The 39-year-old actress isn’t the only celebrity who swears by this particular approach to healthy eating: Tom and Gisele Brady, Gwyneth Paltrow, Jennifer Aniston, Jared Leto, Kirsten Dunst and Victoria Beckham are all fans of the pH Miracle Lifestyle and Diet.
So how exactly does it work? The diet is based on the theory that different types of food leave behind different types of waste — metabolic, respiratory, dietary and environmental by-products can be either acidic, alkaline or neutral.
What we eat, what we drink, what we breath, and what we think produce acidic waste products, if not eliminated via, respiration, urination, defecation and perspiration will be pushed out into the interstitial fluids of the Interstitium will suppress the immune system, and contribute to blood, bone, muscle, organ and glandular damage; conversely, an alkaline lifestyle, including diet will improve health as well as physical integrity of the blood, bone, muscles and major organs of the body, according to Robert O. Young DSc. PhD, Naturopathic Practitioner, author of the pH Miracle, Revised and Updated – https://www.amazon.com/gp/product/0446556181/ref=dbs_a_def_rwt_hsch_vapi_taft_p1_i0
By bringing your interstitial fluids of the Interstitium to an optimal alkaline pH level of 7.365, you can not only reduce the risk for sickness and disease, but also lose acidic weight stored in the fatty tissues, enhance your energy levels, strengthen your immune system and improve digestion and sleep. “That’s my go-to,” Kate has said. “It’s no dairy, wheat, meat, or sugar, and it’s gluten-free. And no wine and no beer — only vodka and tequila, straight up.”
When she sticks to the plan, she gets 60 to 80 percent of her electron intake from foods like tofu, nuts and most fruit and vegetables. She does make an exception for lean meats, “but only occasionally — animal protein has a highly acidic effect on the body and causes inflammation.”
Scientific medical studies have confirmed the pH Miracle theory behind this theory in the prevention and treatment of cancer, diabetes, heart disease, lupus, Cystic fibrosis, healthy weight loss, just to name a few (www.drrobertyoung.com), there’s certainly no harm in fostering healthy eating habits. And for Kate, there’s plenty of room for trial — and even more for error. “If I want to go out and eat at a restaurant with amazing food, I’ll do that,” she said — but she’ll try and “keep it light” beforehand. Kate’s normal day-to-day meals include lentil tacos, gluten-free pasta with veggies, steamed fish “in greaseproof paper,” almond milk-based butternut squash soup, and vegetable stir-fry over brown rice.
But to maintain her incredible figure, Kate does more than just eat right. “If I’m not doing something active, I don’t feel good,” said the Almost Famous star, who’s been practicing Pilates for 15-plus years. Apart from the core-focused routines, she does everything from Soul Cycle to Bella Core (a ballet barre class) to Tracy Anderson to the Brazil Butt Lift DVD series. At home, she has no qualms about locking herself in a room and dancing her feet off. “I’ll turn music on as loud as possible and just get weird. “It just makes me feel so much better.”
Kate, who’s mom to two boys and a girl — Ryder, 14, Bingham, 7, and Rani, 3 months — got a particularly healthy start in life. Her own mother, the still-stunning Goldie Hawn, taught her the value of both smart eating and regular exercise. But that’s not all that matters, she knows. In fact, the secret to a happy life, Goldie’s said, may have less to do with diet and more to do with quiet. Revealed the 72-year-old “glamma” — who’s meditated every day for the past 50 years — “Everyone in Hollywood seemed all screwed up and I didn’t want to be like that… I wanted life to be fuller.”
In 2016, Kate released her self-help book, Pretty Happy: Healthy Ways to Love Your Body Opens a New Window., the culmination of years of dedicated journaling that helped reveal personal patterns. “I’m finally at a place in my life where if someone wants to know what I do to stay healthy, why not tell them?” she explained. She’s also the cofounder of the trendy women’s athleisure line Fabletics, which began as an e-commerce site but now plans to open more than 100 retail locations over the next few years. “I wanted to create something that was more about a lifestyle than an actual fitness brand,” Kate said, adding proudly, “It seemed to really resonate.”
To learn more about healthy alkalizing weight loss and/or weight gain read, The pH Miracle for Weight Loss and learn how the Worlds top athletes and celebrities achieve extraordinary energy, health, fitness and longevity.
The New England quarterback’s alkaline diet has many rules to keep people on track as they attempt this new lifestyle.
Just in time for the Super Bowl!
1. Consume mainly alkaline foods – At least 80%!
2. Pay attention to portion sizes. The plate should include two palm sizes of vegetables and one-palm size of protein
3. Stop eating two to three hours before bed
4. You cannot combine protein and complex carbs for a meal
5. Cannot eat fruit alone
6. Little to no carbs for breakfast
7. Eat foods based on seasons
8. Consume 20oz of alkaline water at a pH of 9.5 right when you wake up
9. If you cannot give up coffee then limit caffeine intake to 200mg (two cups of coffee)
10. No soda, carbonation, milk, dairy, fruit juices, sweetened drinks, and alcohol!
Giving it a try! Toronto-based fitness trainer Keltie O’Connor shared with her followers how she attempted the alkaline diet!
Before the pH Alkaline diet Keltie admitted she was bloated!
After 30 days on the pH Alkaline Diet no more digestive problems and bloating!
To learn more about the pH Miracle lifestyle read The pH Miracle, revised and updated.
To order the pH Miracle Alkaline Lifestyle and Diet book go to: https://www.amazon.com/Robert-O.-Young/e/B001ILKCSU/ref=sr_tc_2_0?qid=1547305442&sr=1-2-ent
The Truth About Alkalizing Your Body Fluids
WARNING – WARNING – WARNING – WARNING
Reading the following article may disrupt just about everything you thought you understood about the anatomy, physiology and the functionality of the human body!
Read at YOUR OWN RISK!
The following article was published by Dr. Robert O. Young, April 12th, 2012, covering his work, research and discovery of the interstitial fluid of the colloidal connective tissue, the largest organ of the body and the alkaline buffering system, a new organ system. Dr. Young also shares his discovery of the true purpose ot the stomach and small intestines and his foundational theory that the human body is alkaline by design although acidic by function.
Please Read and Share this very important break-through research that pre-dates the so-called discovery in 2018 of the interstitial fluids of the interstitium as a NEW ORGAN and NEW ORGAN SYSTEM.
The interstitial fluid of the Interstitium is what I have been referring to as the interstitial fluid of the colloidal connective tissues of the schade for over 25 years.
Many of my critics called me a quack when I suggested over 25 years ago that the largest organ of the human body was NOT the skin but a new organ, undetected by current medical savants, called the interstitial fluid of the colloidal connective tissues.
This organ holds all the impurities of the blood in order to maintain its delicate pH balance at 7.365. The colloidal connective tissue holds these impurities until they can be removed by the lymphatic system via perspiration, urination or defecation.
You will also learn about another new organ system I refer to as the Alkaline Buffering System and the real functionality of the stomach!
Reading the following article may disrupt everything you learned in elementary, secondary, graduate and post graduate school. If you are a medical doctor or a naturopathic doctor you will have to unlearn just about everything you were taught at medical school!
The following is a rebuttal to Dr. Ben Kim, a conventionally trained doctor and Dr. Robert O. Young, a biochemist, nutritionist and a naturopathic practitioner.
Dr. Ben Kim states: Is it true that the foods and beverages you consume cause your blood to become more alkaline or acidic? Contrary to popular hype, the answer is: not to any significant degree.
Dr. Robert O. Young states: The pH of blood and interstitial fluids are constantly being challenged with environmental, dietary, respiratory, and metabolic acids. The body deals with blood acids by eliminating these acids through the four channels of elimination (urination, perspiration, defecation and respiration) and the buffering of acids through the alkaline buffering system in order to maintain the delicate pH balance of the blood plasma and interstitial fluids at 7.365.
Dr. Kim Ben states: The pH of your blood is tightly regulated by a complex system of buffers that are continuously at work to maintain a range of 7.35 to 7.45, which is slightly more alkaline than pure water.
Dr. Robert O. Young states: The pH of your extracellular fluids, which includes the blood plasma and the interstitial fluids are kept at a very narrow range at 7.365 to 7.385. Any pH measurement of blood plasma in excess of 7.385 indicates a condition of compensated acidosis and any pH measurement of blood plasma or interstitial fluids below 7.365 indicates a condition of decompensated acidosis.
When the pH of the blood plasma increases above the 7.385 this is the result of the blood pushing environmental, dietary, respiratory and metabolic acids out into the interstitial fluids of the colloidal connective tissues of the schade as the blood is pulling alkaline mineral salts such as calcium ions from the bones or magnesium ions from the muscles to offset the increase of acids in the blood. It is always a sure sign that as the blood plasma is becoming more alkaline the interstitial fluids of the colloidal connective tissues of the schade are becoming more acidic and this is the cause of ALL inflammatory and degenerative diseases.
When the body tissues, organs, cells, or the alkaline reserves (sodium, calcium, magnesium and potassium) become deficient in alkaline minerals the blood plasma pH will drop below the ideal 7.365 causing decompensated acidosis leading to hemolysis at a pH below 7.365 or a coma and/or death at a pH below 7.2.
Dr. Kim Ben states: If the pH of your blood falls below 7.35, the result is a condition called acidosis, a state that leads to central nervous system depression. Severe acidosis – where blood pH falls below 7.00 – can lead to a coma and even death.
Dr. Robert O. Young states: If pH of the blood plasma drops below 7.365 the result is called decompensated acidosis. If the pH of the blood plasma stays at the ideal 7.365 this is called compensated acidosis. And if the pH of the blood plasma increases above the 7.365 this is called “latent tissue acidosis” in the interstitial fluids of the colloidal connective tissues of the schade.
The blood plasma pH always goes alkaline when the blood pushes out environmental, dietary, respiratory or metabolic acids out into the interstitial fluids of the colloidal connective tissues of the schade. These acids are always deposited into what I call the ‘acid catchers’ which are the connective tissues of the schade. This leads to what I call ‘latent tissue acidosis’.
As acids build-up in the colloidal connective tissues of the schade and if NOT eliminated by the lymphatic system and the out through the channels of elimination, including the skin, lungs, bowels or urine this will result in ALL the connective tissue diseases and degenerative diseases, including ALL cancerous conditions.
Dr. Kim Ben states: If the pH of your blood rises above 7.45, the result is alkalosis. Severe alkalosis can also lead to death, but through a different mechanism; alkalosis causes all of the nerves in your body to become hypersensitive and over-excitable, often resulting in muscle spasms, nervousness, and convulsions; it’s usually the convulsions that cause death in severe cases.
Dr. Robert O. Young states: If the blood plasma pH increases over 7.385 you are in a state of ‘latent tissue acidosis’ of the interstitial fluids on the colloidal connective tissue of the shade and a high risk for cancer. This is what I call the “tee-ter-totter effect”. The body is pulling alkaline minerals into the blood to compensate for an equal amount of acids being pushed out into the interstitial fluids of the connective tissues of the schade to keep the blood plasma in an alkaline state. The result is tissue acidosis which then leads to hypersensitivity and over-excitable nerves, muscle spasms, nervousness and convulsions that can lead to coma or even death. All of these symptoms are NOT a result of too much base or alkalinity but the result of too much acid from the blood being deposited into the interstitial fluids of the colloidal connective tissues of the schade. The cause of ‘latent tissue acidosis’ is caused by an acidic lifestyle and dietary choices.
Dr. Kim Ben states: The bottom line is that if you’re breathing and going about your daily activities, your body is doing an adequate job of keeping your blood pH somewhere between 7.35 to 7.45, and the foods that you are eating are not causing any wild deviations of your blood pH.
Dr. Robert O. Young: The bottom line is when you understand that having an acidic lifestyle and diet does affect the blood plasma and interstitial fluid pH in a negative way.
The blood responds to increased acids from lifestyle and diet by pushing them out into the interstitial fluids of the connective tissues or the colloidal connective tissues of the schade. The foods you eat, the liquids you drink, the air you breath, even your thoughts will effect the pH of blood and then the interstitial fluids of the colloidal connective tissues. The blood is constantly responding to the acidic wastes of lifestyle and diet choices!!!!!!!!!!!
Dr. Kim Ben stated: So what’s up with all the hype about the need to alkalize your body? And what’s to be made of the claim that being too acidic can cause osteoporosis, kidney stones, and a number of other undesirable health challenges?
Dr. Robert O. Young states: The hype about alkalizing your blood and then interstitial fluids of the connective tissues is important because the human body is alkaline by design and acidic by function. This is the foundation for understanding the true cause of ALL sickness and disease.
Dr. Kim Ben states: As usual, the answers to such questions about human health can be found in understanding basic principles of human physiology. So let’s take a look at the fundamentals of pH and how your body regulates the acid-alkaline balance of its fluids on a moment-to-moment basis.
Dr. Robert O. Young states: The problems with current understanding of the basic principals of human physiology is the basic principals do NOT understand that the human body is alkaline by design and acidic by function. Current medical savants DO NOT understand that there is only one health, one sickness, one disease and one treatment. The one health is to maintain the alkaline design of the blood and interstitial fluids of the connective tissues with an alkaline lifestyle and diet. The one sickness and one disease is the over-acidification of the blood and then tissues due to an inverted way of living, eating and thinking. The one treatment is to restore the alkaline design of the body fluids with an alkaline lifestyle and diet. Remember the fish bowl metaphor? It goes like this – When the fish is sick what would you do? Treat the fish or change the water? Remember the fish is only as healthy as the water it swims in.
Dr. Kim Ben states: pH is a measure of how acidic or alkaline a liquid is. With respect to your health, the liquids involved are your body fluids, which can be categorized into two main groups:
1. Intracellular fluid, is the fluid found in all of your cells. Intracellular fluid is often called cytosol, and makes up about two-thirds of the total amount of fluid in your body.
2. Extracellular fluid, is the fluid found outside of your cells. Extracellular fluids are further classified as one of two types:
Plasma, which is fluid that makes up your blood.
Interstitial fluid, which occupies all of the spaces that surround your tissues. Interstitial fluid includes the fluids found in your eyes, lymphatic system, joints, nervous system, and between the protective membranes that surround your cardiovascular, respiratory, and abdominal cavities.
Your blood (plasma) needs to maintain a pH of 7.35 to 7.45 for your cells to function properly. Why your cells require your blood to maintain a pH in this range to stay healthy is beyond the scope of this article, but the most important reason is that all of the proteins that work in your body have to maintain a specific geometric shape to function, and the three-dimensional shapes of the proteins in your body are affected by the tiniest changes in the pH of your body fluids.
Dr. Robert O. Young states: pH is a measurement of the concentrations of hydrogen and hydroxyl ions in a aqueous solution. Any aqueous solutions, including your blood plasma interstitial fluids of the colloidal connective tissues of the schade (the largest organ of the human body) and the intracellular fluids that are saturated in hydrogen ions is less or more acidic and any aqueous solution that is saturated in hydroxyl ions is less or more base or alkaline.
Once again, the human body is alkaline in its design and acidic in its function. Your blood plasma and interstitial fluids of the colloidal connective tissue of the schade needs to be maintained at a delicate pH of 7.365 for your cells to function properly.
I have found in my own blood research that when your pH is stable at 7.365 you find healthy blood which is even in color, even in size and even in shape. The red blood cell is the primary stem cell which becomes all other body cells. And the health of the blood and the interstitial fluid of the colloidal connective tissues of the schade is directly connected to the health of all body cells. It is blood that becomes, liver, heart, brain and skin cells. All red blood cells and then body cells are made up of microzymas.
Microzymas are the foundational, indestructible matter and intelligent matter that makes up all living cells, including the DNA. The tiniest changes in the pH of the body fluids can cause the microzymas in the red blood cells or body cells to change into bacteria, yeast and/or mold. This is how germs are created – from within NOT from without.
Dr. Kim Ben states: The pH scale ranges from 0 to 14. A liquid that has a pH of 7 is considered to be neutral (pure water is generally considered to have a neutral pH). Fluids that have a pH below 7 – like lemon juice and coffee – are considered to be acidic. And fluids that have a pH above 7 – like human blood and milk of magnesia – are considered to be alkaline.
Dr. Robert O. Young states: The pH scale ranges from 0 to 14 with the pH of 7 being the midpoint (pure water may have a pH of 7 but I have found that the pH of pure water has a range of 6.2 to 7.2 – a 10 times exponential swing. I have also found that pure water has an oxidative reduction potential in a range of +50mV to +150mV) which will drain energy from the body. Lemon is an alkaline fruit not an acidic fruit. This is because of its low sugar and high alkaline mineral content of potassium bicarbonate. Lemons do not draw down on the alkaline buffering system and contributes in excess of 10 times in hydroxyl ions (OH-) in relationship to its hydrogen ion content.
Dr. Kim Ben states: It’s important to note that on the pH scale, each number represents a tenfold difference from adjacent numbers; in other words, a liquid that has a pH of 6 is ten times more acidic than a liquid that has a pH of 7, and a liquid with a pH of 5 is one hundred times more acidic than pure water. Most carbonated soft drinks (pop) have a pH of about 3, making them about ten thousand times more acidic than pure water. Please remember this the next time you think about drinking a can of pop.
When you ingest foods and liquids, the end products of digestion and assimilation of nutrients often results in an acid or alkaline-forming effect – the end products are sometimes called acid ash or alkaline ash.
Dr. Robert O. Young states: All food and drink which has a pH of less than 8.4 will cause the production of sodium bicarbonate by the stomach and the release of this sodium bicarbonate via the salivary glands, the pylorus glands, the pancreas, gall bladder and intestinal glands to alkalize whatever ingested. The main purpose of stomach is to prepare the food in a liquid state at a pH of 8.4 for biological transformation into stem cells which takes place in the crypts of the small intestines.
Dr. Kim Ben states: Also, as your cells produce energy on a continual basis, a number of different acids are formed and released into your body fluids. These acids – generated by your everyday metabolic activities – are unavoidable; as long as your body has to generate energy to survive, it will produce a continuous supply of acids.
Dr. Robert O. Young states: Metabolism produces acidic waste products of lactic, uric, citric and glucose if not eliminated will cause dis-ease and then disease. You are only as healthy as the alkaline fluids of the body which includes the extracellular and intracellular fluids.
Dr. Kim Ben states: So there are two main forces at work on a daily basis that can disrupt the pH of your body fluids – these forces are the acid or alkaline-forming effects of foods and liquids that you ingest, and the acids that you generate through regular metabolic activities. Fortunately, your body has three major mechanisms at work at all times to prevent these forces from shifting the pH of your blood outside of the 7.35 to 7.45 range.
Dr. Robert O. Young states: There are seven main sources at work on a daily basis that can disrupt the pH of your body fluids – these forces include acids from the external environment, acids from the foods and liquids ingested, acids from the air you breath, acids from metabolism, acids from cells breaking down or catabolic activity, acids from endogenous bacteria, yeast and mold, and acids from respiration. Your body has an elaborate alkalizing buffering system at work at all times to help prevent through chelation these forces from shifting the pH of the blood plasma pH as well as the interstitial fluid pH at a delicate pH of 7.365. It is important to note that when the alkaline buffering system (a new organ discovered by Dr. Robert O. Young) becomes depleted and acids are being deposited into the interstitial fluids of the colloidal connective tissues and the fatty tissues this is when dis-ease and eventual disease manifests.
Dr. Kim Ben states: These mechanisms are:
- Buffer Systems
Carbonic Acid-Bicarbonate Buffer System
Protein Buffer System
Phosphate Buffer System
Exhalation of Carbon Dioxide
Elimination of Hydrogen Ions via Kidneys
Dr. Kim Ben states: When people encourage you to “alkalize your blood,” most of them mean that you should eat plenty of foods that have an alkaline-forming effect on your system. The reason for making this suggestion is that the vast majority of highly processed foods – like white flour products and white sugar – have an acid-forming effect on your system, and if you spend years eating a poor diet that is mainly acid-forming, you will overwork some of the buffering systems mentioned above to a point where you could create undesirable changes in your health.
Dr. Robert O. Young states; Everything you eat, everything you drink, everything you breath, everything you think and everything you do affects the blood and interstitial fluids in an acidic way to a lesser or greater degree. That is why we age. We do not get old we mold from years of acidic lifestyle and dietary choices. The key to a healthy life or a life of sickness and disease and then eventual death is in the blood! Especially the blood plasma.
Dr. Kim Ben states: For example, your phosphate buffer system uses different phosphate ions in your body to neutralize strong acids and bases. About 85% of the phosphate ions that are used in your phosphate buffer system comes from calcium phosphate salts, which are structural components of your bones and teeth. If your body fluids are regularly exposed to large quantities of acid-forming foods and liquids, your body will draw upon its calcium phosphate reserves to supply your phosphate buffer system to neutralize the acid-forming effects of your diet. Over time, this may lead to structural weakness in your bones and teeth.
Dr. Robert O. Young states: Dr Kim Ben has described the cause of bone loss correctly. It is important to remember that the activation of the pHosphate buffering system does not happen when you are on an alkalizing lifestyle and diet and hyper-perfusing the blood and interstitial fluids of the colloidal connective tissues with alkalinity. The alkaline lifestyle and diet is outlined in my book, The pH Miracle Revised and Updated.
Drawing on your calcium phosphate reserves at a high rate can also increase the amount of calcium that is eliminated via your genito-urinary system, which is why a predominantly acid-forming diet can increase your risk of developing calcium-rich kidney stones.
Dr. Ken Ben states: This is just one example of how your buffering systems can be overtaxed to a point where you experience negative health consequences. Since your buffering systems have to work all the time anyway to neutralize the acids that are formed from everyday metabolic activities, it’s in your best interest to follow a diet that doesn’t create unnecessary work for your buffering systems.
Dr. Robert O. Young states: The protocol that will NOT unnecessarily activate the alkaline buffering systems of the body is outlined in the pH Miracle Revised and Updated by Dr. Robert O. Young.
Dr. Kim Ben states: Generally speaking, most vegetables and fruit have an alkaline-forming effect on your body fluids.
Dr. Robert O. Young states: Generally, all green fruit and vegetables are the ONLY alkalizing foods for the blood and interstitial fluids of the colloidal connective tissues and are critical in building healthy blood and then healthy body cells.
Dr. Kim Ben states: Most grains, animal foods, and highly processed foods have an acid-forming effect on your body fluids.
Dr. Robert O. Young states; All grains, animal foods, dairy products, fermented foods, algae, probiotics, enzymes, high sugar fruit, high sugar vegetables, vinegar, corn, nuts, mushrooms, alcohol, carbonate drinks, sport drinks and tobacco products are acidic to the blood and interstitial fluids of the colloidal connective tissues of the schade (the largest organ of the body) and will activate the alkaline buffering systems.
Dr. Kim Ben states: Your health is best served by a good mix of nutrient-dense, alkaline and acid-forming foods; ideally, you want to eat more alkaline-forming foods than acid-forming foods to have the net acid and alkaline-forming effects of your diet match the slightly alkaline pH of your blood.
Dr. Robert O. Young states: The only way to achieve extraordinary health and fitness is with the pH Miracle Lifestyle and Diet as outlined in The pH Miracle Revised and Updated book.
Dr. Kim Ben states: The following lists indicate which common foods have an alkaline-forming effect on your body fluids, and which ones result in acid ash formation when they are digested and assimilated into your system.
Please note that these lists of acid and alkaline-forming foods are not comprehensive, nor are they meant to be.
If you’re eating mainly grains, flour products, animal foods, and washing these foods down with coffee, soda, and milk, you will almost certainly improve your health by replacing some of your food and beverage choices with fresh vegetables and fruits.
Dr. Robert O. Young states: The four alkalizing food groups are chlorophyll from green fruit and green vegetables, mono and polyunsaturated oils, alkalizing water and finally alkalizing mineral salts. I call this the COWS Plan as outlined in The pH Miracle Revised and Updated book.
Dr. Kim Ben states: The primary purpose of this article is to offer information that explains why I believe that you don’t need to take one or more nutritional supplements or “alkalized water” for the sole purpose of alkalizing your body. Your body is already designed to keep the pH of your body fluids in a tight, slightly alkaline range.
Dr. Robert O. Young states: Your body is alkaline by design but acidic by function and that is why you would be wise to follow an alkaline lifestyle and diet to prevent ALL sickness and disease and remain strong, healthy and fit. Drinking alkaline water is essential to maintaining the healthy alkaline state of ALL your body fluids!!!!!!
Dr. Kim Ben states: The ideal scenario is to make fresh vegetables and fruits the centerpieces of your diet, and to eat small amounts of any other nutrient-dense foods that your appetite calls for and that experience shows your body can tolerate.
Dr. Robert O. Young states: The ideal scenario is to make fresh organic electron-rich green alkalizing fruit and vegetables the centerpiece of your diet with liberal amounts of alkalizing polyunsaturated oils, alkalizing water at a pH of 9.5 and finally alkalizing mineral salts of sodium, potassium, magnesium and calcium.
Dr. Kim Ben states: I hope these thoughts bring some clarity to this often misunderstood health topic.
Dr. Robert O. Young states: I hope these scientific truths brings some clarity to Dr. Kim Ben and others that are confused about the biochemistry, bioenergetics and the importance of alkalizing the blood and interstitial fluids of the colloidal connective tissues of the schade (the largest organ of the human body) with an alkaline lifestyle and diet as outlined in my book, The pH Miracle, revised and updated.
For additional information read The pH Miracle Revised and Updated by Dr. Robert O. Young . You can also go to his personal website at: http://www.drrobertyoung.com
How To Determine the pH of the Interstitial Fluids
YOU ARE THE WORLDS LEADING STAR! AN EXPERT ON YOU! ⭐️
December 30, 2018
If you believe this, you’re in the driver’s seat. Your life, your health, your everything, is yours to create.
But somewhere in the last 100 years or so, we’ve been taken out of the driver’s seat when it comes to our own health.
We’ve been brainwashed, drugged, numbed and controlled!!
We have become so confused about who to listen to and who to believe!!!
Science has advanced beyond what we can see or feel. We’ve been convinced that we’re not expert enough in our selves to be in that driver’s seat. We’ve handed control to what we hope are smarter, more capable hands. Medical doctors, specialists etc.
People are still dying of heart disease, cancer and diabetes.., devastating, horrendous and crazy when you are aware.
We’re in a HEALTH and MORAL crisis!
Our medical system is exceptionally good at handling trauma care. We’re getting better every day at repairing damaged tissue throughout our body. Emergency care is consistently miraculous for accidents and injuries….
But for most people, every day, health or sick care isn’t helping those who are really sick and suffering!!
More and more of us ARE turning to alternative medicine, naturopaths, iridologists, Chinese herbalists.. why?.. because traditional conventional medicine is NOT working.
It’s become almost normal to have chronic pain and disease! And if not now you are going to some time in your future! That’s what we are told!
It’s become normal to take multiple chemical drugs that treat symptoms like antibiotics, and not treat the causes, and they create their own cascade of additional health dilemmas call side-effects!
It’s almost normal to be obese etc! Go to Disneyland Parks and you will see what I mean.
WE can’t afford to live this way!
The government WANTS us to live this way believe it or not! Its become a big fat money making machine! Everyday we are poisoned by almost everything we consume, ingest, and breath!
As much as we’re getting use to it as our new normal, nobody wants to live this way.
It doesn’t feel good!
When we’re not healthy, it’s so much harder to feel, because so much is painful.
So we tune out of feeling.
Without feeling, we lose sight of choice. Without choice, we don’t create what we want. We create what we don’t want.
And that, that’s right this is exactly how they want it to be in this matrix system.
All under their control – sick, tired and then dead!
I want you to be healthy!
Live the best human experience you possibly can!
Not just free from diagnosed disease, but really radiantly healthy.
Healthy feels good!
Feeling good, we get to tune into feeling rather than tune out of it. Tuned in, we get to see what’s possible, and respond to that.
We can FEEL what’s wrong with us.
Our bodies are designed to be able to TELL us and SHOW us!
We just have to cut out the poisons in order to FEEL!
Remove the drugs, the alcohol, the chemicals, pesticides, and clear out our bodies, minds and brains! OUR BLOOD!!!
The first step IS to become ALKALINE!
Yes it’s that simple!
Yes I’m a waking example of the Alkaline lifestyle and diet!
As are many others, some very high profile celebrities have also become enlightened!
This is how you feel, how you see, and how you know what’s wrong with you!
This is how you stop disease in the first place!
According to Dr. Robert O. Young, “THE BLOOD NEVER LIES!”
Many studies show this.
Refer to Robert O Young, CPT, MSc, DSc. PhD, Naturopathic Practitioner at: http://www.drrobertyoung.com and read his book “The Ph Miracle” revised & updated!
We all desire to live inspiringly capable, creative happy lives!
So slow down, take time out.
Tune in. Feel, believe what you feel, and respond to that.
Breathe and move.
Move everything you’ve got in every direction you can, and do it with ease.
No struggle, let the stress go. Let your body shift from defense mode into healing mode.
Start looking after YOU now.
There are amazing souls out that that care, and are experienced in years of research. Robert O Young is one of them! – http://www.drrobertyoung.com
Yes it’s my job to physically train, counsel, and heal someone, but I can assure you I do what I do from passion and empathy.
As a survivor of DV I volunteer at women’s shelters and offer them everything I offer my clients. Everyone deserves a chance to live healthy.
With the New Year approaching maybe you should consider manifesting a few healthy resolutions for yourself!
There are a few spots still avail in our “Enhance Your Life Program” 💚
It’s about time you got really, fully, into you. 💋💋🦅
Namaste ~Leeza Cooper
(You can contact Leeza Copper and begin “Enhancing YOUR LIfe!” at: https://www.facebook.com/leeza.cooper.31?__tn__=%2CdlC-R-R&eid=ARCcG4oMvpaaocp9ZDr_tMAxOGGHrnnYD8ARYFfn0Ki8R_MyMNLq1AGFe9NpEym2re4O7J_qhiETXQ2K&hc_ref=ARTUwt2iutC64zxl_bG2PaO8sOQrrM1xvJBLS9LGCBhRS5btPLl2-2YFF3ZCBAHLLz0
Which brings me to Dr. Dwight Lundell. He’s an experienced heart surgeon and retired Chief of Staff and Chief of Surgery at Banner Heart Hospital in Mesa, Arizona. Not so long ago, Dr. Lundell made the following statement of confession:
“We physicians with all our training, knowledge and authority often acquire a rather large ego that tends to make it difficult to admit we are wrong. So, here it is. I freely admit to being wrong. As a heart surgeon with 25 years experience, having performed over 5,000 open-heart surgeries, today is my day to right the wrong with medical and scientific fact.
I trained for many years with other prominent physicians labeled “opinion makers.” Bombarded with scientific literature, continually attending education seminars, we opinion makers insisted heart disease resulted from the simple fact of elevated blood cholesterol. The only accepted therapy was prescribing medications to lower cholesterol and a diet that severely restricted fat intake. The latter of course we insisted would lower cholesterol and heart disease. Deviations from these recommendations were considered heresy and could quite possibly result in malpractice. It Is Not Working!
These recommendations are no longer scientifically or morally defensible.”
Many doctors are highly admirable people, but they are still human beings. They all make mistakes, they all learn from them, but the really good ones are willing to admit to them.
Cholesterol does not cause heart disease and trying to reduce it with statin drugs is a waste of time, an international group of experts has claimed.
Not surprisingly, Lundell’s statement regarding the medical establishment’s approach to treating heart disease caused a ripple in the medical industry. It challenged the validity of statins – commonly known as cholesterol-lowering medications – such as Lipitor, Crestor, Zocor, and others.
The reason Lundell’s statement created such a buzz is because statins are big business. In the United States alone, about 25% of the population takes statin medications. They cost from as little as $53 per month to more than $600. Pfizer’s Lipitor went on sale in 1997 and its lifetime sales have surpassed $125 billion. AstraZeneca’s Crestor was the top-selling statin in 2013, generating $5.2 billion in revenue that year alone. The statin industry is estimated at around $30 billion in sales per year. Nevertheless, in the United States, more die each year of heart disease than ever before.
Lundell went on to say:
“The discovery a few years ago that inflammation in the artery wall is the real cause of heart disease is slowly leading to a paradigm shift in how heart disease and other chronic ailments will be treated. The long-established dietary recommendations have created epidemics of obesity and diabetes, the consequences of which dwarf any historical plague in terms of mortality, human suffering and dire economic consequences.
I have peered inside thousands upon thousands of arteries. A diseased artery looks as if someone took a brush and scrubbed repeatedly against its wall. Several times a day, every day, the foods we eat create small injuries compounding into more injuries, causing the body to respond continuously and appropriately with inflammation. While we savor the tantalizing taste of a sweet roll, our bodies respond alarmingly as if a foreign invader arrived declaring war. Foods loaded with sugars and simple carbohydrates, or processed with omega-6 oils for long shelf life have been the mainstay of the American diet for six decades. These foods have been slowly poisoning everyone.”
Listen to Dr. Tom Sladic, MD has he explains his understanding of the true cause of Heart Disease:
So What is the True Cause of Heart Disease?
A review of research involving nearly 70,000 people found there was no link between what has traditionally been considered “bad” LDL cholesterol and the premature deaths of over 60-year-olds from cardiovascular disease.
Published in the BMJ Open journal, the new study found that 92 percent of people with a high cholesterol level lived longer. (BMJ Open. Published online June 12 2016)
The authors have called for a re-evaluation of the guidelines for the prevention of cardiovascular disease and atherosclerosis, a hardening and narrowing of the arteries, because “the benefits from statin treatment have been exaggerated”.
High cholesterol is commonly caused by an unhealthy acidic lifestyle and diet, and eating high levels of processed fat in particular, as well as smoking.
It is carried in the blood attached to proteins called lipoproteins and has been traditionally linked to cardiovascular diseases such as coronary heart disease, stroke, peripheral arterial disease and aortic disease.
Co-author of the study Dr Malcolm Kendrick, an intermediate care GP, acknowledged the findings would cause controversy but defended them as “robust” and “thoroughly reviewed”. “What we found in our detailed systematic review was that older people with high LDL (low-density lipoprotein) levels, the so-called “bad” cholesterol, lived longer and had less heart disease.”
Vascular and endovascular surgery expert Professor Sherif Sultan from the University of Ireland, who also worked on the study, said cholesterol is one of the “most vital” molecules in the body and prevents infection, cancer, muscle pain and other conditions in elderly people. He also stated, “lowering cholesterol with medications is a total waste of time and money”.
“Lowering cholesterol with medications for primary cardiovascular prevention in those aged over 60 is a total waste of time and resources, whereas altering your lifestyle is the single most important way to achieve a good quality of life,” he said.
Lead author Dr Uffe Ravnskov, a former associate professor of renal medicine at Lund University in Sweden, said there was “no reason” to lower high-LDL-cholesterol.
Heart Disease and Cholesterol
The graph below shows the famous 10 year Framingham correlation study between cholesterol and coronary heart disease, published in the Lancet in 1986, that big Pharma relies on and sold to the American public at large.
The problem though, as you see in the next graph, after 20 years the correlation shows that high cholesterol saves lives and low cholesterol is a risk factor for heart disease!
Everyone in modern society has heard about cholesterol, and how bad it is. Most do not understand why it exists, and simply see it as a menace that must be eliminated as quickly as possible. This misunderstanding is exactly what the pharmaceutical complex promotes, because it allows them to perpetually treat high cholesterol with drugs like Lipitor. These drugs are prescribed for the remainder of a patient’s lifetime, and when he/she eventually dies of a “thought attack”, family and friends will believe that the disaster was inevitable from “high cholesterol”. The death will not be attributed to other health factors or to the drugs themselves, but to the “high cholesterol”; even though there are no known deaths from cholesterol in human history. It is all very convenient for the drug companies, so long as we do not examine what is up the other sleeve.
I am reminded of restless leg syndrome, whereby the dis-ease was ‘discovered’ immediately after the pharmaceutical for it was patented, as a reason to sell us this useless pharmaceutical drug. Now, restless leg syndrome has been upgraded to a new “disease”. The cause of restless leg syndrome is also the cause of heart disease – retained metabolic and/or dietary acids in the connective and fatty tissues leading to inflammation, induration, ulceration, degeneration and finally death
“Before 1920, coronary heart disease was rare in America — so rare that when a young internist named Paul Dudley White introduced the German Electrocardiograph to his colleagues at Harvard University, they advised him to concentrate on a more profitable branch of medicine. The new machine revealed the presence of arterial blockages, thus permitting early diagnosis of coronary heart disease. But in those days, clogged arteries were a medical rarity, and White had to search for patients who could benefit from his new technology. During the next forty years, however, the incidence of coronary heart disease rose dramatically, so much so that by the mid 1950’s, heart disease was the leading cause of death among Americans.”
— Mary Enig, Ph.D.
The amount of cholesterol that you eat actually has very little relationship with the amount that you have in your blood. When you eat more cholesterol, your body produces less, and when you eat less cholesterol, your body produces more. Another way to say this is like this – when you have more metabolic or dietary acid in your blood and interstitial fluids the body produces more LDL cholesterol, and when you have less metabolic or dietary acid in your blood and interstitial fluids the body produces less cholesterol. Why? Because LDL cholesterol is a buffer or chelator of metabolic and/or dietary waste. Understand? A body usually produces between three and four times the cholesterol that one eats. The amount produced is generally related to how much is needed. Cholesterol is indeed needed and critical for optimal health. The purpose of so-called “bad cholesterol” is not to give us heart attacks, but to buffer acidic metabolic and dietary waste and to repair the damage to arteries or veins from our acidic lifestyles and diets.
Whenever a poor acidic diet and lifestyle leads to damaged arteries, a thick and sticky substance is required to patch them. That substance is known as LDL or “bad cholesterol”. When this damaging behavior is continued, multiple patches are created, leading to what we know as “clogged arteries”. The problem is not the cholesterol, which is doing its wonderful job of preventing our death from internal bleeding. The problem is the fact that the arteries or veins are damaged enough from acidic lifestyle and dietary choices to risk internal bleeding. Blocking a body’s healthy countermeasure only leads to worse problems. It is the pharmaceutical standard of symptom suppression that is like hiding the timer of a time bomb, and then expecting it not to eventually go off. Thus, that so-called “BAD” cholesterol is not “BAD” at all. In fact LDL cholesterol is saving your acidic body from internal bleeding and inevitable death. LDL cholesterol ONLY increases in the presence of excess metabolic, dietary, respiratory and/or environmental acids which increase as a result of what you eat, what you drink and what you think. High LDL cholesterol is a warning sign of your poor acidic lifestyle and dietary choices and the body is in preservation mode. It is trying to protect itself from YOU!
Cholesterol is created to save your life! The following picture is what solidified metabolic acid bound cholesterol looks like in the blood.
Modern medicine spends a lot of time fighting this pitch, instead of the actual causes of arterial damage. Thus, it is not surprising that cholesterol-lowering drugs cause more heart dis-ease and more heart attacks and strokes. A massive portion of the elderly population is taking cholesterol-lowering drugs, even though research shows that the higher their cholesterol levels (especially LDL) the longer that they will live and the less risk for a heart attack or stroke. The graph below illustrates this point! Low cholesterol in the elderly is actually a sign that something is seriously wrong, and a heart attack or stroke may be imminent. Modern medicine has only recently come to accept that at least some cholesterol (LDL and HDL) is good and protective! But when you mention (LDL) cholesterol as “GOOD” you better take cover from current medical savants who will attack you with their ignorance!
Cholesterol is still suppressed with drugs, despite what science would make prudent from the long-term Framingham Study. It also has been proven that these drugs cause high suicide rates. The drugs can lead to personality changes, in a manner similar to (but not as intense as) S.S.R.I. antidepressants.
The anti-cholesterol hysteria began in the 1950’s, when researcher Ancel Keys proposed the Lipid Hypothesis. It stated that cholesterol and saturated fats lead to heart disease. His beliefs were promoted heavily by the new hydrogenated oils industry, which spent obscene amounts of money to convince every one of Keys’ indisputable findings. This successful marketing campaign was on par with similar marketing for fluoride at about the same time. Studies which had oppositional findings to Keys’ were ignored or maligned. As a result of his flawed scientific methodology (subjective cherry picking results to match what he wanted to find) saturated fats like butter and eggs were used less, in exchange for the poisonous trans-fats that are in hydrogenated oils. Heart disease rates have been rising exponentially ever-since.
The French eat more fats than any other group in the world, yet they have lower rates of heart disease. The Japanese eat more fats than Americans, yet have lower rates of heart disease. There are plenty of countries with similar patterns. The French lifestyle especially counters Keys’ hypothesis, and it also provides evidence that resveratrol (found in red or purple grapes) improves heart health. Resveratrol has been shown to reverse atherosclerosis (hardening of the arteries). Maybe, just maybe its being American that causes higher rates in heart attacks. The bottom-line medical research is subjective NOT objective!
Just recently the Food and Drug Administration issued new safety warnings about a popular class of drugs used to control and lower cholesterol levels. The FDA says the drugs, known as statins, can cause several side effects, including cognitive problems such as memory lapses and confusion. But the agency is stressing that the side effects appear to be rare and not serious. I have suggested that taking any drug, like statin drugs that lowers LDL cholesterol without removing acidic lifestyle and dietary choices is a risk for heart attack, stroke and other dis-eases like diabetes. I have lowered cholesterol successfully in all cases of hyper-chlolesterolemia without drugs by just changing the diet and lifestyle to an alkaline pH Miracle lifestyle and diet that restores the alkaline design of the body.
One of my research clients Maren Hale was diagnosed with familial hypercholesterolemia and hyper-triglycerides with LDL’s over 400 mg/dl and triglycerides over 200 mg/dl. She was also overweight. Over a period of four years Maren lost over 70 pounds and lowered her cholesterol and triglycerides to healthy normal ranges on the pH Miracle Lifestyle and Diet. Maren and her family and extended family have been a research study of the University of Utah for familial hypercholesterolemia for over 60 years. Maren was the first of all family members to lower her cholesterol and triglycerides to normal ranges due to her commitment to living a pH Miracle Lifestyle and Diet.
High cholesterol levels should be a warning to most people who inflammation caused by metabolic and dietary acid is present. It is a risk marker, and a symptom that can save your life! Eliminating the LDL cholesterol through drugs is the equivalent to eliminating the thermometer in a room that is too hot. It is illogical, and it does nothing to eliminate the dangerous cause of the symptom being expressed.
LDL cholesterol levels naturally drop whenever the body’s becomes less acidic and more alkaline in the interstitial fluids where acids are stored! And LDL cholesterol should never be forced lower with drugs because they WILL cause a heart attack or stroke! The pH Miracle alkaline lifestyle and diet can reduce LDL cholesterol, but it is never because of a lowered cholesterol intake.
The natural drop in cholesterol and triglycerides happens only when a person stops eating toxic acidic foods, drinking toxic acidic drinks and stops toxic acidic thoughts that produce toxic acidic waste products that destroy the arteries and veins!
Do YOU Understand?
Because healthy arteries and veins do not need patching. Remember that a body typically produces 3-4 times the amount of LDL cholesterol than consumed. The fats that a person eats are therefore comparatively insignificant. Cholesterol will rise whenever the body’s need for cholesterol rises and in direct relationship to the level of acidic thoughts, words and deeds. So acidic trans-fats and inflammatory acidic substances are what need to be avoided. These toxic acidic wastes are what damage the arteries and veins, and a body will be required to do a great deal of patching as a consequence. I will reference to alkalizing or chelating herbs and minerals that lower cholesterol levels naturally later, but alkalizing and chelating herbs and minerals do it by lowering the body’s need for LDL cholesterol, not by forcefully lowering it like pharmaceuticals do.
Studies on the link between cholesterol and heart health have been manipulated for decades. The first studies on eggs showed elevated cholesterol levels because they had used dehydrated eggs, and studies of coconut oil yielded similar results because they had used partially hydrogenated coconut oil to get the results that they wanted. That is why I state that ALL scientific research is subjective NOT objective!!!!!!!!!!!!!!!!!!!!!! Read about it here: http://wp.me/p5ggLY-a5
It is Simple – Cholesterol DOES NOT CAUSE Heart Disease!
Simply stated, without acid caused inflammation being present in the body, there is no way that cholesterol would accumulate on and in the wall of the blood vessel and cause heart disease and strokes. Without acid caused inflammation, cholesterol would move freely throughout the body as nature intended. It is acid caused inflammation from acidic lifestyle and dietary choices that causes cholesterol to become trapped.
Acid caused inflammation is not complicated. The cycle of metabolic and dietary acid inflammation is perfect in how the body releases cholesterol to bind acids that cause inflammation in the first place. However, if we chronically expose the body to injury to acidic poisonous toxins from acidic foods and drinks the human body was never designed to process, a condition occurs called systemic latent tissue acidosis that is the cause of ALL inflammation. Chronic acidic inflammation is just as harmful as acute acidic inflammation and are both caused by an increase of dietary and metabolic acids.
What thoughtful person would willfully expose himself or herself repeatedly to acidic foods, drinks, drugs or other substances that are known to cause injury to the body? Well, smokers, alcohol, coffee black tea, soda pop, energy and sport beverage drinkers perhaps, but at least they made that choice willfully.
The rest of us have simply followed the recommended mainstream acidic diet that is low in polyunsaturated fats, high in acidic carbohydrates and highly acidic animal flesh, not knowing we were causing repeated acidic injury to our blood vessels. This repeated injury creates chronic acidic inflammation leading to heart disease, stroke, diabetes and obesity.
Let me repeat: The injury and inflammation caused from acidic foods, drinks and metabolism in our blood vessels is the cause of stokes, heart attacks, diabetes and obesity and NOT the increase of cholesterol. A low healthy fat and salt diet recommended for years by mainstream medicine will cause strokes, heart attacks, diabetes and obesity.
What are the biggest culprits of chronic acidic inflammation? Quite simply, they are the overload of simple, highly processed carbohydrates (sugar, dairy products, animal flesh, chocolate, coffee, tea, including green tea, alcohol, soda pops, vinegar, peanuts, mushrooms, flour and corn and all the products made from them) and the excess consumption of saturated vegetable oils like soybean, corn and sunflower that are found in many processed foods.
Take a moment to visualize rubbing a stiff brush repeatedly over soft skin until it becomes quite red and nearly bleeding if you kept this up several times a day, every day for five years. If you could tolerate this painful brushing, you would have a bleeding, swollen infected area that became worse with each repeated acid causing injury. This is a good way to visualize dietary and metabolic acids as the brush leading to the inflammatory process that could be going on in your body right now.
Regardless of where the acidic inflammatory process occurs, externally or internally, it is the same. Using Ultrasound I have peered inside thousands upon thousands of arteries. A diseased artery looks as if someone took a brush and scrubbed repeatedly against its wall. Several times a day, every day, the acidic foods we eat create small injuries compounding into more injuries, causing the body to respond continuously and appropriately with increased acid caused inflammation.
While we savor the tantalizing taste of a sweet roll, chocolate or a carbonated drink our body responds alarmingly as if a foreign invader arrived declaring war. ACIDIC foods loaded with sugars and simple carbohydrates, or processed with saturated oils for long shelf life have been the mainstay of the American diet for six decades. These acidic foods have been slowly poisoning everyone.
How does eating a simple sweet roll or a piece a chocolate create a cascade of acid causing inflammation to make you sick?
Imagine spilling acidic sugary syrup on your keyboard and you have a visual of what occurs inside the cell. When we consume simple carbohydrates such as sugar, blood sugar rises rapidly. In response, your pancreas secretes insulin and sodium bicarbonate whose primary purpose is to bind and solidify acids so they do NOT destroy healthy body and blood cells and cause internal bleeding. In addition, the body releases cholesterol to help solidify excess dietary and/or metabolic acids that have NOT been properly eliminated through the four channels of elimination – urination, perspiration, respiration and defecation.
The body solidifies acids to protect healthy tissues, glands and organs from ulceration and then degeneration. After years of an acidic lifestyle and diet solidified acids will build-up on the wall of the arteries and veins leading to atherosclerosis, stroke and heart attack.
What does all this have to do with inflammation? Blood sugar which is a metabolic acid is controlled in a very narrow range. Extra acidic sugar molecules that are not solidified and eliminated through the four channels of elimination will injure the blood vessel wall. This repeated acidic injury to the blood vessel wall causes irritation, inflammation, ulceration and eventual degeneration or heart disease and/or cancer. When you spike your blood sugar levels or acid levels several times a day, every day, with acidic foods or thoughts it is exactly like taking sandpaper to the inside of your delicate blood vessels.
While you may not be able to see it, rest assured, tissue, gland and organ acidosis is present. I have seen it in over 40,000 client/patients spanning over 30 years who all shared one common denominator — dietary and metabolic acid caused inflammation in their veins, arteries, glands, tissues and organs. This is what retained physiological acid looks like in the tissues using full-body thermography to show the acidic red and white hot spots.
Let’s get back to the sweet roll and chocolate. These innocent looking goodies not only contain the acid sugar, they are also fermented and processed in one of many saturated oils. Chips and fries are soaked in soybean oil; processed foods are manufactured with saturated oils for longer shelf life.
If the balance shifts by consuming excessive sugar, animal protein, vinegar, coffee, tea, alcohol, corn, peanuts and saturated oil, the cell membranes will be damaged and the body and blood cells will begin to degenerate causing even more acids leading to greater risk of inflammation and dis-ease.
Today’s mainstream American ACIDIC diet has produced an extreme imbalance in the alkaline design of the body and an increase in dietary and metabolic acids that cause ALL sickness and dis-ease. You read this correctly – ALL sickness and dis-ease is caused by metabolic, dietary, respiratory and/or environmental ADIDS! There are no other causes. Germs and viruses are the symptoms of cellular breakdown and NOT the cause of ANY disease. Simply said, germs do NOT cause dis-ease!
To make matters worse, eating these acidic foods and drinks causes the body to hold on to more fat as a depository for these excess acids that are NOT being properly eliminated through the four channels of elimination. That is why people get fat. The increase in fat is in direct relationship to the increase of acidic foods, drinks and lifestyle choices. The process that began with a sweet roll or a cup of coffee, or a piece of chocolate or a glass of wine turns into a vicious cycle over time that creates heart disease, stroke, high blood pressure, diabetes, obesity and finally, Alzheimer’s disease, as the acid caused inflammatory process continues unabated.
There is no escaping the fact that the more we consume prepared and processed acidic foods, the more we increase the inflammation switch little by little each day. The human body cannot process, nor was it designed to consume, foods packed with sugars, animal flesh, dairy products, vinegar, alcohol, coffee, tea, chocolate, soda pop, mushrooms, peanuts, corn, flour and saturated processed oils.
There is but one answer to quieting acid caused inflammation, and that is returning to foods closer to their natural alkaline state. To build muscle, eat more chlorophyll concentrated alkaline foods.
Choose carbohydrates that are very complex such as colorful fruit and vegetables. Cut out of your diet saturated oils from corn or soybean.
One tablespoon of corn oil contains 7,280 mg of saturated oil; soybean contains 6,940 mg. Instead, use olive oil, avocado oil, hemp oil or fax oil.
Forget the “science” that has been drummed into your head for decades. The science that saturated fat alone causes heart disease is non-existent. The science that saturated fat raises blood cholesterol is also very weak. Since we now know that cholesterol is not the cause of heart disease, the concern about saturated fat having no place on its hydrogen chain to buffer metabolic and dietary acid is real science. It is acid that causes disease and ALL polyunsaturated oils help to buffer excess acids by the carbon chain picking up the hydrogen ion or acid on its unsaturation. In other words, all polyunsaturated fats whether Omega 1, 3, 6 or 9 buffer or neutralize all dietary and/or metabolic acids on their unsaturated carbon.
The cholesterol theory led to the no-fat, low-fat recommendations that in turn created the very acidic foods now causing an epidemic of acid caused inflammation,induration, ulceration and degeneration. Mainstream medicine made a terrible mistake when it advised people to avoid foods high in cholesterol. We now have an epidemic of arterial acidic caused inflammation leading to heart disease and other silent killers.
Government nutrition guidelines recommend a diet high in carbohydrate regardless of the ample evidence of the health risks it promotes. Yet, heart disease and obesity rates have risen in correlation with a reduced intake of dietary fat. The Food Standards Agency states all individuals’ diets should contain “plenty of starchy foods such as rice, bread, pasta and potatoes”. In addition to this, “just a little saturated fat”. This recommendation is a recipe for heart disease and stroke because of its high level of dietary acid.
While science has moved on, nutritional advice lags behind. And in a study published in Open Heart, a group of researchers conclude that national dietary advice on fat consumption issued to millions in the 1970s to reduce the risk of heart disease which suggested that fat should form no more than 30% of daily food intake lacked any solid trial evidence and shouldn’t have been introduced.
While more circumspect, cardiologist Rahul Bahl wrote in a linked editorial:
“There is certainly a strong argument that an over-reliance in public health on saturated fat as the main dietary villain for cardiovascular disease has distracted from the risks posed by other nutrients, such as carbohydrates.”
Fat and High-Carbohydrate Foods
Some fats aren’t good – trans fats, for example, which are mostly man-made – while others, such as monounsaturated fats found in olive oil are seen as having beneficial qualities.
Today, government guidelines recommend that fats should compose no more than 35% of an individual’s daily calorie intake – and that saturated fat, in particular, ought to supply less than 11%.
Fat intake decreased from 36.6% to 33.7% from 1971 to 2006, while the intake of carbohydrates rose from 44.0% to 48.7%. Yet obesity levels have escalated.
There is evidence to also show that carbohydrates can lead to feelings of increased hunger. A recent study in The American Journal of Clinical Nutrition found that eating carbohydrate foods with a high glycemic index (bread, rice, pasta) caused effects on the brain that led to feelings of increased hunger, which could in turn lead to eating more.
Another study in 2013 found high-carb meals could leave you feeling hungrier hours later compared to a low-carb meal with more fibre, protein and fat. The team behind the research attributed this to the plummeting levels of blood sugar that regularly follows high-carb meals.
The Diet-Heart Hypothesis
At the University of Hull they have been also looking at the effects of saturated fats on triglyceride levels – a type of fat (lipid) found in the blood. Using coconut oil because of its high (90%) saturated fat content, we found that when coupled with exercise, it significantly reduced triglyceride levels. A recent Brazilian rat study also found that coconut oil and exercise could lower blood pressure.
So where does our unshakable idea that fat leads to heart disease come from? The diet-heart hypothesis, that low density lipoproteins (LDL) cholesterol is raised in the blood by eating saturated fat, which then leads to clogged arteries and eventual heart disease, is not a credible claim.
This theory linking saturated fat and heart disease has been around since 1955 when Ansel Keys introduced his lipid hypothesis. Despite it being the foundation of dietary recommendations, it has never been proven and we have been advised to avoid certain foods including meat, dairy products and coconuts. And these myths are so deeply embedded in our minds, that recent science advocates have seen how hard it is to challenge established thinking.
Saturated Fat and Cholesterol
When we talk about high-density lipoprotein (HDL) or LDL – often referred to as good and bad cholesterol – we aren’t actually referring to cholesterol itself. These lipoproteins actually carry cholesterol, fat and fat soluble vitamins in the bloodstream. It appears that elevated levels of cholesterol (or more accurately, cholesterol which is transported around the blood by lipioproteins) is correlated with an increase in the risk of heart disease.
However, correlation does not mean causation. Very low cholesterol is linked with an increased risk of death (though not from heart disease). And in the very old, research suggests cholesterol can be protective. So it’s fair to say the relationship between cardiovascular disease and total cholesterol is complex.
Type of cholesterol is important. The “good” (HDL) cholesterol is strongly linked with a reduced risk of heart disease. However, LDL, the “bad” cholesterol, is associated with an increased risk of heart disease. But it turns out that there are in fact subtypes of LDL which make this black and white picture more complicated. The actual size of the LDL particle is significant. Individuals are at a heightened risk of heart disease if they have most small, dense LDL particles, that may more easily lodge in the arteries, as opposed to those who have large LDL particles.
Your blood lipid profile is frequently used as a medical screening tool for abnormalities in lipids (including triglycerides and cholesterol). These blood lipid profile tests can identify approximate risks for cardiovascular disease and specific genetic diseases. Studies have also shown that saturated fats do not harm your blood lipid profile – and can actually improve it. Saturated fats could lower the risk of heart disease by shifting LDL cholesterol from dense small LDL to large LDL.
Numerous short-term feeding trials have shown that an increase in saturated fat consumption leads to a rise in overall LDL. Nevertheless, the result is inconsistent and weak. The methods used in a number of these research studies have been criticised – and plenty of studies support the contrary, that no association exists between total LDL and saturated fat consumption.
Cause and Correlation
If it was true that saturated fat did cause heart disease, then it follows that people who consume more would be at higher risk. But observational studies – again only illustrative of correlation not cause – haven’t shown this. One study looked at a population of 347,747 subjects from a total of 21 studies and concluded that there was “no significant evidence for concluding that dietary saturated fat is associated with an increased risk of coronary heart or cardiovascular disease”. This has also been the conclusion of other reviews.
So What About Randomized Controlled Trials?
One such study divided 12,866 male subjects at a high risk of heart disease into a low-fat or Western diet group. After six years, no difference was found between them. The Women’s Health Imitative, the biggest randomized controlled trial in diet history, comprised of 48,835 postmenopausal women who were also divided into two similar groups and came up with similar findings.
The Cold-Pressed Organic Coconut Oil Connection
If you don’t care for the science, then take an everyday example. Look at the large populations of the Masai in Africa who consume large amounts of saturated fat but have low levels of coronary heart disease. Or the Tokelauans of New Zealand who consume a massive amount of saturated fat through coconuts: more than 60% of their daily calories come from coconuts. These populations have no history of heart disease. And the health benefits of coconut oil are now becoming known more widely.
We are learning so much more about fats and that there is no evidence that saturated fat causes heart disease. Leading nutrition experts have been calling for an amendment to dietary recommendations for more than ten years. But despite these calls and the high-quality evidence assembled throughout the past decade, doctors, governments – and by extension the public – still take extraordinarily little notice. But a decade of research to the contrary would suggest it’s time we moved away from entrenched thinking, towards a more enlightened attitude to saturated fat.
What you can do is choose whole, organic, raw, NON-GMO, alkaline foods your grandmother served and not those your mom turned to as grocery store aisles filled with manufactured acidic foods and drinks. By eliminating acidic causing inflammatory foods and adding essential nutrients from fresh, raw, organic, alkaline unprocessed food, you will reverse years of damage in your arteries and throughout your body from consuming the typical American ACIDIC diet.
To learn more read the following article, THE PH MIRACLE FOR HEART DISEASE – DISCOVER THE TRUTH ABOUT HEART DISEASE, CONGESTIVE HEART FAILURE, ATHEROSCLEROSIS, CHOLESTEROL, HYPERTENSION, STROKE AND MORE! –
To learn more read the following article: https://www.amazon.com/gp/product/B01KBMFRA4/ref=dbs_a_def_rwt_hsch_vapi_taft_p2_i8
To learn more about the work, research, findings of publications of Robert O Young CPT, MSc, DSc, PhD and Naturopathic Practitioner go to: http://www.drrobertyoung.com
- Ravnskov U, Diamond DM, Hama R, et al. Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly: a systematic review
- Office Of Dietary Supplements Fact Sheet: Folate
- Doshi SN, McDowell IF, Moat SJ, Payne N, Durrant HJ, Lewis MJ, Goodfellos J. Folic acid improves endothelial function in coronary artery disease via mechanisms largely independent of homocysteine. Circulation. 2002;105:22-6.
- Doshi SN, McDowell IFW, Moat SJ, Lang D, Newcombe RG, Kredean MB, Lewis MJ, Goodfellow J. Folate improves endothelial function in coronary artery disease. Arterioscler Thromb Vasc Biol 2001;21:1196-1202.
- Wald DS, Bishop L, Wald NJ, Law M, Hennessy E, Weir D, McPartlin J, Scott J. Randomized trial of folic acid supplementation and serum homocysteine levels. Arch Intern Med 2001;161:695-700.
- Jennings E. Folic acid as a cancer preventing agent. Med Hypothesis 1995;45:297-303.
- Freudenheim JL, Grahm S, Marshall JR, Haughey BP, Cholewinski S, Wilkinson G. Folate intake and carcinogenesis of the colon and rectum. Int J Epidemiol 1991;20:368-74.
- Giovannucci E, Stampfer MJ, Colditz GA, Hunter DJ, Fuchs C, Rosner BA, Speizer FE, Willett WC. Multivitamin use, folate, and colon cancer in women in the Nurses’ Health Study. Ann Intern Med 1998;129:517-24.
- A Paoloni-Giacobino, R Grimble, C Pichard. Genetics and nutrition. Clinical Nutrition Volume 22, Issue 5, Pages 429-435 (October 2003)
- Corradaa MM, Kawasab CH, Hallfrischc J, Mullerd D, Brookmeyere R. Reduced risk of Alzheimer?s disease with high folate intake: The Baltimore Longitudinal Study of Aging. Alzheimer’s and Dementia Volume 1, Issue 1, Pages 11-18 (July 2005).
- Wang HX, Wahlin Å, Basun H, Fastbom J, Winblad B, Fratiglioni L. Vitamin B12 and folate in relation to the development of Alzheimer?s disease. Neurology May 8, 2001 vol. 56 no. 9 1188-1194.
- Office Of Dietary Supplements Fact Sheet
- Appel LJ. Nonpharmacologic therapies that reduce blood pressure: A fresh perspective. Clin Cardiol 1999;22:1111-5.
- Simopoulos AP. The nutritional aspects of hypertension. Compr Ther 1999;25:95-100.
- Appel LJ, Moore TJ, Obarzanek E, Vollmer WM, Svetkey LP, Sacks FM, Bray GA, Vogt TM, Cutler JA, Windhauser MM, Lin PH, Karanja N. A clinical trial of the effects of dietary patterns on blood pressure. N Engl J Med 1997;336:1117-24.
- Saris NE, Mervaala E, Karppanen H, Khawaja JA, Lewenstam A. Magnesium: an update on physiological, clinical, and analytical aspects. Clinica Chimica Acta 2000;294:1-26.
- Institute of Medicine. Food and Nutrition Board. Dietary Reference Intakes: Calcium, Phosphorus, Magnesium, Vitamin D and Fluoride. National Academy Press. Washington, DC, 1999.
- Paolisso G, Sgambato S, Gambardella A, Pizza G, Tesauro P, Varricchio H, D’Onofrio F. Daily magnesium supplements improve glucose handling in elderly subjects. Am J Clin Nutr 1992;55:1161-7.
- Altura BM and Altura BT. Magnesium and cardiovascular biology: An important link between cardiovascular risk factors and atherogenesis. Cell Mol Biol Res 1995;41:347-59.
- Ford ES. Serum magnesium and ischaemic heart disease: Findings from a national sample of US adults. Intl J of Epidem 1999;28:645-51.
- Liao F, Folsom A, Brancati F. Is low magnesium concentration a risk factor for coronary heart disease? The Atherosclerosis Risk in Communities (ARIC) Study. Am Heart J 1998;136:480-90.
- Ascherio A, Rimm EB, Hernan MA, Giovannucci EL, Kawachi I, Stampfer MJ, Willett WC. Intake of potassium, magnesium, calcium, and fiber and risk of stroke among US men. Circulation 1998;98:1198-204.
- Elisaf M, Milionis H, Siamopoulos K. Hypomagnesemic hypokalemia and hypocalcemia: Clinical and laboratory characteristics. Mineral Electrolyte Metab 1997;23:105-12.
- Xing JH and Soffer EE. Adverse effects of laxatives. Dis Colon Rectum 2001;44:1201-9.
- Mauskop A, Altura BM. Role of magnesium in the pathogenesis and treatment of migraines. Clin Neurosci. 1998;5(1):24-27.
- Peikert A, Wilimzig C, Kohne-Volland R. Prophylaxis of migraine with oral magnesium: results from a prospective, multi-center, placebo-controlled and double-blind randomized study. Cephalalgia. 1996;16(4):257-263.
- Pfaffenrath V, Wessely P, Meyer C, et al. Magnesium in the prophylaxis of migraine–a double-blind placebo-controlled study. Cephalalgia. 1996;16(6):436-440.
- Wang F, Van Den Eeden SK, Ackerson LM, Salk SE, Reince RH, Elin RJ. Oral magnesium oxide prophylaxis of frequent migrainous headache in children: a randomized, double-blind, placebo-controlled trial. Headache. 2003;43(6):601-610.
- Bendich A. The potential for dietary supplements to reduce premenstrual syndrome (PMS) symptoms. J Am Coll Nutr. 2000;19(1):3-12.
- Rude RK. Magnesium deficiency: A cause of heterogeneous disease in humans. J Bone Miner Res 1998;13:749-58.
- Rude KR. Magnesium metabolism and deficiency. Endocrinol Metab Clin North Am 1993;22:377-95.
- Kelepouris E and Agus ZS. Hypomagnesemia: Renal magnesium handling. Semin Nephrol 1998;18:58-73.
- Ramsay LE, Yeo WW, Jackson PR. Metabolic effects of diuretics. Cardiology 1994;84 Suppl 2:48-56.
- Kobrin SM and Goldfarb S. Magnesium Deficiency. Semin Nephrol 1990;10:525-35.
- Lajer H and Daugaard G. Cisplatin and hypomagnesemia. Ca Treat Rev 1999;25:47-58.
- Tosiello L. Hypomagnesemia and diabetes mellitus. A review of clinical implications. Arch Intern Med 1996;156:1143-8.
- Paolisso G, Scheen A, D’Onofrio F, Lefebvre P. Magnesium and glucose homeostasis. Diabetologia 1990;33:511-4.
- Elisaf M, Bairaktari E, Kalaitzidis R, Siamopoulos K. Hypomagnesemia in alcoholic patients. Alcohol Clin Exp Res 1998;22:244-6.
- Abbott L, Nadler J, Rude RK. Magnesium deficiency in alcoholism: Possible contribution to osteoporosis and cardiovascular disease in alcoholics. Alcohol Clin Exp Res 1994;18:1076-82.
- Rude RK, Shils ME. Magnesium. In: Shils ME, Shike M, Ross AC, Caballero B, Cousins RJ, eds. Modern Nutrition in Health and Disease. 10th ed. Baltimore: Lippincott Williams & Wilkins; 2006:223-247.
- Food and Nutrition Board, Institute of Medicine. Magnesium. Dietary Reference Intakes: Calcium, Phosphorus, Magnesium, Vitamin D, and Fluoride. Washington D.C.: National Academy Press; 1997:190-249.
- Schwartz R, Walker G, Linz MD, MacKellar I. Metabolic responses of adolescent boys to two levels of dietary magnesium and protein. I. Magnesium and nitrogen retention. Am J Clin Nutr. 1973;26(5):510-518.
- Shils ME. Magnesium. In Modern Nutrition in Health and Disease, 9th Edition. (edited by Shils, ME, Olson, JA, Shike, M, and Ross, AC.) New York: Lippincott Williams and Wilkins, 1999, p. 169-92.
- Spencer H, Norris C, Williams D. Inhibitory effects of zinc on magnesium balance and magnesium absorption in man. J Am Coll Nutr. 1994;13(5):479-484.
- Torsten Bohn, Lena Davidsson*, Thomas Walczyk and Richard F. Hurrel Fractional magnesium absorption is signi?cantly lower in human subjects from a meal served with an oxalate-rich vegetable, spinach, as compared with a meal served with kale, a vegetable with a low oxalate content. Laboratory for Human Nutrition, Institute of Food Science and Nutrition, Swiss Federal Institute of Technology, Zurich, Switzerland (Received 27 May 2003 – Revised 7 November 2003 – Accepted 28 November 2003
- FDA Drug Safety Communication: Low magnesium levels can be associated with long-term use of Proton Pump Inhibitor drugs (PPIs)
- Leach RM, Harris ED. Manganese. In: O’Dell BL, Sunde RA, eds. Handbook of nutritionally essential minerals. New York: Marcel Dekker, Inc; 1997:335-355.
- Freeland-Graves J, Llanes C. Models to study manganese deficiency. In: Klimis-Tavantzis DL, ed. Manganese in health and disease. Boca Raton: CRC Press, Inc; 1994.
- Reginster JY, Strause LG, Saltman P, Franchimont P. Trace elements and postmenopausal osteoporosis: a preliminary study of decreased serum manganese. Med Sci Res. 1988;16:337-338.
- Odabasi E, Turan M, Aydin A, Akay C, Kutlu M. Magnesium, zinc, copper, manganese, and selenium levels in postmenopausal women with osteoporosis. Can magnesium play a key role in osteoporosis? Ann Acad Med Singapore. 2008;37(7):564-567.
- Keen CL, Zidenberg-Cherr S. Manganese. In: Ziegler EE, Filer LJ, eds. Present Knowledge in Nutrition. 7th ed. Washington D.C.: ILSI Press; 1996:334-343.
- Carl GF, Gallagher BB. Manganese and epilepsy. In: Klimis-Tavantzis DL, ed. Manganese in health and disease. Boca Raton: CRC Press, Inc; 1994:133-157.
- Blaurock-Busch, E. Wichtige Nahrstoffe fur Gesunde Haut und Haare, Kosmetik Internat. 3/87.
- Collipp, P.J., et al. Manganese in infant formulas and learning disability. Ann. Nutr. Metab. 27(6):488-494, 1983.
- “Guidelines for Niacin Therapy For the Treatment of Elevated Lipoprotein a (Lpa)”. Rush Hemophilia & Thrombophilia Center. August 15, 2002, Revised July 27, 2005. Retrieved 20 November 2009. “facial flushing is a common side effect of niacin therapy that usually subsides after several weeks of consistent niacin use”
- Katzung, Bertram G. (2006). Basic and clinical pharmacology. New York: McGraw-Hill Medical Publishing Division. ISBN 0071451536.
- Greenbaum CJ, Kahn SE, Palmer JP. Nicotinamide’s effects on glucose metabolism in subjects at risk for IDDM. Diabetes. 1996;45(11):1631-1634.
- Lampeter EF, Klinghammer A, Scherbaum WA, et al. The Deutsche Nicotinamide Intervention Study: an attempt to prevent type 1 diabetes. DENIS Group. Diabetes. 1998;47(6):980-984.
- Hageman GJ, Stierum RH. Niacin, poly(ADP-ribose) polymerase-1 and genomic stability. Mutat Res. 2001;475(1-2):45-56.
- Jacobson EL, Shieh WM, Huang AC. Mapping the role of NAD metabolism in prevention and treatment of carcinogenesis. Mol Cell Biochem. 1999;193(1-2):69-74.
- Weitberg AB. Effect of nicotinic acid supplementation in vivo on oxygen radical-induced genetic damage in human lymphocytes. Mutat Res. 1989;216(4):197-201.
- Tang AM, Graham NM, Saah AJ. Effects of micronutrient intake on survival in human immunodeficiency virus type 1 infection. Am J Epidemiol. 1996;143(12):1244-1256.
- Brown RR, Ozaki Y, Datta SP, Borden EC, Sondel PM, Malone DG. Implications of interferon-induced tryptophan catabolism in cancer, auto-immune diseases and AIDS. Adv Exp Med Biol. 1991;294:425-435.
- Murray MF, Langan M, MacGregor RR. Increased plasma tryptophan in HIV-infected patients treated with pharmacologic doses of nicotinamide. Nutrition. 2001;17(7-8):654-656.
- Office of Dietary Supplements Fact Sheet: Vitamin B6
- New SA, Bolton-Smith C, Grubb DA, Reid DM. Nutritional influences on bone mineral density: a cross-sectional study in premenopausal women. Am J Clin Nutr. 1997;65(6):1831-1839.
- New SA, Robins SP, Campbell MK, et al. Dietary influences on bone mass and bone metabolism: further evidence of a positive link between fruit and vegetable consumption and bone health? Am J Clin Nutr. 2000;71(1):142-151.
- Tucker KL, Hannan MT, Chen H, Cupples LA, Wilson PW, Kiel DP. Potassium, magnesium, and fruit and vegetable intakes are associated with greater bone mineral density in elderly men and women. Am J Clin Nutr. 1999;69(4):727-736.
- Ascherio A, Rimm EB, Hernan MA, et al. Intake of potassium, magnesium, calcium, and fiber and risk of stroke among US men. Circulation. 1998;98(12):1198-1204.
- Iso H, Stampfer MJ, Manson JE, et al. Prospective study of calcium, potassium, and magnesium intake and risk of stroke in women. Stroke. 1999;30(9):1772-1779.
- Fang J, Madhavan S, Alderman MH. Dietary potassium intake and stroke mortality. Stroke. 2000;31(7):1532-1537.
- Bazzano LA, He J, Ogden LG, et al. Dietary potassium intake and risk of stroke in US men and women: National Health and Nutrition Examination Survey I epidemiologic follow-up study. Stroke. 2001;32(7):1473-1480.
- Green DM, Ropper AH, Kronmal RA, Psaty BM, Burke GL. Serum potassium level and dietary potassium intake as risk factors for stroke. Neurology. 2002;59(3):314-320.
- Barri YM, Wingo CS. The effects of potassium depletion and supplementation on blood pressure: a clinical review. Am J Med Sci. 1997;314(1):37-40.
- Hajjar IM, Grim CE, George V, Kotchen TA. Impact of diet on blood pressure and age-related changes in blood pressure in the US population: analysis of NHANES III. Arch Intern Med. 2001;161(4):589-593.
- Appel LJ, Moore TJ, Obarzanek E, et al. A clinical trial of the effects of dietary patterns on blood pressure. DASH Collaborative Research Group. N Engl J Med. 1997;336(16):1117-1124.
- Gennari FJ. Hypokalemia. N Engl J Med. 1998;339(7):451-458.
- Shearer MJ. The roles of vitamins D and K in bone health and osteoporosis prevention. Proc Nutr Soc. 1997;56(3):915-937.
- Booth SL. Skeletal functions of vitamin K-dependent proteins: not just for clotting anymore. Nutr Rev. 1997;55(7):282-284.
- Suttie JW. Vitamin K. In: Shils ME, Shike M, Ross AC, Caballero B, Cousins RJ, eds. Modern Nutrition in Health and Disease. 10th ed. Baltimore: Lippincott Williams & Wilkins; 2006:412-425.
- Allison (2001). The possible role of vitamin K deficiency in the pathogenesis of Alzheimer’s disease and in augmenting brain damage associated with cardiovascular disease. Medical hypotheses 57 (2): 151?5. doi:10.1054/mehy.2001.1307. PMID 11461163.
- ODS Fact Sheet on Coumadin – http://ods.od.nih.gov/pubs/factsheets/coumadin1.pdf
- Office of Dietary Suppliments Face Sheet: Vitamin C
- Gokce N, Keaney JF, Jr., Frei B, et al. Long-term ascorbic acid administration reverses endothelial vasomotor dysfunction in patients with coronary artery disease. Circulation. 1999;99(25):3234-3240.
- Audera, C (2001). “Mega-dose vitamin C in treatment of the common cold: a randomised controlled trial”. Medical Journal of Australia 389: 175.
- Hemilä, Harri; Chalker, Elizabeth; Douglas, Bob; Hemilä, Harri (2007). “Vitamin C for preventing and treating the common cold”. Cochrane database of systematic reviews (Online) (3): CD000980.
- Fleming DJ, Tucker KL, Jacques PF, Dallal GE, Wilson PW, Wood RJ (December 2002). “Dietary factors associated with the risk of high iron stores in the elderly Framingham Heart Study cohort”.
- The American Journal of Clinical Nutrition 76 (6): 1375?84.Institute of Medicine. Food and Nutrition Board. Dietary Reference Intakes for Vitamin C, Vitamin E, Selenium, and Carotenoids. Washington, DC: National Academy Press, 2000.
- Weinstein M, Babyn P, Zlotkin S. An orange a day keeps the doctor away: scurvy in the year 2000. Pediatrics 2001;108:E55.
- Hoffman FA. Micronutrient requirements of cancer patients. Cancer. 1985;55 (1 Suppl):295-300.
- Deicher R, Hörl WH. Vitamin C in chronic kidney disease and hemodialysis patients. Kidney Blood Press Res 2003;26:100-6.
- Aishah Al-Jarallah, Fatima Igdoura, Yi Zhang, Christine B Tenedero, Elizabeth J White, Melissa E Macdonald, Suleiman A Igdoura, Bernardo L Trigatti. The effect of pomegranate extract on coronary artery atherosclerosis in SR-BI/APOE double knockout mice. Atherosclerosis. 2013 May ;228(1):80-9. Epub 2013 Mar 7. PMID: 23528829
- Michael Aviram, Mira Rosenblat, Diana Gaitini, Samy Nitecki, Aaron Hoffman, Leslie Dornfeld, Nina Volkova, Dita Presser, Judith Attias, Harley Liker, Tony Hayek. Pomegranate juice consumption for 3 years by patients with carotid artery stenosis reduces common carotid intima-media thickness, blood pressure and LDL oxidation. Clin Nutr. 2004 Jun;23(3):423-33. PMID: 15158307
- GreenMedInfo.com, Pomegranate’s Anti-Inflammatory Properties
- Mahalaxmi Mohan, Harshal Waghulde, Sanjay Kasture. Effect of pomegranate juice on Angiotensin II-induced hypertension in diabetic Wistar rats. Phytother Res. 2009 Dec 17. PMID: 20020514
- Filomena de Nigris, Maria Luisa Balestrieri, Sharon Williams-Ignarro, Francesco P D’Armiento, Carmela Fiorito, Louis J Ignarro, Claudio Napoli. The influence of pomegranate fruit extract in comparison to regular pomegranate juice and seed oil on nitric oxide and arterial function in obese Zucker rats. Nitric Oxide. 2007 Aug ;17(1):50-4. Epub 2007 May 5. PMID: 17553710
- Filomena de Nigris, Sharon Williams-Ignarro, Vincenzo Sica, Lilach O Lerman, Francesco P D’Armiento, Russell E Byrns, Amelia Casamassimi, Daniela Carpentiero, Concetta Schiano, Daigo Sumi, Carmela Fiorito, Louis J Ignarro, Claudio Napoli. Effects of a pomegranate fruit extract rich in punicalagin on oxidation-sensitive genes and eNOS activity at sites of perturbed shear stress and atherogenesis. Cardiovasc Res. 2007 Jan 15;73(2):414-23. Epub 2006 Sep 1. PMID: 17014835
- Yasunori Sawayama, Kyoko Okada, Shinji Maeda, Hachiro Ohnishi, Norihiro Furusyo, Jun Hayashi. Both hepatitis C virus and Chlamydia pneumoniae infection are related to the progression of carotid atherosclerosis in patients undergoing lipid lowering therapy. Fukuoka Igaku Zasshi. 2006 Aug;97(8):245-55. PMID: 17087362
- M Aviram, L Dornfeld, M Rosenblat, N Volkova, M Kaplan, R Coleman, T Hayek, D Presser, B Fuhrman. Pomegranate juice consumption reduces oxidative stress, atherogenic modifications to LDL, and platelet aggregation: studies in humans and in atherosclerotic apolipoprotein E-deficient mice. Am J Clin Nutr. 2000 May ;71(5):1062-76. PMID: 10799367
- Adde FV, Rodrizues JC, Cardoso AL. Nutritional follow-up of cystic fibrosis patients: the role of nutrition education. J Pediatr (Rio J). 2004;80(6):475-82.
- Beckles Willson N, Elliot TM, Everard ML. Omega-3 fatty acids (from fish oils) for cystic fibrosis. Cochrane Database Syst Rev. 2002;(3):CD002201.
- Bope. Conn’s Current Therapy 2010. 1st ed. Philadelphia, PA: Saunders, An Imprint of Elsevier; 2009.
- Bruzzese E, Raia V, Gaudiello G, et al. Intestinal inflammation is a frequent feature of cystic fibrosis and is reduced by probiotic administration. Aliment Pharmacol Ther. 2004;20(7):813-9.
- Cabrera C, Artacho R, Gimenez R. Beneficial effects of green tea — a review. J Am Coll Nutr. 2006;25(2):79-99.
- Campbell, T.M. The China Study, BenBella Books; First Paperback Edition edition (May 11, 2006).
- Caramia G, Cocchi M, Garliardini R, et al. Fatty acids composition of plasma phospholipids and triglycerides in children with cystic fibrosis. The effect of dietary supplementation with an olive and soybean oils mixture. Pediatr Med Chir. 2003;25(1):42-9.
- Chin J. Intestinal microflora: negotiating health outcomes with the warring community within us. Asia Pac J Clin Nutr. 2004;13(Suppl):S24-5.
- Cvetnic Z, Vladimir-Knezevic S. Antimicrobial activity of grapefruit seed and pulp ethanolic extract. Acta Pharm. 2004;54(3):243-50.
- D’Agostino, Russell MW, Huse DM et al. ‘Primary and subsequent coronary risk appraisal: new results from the Framingham Study’, American Heart Journal 2000.
- D’Agostino, Russell MW, Huse DM et al. ‘Primary and subsequent coronary risk appraisal: new results from the Framingham Study’, American Heart Journal 2000.
- D’Agostino, Vasan, Pencina, Wolf, Cobain, Massaro, Kannel. ‘A General Cardiovascular Risk Profile for Use in Primary Care: The Framingham Heart Study’.
- D’Agostino, Wolf, Belanger, Kannel ‘Stroke Risk Profile: Adjustment for Antihypertensive Medication’, Stroke 1994.
- Dolinoy D.C., Weidman J.R., Waterland R.A., Jirtle R.L. (2006). Maternal Genistein Alters Coat Color and Protects Avy Mouse Offspring from Obesity by Modifying the Fetal Epigenome. Environmental Health Perspectives, 114:567-572.
- Dolinoy D.C., Huang D., Jirtle R.L. (2007). Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development. PNAS, 104: 13056-13061.
- Doron S, Gorbach SL. Probiotics: their role in the treatment and prevention of disease. Expert Rev Anti Infect Ther. 2006;4(2):261-75.
- Farrell P, Rosenstein B, White T, et al. Guidelines for Diagnosis of Cystic Fibrosis in Newborns through Older Adults: Cystic Fibrosis Foundation Consensus Report. Journal of Pediatrics. 2008;153(2)
- Ferri. Ferri’s Clinical Advisior 2010. 1st ed. Philadelphia, PA: Mosby, An Imprint of Elsevier; 2009.
- Gonclaves C, Dinis T, Batista MT. Antioxidant properties of proanthocyanidins of Uncaria tomentosa bark decoction: a mechanism for anti-inflammatory activity. Phytochemistry. 2005;66(1):89-98.
- Grey V, Mohammed SR, Smountas AA, et al. Improved glutathione status in young adult patients with cystic fibrosis supplemented with whey protein. J Cyst Fibros. 2003;2(4):195-8.
- Guo R, Pittler MH, Ernst E. Herbal medicines for the treatment of COPD: a systematic review. Eur Respir J. 2006;28(2):330-8.
- Hale LP, Greer PK, Trinh CT, James CL. Proteinase activity and stability of natural bromelain preparations. Int Immunopharmacol. 2005;5(4):783-93.
- Harlan M. Krumholz, MD; Teresa E. Seeman, PhD; Susan S. Merrill, PhD; Carlos F. Mendes de Leon, PhD; Viola Vaccarino, MD; David I. Silverman, MD; Reiko Tsukahara, MD; Adrian M. Ostfeld, MD; Lisa F. Berkman, PhD. Lack of Association Between Cholesterol and Coronary Heart Disease Mortality and Morbidity and All- Cause Mortality in Persons Older Than 70 Years. JAMA. 1994;272(17):1335-1340. doi:10.1001/jama.1994.03520170045034.
- Heggers JP, Cottingham J, Gussman J, et al. The effectiveness of processed grapefruit-seed extract as an antibacterial agent: II
- Mechanism of action and in vitro toxicity. J Altern Complement Med. 2002;8(3):333-40.
- Huang SH, Schall JI, Zemel BS, Stallings VA. Vitamin E status in children with cystic fibrosis and pancreatic insufficiency.J Pediatr. 2006;148(4):556-559.
- Infante P, Redecillas F, Torrent V, et al. Improvement of intestinal function in cystic fibrosis patients using probiotics. An Pediatr. 2008;69(6):501-5.
- Jonsdottir B, Bergsteinsson H, Baldursson O. Cystic Fibrosis–Review. Laeknabladid. 2008;94(12):831-7.
- Kannel, D’Agostino, Silbershatz, Belanger, Wilson, Levy. ‘Profile for Estimating Risk of Heart Failure’ – Arch Intern. Med. 1999.
- Kaati G., Bygren L.O., Pembrey M., Sjostrom M. (2007). Transgenerational response to nutrition, early life circumstances and longevity. European Journal of Human Genetics, 15: 784-790.
- Kormosh N, Laktionov K, Antoshechkina M. Effect of a combination of extract from several plants on cell-mediated and humoral immunity of patients with advanced ovarian cancer. Phytother Res. 2006;20(5):424-5.
- Kucharski R., Maleszka J., Foret S., Maleszka R. Nutritional Control of Reproductive Status in Honeybees via DNA Methylation (2008). Science, 319: 1827-1830 (registration required)
- McCabe H. Riboflavin deficiency in cystic fibrosis: three case reports. J Hum Nutr Diet. 2001;14(5):365-70.
- McGowan P.O., Meaney M.J., Szyf M. (2008). Â Diet and the epigenetic (re)programming of phenotypic differences in behavior. Brain Research, 1237: 12-24 (subscription required).
- Mizejewski GJ, Pass KA. Fatty acids, alpha-fetoprotein, and cystic fibrosis. Pediatrics. 2001;108(6):1370-3.
- Murray KL, Lee CK, Mogayzel PJ Jr, Zeitlin PL, Rosenstein BJ. Dietary supplement use in pediatric patients with cystic fibrosis. Am J Health Syst Pharm. 2008;65(6):562-5.
- National Institutes of Health (U.S. Department of Health and Human Services) www.nih.gov.
- Nutrition Reviews 54: 1-30, 1996. Raised glutathione levels fight the oxdiation of fats circulating in the bloodstream including cholesterol, retarding the process of plaque formation in the arteries leading to most heart attacks and strokes.
- Olveira G, Olveira C. Nutrition, cystic fibrosis and the digestive tract. Nutr Hosp. 2008;23(2):71-86.
- Paterson PG, Juurlink BH. Nutritional Regulation of Glutathione in Stroke. Neurtox Res. 1999 Dec; 1(2): 99-112.
- Parikh, Pencina, Wang, Benjamin, Lanier, Levy, D’Agostino, Kannel, Vasan. ‘A Risk Score for Predicting Near-Term Incidence of Hypertension: The Framingham Heart Study’, Annals of Internal Medicine 2008.
- Pencina, D’Agostino, Larson, Massaro, Vasan. ‘Predicting the 30-Year Risk of Cardiovascular Disease: The Framingham Heart Study’, Circulation 2009.
- Proesmans M, Vermeulen F, De Boeck K. What’s new in cystic fibrosis? From trating symptoms to correction of the basic defect. Eur J Pediatr. 2008;167(8):839-49.
- RahmanI, MacNee W. Oxidative Stress and Regulation of Glutathione in Lung Inflammation. Eur Respir J. 2000 Sep; 16(3):534-54.
- Roum JH, Buhl R, McElvaney NG, et al. Systemic Deficiency of Glutathione in Systic Fibrosis. J Appl Physiol 1993; 75:19-24.
- Rubin BK. The pharmacologic approach to airway clearance: Mucoactive agents. Paediatr Respir Rev. 2006;7 Suppl 1:S215-9.
- Schnabel RB, Sullivan LM, Levy D, Pencina MJ, Massaro JM, D’Agostino RB, Sr., Newton-Cheh C, Yamamoto JF, Magnani JW, Tadros TM, Kannel WB, Wang TJ, Ellinor PT, Wolf PA, Vasan RS, Benjamin EJ. Development of a risk score for atrial fibrillation (Framingham Heart Study): a community-based cohort Lancet 2009;373:739-745.
- Simopoulos AP. Omega-3 fatty acids in inflammation and autoimmune diseases. J Am Coll Nutr. 2002;21(6):495-505.
- The Adult Treatment Panel III, JAMA. 2001.
- Wilson, Meigs, Sullivan, Fox, Nathan, D’Agostino. ‘Prediction of Incident Diabetes Mellitus in Middle-aged Adults: The Framingham Offspring Study,’ Archives of Internal Medicine 2007.
- Young, RO, Sick and Tired, Woodland Publishing, Orem, Utah, 2001.
- Yoon JH, Baek SJ. Molecular targets of dietary polyphenols with anti-inflammatory properties. Yonsei Med J. 2005;46(5):585-96.
- Young, RO, Young, SR, Young, The pH Miracle Revised and Updated, Grand Central Publishing, New York, NY, 2010Pollak O J. 1952, An Etiologic Concept of Atherosclerosis Based on Study of Intimal Alterations after Shock. Circulation;5;539-550. Full free paper at:http://circ.ahajournals.org/cgi/reprint/5/4/539.pdf
- 148. Ross R, Glomset J, Harker L. 1977. Response to injury and atherogenesis. Am J Pathol. Mar;86(3):675-8
- Press release. 2006. New Explanation For The Cause Of Atherosclerosis: The Acidity Theory, Medical News Today, Aug 10 at http://www.medicalnewstoday.com/articles/49244.php
- Press release. 2006. Beyond Lipids: Understanding the Mechanics of Atherosclerosis (press release). UCSD News, July 12. at: http://www.jacobsschool.ucsd.edu/news/news_releases/release.sfe?id=554
- Kaunas R, Usami S, Chien S. 2006 Regulation of stretch-induced JNK activation by stress fiber orientation. Cellular Signalling, Nov;18(11):1924-31 at http://www.ncbi.nlm.nih.gov/pubmed/16581230
- Haga JH, Li Yi-Shuan J. and Chien S. 2007. Molecular basis of the effects of mechanical stretch on vascular smooth muscle cells, Journal of Biomechanics, 40(5):947-60.
- Mesquita QHde. 1979. Myogenic Theory of Myocardial Infarction (Teoria Miogênica do Enfarte do Miocárdio, Gemini, Sao Paulo, SP – Brazil Book in Portuguese language with a summary in English at: http://www.infarctcombat.org/LivroTM/parte8.htm
- Mesquita QHde, Baptista CAS. 1994. Why Myogenic Theory not Thrombogenic Theory. Arq Bras Cardiol, V. 62 (4) – (Official Journal of Brazilian Cardiology Society). Full translated paper at http://www.infarctcombat.org/MTxTT-ABC.pdf
- Fernandes VS et al. 2006, Subclinical atherosclerosis and incipient regional myocardial dysfunction in asymptomatic individuals. The Multi-Ethnic Study of Atherosclerosis (MESA), J Am Coll Cardiol 47: 2420-8 Full free paper at http://content.onlinejacc.org/cgi/content/full/j.jacc.2005.12.075v1
- Marwah R, Doux J, Lee P and Yun A. 2007. Is atherosclerosis a neurogenic phenomenon? Medical Hypotheses, V 69, I 4: 884-887
- Selye H. 1950. The physiology and pathology of exposure to stress: A treatise based on the concepts of the general-adaptation-syndrome and the diseases of adaptation”, Montreal, Acta, Inc. / Selye H et al. 1970 Experimental Cardiovascular Diseases, Volume 1 (History, Cardiovascular Disease, Factors Influencing Cardiovascular Disease); Volume 2 (Histology and Histochemistry, Chemical and Functional Changes, References), Springer-Verlag, Berlin New York
- Cannon WJ. 1914. The emergency function of the adrenal medulla in pain and the major emotions. Am J Physiol. 33:356-372
- Benson JC, Eckert SP, McCleskey EW. 1999. Acid-Evoked Currents in Cardiac Sensory Neurons – A possible mediator of myocardial ischemic sensation, Circulation Research, 84:921-928. Full free paper at http://circres.ahajournals.org/cgi/content/full/84/8/921
- Gianni M et al. 2006. Apical ballooning syndrome or takotsubo cardiomyopathy: a systematic review, European Heart Journal, V27,N13: 1523-1529
- Akashi YJ et al. 2002. Reversible left ventricular dysfunction “takotsubo” cardiomyopathy related to catecholamine cardiotoxicity, J. Electrocardiol 2002; 35:351-356
- Arora S et al. 2006. Transient left ventricular apical ballooning after cocaine use; is catecholamine cardiotoxicity the pathologic link? Mayo Clin Proc. 2006; 81:820-832. Full free paper at http://www.mayoclinicproceedings.com/pdf/8106/8106cr2.pdf
- Wittstein IS et al. 2005. Neurohumoral features of myocardial stunning due to sudden emotional stress, New Engl J Med, Feb 10, V352: 539-548
- Graham LN, Smith PA et al. 2004. Sympathetic neural hyperactivity and its normalization following unstable angina and acute myocardial infarction, Clin Sci (Lond), Jun;106(6):605-11
- Gazes PC, Richardson JA et al. 1959. Plasma catecholamine concentrations in myocardial infarction and angina pectoris, Circulation 19:657-661
- Waldenstrom AP et al. 1978. A possible role of noradrenaline in the development of myocardial infarction, Am Heart J. 95:43-51
- Nadeau RA, de Champlain J. 1979. Plasma catecholamine in acute myocardial infarction, Am Heart J, 98: 548-554
- McCance AJ, Thompson PA, Forfar JC. 1993. Increased cardiac sympathetic nervous activity in patients with unstable coronary heart disease, Eur Heart J, Jun;14(6):751-7
- Makikalio A. 2005. Cardiovascular autonomic and hormonal dysregulation in ischemic stroke with an emphasis on survival, International Journal of Circumpolar Health 64:5
- Korner P. 2007. Essential Hypertension and Its Causes: Neural and Non-Neural Mechanisms. New York, Oxford University Press
- Rainforth MV, Schneider RH, Nidich SI, Gaylord-King C, Salerno JW, Anderson JW. 2007. Stress Reduction Programs in Patients with Elevated Blood Pressure: A Systematic Review and Metaanalysis. Curr Hypertens Rep Dec;9(6):520-8. Full free paper at: http://www.pubmedcentral.nih.gov/articlerender.fcgi?tool=pubmed&pubmedid=18350109
- Barnett PA, Spence JD, Manuck SB, et al. 1997. Psychological stress and the progression of carotid artery disease, J Hypertens 15:49–55
- Kamarck TW, Everson SA, Kaplan GA, et al. 1997. Exaggerated blood pressure responses during mental stress are associated with enhanced carotid atherosclerosis in middle-aged Finnish men: findings from the Kuopio ischemic heart disease study. Circulation,96:3842–8 Full free paper at: http://circ.ahajournals.org/cgi/content/full/96/11/384210
- Jennings JR, Kamarck TW et al. 2004. Exaggerated blood pressure responses during mental stress are associated with enhanced carotid atherosclerosis in middle-aged Finnish men: findings from the Kuopio ischemic heart disease study, Circulation;110:2198-2203. Full free paper at: http://circ.ahajournals.org/cgi/content/full/110/15/2198
- Hauss WH et al. 1990. Adrenaline and noradrenaline as possible chemical mediators in the pathogenesis of arteriosclerosis. Ann N Y Acad Sci 598:91-101
- Matthews KA et al. 1998. Stress-Induced Pulse Pressure Change predicts women’s carotid atherosclerosis, Stroke 29:1525-1530
- Matthews KA, Zhu S, Tucker DC, Whooley MA. 2006. Blood pressure reactivity to psychological stress and coronary calcification in the Coronary Artery Risk Development in Young Adults Study, Hypertension, Mar; 47(3):391-5. Full free paper at http://hyper.ahajournals.org/cgi/content/full/47/3/391
- Ghiadone L et al. 2000. Mental stress induces transient endothelial dysfunction in humans, Circulation102:2473. Full free paper at http://circ.ahajounals.org/cgi/content/full/102/20/2473
- Steptoe A. et al. 2006. Delayed blood pressure recovery after psychological stress is associated with carotid intima-media thickness. Arterioscler. Thromb. Vasc. Biol. Nov, 26(11):2547-51
- Eller NH, Netterstrom. 2007. Psychosocial factors at home and at work and four-years progression in intima-media thickness. In J Behav Med 2007; 14 (1):21-29
- Faramawi et al. 2007. Relation between depressive symptoms and common carotid artery atherosclerosis in American persons > 65 years of age, Am J Cardiol; 99:1610-1613
- Schoner W. 2002. Endogenous cardiac glycosides, a new class of steroid hormones. Eur J Biochem. 268, 2440-2448, Full free paper at http://www.ejbiochem.org/cgi/content/full/269/10/2440
- Nesher M, Shpolansky U, Rosen H, Lichtstein D. 2007.The digitalis-like steroid hormones: New mechanisms of action and biological significance. Life Sci. May 15;80(23):2093-107
- Sophocleus A et al. 2003. Circulating endogenous digitalis-like factors (EDLF) in man is derived from the adrenals and its secretion is ACTH-dependent. J Endocrinol Invest Jul;26(7):668-74
- Weidemann H et al. 2004. Diverse effects of stress and additional adrenocorticotropic hormone on digitalislike compounds in normal and nude mice, Journal of Neuroendocrinology, Vol 16, 458-463. Full free paper at http://physiology.huji.ac.il/pdf/lichtstein/weiden-et-al04.pdf
- Hassan M. AM Qazzaz et al. 2004. De Novo Biosynthesis and Radiolabeling of Mammalian Digitalis-Like Factors. Clin Chem. Mar;50(3):612-20. Full free paper at http://www.clinchem.org/cgi/content/full/50/3/612
- Rose AM, Valdes RJ. 1994. Understanding the sodium potassium pump and its relevance to disease, Clin. Chem. 40/9: 1674-1685 Full free paper at: http://www.clinchem.org/cgi/reprint/40/9/1674
- Vasilyev A, Khater K, and Rakowski RF. 2004. Effect of Extracellular pH on Presteady-State and SteadyState Current Mediated by the Na+/K+ Pump,. J Membr Biol. March 15; 198(2):65–76. Full free paper at: http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1357233
- Li C, Geering K, Horisberger JD. 2006. The Third Sodium Binding Site of Na,K-ATPase Is Functionally Linked to Acidic pH-Activated Inward Current. Membr Biol. 213(1):1-9.
- Mesquita QHde, Baptista CAS. 2002. Cardiotônico: insuperável na preservação da estabilidade miocárdica como preventivo das síndromes coronárias agudas e responsável pela prolongada sobrevida, Ars Cvrandi, maio 35:3. Full free paper at http://www.infarctcombat.org/28anos/digitalicos.html . Summary in English at: http://www.infarctcombat.org/heartnews-16.html
- Mesquita QHde, Baptista CAS et al. 2002. Efeitos do cardiotônico + dilatador coronário na coronáriomiocardiopatia crônica estável, com e sem enfarte prévio, a longo prazo. Ars Cvrandi, setembro;35:7. Full free paper at http://www.infarctcombat.org/qhm/cme.pdf Summary in English athttp://www.infarctcombat.org/heartnews-16.html
- Kern B. 1970. Der Myokard-Infarkt. Haug-Verlag, Heidelberg.
- Gao JRS et al. 2002. Isoform specific stimulation of cardiac Na/K pumps by nM concentrations of glycosides, J Gen Physiol 119:297-312. Full free paper at http://www.jgp.org/cgi/content/full/119/4/297
- Schobel HP et al. 1991.Contrasting effects of digitalis and dobutamine on baroreflex sympathetic control in normal humans, Circulation V84, 1118-1129. Full free paper at: http://circ.ahajournals.org/cgi/reprint/84/3/1118
- Gutman Y, Boonyaviroj P. Naunyn Schmiedebergs. 1977. Mechanism of inhibition of catecholamine release from adrenal medulla by diphenylhydantoin and by low concentration of ouabain (10 (-10) M). Arch Pharmacol Feb;296(3):293-6
- von Ardenne M. 1978. Die Hemmung der mikrozirculation beim myokardinfarkt und das perlingual applizierte g-strophanthin, Arzneimittel-Forsch. 28; 202:
- Pierre SV et al. 2007. Ouabain triggers preconditioning through activation of the NA+, K+-ATPase signalling cascade in rat hearts, Cardiovasc Res, Feb 1;73(3): 488-96
- Pugin J, Dunn-Siegrist I, Dufour J, Tissieres P, Charles PE, Comte R. 2007. Cyclic Stretch of Human Lung Cells Induces an Acidification and Promotes Bacterial Growth, Am J Respir Cell Mol Biol. Oct 5 doi:10.1165/rcmb.2007-0114OC
- Levy B, Gibot S, Franck P, Cravoisy A, Bollaert PE. 2005. Relation between muscle Na+K+ ATPase activity and raised lactate concentrations in septic shock: a prospective study. Lancet. Mar 5-11;365(9462):871-5.
- Schade DS.1982. The role of catecholamines in metabolic acidosis. Ciba Found Symp;87:235-53
- Abarquez RF Jr. 1967. Digitalis in the treatment of hypertension. A preliminary report. Acta Med Philipp. Jan-Mar;3(3):161-70
- Yuan CM, Manunta P, Hamlyn JM et al. 1993. Long-term ouabain administration produces hypertension in rats. Hypertension, 3;22;178-187 Full free paper at: http://hyper.ahajournals.org/cgi/reprint/22/2/178
- Manunta, P., Hamilton, J., Rogowski, A.C., Hamilton, B.P., Hamlyn, J.M. 2000. Chronic hypertension induced by ouabain but not digoxin in the rat:antihypertensive effect of digoxin and digitoxin. Hypertension Research 23 (Suppl), S77–S85.
- Yang Q, Huang W, Jozwik C, Lin Y, Glasman M et al. 2005. Cardiac glycosides inhibit TNF-alpha/NF-kappaB signaling by blocking recruitment of TNF receptor-associated death domain to the TNF receptor. Proc Natl Acad Sci USA Jul 5;102(27):9631-6. Full free paper at http://www.pnas.org/cgi/content/full/102/27/963111
- Sternberg EM. 2001. Neuroendocrine regulation of autoimmune/inflammatory disease, J Endocrinol Jun; 169(3):429-35. Full free paper at http://joe.endocrinology-journals.org/cgi/reprint/169/3/429
- Brum PC, Kosek J, Patterson A et al. 2002. Abnormal cardiac function associated with sympathetic nervous system hyperactivity in mice. Am J Physiol Heart Circ Physiol 283: H1838-H1845. Full free paper at http://ajpheart.physiology.org/cgi/content/full/283/5/H1838#B4
- F. E. Demartini, P. J. Cannon, W. B. Stason, and J. H. Laragh. 1965. Lactic Acid Metabolism in Hypertensive Patients. Science 11 June, Vol. 148. no. 3676, pp. 1482 – 1484
- Sharda S, Gupta SN and Khuteta KP. 1975. Effect on mental stress on intermediate carbohydrate-and lipidmetabolism. Indian J Physiol Pharmacol. Apr-Jun;19(2):86-9.
- Hall JB, Brown DA. 1979. Plasma glucose and lactic acid alterations in response to a stressful exam. Biol Psychol. May;8(3):179-88.
- von Ardenne M, Reitnauer PG. 1989. Increase of perfusion pressure at constant perfusion rate caused by low pH values, Biomed Biochim Acta, 48(4):317-23
- Yasushi Horai et al. 2005. Changes in pH increase perfusion pressure of coronary arteries in the rat. J Pharmacol Sci 97; 400: 407
- Austin C, Wray S. 2000. Interactions Between Ca2+ and H+ and Functional Consequences in Vascular Smooth Muscle, Mini Review, Circulation Research 86:355. Full free paper at: http://circres.ahajournals.org/cgi/content/full/86/3/355
- Kim YM et al. 2005. Contribution of Na_-K_ pump and KIR currents to extracellular pH-dependent changes of contractility in rat superior mesenteric artery, Am J Physiol Heart Circ Physiol 289:792-800 Full free paper at http://ajpheart.physiology.org/cgi/reprint/289/2/H792
- Carter G, Gavin JB. 1989. Endocardial damage induced by lactate, lowered pH and lactic acid in nonischemic beating hearts. Pathology Apr;21(2):125-30
- Sharma AM, Kribben A et al. 1990. Salt sensitivity in humans is associated with abnormal acid-base balance. Hypertension; 16, 407-413. Full free paper at http://hyper.ahajournals.org/cgi/reprint/16/4/407
- Harold T. Edwards, Edward H. Bensley, David B. Dill and Thorne M. Carpenter. 1944. Human Respiratory Quotients in Relation to Alveolar Carbon Dioxide and Blood Lactic Acid After Ingestion of Glucose, Fructose, or Galactose. Journal of Nutrition Vol. 27 No. 3 March, pp. 241-251. Full free paper at http://jn.nutrition.org/cgi/reprint/27/3/241
- Hallfrisch J. 1990. Metabolic effects of dietary fructose. FASEB J, Vol 4; Jun: 2652-2660. Full free paper at http://www.fasebj.org/cgi/reprint/4/9/2652.pdf
- Mesquita QHde. 1982. Aspectos angiográficos coronários e ventriculograficos do primeiro enfarte do miocárdio em coronariopatia crônica silenciosa. Rev. Bras. Med., V 39: N7
- LA Naves and McCleskey EW. 2005. An acid-sensing ion channel that detects ischemic pain. Braz J Med Biol Res, 38 (11) 1561-69 http://www.scielo.br/pdf/bjmbr/v38n11/v38n11a01.pdf
- Vogt AM, Ackermann C, Yildiz M, Schoels W, Kübler W. 2002. Lactate accumulation rather than ATP depletion predicts ischemic myocardial necrosis: implications for the development of lethal myocardial injury, Biochim Biophys Acta Mar 16;1586(2):219- 26.
- Todd GL, Baroldi G, Pieper GM, Clayton FC, Eliot RS. 1985. Experimental catecholamine- induced myocardial necrosis. I. Morphology, quantification and regional distribution of acute contraction band lesions. J Mol Cell Cardiol. Apr 17(4):317- 38.
- Henning RJ, Well MH, Weiner F. 1982. Blood lactate as prognostic indicator of survival in patients with acute myocardial infarction. Circ Shock, 9(3):307-15
- Vikhert AM, Cherpachenko NM. 1985. Histoenzymological characteristics of the myocardium in sudden cardiac death. Arkh Patol 47(7):29-34
- Huang Y, McNamara JO. 2004. “Ischemic Stroke: “Acidotoxicity” Is a Perpetrator”, Cell, Volume 118, Issue 6, 17 September, Pages 665-666Tennant R. 1935. Factors concerned in the arrest of contraction in an ischemic myocardial area. Am J Physiol: 133; 677-682
- Katz AM, Hecht H. H. 1969. The early pump failure of the ischaemic heart. Am J Med: 47; 497-502
- Elharrer V, Zipes D.P. 1977. Cardiac electrophysiologic alterations during myocardial ischaemia. Am J Physiol: 233: H329-345
- Pan HL et al. 1999. Role of protons in activation of cardiac sympathetic C-fibre afferents during ischaemia in cats. J Physiol. Aug 1;518 ( Pt 3):857-66. Full free paper at http://jp.physoc.org/cgi/content/full/518/3/857
- Leake DS. 1997. Does an acidic pH explain why low density lipoprotein is oxidised in atherosclerotic lesions? Atherosclerosis. Mar 21;129(2):149- 57
- Gown MA, Benditt PE. 1982. Lactate dehydrogenase (LDH) isozymes of human atherosclerotic plaques. Am J Pathol 1982, 107:316-321
- Morgan J, Leake DS. 1995. Oxidation of low density lipoprotein by iron or copper at acidic pH. J Lipid Res. Dec;36(12):2504- 12. Full free paper at: http://www.jlr.org/cgi/reprint/36/12/2504
- Patterson RA, Leake DS. 1998. Human serum, cysteine and histidine inhibit the oxidation of low density lipoprotein less at acidic pH. FEBS Lett. Sep 4;434(3):317- 21.
- Naghavi M et al. 2002. pH Heterogeneity of human and rabbit atherosclerotic plaques; a new insight into detection of vulnerable plaque. Atherosclerosis Sep, V 164; 1:27-35
- Khan T, Soller B, Naghavi M, Casscells W. 2005. Tissue pH determination for the detection of metabolically active inflamed vulnerable plaques using near-infrared spectroscopy: an in-vitro feasibility study. Cardiology.; 103(1): 10-6.
- Sneck M, Kovanen PT, Oorni K. 2005. Decrease in pH strongly enhances binding of native, proteolysed, lipolysed, and oxidized low density lipoprotein particles to human aortic proteoglycans, Journal of Biological Chemistry, 280;45: Nov. Full free paper at http://www.jbc.org/cgi/reprint/280/45/37449
- Oorni K and Kovanen PT. 2006. Enhanced extracellular lipid accumulation in acidic environments. Curr Opin Lipidol 17(5);534-40: Oct
- Patterson RA, Horsley ETM, Leake DS. 2003. Prooxidant and antioxidant properties of human serum ultrafiltrates toward LDL: important role of uric acid. Journal of Lipid Research, Vol. 44, 512-521, March Full free paper at http://www.jlr.org/cgi/reprint/44/3/51212
- Hayden MR, Tyagi SC. 2004. Uric acid: A new look at an old risk marker for cardiovascular disease, metabolic syndrome, and type 2 diabetes mellitus: The urate redox shuttle. Nutr Metab (Lond). 1: 10, October 19. Full paper at http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=529248
- McCully KS. 1969. Vascular pathology of homocysteinemia: implications for the pathogenesis of atherosclerosis. Am J Pathology 56:111:28
- Stoney CM. 1999. Plasma homocysteine levels increase in women during psychological stress, Life Sci 64(25):2359-65
- Stoney CM and Engebretson TO. 2000. Plasma homocysteine concentrations are positively associated with hostility and anger, Life Sci 66(23):2267-75
- Hapuarachchi JR, Chalmers AH et al. 2003. Changes in clinically relevant metabolites with psychological stress parameters. Behav Med. Summer;29(2):52-9
- Jerlich A et al. 1999. Correlation of low-density lipoprotein modification by myeloperoxidase with hypocholorous acid formation, Int. J. Clin, Lab, Res 29(4):155-61
- Podrez EA, Abu-Soud HM, Hasen SL. 2000. Myeloperoxidase-generated oxidants and atherosclerosis. Free Radic Biol Med 28:1717–172
- Yang J, Cheng Y, Ji R, Zhang C. 2006. Novel model of inflammatory neointima formation reveals a potential role of myeloperoxidase in neointimal hyperplasia. Am J Physiol Heart Circ Physiol. Dec;291(6):H3087- 93.
- Meuwese MC, Stroes ESG, Hazen SL, et al. 2007. Serum myeloperoxidase levels are associated with the future risk of coronary artery disease in apparently healthy individuals: The EPIC-Norfolk Prospective Population Study..J Am Coll Cardiol 50:159-165
- Wong ML et al. 2000. Acute systemic inflammation up-regulates secretory sphingomyelinase in vivo: A possible link between inflammatory cytokines and atherogenesis, PNAS 97;8681-8686 Full free paper at http://www.pnas.org/cgi/content/full/97/15/8681
- Abela GS. 2006. Plaque Rupture by Cholesterol Crystallization – A Novel Concept for Acute Coronary Syndrome, American College of Cardiology Annual Scientific Session, March 13, Full free paper at http://www.cardiosource.com/rapidnewssummaries/summary.asp?SumID=164
- Malek AM, Alper SL, Izumo S. 1999. Hemodynamic shear stress and its role in atherosclerosis JAMA 282: 2035-2042
- Cheng C et al. 2006. Atherosclerotic lesion size and vulnerability are determined by patterns of fluid shear stress. Circulation 113:2744-2753. Full free paper at at: http://circ.ahajournals.org/cgi/content/abstract/113/23/2744
- Cunningham KS and Gotlieb AI. 2005. The role of shear stress in the pathogenesis of atherosclerosis (Mini review), Laboratory Investigation 85, 9-23, Full free paper at: http://www.nature.com/labinvest/journal/v85/n1/full/3700215a.html
- Texon M. 1957. A hemodynamic concept of atherosclerosis, with particular reference to coronary occlusion. Arch Intern Med 99:418–42
- Imparato AM, Lord JW Jr, Texon M, Helpern M. 1961. Experimental atherosclerosis produced by alteration of blood vessel configuration. Surg Forum 12:245–247.
- Rittersma SZH, van der Wal AC, Koch KT, et al. 2005. Plaque instability frequently occurs days or weeks before occlusive coronary thrombosis. A pathological thrombectomy study in primary percutaneous coronary intervention. Circulation; 111:1160-1165. Full free paper at: http://circ.ahajournals.org/cgi/content/full/111/9/1160
- Ojio S, Takatsy H, et al. 2000. Considerable time from the onset of plaque rupture and/or thrombi until the onset of acute myocardial infarction in humans coronary angiographic findings within 1 week before the onset of infarction. Circulation;102:2063. Full free paper at:http://www.circ.ahajournals.org/cgi/reprint/102/17/206
- Ulrich E. Heidlan, Bodo E. Strauer. 2001. Left ventricular muscle mass and elevated heart rate are associated with coronary plaque disruption, Circulation 104:1477. Full free paper at: http://circ.ahajournals.org/cgi/content/full/104/13/1477
- Baroldi G, Bigi R, Cortigiani L. 2004. Ultrasound imaging versus morphopathology in cardiovascular diseases. Coronary collateral circulation and atherosclerotic plaque. Cardiovascular ultrasound; 3: 6. Full free paper at: http://www.cardiovascularultrasound.com/content/3/1/6
- Roberts W. C. 1974. Coronary Thrombosis and Fatal Myocardial Ischemia. Circulation;49;1-3 Full free paper at http://circ.ahajournals.org/cgi/reprint/49/1/1.pdf
- Rioufol G, Finet G, Andre-Fouet X et al. 2002. Multiple atherosclerotic plaque rupture in acute coronary syndrome: a three-vessel intravascular ultrasound study. Circulation; 106:804-808. Full free paper at: http://www.circ.ahajournals.org/cgi/reprint/01.CIR.0000025609.13806.31v1
- Yasunori Ueda, Masanori Asakura, et al. 2001. The healing process of infarct-related plaque: Insights from 18 months of serial angioscopic follow-up. Am Coll Cardiol, 38:1916-1922.Full free paper at: http://content.onlinejacc.org/cgi/reprint/38/7/1916
- Giorgio Baroldi, Riccardo Bigi and Lauro Cortigiani.2005. Ultrasound imaging versusmorphopathology in cardiovascular diseases. Myocardial cell damage. Cardiovascular Ultrasound 3:32. Full free paper at http://www.cardiovascularultrasound.com/content/3/1/32
- Murakami T, Mizuno S, Takahashi Y, Ohsato K et al. 1998. Intracoronary aspiration thrombectomy for acute myocardial infarction, Am. J Cardiology Oct 1;82 (7):839-44
- Wang HX, Leineweber C, et al. 2007. Psychosocial stress and atherosclerosis: family and work stress accelerate progression of coronary disease in women. The Stockholm Female Coronary Angiography Study. Journal of Internal Medicine 261;245-254
- Richmond AC et al. 2000. Effects of stress reduction on carotid atherosclerosis in hypertensive African Americans, Stroke 31:568-573. Full free paper at: http://stroke.ahajournals.org/cgi/reprint/31/3/568
- Fields JZ et al. 2002. Effect of a multimodality natural medicine program on carotid atherosclerosis in older subjects: a pilot trial of Maharishi Vedic Medicine, American Journal of Cardiology, 89; 8:952-958
- Manchanda SC, Narang R, Reddy KS, Sachdeva U, Prabhakaran D, Dharmanand S, Rajani M and Bijlani R. 2002. Retardation of coronary atherosclerosis with yoga lifestyle prevention, J Assoc Physicians India Jul; 48(7): 687-94 13.
- Rainer Rauramaa et al. 2004. Effects of aerobical physical exercise on inflammation and atherosclerosis in men: The DNASCO Study. Annals of Internal Medicine, 15 June, 140:12:1007-1014,
- Lichtor T et al. 1987.The sympathetic nervous system and atherosclerosis. J Neurosurg Dec;67(6):906-1,Pauletto P et al. 1991. Sympathetic drive and vascular damage in hypertension and atherosclerosis, Hypertension Apr;17(4 Suppl):III75-81.
- Wikstrand J, Berglund G, Hedblad B, Hulthe, Wikstrand J. 2003.Anti-atherosclerotic effects of beta-blockers. Am J Cardiol. Jun 19;91(12A):25H-29H.
- Sipahi I et al. 2007. B-Blockers and progression of coronary atherosclerosis; Pooled analysis of 4 intravascular trials. Annals of Internal Medicine, 3 July, V147; Issue 1: 10-18
- Mesquita QHde, Kerbrie SV, Mari SM, Baptista CA, Monteiro J, Maciel MC. 1978. Preservação funcional do miocárdio isquêmico pelo cardiotonico a longo prazo: recateterização de 29 casos. Medicina de Hoje, março 1978
- Hansson GK. 2005. Inflammation, atherosclerosis and coronary artery disease, NEJM V 352; N16 April 21.
- Malcolm Kendrick. 2007. Are statins overused? Future Lipidol, 2 (5)
- Player MS, King DE, et al. 2007. Psychosocial Factors and Progression From Prehypertension to Hypertension or Coronary Heart Disease, Ann Fam Med ;5(5):403-411. Full free paper at http://www.medscape.com/viewarticle/565806?src=mp.
- Palatini P, Longo D, Zaetta V, Perkovic D, Garbelotto R, Pessina AC. 2006. Evolution of blood pressure and cholesterol in stage 1 hypertension: role of autonomic nervous system activity, J Hypertens.Jul;24(7):1375-81.
- Grassi G, Quarti-Trevano F, Seravalle G, Dell’Oro R. 2007. Cardiovascular risk and adrenergic overdrive in the metabolic syndrome. Nutr Metab Cardiovasc Dis Jul; 17(6): 473-81.
- Choi CS, Kiim YB, Lee FN, et al. 2002. Lactate induces insulin resistance in skeletal muscle by suppressing glycolysis and impairing insulin signaling. Am J Physiol Endocrinol Metab 283: E233–E240, 2002. Full free paper at: http://ajpendo.physiology.org/cgi/content/full/283/2/E233,
- Tentolouris N, Tsigos C, Perea D et al. 2003. Differential effects of high-fat and high-carbohydrate isoenergetic meals on cardiac autonomic nervous system activity in lean and obese women. Metabolism. Nov;52(11):1426- 32.
- Calynn Davis Bunol, 2005. Thesis, Autonomic nervous system modulation of the heart following a high carbohydrate liquid meal, December. Full free paper at http://etd.lsu.edu/docs/available/etd-09082005-165133/unrestricted/Bunol_thesis.pdf
- Erkilla AT, Matthan NR, et al. 2006. Higher plasma docosahexaenoic acid is associated with reduced progression of coronary atherosclerosis in women with CAD. J Lipid Res; 47: 2814-19 Full free paper at http://www.jlr.org/cgi/reprint/47/12/2814.
- Ogilve GK, Fettman MJ et al. 2000. Effect of fish oil, arginine, and doxorubicin chemotherapy on remission and survival time for dogs with lymphoma: A double-blind, randomized placebo-controlled study, Cancer; 88: 1016-28. Full free paper at: http://www3.interscience.wiley.com/cgibin/fulltext/75504731/PDFSTART
- Graziani Y. 1977. Regulation of cyclic AMP level and lactic acid production in Ehrlich ascites tumor cells. Biochim Biophys Acta April 27;497(2):499-506.
- Nazam Ansari N, Bhandari U, Pillai KK. 2007. Protective role of curcumin in myocardial oxidative damage induced by isoproterenol in rats. Hum Exp Toxicol, Dec;26(12):933-8.
- Dernek S et al. 2004. Cardioprotection with resveratrol pretreatment: improved beneficial effects over standard treatment in heart rats after global ischemia. Scand Cardiovasc J Aug;38(4):245-54.
- Al Makdessi S, Sweidan H, Müllner S, Jacob R. 1996. Myocardial protection by pretreatment with Crataegus oxyacantha: an assessment by means of the release of lactate dehydrogenase by the ischemic and reperfused Langendorff heart. Arzneimittelforschung Jan;46(1):25-7.
- Leor J, Goldbourt U et al. 1995. Digoxin and increased mortality among patients recovering from acute myocardial infarction: importance of digoxin dose, Cardiovasc Drugs Ther. Oct;9(5):723-
- Adams KF Jr, Patterson JH et al. 2005. Relationship of serum digoxin concentration to mortality and morbidity in women in the digitalis investigation group trial: a retrospective analysis. J Am Coll Cardiol. Aug 2;46(3):497-504.
- Wycoff C.C. 1969. New Concepts of Digitalis, Calif Med. 1969 December; 111(6): 423–432. Full free paper at http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=1503737.
- T Bjornheden, M Levin, M Evaldsson, O Wiklund. 1999. Evidence of hypoxic areas within the arterial wall in vivo, Arteriosclerosis, Thrombosis and Vascular Biology; 19:870-87.
- Quick AJ. 1935. The effect of exercise on the excretion of uric acid. The Journal of Biological Chemistry. Full free paper at: http://www.jbc.org/cgi/reprint/110/1/107.pdf
- Flierl MA, Rittirsch D, Nadeau BA et al. 2007. Phagocyte-derived catecholamines enhance acute inflammatory injury. Nature Oct 11;449 (7163):721-5
- Lee KS and Klaus W. 1971. The subcellular basis for the mechanisms of inotropic action of cardiac glycosides. Pharmacol Rev 23:193-261
- Wen Y, Leake DS. 2007. Low density Lipoprotein oxidation undergoes within lysosome in cells. Circ.Res. 100;1337-1343. Full free paper at http://circres.ahajournals.org/cgi/content/full/100/9/1337
- Marshall MW and Iacono JM (1976). Changes in lactate dehydrogenase, LDH isoenzymes, lactate, and pyruvate as a result of feeding low fat diets to healthy men and women. Metabolism. 1976 Feb;25(2):169-78.
- Yoshimura T, Miyoshi T, et al. (1986). Effect of high carbohydrate diet on serum lactate
- dehydrogenase isozyme pattern in Japanese young men. Acta Biol Hung. 1986;37(3-4):243-8.
The author of the best-selling pH Miracle books, already bankrupted and criminally convicted of “practicing medicine without a license,” was slammed with a $105 million judgement last month following a civil trial in a case in which a patient alleged negligence and fraud.
The plaintiff is Dawn Kali, a patient who agreed to use his medical protocol. She then sued Robert O. Young Ph.D., claiming he held himself out as a doctor and counseled her to forego traditional cancer treatment and instead use his treatments based on his “alkaline theories.” The reward was more than double what the woman sought. The trial lasted seven days and the jury deliberated less than half a day.
The 45-year-old woman’s oncologist says she has only three or four years to live and has stage four cancer. Kali had previously worked for Young and knew his treatments were outside the practices of orthodox medicine.
I have read Young’s books. I wrote about The pH Miracle for the readers of my Bob Livingston Letter™ (subscription required) back in April 2012. His theories are sound and based on science, according to my years of research.
It’s very simple. A buildup of acid in our bodies leads to death. Our standard American diet promotes acidity with its processed foods, sugars and preservatives.
Cooked and processed foods — the foods that make up the diets of most Americans — are mostly acid. Meat is always acid-forming. There are no alkaline-forming meats. Acid (low pH) causes fermentation and disease. That is why there is such a culture of disease in this country.
Acidity is fertilizer to cell mutation and malignancy. Body acidity interferes with normal cell death or apoptosis. This is the basis of normal cells mutating into malignancy. At all times the healthy body requires an excess of alkaline reserves to support the body skeleton, and equally to keep the critical 1 percent alkaline balance in the blood and the cell tissue. This is the absolute best guarantee against disease. Our glands and organs function properly in exact proportion to the balance of alkaline and acid balance.
It’s possible Young got out over his skis and promised Kali he would “cure” her cancer, though I would guess he’s smarter than that. But if he offered it as a treatment option he did nothing more or less than what mainstream doctors do. So his “crime,” “fraud” and “negligence” was not that his treatments failed. After all, mainstream medicine doesn’t cure disease, it just treats symptoms and covers them over with chemicals.
Young’s “crime,” “fraud” and “negligence” was simply that he was not part of the establishment.
Cancer is serious business. When people hear the “C” word come out of their doctor’s mouth they believe their death warrant has been signed.
It’s also serious business for the medical-industrial complex, which makes big money off cancer treatments and cancer drugs. Oncology is a most damnable plot against cancer patients. The cash register rings for every new cancer patient. Insurance companies contribute to the pain, suffering and death.
In 2014 Americans spent $32.6 billion on oncology drugs and another $11.1 billion supportive care treatments. New cancer drugs cost anywhere from $10,000 to $30,000 per month.
Kali complained that Young was charging her $500 per treatment that consisted of giving intravenous fluids with baking soda. A baking soda treatment was devised and long used by Italian doctor Tullio Simoncini (who has also been stripped of his medical license).
People undergoing cancer treatments die every day. But state-sponsored, officially-approved death is OK. No one thinks twice if someone dies after chemotherapy. And chemotherapy doesn’t cure anyone. In fact, chemotherapy is a catalyst for cancer cell growth, as a study published in Nature Medicine found.
In his book Questioning Chemotherapy, leading author and consultant on cancer treatment Ralph Moss, Ph.D., explains chemotherapy to be an effective treatment option in only 2 percent to 4 percent of cancers: Hodgkin lymphoma, acute lymphocytic leukemia, testicular cancer and choriocarcinoma.
A report funded by the U.S. National Institutes of Health and published in the journal Proceedings of the National Academy of Sciences (PNAS) shows that cancer-fighting drugs actually induce major stem cell damage that causes more cancer. An abstract of the report can be seen here.
The upshot is that even though the chemical cocktails used to treat tumors are sometimes initially effective at stemming more tumor growth and the spread of more tumor cells, the damage they leave behind causes more tumors to develop on stem cells. And given the right environment and genetic background, those tumors could become malignant.
Cutting, burning and poisoning (surgery, radiation and chemotherapy) do nothing to cure cancer, nor can they reverse the underlying mechanisms that triggered or promoted its growth in the first place. Moreover, cancer will likely return somewhere else in the body if conventional methods are your only treatment strategy.
It sounds backward to suggest that cancer treatment is deadlier than cancer. And saying so is certain to earn you scornful looks from the vast majority of people. But what the medical establishment doesn’t reveal is that cancer drugs like chemotherapy are the direct cause of death of thousands of cancer patients each year. Most cancer patients who die don’t die from cancer. They die with cancer.
No doubt, the powerfully toxic drugs can kill cancer cells. But they also don’t discriminate and wreak havoc on healthy cells as well.
And when too many healthy cells are destroyed, so too are the people they make up.
Now back to body pH and maintaining your health; after all, it’s better to keep your body healthy and avoid cancer than to try and treat it once it’s taken hold, right? High pH or about 7.25 is alkaline and increases the oxygen content of the blood and tissue. Cancer cells are anaerobic. If cancer cells get enough oxygen they die, hence the theory behind Young’s protocol.
According to a study conducted by the Arizona Respiratory Center at the University of Arizona and published in the U.S. National Library of Medicine National Institutes of Health’s PubMed.gov, “Acidogenic diets, which are typically high in animal protein and salt and low in fruits and vegetables, can lead to a sub-clinical or low-grade state of metabolic acidosis.” This leads to an increase in the risk of cancer, elevated levels of insulin and a host of diseases.
But you can naturally alter your pH by choosing your foods wisely. The following foods are alkaline-forming and can help your body achieve the proper pH:
- Cayenne pepper
- Lemon water
- Leafy greens
In addition to helping your pH balance, they also contain a host of necessary nutrients, antioxidants and other health benefits.
P.S.: I’ll have more to say about the benefits of keeping your body in an alkaline state in the January issue of The Bob Livingston Letter. You can subscribe here.
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It’s also serious business for the medical-industrial complex, which makes big money off cancer treatments and cancer drugs. Oncology is a most damnable plot against cancer patients. The cash register rings for every new cancer patient. Insurance companies contribute to the pain, suffering and death.
In 2014 Americans spent $32.6 billion on oncology drugs and another $11.1 billion supportive care treatments. New cancer drugs cost anywhere from $10,000 to $30,000 per month.
Kali complained that Young was charging her $500 per treatment that consisted of giving intravenous fluids with baking soda. A baking soda treatment was devised and long used by Italian doctor Tullio Simoncini (who has also been stripped of his medical license).
People undergoing cancer treatments die every day. But state-sponsored, officially-approved death is OK. No one thinks twice if someone dies after chemotherapy. And chemotherapy doesn’t cure anyone. In fact, chemotherapy is a catalyst for cancer cell growth, as a study published in Nature Medicine found.” The Bob Livingston Letter
PS Dawn Kali did not pay for any treatments from Dr. Young. In fact she received over $200,000 i free care because she had no money! Dawn Kali brings true meeting to the words, “No good deed goes unpunished!” During the time from 2010 to April/May of 2013 Dr. Young gave Dawn Kali over $450,000 in services and money.
Why would Dawn do such a thing? Follow the money! It will always lead you back to the real criminals in this case.