep. Suppl. 177, 1944.

[61] Neal, P. A., von Oettingeu, W. F., Dunn, R. C., and Sharpless, N. E.: “Toxicology and Potential Dangers of Aerosols and Residues from Aerosols Containing 3 Percent of DDT. Second Report, ibid., Suppl. 183, 1945.

[62] Nelson, A. A., Draize, 3. H., Woodard, G., et al: “Histopathological Changes Following Administration of DDT to Several Species of Animals”, U. S. Pub. Health Rep. 59: 1009, Aug. 4, 1944.

[63] Neve, Helen: “Toxic Effects of DDT on a Cat”, Vet. Rec. 58: 43, 1946. Vet. Med., Feb. 1947, p. 78.

[64] Niedelman, M. L.: “Contact Dermatitis Due to DDT”, Occup. Med. 1: 391-395, April 1946.

[65] Radeleff, R. D.: “DDT Spray Outmodes Dipping Vat”, Vet. Med. Oct. 1947, pp. 372- 373.

[66] Radeleff, R. D.: “Chlordane Poisoning: Symptomatology and Pathology, Vet. Med. Aug. 1948, pp. 342-347.

[67] Robinson, J. H.: “Harvest Analysis of DDT Residues”, Food Packer, 29: 50-53, 1948.

[68] Riker, W. F., Jr., Huebner, Virginia, R., Raska, S. B., and Cattell, McKeen: “Studies on DDT, Effects on Oxidative Metabolism”, J. Pharmacol. and, Exper. Therap., 88: 327- 332, Dec. 1946.

[69] Sarrett, H. P., and Jandorf, B. J.: “Effects of Chronic DDT Intoxication in Rats on Lipids and Other Constituents of Liver”, ibid., 91: 340-344, Dec. 1947.

[70] Smith, M. I.: “Accidental Ingestion of DDT, with a Note on its Metabolism in Man”, J.A.M.A., 131: 519-520, Juno 8, 1946.

[71] Smith, M. I., and Stohlnian, E. F.: “Pharmacologic Action of 2, 2 his (p-Chlorophenyl) 1,1,1-Trichloroethane and its Estimation in the Tissues and Body Fluid”, Pub. Health Rep., 59: 984, July 28, 1944.

[72] SmIth, M. I., and Stohlman, E. F.: “Further Studies on the Pharmacologic Action of DDT”, ibid., 60: 289, March 16, 1945.

[73] Smith, N. 3.: “Death Following Accidental Ingestion of DDT”, J.A.M.A., 136: 469- 471, Feb. 14, 1948.

[74] Smith, R. F., Fullmes, O. H., and Messenger, P. S.: “DDT Residues on Alfalfa Hay and Seed Chaff”, J. Econ. Entomol. 41: 755-8, 1948.

[75] Strycker, G. V., and Godfroy, B.: “Dermatitis Resulting from Exposure to DDT”, J. Missouri St. M. A., 43: 384-386, June 1948.

[76] Taylor, E. L.: “Danger of Ununction with DDT”, Lancet, 2: 320, Sept. 8, 1945.

[77] Telford, H. S., and Guthrie, J. E.: “Transmission of the Toxicity of DDT Through the Milk of White Rats and Goats”, Science, 102: 647, Dec. 21, 1945.

[78] Thoungh, TI. C.: “Poisonous Effects of DDT on Humans”, Indian M. Ga:. 81: 432, Oct. 1946.

[79] U. S. Dept. Agriculture, “Bureau of Entomology and Plant Quarantine: Now Insecticides in Grasshopper Control”, Bull. E-722, May 1947. Bull. EC.1, March 1948.

[80] U. S. Dept. Agriculture, Bureau of Entomology and Plant Quarantine: “New Insecticides for Controlling External Parasites of Livestock”, Bull. E. 762, Dec. 1948.

[81] Westerfteld, C.: “The Use of DDT in Medicine-A Review”, Vet. Med., Oct. 1946, pp. 355-360.

[82] Wigglesworth, V. D.: “A Case of DDT Poisoning in Man”, Brit M. J. 1: 517, April 14, 1945.

[83] Wilson, J. B.: Are Pesticides Making Your Food Unsafer? Hygiea, Jan. 1949. p. 44.

[84] Woodard, G., Ofner, Ruth B., and Montgomery, C. M.: “Accumulation of DDT in the Body Fat and its Appearance in the Milk of Dogs”, Science, 102: 177-178, Aug. 17, 1945.

[85] Wright, C. S., Doan, C. A., and Haynie, H. C.: “Agranulocytosis Occurring after Exposure to DDT Pyrethrum Aerosol Bomb”, Am. J. Med., 1: 562-567, Nov. 1946.

[86] The Pesticide Residues Amendment of 1954, Pub. L. No. 83-518, ch. 559, 68 Stat. 511 [codified at 21 USC § 346a (1981)]; and the Food Additives Amendments of 1958, Pub. L. No. 85-529, Ch. 4.72 Stat. 1785 [codified at 21 USC § 348 (1981)], respectively.

[87] 20 Fed. Reg. 750 (1955) [codified until repealed at 21 CFR § 120. 1(f) (1956). [88] DDT Regulatory History: A Brief Survey (to 1975). United States Environmental

Protection Agency (EPA).

[89] Ibid.

[90] TIME Magazine, U.S. Edition, March 14, 1994 Vol. 143 No. 11.

(91] Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991.

[92] Peter Duesberg and Brian J. Ellison, Inventing the AIDS Virus, Regnery Pub.,1996. [93] Ibid.

[94] Biskind, MS (1953) “Public Health Aspects of the New Insecticides,” American Journal of Digestive Diseases 20: 331-341.

[95] Peter Duesberg and Brian J. Ellison, Inventing the AIDS Virus, Regnery Pub.,1996. [96] DDT Regulatory History: A Brief Survey (to 1975). United States Environmental

Protection Agency (EPA).

[97] Poliomyelitis: Fact sheet N°114″. World Health Organization. Sep 2016. Retrieved 14 Sep 2016.

[98] Ibid.

[99] DDT Regulatory History: A Brief Survey (to 1975). United States Environmental

Protection Agency (EPA).

[100] Ibid.

[101] Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R.

Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991.

[102] Rea WJ, Johnson AR, Fenyves E, Butler J. Related Articles: The environmental aspects of the post-polio syndrome. Birth Defects Orig Artic Ser. 1987;23(4):173-81. No abstract available. Pub Med ID: 3620615; UI: 87299998.

[103] Ibid.

[104] Casarett and Doull’s Toxicology (1996).

[105) Rea WJ, Johnson AR, Fenyves E, Butler J. Related Articles: The environmental aspects of the post-polio syndrome. Birth Defects Orig Artic Ser. 1987;23(4):173-81. No abstract available. Pub Med ID: 3620615; UI: 87299998.

[106] PubMed ID: 7611631, UI: 95336052 (London, May, 1995)

[107] Pub Med ID: 7611630, UI: 95336051 (Bethesda, MA, May, 1995)

[108] Pub Med ID: 8818905, UI: 96415998 (Lyon, France, Aug., 1996)

[109] Alfredo Morabia (1 January 2004). A History of Epidemiologic Methods and Concepts. Springer. pp. 133–4. ISBN 978-3-7643-6818-0. Retrieved 22 June 2013.

[110] Ibid.

[111] Morton S. Biskind, MD. “Public Health Aspects of the New Insecticides”. American

Journal of Digestive Diseases, New York, 1953, v 20, p331. [112] Ibid.

[113] Young RO (2016) Second Thoughts Concerning Viruses, Vaccines and the HIV/AIDS Hypothesis – Part 2. Int J Vaccines Vaccin 2(3): 00034. DOI: 10.15406/ijvv.2016.02.00034

[114] Dirt and Disease: Polio before FDR Rutgers University Press, 1992. [115] Ibid.

[116] Menkes, John H., Child Neurology, pg. 420, (1995).

[117] A Paralyzing Fear: The Story of Polio in America. Produced by Paul Wagner, Nina Gilden Seavey. Directed, written by Nina Gilden Seavey. Narration written by Stephen Chodorov. With: Narrator: Olympia Dukakis. Camera (Colorlab color), Allen Moore, Reuben Aaronson; editor, Catherine Shields; music, Paul Christianson; associate producers, Tom Wentworth, Malvina Anderson Martin. Reviewed on videocassette, N.Y., March 3, 1998. Running time: 90 min.

[118] FILM REVIEW; Once a Fear Beyond Fear Itself, by STEPHEN HOLDEN, Published: March 4, 1998, New York Times.

[119] Ibid.

[120] Duesberg, Peter and Ellison, Brian J., Inventing the AIDS Virus, Regnery Pub.,1996.

[121] Ibid.

[122] Ibid.

[123] Ibid.

[124] Ibid.

[125] Rose DR (2004). “Fact Sheet—Polio Vaccine Field Trial of 1954.” March of Dimes Archives. (2004).

[126] Ibid.

[127] American Journal of Digestive Diseases, 1953 20:330 [128] Ibid.

[129] Ibid.

[130] Jenkins, D. W.: “A Review of the Insecticide Hexachloro-cyclohexane (‘666’)”, Office of Technical Services, U. S. Department of Commerce, Washington, D.C., No. PB 4034, Med. Div. Rept. No. 56, Sept. 26, 1945.

[131] Biskind, M., “DDT Poisoning and the Elusive ‘Virus X’.” A New Cause for Gastroenteritis.” Am. J. Dig., Vol. 16, Num 3, pg. 79-84, (1949).

[132] Biskind, MS, Bieber, I, “DDT Poisoning A New Syndrome With Neuropsychiatric Manifestations,” American Journal of Psychotherapy, p261, (1949).

[133] Presented before the Select Committee to Investigate the Use of Chemicals in Food Products, United States House of Representatives, U.S. December 12, 1950 Westport, Conn.

[134] “Salk and Sabin: poliomyelitis immunisation”. J Neurol Neurosurg Psychiatry. 75 (11): 1552. doi:10.1136/jnnp.2003.028530. PMC 1738787. PMID 15489385.

[135] H. Rept. No. 2356, 82d Cong., 2d sess. 1 (1952), reprinted in A Legislative History of the Federal Food, Drug and Cosmetic Act and Its Amendments 499 (hereinafter Legislative History)

[136] Scobey, RR, “Is The Public Health Law Responsible For The Poliomyelitis Mystery?” Syracuse, N.Y., Archive of Pediatrics (May, 1951).

[137] White, Mark; Sharon M. McDonnell; Denise H.Werker; Victor M. Cardenas; Stephen B. Thacker (2001). “Partnerships in International Applied Epidemiology Training and Service,”. American Journal of Epidemiology 154 (11): 993–999. doi:10.1093/aje/154.11.993.

[138] Van Nostrand’s Encyclopedia of Science and Engineering, Van Nostrand Reinhold 1995, v 5, p1775

[139] “Salk and Sabin: poliomyelitis immunisation”. J Neurol Neurosurg Psychiatry. 75 (11): 1552. doi:10.1136/jnnp.2003.028530. PMC 1738787. PMID 15489385.

[140] Ralph R. Scobey, MD. “The Poison Cause of Poliomyelitis and Obstructions to Its Investigation.” Archive of Pediatrics, April 1952.

[141] The National Adipose Tissue Survey, reported in Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991, pg. 303.

[142] The National Adipose Tissue Survey, reported in Handbook of Pesticide Toxicology, edited by Wayland J. Hayes, Jr. and Edward R. Laws, Academic Press Inc., Harcourt Brace Jovanovich, Publishers, San Diego, 1991, pg. 303.

[143] Van Nostrand’s Encyclopedia of Science and Engineering (1995), vol. 5, pg.1725. [144] Offit, Paul A. (2007). The Cutter Incident: How America’s First Polio Vaccine Led to

the Growing Vaccine Crisis. Yale University Press. p. 38. ISBN 0-300-12605-0. [145] Albert Sabin to Henry Kumm, Sabin Papers, UC, Pittsburgh Press, 1954. [146] American Journal of Digestive Diseases, 1953 20:330.

[147] Trevelyan, B., Smallman-Raynor, M. and Cliff, A.D., The Spatial Dynamics of Poliomyelitis in the United States: From Epidemic Emergence to Vaccine-Induced Retreat, 1910–1971, Ann Assoc Am Geogr. 2005 Jun; 95(2): 269–293.

[148] Baicus, A., History of Polio Vaccination, World J Virol. 2012 Aug 12; 1(4): 108–114. Published online 2012 Aug 12. doi: 10.5501/wjv.v1.i4.108.

[149] Ibid.

[150] Women’s History Month: “Oveta Culp Hobby” by Senator Kay Bailey Hutchison

Humanities Texas, March 2012.

[151] Harry M. Marks, “The 1954 Salk Poliomyelitis Vaccine Field Trial,” Institute of the History of Medicine, Johns Hopkins University, Baltimore, MD: 2008.

152[ National Museum of American History, “Whatever Happened to Polio?” Time line, http://americanhistory.si.edu/polio/timeline/index.htm (accessed March 28,, 2012).

[153] Abid.

[154] Norrby E., Prusiner S.B., Polio and Nobel Prizes: looking vack 50 years. Ann Neurol.

2007 May;61(5):385-95.

[155] Eloise Batic, You Are There 1955: Ending Polio exhibit text (2012).

[156] Boston Herald newspaper, April 18, 1955, “Drug Companies Expecting Big Profit on

Salk Vaccine”,

[157] Washington Bureau of the Detroit Free Press reports, June 3, 1955.

[158] Michigan University. Poliomyelitis Evaluation Center (1955), An evaluation mof the 1954 poliomyelitis vaccine trials; summary report. Ann Arbor: n.p. , pp. 17-18 as quoted in Marks, Harry M. “The 1954 Salk Poliomyelitis Vaccine Field Trial.” Institute of the History of Medicine, Johns Hopkins University. Baltimore: 2008, p. 20.

[160] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1

[161] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research, 1957:119.

[162] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1

[163] Offit, Paul A. The Cutter Incident: How America’s First Polio Vaccine Led to the Growing Vaccine Crisis, Yale University Press, 2005, pp. 100, 116–19, 133. ISBN 0-300- 10864-8

[164] Ibid.

[165] Smith, JS, “Patenting the Sun: Polio and the Salk Vaccine,” 1st Edition, William

Morrow & Co; 1st edition (April 1990).

[166] Offit PA (2005), “The Cutter incident, 50 years later” (PDF). N. Engl. J. Med. 352 (14): 1411–1412. doi:10.1056/NEJMp048180. PMID 15814877

[167] McBean E., The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1.

[168] Harris RJ et al Contaminant viruses in two live vaccines produced in chick cells. J Hyg (London) 1966 Mar:64(1) : 1-7

[169] McBean E. The Poisoned Needle. Mokelumne Hill, California: Health Research,1957:1

[170] Ibid.

[171] Ibid.

[172] Ibid.

[173] Ii. Results. American journal of public health and the nation’s health. 1955;45:15–48. [PMC free article] [PubMed]

[174] Harper’s Magazine. “’Who is responsible, and why, for the chaotic confusion over the polio inoculations?’ A noted medical journalist disentangles the essential facts.” August, 1955.

[175] Ibid.

[176] Ibid.

[177] American Cancer Society, Volume 8, Issue 1, Pages 1–218, (1955).

[178] Paul JR. A history of poliomyelitis. New Haven, CT: Yale University Press; 1971.

[179] Ibid.

[180] Ibid.

[181] Ibid.

[182] Rogers N. Dirt and disease: Polio before fdr. New Brunswick, NJ: Rutgers University Press; 1992.

[183] Ibid.

[184] Smith, Derek R; Leggat Peter A (2005). “Pioneering figures in medicine: Albert Bruce Sabin–inventor of the oral polio vaccine”. The Kurume medical journal. 52 (3): 111–6. doi:10.2739/kurumemedj.52.111. PMID 16422178

[185] Rose, David, March of Dimes Archives, August 26, 2010. http://www.marchofdimes.org/mission/a-history-of-the-march-of-dimes.aspx

[186] American Journal of Public Health and the Nations Health: May 1956, Vol. 46, No. 5: 547–562. Citation | PDF (2177 KB) | PDF Plus (744 KB)

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[189] O’Hern M. Profiles: Pioneer Women Scientists. Bethesda, MD: National Institutes of Health.

[190] Curtis T, Manson P. Scientist’s Polio Fear Unheeded: How U.S. Researcher’s Warning Was Silenced. The Houston Post 1992:A1 and A12.

[191] Sweet BH, Hilleman MR. The Vacuolating Virus: SV-40. As cited in The polio vaccine and simian virus 40 by Moriarty, T.J. http://www.chronicillnet.org/online/bensweet.html

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[193] Shah K, Nathanson N. Human exposure to SV40. American Journal of Epidemiology, 1976;103:1-12.

[194] Curtis T. The origin of AIDS: A startling new theory attempts to answer the question, “Was it an act of God or an act of man”, Rolling Stone, March 19,1992:57.

[195] Bookchin D, Schumaker J. Tainted Polio Vaccine Still Carries Its Threat 40 Years Later. The Boston Globe, January 26, 1997.

[196] Innis MD. Oncogenesis and poliomyelitis vaccine. Nature, 1968;219:972–3. [197] Soriano F, et al. Simian virus 40 in a human cancer. Nature, 1974; 249:421–4.

[198] Weiss AF, et al. Simian virus 40-related antigens in three human meningiomas with defined chromosome loss. Proceedings of the National Academy of Science, 1975;72(2):609–13.

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[204] Weiner LP, et al. Isolation of virus related to SV40 from patients with progressive multifocal leukoencephalopathy. New England Journal of Medicine, 1972;286:385–90.

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[206] Meinke W, et al. Simian virus 40-related DNA sequences in a human brain tumor. Neurology 1979;29:1590–4.

[207] Krieg P, et al. Episomal simian virus 40 genomes in human brain tumors. Proceedings of the National Academy of Science 1981; 78:6446-50.

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[209] Geissler E. SV40 in human intracranial tumors: passenger virus or oncogenic >hit- and-run= agent? Z Klin Med, 1986;41:493–5.

[210] Geissler E. SV40 and human brain tumors. Progress in Medical Virology, 1990;37:211–22.

[211] Bergsagel DJ, et al. DNA sequences similar to those of simian virus 40 in ependymomas and choroid plexus tumors of childhood. New England Journal of Medicine, 1992;326:988–93.

[212] Martini, M., et al. Human brain tumors and simian virus 40. Journal of the National Cancer Institute, 1995;87(17):1331.

[213] Lednicky JA, et al. Natural Simian Virus 40 Strains are Present in Human Choroid Plexus and Ependymoma Tumors. Virology, 1995;212(2):710–7.

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[216] Carbone, M., et al. SV-40 Like Sequences in Human Bone Tumors. Oncogene, 1996;13(3):527–35.

[217] Pass, HI, Carbone, M., et al. Evidence For and Implications of SV-40 Like Sequences in Human Mesotheliomas. Important Advances in Oncology, 1996:89-108.

[218] Rock, Andrea. The Lethal Dangers of the Billion Dollar Vaccine Business, Money, December 1996:161.

[219] Carlsen, W. Rogue virus in the vaccine: Early polio vaccine harbored virus now feared to cause cancer in humans. San Francisco Chronicle, July 15,2001:7. Research by Susan Fisher, epidemiologist, Loyola UniversityMedical Center.

[220] National Institutes of Health. Zones of Contamination: Globe Staff Graphic.

[221] Bookchin D, Schumacher J. Tainted polio vaccine still carries its threat 40 years later. The Boston Globe, January 26, 1997.

[222] SV-40 Contamination of Polio Vaccine. Well Within Online, (February 3,2001, updated). http://www.nccn.net/~wwithin/polio.htm

[223] Rosa FW, et al. Absence of antibody response to simian virus 40 afterinoculation with killed-poliovirus vaccine of mothers offspring with neurological tumors. New England Journal of Medicine, 1988;318:1469.

[224] Rosa FW, et al. Response to: Neurological tumors in offspring after inoculation of mothers with killed poliovirus vaccine. New England Journal of Medicine, 1988, 319:1226.

[225] Martini F, et al. SV-40 Early Region and Large T Antigen in Human Brain Tumors, Peripheral Blood Cells, and Sperm Fluids from Healthy Individuals. Cancer Research, 1996;56(20):4820–5.

[226] Fisher, Barbara. Vaccine safety consumer group cites conflict of interest in government report on cancer and contaminated polio vaccine link. National Vaccine Information Center (NVIC); Press Release, January 27, 1998.

[227] National Cancer Institute (June 2001).

[228] The Landsteiner and Popper study, first published in Germany, was reported in Robert W Lovett, MD. The Occurrence of Infantile Paralysis in Massachusetts in 1908. Boston Medical and Surgical Journal, pg. 112, July 22, 1909.

[229] Young, RO (2016) Second Thoughts about Viruses, Vaccines, and the HIV/AIDS Hypothesis – Part 1. Int J Vaccines Vaccin 2(3): 00032. DOI: 10.15406/ijvv.2016.02.00032

[230] Young, RO (2016) Second Thoughts Concerning Viruses, Vaccines and the HIV/AIDS Hypothesis – Part 2. Int J Vaccines Vaccin 2(3): 00034. DOI: 10.15406/ijvv.2016.02.00034

[231] Young RO (2016) Second Thoughts Concerning Viruses, Vaccines and the HIV/AIDS Hypothesis – Part 3 HIV/AIDS and the Monomorphic Disease Model. Int J Vaccines Vaccin 2(3): 00035. DOI: 10.15406/ijvv.2016.02.00035

[232] Young RO (2016) Who Had Their Finger on the Magic of Life – Antoine Bechamp or Louis Pasteur?. Int J Vaccines Vaccin 2(5): 00047. DOI: 10.15406/ijvv.2016.02.00047

[233] Peter Duesberg and Brian J. Ellison, Inventing the AIDS Virus, Regnery Pub., 1996. [234] Gerald L. Geison, The Private Science Of Louis Pasteur, Princeton University Press, 1995.

[235] The Landsteiner and Popper study, first published in Germany, was reported in Robert W Lovett, MD. The Occurrence of Infantile Paralysis in Massachusetts in 1908. Boston Medical and Surgical Journal, pg. 112, July 22, 1909.

[236] Shaw D. Unintended casualties in war on polio. Philadelphia Inquirer June 6, 1993:A1.

[237] Moriarty T.J. The polio vaccine and simian virus 40. Online News Index. http://www.chronicillnet.org/online/bensweet.html

[238] Koprowksi H. Tin anniversary of the development of live virus vaccine. Journal of the American Medical Association 1960;174:972–6.

[239] Hayflick L, Koprowski H, et al. Preparation of poliovirus vaccines in a human fetal diploid cell strain. American J Hyg 1962;75:240–58.

[240] Hayflick L, Koprowski H, et al. Preparation of poliovirus vaccines in a human fetal diploid cell strain. American J Hyg 1962;75:240–58.

[241] Koprowski H. In a letter sent to the Congressional Health and Safety Subcommittee, April 14, 1961.

[242] Rock, Andrea. The Lethal Dangers of the Billion Dollar Vaccine Business, Money, December 1996:161.

[243] Scheibner V. Vaccination: 100 Years of Orthodox Research Shows that Vaccines represent a Medical Assault on the Immune System. Blackheath, NSW, Australia: Scheibner Publications, 1993153.

[244] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[245] Carlsen, W. Rogue virus in the vaccine: Early polio vaccine harbored virus now feared to cause cancer in humans. San Francisco Chronicle, July 15,2001:7. Research by Susan Fisher, epidemiologist, Loyola UniversityMedical Center.

[246] Neustaedter R. The Vaccine Guide. Berkeley, California: North Atlantic Books, 1996:107–8

[247] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[248] Essex M, et al. The origin of the AIDS virus. Scientific American, 1988; 259:64–71. [249] Karpas A. Origin and Spread of AIDS. Nature, 1990; 348:578.

[250] Kyle WS. Simian retroviruses, poliovaccine, and origin of AIDS. Lancet, 1992; 339:600–1.

[251] Elswood BF, Stricker RB. Polio vaccines and the origin of AIDS. Medical Hypothesis, 1994:42:347–54.

[252] Myers G, et al. The emergence of simian/human immunodeficiency viruses. AIDS Res Human Retro 1992:8:373–86.

[253] Curtis T. The origin of AIDS: A startling new theory attempts to answer the question “Was it an act of God or an act of man”, Rolling Stone, March 19,1992:57.

[254] O’Hern M. Profiles: Pioneer Women Scientists. Bethesda, MD: National Institutes of Health.

[255] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[256] Curtis T. Expert says test vaccine: backs check of polio stocks for AIDS virus. The Houston Post, March 22, 1992:A-21.

[257] Essex M, et al. The origin of the AIDS virus. Scientific American, 1988; 259:64–71. [258] Karpas A. Origin and Spread of AIDS. Nature, 1990; 348:578.

[259] Kyle WS. Simian retroviruses, poliovaccine, and origin of AIDS. Lancet, 1992; 339:600–1.

[260] Elswood BF, Stricker RB. Polio vaccines and the origin of AIDS. Medical Hypothesis, 1994:42:347–54.

[261] Workshop on Simian Virus-40 (SV-40): A Possible Human Polyomavirus. National Vaccine Information Center, January 27-28, 1997. http://www.909shot.com/polio197.htm (Includes a summary of evidence presented at the Eighth Annual Houston Conference on AIDS.)

[262] Martin B. Polio vaccines and the origin of AIDS: The career of a threatening idea. Townsend Letter for Doctors, January 1994:97–100.

[263] Curtis T. Did a polio vaccine experiment unleash AIDS in Africa? The Washington Post, April 5, 1992:C3+.

[264] Myers G, et al. The emergence of simian/human immunodeficiency viruses. AIDS Res Human Retro 1992:8:373–86.

[265] World Health Organization. T-lymphotropic retroviruses of nonhuman primates. WHO informal meeting. Weekly Epidemiology Records, 1985; 30:269–70.

[266] Ibid.

[267] Elswood BF, Stricker RB. Polio vaccines and the origin of AIDS. Medical

Hypothesis, 1994:42:347–54.

[268] Ohta Y, et al. No evidence for the contamination of live oral poliomyelitis vaccines with simian immunodeficiency virus. AIDS, 1989; 3:183–5.

[269] Huet T, et al. Genetic organization of a chimpanzee lentivirus related to HIV-1. Nature, 1990; 345:356–9.

[270] Desrosiers RC. HIV-1 origins: A finger on the missing link. Nature, 1990;345:288– 9.

[271] Sabin AB. Properties and behavior of orally administered attenuated poliovirus vaccine. Journal of the American Medical Association, 1957; 164:1216–23.

[272] Siehe Ausführungen zu Virchows Leben und Wirkung in WissenschafftPlus Nr. 5/2015 und Nr. 6/2015. 2 Anticontagionism between 1821 and 1867.

[273] Aufsatz von Erwin H. Ackerknecht in der Zeitschrift Bulletin of the History of Medicine, Volume XXII, The Johns Hopkins Press, 1948.

[274] Bech V, Magnus Pv. Studies on measles virus in monkey kidney tissue cultures. Acta Pathol Microbiol Scand. 1959; 42 (1): 75–85.

[275] Nakai M, Imagawa DT. Electron microscopy of measels virus replication. J. Virol. 1969 Feb; 3v (2): 187–97.

[276] Lund GA, Tyrell, DL, Bradley RD, Scraba DG. The molecular length of measles virus RNA and the structural organization of measles nucleocapsids. J. Gen. Virol. 1984 Sep;65 (Pt 9): 1535–42.

[277] Daikoku E, Morita C, Kohno T, Sano K. Analysis of Morphology and Infectivity of Measles Virus Particles. Bulletin of the Osaka Medical College. 2007; 53 (2): 107–14.

[278] Horikami SM, Moyer SA. Structure, Transcription, and Replication of Measles Virus. Curr Top Microbiol Immunol. 1995; 191: 35–50.

[279] Siehe WissenschafftPlus Nr. 1/2014.

[280] Zur Geschichte der frühen Virusforschung. Übersichtsarbeit von Prof. Karlheinz Lüdtke. Reprint 125 des MAX-PLANCK-INSTITUT FÜR WISSENSCHAFTSGESCHICHTE, 89 Seiten, 1999.

[281) The government of the United States of America holds patents on the following viruses: Ebola, Patent number #CA2741523A1, Swine Flu, Patent number 8124101, HIV, Patent number #5676977, the cure for cancer, Patent number #6630507.

Dr. Young’s Recent Scientific Peer-reviewed Published Articles

The Annual Conference on Bacterial, Viral and Infectious Diseases held in Dubai, UAE and published in The Journal of Infectious Diseases & Therapy

Robert Young

The Annual Conference on Bacterial, Viral and Infectious Diseases held in Dubai, UAE and published in The Journal of Infectious Diseases & Therapy

Dr. Robert O. Young presents diplomas and certificates to the scholars and faculty on the joint meeting of the Annual Conferences on Bacterial, Viral & Infectious Diseases and the Neglected Tropical Disease Congress: The Future Challenges, December 5th and 6th, 2018, Dubai, UAE sponsored and published in the Journal of Infectious Diseases and Therapy, December 2018. Volume 6. ISSN: 2332-0877.

 

The following picture is Dr. Young presenting one of our keynote speakers, Dr. Crystal M. James, Tuskegee University, USA on her presentation of research on The Defining Health Security: Neglected Disease in Rural Alabama. I was shocked to learn that there are many counties in Alabama where there are no hospitals and no doctors to provide care. The USA is so far behind in not only providing care but also providing quality care.

 

The second picture shows some of the published information presented by Dr. Robert O Young and Dr. Galina Migalko.

 

Here are just a few of the Professors and Medical Doctors from around the World that presented their research, including Professor Robert O Young and Dr. Galina Migalko.

 

1) Dr Stef Steinstra, Royal Dutch Navy, Netherlands

2) Dr Giulio Tarro, Foundation T& L Beaumont Boneli for cancer research, Italy

3) Dr Ghweil Ali Abderlrahman, South Valley University, Egypt

4) Dr Yusef Nazzal, Zayed University, United Arab Emirates

5) Dr Ghada Al Qassim, Bahrain Medical Society, Kingdom of Bahrain

6) Dr Ruben Bueno Mri, Laboratories Lokinica, Spain

7) Dr Abdul Matin, Majmuuh University, Saudia Arabia

8) Dr Waled Abuhammour, Michigan State University, USA

9) Dr Abdulrahman Abdulhadi Al-Sultan, King Fasal University, Saudi Arabia

10) Dr Arun Kumar Jha, Life Care Hospital, United Arab Emirates

11) Dr Muhammed Adeel Hassan, PRAS Arid Agriculture, University of Pakistan

12) Dr Carl Contini, University of Ferrara, Italy

13) Dr Addulrahman Mohammad, Magrah Hospital and Centers, Eygpt

14) Dr Ibrahim El-Bayonmy, Tanta Faculty of Medicine, Egypt

15) Dr Galina Migalko, Non-invasive Medical Diagnostics, Ukraine

16) Dr Digambar Behera, Institute of Medical Education and Research, India

17) Dr Crystal M James, Tuskegee University, USA

And many more speakers from around the World, including South Africa, Brazil, French Giana, Nigeria, France, Portugal, Australia, just to name a few.

 

To learn more about the work, research, findings and scientific publications of Dr. Robert O Young or Dr Galina Migalko, click on the following links: http://www.drrobertyoung.com or http://www.universalmedicalimaging.com

A Second Thought About Viruses, Vaccines and the HIV, HPV, HEP C, Measles, Mumps, SARS, Hantavirus and Ebola Hypothesis

Micrograph of a solidification of metabolic acid
Parasite or Virus?
So-called Ebola Virus or Parasite?
A Second Thought About Viruses, Vaccines and the HIV/AIDS, HEP C, HPV, Polio, Spanish Flu, Hantavirus, SARS, Measles, Mumps,  and Ebola Hypothesis! – Part 1
“In the sciences, people quickly come to regard as their own personal property that which they have learned and had passed on to them at the Universities and Academies.  If, however, someone else now comes along with new ideas to contradict the credo (that has been recited for years and passed on in turn to others) and in fact, even threaten to overturn it, and all passions are raised against this threat and no methods are left untried to suppress it.  People resist it in every way possible: pretending not to have heard about it; speaking disparagingly of it, as if it were not even worth the effort of looking into the matter.  And so a new truth can have a long wait before finally been excepted.”  – Goethe
Viruses
 
Introduction
The first isolation of the virus was achieved in 1892 by Russian that bacteria hunter Dmitri Ivowski, who gathered fluid from disease , tobacco plants.  He passed this liquid through field for fine enough to retain bacteria; yet to Ivowski’s surprise, the bacteria space free filtrate easily made healthy plants sick.  In 1888, a Dutch botanist, Martinus Wilhelm Beijerinck, repeating the experiment, also recognized that there was an invisible cause and named the infectious agent,  “Tobacco mosaic virus.”  In the same year as Beijerinck’s report, two German scientists, purified a liquid containing ‘filterable viruses’ that caused foot and mouth disease in cattle (viruses were at one time called ‘filterable bacteria’, but eventually the term ‘filterable bacteria’ came to apply only to viruses, and was the words ‘filterable bacteria’ were dropped).  Walter Reed followed in 1901 with a filtrate responsible for yellow fever, and soon dozens of other disease causing viruses were found.
In 1935 , another American, Wendell M. Stanley, went back to the beginning and created pure crystals of tobacco mosaic virus from a filtered liquid solution.  He affirmed that these crystals could easily infect plants, and concluded that a virus was not a living organism, since it could be crystallize like salt and yet still remain infectious.  Subsequently, bacteriologists all over the world began filtering for viruses, and a new era of biology was born – Virology.
Historically, medical science has a baseline on the question of whether any virus is alive.  Originally, it was described as non-living, but is currently said to be an extremely complex molecule or extremely simple microorganism, and is usually referred to as a parasite having a cycle of life.  (The term “Killed” is applied to certain viral vaccines, thus implying an official conviction that viruses are alive.)  Commonly composed of either DNA or RNA cores with protein coverings, and having no inherent reproductive ability, viruses depend upon the host for replication.  They must utilize the nucleic acids of living cells.  They infect to reproduce their proteins (i.e., trick the host into producing them), which are then assembled into new viruses like cars on assembly line.  Theoretically, this is their only means of surviving, and infecting new cells or hosts.
Birth of Virology — a Miscarriage?
Underlying the birth of virology was the doctrine of monomorphism — that all microorganisms (herein called microforms) are fixed species, unchangeable; that each pathological type produces (usually), only one specific disease; that microforms never arise endogenously, i.e., have absolute origin within the host; and that blood and tissues are sterile under healthy conditions.  This last point warrants immediate comment.  Theoretically, under ideal health conditions, the blood might be sterile, though it has the inherent potential to develop morbid microforms, as discussed in my book, Sick and Tired.  Long and repeated observation of live blood in the phase contrast, darkfield microscope, however, shows that the blood can contain various microforms and otherwise asymptomatic host, or in a condition defined as normal or healthy in orthodox terms.  The forms are easily visible before other physical symptoms arise. (Since long and repeated observation has correlated their presence with other disease symptoms and their disappearance with the return of health, they serve as indicators of impending outward signs of disease).
Monomorphism was the cornerstone of developments in 20th-century medical research and treatments.  Refusal by the mainstream to examine fairly, much less except, the demonstrated fact of pleomorphism — that viruses and bacteria (and also yeast, fungi and mold) are evolutions from a small indestructible anatomical element, I referred to as the microzyma.  That microforms can rapidly change their form (evolve and “devolve”) in vivo, one becoming another dependant upon conditions in the inner terrain (environment); that blood and tissues are not necessarily sterile; and that there are no specific diseases, but only specific disease conditions — was the foundation of a latter-day “Galileo debate.”  It is so-called because those who wore the “robes” of scientific authority just like today, reprising the religious fanatics who punished the noted astronomer for his truth, would not be swayed from folly when presented with its contrary theory.  These truths began in earnest with Antoine Bechamp in the 19th-century (who also endured the indignation of a fanatical clergy).
In the early third of the 20th-century, the heated debate took place over ‘filterable bacteria’ versus ‘non-filterable’ bacteria.  This was a major battle concerning micromorphology (discussed briefly below). The orthodox view prevailed: bacterial forms were not small enough to pass, or did not have a smaller, earlier stage.  What passed through ‘bacteria proof’ filters was something else, i.e., viruses.  Standard medical textbooks, long made this filtering distinction between bacteria and viruses.  Subsequently, however, the cellular nature of many filterable forms originally thought to be viruses, such as some mycoplasmas, rickettsias, and various other groups, has been established.  In this writer’s opinion, with the victory of the monomorphic view, deeper understanding of infectious ‘disease’ was lost, setting the stage for cancer, degenerative symptoms, HIV. AIDS, Ebola, Hantavirus, Hep C, HPV, etc.
What You See?
A typical bacteria is about 1 micron in size.  Most filterable bacterial forms now called viruses range in size from .3 microns (300 millimicrons) to .01 microns (10 millimicrons) — particularly in the colloidal range  (.1 to .001 micron). Most of the larger viruses are a third to a quarter the size of the average bacteria.  And size is critical because .3 microns is the resolution limit of modern-day light microscopes.  Thus, as viruses were discovered (except for the very large ones, such as mumps), they required an electron microscope to be seen, especially given the the fact that Royal Rife’s microscope technology and career were destroyed by vested interests.  Unfortunately, electron microscopes and the process of chemical staining disorganize or damage all specimens, whereas Rife’s technology allowed life to proceed and thus evolve under its lens. As viruses became visible to advancing technology, the ratification was that the technology revealed, two minds infected with monomorphism, protein structures deemed foreign in the body.
A New Theory
Formulated by Antoine BeChamp in the 19th-century, the microzymian principal is the basis of the new theory about ‘viruses’.  Recently, this principle holds that in all living organisms are biologically indestructible anatomical elements, which BeChamp called microzymas.  They are independently living organized ferments, capable of producing enzymes and capable of evolving into more complex microforms such as bacteria, yeast or mold.  Bechamp’s thesis, is that disease is a condition of ones internal environment (terrain); that disease (and its symptoms) are “born of us and in us.”; and that disease is not produced by an attack of micro entities, but calls forth their endogenous evolution.
My studies and research suggests that the complexes, science calls viruses and retroviruses originate in the cell, as the microzymian as the principal suggests.  However, they are created in response to an alarming acidic situation (condition of disease) for the purpose of genetic repair.  They are repair proteins, evolved from anatomical elements (microzymas), not pathogenic  microorganisms.
It is known that normal cell activity includes genetic repair.  Both enzymes and proteins must be involved.  What is the mechanism?  Viruses are organized around DNA or RNA, not both.  Thus, they are quite probably intended to repair genetic molecules or other structures, and show up with disease symptoms, because the body needs them.  Since viruses require a living cell/host for reproduction, how do we know that the scenario is not set in motion, for a purpose by the cell (i.e., it’s microzymas), rather than being the result of invasion?  Because disease (disturbance of balance in the organism) is so prevalent, especially that which is not yet becoming indicated by common symptoms, repair proteins may be frequently or constantly present.  A toxified cell may easily suffer localized damage to the genome.  Since most observers are not even aware of the microzymian principal, much less understand or even consider it, and since monomorphism stresses invasion, these proteins complexes are regarded as foreign and disease is attributed to them.
Another note of interest is the size of viruses compared to the microzyma.  Viruses are considered to be some of the smallest biological particles and are frequently of colloidal size: e.g., hepatitis A, 27 nanometers (.027 microns); hepatitis B (.042 microns); polio virus (.03 microns); EBV (.042 microns); HIV (.080 to .12 microns); influenza (.08 to .12 microns); mumps (.15 to .30 microns); smallpox (.30 microns); and, according to BeChamp, the microzyma (.0005 microns).
In his book, ‘The Blood and its Third Anatomical Element’, Bechamp states: “the microzyma is at the beginning and at the end of all organization.  It is the fundamental anatomical element whereby the cellules, the tissues, the organs, the whole of the organism are constituted living . . . . in a state of health, the microzymas act harmoniously and our life is, in every meaning of the word, a regular fermentation.  In the condition of disease, the microzymas do not act harmoniously, the fermentation is disturbed, the microzymas have either change their function or are placed in an abnormal situation by some modification of the median.  The virus is either a self-ordered microzymian polymerization, or (less likely), a structure made by microzymas.  It is envelope in protein which is also composed of microzymas, and could well be thought of as an autonomous molecular tool box.
Along with doctors Glen Dettman and Archie Kalokerinos, I wonder, “whether Bechamp’s writing anticipated, in some respects, the discovery of RNA and DNA?”  Could the genetic structure be the construct, thus a tool, of the microzyma?  They quote a personal communication [1974] from a professor Bayev of the former USSR Academy of Sciences, who discusses his work showing the molecular self- restoration from its parts of pure transfer RNA from brewers yeast is possible.
In my own research I have found molecular restoration similar to that described by Bayev.  In my experiment , I used 10-year-old coagulated capillary blood from a woman with cancer.  With one drop of .9% of sodium chloride, the blood was restored to an appearance and level of activity characteristic of freshly drawn sample of blood.  In other words, the anatomical microzymas of the dry blood were restored to activity.  Even the white blood cells became active again.  One might eagerly asked for explanation of the reversal of polymers made during clotting.  It is unclear at this point how this reversal takes place, except to say that what can evolve apparently has the potential to devolve.  It is observable, however.  For example, I have seen, and recorded on video, rod microforms retro-grading without any visible decomposition from 10 microns in length to the vicinity of .1 micron.
This research supports the very important postulates that the cell is not the smallest living biological unit, as promulgated by conventional medical science.  In fact, a smaller biological unit is the imperishable micros I’m a, which is an organized, living been “of a special category without analog,” said BeChamp, who found them ready to become active in chalk deposits at least 11 million years old.
The Pleomorphic Cycle
I suggest a developmental cycle in vivo consisting of three macro stages: [1] a primitive stage comprising the repair proteins complexes; [2] an intermediate, or bacterial, stage including filterable forms such as the cell wall deficient forms described by Lida Mittman, PhD. [in Cell Wall Deficient Forms, Stealth pathogens]; and [3] a culmination stage consisting of yeast and fungal phases, and then mold, the and phase.  The usual course of development would be from microzyma to repair proteins, and then to bacterium, etc.  However, under certain conditions, such as, for example, it is highly likely that the microzymas can skip the primitive stage and become bacteria directly.  Although these transformations are as astounding as that of a larva to a butterfly, what is equally impressive under observation is in the rapidity with which they can take place — in minutes, even seconds, sometimes.  By the same token, when provoked by acidic conditions and the cycle proceeds to yeast, fungus and then mold, it may occur so rapidly that the bacterial stage, if that happens, has no time to be of any significance.
Thus, symptomgenic microforms can originate within the higher organisms without invasion, via a permutation of the endogenous microzymas when the situation calls for such change.  The situation is an imbalance referred to by Bechamp as a “modification of the median.”  Endogenous evolution is evident under the microscope when bacterial, yeast, and fungal forms are seen coming out of the red blood cells, which initially appear normal.
Biological Basis for the Pleomorphic Cycle
There is a common biological basis for the pleomorphic cycle and its increasing complexity of organization: more complex forms evolve inherently upon the death of an organism for the purpose of recycling its anatomical and chemical structures in the carbon cycle.  The process of rapid evolution [which is reversible] is an essential life process, which, beyond the repair stage, is necessary to return a dead organism to the earth.  The second and third stage microforms degenerate the body’s vital substances and tissues via putrefaction [bacteria] and fermentation [yeast and fungus].  Fermentation results in acidic waste products, which further breakdown tissue.  Disease symptoms, then, especially the degenerative type, are NOT produced by viruses, but manifest as chemical decomposition, or attempted recycling via fermentation and acidic toxins, but with ‘host’ survival processes still, operable.  Obviously, certain other factors may play important roles in producing symptoms, such as heavy-metal toxicity, or state of mind, for example.  Some of the body survival methods also produce symptoms commonly called dis-eases.  An example is eczema, and emergency expulsion of acidic toxins via the pores of the skin.
The aforementioned casual [alarming] situation, or modification of the median, is chronic tissue acidification [pH imbalance] and oxygen deprivation in the blood and tissues due to acidic forming foods, adverse lifestyle, emotional stress, and environmental stress.  This is not an oversimplification!  Acidification/hypoxia biochemically signals a dead host to the microzymas, while creating collapsed areas [dead zone’s] of the colloidal system in the intercellular fluid, and it is the primary physiological disease condition at which the symptoms commonly called specific diseases arise.
Thus, we distinguish between this disease condition and its consequent symptoms, which include both the morbidly evolved microzymas and the physiological science commonly thought of as specific diseases.  As they develop, microforms [bacteria, yeast, fungus and mold] are actually scavenging forms of the microzyma, developed when disease in the cell life requires tissue to be broken up.  These upper development forms are the ones easily visible in the blood before physical symptoms arise.  They disappear, [devolve] when the recycling task is complete, once again becoming microzymas of the earth and/or air.
Virus or Toxin/Acid?
Regarding the early period of virus isolation, a question is whether the unseen entities isolated in filtered fluids were accompanied by the waste products [mycotoxins] of fermentation by yeast and/or fungus of cellular elements, such as DNA.  If virus infiltrates are injected into a host to prove virulence, it is almost certain that easily filterable molecular toxins will be introduced as well.  Could Dr. Stanley’s “pure crystals of tobacco mosaic virus” have been crystallized acidic toxins?  If so, they would certainly be highly symptomgenic, as are exotoxins at the intermediate stage of the cycle, for example.  However, it is not proof of anything that you can create illness by poison injection, except proof of that tautological fact.
In my research utilizing darkfield and phase contrast microscopy, it is common to see acid crystallization’s in the blood.  It is normal for the body to use calcium or other mineral salts, and fats as well, to chelate the acidic waste products from the morbid fermentation of body proteins, fats and sugars.  Such crystal deposits are found in cancer tissue as well.  A malignant tumor removed from the breasts of one of my research clients was found to have numerous calcium deposits attached to it.  It is an attempt to render inactive acidic substances that make our inner streams healthy, poison our cells, and coagulated colloidal systems in blood and intercellular fluid.
The term “virus” is the Latin word for poison, and gives us insight into the immediate cause of disease symptoms — poison is: exotoxins and mycotoxins, and a toxin, exotoxins, and toxins from environmental sources, [many of which are primary or secondary mycotoxins.].  Orthodox medicine is well aware that it is bacterial toxins more than the bacteria them self.  [They feed in-house], that caused the symptoms referred to as infectious disease.  Little if any emphasis is placed on this fine, but important distinction.  Always, the germ is emphasized.  There is little too, no awareness [or knowledge that], either, of the same role played by acidic toxins of the culminate microforms of the pleomorphic cycle.  Their action and the body’s response to them are frequently ascribe to viruses, which do not produce toxins because they are the toxin or acid, but are said to wreck havoc by a number of other means.  However, if they participate in symptom at Genesis in a host it is because they are stimulated to evolve into more complex, toxic genetic forms.  Somewhat less likely is the possibility that they cause damage as a result of erroneous construction or function, for one reason or another — missing mineral nutrients leading to alkaline mineral deficiencies, for example.
Misconception Breeds Contempt
In addition to chemical toxicity, however, what is the impact of the fear [emotional toxicity] that the word “virus” brings to mind and heart?  It has been said that fear it is the most deadly of disease conditions.  If the “disease” kills one person, the fear of it may kill 20.  General prejudice concerning the danger of viruses is fundamental biological error based on Louis Pasteur’s germ theory, and is itself a perpetrator of auto-suggested illness.  For example, in Africa doctors attribute some AIDS sickness to “voodoo death” syndrome, the term for illnesses induced psychologically.  According to one nurse, “we had people who were symptomatically AIDS patients.  They were dying of AIDS, but when they were tested and found out they were negative they suddenly rebounded and are now perfectly healthy.”  Ironically, if the germ theory were found on facts, it would be correct to fear viruses, except there would be few, if any, humans living to discuss the issues.  These so-called pathogenic entities are to researchers, medical practitioners and the press what criminals are to detectives — the focus and justification of their existence.
The Encyclopedia Britannica has this to say about bacteria, which relates also to viruses:
“The common idea of bacteria in the minds of most people is that of the hidden and sinister scourge lying in wait for mankind.  This popular conception is born of the fact that attention was first focused upon bacteria through the discovery, some seven years ago, of the relationship of bacteria to disease in man, and that in its infancy, the study of bacteriology was a branch of medical science.  Relatively few people assigned to bacteria, the important position in the world of living things that they rightly occupied, for it is only a few of the bacteria known today that have developed in such a way that they can live in the human body, and for everyone of this kind, there are scores of others which are perfectly harmless and far from being regarded as the enemies of mankind, must be numbered among his best friends.
It is in fact, no exaggeration to say that upon the activities of bacteria.  The very existence of man depends; indeed, without bacteria there could be no other living thing in the world; for every animal and plant owes its existence to the fertility of the soil, and this in turn depends upon the activity of the microorganisms which inhabit the soil in almost inconceivable numbers.  It Is one of the main objects of this article to show how true is this statement; there will be found in it only passing reference to the organisms which produce disease in man and animals — for information on these see Pathology and Immunity.  [Encyclopedia Britannica, 14th ed., Volume 2, page 899].
The general message of the foregoing article applies even more aptly to viruses in the sense that much fear has been bred and cultivated around them, although they never produce disease symptoms, whereas the acid waste products of bacteria, yeast, fungus and mold do.  The writer of the above understands bacteria, with the exceptions that symptomgenic bacteria found in man and animals do not produce disease.  [Only secondary symptoms], that their precursors are endogenous to higher organisms, and they have not “developed in such a way that they can live in the human body.”  If anything, the reverse is true.  According to one theory of microbiology, microforms have colonized over eons to become higher organisms.  In one sense, then, the human body has developed as a specialized environment for them.
An important dimension of the bacterial dependence of higher life forms is the floral population in the human digestive tract.  Literally, these “foreign species” keep us alive.  Most bacteria have the same underlying function, whether found in the soil, sewage, in the human digestive tract, or elsewhere in nature: they are an essential part of the life processes of hire organisms.  They will not or cannot attack healthy cells or tissues, but certain ones will recycle sick or dead tissue in much the same way insect pests are drawn to weaker plants.  As Bechamp said, “nothing is the prey of death; all things are the prey of life.”
Following in the wake of misconceptions arising from the fundamental biological error known as the germ theory of disease, defying infiltrates of disease tissue as a newly discovered infectious microforms was the birth of a major corollary error in bio science.
Viral Behavior Reconsidered
Listed below are ways of viruses are said to disrupt or destroy host cells.  According to orthodox medical science and the germ theory advocates.  Following each in its italics is a different interpretation following from microzymian principle:
1.  Viral proteins insert into the host cells , plasma membrane and directly damage its integrity , to promote cell fusion [HIV, measles, and herpes viruses.].
Proteins are attempting to repair membrane damage, or enter cells to repair other proteins.  There is the question as to whether viruses on cell walls are coming or going.  In both cases, it would be a matter of whether or not a cell has been disturbed by excess fermentation and acidity.  But in the former case, the cell would be dysfunctional before attachment occurs, thus requiring the repair complex.  Another possibility, perhaps remote, is that dysfunctional receptors on cells are in need of repair, or they are covered by these complexes to inactivate malfunction of the cells.  Positive electrical charges in a compromised acidic terrain, primarily on acidic molecules from fermentation’s, discharge cell membranes and act as mortar to stick cells together causing rouleau and clotting.
2.  Viruses inhibit a host cell DNA, RNA, or protein synthesis.  For example, polio virus inactivates cap-binding protein, which is essential for protein synthesis, directed by capped host cell mRNA’s, while allowing protein synthesis from uncapped polio virus in mRNA’s.
Protein inactivation is probably being done by fermentation or by acidic toxins from fermentation, while “poliovirus” is produced in the cell to reverse the damage.
3.  Viruses replicate efficiently and lyse host cells, e.g., liver cells by yellow fever, and neurons by poliovirus.
Highly unlikely.  The lysing is more likely caused by acidic mycotoxicosis, or by free radicals released in response to mycotoxic stress, or from other sources [I lysine radiation, for example].  Repair particles are residual after cell wall disruption.
4.  Slow-virus infections [e.g., sub acute sclerosing panencephalitis caused by the measles virus] culminate in severe progressive disease is after a long latency period.
How is this demonstrated?  Perhaps “latency” is a period of unsuccessful or attempted repair that eventually falters.  Symptomology naturally appears in the weakest parts of the body.  Excess acidity is always a systemic problem that localizes, just as cancer is a systemic acidic condition that localizes, even though it its symtogenic influence may later spread.
5.  Viral antigen proteins on the surface of the host cells are recognized by the immune system, and the host lymphocytes attack, the virus infected cells [e.g., liver cells infected with hepatitis B].
Liver cells are damaged beyond repair by exotoxins and mycotoxicosis, and the immune system, our elaborate janitorial service, is cleaning out the garbage.  Perhaps the repair protein antigen is expressed to signal any in response [because the cell is beyond repair], which is one explanation for why there are antibodies to these proteins.
6.  Viruses damage cells involved in the host anti-microbial defense, leading to secondary infections.
The function of immune cells are damaged by bacterial or fungal waste products/acidic and/or overworked by toxic acidic overload, preventing proper cleanup and elimination of disharmonious, symptomgenic elements.
7.  Viral killing the one cell type causes the death of other cells that depend on them, e.g., degeneration of muscle cells enervated by the attack of poliovirus on motor neurons.
Once again, a misinterpretation and lack of understanding that is not viral microforms that damage neurons.  Acidic toxins from bacteria, yeast, fungus and mold — as well as the ferments of glucose, uric acid from proteins, hormones and acetic acids from fats — produce, or influence the body to produce, dis-ease  or inflammatory symptoms.  Not recognizing “virus,” for what it is, observers attribute dis-ease or disease to it.
8.  Host cell responses to viruses include metabolic derangement and transformations resulting in neoplastic changes.
Metabolic derangement has occurred prior to the appearance of repair proteins, due to toxic overload in the cell.  It is more likely that the proteins attempt to prevent cell transformation, and that cancerous development is cell conversion from primarily oxidative to wholly fermentation of metabolism, mediated by yeast, fungus and mold.
9.  According to orthodox theory, viruses enter a host cell and replicate at the host’s expense.  Replication is accomplished using enzymes, which are distinct for each virus family.  For example, RNA polymerase is used by negative stranded RNA viruses degenerates positive stranded mRNA, or as reverse transcriptase is used by retroviruses to generate DNA from their RNA template and to integrate that DNA into the host genome.
It is normal for repair proteins to generate enzymes or acidic waste products as they do their work of repair.
10.  one reason suggested for viral tropism [the tendency to infect some cells, but not others] is the presence or absence of host cell receptors that allow the virus to attach.  It is said, for example, that HIV binds to the proteins [CD4] involved with antigen presentation on a helper.  The lymphocytes, that Epstein-Barr virus binds to the complement receptor [CD2] on macrophages, that rabies virus binds to the acetylcholine receptor on neurons, and that rhino viruses bind to the adhesion proteins [ICAM-1] on mucosal cells.
See answer to number 1 above.
Theoretically, once attach, the entire virion, or a portion containing the genome and essential polymerases, penetrates into the cell saddle plasma in one of three ways: [one] translocation of the entire virus across the plasma membrane; [two] receptor mediated endocytosis of the virus and fusion with endosomal membranes; or , [three] fusion of the viral envelope with the cell membrane.  Theory suggests that within the cell the virus uncoats, separating its genome from its structional components and losing its infectivity before replication.  In either the nucleus or the cytoplasma, newly synthesize viral genomes and capsid proteins are assembled into progeny virions, which may then bud to the plasma membrane.  Unencapsulated viruses may be released also, directly through the membrane.
It is interesting, however, that viruses can somehow choose the “infection.”  To be aborted, latent or persistent, meaning respectively: [one] viral infections with incomplete replication cycles; [two] persisting in the cryptic state, like herpes zoster within a dorsal root ganglion, which suddenly becomes active to produce shingles; [three continuously synthesized virions, with or without altered cell function [e.g., hepatitis B].  These three ideas, especially latency, have arisen as feeble excuses for the untenable virus theory.
11.  In order for viruses to reproduce, they must complete the following four steps:
a] Adsorption and penetration of the cell.  The viral particle binds to the host cell membrane.  This is unusually a specific interaction in which a viral encoded protein on the capsid or a glycoprotein embedded in the virion envelope binds to a host cell membrane receptor and is then internalized.  This internalization occurs by endocytosis or by fusion of the virion envelope with the host cell membrane.
This is the mechanism whereby the viral particle enters the cell for the purposes of carrying out repairs to the damaged DNA or RNA.
b) Uncoating of the virus, so that the nucleic acid can be released from the capsid into the nucleus or cytoplasm.
Repair work may require uncoating.  An uncoated “virus” in the saddle plasma, may have, from the nucleus and not yet have a code, as in the case of hepatitis B , according to medical science.  A coat is then created to protect the nucleic acid, to make a communicative or response to protein complex, or to allow exiting the cell for remote function or for neutralization and recycling by the immune system.
c) Synthesis and assembly of viral products, as well as in addition of the host cell’s own DNA, RNA and protein synthesis.
Protein complex is produced in response to an alarming acidic situation — fermentation and mycotoxic stress — are capable of self-reported replication.  As suggested by Bechamp, the microzyma is specific for each organ, therefore specific repair proteins will be needed for specific cells that make a specific organ that are being disturbed by dietary and/or metabolic acidic waste products.  There is the question of why the great numbers in some cases.  One possibility is simply over reaction; for example, fever can be extreme.  Why?  To remove dietary, metabolic acids or acids from bacteria, yeast, fungus and/or mold.
d) And finally, release of virions from the host cell either by budding or lysis.
[1] Complexes leave the cell for remote function or to be neutralize; [2] repairs have failed, and complexes are released prior to or during the breakdown of the cell by acidic toxins or the immune system.
Further Considerations
Virologists referred to certain microforms as passenger viruses, which are present in asymptomatic situations, riding on their host genetic molecule like a passenger.  To the conventional mind searching for new diseases or for viral cause of unexplained ones, they are most interesting, because the status virologist in the scientific community depends upon the pathogenic potential of the viruses they study.  Due to their location, passenger viruses are thought to have much disease potential, thus their true function goes unnoticed.  These colloidal passengers are the silent majority of animal and human intranuclear proteins essential for genetic repair.
Kalokerinos and Dettman quote Dr. Fred Klenner regarding the changeability of viruses, “I am of the opinion that virus units have the potential of going from one type to another by altering their protein coat.  We see chickenpox at Thanksgiving, mumps at Christmas, read measles in the spring, and polio and Coxsackie in the summer.”  Seasonal appearance of different forms may be mediated by variations of imbalance in the biological terrain or nutritive median due to the fermentation of dietary excesses such as sugar and animal proteins that accompany holidays and seasons, calling for different repair proteins.  For example, outbreaks of polio have been associated with sugar consumption in summer.  Various psychoemotional stresses correspond to the seasons as well.”
Supporting the general idea of dietary culpability is a statement published by the great English physician, Sir Robert McCarrison in 1936: “obsessed with the invisible microbe, virus, protozoa as all-important excite tens of disease, subservient to lavatory methods of diagnosis, hidebound by our system of nomenclature, we have to forget the most fundamental of all rules for the physician, but the right kind of food [nutrition] is the most important single factor in the promotion of health and the rhonchi to food.  The most important single factor in the promotion of disease.”
Six years before BeChamp identified the microzyma as a ferment and, with his devoted associate, Professor Estor, began a 13 year odyssey of research into its nature. Florence Nightingale published a statement about the germ theory,  In ‘Notes on Nursing’, first addition, 1860, she said of infection:
“Diseases are not individuals arranged in classes, like cats and dogs, but conditions growing out of one another.
Is it not living in a continual mistake to look upon diseases, as we do now, as separate entities, which must exist, like cats and dogs, instead of looking upon them as conditions, like a dirty and a clean condition, and just as much under our own control; or rather, as the reactions of kindly nature against the conditions in which we have placed ourselves?
I was brought out . . . . distinctly to believe that smallpox, for instance, was a thing of which there was once a first specimen in the world, which went on propagating itself in a perpetual chain of dissent, just as much as that there was a first dog, [or a first pair of dogs], and that smallpox would not begin itself anymore than a new dog would begin without there having been a parent dog.
Since then, I have seen it with my eyes and smelt it with my nose smallpox growing up in the first specimens, ear in close rooms or in overcrowded wards, where it could not by any possibility have been ‘caught’,  but must have begun.  Nay, more, I have seen diseases begin, grow up, and pass into one another . . . . I have seen, for instance, with a little overcrowding, continued fever grow up; and with a little more, typhoid fever; and when little more, typhus, and all in the same ward or hut.
Would it not be far better, truer, and more practical, if we looked upon disease in this light?  For diseases, as all experience shows, are adjectives, not noun- substantives.”
That is, symptoms [called diseases] are described first of the situation.
I find legitimate BeChamp’s conclusion that what are called germs of the air are fundamentally microzyma’s of beings, which are being consumed by the recycling process, i.e., some kind of vegetative digestion — putrefaction or fermentation.  In short, there are no pre-existing disease germ species.  The principals of microbial medicine constitute a fundamental biological ERROR!!!!!!  As BeChamp said, “the microbial doctrine is the greatest scientific silliness of this age.”  This is not to say there is no transmission, only that invasion is not necessary for symptogenesis, nor is it the primary mechanism for illness.  It is to say that for transmission to take place, susceptibility in the form of a compromised terrain must pre-exist in the receiver, who was then likely to be ill anyway.  With the exception of the immune component in the mucosal barrier, primary host “resistance” is a function of terrain condition rather than immunity per se.
Phantom Viruses
 
Hepatitis
Hepatitis can be a painful symptom that has yielded profitable virus hunting opportunities in recent years.  Although there are several categories of this disorder, three main varieties of what is called “acute viral hepatitis” exist: Type A [formally, ‘Infectious hepatitis’], Type B [formally ‘Serum hepatitis’], and hepatitis Type C (formally ‘non A, non-B’].  The corresponding viruses are HIV, HBV, and the non-A, non-B ‘group’, now called C. Type A is said to be caused by an RNA virus, spread primarily by fecal contamination of water and food, with blood and secretions also possibly being infectious [but it is due to the acidic toxins associated with unsanitary conditions].  Hepatitis B, discovered in the sixties, is said to be caused by a DNA virus, which replicates in the hepatocyte nucleus and receives its surface coat in the cytoplasma.  It is said to be transmitted by transfused blood or blood products, or via common use of needles by intravenous drug users [but it is due primarily to over-acidification from the drugs, especially heroine.  The exchange of body fluids into the blood, whether by sterilize needles, abusive sexual activity, eccentric sexual activity, etc. can also play a role overtime, because of repeated immune stress caused by foreign proteins].  Third World babies with poor nutrition and unsanitary conditions around the time of birth are also susceptible.
The third type of hepatitis, discovered in the seventies, is found among drug users and alcoholics, and accounts for 80 to 90% of hepatitis caused by blood transfusion.  It is thus akin to B type and was at first thought by scientists to be hepatitis B until thorough testing a subject revealed no virus B nor A, for that matter.  It was thus called “non-A, non-B” hepatitis and thought to be at least two viruses and perhaps more.
In 1987 scientists believed they found a single virus causing the third type, what is known today as the hepatitis C virus.  However, what they identified was an antibody, they associated with a virus.  Now, just as with HIV, they could test patients for antibodies against an elusive or invisible phantom virus.  With this new observation, however, new paradoxes confronted the viral hypothesis.  Huge numbers of people testing positive for the Phantom C virus never developed any symptoms.  Hepatitis C is truly the result of an over-acidification or toxification of the largest filter organ in the body by such substances as lactic acid, acetylaldehyde and ethanol alcohol — not the disease of a pathological phantom virus.  It is interesting to note also that all these hepatitis viruses have incubation periods of two to 25 weeks, violating Farr’s law, [see below], yet are not classified as slow viruses.  Also, the point at which a “natural invasion” takes place, as opposed to a highly artificial in objective one, and thus, how true incubation periods are determined, is another interesting question.  Bottom-line there is no Hepatitis C virus.
Hantavirus
A recent example of unwarranted panic in American bio medicine was the eminent hantavirus of 1994.  Presumably, it had jumped species, from mouse to man [the American Navajo Indians].  However, after supposedly killing a number of people, this phantom virus apparently made peace with the Indians and retired to its mouse reservoir.  The virus failed to materialize.  A front-page article in the San Francisco Chronicle reported that CDC “epidemiologist across the nation are carefully monitoring the deer mouse population and the level of virus within it.”  But all that was left to discover of the former.  “Navajo flu” by the CDC epidemiologist [shown in their space suits] were healthy mice in the mountains.  The Navajo flu is nothing new to the Native Americans and is most likely tied to sanitation, nutrition and lifestyle.
Ebola
In May 1995 , the CDC announced the new, threatening Ebola virus.  The deadly killer virus was expected to leave its hidden reservoir in the rain forest of Africa to claim Europe and the United States.  An article in Time magazine was peppered with men in space suits and color electron micrographs of the virus  [even though electron microscopes cannot take color pictures and the pictures were of parasites].  A CDC virologist suggested the virus could leave the rain forest a if “we get a virus that is both deadly to man and transmitted in the air.”  We are thus asked to fear the false image of virus somehow being launched into the air, perhaps by injection from a host, and then floating on a killer breeze to other lands.  A more imaginable scenario was suggested by European epidemiologist who heads the United Nations AIDS program.  Echoing the the CDC’s alarm, he stated, “it’s theoretically feasible.  Then infected person from Kuwait could go to Tunisia, get on a plane to New York, fall ill, and present transmission risk there.”  But within a month, the virus had disappeared in Africa, and not a single Ebola case was reported in the United States or Europe.
The World Health Organization announced on December 19, 1995 that the Ebola virus epidemic that killed 245 people in West Africa was over.  All tests on any remaining suspected cases were negative.  A somewhat unsettling revelation was that every Ebola outbreak in Africa, “is associated to have spread to public hospitals.”  As it turned out, it was associated with reused hypodermic needles in these hospitals.  Just like hantavirus, Ebola vanished, never to be heard from again, until NOW!  Most interesting is that this so-called epidemic, as epidemics will, stopped without vaccines or other drugs.  Consider the impact such stories have made upon our minds and on the way we view and understand germs.  What’s next in the virodrama, the Andromeda strain?  NO!  Here we go again with the same old phantom viral story!
There is one insidious possibility that must be mentioned in passing.  Some mysterious outbreaks of the past have shown years later to have been man-made.  In some cases, government agency have used the public to test releases of organisms and weak biochemical acidic toxins in order to verify, through medical reports, expectations of bio-warfare activity.  These incidents and the whole story of such behavior is well documented in the book, all higher forms of killing by Robert Harris and Jeremy Paxman [Hill and Wang, 1982].  In this scenario, the cause of such an incident would be constructed officially, or left as a mystery, in order to draw attention away from the truth.
Resources:
  1. To read Part 2 and Part 3 and for all references for this article read Sick and Tired by Dr. Robert O. Young – http://www.phoreveryoung.com, http://www.phmiracle.com or http://www.phmiraclebooks.com

How Many Deaths Due To Measles In The Last 10 Years – NOW The TRUTH – NONE!!!!!!!!!!!!!!!!

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Brian Shilhavy

With the measles and measles vaccine debate reaching a near frenzy on the Internet, it is always nice to throw some cold hard facts on the firestorm currently raging in the measles debate.

So here are some easily verifiable facts regarding deaths associated with measles in the United States for the past 10 years, and deaths associated with measles vaccines during the same 10 year period.

First, the Centers for Disease Control and Prevention (CDC) keeps a weekly tally of disease outbreaks, including deaths. According to a statement made by Dr. Anne Schuchat, the director of CDC’s National Center for Immunization and Respiratory Diseases, in an Associated Press story picked up by Fox News on April 25, 2014:

There have been no measles deaths reported in the U.S. since 2003

The weekly CDC Morbidity and Mortality Weekly Reports (MMWR) since that date have not revealed any measles deaths either.

And while health authorities are blaming measles outbreaks in recent years on unvaccinated children, when you mention the fact that nobody is dying from measles in the U.S., they are quick to turn around and claim vaccines have eliminated measles deaths (even though they cannot eliminate the disease itself apparently.)

Besides the obvious contradiction in reasoning with such a claim, the historical evidence just does not support it either:

Death by Measles Vaccines

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What about deaths associated with the measles vaccine during the same time period?

The U.S. Government keeps a database of reports called The Vaccine Adverse Event Reporting System (VAERS). The database is available to the public, and there is a search portal the public can use at Medalerts.org.

We ran a search for a ten year period for deaths reported with measles vaccines, including a few that are no longer in production. The search result contained 108 deaths over this period, associated with four different measles vaccines sold in the United States during the past 10 years.

VAERS-Measles-Vaccine-Deaths

Today, one can only purchase a measles vaccine in combination with the mumps and rubella vaccines (MMR Vaccine).

When searching for just the MMR vaccine during the past 10 years, 96 deaths were reported:

MMR-deaths

Anyone with a computer and Internet access can search this database by visiting MedAlerts.org.

This database reflects only deaths that were reported during the time frame, and therefore probably reflects a much lower number than actual deaths, since most doctors and health authorities believe vaccines are safe, and would not normally attribute a death to a vaccine and actually report it.

The U.S. Government Settlements on Measles Vaccine Injuries

The other place to find facts regarding injuries and deaths due to the measles vaccine is to look at U.S. Government settlements for MMR vaccine injuries and deaths. The U.S. public is largely unaware that manufacturers of vaccines have been given legal immunity from being prosecuted in civil court for vaccine injuries and deaths, since 1986. If someone is injured or killed by a vaccine, they have to sue the U.S. Government in a special “vaccine court.”

The Department of Justice issues quarterly reports on claims and settlements, and one can search for specific vaccines settlements  at the United States Federal Courts website.

As search here for “measles” returns a result of 111 claims settled for the MMR vaccine since 2004. Some of them are for settlements due to deaths related to the MMR vaccine, as determined by the judge.

Measles-vaccine-settlements

We did not click on and read each decision to find out how many resulted in deaths, but if we get a few reports from others who are willing to do so, and the numbers match, we will update this story with the actual death figure.

It takes many years to win a case in this vaccine court, so this probably represents only a tiny fraction of actual injuries and deaths due to the MMR vaccine.

Conclusion: Measles Vaccine Enthusiasm based Largely on Fear and Beliefs

We fully realize that those who believe in the value of vaccines will probably not be persuaded by these facts, which anyone with a computer and Internet access can verify from U.S. Government sources.

Having now published a few stories on the measles issue, and having received many hundreds of comments, it has become very clear to me that those who have strong opinions on the measles vaccine are based more on fear and beliefs, than on facts or science. Any attempt by these vaccine proponents to force their beliefs on the rest of the U.S. public should be vigorously opposed.

See Also:

Dr. Rowen: Measles Spread by those Vaccinated

The Truth About Measles the Mainstream Media is Suppressing

Arizona Cardiologist Responds to Critics Regarding Measles and Vaccines

UCLA Medical Center Pediatrician: Let Parents Choose on Measles Vaccine